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KayleighLara

NervousSystemLabReport

Thislabreportisaboutactionpotentialandtheelectricactivityinthenormalcellmembrane
environment,environmentsmodifiedbyvarioustoxins,andactionpotentialsobservedinhuman
reflexes.Inresponsetoastimulus,theexcitablecellmembraneofaneuroncellgoesfroma
restingstatetodepolarization,followedbyrepolarizationtothatrestingstateagain.Thisprocess
iscalledactionpotential.Depolarizationoccurswithinaneuronwheresodiumionsenterthecell
andraisethechargefromanegativetoapositivelevelreversingthestateoftheresting
potential.Repolarizationoccurswhenthemembraneofaneuronbecomesmorepermeable,
whichallowspotassiumionstodiffuseoutoftheneuroncausingthemembranetoreturntothe
restingstate.Afterrepolarization,theperiodwhenaxonscantbestimulatedtogenerateanew
actionpotentialiscalledtherefractoryperiodwhichlimitsimpulsestoanaverageof100per
second.Thefollowingfigureswilladdressactionpotentialandtheactionpotentialdatacollected
fromspecificcranialandperipheralnerves.

Figure1
:Thislinegraphshowshowactionpotentialiscausedbydifferentamountsofvoltage
andatwhattimeintervalsdepolarizationandrepolarizationintheneuronbeginsandends.

Figure2
:Thistoxingraphpresentsthedifferenttoxinsincorrelationwiththeirnegativeeffects
onaneuronthatenablesitfromgoingthroughthenormalstagesofactionpotential.

Figure3
:Thischartandimagecontains
thetwelvemajorcranialnervesonasheep
brainand
theirfunctionsthat,throughaction
potential,theyareabletoprovidetothe
body.


Exampleofthereflexarc

Figure4:
The
pictures
above
(takenby
author)
showhow
astimulus(
reflex
mallet)
affectsa
receptor
andcauses
sensory/afferentneuronstobesenttothespinalcord.Oncesenttothespinalcordingoes
throughtheinterneuronwhichprocessestheneuron'sinformationandsendsittothemotor/
efferentneuron(musculocutaneous).Thisprocessiscarriedoutbyactionpotentialwhich
increaseasthemusclestretchesandthedecreasesasthemusclecontracts.

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