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OBJECTIVES
P wave
P axis in range 0 to +90
P waves upright in I, II & aVF
P wave duration 0.06s +/- 0.02s in children
Max P duration 0.1s in children & 0.08s in
infants.
E.g if P axis is in range of +90 to + 180 what
would u suspect in a normal healthy child?
P wave
P wave + physiologic delay in AV node
(PQ segment)
Varies with age & HR.
Age increases, HR decreases & PR interval
increases in duration
With the exception the PR interval is
longer in duration at Birth than at infants
period
PR Interval
PR Interval
1 yr 70-150
5 yr 80-160
10 yr 90-170
Ventricle Dominance
Fetal heart pumps blood to high resistance
pulmonary circuit, so RV pressure high
After birth:
Pulmonary vascular resistance falls
RV muscularity recedes
RV contribution to ECG diminishes
Systemic vascular resistance changes:
increased LV size until > than RV (1 month)
6 months: RV/LV ratio similar to adults
Shift from newborn RV dominance to LV
dominance by 1 yr
RV dominance: R wave is larger than S wave
in V1
Heart Changes
LV/RV Weight Ratio
U wave
Long QT syndrome in 3 yr old
ABNORMAL
PAEDIATRIC ECGs
Ventricular Hypertrophy
Voltage Criteria: Depend on age adjusted
values for R and S wave amplitudes
R wave (S wave) R wave (S wave)
amplitude (mm) amplitude (mm)
AGE V1 V6
Birth 5-26 (1-23) 0-12 (0-10)
6m 3-20 (1-17) 6-22 (0-10)
1 yr 2-20 (1-20) 6-23 (0-7)
5 yr 1-16 (2-22) 8-25 (0-5)
10 yr 1-12 (3-25) 9-26 (0-4)
RVH
RV systemic
ventricle: RVH
RAD
Dominant R in R
precordial leads
Case: 6 m old with Cyanotic Episodes:
ToF and RVH
Tall R in
V1,
reciprocal
S in V6
qR in V3R
and V4R
RAD 120*
Upright T
V1-V3
(should be
inverted)
LVH
Useful ECG Features
Deep Qs in L precordial leads
Lateral ST depression and T wave inversion
Some Congenital Heart
Defects and ECG
Manifestations
Anomalous L coronary Aortic Stenosis
artery LVH
Anterolat MI
Anomalous Coarctation
pulm
venous return < 6m: RBBB or RVH
Total: RAD, RVH, RAH > 6m: LVH, N, RBBB
Partial RVH or RBBB Patentductus
arteriosus
Small shunt: N
Mod: LVH, +/- LAH
Large: CVH, LAH
Some Congenital Heart
Defects and ECG
Manifestations
Persistent truncus Transposition
arteriosus Intact septum: RVH,
LVH or CVH RAH
Pulmatresia (and VSD and/or PS: CVH,
hypoplastic RV) RAH, or CAH
LVH Corrected
Tetralogy of Fallot transposition
RAD, RVH, +/- RAH AV blocks, WPW, LAH
or CAH, absent Q in
V5/V6, and qR in V1
ABNORMALITIES OF
RATE AND RHYTHM
Abnormal HR
SIMILARITIES DIFFERENCES
Conduction Kids:fast HR that
pathways
same, so waveforms slows with age,
(P, QRS, T) same, and shorter N intervals
that prolong with
waveform timing age, and diminution
measured the same of RV dominance
(i.e., PR, QRS, QT
interval) Sinusbradycardia,
sinus arrhythmia
Identical
approach to and SVT most
ECG analysis common
arrhythmias in kids
Findings that may be N
HR > 100 bpm
Right precordial T wave inversion
Dominant R precordial R waves
Short PR and QT intervals
Short P wave and short QRS duration
Inferior and lateral Q waves
REFERENCES
ABC of clinical electrocardiograpy. Paediatric
electrocardiography. Goodacre S, McLeod K. BMJ Volume
324. June 8, 2002. Pgs 1382-1385
ECG INTERPRETATION: WHAT IS DIFFERENT IN CHILDREN?
Mowery, Bernice, Suddaby, Elizabeth C., Pediatric Nursing,
0097-9805, May 1, 2001, Vol. 27, Issue 3.
How to interpret Paediatric ECG by Gunneroth