You are on page 1of 2

See discussions, stats, and author profiles for this publication at: https://www.researchgate.

net/publication/273577854

Crack Lung: Cocaine induced lung injury

Article  in  QJM: monthly journal of the Association of Physicians · March 2015


DOI: 10.1093/qjmed/hcv064 · Source: PubMed

CITATIONS READS

3 245

4 authors:

Rushikesh Shah Arpan Patel


Emory University State University of New York Upstate Medical University
92 PUBLICATIONS   82 CITATIONS    10 PUBLICATIONS   16 CITATIONS   

SEE PROFILE SEE PROFILE

Omar Mousa Divey Manocha


State University of New York Upstate Medical University State University of New York Upstate Medical University
124 PUBLICATIONS   117 CITATIONS    47 PUBLICATIONS   90 CITATIONS   

SEE PROFILE SEE PROFILE

Some of the authors of this publication are also working on these related projects:

Effects of 2010 ACGME Regulations on Internal Medicine Residency Programs in the Northeast. View project

Volumetric Laser Endomicroscopy in the Biliary and Pancreatic Ducts: a Feasibility Study with Histological Correlation View project

All content following this page was uploaded by Rushikesh Shah on 02 September 2015.

The user has requested enhancement of the downloaded file.


QJM Advance Access published April 13, 2015

QJM: An International Journal of Medicine, 2015, 1

doi: 10.1093/qjmed/hcv064
Advance Access Publication Date: 14 March 2015
Clinical Picture

CLINICAL PICTURE

Crack lung: cocaine-induced lung injury


A 31-year-old female presented with acute onset shortness of cocaine. The management is supportive with maintaining oxy-
breath and chest pain. She had a past medical history of co- genation with O2 supplementation. Cocaine-induced pulmon-
caine abuse and agreed that she used cocaine 2 days prior to ary embolism although rare has been reported previously.
presentation. On admission, her vitals included Blood Pressure Underlying mechanism is believed to be stasis in pulmonary
of 160/70, Heart Rate—110, Respiratory Rate—36 with oxygen circulation leading to in situ thrombosis.
saturation of 78%. Chest X-ray showed some interstitial opac-
ities. CT scan of the chest was performed, which revealed dif-
Photographs and text from: R. Shah, SUNY Upstate Medical
fuse ground glass opacity consistent with cocaine-induced
University, 50 Presidential Plaza, Syracuse, NY 13202, USA;
lung injury—crack lung (Figure 1A) as well as cocaine-induced
A. Patel, SUNY Upstate Medical University, 153 Summerheaven
in situ thrombosis (Figure 1B). She was treated with anticoagu-
drive, East Syracuse, NY 13057, USA; O. Mousa and D. Manocha,
lation and supportive measures including oxygen. She was fi-
SUNY Upstate Medical University, 750 E Adams Street, Syracuse,
nally discharged home on Coumadin as well as continues

Downloaded from by guest on August 22, 2015


NY 13210, USA. email: rushishah.17@gmail.com.
home oxygen. Crack lung occurs due to diffuse alveolar dam-
age and alveolar hemorrhage that occurs 48 h after smoking Conflict of Interest: None declared.

Figure 1. A. Cocaine-induced diffuse alveolar hemorrhage. B. Cocaine-induced in situ lung thrombosis (shown by arrow).

C The Author 2015. Published by Oxford University Press on behalf of the Association of Physicians.
V
All rights reserved. For Permissions, please email: journals.permissions@oup.com

View publication stats

You might also like