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2. how is the comparison between Previous studies found that spasticity played a major role in the √
pathogenesis of HSP,9-11 which could be improved by electrical
the study result and other
stimulation.50 In this trial, we did not observe improvements in the
studies? spasticity of shoulder adductors and internal rotators after treatment. Although the
MAS is not adequate for the evaluation of
spasticity, it was unlikely that NMES or TENS relieved pain by
reducing spasticity; this phenomenon was inconsistent with the
results of previous studies
3. how is the biological plausibility Effects of low-frequency electrical stimulation for √
reducing HSP lie in decreasing the excitability of nerve C fibers,40
of study result?
and antidromic conduction stimulation of Ab fibers in the dorsal
columns in the stimulated segment.41 In addition, they seem to
modify the release of g-aminobutyric acid, calcitonin gene-related
peptide, substance P, adrenaline, serotonin, and alanine.
4. can the results of the study be
generalized?
5. how is the clinical significance These results are in accordance with our findings. Overall, the 8-week intervention √
of the study result? did not result in any significant difference in the AROM/PROM of
the shoulder in the 3 directions between NMES and TENS, as well
as compared to those of the control group. Thus, we should not
overestimate the efficacy of electrical stimulation on the
improvement of the shoulder ROM