Professional Documents
Culture Documents
1. excessive heat
2. Chemicals
3. fire/steam
4. radiation
5. electricity
6. hot water
1
BURNS
Results in 10-20 thousand deaths
annually
Survival best at ages 15-45
Children, elderly, and diabetics
Survival best burns cover less than
20% of TBA
2
TYPES OF BURNS
Thermal
exposure to flame or a hot object
Chemical
exposure to acid, alkali or organic substances
Electrical
result from the conversion of electrical energy into
heat. Extent of injury depends on the type of
current, the pathway of flow, local tissue resistance,
and duration of contact
Radiation
result from radiant energy being transferred to the
body resulting in production of cellular toxins
3
Chemical Burn
4
Chemical Burn
Electrical Burn
6
BURN WOUND ASSESSMENT
Classified according to depth of injury
and extent of body surface area
involved
Burn wounds differentiated depending
on the level of dermis and
subcutaneous tissue involved
1. superficial (first-degree)
2. deep (second-degree)
3. full thickness (third and fourth
degree)
7
8
SUPERFICIAL BURNS
(FIRST DEGREE)
10
SUPERFICIAL DERMAL BURNS
(superficial 2nd DEGREE)
12
13
15
DEEP (SECOND DEGREE)
16
17
19
20
FULL THICKNESS (THIRD/FOURTH
DEGREE)
Destruction of all skin layers
Requires immediate hospitalization
Dry, waxy white, leathery, or hard
skin, no pain
Exposure to flames, electricity or
chemicals can cause 3rd degree
burns
21
22
23
Calculation of Burned Body Surface
Area
Calculationof Burned
Body Surface Area
25
TOTAL BODY SURFACE AREA
(TBSA)
Superficial burns are not involved in
the calculation
Lund and Browder Chart is the most
accurate because it adjusts for age
Rule of nines divides the body –
adequate for initial assessment for
adult burns
26
Lund Browder Chart used for
determining BSA
28
29
Physiology
VASCULAR CHANGES RESULTING
FROM BURN INJURIES
Circulatory disruption occurs at the
burn site immediately after a burn
injury
Blood flow decreases or cease due to
occluded blood vessels
Damaged macrophages within the
tissues release chemicals that cause
constriction of vessel
Blood vessel thrombosis may occur
causing necrosis
31
FLUID SHIFT
Occurs after initial vasoconstriction,
then dilation
Blood vessels dilate and leak fluid into
the interstitial space
Known as third spacing or capillary
leak syndrome
Causes decreased blood volume and
blood pressure
Occurs within the first 12 hours after
the burn and can continue to up to 36
hours 32
FLUID IMBALANCES
Occur as a result of fluid shift and cell
damage
Hypovolemia
Metabolic acidosis
Hyperkalemia
Hyponatremia
Hemoconcentration (elevated blood
osmolarity, hematocrit/hemoglobin)
due to dehydration
33
CURLING’S ULCER
Acute ulcerative gastro duodenal
disease
Occur within 24 hours after burn
Due to reduced GI blood flow and
mucosal damage
Treat with PPI
Watch for sudden drop in
hemoglobin
34
PHASES OF BURN INJURIES
Emergent (24-48 hrs)
Acute
Rehabilitative
35
EMERGENT PHASE
Immediate problem is fluid loss, edema,
reduced blood flow (fluid and electrolyte shifts)
Goals:
1. secure airway
2. support circulation by fluid replacement
3. keep the patient comfortable with analgesics
4. prevent infection through wound care
5. maintain body temperature
6. provide emotional support
36
EMERGENT PHASE
Knowledge of circumstances surrounding the
burn injury
Obtain pre-burn weight (dry weight) to
calculate fluid rates
Calculations based on weight obtained after
fluid replacement is started are not accurate
because of water-induced weight gain
Height is important in determining body
surface area (BSA) which is used to
calculate nutritional needs
Know client’s health history because the
physiologic stress seen with a burn can
make a latent disease process develop
symptoms
37
CLINICAL MANIFESTATIONS IN THE
EMERGENT PHASE
41
CLINICAL MANIFESTATIONS
Sympathetic stimulation during the
emergent phase causes reduced GI motility
and paralytic ileus.
42
SKIN ASSESSMENT
Assess the skin to determine the size and
depth of burn injury.
43
IV FLUID THERAPY
Fluid
replacement formulas are calculated
from the time of injury and not from the
time of arrival at the hospital.
Diuretics
should not be given to increase
urine output.
44
FLUID RESUSCITATION IN THE
EMERGENCY ROOM
Colloid formulas
Evans Normal saline 1.0 mL/kg/% burn 1.0 mL/kg/% burn 2000 mL
Crystalloid formulas
Hypertonic saline solution (Monafo)—Volume to maintain urine output at 30 mL/h; fluid contains 25 mEq Na/L
Modified hypertonic (Warden)—Lactated Ringer's + 50 mEq NaHCO3 (180 mEq Na/L) for 8 h to maintain urine output at 30–50 mL/h;
lactated Ringer's to maintain urine output at 30–50 mL/h beginning 8 h postburn
Dextran formula (Demling)—Dextran 40 in saline: 2 mL/kg/h for 8 h; lactated Ringer's: volume to maintain output at 30 mL/h; fresh
frozen plasma: 0.5 mL/kg/h for 18 h beginning 8 h postburn
SOURCE: Reproduced with permission from Warden GD: Burn shock resuscitation. World J Surg 16:16, 1992.
TETANUS
PROPHYLAXIS
GASTRIC
DECOMPRESSION
PAIN CONTROL
PSYCHOSOCIAL CARE
ESCHAROTOMY
ACUTE PHASE OF BURN
INJURY
• Lasts until wound closure is complete
• Care is directed toward continued assessment
and maintenance of the cardiovascular and
respiratory system
• Pneumonia is a concern which can result in
respiratory failure requiring mechanical
ventilation
48
LOCAL AND SYSTEMIC SIGNS OF
INFECTION- GRAM NEGATIVE
BACTERIA
Pseudomonas, Proteus
May lead to septic shock
Conversion of a partial-thickness injury to a full-
thickness injury
Ulceration of health skin at the burn site
Erythematous, nodular lesions in uninvolved skin
Excessive burn wound drainage
Odor
Sloughing of grafts
Altered level of consciousness
Changes in vital signs
Oliguria
GI dysfunction such as diarrhea, vomiting
Metabolic acidosis
49
S aureus P aeruginosa C albicans
Course Slow onset over 2–5 Rapid onset over 12–36 Slow (days)
days hours
51
DRESSING THE BURN WOUND
After burn wounds are cleaned and
debrided, topical antibiotics are
reapplied to prevent infection
Standard wound dressings are
multiple layers of gauze applied over
the topical agents on the burn
wound
52
REHABILITATIVE PHASE OF BURN
INJURY
Started at the time of admission
Technically begins with wound closure
and ends when the client returns to
the highest possible level of
functioning
Provide psychosocial support
Assess home environment, financial
resources, medical equipment,
prosthetic rehab
Health teaching should include
symptoms of infection, drugs
regimens, f/u appointments, comfort
measures to reduce pruritus
53
DIET
Initially NPO
Begin oral fluids after bowel sounds
return
Do not give ice chips or free water
lead to electrolyte imbalance
High protein, high calorie
54
SKIN GRAFTS
Done during the acute phase
Used for full-thickness and deep
partial-thickness wounds
55
POST CARE OF SKIN GRAFTS
Maintain dressing
Use aseptic technique
Graft should look pink if it has taken
after 5 days
Skeletal traction may be used to
prevent contractures
Elastic bandages may be applied for
6 mo to 1 year to prevent
hypertrophic scarring
58
BURNS !!
B Breathing & Body Image
U Urinary output
N Nutrition
5. Start IV’s –
Two large bore IV’s (for major burns)
Fluid resuscitation is particularly
important!
(burns cause increased vascular
permeability with a concurrent
reduction in cardiac output, a
response that lasts at least 12 hours
post injury.)
Management | 10-Steps (Cont.)