MULTIPLE CHOICE QUESTIONS (GYNECOLOGY)
CHAPTER 1: DISORDERS OF MENSTRUATION AND AUB
PHYSIOLOGY OF MENSTRUATION
1. The amount of blood loss in each menstrual cycle is
about- (PGMEE 2012-13)
a. 10cc b. 35cc
c. 50cc d. 100cc
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P.
66;Guyton 12th/e p. 996; Ganong 24th/e p. 404; Berek &
Novak’s Gynecology 15th/e page 404, table 14.9 and 14.10]
Explanation
ƒƒ In normal menstrual cycle-
ƒƒ Menstrual cycle frequency 24-38 days
ƒƒ Duration of flow
ƒƒ Volume of flow
4-8 days
4-80 ml
ƒƒ The average blood flow per cycle is 35 ml.
,2
Es
ƒƒ Disorders of mensuration-
ƒƒ Polymenorrhoea If frequency of cycle is less than 21
days.
ƒƒ Oligomenorrhoea If frequency of cycle is less than 35
iM
days.
ƒƒ Amenorrhoea If no menstruation has occurred in 90
days.
ƒƒ Menorrhagia If cycles are regular and amount of
PR
blood flow is more than 80 ml, or
duration of blood flow is more than 8
days, or both.
ƒƒ Menometrorrhagia If cycles are irregular and amount
of blood flow is more than 80 ml, or
duration of flow is more than 8 days,
or both.
2. Maximum estrogen level in menstrual Cycle is-
 (PGMEE 2012-13)
a. 100-200 pg/ml b. 200-300 pg/ml
c. 300-400 pg/ml d. 400-500 pg/ml
 [Ref: Shaw’s Gynae 15th/e p. 47]
3. Normal blood loss in menstruation is?
 (PGMEE Aug 13 Pattern)
a. 20 to 40 ml b. 40 to 80 ml
c. 80 to 120 ml d. 120 to 150 ml
[Ref: Guyton 12th/e p. 996; Ganong 24th/e p. 404; Dutta’s
Obs. 8th/e, pg. 23-27,30,46; Holland Brew’s Obs.4th/e, pg.16-
19,28; Shaw’s Gynae. 16th/e, pg.10]
Explanation
ƒƒ See question 1
4. Regeneration of Endothelium after menstruation takes
about- (PGMEE 2012-13)
a. 2 days b. 5 days
c. 10 days d. 15 days
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 71;
Shaw’s Gynae 15th/e p. 35]
5. What is seen in luteal phase- (PGMEE 2012-13)
/e
a. Increased progesterone levels
b. Decreased progesterone levels
c. Decreased estrogen level
d. None of the above
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 74
Shaw’s Gynae 15th/e p. 46]
Explanation
ƒƒ Regeneration of endometrium starts even before
1.
2.
b
d
3. a
menstruation ceases and is completed 2-3 days after end 4. a
of menstruation. Thickness of endometrium at this time is
5. a
2 mm.
6. a
6. Estrogen Level in follicular phase of menstrual Cycle-
7. a
 (PGMEE 2012-13)
8. d
a. 100-200 pg/ml b. 200-300 pg/ml
c. 300-400 pg/ml d. 400-500 pg/ml 9. c
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 74 fig
8.10;Shaw’s Gynae 15th/e p. 48]
7. Rescue hormone for corpus leutum is?
a. hCG b. Progesterone
c. hPL d. Estrogen
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 70]
DYSMENORRHOEA
8. Which of the following is a cause of dysmenorrhea:
 (PGMEE 2015)
a. Adenomyosis b. Fibroid
c. Endometriosis d. All of the above
 [Ref: Shaw’s Gyanecology 16th/e p. 471]
9. Dysmenorrhoea is due to? (PGMEE 2014)
a. Ovulation b. Decreased progesterone
c. Increased progesterone d. Secretory epithelium
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 147;
Berek & Novak’s Gynecology 15th/e p. 481]
Explanation
ƒƒ Etiology of dysmenorrhea includes excessive or imbalanced
amount of prostanoids secreted from the endometrium 817
during menstruations.
 PRIMES (Volume II)

ƒƒ Decline in progesterone levels in late luteal phase
triggers released of lytic enzymes, resulting in release of
phospholipids with generation of arachidonic acid and
COX pathway resulting in synthesis of prostanoids. This
increased synthesis of prostanoids lead to higher uterine
tone with high amplitude uterine contractions – thus
causing dysmenorrhea.
10. Mittelschemerz is- (PGMEE 2013-14)
a. Pain just before menstruation
b. Pain at the time of ovulation
c. Pain 5 days after ovulation
d. Pain during menstruation
 [Ref: Jeffcoate’s Gynae. 8th/e, pg. 90]
11. Mainstay of treatment for primary dysmenorrhea is- c. Hydatidiform mole
 (PGMEE 2014)
a. Non-steroidal anti-inflammatory drugs
17. Oligomenorrhoea means -  (PGMEE 2013- 14)
a. Cycle <20 days b. Cycle more than 45 days
c. Cycle more than 28 days d. Cycle more than 35 days
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 153;
Shaw’s Gynae 15th/e p. 283]
18. Age of metropathia hemorrhagica is- (PGMEE 2013-14)
a. 20-25 years b. 50-55 years
c. 60-65 years d. 40-45 years
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 155;
Shaw’s Gynae 15th/e p. 302]
19. Swiss cheese pattern endometrium is seen in
a. Carcinoma endometrium  (PGMEE 2015)
b. Metropathia hemorrhagica
d. Halban’s disease
 [Ref: Textbook of gynecology by Rao:65]

/e
b. Dicyclomine
c. Hyoscine 20. Drug not used commonly for menorrhagia-
d. Paracetamol  (PGMEE 2012-13)
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 148] a. Methergin b. Ormiloxifene

,2
c. Danazol d. NSAIDS
12. Drug of choice in premenstrual syndrome-
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 157;
 (PGMEE 2012-13)
10. b Shaw’s Gynae 15th/e p. 304]
a. Antipsychotics b. SSRI
11. a c. OCP d. Depo progesterone Explanation
Es
12. b [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 150; ƒƒ A only, ormeloxifen or centchroman or Saheli is non
13. d Shaw’s Gynae 15th/e p. 297] hormonal drug used for contraception and also for
14. d menorrhagia.
15. a ABNORMAL UTERINE BLEEDING
16. a 13. Endometrial biopsy is usually performed between :- POSTMENOPAUSAL BLEEDING
iM

17. d  (PGMEE 2016-17) 21. Post menopausal endometrial thickness is: -

18. d a. 1-5 days of menstrual cycle  (PGMEE 2012-13)
19. b b. 7-14 days of menstrual cycle a. 1-3 mm b. 8-9 mm
c. 5-7 days of menstrual cycle c. 5-7 mm d. 6-8 mm
20. a,b
d. 21-28 days of menstrual cycle
PR

21. a [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P463;

 [Ref: D.C Dutta text book of gynaecology 7th e p 156-158
22. a
23. c 14. Metrorrhagia is produced by the following except- 22. Most common cause of postmenopausal vaginal bleeding
 (PGMEE 2013-14)
a. Polyp b. CA endometrium
c. IUD d. Intramural fibroid
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P153]
15. Anovulatory DUB is due to?
 (PGMEE 2013-2014; PGMEE 2013)
a. Absence of progesterone
b. Excess of estrogen
c. Hypothalamic pitutary defect
d. High progesterone
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 155]
16. Most common cause of AUB is? (PGMEE 2014)
a. Anovulatory b. Ovulatory
c. Coagulopathy d. Pregnancy related
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 154]
Shaw’s Gynae 15th/e p. 36]
is? (PGMEE 2010)
a. Endometrial carcinoma b. Ovarian tumor
c. Carcinoma vulva d. Carcinoma cervix
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, Page no. 71,
Jeffcoate’s Principles of Gynaecology, 8th/e, Page no. 576-578]
23. Investigation of choice in post-menopausal bleeding is?
 (PGMEE 2010)
a. Fractional curettage b. Coagulation profile
c. Endometrial biopsy d. D and C
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P463;
Shaw’s Textbook of Gynaecology, 15th/e, Page no. 71-72;
Jeffcoate’s Principles of Gynaecology, 7th/e, P. 13, 613-614;
Berek & Novak’s Gynecology, 15th/e, P. 1251]

818
 CHAPTER 2: INFERTILITY
CAUSES
MALE INFERTILITY
1. Which of the following is true about obstructive azoosper-
mia- (PGMEE 2009)
a. FSH and LH
b. Normal FSH and Normal LH
c. FSH, Normal LH
d. LH, Normal FSH
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,
P191;Shaw 15th/e p. 210 & 13th/e p. 203; Oxford textbook of
Medicine 4th/e p. 283; Dewhurst Obs & Gynaec 10th/e p. 449;
Obstetrics & Gynaecology by James Draycott 1999 p. 137;
Berek & Novak’s Gynecology 15th edition page 1146.]
2. Pt. with low testosterone & low count can show all except- 
a. Decreased FSH  (PGMEE 2012-13)
,2
b. Decreased LH
c. Increased FSH
d. Obstruction of spermatic duct
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P190-191
Shaw’s Gynae 15th/e p. 204]
Es
FEMALE INFERTILITY
3. What is the % of tubal causes of female infertility?
 (PGMEE 2014)
iM
a. 7% b. 19%
c. 26% d. 52%
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P188;
Berek & Novak’s Gynecology 15th edition page 1134.]
PR
Explanation
ƒƒ Tubal factors account for 30-40% causes for female
infertility.
4. In hypergonadotropic hypogonadism FSH level is? 
 (PGMEE 2014)
a. <20 m IU/ml b. >20 m IU/ml
c. <40 m IU/ml d. >40 m IU/ml
[Ref: Speroff’s Clinical Gyaecologic Endocrinology and
Infertility 8th/e p. 443; D. C. Dutta’s Textbook of gynaecology,
7th/e, P374
5. A patient with infertility was prescribed bromocriptine by
the gynecologist? What would be the cause? 
a. Hypogonadotropic hypogonadism  (AIIMS Nov’ 2017)
b. Premature ovarian failure
c. Hyperprolactinemia
d. PCOS
 Ref: Speroff 8th e.p.1328
Explanation
ƒƒ Dopamine agonists are the treatment of choice for hyper-
prolactinemic infertile women with ovulatory dysfunction
who wish to conceive.
6. In hypogonadotropic hypogonadism, what all can be seen
except.  (AIIMS Nov’ 2017)
a. Low GNRH b. Low LH & FSH
c. Low estradiol d. Hyperprolactinemia
 Ref: Speroff 8th e p.1295
Explanation
ƒƒ WHO Group I: Hypogonadotropic Hypogonadal
Anovulation. The group accounts for approximately
5–10% of anovulatory women and includes those with low
or low-normal serum follicle-stimulating hormone (FSH)
concentrations, and low serum estradiol levels, due to absent
or abnormal hypothalamic gonadotropin-releasing hormone
(GnRH) secretion or pituitary insensitivity to GnRH.
/e
Examples include women with hypothalamic amenorrhea
relating to physical, nutritional, or emotional stress, weight
loss, excessive exercise, anorexia nervosa and its variants,
Kallmann syndrome, and isolated gonadotropin defi ciency.
Women in the group may require hypothalamic-pituitary
imaging to exclude a mass lesion.
ƒƒ WHO Group II: Eugonadotropic Euestrogenic 1. b
Anovulation. This group is the largest, including 75–85%
2. d
of anovulatory women, and is characterized by normal
3. c
serum FSH and estradiol levels and normal or elevated LH
concentrations.7 The most common examples are women 4. b
with polycystic ovary syndrome (PCOS), some of whom 5. c
ovulate at least occasionally. Women with PCOS should 6. d
be screened for type 2 diabetes mellitus before treatment, 7. b
due to the fetal risks associated with untreated diabetes.8
Weight loss generally is the best initial treatment for those
who are obese because it can, by itself, restore ovulatory
function
ƒƒ WHO Group III: Hypergonadotropic Anovulation. The
group accounts for approximately 10–20% of anovulatory
women and includes those with elevated serum FSH
concentrations; most, but not all, have amenorrhea. The
classic example is premature ovarian failure, due to
follicular depletion, and few respond to treatment aimed at
ovulation induction.
ƒƒ Hyperprolactinemic Anovulation. Approximately 5–10%
of anovulatory women have hyperprolactinemia, which
inhibits gonadotropin secretion. Consequently, serum
FSH concentrations generally are low or low-normal
and serum estradiol levels also tend to be relatively low.
Most hyperprolactinemic women have oligomenorrhea
or amenorrhea. When hyperprolactinemia cannot be
attributed confi dently to coexisting hypothyroidism or to
medications, hypothalamic-pituitary imaging is indicated to
exclude a mass lesion.
7. Cause of infertility in hypothyroidism- (PGMEE 2012-13)
a. Decrease prolactin
b. Increased prolactin
c. Both
d. None
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P385] 819
 PRIMES (Volume II)

Explanation 12. Ovarian reserve is measured by- (PGMEE 2013-14)

a. FSH b. LH
ƒƒ Reasons for infertility in females with hypothyroidism –
c. FSH & LH d. ESTRADIOL
○○ Anovultion
○○ hyperprolactinemia [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P437;
○○ Luteal phase defect Shaw’s Gynae 15th/e p. 47, 216; Speroff 7th/e p. 444-448]
○○ Sex hormone imbalance 13. Semen examination can be done: (PGMEE 2013-14)
○○ Autoimmune factors. a. Immediately in semi solid form
8. A 35 year old nulliparous women with primary infertility b. After liquefaction
presents with adenexal mass and CA -125 level of >60 U/ c. Within 15-30 minutes of liquefaction
mL. What is the most likely diagnosis? -  d. 1½-2 hr irrespective of liquefaction
a. Ovarian cancer  (AIIMS May’2010) [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P190,
b. Endometrioma Shaw’s Gynae 15th/e p. 203]
c. Borderline ovarian tumour
d. Tuberculosis 14. On which day LH & FSH should be measured-
 (PGMEE 2013-14)
Explanation a. 1-3rd day b. 7th day
c. 1-4th day d. 10th day

/e
D/d of infertility in adenexal masses  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P]
Clinical 15. Normal sperm motility normal according to WHO is-
Age & parity Cl/f CA 125
condition  (PGMEE 2012-13)

,2
Ovarian Elderly Abdominal ↑ in 50% a. > 20% b. > 30%
Ca distension, of epithelial c. > 40% d. > 50%
8. b mass tumours [Ref: C. Dutta’s Textbook of gynaecology, 7th/e, P 190;Shaw’s
abdomen Gynae 15th/e p. 204; Berek & Novak’s Gynecology 15th
9. a
Borderline Middle age Normal edition page 1141, table 32.2.]
Es
10. d
ovarian
11. a Explanation
tumours
12. a ƒƒ WHO Guidelines for semen analysis-
Endometrioma Middle age Painful ↑ in > 80%
13. d
adenexal 1992 2010 guidelines
14. a mass, 10 guidelines
iM

15. d infertility
16. a ƒƒ Volume 2 ml ≥ 1.5 ml
TB Any age ↑ Painless May be ↑
17. a adenexal ƒƒ Sperm concentration 20 million/ml ≥ 15 million/ml
mass ƒƒ Sperm motility 50% ≥ 32%
PR

10 infertility progressive, or progressive

>25% rapidly
INVESTIGATIONS progressive
9. Aspermia:- (PGMEE 2016-17) ƒƒ Morphology (strict >15% normal ≥ 4% normal
a. No semen b. No sperm in semen criteria) forms forms
c. Dead sperm in semen d. Low semen volume
ƒƒ White blood cells < 1 million/ml < 1 million/ml
 [Ref: D.C Dutta text book of gynaecology 7th e p190]
ƒƒ Immunobead or < 10% coated < 50%
10. Anovulation may be diagnosed with:- (PGMEE 2016-17) mixed antiglobulin with antibodies
a. Low LH b. High FSH reaction test
c. Static elevated estrogen d. Low progesterone
[Ref: speroff 8th e p.1162; D.C Dutta text book of gynaecology 16. The confirmatory test for presence of semen is? 
7th e p 193]  (PGMEE Nov. 13 Pattern)
a. P 30 b. Casein
11. All of the following are markers of ovarian reserve except-
c. Amylase d. Alpha feto protein
 (PGMEE 2015)
a. Inhibin A b. Estradiol concentration 17. Fertile period of female is measured by-
c. Inhibin B d. Ovarian volume  (PGMEE 2012-13)
a. LH b. FSH
[Ref: C. Dutta’s Textbook of gynaecology, 7th/e, P 437;Speroff’s c. Estrogen d. Oxytocin
Clinical Gyaecologic Endocrinology and Infertility 8th/e p.
1147]  [Ref: Shaw’s Gynae 15th/e p. 297]
820
Gynecology  Chapter 2 Infertility (MCQs)

18. Inhibin B levels as test of ovarian reserve is measured on? 24. Treatment of immunological infertility is? 
 (PGMEE 2011)  (PGMEE Nov 13 Pattern)
a. Day 2 of menstruation b. Day 3 of menstruation a. ICSI b. GIFT
c. Day 4 of menstruation d. Day 5 of Menstruation c. IVF d. IUI
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 45; Berek & [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P203;Novak
Novak’s Gynecology 15th edition page 1149.] 15th/e ch. 32p. 1134; Harrison 17th/e ch. 341; Danforth’s Obs
& Gynae 10th/e p. 706; Berek & Novak’s Gynecology 15th
Explanation edition page 1163.]
ƒƒ Inhibin B levels are measured on 5th day of ovarian
Explanation
stimulation.
19. Best diagnosis of ovulation is by?  (PGMEE 2012) ƒƒ Immunological factors for infertility have not been found to
a. Ultrasound b. Chromotubation affect IVF outcomes.
c. Endometrial biopsy d. Laparscopy 25. All are indications of intra uterine insemination except: 
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P194 a. Vicid cervical mucus  (PGMEE 2010)
Novak’s Gynae 14th/e p. 1207; Shaw’s 13th/e p. 33] b. Immune factor of sperms
c. Tubal blockade
20. Best test for ovulation- d. Oligozoospermia

/e
 (PGMEE 2012-13; PGMEE Nov 12 Pattern)
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P203;
a. Serum estrogen b. Serum progesterone
Shaw’s Textbook of Gynaecology, 15th/e, Page no. 207
c. Both d. None
Jeffcoate’s Principles of Gynaecology, 7th/e, P. 724-6 Berek &
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P196;

,2
Novak’s Gynecology, 15th/e, P. 1145, 1153
Shaw’s Gynae 15th/e p. 216]
26. AID the term used to describe the artificial insemination
21. Dye used in Hysterosapingography- (PGMEE 2012-13) achieved by using sperms of-  (PGMEE 2012-13)
a. Conray 420 b. Renografin-60 18. d
a. Husband b. Donor
c. Toluidine blue d. None of the above 19. a
c. Both d. None
Es
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e486] 20. b
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P204]
21. b
TREATMENT ASSISTED REPRODUCTIVE TECHNIQUES 22. a
22. Vasectomy reversal in primary infertility is done within 23. a
27. Which of the following is not an assisted reproductive
how many years of vasectomy- (PGMEE2012-13) technique (ART)? (PGMEE Aug 12 Pattern) 24. c
iM

a. 5 yrs b. 6 yrs a. ZIFT b. GIST 25. c

c. 7 yrs d. 8 yrs c. IVF d. Artificial insemination 26. b
 [Ref: speroff 8th p.936; Shaw’s Gynae 15th/e p. 205] [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P204; 27. d
23. Drug of choice for ovulation induction- (PGMEE 2012-13) COGT 2009/e Chapter 58; Danforth’s 10/e p. 710]
PR

a. Clomiphene b. FSH
c. LH d. hCG
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,
P199;Shaw’s Gynae 15th/e p. 217]

821
 CHAPTER 3: CONTRACEPTION

METHODS 7. Ideal contraceptive in RHD- (PGMEE 2012-13)

1. Which of following is/are not sexual awareness method of
contraception? (PGMEE 2016)
a. Withdrawal method b. Barrier methods
c. Cervical mucus d. MTP kit
e. Symptothermal method
[Ref: D.C Dutta text book of gynaecology 7th e p 414]
2. Knaus-Ogino method is what type of contraceptive meth-
od? (PGMEE Aug 13 Pattern)
a. Calender method
b. Withdrawal method
c. Barrier method
d. Cervical mucous rhythm method
a. Barrier b. IUCD
c. OCPs d. DMPA
[Ref: Park 20th/e p. 425; Shaw’s Gynae 15th/e p. 244; Dutta
Obs 7th/e p. 545]
8. Today vaginal sponge failure rate is?  (PGMEE Nov. 12
Pattern)
a. 5% b. 9%
c. 16% d. 20%
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,
P413;speroff clinicalgynaecologic endocrinology& infertility
8th ep.1123
Lowest expected in nulliparous is 9%, lowest expected in

/e
3. Billing’s method of contraception is a: multiparous is 20%
 (PGMEE 2010; PGMEE Aug 12 Pattern)
IUCD
a. Barrier method
b. Hormonal method 9. Pregnancy occurred with IUCD in situ, the strings are

1.
2.
d
a


c. Behavioral method
d. None

,2
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 223
Jeffcoate’s Principles of Gynaecology, 7th/e, P. 787-788 Berek




however not visible, all of the following can be done except-
a. Leave in situ 
b. Remove under USG guidance
c. Remove using hysteroscope
d. Remove using IUCD hook
(PGMEE 2015)
Es
3. c & Novak’s Gynecology, 15th/e, P. 218]
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P396;
4. c 4. Contraception failure defined in terms of?(PGMEE 2009)
Speroff’s Clinical Gynaecologic Endocrinology and Infertility
5. c a. Per woman years b. Per 10 woman years
8th/e p. 1109]
c. Per 100 woman years d. Per 1000 woman years
6. b 10. IUD in situ does not cause- (PGMEE 2015)
7. a [Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 223;
iM

Jeffcoate’s Principles of Gynaecology, 7th/e, P. 787-788; a. Pregnancy

8. b b. Ectopic pregnancy
Berek & Novak’s Gynecology, 15th/e, P. 218
9. d c. Pain in abdomen
d. Intrauterine malformation
10. d TEMPORARY METHODS
11. c  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P395-6]
PR

12. a
BARRIER METHODS
5. Identify the contraceptive device shown in the picture.
 (PGMEE 2016-17)
a. Diaphragm b. Male condom
c. Female condom d. Diva cup
 [Ref: D.C Dutta text book of gynaecology 7th e p 412]
6. HIV positive couple with pregnant female, contraceptive of
choice is- (PGMEE 2012-13)
a. Abstinence b. Condoms
c. No restrictions d. OCPs
822
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P412;Dutta
Obs 7th/e p. 303]
11. Which IUCD has the maximum chances of development of
ectopic pregnancy:- (PGMEE 2015)
a. Levenogestrol b. Cu T 380 A
c. Progestasert d. Cu T 200
[Ref: Speroff’s Clinical Gynaecologic Endocrinology and
Infertility 8th/e p. 1105]
12. When is copper T inserted- (PGMEE 2013-14)
a. 3 days after periods are over
b. Within 10 days of menstrual cycle
c. PID just before menstruation
d. Just after menstruation
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P393]
Explanation
ƒƒ Best time for Cu-T insertion is during menses, reason –
pain due to Cu-T insertion is masked by dysmenorrhea and
confirmation of non pregnant state occurs.
ƒƒ Else it can be inserted any time after exclusion of PID or
Pregnancy.
Gynecology  Chapter 3 Contraception (MCQs)

13. Absolute contraindication for IUCD is-(PGMEE 2013-14) 18. The most common side effect of IUD insertion is-
a. Menorrhagia b. Vaginal bleeding  (DNB 2007)
c. Fibroid d. Purulent discharge a. Bleeding b. Pain
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P393; c. Pelvic infection d. Ectopic pregnancy
Shaw’s 15th/e p. 227]  [Ref: Park’s textbook of PSM 22nd/e p. 459]
14. About IUCD all are true except: (PGMEE Nov 13 Pattern) Explanation
a. Multiload 375 is a third generation IUCD
ƒƒ Side effects and complications of IUDs
b. Lippes loop and Cu T 200 have same pregnancy rate
○○ Bledding –most common
c. IUCD can be used as emergency contraception
○○ Pain – second major side effect
d. LNG IUD has half life of 10 years
○○ Pelvic infection- PID
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P392-393; ○○ Uterine perforation
Novak’s 14th/e ch. 10t. 102; William’s 22nd/e ch. 32] ○○ Pregnancy
15. Women with menorrhagia IUCD of choice:  ○○ Ectopic pregnancy
 (PGMEE 2012-13) ○○ Expulsion
a. NOVA T b. LNG IUD ○○ Mortality – extremely rare
c. Mirena d. Gyne fix ƒƒ Most common side effect requiring removal of the device

/e
- pain
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P397;
ƒƒ The commonest complaint of women fitted an IUCD (inert
Shaw’s Gynae 15th/e p. 303]
or medicated) is increased vaginal bleeding. It accounts for
16. Intrauterine contraceptive device with effective life of 10 10-20 percent of all IUCD removals. The bleeding may take

,2
years is? (PGMEE Nov. 12 Pattern) one or more of all the following forms: greater volume of
a. Cu T 380A b. Progestasert blood loss during menstruation, longer menstrual periods
c. Cu T 200 d. Mirena or mid-cycle bleeding. Copper devices seem to cause less
13. d
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P393; average blood loss. Menstrual blood loss is consistently
lower when hormone-releasing devices are used. If the 14. a
Novak’s 14th/e Chapter 10]
Es
bleeding is heavy or persistent or if the patient develop 15. c
17. Cu T 380A IUCD should be replaced once in- anemia despite the iron supplement the IUCD should be 16. a
 (PGMEE pattern 2008) removed. 17. d
a. 4 yrs b. 6 yrs
19. Mirena releases ____­­ microgram of LNG/day? 18. a
c. 8 yrs d. 10 yrs
 (PGMEE Nov. 12 Pattern) 19. a
iM

 [Ref: Park’s textbook of PSM 22nd/e p. 458] a. 20 b. 55

20. a
c. 65 d. 380
Explanation 21. a
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P393;
ƒƒ Cu-T-380A is approved for use for 10 years. However, the 22. d
Novak’s 14th/e Chapter 10]
Cu-T-380A has been demonstrated to maintain its efficacy
PR

over at least 12 years of use. The CuT-200 is approved 20. Which of the following has the least risk of ectopic preg-
for 4 years and the Nova T for 5 years. The progesterone- nancy? (PGMEE 2011)
releasing IUD must be replaced every year because the a. Condoms b. OC pills
reservoir of progesterone is depleted in 12-18 months. The c. Copper T d. Tubectomy
levonorgestrel IUD can be used for at least 7 years, and [Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 230, 267;
probably 10 years. Jeffcoate’s Principles of Gynaecology, 7th/e, P. 789, 799;
Device Pregnancy Expulsion Life span Berek & Novak’s Gynecology, 15th/e, P. 224]
rate(%) rate (%) (years)
First generation: 3.0 19.1 -
STEROIDAL
Lippe’s loop
ORAL CONTRACEPTIVE PILLS
Second
generation : 21. OCP will b protective to:- (PGMEE 2016-17)
a. Ovarian carcinoma b. Breast cancer
Cu T-200 3.0 7.8 3
c. Cervical cancer d. Liver cancer
CuT-380A 0.5-0.8 5 10  [Ref: D.C Dutta text book of gynaecology 7th e p 402]
NovaT 0.7 5.8 5 22. Regarding OCPs all are true EXCEPT? (PGMEE 2016)
Multiload 250 0.5 2.2 3 a. Can increase thromboembolic events
b. Protects from benign conditions of breast
Third generation : c. Protects against endometrial cancer
Progestasert 1.8 3.1 1 d. Decreases bone mineral density 823
 [Ref: D.C Dutta text book of gynaecology 7th e p 401-2]
 PRIMES (Volume II)

23. Absolute contraindications of OCP are all except-
 (PGMEE 2015)
a. Sickle cell disease b. Breast cancer
c. Ischemic heart disease d. Hepatoma
 [Ref: Dutta’s Obstetrics 8th/e p. 623]
24. Which of the following is not a mechanism of action of
OCPs- (PGMEE 2015)
a. Inhibition of ovulation
b. Alteration of cervical mucus
c. Out of phase endometrium
d. None of the above
 [Ref: Dutta’s Obstetrics 8th/e p. 622]
25. Lowest amount of estrogen in OCP is-
 (PGMEE 2015)
a. 15 microgram b. 20 microgram
c. 30 microgram d. 35 microgram
30. OC pills must be started on- (PGMEE 2012-13)
a. 5th day b. 3rd day
c. 1st day d. When menses cease
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P399]
31. Non-contraceptive metabolic effect of estrogen are all
except- (PGMEE 2012-13)
a. Increased fatty acids
b. Increased plasma lipid and lipoproteins
c. Increased total cholesterol
d. Decreased HDL
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P402]
32. OCPs are contraindicated in? (PGMEE 2011)
a. Heart disease b. Breast cancer
c. Thromboembolism d. All of the above
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 232;[Ref:

/e
D. C. Dutta’s Textbook of gynaecology, 7th/e, P401;Jeffcoate’s
[Ref: Speroff 8the p.966; Practical Obstetrics and
Principles of Gynaecology, 7th/e, P. 801-804;Berek & Novak’s
Gynaecology, Virkhud p. 188]
Gynecology, 15th/e, P. 215]
26. What is to be done if 2 OCP is missed on day 17-18 of the

,2
33. Least failure rate is of- (PGMEE 2012-13)
cycle- (PGMEE 2015)
a. OC pills b. IUDs
a. Use back up contraceptive
c. Condom d. DMPA
b. Take 2 pills on the next 2 days
23. a [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P397,401;
c. Continue taking single pill per day
24. d d. Both a and b Speroff’s gynecology – Section III- contraception and Berek &
Es
25. b Novak’s Gynecology 15th edition page 215.]
 [Ref: Dutta’s Obstetrics 8th/e p. 624; Speroff’s
26. a gynecology – Section III- contraception-] 34. OCP decrease risk of - (PGMEE 2012-13)
27. d a. Cervical ca b. Endometrial ca
28. a Explanation
c. Vaginal ca d. Liver carcinoma
29. c If 2 pills are missed  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P401]
iM

30. c ƒƒ In first 2 weeks – take 2 pills daily for 2 days and then finish 35. OC pills decrease risk of- (PGMEE 2012-13)
31. d the pack + use of back up method advised for 7 days. a. Stroke b. CVD
32. d ƒƒ During 3rd week – 2 options – c. Endometrial ca d. Hepatic adenoma
33. a,b ○○ Start new pack and use back up method immediately and  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P401]
PR

34. b for 7 days, or

○○ Take daily pill upto next Sunday and then start new pack
35. c
and use back up method immediately and for 7 days.
36. b
37. a 27. OCP causes? (PGMEE 2015; Nov 13 Pattern)
a. Simple hyperplasia b. Atypical hyperplasia
c. Endometrial proliferation d. Endometrial atrophy
[Ref: speroff 8the p.974; Endometrial cytology with tissue
correlations-John A. Maksem, Stanley J. Robby, John W.
Bishop p. 94]
28. OCP does not prevent? (PGMEE 2015; Nov 13 Pattern)
a. Breast cancer b. Endometrial cancer
c. Ovarian cancer d. Ovarian cysts
36. Which of the following causes OCP failure?
 (PGMEE Nov. 12 Pattern)
a. Carbamazepine b. Rifampicin
c. NSAIDS d. Ethambutol
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P400;KDT
7th/e p. 317]
37. Which of the following progesterones is preferred in
combination with estrogen in Low dose Oral contraceptive
pills- (PGMEE 2011)
a. Desogestrel b. Norethisterone
c. Norgestrel d. Levonorgestrel

[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P401; [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,
Novak’s gynae 14th/e p. 275] P398;Harrison’s Endocrinology 2nd/e p. 199; CMDT 2011/e
p. 743; Drug Benefits and Risk: International Textbook of
29. OC pills decrease the risk of- (PGMEE 2012-13) clinical Pharmacology 2nd/e p. 462]
a. Stroke b. CVD
c. Endometrial ca d. Hepatic adenoma
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P401]

824
Gynecology 
38. Which of the following methods of contraception should be
avaoided in women with epilepsy- (PGMEE 2011)
a. Oral contraceptive pills b. IUCD
c. Condoms d. Diaphragm
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,
P400;Shaw’s 13th/e p. 228; Berek & Novak’s Gynaecology
14th/e p. 286; Wyllie’s Treatment of Epilepsy: Principles &
Practice (Lippincott Williams & Wilkins] 2010/e p. 657]
39. Role of OCP in cervical cancer is-
a. Predisposes to squamous cell carcinoma of the cervix
b. Increases chances of Human Papilloma virus infection
c. Predisposes to adenocarcinoma of the endocervix
d. Has a preventive effect towards the carcinoma of cervix
[Ref: Speroff clinical endocrinology 8th e pg 996;CGDT
10th/e p. 834; Shaw Gynae 14th/e p.
CENTCHROMAN
40. Dose of centchroman is- (PGMEE June 14 Pattern)
a. 30 mg b. 60 mg
,2
c. 90 mg d. 20 mg
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P415]
41. Centchroman is a? (PGMEE 2009)
a. Female oral contraceptive
Es
b. Oxytocic
c. Tocolytic
d. Male contraceptive
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P415]
PROGESTERONE ONLY PILL (POP)
iM
42. Progesterone only pills act by:- (PGMEE 2018)
a. Cervical mucous thickening
b. Prevention of implantation
PR
c. Inhibition of ovulation
d. Interfering sperm motility
[Ref speroff 8th p.1037; d.c. dutta text book of
gynecology p.403 7th]
Explanation
POP
ƒƒ POP works mainly by making cervical mucous thick
and viscous . thereby prevents sperm penentration.
Endometrium becomes atrophic, so blastocyst implantation
is also hindered. In about 2 % cases ovulation is inhibhited
and 50% ovulate normally.
ƒƒ After taking a progestin-only minipill, the small amount of
progestin in the circulation (about 25% of that in combined
oral contraceptives) will have a significant impact only
on those tissues very sensitive to the female sex steroids,
estrogen and progesterone. The contraceptive effect is
more dependent on endometrial and cervical mucus effects,
because gonadotropins are not consistently suppressed.
The endometrium involutes and becomes hostile to
implantation, and the cervical mucus becomes thick and
impermeable. Approximately 40% of patients will ovulate
normally. Tubal physiology may also be affected
Chapter 3 Contraception (MCQs)
43. Pelvic inflammatory Disease prevented by:-
 (PGMEE 2016-17)
a. Tubal block b. IUC
c. Progesterone only pill d. Condom
 [Ref: D.C Dutta text book of gynaecology 7th e p403; Berek
& Novak’s Gynecology 15th edition page 219]
44. Which is not a side effect of POP (Progestin only pill)-
a. Ectopic pregnancy  (PGMEE 2015)
b. Increased risk of diabetes mellitus
c. Ovarian cysts
d. Venous thromboembolism
[Ref: Dutta’s Obstetrics 8th/e p. 627; Speroff’s Clinical
Gyanecologic Endocrinology and Infertility, 8th/e p. 1037]
45. Contraceptive of choice during lactation is-
 (PGMEE 2013-14)
a. POP b. PC pills
/e
c. IUD d. Minena
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P403]
46. Minipill, side effect is- (PGMEE 2012-13)
a. Break through Bleeding b. PID
c. Endometrial CA d. Thromboembolism
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P403] 38. a
47. Lactating mother OCP of choice- (PGMEE 2012-13) 39. a,c
a. Progestin only pil b. Combined OCP 40. a
c. Estrogen only pill d. None 41. a
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P403] 42. a
48. Side effect of progesterone only pill are all except- 43. d
 (PGMEE 2012-13) 44. d
a. Irregular bleeding b. Amenorrhea 45. a
c. Decreased lactation d. Weight gain 46. a
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P403Shaw’s 47. a
Gynae 15th/e p. 233] 48. c
49. Progesterone only pill not given after-  (PGMEE 2012-13) 49. a
a. 24 hrs b. 48 hrs 50. b
c. 72 hrs d. 96 hrs
51. c
 [Ref: D.C. Dutta’s Textbook of gynaecology, 7th/e, P403]
50. The most common side effect of POP (progesterone only
pills) is? (PGMEE 2012)
a. Hypertension b. Irregular bleeding
c. Acne d. DVT
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 233,
Jeffcoate’s Principles of Gynaecology, 7th/e, P. 811, [Ref: D.
C. Dutta’s Textbook of gynaecology, 7th/e, P403
INJECTABLE CONTRACEPTIVES AND IMPLANTS
51. DMPA 150mg subcutaneous is to be repeated every:-
 (PGMEE 2016)
a. 1 month b. 2 months
c. 3 months d. 1 year
[Ref: D.C Dutta text book of gynaecology 7th e p403; From
ROAMS 13th/e pg. 652]
825
 PRIMES (Volume II)

52. Contraceptive failure is minimum with?
 (PGMEE Nov. 12 Pattern)
a. Combined oral contraceptive
b. Injectable hormonal contraceptive
c. Hormonal IUCD
d. Subdermal implant (Norplant]
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P404]
53. Which of the following is not an adverse effect of DMPA? a. Estradiol b. Levonorgestrel
 (PGMEE Nov. 12 Pattern)
a. Slight blood glucose elevation
b. Disturbance of lactation
c. PID
d. Bone loss
 [Ref: Speroff 8th e. p.1085]
54. Depot MPA (DMPA), all are true except (PGMEE 2012-13)
a. Dysmenorrhea
60. Emergency contraceptive should must be started with in
how much time after unprotected intercourse-
 (PGMEE 2012-13)
a. 24 hrs b. 48 hrs
c. 72 hrs d. 96 hrs
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P404]
61. Emergency contraceptive contains? (PGMEE 2009)
c. PGE2 d. Estrone
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P404
62. Following unprotected intercourse, Emergency Contra-
ceptive levonorgestrel 0.75 mg is taken by the patient.
What are the doses that she should take subsequently? 
a. 1 dose of 0.75 mg after 12 hours  (AIIMS Nov’ 2017)
b. 2 dose of 0.75 mg after 12 hours
c. 3 dose of 0.75 mg after 12 hours

/e
b. Dyslipidemia d. 4 dose of 0.75 mg after 12 hours
c. Do not prevent STDs  Ref: Dutta 7th e p. 405
d. Can be given in breast cancer
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P404; Explanation

52.
53.
d
b


Shaw’s Gynae 15th/e p. 234]
55. Duration of contraception after DMPA is-

a. 21 months
,2
(PGMEE 2012-13)
b. 1-3 months
ƒƒ Levonorgestrel (e.pills) 0.75 mg two doses given at 12 hrs
interval. The two tablets 1.5 mg can be taken as a single
dose also. The first dose should be taken within 72 hours
may be taken up to 120 hrs.
Es
54. d c. 3-6 months d. >6 months
55. c
STERILIZATION
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P404]
56. d 63. Most common site of tubectomy is:- (PGMEE 2016-17)
56. Long acting revesible contraceptives (LARC) are all a. Isthumus
57. b
except- (PGMEE 2015; Nov 13 Pattern) b. Ampulla
58. a a. Implanon b. DMPA c. Interstitial
iM

59. a c. IUCD d. OCPs d. Infudibular

60. c [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P398,  [Ref: D.C Dutta text book of gynaecology 7th ep.408]
61. b speroff 8th e p1059.
64. Contraceptive failure rate of Pomeroy’s method is?
62. a
 (PGMEE Nov. 12 Pattern)
EMERGENCY
PR

63. a a. 1/1000 cases b. 2/1000 cases

64. d 57. Single dose of drug used in emergency contraception - c. 3/1000 cases d. 4/1000 cases
65. d a. Levonorgestrol 0.75 mg  (PGMEE 2015)
[Ref: speroff 8th p.912; Berek & Novak’s Gynae 14th/e p. 287;
66. d b. Levonorgestral 1.5 mg
Shaw’s 13th/e p. 236]
c. Desogestrel 1.5 mg
67. c 65. Which of the following is not a ligation technique?
d. Ethinyl estradiol + levonorgestrel
[Ref: Shaw’s Gyanecology 16th/e p. 279; Speroff’s Clinical  (AIIMS Nov 14)
Endocrinology and Infertility 8th/e p. 1040] a. Irving b. Parkland
c. Pomeroy d. Essure
58. Not an emergency contraception?  (PGMEE 2013-14)
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,p.416
a. IUCD
b. LNG 66. Pomeroy technique is for- (PGMEE 2012-13)
c. Mifepristone a. Tubal ligation
d. Combined estrogen and progesterone b. Laprscopy
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P404 c. Hysteroscopy
Shaw’s Gynae 15th/e p. 551] d. Minilaparotomy
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,409
59. I pill is used when- (PGMEE 2012-13)
a. Accidental sexual exposure 67. Maximum success of reversal after tubal ligation with-
b. OCP forgotten  (PGMEE 2012-13)
c. Of choice in young a. Cauterization b. Pomeroy’s technique
d. All of the above c. Clip method d. Fimbriectomy
826
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P405]  [Ref: speroff 8th e p.935]
Gynecology  Chapter 3 Contraception (MCQs)

68. Laproscopic tubal ligation contraindication- along the antimesentric border to expose the muscular tube
a. Post partum state  (PGMEE 2012-13) about 3-5 cm of tube is excised. The ligated proximal stump
b. Post MTP is buried beneath the serous coat. Distal stump is open in
c. Gynaecologic malignancies peritoneal cavity.
d. 3 pervious child birth ƒƒ Irving : the tube is ligated on ethier side and mid ortion
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,p.409] of tube is excised. The free medial end is turned back and
buried in posterior uterine wall creating a myometrial
69. Which one of the following techniques of tubectomy is tunnel.
depicted in the diagram below? (PGMEE 2015) ƒƒ ESSURE: Hysteroscopic tubal ligation with nickel titanium
alloy.

/e
,2 68. a
69. c
Es
70. d
iM

a. Kroener technique b. Irving technique

c. Pomeroy technique d. Uchida technique
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, 409,
speroff 8th ep921
70. Technique in which fimbriectomy is done:- (PGMEE 2018)
PR

a. Uchida technique
b. Irving’s method
c. Modified Pomeroy method
d. Kroener
Ref: D.C. dutta text book of gynecology p.408-409 7th e;
 Speroff 8th e p.921-931

Explanation
ƒƒ Fimbriectomy is done in Kroener procedure : ampullary
end of tube is ligated and dissected .
ƒƒ Pomeroy : a loop is made by holding the tube by an allis
forceps such a way that the major part of loop consist
mainly of isthumuns and ampullary part . about 1-1.5 cm of
loop is excised (Loop-ligate-cut)
ƒƒ Modified Pomeroy : Loop-ligate-cut and crush the end.
ƒƒ Uchida : a saline solution is injected subserosaly in the mid
portion of tube to create bleb. The serous coat is incised

827
 CHAPTER 4: BENIGN DISORDER OF REPRODUCTIVE TRACT

BENIGN LESIONS OF VULVA AND VAGINA 9. Most common degeneration of fibroids-(PGMEE 2012-13)
a. Calcareous b. Hyaline
1. In what condition does vulvectomy has to be performed c. Red d. Cystic
sometimes- (PGMEE 2012-13) [Ref: D.C Dutta text book of gynaecology 7th e p.224;
a. Granuloma inguinale b. Chlamydia trachomitis Jeffcoate 7th/e p. 501]
c. Herpes simplex d. Candidial infection
10. Pain in fibroid is due to all except: (PGMEE 2011)
 [Ref: D.C Dutta text book of gynaecology 7th e p.124] a. Red degeneration b. Sarcomatous degeneration
2. Lichen sclerosis of vulva false is (PGMEE 2012-13) c. Hyaline degeneration d. Torsion
a. It is infective [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P224;Shaw’s
b. Usually occurs in old age Textbook of Gynaecology, 15th/e, P. 353-355;Jeffcoate’s
c. May b associated with autoimmune disease Principles of Gynaecology, 7th/e, P. 500-502]
d. Due to decreased estrogen
11. Fibroid uterus may present with all of the following ex-
[Ref: D.C Dutta text book of gynaecologyp7th e.211 Shaw’s cept-  (PGMEE 2007)
Gynae 15th/e p. 124-125] a. Amenorrhoea b. Pelvic mass

/e
c. Infertility d. Polymenorrhea
BENIGN LESIONS OF CERVIX [Ref: D.C Dutta text book of gynaecology 7th ep.225; Shaws
15th/e p. 356]
3. True about nabothian cyst all except- (PGMEE 2015)

,2
a. It is pre-malignant 12. A 40 year old female presents with history of abnormal
b. It is a pathology of the cervix uterine bleeding underwent hysterectomy. Post hysterec-
c. It is seen in chronic irritation and inflammation tomy specimen is shown below. Most likely diagnosis is:-
1. a  (PGMEE 2018)
d. Squamous epithelium occludes the mouth of the glands
2. a a. Lipoma b. Endometrial carcinoma
 [Ref: Shaw’s Textbook of Gyanecology 16th/e p. 172]
Es
3. a c. Adenomyosis d. Leiomyoma
4. c
5. b
BENIGN LESIONS OF UTERUS
6. a FIBROID
7. b
iM

8. b 4. Treatment of choice of a 28 weeks pregnant female with

9. b pain due to fibroid of fundus of size 10 × 10cm is? 
 (PGMEE Pattern 2015)
10. c
a. Myomectomy b. Hysterectomy
11. a c. Conservative d. Arterial embolization
PR

12. d
 [Ref: D.C Dutta text book of gynaecology 7th ep.224]
13. d
14. b 5. Red degeneration of fibroid is seen in:- (PGMEE 2015)
15. b
a. First trimester b. Second trimester POLYPS
c. Third trimester d. Any of the above
BENIGN LESIONS OF OVARY
 [Ref: D.C Dutta text book of gynaecology 7th ep.224]
6. Drugs given for fibroid-  (PGMEE 2013-14) 13. Ovarian fibroma develops from? (PGMEE Pattern 2015)
a. GnRH agonist b. Multivitamins a. Germ cells b. Sex cord
c. Mesoprost d. Isoxsuprine hydrochloride c. Epithelium d. Stroma
[Ref: D.C Dutta text book of gynaecology 7th ep. 229; Shaw’s  [Ref: D.C Dutta text book of gynaecology 7th ep.237]
Gynae 15th/e p. 359]
14. Most common complication of dermoid cyst is-
7. Red degeneration of fibroid is seen in-  (PGMEE 2013-14)  (PGMEE 2015)
a. Early pregnancy b. Mid pregnancy a. Malignant degeneration b. Torsion
c. Pueperium d. Nulliparous women c. Cyst Rupture d. None of the above
 [Ref: D.C Dutta text book of gynaecology 7th ep.224] [Ref: Shaw’s Gynaecology 16th/e p. 439, William’s
Gynaecology 2nd/e p. 267]
8. Torsion of fibroid depends upon-  (PGMEE 2012-13)
a. Infection b. Site of origin 15. Meig syndrome is associated with-  (PGMEE 2013-14)
c. Both d. None a. Brenner tumor b. Fibroma
[Ref: D.C Dutta text book of gynaecology 7th e p.225; Shaw’s c. Choriocarcinoma d. Teratoma
828
Gynae 15th/e p. 355-356] [Ref: D.C Dutta text book of gynaecology 7th ep.241;Shaw’s
Gynae 15th/e p. 381]
 CHAPTER 5: PREMALIGNANT AND MALIGNANT DISORDERS IN GYNAE
CERVIX
1. Most common cause of Cervical neoplasia is?
 (PGMEE 2015)
a. HPV-6 b. HPV-11
c. HPV-16 d. HHV
[Ref: https://aidsinfo.nih.gov/guidelines/html/]
2. 100/0/0 maturation index denotes (PGMEE 2015)
a. Atrophic smear b. Pregnancy
c. Reproductive age female d. None
[Ref: Dutta Gyne 4th ed:105]
3. Most common chemotherapeutic agent used in cervical
cancer:- (PGMEE 2016-17)
a. Etoposide b. Cisplatin
c. Ironotecan d. Methotrexate
[Ref: Harrison’s 19th/e pg. 596]
,2
4. How many years once pap smear done in female accoprding
to ACOG 2013 guidelines:- (PGMEE 2016-17)
a. Cervical cancer screening should start at age 21 yrs
b. Women 21-29 yrs pap test every 3 yrs
c. Women 30-65 paptest and HPV test (cotesting]every 5yrs
Es
or pap alone every 3yrs
d. None
 [Ref: Internet]
5. Gardasil vaccine used to prevent:- (PGMEE 2016-17)
a. Ca of cervix b. Breast ca
iM
c. Colon ca d. Ovary ca
 [Ref: D.C Dutta text book of gynaecology 7th e p268]
6. Gardasil vaccine for HPV contains which strains of virus?
 (PGMEE 2015)
PR
a. HPV 6, 11 b. HPV 6, 11, 16, 18
c. HPV 16, 18 d. HPV 16, 18, 31, 33
 [Ref: Shaw’s Gynaecology 16th/e p. 495]
7. Treatment options for CIN III include all of the following
except- (PGMEE 2015)
a. Hysterectomy b. Wertheim’s hysterectomy
c. LLETZ d. Conization
 [Ref: Shaw’s Gynaecology 16th/e p. 494]
8. 40 yr female with abnormal cervical cytology, pap smear
suggestive of CIN III, next step in management is-
a. Surgery with adjuvant chemoradiation  (PGMEE 2015)
b. Colposcopy and biopsy
c. Test for HPV and follow up after 3 months
d. Hysterectomy [Ref: Shaw’s Gyanecology 16th/e p. 485]
9. Green filter is used to visualize the following in colposcopy:-
a. Necrotic areas  (PGMEE 2015)
b. Measure actual size of lesion
c. Vascular pattern of cervix
d. None of the above
[Ref: Advanced health assessment of women By Helen 2nd/e
p. 531]
10. Magnification obtained by colposcopy is- 
 (PGMEE 2013-14)
a. 1-2 times b. 2-5 times
c. 15-25 times d. 10-20 times
 [Ref: D.C Dutta text book of gynaecology 7th e p.93]
11. A 35-year-old multiparous woman came with post coital
bleeding. What is the next step.? (AIIMS Nov’ 2017)
a. Pap smear b. Cryotherapy
c. Cone biopsy d. 4 quadrant biopsy
 Ref: Berek and Hacker’s G. Oncology 15th e p.
Explanation
ƒƒ Pap smear is the initial screening method for pre invasive
/e
cervical lesions.
ƒƒ After getting pap smear report, the patient can undergo
HPV DNA testing, colposcopy, or may require to repeat the
Pap smear.
ƒƒ If on initial examination the patient has a grossly visible
lesion, cervical biopsy may be taken at the junction of the
lesion and normal cervical tissue. 1. c
ƒƒ Cryotherapy: Women with persistent cervical discharge in 2. a
whom pre invasive lesions have been ruled out, who have 3. b
received antibiotic therapy. 4. b
ƒƒ Cone Boiopsy (Conization): Performed in women with 5. a
cervical intra epithelial neoplasia as a diagnostic procedure 6. b
to rule out invasive cancer which cannot be seen with a 7. b
simple biopsy.
8. b
ƒƒ 4 quadrant biopsy: Done in colposcopy with abnormal
9. c
findings (diffuse aceto-white or reduced iodine uptake
areas) but no definite lesion. Colposcopy should be done in 10. c
women with unexplained post coital bleeding or discharge 11. a
not responding to antibiotic therapy. 12. b
12. A 38 year old female’s pap smear shows HSIL. What will 13. c
be the next step?  (PGMEE 2013) 14. a
a. Repeat pap
b. Colposcopy
c. Currentage
d. Hysterectomy
 [Ref: Shaw’s Gyanecology 16th/e p. 485]
13. PAP smear from cervix shows HSIL grade CIN, next step
is- (PGMEE 2012-13)
a. Coagulation b. Conization
c. Colposcopy & biopsy d. Cryosurgery
[Ref: D.C Dutta text book of gynaecology 7th e p.267; Shaw’s
Gynae 15th/e p. 470]
14. Cervical cytology smear reveal CIN2- next step- 
 (PGMEE 2012-13)
a. Colposcopy b. Cryocautery
c. Hysterectomy d. Laser ablation
[Ref: D.C Dutta text book of gynaecology 7th e p.267; Shaw’s
829
Gynae 15th/e p. 404]
 PRIMES (Volume II)

15. HPV infects first which cells in cervix-  (PGMEE 2012-13) 22. First symptom of vulval cancer is- (PGMEE Pattern 2015)
a. Basal and parabasal cells a. Ulcerated lesion b. White discharge
b. Corneal cells c. Mass in the groin d. Pruritis
c. Granulosa cell d. Spinus cells [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P275;Shaw’s
[Ref: D.C Dutta text book of gynaecology 7th e p.265;Shaw’s Gyane 16th/e p. 475;berek & Novak’s Gyanecology 15th/e p.
Gynae 15th/e p. 84; Internet] 1432]
16. Unsatisfactory colposcopy refes to failure to visualize: 23. Vulval carcinoma accounts for what percentage of genital
 (PGMEE 2010) tract malignancies? (PGMEE 2015)
a. Fallopian tubes b. Transformation zone a. 0.5-1% b. 3-5%
c. Fornices d. Ectocervix c. 7-11% d. 13-15%
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P94;Shaw’s  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P274]
Textbook of Gynaecology, 15th/e, P. 85, 403, 497-499; Berek
24. Uethral involvement of Vulval carcinoma without inguino
& Novak’s Gynecology, 15th/e, P. 575-579
femoral nodes is seen in? (PGMEE Pattern 2015)
a. Stage I b. Stage II
UTERUS c. Stage III d. Stage IV
17. Treatment of simple hyperplasia of endometrium is-  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P276]

/e
 (PGMEE 2013-14)
a. Progesterone b. Estrogen 25. Uethral involvement of Vulval carcinoma with inguino
c. Hysterectomy d. Cryosurgery femoral nodes is seen in? (PGMEE Pattern 2015)
a. Stage I b. Stage II
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P271]

,2
c. Stage III d. Stage IV
CARCINOMA VULVA  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P276]
15. a 26. Treatement of choice of early vulvar carcinoma is?
18. Most common malignancy of vulva is- (PGMEE 2015)
16. b  (PGMEE Pattern 2015)
a. Squamous cell carcinoma
Es
17. a b. Melanoma a. Radiotherapy b. Hormone therapy
18. a c. Bartolin’s gland tumor c. Radical vulvectomy d. Chemotherapy
19. c d. Adenocarcinoma  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P277]
20. b [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P275; Shaw’s
21. b Gyane 16th/e p. 478; Berek & Novak’s Gyane 15th/e p. 1432] CARCINOMA VAGINA
iM

22. d 19. Predisposing factor for Carcinoma vulva are all except: 27. Sarcoma botryoides in vagina is seen in which age-
23. b a. Smoking  (PGMEE 2015)  (PGMEE 2012-13)
24. b b. Human papilloma virus (HPV) infection a. Less than 8 yrs b. 8-16 yrs
c. Fibroepithelial polyps c. 16-24 yrs d. > 24 yrs
25. c
d. Leukoplakia  [Ref: D.C Dutta text book of gynaecology 7th e p.323]
PR

26. c
27. a  [Ref: Robbins 9 /e pg. 997]
th
28. Sarcoma botryoides is a type of? (PGMEE 2015)
28. a 20. Most common extra-mammary site of Paget’s disease:- a. Embryonal Rhabdomyosarcoma
(PGMEE 2018) b. Pleomorphic Rhandomyosarcoma
29. d
a. Vagina b. Vulva c. Leimyoma
30. c d. Rhabdomyoma
c. Uterus d. Fallopian tubes
 Ref : D.C. Dutta text book of gynecology p.212 7th e  [Ref: Robbins 9th/e pg. 1222]

Explanation CARCINOMA CERVIX

ƒƒ Extramammary pagets disease of vulva is a rare condition
seen in postmenopausal female. It present with pruritus.
On examination, the lesion appears florid ,eczematous
with erythem and excoriation. It can be associated with
underlying adenocarcinoma of GI tract, urinary tract and
the breast. Treatment is surgical excision.
21. True regarding vulval cancer is all except- (PGMEE 2015)
a. HPV virus is causative factor
b. Iliac group of nodes are primary site of metastasis
c. Smoking is a risk factor
d. Vulectomy is the treatment for Bowen’s disease
830 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P275-6;
Shaw’s Gyanecology 16th/e p. 478]
29. Ca cervix risk factors are all except:- (PGMEE 2016-17)
a. Early sexual life b. Multiple children
c. Multiple sexual partner d. Nulliparity
 [Ref: D.C Dutta text book of gynaecology 7th e p 264]
30. During radiation therapy of cervical cancer which LN is/
are excluded (PGMEE 2016)
a. External iliac
b. Common iliac
c. Inguinal
d. Sacral (not mentioned anywhere]
e. Obturator
 [Ref: D.C Dutta text book of gynaecology 7th e p422]
Gynecology  Chapter 5 Premalignant and Malignant Disorders in Gynae (MCQs)

31. Radiotheraphy (external beam) at point A in Ca Cervix
requires:- (PGMEE 2016-17)
a. 4500 cGy b. 5500 cGy
c. 6500 cGy d. 7500 cGy
 [Ref: D.C Dutta text book of gynaecology 7th e p 289]
32. Staging of cervical cancer is mainly by- (PGMEE 2015)
a. CT Scan b. Exploratory laparotomy
c. MRI d. Clinical staging
 [Ref: Shaw’s Gyanecology p. 399]
33. Agent associated with carcinoma cervix- (PGMEE 2015)
a. Herpes virus
b. Calymmatobacterium granulomatis
c. Human Papilloma Virus
d. Trichomoniasis
 [Ref: Shaw’s Gynaecology 16th/e p. 495]
40. Most common presentation of cervical cancer is-
 (PGMEE 2013-14)
a. Deep pelvic pain b. Rectal pain
c. Bleeding per vaginum d. Weight loss
[Ref: D.C Dutta text book of gynaecology 7th e p.284; Shaw’s
Gynae 15th/e p. 409]
41. Radiation point A in ca cervix-  (PGMEE 2013-14)
a. 8000 cGy b. 6000 cGy
c. 10000 cGy d. 4000 cGy
[Ref: D.C Dutta text book of gynaecology 7th e p.289; Shaw’s
Gynae 15th/e p. 436]
42. Radical hysterectomy in stage 1b ca cervix better than ra-
diotherapy all are true except-  (PGMEE 2013-14)
a. Chance of survival more
b. Chance of recurrence less
c. Ovary function can be preserved

/e
34. Most common carcinoma in pregnancy is- (PGMEE 2015) d. Less complicated
a. Oropharynx b. Colon
[Ref: D.C Dutta text book of gynaecology 7th e p.290; Shaw’s
c. Ovary d. Cervical
Gynae 15th/e p. 413]
[Ref: DeSwiet’s Medical Disorders in obstetric practice 5th/e

,2
p. 553] 43. Dose of radiation for early and locally advanced cancer
cervix at point A during brachytherapy- (AIIMS Nov 2013)
35. Most common gynecological malignancy in affluent obese a. 70-75 Gray and 75-80 Gray
women is:-  (PGMEE 2015) 31. d
b. 75-80 Gray and 80-85 Gray
a. Ovarian b. Cervical c. 80-85 Gray and 85-90 Gray 32. d
Es
c. Vaginal d. Endometrial d. 85-90 Gray and 90-95 Gray 33. c
[Ref: Cancer Rehabilitation: Principles and practice by [Ref: Radiation Oncology/Cervix/Bachytherapy (wikiPedia], 34. d
Michael O’ Dell p. 279] Berek’s and Novak’s 15th/e] 35. d
36. What is the stage of carcinoma cervix involving body of 44. All of the following are risk factors of cervical cancer 36. a
uterus- (PGMEE 2015) except- (AIIMS Nov 2013) 37. c
iM

a. Stage I b. Stage II a. Low parity b. Multiple partners 38. d

c. Stage III d. Stage IV c. Early sexual intercourse d. Smoking 39. a
 [Ref: Shaw’s Gynaecology 16th/e p. 501] [Ref: D.C Dutta text book of gynaecology 7th e p.285; Berek’s 40. c
37. Most common cause of death in cervical cancer is- and Novak’s 15th/e] 41. a
PR

a. Metastasis to vital organs  (PGMEE 2015) 45. Drug of choice for chemotherapy of cervical cancer is? 42. a
b. Haemorrhage  (PGMEE 2013) 43. c
c. Renal infection a. Cisplatin b. Vincristine
d. Infection 44. a
c. Cyclophosphamide d. Etoposide
45. a
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P282] [Ref: D.C Dutta text book of gynaecology 7th e p.289; Berek
46. c
38. Risk factor for cervical carcinoma is- (PGMEE 2015) & Novak’s gynae 14th/e p. 1431]
47. d
a. Low socioeconomic status 46. Management of stage IB1 cancer cervix in a 34 weeks
b. Smoking pregnant female is? (PGMEE 2013)
c. Human papilloma virus a. Vaginal delivery followed by chemotherapy
d. All of the above b. Classical caesarean followed by chemotherapy
 [Ref: Shaw’s Gyanecology 16th/e p. 495] c. Classical caesarean with radical hysterectomy at delivery
d. Classical caesarean followed by radical hysterectomy after
39. Organism causing cervical carcinoma is? (PGMEE 2014)
6 weeks
a. HPV b. HAV
c. HBV d. HCV [Ref: D.C Dutta text book of gynaecology 7th e p.291; Novak’s
gynae 14th/e p. 1439; Shaw’s Gynae 13th/e p. 390]
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P262;
47. In FIGO staging of cancer cervix investigation includes all
Jeffcoate’s Principles of Gynaecology, 8th/e, P. 387-388,
except- (PGMEE 2012-13)
392, 436 Berek & Novak’s Gynecology, 15th/e, P. 580-581;
a. CT scan b. PET scan
Progress in Obstetrics & Gynaecology, John Studd, Vol. 18,
c. MRI d. Laparoscopy
Ch. 19, P. 288-290]
[Ref: Shaw’s Gynae 15th/e p. 409; D.C Dutta text book of 831
gynaecology 7th e p.281]
 PRIMES (Volume II)

48. Cervical carcinoma is due to HPV-  (PGMEE 2012-13)
a. 6, 8 b. 16, 18
c. 30, 32 d. 15, 17
[Ref: D.C Dutta text book of gynaecology 7th e p.265; Shaw’s
Gynae 15th/e p. 400]
49. What are the strains included in nonavalent HPV vaccine.
a. 6, 11, 16, 18, 31, 33, 45, 52, 58  (AIIMS Nov’ 2017)
b. 6, 11, 16, 18, 31, 33, 58, 72, 73
c. 7, 11, 16, 18, 31, 45, 58, 72, 73
d. 6, 11, 16, 18, 31, 33, 58, 45, 103
Ref: Gardasil 9 protects against additional HPV TYPES
www.cancer.gov
Explanation
ƒƒ Cervarix is a bivalent vaccine: –16, 18
ƒƒ In December 2014 FDA approved the 9-valent vaccine
56. Ca cervix staging done by-  (PGMEE 2012-13)
a. CT b. MRI
c. Clinical findings d. Histopathology
[Ref: D.C Dutta text book of gynaecology 7th e p.281; Shaw’s
Gynae 15th/e p. 409]
57. Treatment of stage IIB of cervix is-  (PGMEE 2012-13)
a. Chemoradiation b. Hysterectomy
c. Radiation d. Chemotherapy
[Ref: D.C Dutta text book of gynaecology 7th e p.290;Shaw’s
Gynae 15th/e p. 415]
58. Radiation to point A in cervix is-  (PGMEE 2012-13)
a. 8000 rad b. 6000 rad
c. 10000 rad d. 4000 rad
[Ref: D.C Dutta text book of gynaecology 7th e p.289;Shaw’s
Gynae 15th/e p. 436]

Gardasil 9 which protects against infection with HPV types
6,11,16,18,31,33,45,52 and 58
50. Staging of ca cervix with endometrial involvement-
 (PGMEE 2012-13)
/e
59. A women is diagnosed with cervical carcinoma stage 2b,
treatment of choice is-  (PGMEE 2012-13)
a. Hysterectomy b. Chemoradiation
c. Primary radiation d. None of the above

48.
49.
b
a


a. Stage 1
c. Stage 3
b. Stage 2
d. Stage 4

,2
[Ref: D.C Dutta text book of gynaecology 7th e p.282; Shaw’s
Gynae 15th/e p. 412]
[Ref: D.C Dutta text book of gynaecology 7th e p.290;Shaw’s
Gynae 15th/e p. 414]
60. In Brachytherapy for Carcinoma cervix, dose of radiation
at point A is? (PGMEE 2013)
Es
50. b 51. FIGO staging with cervical carcinoma involving parame- a. 1000 rad b. 4000 rad
51. b trium- (PGMEE 2012-13) c. 8000 rad d. 10000 rad
52. d a. IIa b. IIb
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P289;
53. c c. IIIa d. IIIb
Novak’s gynaw 13th/e ch. 31]
54. c [Ref: D.C Dutta text book of gynaecology 7th e p.282; Shaw’s
61. Rate limiting dose of radiotherapy to rectum and bladder
iM

Gynae 15th/e p. 412]

55. c in gynaecologic malignacies-  (PGMEE 2012-13)
56. c 52. Hydronephrosis is seen in which stage of Ca cervix- a. 4000cGy b. 5000cGy
57. a  (PGMEE 2012-13) c. 6000cGy d. 7000cGy
a. 2a b. 2b
58. a  [Ref: Shaw’s Gynae 15th/e p. 438]
c. 3a d. 3b
PR

59. b
[Ref: D.C Dutta text book of gynaecology 7th e p.282; Shaw’s 62. Ca cervix IIIB is treatment of choice is- (PGMEE 2012-13)
60. c a. Radiotherapy b. Chemotherapy
Gynae 15th/e p. 412]
61. b c. Chemoradiation d. Surgery
53. Treatment of stage IIIB in Ca cervix is- (PGMEE 2012-13)
62. c [Ref: D.C Dutta text book of gynaecology 7th e p.290; Shaw’s
a. Wertheim’s hystrectotomy
63. c b. Mitra operation Gynae 15th/e p. 414]
64. c c. Chemoradiation 63. Vaccine preventable cancer is? (PGMEE 2012)
d. Primary radiotherapy a. Ovarian cancer b. Breast cancer
[Ref: D.C Dutta text book of gynaecology 7th e p.290;Shaw’s c. Cancer cervix d. Endometrial cancer
Gynae 15th/e p. 414] [Ref: D.C Dutta text book of gynaecology 7th e p.268; Novak’s
54. Carcinoma cervix screening are all except-  gynae 15th/e Chapter 19 p. 581]
 (PGMEE 2012-13) 64. A case of carcinoma cervix who earlier received
a. PAP smear b. Liquid based cytology radiotherapy, relapses with new lesion, what should be the
c. CT and MRI d. Acetowhite areas next line of management? (PGMEE 2010)
[Ref: D.C Dutta text book of gynaecology 7th e p.91-95;Shaw’s a. Repeat radiotherapy
Gynae 15th/e p. 402-403] b. Complete hysterectomy
55. Cervical cancer 4mm deep invasion and 7 mm spread, c. Pelvic exenteration
stage is- (PGMEE 2012-13) d. Chemotherapy
a. IA b. IA 1 [Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 415, 438-
c. IA 2 d. IB 439 Jeffcoate’s Principles of Gynaecology, 8th/e, P. 443-444,
832
[Ref: D.C Dutta text book of gynaecology 7th e p.282;Shaw’s 449-450]
Gynae 15th/e p. 412]
Gynecology  Chapter 5 Premalignant and Malignant Disorders in Gynae (MCQs)
65. About vaccination for carcinoma cervix, all are true
EXCEPT: (PGMEE 2010)
a. Does not require further examinations
b. Administered intramuscularly at 0, 2 and 6 months
c. Bivalent vaccine is more efficient than Quadrivalent
d. Can be taken with other live vaccines at different site
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P268;
Shaw’s Textbook of Gynaecology, 15th/e, P. 406-407 Berek
& Novak’s Gynecology, 15th/e, P. 581-582; Progress in
Obstetrics & Gynaecology, John Studd, Vol. 18, Ch. 19, P.
293-295]
66. Most commonly associated human papilloma virus with
cancer cervix is? (PGMEE 2011)
a. HPV 16 b. HPV 24
c. HPV 32 d. HPV 36
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P265]
67. Koilocytes with perinuclear halo on pap smear is pathog-
nomonic of? (PGMEE 2010)
a. HPV infection b. Bacterial vaginosis
,2
c. Dysplasia d. Metaplasia
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P265]
68. Management of stage IIB cancer cervix is? (PGMEE 2009)
a. Hysterectomy
b. Radiotherapy in combination with chemotherapy
Es
c. Chemotherapy
d. Radiotherapy
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P290]
CARCINOMA ENDOMETRIUM
iM
69. Which histopathological variant of endometrial malignan-
cy has the poorest prognosis:- (PGMEE 2016-17)
a. Clear cell carcinoma
b. Adenocarcinoma
PR
c. Adenosquamous CA
d. Mucinous adenocarcinoma
 [Ref: D.C Dutta text book of gynaecology 7th e p293]
70. Most common histological subtype of cancer endometrium
is?  (PGMEE Pattern 2015)
a. Endometrioid adenocarcinoma
b. Mucinous carcinoma
c. Clear cell carcinoma
d. Squamous cell carcinoma
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P293]
71. Treatment of stage –I endometrial carcinoma is?
 (PGMEE Pattern 2015)
a. Radiotherapy b. Chemotherapy
c. Hormone therapy d. Surgery
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P296]
72. Most common histological variety of uterine carcinoma is?
 (PGMEE 2014)
a. Squamous cell carcinoma b. Columnar cell carcinoma
c. Adeno carcinoma d. Mixed carcinoma
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P293]
73. Serous carcinoma of endometrium is associated with which
mutation? (PGMEE Aug 13 Pattern)
a. p53 b. PTEN
c. K ras d. p 16k
[Ref: D.C Dutta text book of gynaecology 7th e p.294; Robbin’s
8th/e p. 1034]
74. Carcinoma endometrium is not associated with- 
 (PGMEE 2011-2013)
a. Fibromyoma b. Dysgerminoma
c. Granulosa cell tumor d. Endometrial hyperplasia
[Ref: D.C Dutta text book of gynaecology 7th e p.294; Shaw’s
Textbook of Gynaecology, 15th/e, P. 378, 380, 416-418; Jeffcoate’s
Principles of Gynaecology, 8th/e, P. 473, 500, 501, 507]
75. Staging of endometrial ca with involvement of paraaortic
lymph node involvement- (PGMEE 2012-13)
a. IIIC b. IIB
/e
c. IIIA d. IIIB
[Ref: D.C Dutta text book of gynaecology 7th e p.295Shaw’s
Gynae 15th/e p. 420]
76. Endometrial carcinoma involving cervix, stage is-
 (PGMEE 2012-13)
a. 1 b. 2 65. a
c. 3 d. 4
66. a
[Ref: D.C Dutta text book of gynaecology 7th e p.295 Shaw’s
67. a
Gynae 15th/e p. 420]
68. b
77. The following are risk factors for carcinoma endometrium 69. a
EXCEPT- (PGMEE 2012)
70. a
a. Early menopause b. Endometrial cell carcinoma
c. Nulliparity d. Obesity 71. d
72. c
[Ref: D.C Dutta text book of gynaecology 7th e p.294;Shaw’s
Gynecology 15th/e p. 420] 73. a
74. b
78. A 53-year post-menopausal female, with endometrial
hyperplasia with atypia what is the management. 75. a
 (AIIMS Nov’ 2017) 76. b
a. Hysterectomy (Type I) b. Progesterone 77. a
c. MIRENA d. Endometrial 78. a
 [Ref: Dutta 7th e p.271-272]
Explanation
ƒƒ In young premenopausal patients with endometrial
hyperplasia
○○ Without atypia : cyclic progestogen therapy for 6 to 9
months
○○ With atypia : therapy with progestin for 6 to 9 months.
Periodic endometrial sampling every 3 months is
essential. Hysterectomy is best treatment at any age
with atypical endometrial hyperplasia because of risk of
invasive cancer.(ACOG)
ƒƒ In perimenopausal and postmenopausal women
○○ Hyperplasia with atypia : hysterectomy with bilateral
salpingo-oophorectomy.
○○ Hyperplasia without atypia : continuous progestin
therapy may be considered. However , hysterectomy with
bilateral salpingo-oophorectomy is done as an alternative
as isk of carcinoma increases with age. 833
 PRIMES (Volume II)
CARCINOMA OVARY
79. M/C ovarian cancer with menorrhagia:(PGMEE 2016-17)
a. Sex cord tumors b. Germ cell tumors
c. Epithelial cell tumors d. None
 [Ref: D.C Dutta text book of gynaecology 7th e p316-317]
80. AFP is a marker of:- (PGMEE 2016-17)
a. Liver cancer b. Yolk sac cancer
c. Pancreas cancer d. Dysgerminoma
 [Ref: D.C Dutta text book of gynaecology 7th e p 316]
81. All are Sex cord stromal tumor except:- (PGMEE 2016)
a. Granulosa cell tumor b. Leydig cell tumor
c. Yolk sac tumor d. Thecoma
 [Ref: D.C Dutta text book of gynaecology 7th e p 316]
82. Call exner body seen in:- (PGMEE 2012-17)
a. Granulosa cell tumor b. dysgerminoma
c. Volk sac tumor d. Sertoli-leydig cell tumor
 [Ref: D.C Dutta text book of gynaecology 7th e p 316]
,2
83. Call Exner bodies are seen in:  (PGMEE 2012-17)
a. Dysgerminoma b. Mucinous cystadenoma
c. Sex cord stromal tumour d. Yolk sac tumour
79. a
[Ref: D.C Dutta text book of gynaecology 7th e p 316 ; Robbins
80. b 9th/e pg. 1029]
Es
81. c
84. A lady with abdominal mass was investigated. On surgery,
82. a
she was found to have b/l ovarian masses with smooth surface.
83. c
On microscopy they revealed mucin secreting cells with signet
84. b ring shapes. What is your diagnosis? (AIIMS May 2015)
85. d a. Dysgerminoma
iM
86. d b. Krukenberg tumour
c. Primary Adenocarcinoma of the ovaries
87. a
d. Dermoid cyst
88. a
[Ref: Robbins Pathology 9th Ed/Pg 1032; Robbins Pathology
89. a
8th ED/PG 1050; Shaw’s Textbook of Gynaecology 15/e pg 425]
PR
90. b
85. One of the following is a germ cell tumor of ovary:
91. c
 (PGMEE 2015-2016)
92. b
a. Granulosa cell tumor b. Mucinous cystadenoma
93. c c. Brenner tumor d. Benign cystic teratoma
94. d  [Ref: Robbins 9th/e pg. 1023]
95. c
86. Rokitansky protuberance is associated with which of the
96. c
following tumor of ovary: (PGMEE 2016-17)
97. a a. Dysgerminoma b. Mucinous cystadenoma
c. Sex cord stromal tumour d. Mature teratoma
 [Ref: Robbins 9th/e pg. 1029]
87. Which of these tumors is unique to pregnancy?
 (PGMEE 2015)
a. Luteoma b. Serous cystadenoma
c. mucinous cystadenoma d. Teratoma
 [Ref: Robbins 9th/e pg. 1044-1070]
88. Most common ovarian tumor in pregnancy is?
 (PGMEE 2015)
a. Dermoid cyst b. Serous cysyadenoma
834 c. Gonadoblastoma d. Theca cell tumor
 [Ref: Shaw’s 16th/e p. 439]
89. Not an epithelial cancer of ovary- (PGMEE 2015)
a. Granulosa cell tumor b. Choriocarnioma
c. Teratoma d. Endodermal sinus tumor
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P237]
90. AFP is raised in- (PGMEE 2015)
a. Choriocarcinoma b. Endodermal sinus tumor
c. Teratoma d. Dysgerminoma
[Ref: D.C.Dutta’s Textbook of gynaecology, 7th/e, P315;Shaw’s
Textbook of Gynaecology 16th/e p. 522]
91. Sex cord ovarian tumors are all except- (PGMEE 2015)
a. Thecoma
b. Sertoli – Leydig cell tumors
c. Teratoma
d. Granulosa cell tumors
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P237]
/e
92. Torsion as a complication is seen with which tumor-
 (PGMEE 2015)
a. Dysgerminoma b. Teratoma
c. Serous cystadenoma d. Granulosa cell tumor
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P240;
Shaw’s Gynaecology 16th/e p. 445; Break and Novak’s
Gynaecology 15th/e p. 1457]
93. Cut-off value for Ca-125 is? (PGMEE Pattern 2015)
a. 15 units/ml b. 25 units/ml
c. 35 units/ml d. 45 units/ml
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P311
94. Which of the following ovarian tumors is most radiosensi-
tive- (PGMEE 2015)
a. Brenner tumor b. Carcinoid
c. Serous CystadenoCA d. Dysgerminoma
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P315;
Shaw’s Gynaecology 16th/e p. 441]
95. Bilateral ovarian carcinoma + capsule involvement + ascites
+ paraaortic LN. Identify the stage:-  (PGMEE 2013-14)
a. 1C b. 2C
c. 3C d. 4C
[Ref: D.C Dutta text book of gynaecology 7th e p307; Shaw’s
Gynae 15th/e p. 427]
96. Best to defect early ovarian Ca- (PGMEE 2013-14)
a. CT b. TCS
c. USG d. X-ray pelvis
[Ref: D.C Dutta text book of gynaecology 7th e p 310; Shaw’s
Gynae 15th/e p. 427]
97. 51 yr f with abdominal mass & ascites. On H/P ovarian Ca
is +ve for- (PGMEE 2012-13)
a. Ca 125 b. Ca 19-9
c. AFP d. hCG
[Ref: D.C Dutta text book of gynaecology 7th e p311; Harrison
16th/e p. 554; Novak’s 15th/e p. 1371]
Explanation
ƒƒ Women with BRCAI gene mutation have 45% lifetime risk
of ovarian cancer and those with BRCAII mutation have
25% risk.
Gynecology  Chapter 5 Premalignant and Malignant Disorders in Gynae (MCQs)
ƒƒ These women should be screening annually with TVS
and CA 125 testing, and prophylactic oophorectomy is
recommended by age 35year whenever child bearing is
completed b/c of the high risk of disease.
ƒƒ Confusion clears after reading these lines :“For patients
with genetic mutations predisposing them to increase risk,
prophylactic oophorectomy is performed with or without
mastectomy “as well”.
98. Ovarian cancer drugs used are- (PGMEE 2012-13)
a. Paclitaxel + doxorubicin b. Cisplatin + doxorubicin
c. Docetaxel + doxorubicin d. Paclitaxel + cisplatin
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P312;
Shaw’s Gynae 15th/e p. 442]
99. Chemotherapy for epithelial ovarian carcinoma - 
 (PGMEE 2012-13)
a. BEC b. CHOP
c. MOPP d. None of the above
 [Ref: Shaw’s Gynae 15th/e p. 442]
 [Ref: D.C Dutta text book of gynaecology 7th e p312]
,2
100. Ca ovary with left supraclavicular LN, stageis- 
 (PGMEE 2012-13)
a. IV b. III
c. II d. I
[Ref: D.C Dutta text book of gynaecology 7th e p307;Shaw’s
Es
Gynae 15th/e p. 427]
101. Most common malignant ovarian tumor-
a. Dysgerminoma  (PGMEE 2012-13)
b. Germ cell tumour
c. Serous cystadenocarcinoma
iM
d. Yolk sac tuomour
[Ref: D.C Dutta text book of gynaecology 7th e p305;Shaw’s
Gynae 15th/e p. 374]
102. Pseudomyxoma peritonei is seen in which ovarian tumor-
PR
 (PGMEE 2012-13)
a. Dysgerminoma b. Germ cell tumour
c. Mucinous cystadenoma d. Serous cystadenoma
[Ref: D.C Dutta text book of gynaecology 7th e p305;Shaw’s
Gynae 15th/e p. 374]
Explanation
ƒƒ Pseudomyxoma peritonei 111. Bilateral ovarian carcinoma with breech in capsular wall
Seen in mucinous cystadenoma .If tumour ruptures ,it may
lead to formation of pseudomyxoma peritonei & the viscera
show extensive adhesions.
ƒƒ Other causes are----
○○ Rupture of mucocele of appendix
○○ Mucinous ovarian cysts
○○ Mucin secreting intestinal adeno carcinoma
○○ Mucin secreting ovarian adenocarcinoma
(Mucinous cystadenoma)
○○ Colloid carcinoma of stomach/ colon
103. Initial drug for ovarian cancer-  (PGMEE 2012-13)
a. Carboplatin b. Doxorubicin
c. Ifosfamide d. Methotrexate
[Ref: D.C Dutta text book of gynaecology 7th e p312;Shaw’s
Gynae 15th/e p. 442]
104. True about dysgerminoma- (PGMEE 2012-13)
a. Highly aggressive
b. Managed conservatively in young girls
c. Usually seen in old patients
d. Secrets male sex hormones
[Ref: D.C Dutta text book of gynaecology 7th e p314-15;
Shaw’s Gynae 15th/e p. 378-379]
105. AFP is increased in which ovarian tumor-(PGMEE 2012-13)
a. Choriocarcinoma b. Granulose cell tumour
c. Sarcoma d. Endodermal sinus tumour
[Ref: D.C Dutta text book of gynaecology 7th e p315;Shaw’s
Gynae 15th/e p. 424]
106. Age group for dysgerminoma is-  (PGMEE 2012-13)
a. 10-20 yrs b. 30-40 yrs
c. 50-60 yrs d. > 60 yrs
[Ref: D.C Dutta text book of gynaecology 7th e p314: Shaw’s
/e
Gynae 15th/e p. 378]
107. MC ovarian tumors originate from- (PGMEE 2012-13)
a. Mullerian epithelium b. Stroma
c. Germ cells d. Connective tissue
[Ref: D.C Dutta text book of gynaecology 7th e p305;Shaw’s
text book of gynaecology 15th/e p. 373] 98. d
108. Recent biomarker for early detection of surface epithelial 99. d
tumours of ovary- (PGMEE 2012-13) 100. a
a. Ca 125, b. Ca 19-9 101. c
c. Ca 15-5 d. Osteopontin 102. c
[Ref: D.C Dutta text book of gynaecology 7th e p311; Harrison 103. a
16th/e p. 554; Novak’s 15th/e p. 1371]
104. b
109. A unilateral ovarian tumor spreads to peritoneum but not 105. d
to uterus. Its stage would be? (PGMEE Aug. 12 Pattern) 106. a
a. Stage IB b. Stage IC
107. a
c. Stage IIA d. Stage IIB
108. a
[Ref: D.C Dutta text book of gynaecology 7th e p307]
109. b
110. What is the FIGO staging of carcinoma ovary with 110. a
negative nodes, limited to true pelvic microscopic implants
111. a
on peritoneal surface? (PGMEE 2011)
a. III A b. III B 112. b
c. III C d. None 113. a
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P307
with ascites peritoneal washings and positive cytology.
What is the stage of the carcinoma? (PGMEE 2010)
a. Ic b. IIc
c. IIIc d. IVc
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P307
112. Most common germ cell tumor ovary is? (PGMEE 2009)
a. Dysgerminoma b. Dermoid
c. Carcinoids d. Struma ovary
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P239
113. Drug used in ovarian carcinoma? (PGMEE 2009)
a. Cisplatin b. Dacarbazine
c. Cycloposphamide d. Methotrexate 835
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P312]
 PRIMES (Volume II)

GESTATIONAL TROPHOBLASTIC DISEASE
114. Partial mole is :- (PGMEE 2016-17)
a. Diploid b. Triploid
c. Gynogenesis d. Organs are not form at all
[Ref: Williams Obstetrics 24th e p.397From ROAMS 13th/e
pg. 682]
115. Risk factors for molar gestation are all of the following
except- (PGMEE 2015)
a. Oriental countries
b. Disturbed maternal immune mechanism
c. Faculty nutrition
d. Higher ratio of maternal/paternal chromosomes
 [Ref: William’s Obs 24th/e p. 397]
116. Common misdiagnosis of partial mole is- (PGMEE 2015)
a. Ectopic pregnancy b. Choriocarcinoma
122. Complete mole not seen is- (PGMEE 2013-14)
a. XXX chromosome b. YYY
c. 46XX d. 46XY
 [Ref: Williams Obstetrics 24th e pg 397-398]
123. Most common presenting feature of complete mole is-
 (PGMEE 2013-14)
a. Vomiting b. Amenorrhoea
c. Headache d. Bleeding per vaginum
[Ref: Williams Obstetrics 24th e pg 398; Shaw’s Gynae 15th/e
p. 254, 255]
124. Androgenic XX chromosome is- (PGMEE 2013-14)
a. Partial mole b. Complete mole
c. Turner’s syndrome d. Stein leventhal syndrome
[Ref: Williams Obs 24th e pg 397-8 Dutta Obs 7th/e p. 191]
125. Partial mole is? (PGMEE Aug 13 Pattern)

/e
c. Complete mole d. Threatened abortion a. Haploid b. Diploid
 [Ref: Dutta’s Obstetrics 8th/e p. 230] c. Triplod d. Polyploid
[Ref: Williams Obstetrics 24th e pg 397;Danforth’s obstetrics
117. True about placental site trophoblastic disease is-
and hynecology 10th/e p. 1074; Dutta Obs 6th/e p. 200]

,2
a. Secretes human placental lactogen  (PGMEE 2015)
b. Highly Malignant behavior 126. Most common site of metastasis in choriocarcinoma is?
c. Contains syncytiotroblasts mainly  (PGMEE Aug 13 Pattern; june 2011)
114. b d. Hysterectomy followed by chemoradiation is the treatment a. Liver b. Lungs
115. d of choice. c. Ovaries d. Brain
Es
116. d [Ref: Shaw’s Gyane 16th/e p. 313; Dutta’s Obstetrics 8th/e p. 231] [Ref: D.C Dutta text book of gynaecology 7th e p300; Shaw’s
117. a 14th/e p. 233
118. A left side ovarian tumor with HCG 4 IU/L, Normal AFP,
118. a LDH raised. Which of the following is the most likely 127. Which is distinguishing feature of complete mole from
119. d diagnosis?  (AIIMS Nov’ 2017) partial mole- (PGMEE 2012-13)
120. d a. Dysgerminoma  a. Hydropic changes in proliferating villi
iM

121. a b. Endodermal germ cell tumor b. Proliferation of blood vessels into villi
122. a c. Mixed germ cell tumor c. Mutation of p-57 gene in chorionic cells
d. Teratoma[Ref: Dutta 7th e p.314-316] d. Presence of fetus
123. d
Explanation [Ref: Dutta’s Obs. 8th/e, pg. 230; Holland Brew’s Obs.4th/e,
124. b
pg. 250; Williams Obs., 23rd/e, pg. 258]
PR

125. c ƒƒ Dysgerminoma : tumor markers alpha feto orotein, HCG,

126. b lactate dehydrogenase may be positive . 128. What distinguishes placental site trophoblastic tumor
ƒƒ Endodermal sinus tumor: tumor marker alpha feto protein from choriocarcinoma- (PGMEE 2012-13)
127. a
ƒƒ Mixed germ cell tumor : tumor markers HCG and AFP a. Low β hCG
128. a ƒƒ Teratoma : tumor marker AFP others are CA125, CA 19-9, b. Low β hCG and low HPL
129. b CEA. c. High β hCG and low HPL
130. a 119. True about hydatidiform mole is:- (PGMEE 2015) d. High β hCG and high HPL
a. Highest incidence in India [Ref: Dutta’s Obs. 8th/e, pg. 231; Holland Brew’s Obs.4th/e,
b. It is principally a disease of amnion pg. 254-255; Williams Obs., 23rd/e, pg. 262]
c. Embryo is present
129. Partial mole, not true is:- (PGMEE 2012-13)
d. Vesicle fluid is rich in hCG
a. Fetus is present
 [Ref: Dutta’s Obs 7th/e p. 190] b. Diffuse trophoblastic hyperplesia
120. Hydatidiform mole follow up reliable test:-(PGMEE 2015) c. Theca lutein cysts uncommon
a. Clinical examination b. USG d. Uterine size is more than the period of amenorrhoea
c. Serum estradiol levesl d. Beta HCG [Ref: Dutta’s Obs. 8th/e, pg. 224,230; Holland Brew’s
 [Ref: Williams obstetrics 24the p.400] Obs.4th/e, pg. 250-251; Williams Obs., 23rd/e, pg. 258]

121. Poor prognostic factor for hydratidiform mole is- 130. Choriocarcinoma diagnosis is by-  (PGMEE 2012-13)
 (PGMEE 2015) a. USG b. CT
a. WHO score > 8 b. Prior molar pregnancy c. MRI d. X-RAY
c. No prior chemotherapy d. Metastasis to lung [Ref: D.C Dutta text book of gynaecology 7th e p301;Dutta
836
[Ref: Williams obstetrics 24th e p402; Dutta’s Obstetrics 8th/e Obs 7th/e p. 193]
p. 221, Shaw’s Gyanecology 16th/e p. 517]
Gynecology  Chapter 5 Premalignant and Malignant Disorders in Gynae (MCQs)

131. Karyotype of complete mole is? 135. Which of the following is bad prognostic factor for chorio-
 (PGMEE 2009, 2011,2012-13) carcinoma? (PGMEE 2011)
a. 46XX b. 46YY a. Full term pregnancy b. Short duration
c. 69 XXX d. 69 XXY c. Abortion d. Low beta hCG
[Ref: Dutta’s Obs. 8th/e, pg. 222; Holland Brew’s Obs.4th/e,  [Ref: D . C. Dutta’s Textbook of gynaecology, 7th/e, P301]
pg. 250; Williams Obs., 23rd/e, pg. 258]
136. Which of the following is a risk factor for choriocarcinoma?
132. Chemotherapy for choriocarcinoma, DOC is-   (PGMEE 2011)
 (PGMEE 2012-13) a. β hCG < 40,000 mlU/mL b. After full term pregnancy
a. Methotrexate b. Cyclophosphamide c. Lung metastasis d. None
c. Cisplatin d. Doxorubicin  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P301]
[Ref: D.C Dutta text book of gynaecology 7th e p302;Dutta
137. Most common gestational trophoblastic disease following
Obs 7th/e p. 196]
hydatidiform mole is? (PGMEE 2011)
133. High risk gestational trophoblastic disease according to a. Invasive mole
WHO is a pre treatment hCG level higher than? b. Placental nodule
 (PGMEE 2012) c. Placental site trophoblastic tumor
a. 100 IU/L b. 1000 IU/L d. Choriocarcinoma

/e
c. 10,000 IU/L d. 1,00,000 IU/L  [Ref: Williams Obs., 23rd/e, pg. 262]
[Ref: Holland Brew’s Obs.4th/e, pg. 254; Novak’s Gynae.
138. In case of hydatidiform mole, investigation used for diag-
15th/e, pg. 1464, 1469]
nosis is? (PGMEE 2011)

,2
134. Best prognostic indicator of gestational trophoblastic a. Chest X- ray b. USG
disease is: (PGMEE Nov. 12 Pattern) c. hCG titer d. All of the above
a. Uterine size b. Theca lutein cysts [Ref: Dutta’s Obs. 8th/e, pg. 225; Holland Brew’s Obs.4th/e, 131. a
c. β-hCG d. Stage of disease pg. 252; Williams Obs., 23rd/e, pg. 260]
132. a
[Ref: Holland Brew’s Obs.4th/e, pg. 254; Novak’s Gynae.
Es
15th/e, pg. 1469] 133. d
134. c
135. a
136. b
137. a
iM

138. b
PR

837
 CHAPTER 6: INFECTION OF GENITAL TRACT

ACUTE AND CHRONIC PELVIC INFECTION 6. PID is most commonly caused by-
 (PGMEE 2013; Nov.12 Pattern)
1. Most common cause of pelvic inflammatory disease is- a. Chlamydia b. Mycoplasma
a. Sexually transmitted disease  (PGMEE 2015) c. Tubercular bacillus d. E. Coli
b. Puerperal sepsis
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P107;
c. Pelvic peritonitis
Shaw’s Gynae 15th/e p. 445-446]
d. IUCD [Ref: Shaw’s 16th/e p. 177]
2. Pelvic inflammatory disease commonly complicates as:-  7. PID is not aggravated by?  (PGMEE 2012)
a. Pyometra  (PGMEE 2018) a. Cervical cap b. Diaphragm
b. Senile endometritis c. IUCD d. OCP
c. Uterine adhesions/synachiae  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P401
d. Hematocolpos
8. Gold standard for diagnosis for PID is:  (PGMEE 2010)
[Ref: D.C. Dutta text book of Gynecology pg.108 7th e; a. Clinical triad of pain, fever and cervical tenderness
 Speroff 8th e p.921-931] b. USG

/e
c. Diagnostic laparoscopy
Explanation
d. Histologic confirmation of endometritis
Pelvic Inflammatory Disease (PID): Diagnostic criteria [Ref: Shaw’s Textbook of Gynaecology, 15th/e, Page no. 449,
450;Jeffcoate’s Principles of Gynaecology, 7th/e, P. 359, 360;

1.
2.
a
a
Criteria
Minimal
criteria
Additional
Parameters

2. Cervical motion tenderness

1. Oral temp > 38.3oC
,2
1. Tenderness in lower abdomen & adenexa


Berek & Novak’s Gynecology, 15th/e, P. 565
9. Posterior colpotomy is done for?
a. Pelvic abscess
c. Pelvic haematocele
(PGMEE 2010)
b. Ovarian abscess
d. All
Es
3. a criteria 2. Mucopurulent Cx/vaginal discharge
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 453;Te
4. a 3. Leukocytes abundance in cervical smear
Linde’s Operative Gynecology, 9th/e, P. 1084 683-684
5. a 4. Raised CRP and or ESR
5. Laboratory evidence of +ve cervical infection 10. Contraindication to dilatation and curettage (D & C) :-
6. a
with gonorrhea or chlamydia trachomatis a. Abnormal uterine bleeding (AUB)  (PGMEE 2018)
7. d b. Post menopausal bleeding
iM

Definitive 1. Histopathologically evidence of endometritis

8. a c. Tuberculosis
criteria on biopsy.
9. a 2. Radiological evidence (TVS/MRI) of thickened d. Pelvic inflammatory disease
10. d fluid filled tubes  [Ref: Dutta 7th e p.484]
11. d 3. Laparoscopic evidence of PID
Explanation
PR

Complications 1. Early: Ectopic pregnancy, abortions, endome-

of PID tritis, infertility Indications of Dialatation and Curettage:
2. Late: Secondary amenorrhea, pyometra ƒƒ Diagnostic
○○ Infertility
3. PID after insertion of IUD is seen in how many weeks- ○○ AUB
 (PGMEE 2013-14) ○○ Pathologic amenorrhea
a. 3 b. 5 ○○ Endometrial tuberculosis
c. 7 d. 14 ○○ Postmenopausal bleeding
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P395] ○○ Chorionepithelioma
4. Criteria for PID is? (PGMEE Nov 13 Pattern) ƒƒ Therapeutic
a. Tubo ovarian abscess on USG ○○ AUB             ○  Removal of IUD
b. Cervical erosion ○○ Endometrial polyp     ○  Incomplete abortion
c. Temperature more than 37.5 C ƒƒ Combined
d. Infertility ○○ AUB
○○ Endometrial polyp
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,
P108;Danforth’s obstetrics and gynecology 10th/e ch. 34] GENITAL TUBERCULOSIS
5. Most common cause of pyometra- (PGMEE 2012-13) 11. Most common site involved in genital TB- (PGMEE 2015)
a. Endometritis b. CA endometrium a. Vulvo-vaginal part b. Endometrium
c. Ca cervix d. Radiation c. Ovaries d. Fallopian tubes
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P138 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,114; Shaw’s
838 Shaw’s Gynae 15th/e p. 324] Textbook of Gyanecology 16th/e p. 188]
Gynecology  Chapter 6 Infection of Genital Tract (MCQs)

12. Vulvo vaginal tuberculosis is seen in how much percentage
genital tuberculosis- (PGMEE June 14 Pattern)
a. 10-15% b. 20-30%
c. 50-60% d. 1-2%
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,114;Shaw’s
Gynae 15th/e p. 154]
13. Most common presentation of genital TB-
 (PGMEE 2013-14)
a. Infertility b. Polymenorrhoea
c. Vaginal discharge d. Pelvic pain
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,115;Shaw’s
Gynae 15th/e p. 154-156; William’s Gynae 1st/e p. 423]
14. Most common symptom of uterine tuberculosis is? 
 (PGMEE Nov 13 Pattern)
a. Asymptomatic b. Abnormal uterine bleeding
c. Infertility d. Foul smelling discharge
16. TB uterus all is true except- (PGMEE 2012-13)
a. Mostly secondary
b. Increase incidence of ectopic pregnancy
c. Involvement of endosalpinx
d. Most common is ascending infection
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,113-
115;Shaw’s Gynae 15th/e p. 154-155]
17. Fallopian tube tuberculosis- (PGMEE 2012-13)
a. Most common type of genital TB
b. Size of the tubes is unchanged
c. Is asymptomatic
d. Primary focus of infection is always in fallopian tubes
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,113-
114;Shaw’s Gynae 15th/e p. 154]
18. Beaded feel of fallopian tube on HSG is seen in- 
 (PGMEE 2012-13)

/e
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,115;Current a. TB b. Chlamydia
progress in obstetrics and gynaecology vol-1 ch. 18, p. 306- c. Gonococcal infection d. Syphillis
309; Danforth’s obstetrics] [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P116;

,2
Explanation Shaw’s Gynae 15th/e p. 157]
19. Beading is seen in case of genital TB in women in-
Tuberculosis
 (PGMEE 2012-13) 12. d
ƒƒ Tuberculosis is the m/c cause of primary infertility with a. Tubes b. Ovary 13. a
adenexal mass in India. CA 125 c/b raised. Ovarian c. Cervix d. Vagina
Es
cancer or borderline tumours of ovary are rare cause of 14. c
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,116;Shaw’s 15. b
primary infertility. Distension of abdomen, ascites, and
Gynae 15th/e p. 157]
pain abdomen are common presenting features in these 16. d
situations. 20. Genital tuberculosis spreads through: (PGMEE 2010) 17. a
○○ Endometrioma is a/w cyclical pain. a. Hematogenous route b. Ascending infection 18. a
iM

ƒƒ D/d of infertility in adenexal masses: c. Direct contact d. Lymphatic route

19. a
Clinical Age & parity Cl/f CA 125 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, 113;Shaw’s 20. a
condition
Textbook of Gynaecology, 15th/e, P. 154;Jeffcoate’s Principles 21. a
Ovarian Elderly Abdominal ↑ in 50% of Gynaecology, 7th/e, P. 323;Progress in Obstetrics & 22. b
PR

Ca distension, of epithelial Gynaecology, John Studd, Vol. 18, Ch. 27, P. 397
mass tumours
abdomen 21. Most common site of genital tuberculosis is?
 (PGMEE 2010)
Borderline Middle age Normal a. Fallopian tubes b. Ovary
ovarian c. Uterus d. Fimbriae
tumours
Endometrioma Middle age Painful ↑ in > 80% [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,114;;Shaw’s
adenexal Textbook of Gynaecology, 15th/e, P. 154-5;Jeffcoate’s
mass, 10 Principles of Gynaecology, 7th/e, P. 324;Progress in Obstetrics
infertility & Gynaecology, John Studd, Vol. 18, Ch. 27, P. 400
TB Any age ↑ Painless May be ↑
adenexal SEXUALLY TRANSMITTED INFECTION
mass
10 infertility TRICHOMONAS

15. Genital tuberculosis most commonly disseminates by?  22. Strawberry cervix is seen in-  (PGMEE 2015; 2012-13)
 (PGMEE Aug 13 Pattern) a. Ureaplasma urealyticum
a. Lymphatic route b. Hematogenous route b. Trichomoniasis
c. Local spread d. None c. Bacterail vaginosis
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,113; A d. Chalmydia
comprehensive textbook of obs & gynae by Sadhna Gupta [Ref: Shaw’s Gyanecology 16th/e p. 163; William’s Gyanecol- 839
1st/e p. 215] ogy 2nd/e p. 84]
 PRIMES (Volume II)

23. A 28 year old primigravida with 32 weeks of gestation
comes with complain of thin, frothy, profuse discharge
through the vagina since yesterday. She was advised USG
which showed Single live intrauterine gestational sac with
FL and AC as adequate. What is the diagnosis?
 (PGMEE 2015)
a. PPROM b. Candidiasis
c. Normal finding d. Trichomoniasis
[Ref: Shaw’s Textbook of Gynaecology 16th/e p. 163, William’s
Gyanecology 2nd/e p. 84]
24. Which of the following leads to greenish discharge per
vaginum- (PGMEE 2015)
a. Chlamydia b. Bacterial vaginosis
c. Ureaplasma urealyticum d. Trichomoniasis
[Ref: Shaw’s Textbook of Gynaecology 16th/e p. 163, William’s
Gynaecology 2nd/e p. 84]
31. Surest sign of salpingitis is- (PGMEE 2012-13)
a. Discharge of seropurulent pus from fimbrial end of tubes
b. Low back pain
c. Pyuria
d. Flank tenderness
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,134; Shaw’s
Gynae 15th/e p. 145]
Explanation
ƒƒ These lines from Shaw’s give us an idea about the answer.
ƒƒ ‘The sure sign of salpingitis is the discharge of seropurulent
fluid from the fimbrial end of the tube, without which the
diagnosis cannot be justified at laparotomy as the peritoneal
surface may be inflamed in pelvic peritonitis due to any
other cause.’
ƒƒ As Fallopian tube is the first site to be involved and
simultaneously there is also involvement of ovary as it lies

25. All of the following cause endometritis except-
 (PGMEE 2015)
a. IUCD b. Septic abortion
c. Trichomoniasis d. Gonorrhea
/e
in close proximity to the fimbrial end of fallopian tube.
ƒƒ Laparoscopic Examination are not use in routine, the pus
extruded from the fimbrial end and adhesions are sure sign
of PID.

23.
24.
d
d

26. Strawberry vagina causative organism is? 
 ,2
[Ref: Shaw’s Gynaecology 16th/e p. 175]

(PGMEE June14 Pattern)

a. Trichomonas vaginalis b. Candida albicans


32. Hanging drop preparation used for?


(PGMEE 2009)
a. Trichomonas vaginalis b. Gardenella vaginalis
c. Mobilincus d. Candida albicans
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,134]
Es
25. c c. H. vaginalis d. Syphilis CHLAMYDIA
26. a [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,134 ;Shaw
27. a 15th/e p. 146; William Gynae 1st/e p. 64] 33. Drug of choice for chlamydia in pregnancy is-
28. a 27. A lady presents with greenish discharge and strawberry  (PGMEE 2015)
29. a cervix is due to infection of? (PGMEE 2014) a. Metronidazole b. Azithromycin
iM

30. a a. Trichomonas vaginalis c. Doxycycline d. Erythromycin

31. a b. Hemophilus vaginalis [Ref: De Swiet’s Textbook of Medical Disorders in Obstetric
32. a c. Candida Practice 5th/e p. 407; Berek & Novak’s Gynecology 15th
d. Herpes simplex edition page 564, table 18.3]
33. b,c
PR

 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,134 Explanation

34. d
35. b 28. A young sexually active female has intensive pruritus and ƒƒ Both Azithromycin and Doxycycline are 1st line drugs for
36. a watery discharge, smear shows- (PGMEE 2013-14) Chlamydia.
a. Trichomonas vaginalis
34. Bacteria responsible for ectopic pregnancy is-
b. Candidia vaginitis
 (PGMEE 2015)
c. Gardenlla vaginalis
a. Staphylococcus b. Peptostreptococcus
d. HIV
c. Trichomonas vaginalis d. Chlamydia
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, 134]
 [Ref: Dutta’s Obstetrics 8th/e p. 207]
29. Treatment for trichomonas vaginalis is-(PGMEE 2013-14)
35. Usual causative organism of endocervicitis is? 
a. Metronidazole
 (PGMEE Aug. 13 Pattern)
b. Azithromycin
a. Herpes simplex virus b. Chlamydia
a. Ciprofloxacin
c. Trichomoniasis d. Candida
d. Abortions
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,123; Har-
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,134 ;Shaw’s
rison 17th/e p. 124]
Gynae 15th/e p. 121]
36. Most common cause of salpingitis- (PGMEE 2012-13)
30. History of yellow green watery discharge and pruritus-
a. Chlamydia b. Mycoplasma
 (PGMEE 2013-14)
c. Tubercular bacillus d. E. Coli
a. Trichomonas vaginalis b. Candida
c. Bacterial vaginosis d. Clamydia trachomatis [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P139,
Shaw’s Gynae 15th/e p. 445-446]
840 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,134;Shaw’s
Gynae 15th/e p. 145, 131]
Gynecology 
37. Woman presenting with chlamydial vaginal discharge.
DOC is- (PGMEE 2012-13)
a. Ciprofloxacin b. Doxycyclin
c. Metronidazole d. Azithromycin
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,123Shaw’s
Gynae 15th/e p. 145]
38. Which of the following is not caused by Chlamydia? a. Uterus b. Cervix
 (PGMEE Nov. 12 Pattern)
a. Vulvitis b. Salpingitis
c. Urethritis d. Cervicitis
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, pg.123;
Harrison 18th/e Chapter 176]
39. Fitz-Hugh-Curits syndrome is associated with- 
 (PGMEE Aug. 12 Pattern)
a. Chlamydial infection b. Genital tuberculosis
c. Syphilis d. Candida infection
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, 107; Har-
rison 17th/e p. 1073, 830]
Explanation
,2
ƒƒ Fitz hugh Curtis syndrome is caused by both Chlamydia
and Gonorrhoea, but 90% is caused by chlamydia
40. Chlamydia can cause infertility due to? (PGMEE 2009)
a. Salpingitis b. Endometritis
Es
c. Oophritis d. Cervicitis
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,123
CANDIDIASIS
41. Curdy vaginal discharge is seen in which infection-
iM
 (PGMEE 2012-13)
a. Bacterial vaginosis b. Candidiasis
c. Trichomoniasis d. Chlamydia
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,134 Shaw’s
PR
Gynae 15th/e p. 146]
42. Smear of vaginal discharge shows budding yeast cells.
Causative agent is? (PGMEE 2011)
a. Candida b. Mobilincus
c. Trichomonas d. Chlamydia
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,135]
GONORRHOEA
43. Most common organism causing salpingitis? (PGMEE June
14 Pattern)
a. N. gonorrhoeae
b. C. trachomatis
c. HSV
d. Mycoplasma
 [Ref: William’s Gynaecology 2nd/e p. 594]
44. Fitz Hugh Curtis syndrome is seen in- (PGMEE 2013-14)
a. Gonorrhoea b. Trichomonas
c. T.B. d. Herpes
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,107;Shaw’s
Gynae 15th/e p. 486]
Chapter 6 Infection of Genital Tract (MCQs)
45. Uretheral discharge is seen in- (PGMEE 2012-13)
a. Gonorrhoea b. Chlamydia
c. Herpes d. Candida
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,121; IAD-
VL Textbook of Dermatology 3rd/e p. 1849]
46. Gonorrhoea first involves? (PGMEE Nov.12 Pattern)
c. Vulva d. Vagina
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,120;Novak’s
14th/e p. 549; Danforth’s 10th/e p. 614]
SYPHILIS
47. Syphilis causes- (PGMEE 2013-14)
a. Still births b. Macrosomia
c. IUGR d. Abortions
/e
 [Ref: Dutta Obs 7th/e p. 295]
48. Drug of choice in pregnant women with Secondary Syphilis
is? (PGMEE Nov.12 Pattern)
a. Doxycycline b. Benzathine penicillin
c. Ceftriaxone d. Cotrimoxazole
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,123;
Danforth’s Obstetrics & Gynecology 10th/e p. 613] 37. d
38. a
49. Hutchinson’s traid in congenital syphilis include: (AIIMS
39. a
Nov’ 2017)
a. Interstitial keratitis, eighth nerve involvement, 40. a
Hutchinson’s teeth 41. b
b. Interstitial keratitis, mulberry teeth, periostitis 42. a
c. Periostitis, mulberry teeth, eighth nerve involvement
43. a
d. Hutchinson’s teeth. eighth nerve involvement, peritonitis
44. a
[Ref: Nelson 19th edition – chapter 210; Avery diseases of 45. a
 newborn – page 517]
46. b
Explanation 47. a
48. b
ƒƒ Syphilis is caused by infection with the spirochete Treponema
49. a
pallidum. In adults, this spirochete is transmitted through
50. c
sexual contact, but infants acquire the infection from their
mothers, either in utero or during delivery.
ƒƒ Syphilis in infants is acquired primarily by transplacental
transmission, which can occur at any time during pregnancy
but ordinarily occurs during 16 to 28 weeks’ gestation.
ƒƒ Classical triad in syphilis include Deafness (SNHL) +
interstitial keratitis + Hutchinson’s teeth (notched upper
central incisors) & mulberry molars (lower molars). Also k/
as Hutchinson’s triad
ƒƒ Details given in discussion section90
INFECTION OF INDIVIDUAL ORGANS
50. Bartholin cyst treatment of choice- (PGMEE 2012-13)
a. Excision
b. Antibiotics
c. Marsupialization
d. Drainage
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,133;Shaw’s 841
Gynae 15th/e p. 125]
 PRIMES (Volume II)

51. Treatment of choice for Bartholin’s abscess is? 57. Investigation for bacterial vaginosis is? (PGMEE Nov. 12
(PGMEE 2012) Pattern)
a. Incision drainage b. Marsupialisation a. KOH test b. Culture
c. Vulvectomy d. Cystectomy c. Gram stain d. Microscopy
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, 132; [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,125;Novak’s
Jeffcoate’s Principles of Gynaecology, 8th/e, P. 307-308, 418- 14th/e p. 544]
419; Te Linde’s Operative Gynecology, 9th/e, P. 872-874]
58. Not required for the diagnosis of vaginosis is?
(PGMEE 2012)
BACTERIAL VAGINOSIS
a. Whiff’s test positive b. pH > 4.5
52. Amsel criteria is for- (PGMEE 2015) c. Plenty of lactobacilli d. Clue cells
a. HELLP Syndrome
b. Bacterial vaginosis [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,125; Shaw’s
c. Ovarian ectopic pregnancy Textbook of Gynaecology, 15th/e, P. 131-132; Jeffcoate’s
d. Antiphospholipid antibody syndrome Principles of Gynaecology, 7th/e, P. 345-346; Berek & Novak’s
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, 125 Gynecology, 15th/e, P. 558-559; Progress in Obstetrics &
Gynaecology, John Studd, Vol. 15, Ch. 12, P. 186]
DeSwiet’s Medical Disorders in Pregnancy 5th/e p. 405]

/e
59. Clue cells are seen in? (PGMEE 2011)
53. Malodorous vaginal discharge is due to- (PGMEE 2015)
a. Bacterial vaginosis
a. Neisseria gonorrhea b. Bacterial vaginosis
b. Toxoplasmosis
c. Trichomonas vaginalis d. Chlamydia trachomatis
c. Syphilis

51.
52.
a
b Explanation
,2
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/
e,125;DeSwiet’s Medical Disorders in Pregnancy 5th/e p. 405;
Berek & Novak’s Gynecology 15th edition page page 560]

d. Herpes virus

[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,125

60. Whiff test is done for?
a. Bacterial vaginosis
(PGMEE 2011)
Es
53. b
ƒƒ Trichomonas vaginalis causes malodourous discharge, b. Genital tuberculosis
54. d Bacterial vaginosis causes fishy odour discharge. c. Syphilis
55. a 54. Fishy odor is seen in vaginitis by?  (PGMEE June 14 d. Candida
56. d Pattern)  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,125]
57. d a. Trichomonas b. Candidia
iM

61. Bacterial vaginosis does not include? (PGMEE 2010)

58. c c. Chylamdia d. Gardnerella
a. Profuse creamy discharge
59. a [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,125;Shaw b. Absent leucocytes
60. a 15th/e p. 131, 132; Williams Gynae 1st/e p. 51] c. Positive whiff test
61. a d. Clue cells present
55. Amsel criteria for the diagnosis of bacterial vaginosis does
PR

62. b 62. Vaginal emphysematous bulla, which is false-

not include? (PGMEE AUG. 13 Pattern)
63. d a. Presents with vaginal discharge  (PGMEE 2012-13)
a. Plenty of lactobacilli b. pH > 4.5
b. Leads to ulceration
c. Whiff’s test positive d. Clue cells
c. Usually occur in pregnant patients
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,125;Shaw’s d. May occur secondary to genital tract infection
13th/e p. 129; Novak’s Gynae 12th/e p. 192; CGDT 9th/e p. 654]  [Ref: Shaw’s Gynae 15th/e p. 135]
56. True about genital infection is?  (PGMEE 2012) 63. Most common cause of pyometra is- (PGMEE 2015)
a. Thin frothy secretions associated with monilial infection a. PID
b. Tetracycline is drug of choice for trichomonas vaginalis b. Tubercular endometritis
c. Patients and partners are given metronidazole for monilial c. Carcinoma cervix
infection d. Senile endometritis
d. Clue cells are associated with gardenella vaginalis  [Ref: Dutta’s Obstetrics 6th/e p. 168]
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, 134, 125,
135-136 Shaw’s 14th/e p. 132]

842
 CHAPTER 7: DISPLACEMENT AND PROLAPSED UTERUS
PROLAPSE ANATOMY
1. Strongest support of uterus is (PGMEE 2012-13)
a. Mackenrodt’s ligament a. Urogenital diaphragm b. Pubocervical ligament
b. Broad ligaments
c. Round ligaments
d. Uterosacral ligaments
[Ref: D.C. Dutta’s Textbook of gynaecology, 7th/e, P 166;
Shaw’s Gynae 15th/e p. 181]
Explanation
ƒƒ Uterosacral/cardinal ligament is the strongest support of
uterus.
,2
Es
iM
PR
Explanation
Perineal body
ƒƒ Perineal body structures are best visualized in the axial
plane, revealing three distinct regions: superficial, mid and
deep.
ƒƒ In the superficial portion at the level of the vestibular bulb,
the bulbospongiosus (BS) inserts into the lateral margins of
the perineal body, while the superficial transverse perinei
and external anal sphincter traverse the region.
ƒƒ In the mid-region at the proximal end of the superficial
transverse perinei, the puboperinealis muscle (one
component of the pubovisceral muscle) inserts into the
lateral margins of the perineal body and in some individuals
can be seen to cross the midline. The pubo-analis and
internal anal sphincter extend into the perineal body’s most
deep region at the level of the midurethra.
ƒƒ Weakness/injury to pubococcygeus & iliococcygeus
muscles causes the elevator plate to sag down resulting in
prolapse.
2. Not a support of uterus – (PGMEE 2012-13)
c. Perineal body d. Levator ani muscle fibers
 [Ref: Internet]
3. The deepest, part of the perineal body if cut/damaged can
lead to rectocele, cystocele and uterine prolapse. It is due to
damage of: (AIIMS Nov’ 2017)
a. Ischiocavernosus b. Bulbocavernosus
c. Pubococcygeus    d. External and sphincter
[Ref: Internet - https://www.ncbi.nlm.nih.gov/pmc/articles/
PMC3353407/)
/e
1.
2.
d
d
3. c
4. c
5. b
6. d
CLINICAL FEATURES AND DIAGNOSIS
4. Cystocele is formed by laxity and descent of:-
a. Ant upper 1/3 vagina  (PGMEE 2016-17)
b. Post upper 1/3 vagina
c. Anterior upper 2/3rd of vaginal wall
d. Posterior upper 2/3rd of vaginal wall
 [Ref: D.C Dutta text book of gynaecology 7th e p168]
5. Decubitus ulcers are due to -  (PGMEE 2013-14)
a. Due to trauma
b. Due to venous congestion
c. Due to friction created by thighs
d. Due to
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 170]
6. Patient with history of vaginal prolapse with ulcer on it.
Diagnosis- (PGMEE 2013-14)
a. Carcinoma b. Pressure erosion
c. Syphilis d. Decubitus ulcer
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 170; 843
Shaw’s Gynae 15th/e p. 335]
 PRIMES (Volume II)

SURGICAL MANAGEMENT 13. Management of third degree utero-vaginal prolapse in

7. What is the preferred treatment of complete prolapse in a
female with completed family? (PGMEE 2015)
a. Pessary b. Vaginal hysterectomy
c. Sling surgery d. Le Forte’s repair
 [Ref: Shaw’s Gynaecology 16th/e p. 357]
8. Le Fort repair is done for-
 (PGMEE Nov 13 Pattern; PGMEE 2015)
a. VVF b. RVF
c. Vault prolapse d. Uterovaginal descent
[Ref: Shaw’s Gynaecology 16th/e p. 360][Ref: D. C. Dutta’s
Textbook of gynaecology, 7th/e, P180;Danforth 10th/e p. 867;
Shaw’s 14th/e p. 308
9. Le Fort’s operation is done in? (PGMEE Nov 13 Pattern)
a. Young patient with utero-vaginal prolapse
woman who wants to have a child in future is?
a. Fothergill’s repair  (PGMEE 2009)
b. Shirodkar’s modified sling operation
c. Manchester operation
d. Le Fort’s repair
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 339-344;
Jeffcoate’s Principles of Gynaecology, 8th/e, P. 266]
14. Purandare operation is indicated for?
 (PGMEE Aug. 12 Pattern)
a. Elongated cervix b. Missed IUD
c. Incompetent cervix d. Nulliparous prolapse
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P182
15. A female underwent cervical encirclage. Which of the
following is done to check that ring pessary is placed
correctly:- (PGMEE 2018)

/e
b. Elderly patient with utero-vaginal prolapse a. Insufflations of rectum between ring pessary and vagina
c. Multiparous with utero-vaginal prolapse b. Valsalva maneuver
d. Pregnant patient with utero-vaginal prolapse c. Ask her to urinate and look if ring is coming out
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P180; d. All of the above

7.
8.
9.
b
d
b


a. Sling operation
,2
Danforth 10th/e p. 867; Shaw’s 14th/e p. 308]
10. Treatment for young woman with prolapsed uterus is-
(PGMEE 2012-13)
b. Anterior colporrhaphy

PROLAPSE IN PREGNANCY
16. 3rd degree genital prolapse is early (first trimester) of
pregnancy is managed by ? 
a. Ring pessary
(PGMEE Aug 13 Pattern)
Es
c. Posterior colporrhaphy d. Manchester operation
10. a b. Fothergill’s repair
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P182;
c. Le fort’s repair
11. a Shaw’s Gynae 15th/e p. 339]
d. Right transvaginal sacrospinous colpopexy
12. b 11. Prolapse of uterus in nulliparous women, treatment is- [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 173;
13. b a. Sling used involving rectus sheath  (PGMEE 2012-13) Shaw’s 14th/e p. 308; Danforth’s 10th/e p. 867]
iM

14. d b. Anterior colporrhaphy

c. posterior colporrhaphy 17. When ring pessary is removed during pregnancy? 
15. b
d. Manchester operation  (PGMEE Nov. 12 Pattern)
16. a a. Before labour b. 1st trimester
17. c [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,p.182
c. 2nd trimester d. 3rd trimester
Shaw’s Gynae 15th/e p. 339]
PR

18. d  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P173]

12. Which surgery uses rectus sheath to prevent prolapse -
 (PGMEE 2012-13)
a. Khanna operation b. Purandare operation
c. Shirodkar operation d. Le Forte repair
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P182 ;
Shaw’s Gynae 15th/e p. 342]
18. For uterine prolapse in pregnancy, Ring pessary can be
inserted upto- (PGMEE 2012-13)
a. 12 weeks b. 14 weeks
c. 16 weeks d. 18 weeks
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P173]

844
 CHAPTER 8: URINARY PROBLEMS, GENITAL FISTULAE AND
GENITAL TRACT INJURIES

URINARY INCONTINENCE 5. 48 yr, 7 day post hysterectomy fever, burning micturition

with dribbling of urine but able to pass urine voluntarily.
1. Surgery for genuine stress urinary incontinence is? Diagnosis is-  (PGMEE 2012-13)
a. Kelly’s plication  (PGMEE June14 Pattern) a. VVF b. UVF
b. Retropubic urethropexy
c. Urge incontinence d. Stress incontinence
c. Haltain’s operation
d. Spinelli’s operation [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P349;
Jeffcoate 17th/e p. 263-265; Shaw’s Gynae 15th/e p. 183]
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P331;
Shaw’s Gynae 15th/e p. 174] 6. A 52 years old lady presents with constant leakage of urine
and dysuria two weeks after a complicated total abdominal
2. Bonney’s test is used to determine-  (PGMEE 2013-14)
hysterectomy. A diagnosis of Vesicovaginal fistula is
a. Uterine prolapsed
suspected. The most important test for the diagnosis is:
b. Stress urinary incontinence
 (PGMEE 2010)
c. Vesicovaginal fistula

/e
d. Uteric fistula a. Triple swab test b. IVP
c. Cystoscopy d. Urine culture
 [Ref: Shaw’s Gynae 15th/e p. 191]
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P345;
3. Continuous incontinence of urine is seen in-  William’s Gynaecology 1st/e p. 574]




a. VVF 
b. Vesicoperitoneal
c. Ureterovaginal fistulae
d. Uretrovaginal ,2
(PGMEE 2013-14)

[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P349;

URINARY INFECTION
7. Genito urinary TB is characterized by:-(PGMEE 2016-17)
a. Asymptomatic bactriuria b. Symptomatic bactriuria
1.
2.
b
b
Es
c. Hematuria d. Sterile pyuria 3. c
Shaw’s Gynae 15th/e p. 185] 4. c
[Ref: D.C Dutta text book of gynaecology 7th e p 339;From
5. b
URINARY FISTULAS ROAMS 13th/e pg. 690]
6. a
4. VVF in obstructed labour is repaired­after a gap of 8. In a 25 year old female, cystitis is best treated by: 7. d
iM

 (PGMEE 2012-13)  (PGMEE 2012)

a. Immediately b. 3 weeks
c. 3 months d. 6 months
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P346;
Shaw’s Gynae 15th/e p. 192]
PR
8. d
a. Cephelexin b. Amoxicillin
c. Norfloxacin
 d. Nitrofurantoin
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 178-179;
Berek & Novak’s Gynecology, 15th/e, P. 570-571]

845
 CHAPTER 9: ENDOMETRIOSIS

ENDOMETRIOSIS 8. Gold standard diagnostic technique for diagnosis of endo-

1. Which is the following yields definite diagnosis of endome-
triosis (PGMEE 2016-17)
a. MRI b. Ca-125
c. USG abdomen and Pelvis d. Laparoscopic evaluation
 [Ref.: D.C Dutta text book of gynaecology 7th e p251]
2. If endometriosis occurs in lung then what will be term use
for that.....:- (PGMEE 2016-17)
a. Vascular theory b. Implantation
c. Coelomic metaplasia d. Retrograde menstruation
 [Ref: D.C Dutta text book of gynaecology 7th e p249]
3. Sampson’s theory is pathogenesis for  (PGMEE 2016-17)
metriosis -  (PGMEE 2013; Nov 12 Pattern; June 2009)
a. Laparoscopy b. MRI
c. Ultrasound d. Ca 125 level
[Ref: D.C.Dutta text book of gynaecology 7the p.251; Shaw’s
Gynae 15th/e p. 470]
9. Most common site of endometriosis- (PGMEE 2011-2013 )
a. Ovary b. FT
c. LSCS Scar d. Colon
[ Ref: D.C.Dutta text book of gynaecology 7the p.252;Shaw’s
Textbook of Gynaecology, 15th / e, P. 466;Jeffcoate’s Principles
of Gynaecology, 7th / e, P. 370-372]
10. Drug commonly used in t/t of endometriosis-

/e
a. Endometriosis  (PGMEE 2012-13)
b. Fibriod a. LH b. GnRH
c. Ca endometrium c. MPA d. FSH
d. Cervical intraepithelial noeplasia
[Ref:D.C.Dutta text book of gynaecology 7the p.254; Shaw’s

1.
2.
d
a


Explanation
Theory of Endometriosis ,2
[Ref.: D.C Dutta text book of gynaecology 7th e p249]

ƒƒ SAMPSON’S theory (most accepted theory) is theory of


Gynae 15th/e p. 473]
11. 1996 ASRM classification of endometritis includes-

a. Intensity of pain b. Location

(PGMEE 2012-13)
Es
3. a c. Size and location both d. Number
4. a retrograde menstruation
ƒƒ Theory of coelomic metaplasia [Ref: Leon and speroff clinical endocrinology pg1237, 8th
5. d edition]
ƒƒ Immune mediate theory: Causes is deficiency of both cell
6. a mediated & humoral Immunity Explanation
7. d ƒƒ Genetic predisposition: Gene is K-ras. If 1st degree relative
iM

8. a affected → Risk is 7 times higher ASRM classification of Endometriosis

9. a ƒƒ Theory of lymphatic & vascular spread.
State Progression Tissue description
10. b,c 4. Chocolate cyst is found in  (PGMEE 2016-17)
11. c a. Endometriosis b. Ovarian mucinous cyst I Minimal (1-5) 2-3 superficial implants
PR

12. d c. Fallopian cyst d. Cornual cyst
13. d  [Ref: D.C Dutta text book of gynaecology 7th e p252]
5. Drugs used in endometriosis -  (PGMEE 2015)
a. Letrozole b. Mifepristone
c. Combined oral contraceptives
d. All of the above
[Ref: D.C.Dutta text book of gynaecology 7the p.254;Shaw’s
Gyanecology 16th/e p. 417]
6. Not seen in endometriosis is? (PGMEE 2012)
a. Vaginal discharge b. Dysmennorrea
c. Pelvic pain d. Dyspareunia
 [Ref: D.C.Dutta text book of gynaecology 7the p.250]
Explanation
ƒƒ Presentation → M/c presentation: pain
ƒƒ M/c dysmenorrhea (secondary) > Chronic pelvic pain >
dyspareunia > Lower backache.
ƒƒ M/c cause of 20 dysmenorrhea: Endometriosis > PID
7. Pain in endometriosis due to- (PGMEE 2013)
a. PGI b. PG E1
846
c. PG E2 d. PG F2α
 [Ref: D.C.Dutta text book of gynaecology 7the p.250-1]
II Mild (6-15) Appearance of more implants within
deeper layers of tissue
III Moderate Many deep implants in combination
(16-40) with minor/small endometriomas on
one or both ovaries. May also present
filmy adhesions.
IV Severe (>40) Persistence of deep implants, enlarge-
ment of endometriomas on one or both
ovaries, development of dense adhesions.
ADENOMYOSIS
12. Treatment of choice for perimenopausal adenomyosis is?
 (PGMEE 2013)
a. OCPs b. GnRH agonists
c. LNG IUS d. Hysterectomy
[Ref: D.C.Dutta text book of gynaecology 7the p.258; Novak’s
gynae 14th/e p. 521]
13. Definitive management of adenomyosis is
 (PGMEE 2012-13)
a. GNRH analogue b. Danazole
c. LH d. Hysterectomy
[Ref: D.C.Dutta text book of gynaecology 7the p.258; Shaw’s
Gynae 15th/e p. 475]
 CHAPTER 10: ENDOSCOPY AND OPERATIVE GYNECOLOGY

ENDOSCOPY AND OPERATIVE GYNECOLOGY
1. What is the contraindication of laparoscopic sterilization-
a. Post partum state (PGMEE 2012-13)
b. Post MTP
c. Gynaecologic malignancies
d. 3 previous child birth
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P409]
2. Intrauterine adhesions best seen by- (PGMEE 2013-14)
a. USG b. CT
c. Hysteroscopy d. MRI
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P 378]
3. Which of the following cannot be treated by laparoscopy-
 (PGMEE 2012-13)
a. Ectopic pregnancy b. Sterilization
c. Non descent of uterus d. Genital prolapse
7. 35 year old with history of repeated D&C. She now has
secondary ammenorhea. What is your diagnosis-
 (PGMEE 2012-13)
a. Hypothyroidism b. Kallman syndrome
c. Sheehan’s syndrome d. Ashermann’s syndrome
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P 378,486]
8. Which is least injured in gynaecological procedures-
a. Ureter at pelvic brim  (PGMEE 2012-13)
b. Renal pelvis
c. Urinary bladder
d. Ureter at infundibulopelvic ligament
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P493;
Shaw’s Gynae 15th/e p. 184]
9. Radical hysterectomy is named after-  (PGMEE 2012-13)
a. Wertheim’s b. John clark
c. Meigs d. Mitra

/e
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P506]  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P499]

Explanation 10. Hysterectomy when done through broad ligament causes

injury to? (PGMEE Aug 12 Pattern)

,2
ƒƒ Genital prolapse. Prolapse surgeries cant be done a. Bladder
laparoscopically. b. Ureter
4. Not a laparoscopy instrument- (PGMEE 2012-13) c. Urethra 1. a
a. Trocar b. Pneumoperitoneum needle d. Transverse colon 2. c
c. Doyen’s retractor d. Fiberoptic camera [Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P349,496;
Es
3. d
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P504] Shaw’s 13th/e p. 14; Danforth 10th/e p. 456]
4. c
5. All of the following are used in hysteroscopy except- 11. During hysterectomy, ureter is liable for injury at? 5. b
 (PGMEE 2012-13) a. Where it enters bladder wall  (PGMEE 2010) 6. b
a. CO2 b. O2 b. Where it crosses pelvic brim
7. d
c. Normal saline d. Dextrose
iM

c. Where it crosses uterine artery

d. None 8. b
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P511;
Shaw’s Gynae 15th/e p. 493] 9. a
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P349;Te
Linde’s Operative Gynecology, 9th / e, P. 1084] 10. b
OPERATIVE GYNECOLOGY 11. c
12. Hysterosalphingography is done during? (PGMEE 2010)
PR

a. Secretory phase b. Luteal phase 12. c

6. In total abdominal hysterectomy following are removed-  c. Follicular phase d. During menstruation
a. Uterus  (PGMEE 2012-13)
b. Uterus & cervix [Ref: Shaw’s Textbook of Gynaecology, 15th / e, P. 211-3, 501-4
c. Uterus, cervix & fallopian tube Jeffcoate’s Principles of Gynaecology, 7th / e, P. 709-14 Berek
d. Uterus, cervix, fallopian tube & ovary & Novak’s Gynecology, 15th / e, P. 1157]
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th / e, P 490]

847
 CHAPTER 11: HORMONES IN PRACTICE
MIFEPRISTONE
1. Mifepristone can be given upto how many days-
 (PGMEE 2015)
a. 49 days b. 63 days
c. 78 days d. 93 days
 [Ref: Dutta’s Obstetrics 8th/e p. 203]
2. Dose of mifepristone in MTP? (PGMEE 2013-14)
a. 600mg b. 400mg
c. 200mg d. 100mg
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P444]
3. Mifepristone may be used for all of the following Except-
 (PGMEE 2011)
a. Threatened Abortion b. Molar pregnancy
c. Fibroids d. Ectopic pregnancy
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 444]
1.
2.
b
c
a. Ovarian cancer
SERM
c. Endometrial cancer
,2
4. Adverse effect of tamoxifen therapy is:-(PGMEE 2016-17)
b. Breast cancer
d. Cervical cancer
Es
3. a
 [Ref: D.C Dutta text book of gynaecology 7th e p 441]
4. c
5. c 5. SERMs are- (PGMEE 2013-14)
a. Agonist on estrogen receptor
6. a
b. Antagonist on estrogen receptor
7. c c. Some are agonist some antagonist on estrogen receptor
iM
8. d d. Used due to reduced chances of hot flushes, thromboem-
9. c bolism
10. a  [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P441]
11. d
6. GnRH analogue are all except- (PGMEE 2012-13)
PR
12. a
a. Soserelin b. Gonadorlein
c. Goserelin d. Buserelin
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P434]
MENOPAUSE AND HRT
7. Hot flushes in menopause is due to changes in:-
 (PGMEE 2016-17)
a. Prolactin b. GnRH ƒƒ Indications of HRT are
c. Estrogen d. Progesterone
 [Ref: D.C Dutta text book of gynaecology 7th e p 48]
8. Hot flushes are experienced as a result of- (PGMEE 2015)
a. Decreased estrogen
b. Increased noradrenaline
c. Increased noradrenaline and estrogen
d. Increased noradrenaline and decreased estrogen
[Ref: SPEROFF 8th e p.696; D. C. Dutta’s Textbook of
gynaecology, 7th/e, P48;Shaw 16th/e p. 68]
848
9. Drug of choice for vasometer symptoms in post menopausal
women is? (PGMEE 2014)
a. Tamoxifen b. Medroxyprogesterone
c. Clonidine d. Conjugates estrogen
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P50;
Shaw’s Textbook of Gynaecology, 15th/e, Page no. 63, 69;
Jeffcoate’s Principles of Gynaecology, 7th/e, P. 868-869;
Berek & Novak’s Gynecology, 15th/e, P. 1235, 1238
10. In a postmenopausal female, which hormone increases-
 (PGMEE 2012-13)
a. FSH b. Estrogen
c. GH d. None of the above
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P49;Shaw’s
/e
Gynae 15th/e p. 62]
11. HRT is given in- (PGMEE 2012-13)
a. Symptomatic postmenopausal women
b. Following hysterectomy
c. Because a women has asked for it
d. All of the above
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P50;
Shaw’s Gynae 15th/e p. 66]
Explanation
HRT (Hormone Replacement Therapy)
ƒƒ HRT is required in menopausal women who are symptomatic,
high risk of cardio vascular diseases, osteoporosis.
○○ Estrogen component of HRT have cardioprotective
effect. It increases HDL and ¯ses cholesterol and TG. So
it decreases risk of CAD.
○○ It is used for osteoporosis in menopausal women.
Oestrogen, progesterone, tibolone and raloxifene are
beneficial in osteoporosis. HRT prevents bone resorption
and bone mineral density.
○○ Estrogen component can cause increase risk of breast
cancer in multiparous females.
○○ Carcinoma of endocervix can occur. Carcinoma
of endometrium occurs with increase estrogen
supplementation without progesterone.
○○ Postmenopausal women with osteoporosis
○○ Adult hypopituitarism
ƒƒ Remember :
○○ OCPs are protective against benign diseases of breast
(e.g. fibroadenoma) and malignancy of endometrium and
uterus.
12. HRT improves- (PGMEE 2012-13)
a. Bone density b. Coronary artery disease
c. Dementia d. Endometrial cancer
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P50]
Gynecology  Chapter 11 Hormones in Practice (MCQs)

13. Post menopausal HRT decreases incidences of which
malignancy- (PGMEE 2012-13)
a. Breast a. Increase progesterone b. Increase androgens
b. Colorectal
c. Ovarian
d. Endometrium
 [Ref: speroff 8th e p.842; Shaw’s Gynae 15th/e p. 66]
14. What is false about post menopausal state-
 (PGMEE 2012-13)
a. Low LH b. Low estrogen
c. High FSH d. High androgen
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 46;
Shaw’s Gynae 15th/e p. 62]
15. Which of the following is true about menopause?
 (PGMEE Nov 12 Pattern)
c. Incraese FSH d. Increase estrogen
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 47;
Novak’s gynae 12th/e p. 450-452]
16. Absolute contraindication of HRT is? (PGMEE 2011)
a. Osteoarthritis b. Endometriosis
c. Heart disease d. Breast carcinoma
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P.50;
Shaw’s Textbook of Gynaecology, 15th/e, P. 66-67 Jeffcoate’s
Principles of Gynaecology, 7th/e, P. 881-882 Berek & Novak’s
Gynecology, 15th/e, P. 1245]

/e
,2 13.
14.
b
a
Es
15. c
16. d
iM
PR

849
 CHAPTER 12: PCOS AND POF

PCOS 7. Stein Levinthal syndrome what hormone is raised-

1. Treatment of PCOD for infertility (PGMEE 2016-17)
a. Ovulation induction b. OC pills
c. Metformin d. Laproscopic drilling
[Ref: D.C Dutta text book of gynaecology 7th e p.382;From
ROAMS 13th/e pg. 695]
2. USG ovary showing multiple small cystic structures.Ovary
is increased in size and there is string of pearls appearance).
The patient complains of irregular menstruation. The
MOST likely diagnosis on the basis of the investigation can
be:-  (PGMEE 2016-17)
 (PGMEE 2013-14)
a. LH b. FSH
c. GnRH d. Progesterone
[Ref: D.C. Dutta’s Textbook of gynaecology, 7th/e, P378-379]
Explanation
 Persistently elevated LH
ƒƒ PCOD / Polycystic ovarian syndrome or disease was
earlier known as Stein leventhal syndrome. 15 % of female
population suffers from PCOS and the patients are mostly
15 to 25 yrs of age.
ƒƒ PCOS includes chronic non ovulation and hyperandrogen-

/e
emia a/w normal or raised oestrogen,raised LH, and low
FSH/LH ratio
ƒƒ In a typical case USG alone is adequate to confirm
the diagnosis. In other cases, low FSH/LH ratio,raised

1.
2.
a
c ,2 testosterone, androstenedione, DHEA will be observed.
ƒƒ DHES (Sulfated forms of DHEA) is secreted exclusively by
adrenals and its secretion is increased in 50% case of PCOS
and in adrenogenital syndrome esp. 3 b - HSD deficiency.
ƒƒ About 25% of patients with PCOS exhibit elevated Prolactin
Es
3. c levels. (Ref. Novak’s Gynaecology 13th/e p. 878)
4. a ƒƒ There is an increase in LH pulse frequency as a result of
5. a a. Cystic adenocarcinoma ovary GnRH pulse frequency. In some patients with PCOS,
6. a b. Chocolate cyst bromocriptine has reduced LH levels and restored ovulatory
7. a c. PCOS functions. (Ref. Novak’s Gynaecology 13th/e p. 878)
iM

8. d d. Ovarian cyst 8. A 16 years old girl came for evaluation of hirsutism, irregular
9. a  [Ref: D.C Dutta text book of gynaecology 7th e p.379] bleeding and infertility, diagnosed as PCOS. Which of the
following drugs should not be given? (AIIMS Nov 2013)
10. b 3. Hirsutism is graded on the basis of:- (PGMEE 2016-17)
a. Spironolactone
a. Turner score
b. Tamoxifen
PR

b. Keilland score
c. Ferriman-Galleway score
d. Testosterone score
 [Ref: speroff 8th e p.543]
4. Which is not increased in PCOS(PGMEE 2015)
a. Sex hormone binding globulin
b. Estrogen
c. Insulin
d. Luetinizing hormone
[Ref: D.C Dutta text book of gynaecology 7th ep.380;Shaw’s
Gynaecology 16th/e p. 431]
5. Regarding PCOD all are true except- (PGMEE 2013-14)
a. High FSH/LH b. High DHEA
c. Raised LH d. Estrogen
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P379;
Novak’s Gynae 15th/e p. 1076; Shaw’s Gynae 15th/e p. 217]
6. Stein Leventhal syndrome is- (PGMEE 2013-14)
a. PCOD b. Turner’s syndrome
c. Swyer’s syndrome d. Klinefelter’s syndrome
850 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P378]
c. OCPs
d. Clomiphence citrate
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P382;
Berek & Novak’s Gynecology 15th edition – page- 1152.]
Explanation
ƒƒ Clomiphene citrate is drug of choice for ovulation induction
in patients with infertility secondary to PCOS, not for
treatment of PCOS.
9. Commonest cause of anovulatory infertility-
 (PGMEE 2012-13)
a. PCOS b. TB
c. Endometriosis d. Thyroid dysfunction
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P378;
Shaw’s Gynae 15th/e p. 209]
10. True for PCOD is- (PGMEE 2012-13)
a. Increased FSH b. Increased LH
c. Increased SHBG d. Increased FSH/LH ratio
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P379;
Shaw’s Gynae 15th/e p. 370]
Gynecology  Chapter 12 PCOS and POF (MCQs)

11. Enzymes responsible for conversion of androgen into 15. 35 yr old with 4 months amenorrhea with increased FSH,
estrogen are all except- (PGMEE 2012-13) decreased estrogen. What is the diagnosis-(PGMEE 2012-13)
a. Aromatase a. Premature ovarian failure
b. Sulphatase b. PCOD
c. Fumarase c. Pituitary failure
d. 3 beta hydroxy steroid dehydrogenase d. Hypothalamic failure
 [Ref: Speroff 8th e p.775] [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P383;
12. Hyperstimulation syndrome, true is- (PGMEE 2012-13) Shaw’s Gynae 15th/e p. 70, 290]
a. Usually seen in late pregnancy 16. Oestrogen levels in premature ovarian failure are below?
b. hCG is the treatment of choice  (PGMEE Aug 12 Pattern)
c. Raised LH is responsible in PCOD a. 10 pg/ml b. 20 pg/ml
d. Vascular permeability is decreased c. 40 pg/ml d. 80 pg/ml
 [Ref: Dutta gynae 7th e p.437] [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P383;Prin-
13. Which hormone is increased in PCOS?  (PGMEE 2015) ciples and Practice of Endocrinology and metabolism p. 957]
a. LH b. FSH 17. Premature ovarian failure with good ovarian reserve next
c. Inhibin d. Estrogen step in management-

/e
 [Ref: Robbins 9th/e pg. 1022] a. Ovulation induction
b. In vitro fertilization
PREMATURE OVARIAN FAILURE c. Intrauterine sperm insemination
d. Genetic counseling

,2
14. Which is increased in premature ovarian failure:
 (PGMEE 2015) [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P200;
a. Sr. FSH b. Sr. Estradiol Shaw’s Gynae 15th/e p. 215]
c. Sr. Inhibin B d. Both B and C 11. c
[Ref: Shaw’s Gyanecology 16th/e p. 74; Speroff’s clinical 12. c
Es
Gynaecologic Endocrinology and Infertility 8th/e p. 449] 13. a
14. a
15. a
16. b
17. a
iM
PR

851
 CHAPTER 13: AMENORRHEA

PRIMARY AMENNORRHEA also with an assortment of other structural X chromosome

1. Primary amenorrhea is called if menstruation not occurred
by the age of? (PGMEE 2014)
a. 13 years b. 15 years
c. 16 years d. 18 years
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 371;
Berek & Novak’s Gynecology 15th edition page 1036.]
Explanation
ƒƒ According to latest guidelines- primary amenorrhoea is
○○ Absence of initiation of menses in female of 15 years of
age with development of secondary sexual characters, or,
○○ Absence of initiation of menses in females of 13 years of
abnormalities (deletions, ring and iso-chromosomes), any
of which may be present in all or only in some of the cells of
the body (mosaicism), depending on the stage of embryonic
development at the time they arise.
ƒƒ The diagnosis of Turner syndrome generally can be
made easily, based on the phenotype and findings of
hypergonadotropic hypogonadism. A karyotype is
definitive, and specifically indicated, in part because it may
reveal a cell line containing a Y chromosome otherwise not
suspected or identified (e.g., 45,X/46,XY); approximately
5% of women with Turner syndrome have a karyotype
containing all or part of a Y chromosome.
ƒƒ Whereas it is important to identify a Y chromosome

/e
age without development of secondary sexual characters. because affected individuals are at significant increased
risk for developing gonadoblastoma (20–30%), that risk
GONADAL DYSGENESIS appears lower (5–10%) in women with Turner syndrome,
2. XO is seen in: (PGMEE 2016-17) and limited to those having detectable Y chromosome on

,2
a. Turner’s b. Edward’s their karyotype.
c. SuperFemale d. Klinefelter’s syndrome ƒƒ In all patients under age 30 with a diagnosis of ovarian failure,
a karyotype should be obtained to exclude chromosomal
1. b [Ref : D.C Dutta text book of gynaecology 7th e p363] translocations, deletions, and mosaicism that might offer
2. a 3. Patient with XO syndrome attains puberty which hormone an obvious explanation. A karyotype also identifies those
Es
3. c given after puberty :- (PGMEE 2016-17) having a Y chromosome in whom gonadectomy is indicated
4. a a. Oestrogen b. Progesterone due to the significant risk for malignant transformation in
5. a c. E+P d. Thyroid hormone occult testicular elements (20–30%).
6. a  [Ref: speroff 8th e p.380] ABNORMAL GENITAL ANATOMY
7. b 4. Chromosomal abnormality in klinefelter’s syndrome :-
iM

8. d  (PGMEE 2016-17) CYRPTOMENORRHEA

9. a a. 47XXY b. 45XO
c. 46XX d. 47XYY 7. Cryptomenorrhoea and amenorrhoea, other symptom is:- 
 [Ref: D.C Dutta text book of gynaecology 7th e p365] a. Abdominal lump  (PGMEE 2016-17)
b. Cyclical abdominal pain
PR

5. A 16-years old female presents with primary amenorrhea c. Short stature

and raised FSH level. On examination, her height was 58
inches. What would be the histopathological finding in the
ovary? (AIIMS Nov 2013)
a. Absence of oocytes in the ovaries (streak ovaries]
b. Mucinous cystadenoma
c. Psammoma bodies b. Seen in cases of imperforate hymen
d. Hemorrhagic corpus luteum
[Ref: Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e,
P. 363;Berek’s and Novak’s 15th/e, Clinical gynecology
Endocrionology and infertility, 8th/e]
6. A patient with amenorrhea, infantile uterus on karyotyping
showed 450X0/46XY? How would you manage.
a. Bilateral orchidectomy  (AIIMS Nov’ 2017)
b. Vaginopathy
c. Resection of clitoris
d. Hormone replacement therapy
 Ref: Speroff 8th e p 449,461-2
Explanation
852 ƒƒ Turner syndrome is a well known and thoroughly studied
disorder, classically associated with a 45,X karyotype, but
d. Poor secondary sexual characters
 [Ref: D.C Dutta text book of gynaecology 7th e p371]
8. Cryptomenorrhoea true is:- (PGMEE 2015)
a. Retention of urine may be seen
c. Hematocolpos is present
d. All the above
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 371
9. Emergency presentation of imperforate hymen is?
 (PGMEE Nov 13 Pattern)
a. Retention of urine b. Cyclic pain
c. Mass abdomen d. Hematocolpos
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 371;Novak’s
gynae 14th/e p. 1049; William’s gunae 13th/e ch. 41]
Explanation
ƒƒ Pelvic hematocoele may cause urinary retention but
there will be no amenorrhoea.
� Impacted cervical fibroid cause urinary retention +
amenorrhoea.
Gynecology  Chapter 13 Amenorrhea (MCQs)

10. What is the most common cause of the condition shown in 12. Primary amenorrhea, absent uterus, normal pubic hair.
following picture? (PGMEE Nov 13 Pattern) Diagnosis - (PGMEE 2015)
a. Mullerian agenesis
b. WNT 4 syndrome
c. Hand foot genital syndrome
d. Testicular feminization syndrome

[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 373

Shaw’s Gynaecology 16th/e p. 129] William’s Gynaecology
2nd/e p. 496]
13. In MRKH syndrome, which among the following is absent?
 (PGMEE 2015)
a. Pubic hair development b. Breast development
c. Ovary d. Vagina
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 373
a. MRKHS Shaw’s Gynaecology 16th/e p. 129]

/e
b. Transverse vaginal septum 14. An 18 yrs young female presents with primary amenorrhea,
c. Imperforate hymen cyclic abdominal pain and voiding difficulties. On examination
d. Segmental Mullerian agenesis breast and pubic hair are well developed but vagina can’t be
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 371; appreciated. Diagnosis is- (PGMEE 2012-13)

Explanation
,2
Novak’s gynae 14th/e p. 1049; William’s gynae 13th/e ch. 41]

ƒƒ Imerforate hymen can lead to hematocolpos, which

is observed in young girls. These girls present




a. Mayer-Rokitansky-Kuster Hauster Syndrome
b. Turner syndrome
c. Noonan syndrome
d. Kallmann syndrome
10.
11.
c
a
Es
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 373] 12. a
with colicky abdominal pain which is often cyclic.
15. Normal development of a female with absent uterus and 13. d
Menarche has not yet set in. However secondary
vagina indicates? (PGMEE Nov.12 Pattern) 14. a
sexual characters are well developed. Vagina gets
a. Turner’s syndrome 15. c
distended with the menstrual blood and becomes
b. Gonadal dygenesis 16. b
tense. O/ E a bluish bulging membrane is recognized.
iM

c. Mullerian agenesis 17. b

MULLERIAN ANOMALIES d. Testicular feminizing syndrome

11. Gene for Mullerian inhibiting hormone is located on short [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 373;
arm of (PGMEE 2018) Shaw’s 13th/e p. 87]
PR

a. Chromosome 19 b. Chromosome 22 16. -----is seen in Rokitansky Küstner syndrome is?

c. Chromosome X d. Chromosome Y
 [Ref speroff 8th e p.342.]
Explanation
ƒƒ The gene encoding AMH is located on the short arm of
chromosome 19 (19p13.3).
ƒƒ AMH is a member of the TGF-b superfamily family of
growth and differentiation factors that includes inhibin and
activin. Like other members of the TGF-b superfamily, AMH
signaling is mediated via a heterodimeric receptor consisting
of a type I and a type II serine/threonine kinase receptor.
ƒƒ The specific type II receptor that binds AMH are called
AMHR2. The gene encoding AMHR2 is located on
chromosome 12 (12q13).
ƒƒ AMH gene expression is induced by SOX9 in Sertoli cells
soon after testicular differentiation and results in the
ipsilateral regression of the müllerian ducts by 8 weeks
of gestation, before the emergence of testosterone and
stimulation of the wolffi an ducts.
ƒƒ Inactivating mutations of AMH or AMHR2 result in
persistent müllerian ducts in males
(PGMEE 2010)
a. Ovarian agenesis b. Vaginal aplasia
c. Septate uterus d. Bicornuate uterus
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P373;
Shaw’s Textbook of Gynaecology, 15th/e, P. 94-96; Jeffcoate’s
Principles of Gynaecology, 8th/e, P. 182-184; Berek & Novak’s
Gynecology, 15th/e, P. 1007, 1046-1047
TESTICULAR FEMINIZING SYNDROME /
ANDROGEN INSENSITIVITY SYNDROME
17. What is Reifenstein syndrome? (PGMEE 2014)
a. Associated with gonadal dysgenesis
b. Partial androgen insensitivity syndrome due to receptor
mutation
c. Associated with mental retardation
d. 5-alpha reductase deficiency associated with perineo-
scrotal hypospadias
[Ref: Shaw’s Textbook of Gynaecology, 15th/e, P. 112, f.8.6
Jeffcoate’s Principles of Gynaecology, 8th/e, P. 214 853
 PRIMES (Volume II)
18. A 17 year-old female complains of primary amenorrhoea. She
has bilateral inguinal hernias, normal sexual development
but no pubic hair. USG shows absent uterus and vagina.
What is the most likely diagnosis? (PGMEE 2012)
a. Mullerian agenesis a. Anosmia
b. Androgen insensivity syndrome
c. Turner’s syndrome d. None
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 373;
Shaw’s 15th/e p. 95; 14th/e p. 81
Explanation
ƒƒ Testicular feminization is char/ by lack of axillary and
public hairs, absent uterus and upper vagina (Vaginal
pouch present) but normal breast development. Full
sexual development is achieved by 18-20 years of
age. Also k/as Androgen insensitivity Syndrome.
Effects of androgens do not manifest in the patient →
No axillary and public hairs
○○ B/L inguinal hernia is nothing but presence of testes in
inguinal region.
,2
ƒƒ Turner’s Syndrome is char/ by primary amenorrhea
+ lack of secondary sexual characters (no breast
development).
18. b
ƒƒ Rokitansky Küster Hauser Syndrome (RMKH
19. b
syndrome) is char/ by eugonadotropc prim
Es
20. c
amenorrhea, absent/ non-functional uterus and
21. a
vestigial vagina. Ovulation and secondary sexual
22. b characters are normal (normal breast) since ovaries
23. a are functional.
24. a 19. A female coming with normal breast development but
iM
25. a scanty pubic hair is suffering from? (PGMEE 2011)
26. d a. Gonadal disgenesis
b. Testicular feminizing syndrome
27. b
c. Mullerian agenesis
28. b
d. Turner’s syndrome
PR
29. d
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P373; Shaw’s
Textbook of Gynaecology, 15th/e, P. 95-96, 110-112; Jeffcoate’s
Principles of Gynaecology, 8th/e, P. 182-184, 212-214, 224-225
20. A 15 year old female presents with primary amenorrhea.
Her breasts are Tanner 4 but she has no axillary or public
hair. The most likely diagnosis is- (PGMEE 2006)
a. Turner’s syndrome
b. Mullerian agenesis
c. Testicular feminization syndrome
d. Premature ovarian failure
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 373;
Harrison 16th/e p. 2218]
21. Which type of abnormality in sexual development has best
prognosis? (AIIMS Nov 2013)
a. Congenital adrenal hyperplasia
b. Mixed gonadal dysgenesis
c. Androgen insensitivity syndrome
d. True hermaphroditism
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P367-368]
854
KALLMAN SYNDROME
22. Kallmans syndrome is associated with all of the following
except- (PGMEE 2015)
b. Excess stimulation of the HPO axis
c. Amenorrhea
d. Genetic mutation
[Ref: Speroff clinical endocrinology, 8th/e p. 492; William’s
Gynaecology 2nd/e p. 447]
23. A girl with normal stature and minimal or absent pubertal
development is seen in- (AIIMS Nov 14)
a. Kallman syndrome
b. Testicular feminization sundrome
c. Turner’s syndrome
d. Pure gonadal dysgenesis
[Ref: Nelson textbook of Pediatrics 19th/e p. 1956]
/e
24. A 16 year old girl presents with primary amenorrhoea
and lack of secondary sexual characters with anosmia, the
most probable diagnosis is? (PGMEE Aug 13 Pattern)
a. Kallmann’s syndrome b. Turner’s syndrome
c. Down’s syndrome d. Klienfelter’s syndrome
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 383,373
SECONDARY AMENORRHEA
25. Most common feature of Ashermann syndrome is:-
 (PGMEE 2016-17)
a. Hypomenorrhoea b. Amenorrhoea
c. Infertility d. Dyspareunia
[Ref: D.C Dutta text book of gynaecology 7th e p 378; speroff
8th e p.459]
26. HSG of Uterus showing adhesions. The patient is suffering
from:- (PGMEE 2016-17)
a. Septate uterus b. Fibroma
c. Bicornuate uterus d. Ashermann’s syndrome
 [Ref: D.C Dutta text book of gynaecology 7th e p 378]
27. Investigation of choice for endometrial synechiae is-
 (PGMEE Aug 13 Pattern)
a. Endometrial sampling b. Hysteroscopy
c. Colposcopy d. Ultrasound
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 378
Novak’s 15th/e p. 786-2 & 14th/e p. 787; Shaw’s Gynae 15th/e
p. 494, Williams Gynae 1st/e p. 950-1]
28. False about Ashermann’s syndrome :-  (PGMEE 2012-13)
a. Associated with menstrual irregularities
b. Progesterone challenge test is positive
c. Synechiae formation in uterus
d. May be secondary to TB
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 378]
29. Not a cause for secondary amenorrhoea(PGMEE 2012-13)
a. Pregnancy b. PCOD
c. Cushing syndrome d. Turner syndrome
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 371-
377; Shaw’s Gynae 15th/e p. 287—288]
Gynecology  Chapter 13 Amenorrhea (MCQs)

30. Sheehan’s syndrome is due to- (PGMEE 2012-13)
a. Ovarian necrosis b. Hypothalamus necrosis
c. Pituitary necrosis d. Thyroid necrosis
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 384]
31. Most common cause of Sheehan’s syndrome :-
 (PGMEE 2012-13)
a. PPH b. Tubercular endometritis
c. Amenorrhea d. Oligomenorrhea
 [Ref D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 378]
32. All are causes of anovulatory amenorrhoea except-
 (PGMEE 2012-13)
a. PCOD b. Hyperprolactemia
c. Gondal dysgenesis d. Drugs
 [Ref: Leon & speroff 8th e pg 454]
MALFORMATION OF THE FEMALE
34. Investigation of choice for septate uterus is?
 (PGMEE Nov 13 Pattern)
a. HSG b. USG
c. MRI d. Laproscopy
[Ref: Internet, Danforth’s obstetrics and gynecology 10th/e p.
550; William’s 22nd/e ch. 40]
35. Unilateral dysmenorrhoea occurs in?
 (PGMEE Aug 13 Pattern)
a. Uterus didelphys
b. Bicornuate uterus
c. Septate uterus
d. Uterus with rudimentary horn
 [Ref: Jeffcoates principles of gynaecology 7th/e p. 202]
36. The diagnosis of uterine anomalies is-
a. Hysterosalpingography b. MRI
c. CT Scan d. Plain radiography

/e
[Ref: Shaw’s Gynae 15th/e p. 501]
GENERATIVE ORGANS
33. Most common congenital uterine anomaly is-
 (PGMEE 2013-14)


a. Bicronuate uterus
c. Unicornuate uterus
b. Septate uterus
d. Arcuate uterus

,2
[Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P 35,Leon
Speroff 7th/e p. 132]
30.
31.
c
a
Es
32. c
33. b
34. c
35. d
36. a
iM
PR

855
 CHAPTER 14: MISCELLANEOUS

PUBERTY AND ITS DISORDERS
1. 1st sign of puberty:- (PGMEE 2008, 2016-17)
a. Growth spurt b. Pubic hair growth
c. Breast budding d. Menstruation
 [Ref: D.C Dutta text book of gynaecology 7th e p 39]
2. All are true about precocious puberty except-(PGMEE 2015)
a. Most common cause is constitutional
b. Secondary sexual characters before the age of 6 years
c. Menstruation before the age of 10 years
d. Secondary sexual characters before the age of 8 years
[Ref: Shaw’s Gyanecology 16th/e p. 59; Speroff Clinical
Endocrinology 8th/e p. 408]
9. Lithotomy position increase vaginal opening by how many
cm- (PGMEE 2013-14)
a. 1 cm b. 2 cm
c. 3 cm d. 4cm
 [Ref: Dutta Obs 7th/e p. 93]
10. Endometrial biopsy is usually done at- (PGMEE 2013-14)
a. Just before menstruation
b. 10-12 days after menstruation
c. Just after menstruation
d. At the time of ovulation
 [Ref: Shaw’s Gynae 15th/e p. 86]
11. Best way to look at endometrial activity is by-
 (PGMEE 2013-14)

/e
3. First sign of puberty in females is? (PGMEE 2009) a. HSG b. Biopsy
a. Pubrache b. Thelarche c. USG d. Colposcopy
c. Increase in height d. Menarche
12. Pseudocyesis is common in:- (PGMEE 2016-17)
 [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 39] a. Woman nearing menopause

1.
2.
c
b



,2
4. All are true about puberty menorrhagia except:
(PGMEE 2010)
a. Hematinics and hormone therapy is the treatment of choice
b. Endometrial biopsy confirms diagnosis


b. Young women with multiple contacts


c. Contraceptive failure
[Ref: D.C Dutta text book of gynaecology 7th e p 471]
13. Most radiosensitive phase of cell cycle is:-
Es
3. b c. Routine screening for bleeding disorder is done
 (PGMEE 2016-17)
4. b d. Associated with anovulatory bleeding
a. Early s phase b. Late s phase
5. a [Ref: D. C. Dutta’s Textbook of gynaecology, 7th/e, P. 43,156 c. G2M d. G1S
6. c Shaw’s Textbook of Gynaecology, 15th/e, Page no. 301-2 Berek
 [Ref: D.C Dutta text book of gynaecology 7th e p 412]
& Novak’s Gynecology, 15th/e, P. 393-4; Progress in Obstetrics
7. c
& Gynaecology, John Studd, Vol. 18, Ch. 20 , P. 305-6] 14. Vaginimus is:- (PGMEE 2016)
iM

8. b a. Painful sexual intercourse

9. b 5. Most appropriate time for breast self-examination is? 
b. Premature orgasm
a. A week after menstruation starts  (PGMEE 2010)
10. a c. Inability to initiate sexual intercourse
b. A day after menstruation ends
11. b d. Excesscive desire
c. During menstruation
PR

12. a d. Before ovulation
13. c [Ref: Jeffcoate’s Principles of Gynaecology, 8th/e, P. 169;
14. a Berek & Novak’s Gynecology, 15th/e, P. 482 & 656
15. b
6. Perineal tear involving all layer except anal mucosa
belongs to (PGMEE 2016-17)
a. 1° b. 2°
c. 3° d. 4°
 [Ref: D.C Dutta text book of gynaecology 7th e p353]
7. Following delivery, tear involves perineum, external and
sphinctor with intact mucosa, grade of tear is-
 (PGMEE 2015)
a. First degree b. Second degree
c. Third degree d. Fourth degree
 [Ref: Dutta’s Obstetrics 8th/e p. 489]
8. PAP smear invented by? (PGMEE 2013)
a. John papanicolaou b. George Papanicolaou
c. Vladimir papanicolaou d. Ben papnicolau
[Ref: Cervical cancer: Current and emerging trends in detec-
tion and treatment by Heather Hasan p. 28]
856
 [Ref: D.C Dutta text book of gynaecology 7th e p.469]
15. Which of the following is true about vulvodynia?
a. Surgery for localized provoked pain  (PGMEE 2016)
b. Pain without lesion
c. Strong association with irritable bowel syndrome
d. Hyperalgesia to touch
 [Ref: D.C Dutta text book of gynaecology 7th e p.215] 1. (C)
Imperforate hymen
Explanation
Kartagener Syndrome
ƒƒ Testicular feminization is char/ by lack of axillary and
public hairs, absent uterus and upper vagina (Vaginal)
ƒƒ Kartagener Syndrome is situs inversus, chronic
sinusitis, and bronchiectasis. Kartagener syndrome is
a/w male infertility d/to absent or poor sperm motility.
In females it manifest as tubal dysmotility and
infertility. Primary ciliary dyskinesia (PCD) includes
ciliary ultrastructural changes (lack of dynein arms,
microtubular transpositions, compound cilia, radial
spokes and nexin link defects etc. All these changes
result in immotile cilia syndrome.