0015TH00561015 981847q PDF

DIAGNOSTIC REPORT
CLIENT CODE : C000104835 Cert. No. M-2261
CLIENT'S NAME AND ADDRESS : SRL LIMITED

WELL BEING PATH CARE B-22, SECTOR-62
SHOP NO. 3, GROUND FLOOR, ROYAL AVENUE, NOIDA, 201301
SARFABAD, SECTOR-73, UTTAR PRADESH, INDIA
NOIDA 201307 Tel : 0120-2403338, Fax :
UTTAR PRADESH INDIA CIN - U74899PB1995PLC045956
9953526284 Email : customercare.noida@srl.in
PATIENT NAME : MRS. CHHAYA GUPTA PATIENT ID : FH.8219071
ACCESSION NO : 0015TH005610 AGE : 32 Years SEX : Female DATE OF BIRTH : 03/06/1988
DRAWN : 11/08/2020 09:43 RECEIVED : 11/08/2020 13:48 REPORTED : 11/08/2020 14:50
REFERRING DOCTOR : DR. RAKESH OJHA CLIENT PATIENT ID :
Test Report Status Preliminary Results Biological Reference Interval Units
LIVER & KIDNEY PROFILE
ASPARTATE AMINOTRANSFERASE, SERUM

ASPARTATE AMINOTRANSFERASE (AST/SGOT) 10 Upto 32 U/L
METHOD : UV WITHOUT P5P
ALANINE AMINOTRANSFERASE, SERUM

ALANINE AMINOTRANSFERASE (ALT/SGPT) 9 Upto 33 U/L
METHOD : UV WITHOUT P5P
ALKALINE PHOSPHATASE, SERUM

ALKALINE PHOSPHATASE 44 35 - 105 U/L
METHOD : PNPP, AMP BUFFER-IFCC
BILIRUBIN (TOTAL, DIRECT, INDIRECT), SERUM

BILIRUBIN, TOTAL 0.26 UPTO 1.2 mg/dL
METHOD : DIAZONIUM ION, BLANKED (ROCHE)
BILIRUBIN, DIRECT 0.11 0.00 - 0.30 mg/dL

METHOD : DIAZOTIZATION
BILIRUBIN, INDIRECT 0.15 0.00 - 0.60 mg/dL

METHOD : CALCULATED PARAMETER
GAMMA GLUTAMYL TRANSFERASE, SERUM

GAMMA GLUTAMYL TRANSFERASE (GGT) 28 5 - 36 U/L
METHOD : G-GLUTAMYL-CARBOXY-NITROANILIDE-IFCC
LACTATE DEHYDROGENASE, SERUM

LACTATE DEHYDROGENASE 168 135 - 214 U/L
METHOD : L TO P, IFCC
ALBUMIN+GLOBULIN+A/G RATIO, SERUM

ALBUMIN 3.6 Low 3.97 - 4.94 g/dL
METHOD : BROMOCRESOL GREEN
GLOBULIN 2.3 2.0 - 4.0 g/dL

ALBUMIN/GLOBULIN RATIO 1.6 RATIO
TOTAL PROTEIN, SERUM
TOTAL PROTEIN 5.9 Low 6.6 - 8.7 g/dL
METHOD : BIURET,SERUM BLANK,ENDPOINT
SERUM BLOOD UREA NITROGEN

BLOOD UREA NITROGEN 10 6 - 20 mg/dL
METHOD : UREASE - UV
CREATININE, SERUM
CREATININE 0.48 Low 0.50 - 0.90 mg/dL
Page 1 Of 7
DIAGNOSTIC REPORT

NOIDA 201307 Tel : 0120-2403338, Fax :
METHOD : ALKALINE PICRATE-KINETIC
Reference:
Circulation 1997 Oct 21;96(8):2520-5.
Current Atherosclerosis Reports 2007, 9:367–374
Indian J Med Res. 2015 Jan; 141(1): 68–74
Indian Heart Journal 69 (2017) 382–392
Diabetes Care 2017;40:529–537
*The background is coloured according to the risk levels of Triglycerides and HDL as per current worldwide guidelines.
Green zone: Desirable
Red zones: increasingly darker shades (1 to 4) as per risk level increase.
BUN/CREAT RATIO
BUN/CREAT RATIO 20.83 High 5.00 - 15.00
ELECTROLYTES (NA/K/CL), SERUM

SODIUM 140 136 - 145 mmol/L
METHOD : ISE INDIRECT
POTASSIUM 4.30 3.5 - 5.1 mmol/L

CHLORIDE 102 98 - 107 mmol/L

URIC ACID, SERUM

URIC ACID 2.9 2.4 - 5.7 mg/dL
METHOD : URICASE, COLORIMETRIC
URINALYSIS RESULT PENDING
Interpretation(s)
ASPARTATE AMINOTRANSFERASE, SERUM-Aminotransferase (AST) is an enzyme found in various parts of the body .AST is found in the liver, heart, skeletal muscle, kidneys,
brain, and red blood cells, and it is commonly measured clinically as a marker for liver health. AST levels increase during chronic viral hepatitis, blockage of the bile duct,
cirrhosis of the liver, liver cancer, kidney failure, hemolytic anemia, pancreatitis, hemochromatosis. AST levels may also increase after a heart attack or strenuous activity.
ALANINE AMINOTRANSFERASE, SERUM-Alanine aminotransferase (ALT) test measures the amount of this enzyme in the blood. ALT is found mainly in the liver, but also in
smaller amounts in the kidneys, heart, muscles, and pancreas. It is commonly measured as a part of a diagnostic evaluation of hepatocellular injury, to determine liver
health. . AST levels increase during acute hepatitis, sometimes due to a viral infection, ischemia to the liver, chronic hepatitis, obstruction of bile ducts, cirrhosis.
ALKALINE PHOSPHATASE, SERUM-Alkaline phosphatase (ALP) is a protein found in almost all body tissues. Tissues with higher amounts of ALP include the liver, bile ducts,
Page 2 Of 7
DIAGNOSTIC REPORT

NOIDA 201307 Tel : 0120-2403338, Fax :
and bone. Elevated Alkaline Phosphaqtase levels are seen in Biliary obstruction,Osteoblastic bone tumors, osteomalacia, hepatitis, Hyperparathyroidism,Leukemia,
Lymphoma,Paget''''''''s disease,Rickets,Sarcoidosis etc. Lower-than-normal ALP levels seen in Hypophosphatasia, Malnutrition, Protein deficiency,Wilson''''''''s disease .
BILIRUBIN (TOTAL, DIRECT, INDIRECT), SERUM-BILIRUBIN (TOTAL, DIRECT, INDIRECT), SERUM
Bilirubin is a yellowish pigment found in bile and is a breakdown product of normal heme catabolism. Bilirubin is excreted in bile and urine, and elevated levels may give
yellow discoloration in jaundice.Elevated levels results from increased bilirubin production (eg, hemolysis and ineffective erythropoiesis), decreased bilirubin excretion (eg,
obstruction and hepatitis), and abnormal bilirubin metabolism (eg, hereditary and neonatal jaundice). Conjugated (direct) bilirubin is elevated more than unconjugated
(indirect) bilirubin in Viral hepatitis, Drug reactions, Alcoholic liver disease Conjugated (direct) bilirubin is also elevated more than unconjugated (indirect) bilirubin when
there is some kind of blockage of the bile ducts like in Gallstones getting into the bile ducts, tumors & Scarring of the bile ducts. Increased unconjugated (indirect) bilirubin
may be a result of Hemolytic or pernicious anemia, Transfusion reaction & a common metabolic condition termed Gilbert syndrome, due to low levels of the enzyme that
attaches sugar molecules to bilirubin.
Total Bili-
Source: Wallach"s Interpretation of Diagnostic tests, 9th ed
Direct Bili -
Source: Tietz Text book of Clinical Chemistry & Molecular Diagnostics, 4th ed
d
GAMMA GLUTAMYL TRANSFERASE, SERUM-Gamma glutamyl transferase (GGT) is an enzyme found in cell membranes of many tissues mainly in the liver, kidney, and
pancreas. It is also found in other tissues including intestine, spleen, heart, brain, and seminal vesicles. The highest concentration is in the kidney, but the liver is considered
the source of normal enzyme activity. Serum gamma-glutamyl transferase (GGT) has been widely used as an index of liver dysfunction. Elevated serum GGT activity can be
found in diseases of the liver, biliary system, and pancreas .Conditions that increase serum GGT are obstructive liver disease, high alcohol consumption, and use of
enzyme-inducing drugs etc.
LACTATE DEHYDROGENASE, SERUM-LDH is an enzyme that helps in energy production. It is present in almost all of the tissues in the body and its levels rise in response to
cell damage. LDH levels help to diagnose lung disease, lymphoma, anemia, and liver disease. They also help determine how well chemotherapy is working .A
higher-than-normal level may indicate:Blood flow deficiency (ischemia), Heart attack, Hemolytic anemia, Infectious mononucleosis, Liver disease (for example, hepatitis),Low
blood pressure,Muscle injury, muscular dystrophy, New abnormal tissue formation usually cancer, Pancreatitis and Stroke.
ALBUMIN+GLOBULIN+A/G RATIO, SERUM-ALBUMIN+GLOBULIN+A/G RATIO, SERUM
Serum total protein,also known as total protein, is a biochemical test for measuring the total amount of protein in serum..Protein in the plasma is made up of albumin and
globuli.
Higher-than-normal levels may be due to: Chronic inflammation or infection, including HIV and hepatitis B or C, Multiple myeloma, Waldenstrom''''s disease
Lower-than-normal levels may be due to: Agammaglobulinemia, Bleeding (hemorrhage),Burns ,Glomerulonephritis, Liver disease, Malabsorption,Malnutrition,Nephrotic
syndrome,Protein-losing enteropathy etc.Human serum albumin is the most abundant protein in human blood plasma. It is produced in the liver. Albumin constitutes about
half of the blood serum protein. Low blood albumin levels (hypoalbuminemia) can be caused by:Liver disease like cirrhosis of the liver, nephrotic syndrome, protein-losing
enteropathy,Burns,,hemodilution, increased vascular permeability or decreased lymphatic clearance,malnutrition and wasting etc.
TOTAL PROTEIN, SERUM-Serum total protein,also known as total protein, is a biochemical test for measuring the total amount of protein in serum..Protein in the plasma is
made up of albumin and globulin
Higher-than-normal levels may be due to: Chronic inflammation or infection, including HIV and hepatitis B or C, Multiple myeloma, Waldenstrom''''''''s disease
Lower-than-normal levels may be due to: Agammaglobulinemia, Bleeding (hemorrhage),Burns,Glomerulonephritis, Liver disease, Malabsorption, Malnutrition, Nephrotic
syndrome,Protein-losing enteropathy etc.
SERUM BLOOD UREA NITROGEN-Causes of Increased levels
Pre renal
• High protein diet, Increased protein catabolism, GI haemorrhage, Cortisol, Dehydration, CHF Renal
• Renal Failure
Post Renal
• Malignancy, Nephrolithiasis, Prostatism
Causes of decreased levels

• Liver disease
• SIADH.
CREATININE, SERUM-Higher than normal level may be due to:
• Blockage in the urinary tract
• Kidney problems, such as kidney damage or failure, infection, or reduced blood flow
• Loss of body fluid (dehydration)
• Muscle problems, such as breakdown of muscle fibers
• Problems during pregnancy, such as seizures (eclampsia)), or high blood pressure caused by pregnancy (preeclampsia)
Lower than normal level may be due to:

• Myasthenia Gravis
• Muscular dystrophy
ELECTROLYTES (NA/K/CL), SERUM-ELECTROLYTES (NA/K/CL), SERUM
Sodium levels are Increased in dehydration, cushing''''''''s syndrome, aldosteronism & decreased in Addison''''''''s disease, hypopituitarism,liver disease. Hypokalemia (low K)
is common in vomiting, diarrhea, alcoholism, folic acid deficiency and primary aldosteronism. Hyperkalemia may be seen in end-stage renal failure, hemolysis, trauma,
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DIAGNOSTIC REPORT

NOIDA 201307 Tel : 0120-2403338, Fax :
Addison''''''''s disease, metabolic acidosis, acute starvation, dehydration, and with rapid K infusion.Chloride is increased in dehydration, renal tubular acidosis (hyperchloremia
metabolic acidosis), acute renal failure, metabolic acidosis associated with prolonged diarrhea and loss of sodium bicarbonate, diabetes insipidus, adrenocortical hyperfuction,
salicylate intoxication and with excessive infusion of isotonic saline or extremely high dietary intake of salt.Chloride is decreased in overhydration, chronic respiratory acidosis,
salt-losing nephritis, metabolic alkalosis, congestive heart failure, Addisonian crisis, certain types of metabolic acidosis, persistent gastric secretion and prolonged vomiting,
URIC ACID, SERUM-Causes of Increased levels
Dietary
• High Protein Intake.
• Prolonged Fasting,
• Rapid weight loss.
Gout
Lesch nyhan syndrome.
Type 2 DM.
Metabolic syndrome.
Causes of decreased levels

• Low Zinc Intake
• OCP’s
• Multiple Sclerosis
Nutritional tips to manage increased Uric acid levels

• Drink plenty of fluids
• Limit animal proteins
• High Fibre foods
• Vit C Intake
• Antioxidant rich foods
HAEMATOLOGY
COMPLETE BLOOD COUNT, EDTA WHOLE

BLOOD/SMEAR
BLOOD COUNTS
HEMOGLOBIN 11.5 Low 12.0 - 15.0 g/dL
METHOD : SPECTROPHOTOMETRY
RED BLOOD CELL COUNT 3.42 Low 3.8 - 4.8 mil/µL

METHOD : ELECTRICAL IMPEDANCE
WHITE BLOOD CELL COUNT 7.20 4.0 - 10.0 thou/µL

PLATELET COUNT 336 150 - 410 thou/µL

RBC AND PLATELET INDICES

HEMATOCRIT 34.8 Low 36.0 - 46.0 %
MEAN CORPUSCULAR VOL 101.8 High 83.0 - 101.0 fL

METHOD : DERIVED/COULTER PRINCIPLE
MEAN CORPUSCULAR HGB. 33.8 High 27.0 - 32.0 pg

MEAN CORPUSCULAR HEMOGLOBIN 33.2 31.5 - 34.5 g/dL

CONCENTRATION
RED CELL DISTRIBUTION WIDTH 17.2 High 11.6 - 14.0 %
Page 4 Of 7
DIAGNOSTIC REPORT

NOIDA 201307 Tel : 0120-2403338, Fax :
MEAN PLATELET VOLUME 7.1 6.8 - 10.9 fL

WBC DIFFERENTIAL COUNT

SEGMENTED NEUTROPHILS 61 40 - 80 %
METHOD : VCS TECHNOLOGY/ MICROSCOPY
ABSOLUTE NEUTROPHIL COUNT 4.39 2.0 - 7.0 thou/µL

EOSINOPHILS 1 1.0 - 6.0 %

ABSOLUTE EOSINOPHIL COUNT 0.07 0.02 - 0.50 thou/µL

LYMPHOCYTES 35 20 - 40 %
ABSOLUTE LYMPHOCYTE COUNT 2.52 1.0 - 3.0 thou/µL

MONOCYTES 3 2.0 - 10.0 %

ABSOLUTE MONOCYTE COUNT 0.22 0.2 - 1.0 thou/µL

BASOPHILS 0 0-1 %
ABSOLUTE BASOPHIL COUNT 0.00 Low 0.02 - 0.10 thou/µL

DIFFERENTIAL COUNT PERFORMED ON: EDTA SMEAR
Interpretation(s)
BLOOD COUNTS-The cell morphology is well preserved for 24hrs. However after 24-48 hrs a progressive increase in MCV and HCT is observed leading to a decrease in MCHC.
A direct smear is recommended for an accurate differential count and for examination of RBC morphology.
RBC AND PLATELET INDICES-The cell morphology is well preserved for 24hrs. However after 24-48 hrs a progressive increase in MCV and HCT is observed leading to a
decrease in MCHC. A direct smear is recommended for an accurate differential count and for examination of RBC morphology.
COAGULATION
PROTHROMBIN TIME, PLASMA
PROTHROMBIN TIME (PT) 13.4 11.42 - 14.78 SECONDS

METHOD : VISCOSITY BASED DETECTION SYSTEM
INTERNATIONAL NORMALIZED RATIO (INR) 1.02 < 1.5 RATIO

MEAN NORMAL PT 13.1 13.1 SECONDS
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DIAGNOSTIC REPORT

NOIDA 201307 Tel : 0120-2403338, Fax :
METHOD : VISCOSITY BASED DETECTION SYSTEM AND CALCULATION
ACT PARTIAL THROMBO PLASTIN TIME

(APTT), PLASMA
APTT 30.7 24.58 - 31.78 SECONDS

METHOD : VISCOSITY BASED DETECTION SYSTEM
APTT CONTROL 28.1 28.1 SECONDS

METHOD : VISCOSITY BASED DETECTION SYSTEM AND CALCULATION
Interpretation(s)
PROTHROMBIN TIME, PLASMA-Prothrombin Time measures the integrity of the extrinsic pathway and the adequacy of critical coagulation factors involved in it, namely, Factor
VII. This test is therefore, used for monitoring oral anticoagulation therapy which lowers the levels of multiple vitamin K dependent coagulation factors in blood (Factors II,
VII, IX and X) including Factor VII. The result of PT is expressed as International Normalized Ratio (INR) to neutralize the influence of variable sensitivity of the reagents
(thromboplastin) used in the assay by different laboratories.
Prolonged PT/INR is observed in hereditary or acquired deficiency of the relevant coagulation factors, vitamin K deficiency, liver disease, specific coagulation factor inhibitors
and nonspecific inhibitors of PT (eg, monoclonal immunoglobulins, elevated fibrin degradation products).
The following INR ranges are recommended for achieving optimal anticoagulation in different clinical conditions:
Diagnosis Target INR
Treatment of venous thrombosis 2.0 - 3.0

Treatment of pulmonary embolism 2.0 - 3.0
Prevention of systemic embolism 2.0 - 3.0
Tissue heart valves 2.0 - 3.0
Hypercoagulable states 2.0 - 3.0
Atrial fibrillation 2.0 - 3.0
Mechanical prosthetic valves (high risk) 2.5 - 3.5
Bileaflet mechanical valve in aortic position 2.0 - 3.0
ACT PARTIAL THROMBO PLASTIN TIME(APTT), PLASMA-The activated partial thromboplastin time (APTT) reflects the activities of most of the coagulation factors, including
factor XII and other ""contact factors"" (prekallikrein [PK] and high molecular weight kininogen [HMWK]) and factors XI, IX, and VIII in the intrinsic coagulation pathway, as
well as coagulation factors in the common coagulation pathway that include factors X, V, II and fibrinogen (factor I). The APTT also depends on phospholipid (a partial
thromboplastin) and ionic calcium, as well as the activator of the contact factors (eg, silica) present in the reagent, but reflects neither the integrity of the extrinsic
coagulantion pathway that includes factor VII and tissue factor, nor the activity of factor XIII (fibrin stabilizing factor). The APTT is variably sensitive to the presence of
specific and nonspecific inhibitors of the intrinsic and common coagulation pathways, including lupus anticoagulants or antiphospholipid antibodies. It is useful for monitoring
unfractionated heparin therapy, for screening for certain coagulation factor deficiencies, detection of coagulation inhibitors such as lupus anticoagulant, specific factor
inhibitors, and nonspecific inhibitors.
APTT “mixing” studies:

Poor or partial correction of the abnormal result by normal plasma may be observed in the presence of coagulation factor inhibitors, anticoagulant drugs such as heparin or
direct thrombin inhibitors. Total correction indicates coagulation factors deficiency.
**End Of Report**
Please visit www.srlworld.com for related Test Information for this accession
Dr. Shyla Goel,M.B.B.S ,DCP

Pathologist
Page 6 Of 7
DIAGNOSTIC REPORT

NOIDA 201307 Tel : 0120-2403338, Fax :
CONDITIONS OF LABORATORY TESTING & REPORTING
1. It is presumed that the test sample belongs to the 5. The results of a laboratory test are dependent on
patient named or identified in the test requisition form. the quality of the sample as well as the assay
2. All Tests are performed and reported as per the technology.
turnaround time stated in the SRL Directory of services 6. Result delays could be because of uncontrolled
(DOS). circumstances. e.g. assay run failure.
3. SRL confirms that all tests have been performed or 7. Tests parameters marked by asterisks are excluded
assayed with highest quality standards, clinical safety & from the “scope" of NABL accredited tests. (If
technical integrity. laboratory is accredited).
4. A requested test might not be performed if: 8. Laboratory results should be correlated with clinical
a. Specimen received is insufficient or inappropriate information to determine Final diagnosis.
specimen quality is unsatisfactory 9. Test results are not valid for Medico- legal purposes.
b. Incorrect specimen type 10. In case of queries or unexpected test results please
c. Request for testing is withdrawn by the ordering call at SRL customer care (Toll free: 1800-222-000).
doctor or patient Post proper investigation repeat analysis may be carried
d. There is a discrepancy between the label on the out.
specimen container and the name on the test
requisition form
SRL Limited
Fortis Hospital, Sector 62, Phase VIII,

Mohali 160062
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DIAGNOSTIC REPORT

CLIENT CODE : C000104835 Cert. No. M-2261

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : MRS. CHHAYA GUPTA PATIENT ID : FH.8219071

ACCESSION NO : 0015TH005610 AGE : 32 Years SEX : Female DATE OF BIRTH : 03/06/1988

DRAWN : 11/08/2020 09:43 RECEIVED : 11/08/2020 13:48 REPORTED : 11/08/2020 14:50

REFERRING DOCTOR : DR. RAKESH OJHA CLIENT PATIENT ID :

Test Report Status Preliminary Results Biological Reference Interval Units

LIVER & KIDNEY PROFILE

ASPARTATE AMINOTRANSFERASE, SERUM

ALANINE AMINOTRANSFERASE, SERUM

ALKALINE PHOSPHATASE, SERUM

BILIRUBIN (TOTAL, DIRECT, INDIRECT), SERUM

BILIRUBIN, DIRECT 0.11 0.00 - 0.30 mg/dL

BILIRUBIN, INDIRECT 0.15 0.00 - 0.60 mg/dL

GAMMA GLUTAMYL TRANSFERASE, SERUM

LACTATE DEHYDROGENASE, SERUM

ALBUMIN+GLOBULIN+A/G RATIO, SERUM

GLOBULIN 2.3 2.0 - 4.0 g/dL

SERUM BLOOD UREA NITROGEN

CLIENT CODE : C000104835 Cert. No. M-2261

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : MRS. CHHAYA GUPTA PATIENT ID : FH.8219071

ACCESSION NO : 0015TH005610 AGE : 32 Years SEX : Female DATE OF BIRTH : 03/06/1988

DRAWN : 11/08/2020 09:43 RECEIVED : 11/08/2020 13:48 REPORTED : 11/08/2020 14:50

REFERRING DOCTOR : DR. RAKESH OJHA CLIENT PATIENT ID :

Test Report Status Preliminary Results Biological Reference Interval Units

METHOD : ALKALINE PICRATE-KINETIC

ELECTROLYTES (NA/K/CL), SERUM

POTASSIUM 4.30 3.5 - 5.1 mmol/L

CHLORIDE 102 98 - 107 mmol/L

URIC ACID, SERUM

URINALYSIS RESULT PENDING

CLIENT CODE : C000104835 Cert. No. M-2261

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : MRS. CHHAYA GUPTA PATIENT ID : FH.8219071

ACCESSION NO : 0015TH005610 AGE : 32 Years SEX : Female DATE OF BIRTH : 03/06/1988

DRAWN : 11/08/2020 09:43 RECEIVED : 11/08/2020 13:48 REPORTED : 11/08/2020 14:50

REFERRING DOCTOR : DR. RAKESH OJHA CLIENT PATIENT ID :

Test Report Status Preliminary Results Biological Reference Interval Units

Causes of decreased levels

Lower than normal level may be due to:

CLIENT CODE : C000104835 Cert. No. M-2261

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : MRS. CHHAYA GUPTA PATIENT ID : FH.8219071

ACCESSION NO : 0015TH005610 AGE : 32 Years SEX : Female DATE OF BIRTH : 03/06/1988

DRAWN : 11/08/2020 09:43 RECEIVED : 11/08/2020 13:48 REPORTED : 11/08/2020 14:50

REFERRING DOCTOR : DR. RAKESH OJHA CLIENT PATIENT ID :

Test Report Status Preliminary Results Biological Reference Interval Units

Causes of decreased levels

Nutritional tips to manage increased Uric acid levels

COMPLETE BLOOD COUNT, EDTA WHOLE

RED BLOOD CELL COUNT 3.42 Low 3.8 - 4.8 mil/µL

WHITE BLOOD CELL COUNT 7.20 4.0 - 10.0 thou/µL

PLATELET COUNT 336 150 - 410 thou/µL

RBC AND PLATELET INDICES

MEAN CORPUSCULAR VOL 101.8 High 83.0 - 101.0 fL

MEAN CORPUSCULAR HGB. 33.8 High 27.0 - 32.0 pg

MEAN CORPUSCULAR HEMOGLOBIN 33.2 31.5 - 34.5 g/dL

RED CELL DISTRIBUTION WIDTH 17.2 High 11.6 - 14.0 %

CLIENT CODE : C000104835 Cert. No. M-2261

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : MRS. CHHAYA GUPTA PATIENT ID : FH.8219071