Medical – Surgical Nursing
(NUR 155) | BATCH 2023
COLLEGE OF NURSING – PHINMA UNIVERSITY OF PANGASINAN
HYPERTENSION
- Sustained, elevated, systemic, arterial blood pressure; diastolic
elevation more serious, reflecting pressure on arterial wall during
resting phase of cardiac cycle
- Persistent elevation of blood pressure for 2 or more consecutive
readings
CLASSIFICATIONS
(according to type and degree of severity)
1. Systolic and Diastolic Hypertension
a. Systolic HPN is systolic BP of 140 mmHg or higher RISK FACTORS:
b. Diastolic HPN is diastolic BP of 90 mmHg or higher Non-modifiable
- Family history
2. Primary and Secondary HPN - Age
a. Primary/Essential/Idiopathic HPN – occurs in 90-95% of - Sex
patients; etiology unknown; diastolic pressure is > 90
- Ethnic group
mmHg, and other causes of hypertension are absent
b. Secondary HPN – occurs in remaining 5-10% usually of
Modifiable
renal, endocrine, neurogenic or cardiac in origin
- Stress
3. “White-Coat” Hypertension
- Obesity
- HPN in a person who is actually normotensive except when
- Diet
his/her BP is measured by a healthcare professional
- Sedentary Lifestyle
- Substance/Drug Use:
4. Malignant Hypertension
- Cigarette Smoking
- Uncontrollable and may arise from both types and certain
- Alcohol
drugs (e.g., anesthesia)
- An emergency condition characterized by diastolic BP above - Birth Control Pills
120 mmHg - Caffeine
- Smoking cessation
5. Labile (prehypertensive)
- A fluctuating blood pressure increases during stress,
PATHOPHYSIOLOGY:
otherwise normal or near normal
Four control systems that play major roles in maintaining blood pressure
1. Arterial baroreceptor system
2. Regulation of body fluid volume
3. Renin-Angiotensin-Aldosterone system
4. Vascular Autoregulation
Clinical Manifestations:
Subjective
- Headache
- Lightheadedness
- Tinnitus
- Easy fatigability
- Visual disturbances
PADAWAN, ANGELIKA R.| 3BSN3 1
Medical – Surgical Nursing
(NUR 155) | BATCH 2023
COLLEGE OF NURSING – PHINMA UNIVERSITY OF PANGASINAN
- Palpitations 5. Calcium-Channel Blockers (Calcium Antagonists)
- Brief lapses in memory - Action: Inhibit the entry of calcium into the heart and
vascular smooth muscle
Objective Decreases cardiac output
- BP greater than 140/90 Dilate blood vessels
- Retinal changes Lowers blood pressure
- Possible hematuria - Drugs:
- Cardiac hypertrophy Verapamil
Nifedipine
Diltiazem
Medical Management:
- Lifestyle modification
- Weight reduction
5. ACE Inhibitors
- Sodium restriction - Action: Suppress RAAS by blocking conversion of AI to AII
- Dietary fat modification - Drugs: PRIL
- Consideration: First dose – watch for hypotension and loss of taste
- Exercise
6. ARBs
- Relaxation techniques
- Block the vasoconstrictive effect of RAAS by blocking receptor sites
of AII
- Drugs: SARTAN
ANTIHYPERTENSIVE DRUGS
1. Alpha-Adrenergic Blockers GENERAL CONSIDERATIONS ON
- Action: decreases peripheral vascular resistance; relaxes ANTIHYPERTENSIVE MEDICATIONS:
smooth muscle of bladder/prostate Pressure monitoring (blood)
- Drugs: Medications that end in SIN
Rise slowly
2. Central Alpha Agonists Eating must be considered
- Action: decrease the release of adrenergic hormones from the
Stay on medications
brain (medulla oblongata) which decreases peripheral vascular
resistance and reduces cardiac contractility Skipping or stopping is a “No-no”
- Drugs: Clonidine (Catapress); Guanabenz (Wytensin); Undesirable responses assessment
Methyldopa (Aldomet)
- Consideration: Take last dose of the day at bedtime to Remind to exercise
minimize drowsiness during the day Eliminate smoking
3. Beta-Adrenergic Blockers
- Action: these drugs exert antihypertensive effects by:
Reducing contractility
Reducing release of renin
Reducing the cardiac output
- Drugs: medications that end in “olol” (e.g., Metropolol,
Propanolol, Nadolol)
4. Vasodilators
- Action: Direct relaxation of vascular smooth muscle
- Drugs: Hydralazine (Apresoline); Nitroprusside
- Considerations:
Assess for peripheral edema of the hands and feet
Take with food
Review of BP
PADAWAN, ANGELIKA R.| 3BSN3 2