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(English Vers) Laporan Pendahuluan Upload
(English Vers) Laporan Pendahuluan Upload
BASIC CONCEPTS
1. DEFINITION
Morbili is an acute, infectious viral disease characterized by 3 stages, namely the
prodormal (kataral) stage, eruption stage and convalisensive stage, which is
manifested by fever, conjunctivitis andhead spotting (Children's Health Sciences
Edition 2, 1991). FKUI). Morbili is a common infectious childhood disease usually
characterized by mild major symptoms, a rash similar to mild measles or fever,
scarlet, enlargement and spleen pain (Children's Health Sciences vol 2, Nelson, EGC,
2000)
2. ETIOLOGY
The cause is the morbili virus found in the nasopharynce secretion and blood during
the prodormal period up to 24 hours after the spots appear. This virus is an RNA virus
that belongs to the family Paramiksoviridae, genus Morbilivirus. The way it is
transmitted is with droplets of infection.
3. PATHOPHYSIOLOGY
The cause of measles is measles virus (MV), genus of morbili virus,
familyparamyxoviridae. The virus becomes inactive when exposed to heat, light,
acidic pH, ether, and trypsin and lasts less than 2 hours in the open air. The measles
virus is transmitted through droplets, attaches and multiplies to the nasopharyngeal
epithelium. The virus enters through the respiratory tract especially the upper part,
also possibly through the tear glands.
Two to three days after invasion, replication and colonization continues in the
regional lymph nodes and the first viremia occurs. The virus spreads in all
reticuloendothelial systems and follows a second viremia after 5-7 days of initial
infection. The presence of giant cells and the inflammatory process is the pathological
basis of rash and peribronchial infiltration of the lungs. There are also udema, dams
and bleeding that spread to the brain. Colonization and spread of the epithelium and
skin cause coughs, colds, red eyes (3 C: coryza, cough and conjuctivitis)and fevers
that are getting higher. Symptoms of heat, cough, cold are getting heavier and on the
10th day since the beginning of infection (on the day the patient comes into contact
with the source of infection) begins to appear a rash of makulopapuler reddish color.
The virus can multiply also in the central nervous system and cause clinical symptoms of
encefalitis. After the convelesent period on the descent and hypervascularization subsides
and causes the rash to get darker, turning into desquamation and hyperpigmentation. This
process is caused because at first there is pericular bleeding and lymphocyte infiltration.
1. CLINICAL SYMPTOMS
The period of budding / incubation of the disease lasts approximately 10-20 days and the
onset of symptoms that are divided into 3 stages:
The prodormal stage lasts for 4-5 days characterized by ringa to moderate fever, mild dry cough,
coryza, photophobia and conjunctivitis. Towards the end of the catalytic stage aand 24 hours before
enantema arises, corplic spotting is patognomonic for morbili, but is very rare. The koplic
patches are gray white, as big as the tip of a needle and surrounded by erythema.
The localization is webcosed openly opposite the molar below, but can spread irregularly
on the entire surface of the cheek. Although rare, they can also be found in the middle of the
lower lip, palate and lacrimal karankula. The patches appear and disappear quickly within 12-18
hours. Sometimes the prodormal stage is severe because it is accompanied by a sudden high fever
accompanied by convulsions and pneumoni. Peripheral blood images are lymphocytosis and
leukopenia.
B. Stage of eruption
Coryza and the coughing increased. Embossed erythema / red dots are hammered durum and
palate mole. The occurrence of erythema in the form of macula macula is accompanied by an
increase in body temperature. Erythema arises behind the ear at the top of the lateral nape of
the neck, along the hair and the lower back. Sometimes there is primary bleeding on the
skin. Itching, swollen face. There is an enlargement of the lymph nodes in the angle of the
mandible and in the area of the back neck. There is also a little splenomegaly, not
infrequently accompanied by diarrhea and vomiting. A variation of this common morbily is
"Black Measles" which is a morbily accompanied by bleeding on the skin, mouth, nose and
digestivus truck.
c. Stadium convalescence
Reduced eruptions leave older colored marks (hyperpigmentation) that can disappear on their
own. In addition to hyperpigmentation in Indonesian children, it is often found that scaly skin
is also found. Hyperpigmentation is a patognomonic symptom for morbili. In other diseases
with erythema or exantema the skin rash disappears without hyperpigmentation. The
temperature drops until it becomes normal unless there are complications.
Complications
b. Pneumonia/bronchopneumoni
c. Encefalitis
d. Bronchiolitis
6. NURSING DIAGNOTIC
On blood tests, the number of leukocytes is normal or increases if there are complications of
bacterial infection. IgM antibody screening is the fastest way to confirm the presence of an
acute measles infection. Because IgM may not be detectable in the first 2 days of rash, an
IgM blood test is done on the third day to avoid false negatives. IgM begin to be difficult to
measure at 4 weeks after the rash appears.
While IgG antibodies can be detected 4 days after the rash appears, most IgG can be detected
1 week after onset to 3 weeks after onset. IgG can still be found until a few years
later. Measles virus can be isolated from urine, nasopharyngeal aspirates, blood given
heparin, and throat swabs during prodromal times up to 24 hours after spotting. The virus can
remain active for at least 34 hours at room temperature.
7. NURSING MANAGEMENT
There are indications of sedative, antipyretic drugs to overcome high fever. Rest in bed and
adequate fluid intake. It may be necessary to humidicate the room for people with laryngitis
or cough bothersome and better simplify the warm room temperature.
The management of the cited:
a. Vitamin A
B. CONCEPT OF NURSING
1. Assessment
a. Self-identity
b. History of Immunization
c. Contact with an infected person
d. Physical examination:
1) Eyes: conjunctivitis, photophobia
2) Head: Headache
3) Nose: There are many secret, influenza, rhinitis / koriza, nasal bleeding (in stad
eripsi).
4) Mouth & lips: Mucosa dry lips, stomatitis, cough, mouth feels bitter.
5) Skin: Skin surface (dry), turgor skin, itching, maecular rash on the neck, face, arms
and legs (on stad. Convalence), evitema, heat (fever).
6) Breathing: Breathing pattern, RR, cough, shortness of breath, wheezing, renchi,
sputum.
7) Growth and Development: BB, TB, BB Born, Growth and Development R /
immunization.
8) Defecation Pattern: BAK, BAB, Diarrhea
9) Nutritional Status: intake – food output, appetite
3. NURSING ASSESMENT
A. A.A high risk of infection is associated with your generator and infectious agent.
Expected results:
3) The child does not show evidence of complications such as infection and dehydration.
INTERVENTION
Rational:
Expected results:
1) The skin and mucous membranes are clean and free from irritation.
2) The child shows evidence of minimum discomfort.
Intervention:
Expected results:
Intervention:
1) Explain the reasons for isolation and the use of special vigilance.
Rationale: to improve the child's understanding of the discussion.
2) Let the child play gloves and masks
Rationale: to facilitate positive coping.
3) Provide redirection activities
Rationale: to perform the right activities and interact
4) Encourage parents to stay with the child during hospitalization.
Rationale: to lower separation and provide closeness.
5) Prepare your child's peers for changes in physical appropriity
Rational: to encourage peer acceptance
D). A high risk of skin integrity damage is associated with pruritus scratching Expected
results: the skin remains intact
Intervention:
1) Keep your nails short and clean
Rationale: to minimize trauma and secondary infections.
2) Wear gloves or elbow restrein
Rationale: to prevent scratching
3) Give clothes that are thin, loose, and not irritating.
Rationale: because excessive heat can increase itching.
4) Cover the 19/30s (long sleeves, trousers, one-layer clothing).
Rationale: to prevent scratching
5) Give a softening lotion (just a little on the open lesion).
Rationale: because in open lesions the absorption of the drug increases to decrease pruritus.
E. Changes in family processes are related to children suffering from acute illness.
Expected results:
Intervention:
F. Nutritional changes less than the body needs are associated with failure to digest
or inability to digest food or absorption of nutrients which is necessary.
Expected results:
Rationale: Symptoms of intestinal gastro can indicate the effects of anemia (hypoxia) on
organs.
Expected results:
Intervention:
Takipnea usually exists to some degree and can be found in reception or during stress or
the presence of an acute infection process.
4) Maintain environmental pollution, for example; dust, smoke, and pillow feathers
associated with individual conditions.
Rationale: the originator of a type of respiratory allergic reaction that can be an acute
episode.
Cough can be persistent but ineffective, especially when the patient is elderly, acute pain,
or weakness. Coughing is most effective in a high sitting position or head down after
percussion
EVALUATION
A. Active immunization
This can be achieved by using a live measles vaccine that has been attenuated. The first
living vaccine to be used was the Edmonston B strain. The subsequent weakening of the
Edmonston B strain brought about the widespread development and use of the Schwartz and
Moraten strains. The vaccine is given subcutaneously and causes long-lasting immunity.
In the investigation, it turned out that the immunity began to decrease 8-10 years after
vaccination. It is recommended that routine measles vaccination cannot be done before the
baby is 15 months old because before the age of 15 months it is estimated that the child
cannot form antibodies properly because there are still antibodies from the mother. In
communities where measles is endemic, immunization can be given when the baby is 12
months old.
B. Passive immunization (immunoglobulin)
Passive immunization with collected adult serum, collected healing stage serum, collected
placental globulin (gama globulin plasma) can provide effective results for the prevention or
weakening of measles. Measles can be prevented with serum immunoglobulin at a dose of
0.25 ml/kg BB im and given for 5 days after exposure or as soon as possible.
Indication:
2) Infants aged < 12 months who are directly exposed to measles patients have a high risk for
developing complications of this disease, so immunoglobulin should be given as soon as
possible within 7 days of exposure. After that the MMR vaccine is given as soon as possible
until the age of 12 months, at intervals of 3 months after immunoglobulin administration.
References
Tarwoto dan Wartonah. (2000). Kebutuhan Dasar Manusia dan Proses Keperawatan.
Salemba Medika : Jakarta.
http://pediatricinfo.wordpress.com/2008/07/09/campak-morbili-measles-rubeola/
http://www.scribd.com/doc/22319650/asuhan-keperawatan-anak-morbili