PATHOPHYSIOLOGY

unknown etiology
Predisposing factors Precipitating factors
Age preeclampsia in previous
Sex decreased placental pregnancy
Family History perfusion multigravidity
Placental production of endothelin Diet (high in cholesterol,
saturated fat, and
vasospam sodium intake)

Endothelial cell damage

vasoconstriction

hypertension

Platelet cluster at the site of

endothelial damage

rise in peripheral
resistance

reduced blood
supply
 decreased perfusion in placenta

bleeding in the decidua basalis

hematoma formation

further separation of the placenta

from the uterine wall

abruptio placenta

partial separation total separation

Marginal Central –
– vaginal conceale massive vaginal
bleeding d bleeding or
bleeding conceaaled
hemmorhage
 if treated: if not treated: if treated: if not treated:

Nsg. management Nsg. management

- bed rest Fetal risks include acute and - monitor VS poor prognosis
chronic uteroplacental
- monitor vital sign
insufficiency
diet modification
maternal and
Medical
Medical Brain ischemia causes the later
management fetal death
management emergence of convulsions
- IV fluid and
- medication electolyte
administration replacement
Eclampsia
-meds
good
prognosis Occurrence of good prognosis
seizures and can
cause coma

poor prognosis

DEATH