Professional Documents
Culture Documents
Educational-scientific-production complex
"International University of Kyrgyzstan"
Quality Management System
Educational-methodical complex of the discipline "dentistry"
PLO "General Medicine" (for foreign citizens) (5 years) ISM IUK
"APPROVED"
Vice-rector for educational and administrative work
prof. Musa kyzy Alina
______________________
"__" ____________ 2021
"Dentistry"
main educational program
in the specialty General Medicine (for foreign citizens)
Bishkek 2021
1
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Educational-methodical complex of the discipline "dentistry"
PLO "General Medicine" (for foreign citizens) (5 years) ISM IUK
Table of contents
1. The work program of the academic discipline ..........................................................................................5
1.1. Explanatory note ..............................................................................................................................5
1.2. Recommended educational technologies ...........................................................................................7
1.3. The scope of the discipline and types of educational work ................................................................8
1.4. Discipline structure ............................................................................................................................1
1.4.1. Thematic plan for the study of the discipline (by semester)........................................................1
1.4.2. Organization of students' independent work ...............................................................................1
1.4.3. Evaluative Assessment Tools ......................................................................................................3
1.4.4. Course policy and assessment criteria .........................................................................................6
1.4.5. Educational-methodical and informational support of the discipline ..........................................7
1.4.6. Logistics of the discipline............................................................................................................8
1.4.7. Student research work .................................................................................................................8
2. Teaching materials ....................................................................................................................................9
2.1. Lecture notes ......................................................................................................................................9
2.2. Development of practical / seminar / laboratory classes ................................................................125
3. Methodical recommendations for students............................................................................................141
3.1. Methodical recommendations for students on the study of the discipline .....................................142
3.2. Methodical recommendations for the implementation of practical / seminars, laboratory work. ..143
3.3. Methodical recommendations for the implementation of independent work. ................................143
3.4. Methodological instructions for the implementation of abstracts, reports, essays .........................144
3.5. Methodical instructions for preparation for the final certification. ................................................145
3.6. Methodical recommendations for the student's research work.......................................................145
4. Glossary.................................................................................................................................................147
5. Reference materials and applications ....................................................................................................150
2
Non-profit educational institution
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Educational-methodical complex of the discipline "dentistry"
PLO "General Medicine" (for foreign citizens) (5 years) ISM IUK
"APPROVED"
Vice-rector for educational and administrative work
prof. Musa kyzy Alina
________________________
"__" ____________ 20__
"STOMATOLOGY"
Full-time education
Well 4
Semester 8
Credit / Exam (semester) 8
Total Curriculum Credits 4
Total curriculum hours 144
Bishkek 2021
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The work program for the discipline "Otorhinolaryngology" has been developed in
accordance with the requirements of the State Educational Standard of Higher
Professional Education in the specialty 560001 General Medicine.
Work program agreed with the educational and methodological sector of ISM,
EMD
Chief specialist of EMD ______________ ___________________
(Full name) (signature)
"____" _________ 20__
Work program agreed with the head of the main educational program in the
direction / specialty 560001 General Medicine (for foreign citizens)
Head of the PLO ________________________ ___________________
(Full name) (signature)
"____" _________ 20__
Work program agreed with the department of monitoring and quality of the UNPK
"MUK"
Director of DMaQ ___________________ ___________________
(Full name) (signature)
"____" _________ 20__
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− Histology
− Oral hygiene
− Normal anatomy
− Pathological anatomy
− Normal physiology
− Pathological physiology
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Non-profit educational institution
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1.4.1. Thematic plan for the study of the discipline (by semester)
Auditory lessons
Student independent
practical lessons
midterm control
classroom work
Total hours on
sections and topics
technologies,
performance
competence
educational
of teaching
disciplines
academic
seminars
(lectures and practical exercises)
Formed
lectures
SRSP
work
Used
Module 1 T
1 Caries, examination 2 2 4 2 2 SLK-3, LV / PL NS,
methods, etiology, ZK, R
classification, diagnosis, IK-4,
treatment methods. PK-6
1
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Abbreviation for designations of educational technologies, methods and methods of teaching:traditional lecture (L), lecture-
visualization (LP), problem lecture (LP), lecture-press conference (LPK), lesson-conference (LC), training (T), debate (D),
brainstorming (MSH) , master class (MC), "round table" (CC), activation of creative activity (ATD), regulated discussion (RD),
discussion of the forum type (F), business and role-playing educational game (CI, RI), small group method (MG), classes using
simulators, simulators (TP), computer simulation (CS), analysis of clinical cases (CS), preparation and protection of medical
history (IB), use of computer training programs (COP), interactive atlases (IA), attending medical conferences, consultations (VC),
participation in scientific and practical conferences (NPK), congresses, symposia (Sim), educational and research work of a student
(UIRS), conducting subject Olympiads (O),preparation of written analytical works (AR), preparation and defense of abstracts (P),
design technology (PT), excursions (E), distance educational technologies (DOT).
Reduction of forms of current and midterm monitoring of academic performance: T - testing, Pr - assessment of the development
of practical skills (abilities), ЗС - solving situational problems, КР - control work, КЗ - control task, IB - writing and protecting a
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case history, CL - writing and protecting a curatorial sheet, R - writing and defense of the abstract, C - interview on control
questions, D - preparation of a report, etc.
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Non-profit educational institution
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Quality Management System
Educational-methodical complex of the discipline "dentistry"
PLO "General Medicine" (for foreign citizens) (5 years) ISM IUK
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Non-profit educational institution
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5) the progressive loss of tooth tissue (enamel and dentin) is not enough identified
etiology
2. Fluorosis is:
1) an endemic disease caused by fluoride intoxication with excess content
it in drinking water. 2) a developmental defect, consisting in the
underdevelopment of the tooth or its tissues
2) fusion, fusion and bifurcation of teeth
3) violation of enamel formation, expressed by a systemic violation of the
structure
mineralization of dairy and permanent teeth
4) the progressive loss of tooth tissue (enamel and dentin) is not enough identified
etiology
3. Tooth erosion is:
1) an endemic disease caused by fluoride intoxication with excess content
its in drinking water
2) a developmental defect, consisting in the underdevelopment of the tooth or its
tissues
3) violation of enamel formation, expressed by a systemic violation of the
structure and
mineralization of deciduous and permanent teeth
4) the progressive loss of tooth tissue (enamel and dentin) is not enough identified
etiology
5) fusion, fusion and bifurcation of teeth
4. Imperfect amelogenesis is:
1) an endemic disease caused by fluoride intoxication with excess content
its in drinking water
2) a developmental defect, consisting in the underdevelopment of the tooth or its
tissues
3) fusion, fusion and bifurcation of teeth
4) violation of enamel formation, expressed by a systemic violation of the
structure and
mineralization of deciduous and permanent teeth
5) the progressive loss of tooth tissue (enamel and dentin) is not enough identified
etiology
5. Anomaly of tooth development is:
1) an endemic disease caused by fluoride intoxication with excess content
its in drinking water
2) a developmental defect, consisting in the underdevelopment of the tooth or its
tissues
3) fusion, fusion and bifurcation of teeth
4) the progressive loss of tooth tissue (enamel and dentin) is not enough identified
etiology
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The results of the 2 modules are added up and the average score is displayed.
Scoring Policy Module 1 Module 2
Attendance 20 points 20 points
Classroom work (activity in discussions, 20 points 20 points
during oral questioning, working with a
glossary, etc.)
Independent work: essay, report 20 points 20 points
Total by module (testing) 40 credits 40 credits
Total by discipline: More than 60 points
Offset
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Final control in the form of offset is carried out based on the results of attendance,
current and midterm (modular) control.
Final control form - offset.
To assess the student's progress, the following scale of correspondence between
grades and points is used:
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The results of work with scientific monographs and articles are discussed in practical
classes.
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2. Teaching materials
Educational and methodological materials (UMM), as methodological support of the
discipline, are presented in the form of lecture texts, seminar developments, practical
classes, both in print and in electronic form.
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Caries (lat. caries decay) - a complex, slowly flowing pathological process that
occurs in hard tissues toothand developing as a result of the complex impact of
adverse external and internal factors. At the initial stage of development, caries is
characterized by focaldemineralization inorganic part enamels and the destruction
of its organic matrix... This ultimately leads todestruction hard tooth tissues with the
formation of a cavity in dentine, and in the absence of treatment - to the occurrence
of inflammatory complications from pulp and periodontium...
Epidemiology
Caries is the most common human disease (over 93% of people). In childhood, it
ranks first among chronic diseases and occurs 5-8 times more often than the disease,
which ranks second in prevalence -bronchial asthma... According to different
authors, from 80 to 90% of children with milkbite, about 80% of adolescents have
carious cavities at the time of graduation, and 95-98% of adults have filled teeth.
Statistics show that in the equatorial regions (Africa, Asia) caries is less common
than in the circumpolar regions (Scandinavia, North America). In developing
countries, there is also a higher incidence of tooth decay.
Etiology
Currently, the occurrence of dental caries is associated with local changes pH on the
surface of the tooth under dental plaque due to fermentation (glycolysis)
carbohydratescarried out microorganisms, and the formation of organic acids...
Acid-forming streptococci (Streptococcus mutans, Str. sanguis, Str. mitis, Str.
salivarius), which are characterized by anaerobicfermentation and some
lactobacillus...
When considering the mechanisms of the occurrence of dental caries, attention is
drawn to the variety of various factors, the interaction of which determines the
emergence of a focus of demineralization: microorganisms of the oral cavity, the
nature of nutrition (amount of carbohydrates), diet, the amount and quality of
salivation (remineralizing potential of saliva, buffer properties, nonspecific and
specific saliva protection factors), shifts in the functional state of the body, the
amount fluorineentering the body, the influence of the environment, etc. However,
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the main factors for the occurrence of caries are as follows: caries susceptibility of
the tooth surface, cariogenic bacteria, fermentable carbohydrates and time.
Although tooth enamel is the hardest tissue of the human body (hardness 5 ... 6 by
Mohs scale, which roughly corresponds to feldspar), hydroxyapatites of enamel
prisms are too sensitive to an increase in acidity, which gives rise to the onset of
their destruction already at pH 4.5.
Determining factors that affect the intensity of caries development:
● the presence of a large amount of carbohydrates in the diet, in particular,
sucrose - the main component of edible sugar, which is under the action of enzymes
saliva easily forms glucose as a substrate for anaerobic glycolysis... Regular removal
of plaque counteracts the development of caries (therefore, you need to brush your
teeth);
● saturation of tooth enamel with fluoride ions (F>). Fluorapatites, which are
formed as a result of the replacement of OH> -groups and hydroxyapatites of the
enamel with fluorine ions, are resistant to the destructive effect of acidic equivalents
of saliva. The most effective anti-carious effect of fluorides is observed when they
are found in the body during the development of teeth and their mineralization. By
recommendationsworld health organization, fluoridation drinking water to a
concentration of 1 mg F> / ml leads to a decrease in the incidence of caries in the
population by 30-50%
Cariogenic situation
A cariogenic situation is a condition of the oral cavity that forms many factors and
determines the propensity for the disease. They reveal a tendency to illness by
examining the oral cavity and performing a series of tests (staining the enamel,
determining plaque, traces of bacteria, resistance of hard tissues to acids). With the
help of tests, the dentist determines the degree of susceptibility of hard tissues: high,
medium or low resistance to caries.
There are general and local causes of caries. Local factors include factors that are
located and act in the oral cavity. General - the condition and diseases of the body,
other causes of dental caries.
Tooth enamel- the hardest tissue in the human body. It is 96% mineral,
mainlyhydroxyapatites, which are very susceptible to acids, therefore, the
destruction of enamel begins already at a pH of 4.5. Caries susceptibility of the tooth
surface depends on many factors:
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Cariogenic bacteria
In the oral cavity, many bacteria, but in the process of plaque formation and
subsequent enamel demineralization, mainly acid-forming streptococci
(Streptococcus mutans, Streptococcus viridans, Str. sanguis, Str. mitis, Str.
salivarius), which are characterized by anaerobic fermentation and lactobacillus
(Lactobacillus).
Within a few minutes after taking carbohydrates, especially sucrose, there is a
decrease in pH from 6 to 4. In dental plaque, except lactic acid, which is directly
formed during the fermentation of carbohydrates, are found formic, oil, propionic
and other organic acid...
Anti-caries flora
Recently it was discovered that in addition to bacteria, the vital activity of which
destroys tooth enamel, there are bacteria that counteract this process. Robert Burne
and Marcelle Nascimento fromUF College found streptococcus A12 in plaque, a
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previously unknown strain that helps neutralize acids in the oral cavity by
metabolizing arginine
Fermentable carbohydrates
It is the acids formed during the fermentation of carbohydrates that lead to the
destruction of the surface of the tooth enamel. The presence and activity of
fermentation in the plaque depends on the quantity and quality of available
carbohydrates. Most intensefermentation sucrose, less intense - glucose and
fructose... Mannit, sorbitol and xylitol also penetrate into dental plaque, however,
due to the low activity of the enzyme that converts them into fructose, they are not
dangerous. Starchbeing polysaccharide, in its pure form is not karyogenic, since its
molecules do not penetrate into dental plaque. However, food processing can destroy
the molecular structure of starch and increase its cariogenicity.
Time
The frequency with which a tooth is exposed to cariogenic acid influences the
likelihood of tooth decay. After every meal that contains sugar, microorganisms
begin to produce acids that destroy tooth enamel. Over time, these acids are
neutralized by the buffering properties of saliva and partially demineralized enamel.
After each period of exposure to acids on the tooth enamel, the inorganic mineral
components of the tooth enamel dissolve and can remain dissolved for 2 hours. If
carbohydrates are taken periodically throughout the day, then the pH will be low for
a long time, the buffering properties of saliva do not have time to restore the pH, and
there is a possibility of irreversible destruction of the enamel surface.
As shown earlier, the speed of the carious process depends on many factors, the
process that has begun may slow down if fluoride is used, but on average, caries of
the contact surfaces in permanent teeth progresses slowly and the cavity can form
within 4 years. Since the root of the tooth is covered with softer tissue -cement, root
caries develops 2.5 times faster than in the enamel area.
If the oral hygiene is extremely unsatisfactory and the diet is rich in sugar, tooth
decay can develop literally a few months after a tooth erupts.
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● Suspended caries.
● Odontoplasia.
● Another.
● Unspecified.
Process depth classification
Superficial caries.
At the next stage, the untreated chalk stain becomes rough due to the
demineralization of the enamel. In this case, the enamel-dentin junction is not
affected. The tooth in this place hurts from hot, cold food, and also becomes sensitive
to sweet and sour (possibly painless). Treatment of this stage of cervical caries will
consist in grinding the area of the tooth affected by caries. Then, by analogy with
the treatment of the previous stage, remineralizing therapy is performed. (But if
caries is localized in fissures or on contact surfaces (these areas are susceptible to
caries, since there are good conditions for fixing food, and they are difficult to
cleanse), then remotherapy will be useless, so you can prepare the affected area
"according to all the rules." and seal).
Medium caries.
At the third stage, caries begins to penetrate deeper. The enamel-dentin junction is
affected. Serious damage to the tooth becomes clearly visible. Pain sensations
become more frequent, intense and prolonged. Treatment of medium caries consists
in removing the damaged part of the tooth, treating it with medications and installing
a filling.
Deep caries.
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When damage to an untreated tooth reaches the peri-pulpal dentin, the stage of deep
caries begins. The treatment procedure consists in removing the damaged part of the
tooth, treating it with medications, introducing remineralizing drugs and installing a
filling. In the absence of treatment for this disease, further destruction of dentin and
damage to the pulp (nerve) of the tooth occurs -pulpitis and, as its further outcome,
periodontitis...
B) Complicated caries (pulpitis, periodontitis).
This classification takes into account the depth of the process, which is important
for choosing a treatment method.
Caries in the spot stage, superficial caries, medium caries with small defects are not
visible on radiograph... It is very important to differentiate caries from other
diseases:
caries in the spot stage must be differentiated from such non-carious lesions, how
hypoplasia and fluorosis;
common between these diseases: the presence of spots, indications of eom
(electrodontometry) is normal, no subjective discomfort. Differences: caries, unlike
these two diseases, can be stained with special dyes, fluorosis and hypoplasia also
occur before teething, and caries after; caries occurs in caries-susceptible areas, and
they are in atypical (caries-resistant) places;
superficial caries must be distinguished from such non-carious diseases, how wedge-
shaped defects and erosion of hard tissue...
Classification by the severity of the process
Clinical observations have established that the severity and speed of the carious
process determine the methods and tactics of treatment. Based on their results, T.V.
Vinogradova proposed a classification of caries according to the severity and
prevalence of the carious process:
compensated form. In this form, the average intensity of caries is less than the
average intensity for this age group. The process is slow, the existing carious cavities
are lined with hard pigmented dentin (chronic caries);
subcompensated form. The average intensity of caries is equal to the average for this
age group;
decompensated form, or "acute caries". The average caries intensity is much higher
than the average for this age group. With this form, the carious process is intense,
15
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there are multiple carious cavities that are filled with soft dentin. The extreme form
of acute caries is the so-called "systemic damage" of teeth by caries, in which there
is damage to all or almost all of the teeth in the areanecks...
Localization classification
In the textbook "Dentistry" edited by V. Kozlov, the following division of the types
of caries is also proposed:
● fast flowing;
● slow flowing;
● stabilized.
Clinical picture
Spot stage
Demineralization begins with the loss of the natural gloss of the enamel and the
appearance of matte, white, light brown and dark brown spots. The appearance of
16
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this zone is a consequence of the loss of mineral substances by the tooth tissues, in
particularcalcium salts, which leads to a violation of the structure of the enamel.
A white carious spot, depending on the severity of the process, can have two
development paths:
● a white carious spot (progressive demineralization) later turns into superficial
caries due to a violation of the integrity of the surface layer;
● the process slows down, stabilizes, and due to the penetration of organic dyes,
the enamel changes color. It should be understood that the stabilization is temporary
and sooner or later a tissue defect will appear at the site of the pigmented spot.
Caries in the spot stage is usually asymptomatic; very rarely, in the acute course of
the process (white spot), sensitivity to chemical and thermal stimuli may occur.
However, the carious stain stains wellmethylene blue... For this reason, it is this
substance that is used for diagnostic purposes.
With deep caries, there are significant changes in dentin, which causes complaints.
Patients indicate short-term pains from mechanical, chemical and thermal stimuli,
which disappear after their elimination.
Examination reveals a deep carious cavity filled with softened dentin. Probing the
bottom of the cavity is painful. Due to the fact that dentin is more susceptible to
carious processes, the cavity in dentin is usually more extensive than the enamel
entrance.
In some cases, symptoms may appear pulpitis: aching pain in the tooth after removal
of the irritant.
Diagnostics
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With deep forms of caries, it is usually not difficult. The use of a dental mirror and
probe makes it easy to find and diagnose cavities. Cavities localized on the
"proximal" (touching) cavities of the teeth (class II according to Black) constitute a
certain complexity. In such cases, thermal diagnostics (cold) andradiograph teeth.
The X-ray also helps diagnose “caries in the stage of stains", In which the integrity
of the enamel is not compromised and the probe is not delayed. Diagnosis of caries
in the stain stage can also be carried out by staining the tooth surface with a
solutionmethylene blue or caries detector (includes magentapink) (the changed areas
are colored) - using this method you can differentiate caries in the stage of stains
from hypoplasia and fluorosis... You can also dry the surface of the tooth - in this
case, the carious surface loses its shine, but this sign is more difficult to distinguish,
therefore it is less reliable than a test with methylene blue.
To find and diagnose "latent" and initial caries, you can use "transluminescence" -
shining through the tooth from the opposite side with a strong light source, such as
dental photopolymerizer...
remineralizing therapy
Treatment of superficial, medium and deep caries is carried out dissection (removal
of the affected tissue) followed by replacement, filling carious cavity.
Stages of carious cavity processing:
1. definition occlusal points;
2. anesthesia tooth[eighteen] (application, infiltration, conduction, anesthesia);
3. cleaning a tooth from food plaque with a brush and paste or dental
sandblasting (Air flow and etc.);
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Similar techniques are under research and development. So, doctors from the French
National Institute of Health and Medical Research (INSERM) in 2010 discovered a
method of treating a patient with dental caries without surgery. Melanocyte
stimulating hormones, introduced into the carious cavity or applied nearby, stimulate
the cells of the tooth tissue to multiply and heal damage. Experiments on mice have
shown that teeth are restored within a month [source not specified 278 days]...
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studies show the opposite results. So, according toAmerican Dental Association,
researchers from several groups agreed that local anesthesia and dental treatment
during pregnancy are safe for the fetus
Another prospective study published in August in the Journal of the American
Dental Association also confirms that the use of local anesthesia during pregnancy
does not harm the fetus. During the work, 210 pregnant women were monitored,
who received dental treatment (53% in the first trimester) using local anesthesia.
Their data were compared with data from a control group of 794 women who had
not been exposed to any teratogens during pregnancy. The results of the study did
not show any difference in the incidence of complications of pregnancy and
miscarriage between the two groups, and there was also no difference in terms of
delivery and fetal weight. Most often, dental treatment included: endodontic
treatment (43%), tooth extraction (31%), tooth restoration (21%).
The safety of X-ray examination for the treatment of teeth in pregnant women is
confirmed by another independent study conducted in Helsinki... In the course of the
work, it was shown that even the use of such protective devices as a lead apron for
the mother is not justified, since the radiation dose is very small and does not cause
complications in the fetus.
Prophylaxis
Based on modern ideas about the occurrence of caries teeth, its prevention should be
carried out in two directions:
● elimination of a cariogenic situation in the oral cavity;
● increased caries resistance of tooth tissues.
The main measures prevention caries are
● regular oral care;
● decrease in consumption Sahara;
● usage fluorinated toothpastes;
● adherence to the correct technology brushing teeth;
● Flossing before or after brushing your teeth
● use of solutions 0.1-0.2% chlorhexidine for daily mouthwash or chlorhexidine
toothpastes;
● use mouthwash;
● use xylitol at least 3 times a day after meals in the form of xylitol chewing
[23]
gum ;
● preventive examination at dentist at least once every six months.
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Sealing (sealing) of fissures and blind pits with special flowable polymers
(composites based on methacrylate,polyurethane) allows you to protect the most
likely places of caries formation (fissures) and reduces the growth of caries by up to
90%.
Diet correction
One of the main risk factors for the development of caries is the presence of sugars
in the diet. A promising direction of prevention is limiting carbohydrates in the diet
of children and replacingSahara non-cariogenic products (sorbitol, xylitol) in infant
formula and confectionery... Milk and some varieties cheesealso contribute to the
remineralization of enamel. Usagechewing gum (especially with xylitol) also has a
positive role. First, chewing gum removes food debris and, in part, plaque from the
fissures of the teeth; secondly, the act of chewing leads to the release of a large
amount of saliva, and the active components that make up some chewing gums
(calcium lactate or pyrophosphate and tripolyphospha]), improves remineralization
of the enamel surface and reduces the formation of supragingival calculus
For the prevention of caries V. K. Leontiev the "culture of carbohydrate
consumption" was proposed
1. do not use sweet as the last meal when eating;
2. do not eat sweets between meals;
3. do not eat sweets at night;
4. if you violate these rules, you should brush your teeth, chew sugar-free gum,
or eat hard vegetables or fruits that cleanse your mouth.
Sour and sweet drinks are best drunk through straw... Sucking is also
harmfullollipopscontaining sugar.
In 2020, scientists have established benefits for the fight against caries of traditional
Sichuan pickled cucumbers... Of these,strain bacteria Lactobacillus plantarum K41,
which has shown a strong inhibitory effect against the formation of biofilms the main
carious bacterium Streptococcus mutans
Oral hygiene
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Oral hygiene plays an important role in the prevention of diseases such as tooth
decay, gingivitis, periodontitis... Timely removal of plaque not only stops the carious
process, but also leads to a curegingivitis (cm. teeth cleaning methods).
Fluoridation
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Topic 2. Pulpitis
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Pulpitis - inflammation internal tissues tooth (pulp). The pulp is located inside the
canal and contains the nerve, vessels, cells of the connective tissue and provides
nutrition to the hard tissues of the tooth from the inside. Pulpitis develops when an
infection enters the tooth, most often as a result of a long-term currentcaries...
Common manifestations are increased tooth sensitivity to temperature, throbbing
pain, in rare cases, asymptomatic course is possible. Depending on the duration of
the course, it can be acute and chronic.
In the absence of treatment, pulpitis can lead not only to tooth loss, but also to the
transition of the inflammatory process on the tissues of the jaw and sepsis(mainly in
people with severely reduced immunity), so it needs to be treated. Currently,
methods have been developed that allow to treat pulpitis without completely
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destroying the pulp, that is, preserving the viability of the nerve and feeding vessels
(methods of vital amputation)
Etiology
Pulp inflammation is always caused by infection in the pulp chamber. This can
happen in two ways: intradentally (through the crown of the tooth) and retrograde
(through the apical (located at the apex of the tooth) orifice). Most often, pulpitis is
a complicationcaries... Sometimes it can be the result of incorrect actions of the
doctor (grinding a tooth under orthopedic structures, poor qualityfilling materials,
surgical interventions on the periodontium, exposure to chemicals). Cases of
retrograde pulpitis (that is, infection throughapical foramen tooth
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○ the penetration of microbes from the carious cavity through the dentinal
tubules after preparation, when taking an impression under pressure (a very rare
way);
○ retrograde pulpitis - the ingress of microbes through the apical foramen in
osteomyelitis, sepsis, through the lateral branches of the root canal, after curettage
of deep periodontal pockets.
Classification
MMOMA classification (1989)
1. acute pulpitis:
1. focal purulent
2. diffuse purulent
2. chronic pulpitis
1. fibrous
2. gangrenous
3. hypertrophic
3. exacerbation of chronic pulpitis
1. exacerbation of chronic fibrous pulpitis
2. exacerbation of chronic gangrenous pulpitis
4. condition after partial or complete removal of the pulp.
ICD-10 classification - used for diagnosis in most dental clinics in the world. The
correspondence to the shape of the pulpitis according to the MMSI is indicated in
brackets.
● K04 - diseases of the pulp and periapical tissues
● K04.0 pulpitis
● K04.00 - initial (pulp hyperemia) (according to MMOMA deep caries)
● K04.01 - acute (according to MMSI, acute focal pulpitis)
● K04.02 - purulent (pulp abscess) (acute diffuse according to MMSI)
● K04.03 chronic (according to MMSI - chronic fibrous pulpitis)
● K04.04 chron. ulcerative pulpitis (according to MMSI - chronic gangrenous
pulpitis)
● K04.05 - pulp polyp (according to MMSI - chronic hypertrophic pulpitis)
● K04.08 - other specified pulpitis
● K04.09 - Unspecified pulpitis
● K04.1 pulp necrosis (pulp gangrene)
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Pathogenesis
Pulpitis are classified as acute and chronic. Acute pulpitis is understood as a
condition when the infection has penetrated into the pulp with a closed pulp chamber
(through a thin wall of a tooth destroyed by caries). Acute pulpitis initially has a
focal character and proceeds as serous inflammation (serous pulpitis), then
appearspurulent exudate(purulent pulpitis). In this case, very severe pains appear
due to the accumulation of purulent exudate in a closed pulp chamber. Chronic
pulpitis is most often the outcome of an acute one.
Chronic pulpitis is divided into fibrous, hypertrophic and gangrenous. The main
form of chronic pulpitis is fibrous pulpitis, in which fibrous connective tissue grows.
With hypertrophic pulpitis occurshypertrophicproliferation of pulp tissue through an
open carious cavity. With gangrenous pulpitis, tissue decay is found in the coronal
pulp. In the root pulp is foundgranulation tissue...
Acute pulpitis
Acute focal
The inflammation is in the projection of the horn of the pulp. This stage lasts about
2 days. The causative tooth is very sensitive to thermal stimuli (mainly to cold), and
the pain intensifies and continues after the removal of the stimulus (in contrast
tocaries). Percussion (tapping) of the tooth is insensitive or insensitive
(unlikeperiodontitis).
Acute diffuse
The main signs of acute diffuse pulpitis are strong, radiating (spreading) along the
branches trigeminal nervepains that get worse at night. The pains are intermittent.
Quite often, patients note pains of a different nature in the temporal region.
Chronic pulpitis
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Purulent pulpitis can occur with significant pain, while the tooth is sensitive to hot,
and cold soothes the pain.
Focal pulpitis
The presence of a carious cavity filled with softened dentin. The walls and bottom
of the cavity are dense. Probing sharply painful at the bottom of the cavity closer to
the horn of the pulp. The tooth cavity is closed. On X-ray, the carious cavity is close
to the tooth cavity. Vertical percussion is painless. EDI - 15-20 μA
Acute diffuse
A similar pain is characteristic of an acute attack. The carious cavity is deep, with a
large amount of softened dentin. Probing painful throughout the bottom. Vertical
percussion is painful. On the roentgenogram - the carious cavity communicates with
the tooth cavity. There are no changes in the periodontium. EOD - 20-35 μA.
Chronic
Fibrous
The cavity can be either closed or open. Probing is painful. Slowly increasing pain
from hot or cold is inherent. Percussion is also painless. EDI - 20-40 μA
Gangrenous
The cavity is open. Increasing pain under the influence of heat. With percussion,
slight pain is possible, EDI - 40-80 μA
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Treatment
Serous pulpitis is considered reversible with proper treatment (Ca-containing
medicinal pads for fillings, antibiotic dressings, etc.), which is aimed at alkalizing
the tooth cavity, with the subsequent formation of secondary dentin. The indications
for this (biological) method of treatment are young age (up to 30 years), the absence
of chronic diseases and sufficient caries resistance (resistance of tooth tissues to the
carious process). Partial removal of the pulp (pulp amputation) is also possible, but
it is now rarely used, mainly in milk teeth and teeth with an unformed apex (up to
two years after the eruption of the tooth).
In the treatment of acute purulent and chronic pulpitis, mechanical and drug
treatment is performed root canal systems (depulpation - "removal of the nerve")
and filling (airtight obturation) channel... This type of treatment is divided into
devital and vital extirpation.
Devital extirpation
In this method, there is a complete destruction of the pulp of the tooth. Removal of
the neurovascular bundle is carried out in 2 visits. After anesthesia, a message is
created with the cavity of the tooth (pulp chamber) and a devitalizing paste is applied
(popularly referred to as "arsenic", But in fact it is almost never used, but is replaced
by a paste based on paraformaldehyde and anesthetic, it can be left on for a week,
and its toxicity is much lower).
Vital amputation
It allows you to preserve the viability of the nerves and vessels that feed the tooth,
which ensures normal trophism of the tooth tissues and prevents the development of
periapical complications. Studies by a number of authors have shown that after vital
amputation, the root pulp retains its viability, producing secondary dentin.
Removal of the neurovascular bundle and its obturation is carried out under
anesthesia in one visit in the absence of pronounced inflammation, passing into the
periodontium. In case of spread of inflammation beyond the root system, a medicinal
substance is left in the canal (for antiseptics and relieving inflammation). During the
treatment, it is necessary to take at least two images: the first one - before the start
of treatment, to assess the length and structure of the canals; the second - after, to
assess the quality of the filling of the canals. A pulped tooth subsequently needs
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Effects
After depulpation, the tooth becomes "dead" (its blood supply is cut off). Since
complete sterilization of such a tooth is impossible, bacteria that are protected from
immunity and antibiotics can develop in it. Hypothetically, they can cause an
infection in the body if the immune system is seriously weakened, for example, in
the treatment of cancer.
Complications
Chronic pulpitis without proper treatment, with inadequate treatment of the root
canal system, with leaking obturation of the canal or leaking tooth restoration (seal,
tab, onlay, crown) can go to periodontitis...
Prophylaxis
The main task is to prevent the onset and development of diseases at the earliest
stages.
Types of prevention of pulpitis:
1. Primary: Includes a number of activities that are quite cheap and simple
(drawing up a diet, individual and professional care and selection of hygiene
products, the use of fluorine drugs), the regular implementation of which will several
times reduce the risks of pulpitis spread.
2. Secondary: It is used in cases where the disease has already arisen and is
aimed at preventing complications (for example, treatment of caries as prevention
of pulpitis and periodontitis).
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3. Educational technologies
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4. Basic and additional literature:
1. Therapeutic dentistry, E. V. Borovskiy and sov., Moscow., 1998;
2. Dentistry, N. N. Bazhanov, "Medicine", Moscow, 1990
3. L.N. Maksimovskaya,
4. P.I. Roshchin.
5. Medicines in dentistry: Endodontics
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1955; Sobkowiak, 1979). The differences concern only details that are not of
fundamental importance. The most widespread classification of G.I. Lukomsky
(1955), including the following forms of the disease: 1. Acute periodontitis:
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entry of infection into the periodontium through one of the branches of the root
canal.
ETIOLOGY AND PATHOGENESIS. Periodontitis in the overwhelming majority
of cases is of infectious origin and occurs under the influence of nonspecific
pathogens, most often staphylococci, acting alone or in combination with another
microflora. A feature of this infectious process is the lack of immunity and even a
decrease in the body's resistance to re-introduction of the pathogen. In the
pathogenesis of periodontitis, great importance is attached to the allergic
restructuring of the reactivity of the periodontium, which occurs under the influence
of sensitization by microorganisms, toxins and products of pulp decomposition (BC
Ivanov, 1984). In recent years, much attention in the pathogenesis of the disease has
been paid to anaerobic streptococci and bacteroids, which were found during
bacteriological examination of root canals and granulomas (A.I. Marchenko et al.,
1984).
The infection penetrates into the periodontium through the root canal, the bottom of
the gingival pocket, by hematogenous and lymphogenous routes, by continuation. It
should be noted that the hematogenous and lymphogenous pathways of the onset of
periodontitis have not been convincingly proven. We observed periodontitis and
retrograde pulpitis in periodontitis, osteomyelitis, that is, in those cases when the
periodontium, and subsequently the pulp, were involved in inflammation, which
spread along the length, possibly with the participation of blood and lymph vessels.
Microorganisms enter the dental pulp tissue through the carious cavity. If these
microorganisms are devoid of pathogenic properties, then their first contact with the
pulp tissues may not be accompanied by the development of a pronounced
inflammatory reaction. However, the penetration of the waste products of
microorganisms through the lymphatic vessel system into the regional lymph nodes
with their subsequent fixation by immunocompetent cells already at this stage of the
development of the pathological process may be accompanied by the production of
antibodies and sensitization of the body. A new entry of the same non-pathogenic
microbes into the pulp tissue of a sensitized organism may be accompanied by the
development of allergic inflammation. In some cases, the penetration of
microorganisms is preceded by the occurrence of sensitization of the organism to the
identical microflora of some other infectious focus, and the very first introduction of
microbes into the dental pulp may be accompanied by the development of allergic
inflammation.
CLINIC. In acute serous periodontitis, aching, mild pains in the tooth appear,
aggravated by biting. Pain usually does not radiate and patients correctly indicate
the localization of the affected tooth. With prolonged pressure on the tooth (closing
the jaws), the pain subsides somewhat. No soft tissue swelling. Regional lymph
nodes increase slightly, slightly painful. There is little tooth mobility and positive
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Objectively, on the alveolar process of the jaw, in the area of the affected tooth, it is
usually possible to detect a fistulous passage with purulent discharge. The
localization of fistulas is different. Most often, they open in the projection area of
the apex of the tooth root from the vestibular side. This is because the outer wall of
the alveoli is thinner. Around the mouth of the fistulous tract, there is often an
overgrowth of bleeding pink granulations. Instead of a fistula, in some cases, a scar
can be found, which indicates that the functioning fistula has closed. With
granulating periodontitis, fistulous passages can open on the skin, the localization of
which is quite typical and depends on the location of the causative teeth. Cutaneous
fistulas can be localized in the corner of the eye (the reason is the upper canine), in
the buccal region (in the upper part - from the first upper molar, less often -
premolars), in the chin (from the lower incisors and canine). We did not observe
fistulas on the neck with granulating periodontitis.
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The mucous membrane of the gums in the area of the affected tooth is edematous,
hyperemic and pasty. This type of periodontitis is characterized by the symptom of
"vasoparesis". It consists in the fact that when pressing on the mucous membrane of
the gums with a button-like instrument or the blunt side of the tweezers, its blanching
is noted, which is slowly replaced by persistent hyperemia (G.I. Lukomsky, 195E).
This is due to the fact that the decay products in the focus of inflammation cause
persistent paresis of the vasomotor nerves, which leads to disturbances in vascular
tone, congestion and swelling of the gum area. A painful infiltration is felt in the
projection of the root apex.
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With granulomatous periodontitis, using X-ray examination at the apex of the tooth
root, it is possible to detect a focus of destruction of bone tissue, which has a rounded
shape and fuzzy smooth edges. We distinguish between apical, apicolateral, lateral
and interroot granulomas. Apical granuloma is localized strictly at the apex of the
tooth root, lateral - on the side of the tooth root, apicolateral - on the side of the apex
of the tooth root. The tops of the roots of the teeth facing the granuloma are often
resorbed. In multi-rooted teeth, the granuloma can be located in the place of the
deltoid branching of the root canal - interroot granuloma
According to E.V. Borovsky et al. (1973), the diameter of the granuloma usually
does not exceed 0.5 cm, and the size of the cystogranuloma ranges from 0.5-0.8 cm.
Microscopic examination can reveal that the dental granuloma in appearance
resembles a spherical or oval sac. It is surrounded by a dense shell with a smooth
surface and one edge can be tightly soldered to the root of the tooth. Distinguish
between simple, complex and cystic granulomas. A simple granuloma consists of
maturing granulation tissue, which is delimited at the periphery by a fibrous capsule.
In a complex granuloma, an overgrowth of epithelial cords can be found. With
vacuolar dystrophy and disintegration of epithelial cells in the central parts of the
granuloma, a cavity is gradually formed, lined with the epithelium of the cystic
granuloma.
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We have observed patients with granulomas for many years. A granuloma localized
near the outer plate of the jaw (more often in the region of the buccal roots of the
upper molars) can cause destruction of the thin bone wall and grow under the
periosteum, forming a subperiosteal granuloma. In this case, in the projection of the
tops of the roots, a hemispherical formation with clear boundaries, a smooth surface,
dense, slightly painful on palpation, covered with an unchanged mucous membrane
is determined. The high pressure of the contents in the granuloma determines its
density, which often simulates the presence of a tumor. On the roentgenogram, the
subperiosteal granuloma does not differ from the usual ones.
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According to A.P. Grokholsky (1994), long-term results are the main criterion in
assessing the methods of conservative treatment of periodontitis. (In terms of 3 to 6
years), obtained on the basis of clinical and radiological studies. 3 months after high-
quality filling of the root canals, partial restoration of bone tissue in the peri-apical
region is observed, after 6 months - significant restoration of bone tissue, and after
12 months - its complete restoration. Long-term results of treatment in terms of 3 to
7 years indicate the presence of favorable treatment outcomes in 80-90% of cases.
Features of the course of periodontitis in children, according to N.M. Chuprynina
(1985), the following:
• in milk and unformed permanent teeth, periodontitis often occurs with a closed
tooth cavity and with a shallow carious cavity;
• in milk teeth, the granulating form of chronic periodontitis prevails, while
pathological resorption of the roots of these teeth is often observed;
• the granulating form of periodontitis in young children is accompanied by the
formation of a fistula on the gums more often than in adolescents and adults;
• quite often, with all forms of periodontitis, bone rarefaction is observed in the area
of root bifurcation, which is more significant than the apex of the roots;
• necrosis of the pulp and death of the growth zone in chronic periodontitis of
unformed teeth lead to the cessation of root formation;
• the granulating process at the root of the milk tooth, spreading to the follicle of the
corresponding permanent tooth, can disrupt its development;
• in multi-rooted teeth, different roots may have different forms of chronic
inflammation;
• granulating form more often than in adults, it can be accompanied by chronic
lymphadenitis, and sometimes periosteal reaction.
In the elderly, the clinical course of each form of periodontitis has its own
characteristics, which must be taken into account when diagnosing the disease and
choosing a treatment method. First of all, acute periodontitis is very rare in the
elderly. We observed acute periodontitis in young people due to trauma of a very
diverse nature (household, sports, etc.), while in elderly people, the so-called
traumatic periodontitis is, as a rule, a chronic form. In other words, traumatic chronic
periodontitis is characterized by a chronic course from the very beginning of its
onset. This feature of the course of periodontitis in old people is explained by the
fact that it occurs due to the action of a constant traumatic factor, and not, as in young
people, a one-time trauma.
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Acute forms of odontogenic periodontitis in old people are practically not observed,
although an exacerbation resembling a picture of acute periodontitis is a fairly
frequent phenomenon. And if chronic periodontitis can be asymptomatic for years
in people of all age groups, then in the stage of exacerbation, its course has some
difference, depending on the age of the patients. So, in old people, the exacerbation
of chronic periodontitis is less pronounced. This also applies to the pain reaction,
and edema of the surrounding soft tissues, and the general condition of the body.
Much less frequently than in young people, regional lymphadenitis and
adenophlegmon occur. Usually, even with the rapid course of periodontitis, only the
formation of a subperiosteal abscess occurs along the transitional fold near the
causative tooth. After opening the abscess (independently or with the help of a
doctor), fistulas often remain with purulent discharge. Fistulas occur, as a rule, from
the vestibular side of the alveolar process in the projection of the tooth root. Only
with a disease of the lateral incisors, first premolars and molars of the upper jaw,
sometimes fistulas can occur in the palate. The localization of the fistula does not
depend on age, but in older people with reduced regenerative capabilities of the
body, long-standing fistulas rarely close. They can exist for years, and therefore the
exacerbation of periodontitis may not occur for a number of years. With a long-term
illness, pus can flow through the periodontal fissure, in these cases, the fistula is
found in the periodontal pocket. The indicated localization of fistulas, the absence
of lush granulations in the circumference of their mouths, scanty purulent discharge
from them, long-term functioning without a tendency to close the fistulous tract is
characteristic of periodontitis,
Some features of the radiographic image in the elderly should be noted. So, with
fibrous periodontitis on the roentgenogram, the periodontal gap may not be widened,
but narrowed to the limit or may not be detected at all. Sometimes the root of the
tooth on the roentgenogram looks thickened, as with hypercementosis. In
granulomatous periodontitis, the bone tissue at the edges of the granuloma more
intensively than in the neighboring areas, delays X-rays and therefore looks
sclerosed on the X-ray. Moreover, the areas of the bone facing the granuloma and
constituting its outer border have clear, even edges. And the outer sections of the
sclerosed bone sections have uneven, fuzzy edges. Similar changes in the bone in
the circumference of the focus can be observed on the roentgenogram and with
granulating periodontitis.
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Root apex resection, apicoectomy, or, rather, granulemectomy (this operation can be
performed while preserving the root apex), aims to eliminate a chronic pathological
focus in the bone, while preserving the tooth.
More often, the operation is performed on single-rooted teeth of the upper and lower
jaws, less often on small and large molars. This is explained, on the one hand, by the
cosmetic value of the anterior teeth, and, on the other hand, by the convenience of
performing this operation and the possibility of thorough filling of the root canals.
Malmstrom M. et. al. (1982) tested the effectiveness of retrograde (before surgery)
and direct (before surgery) filling of teeth canals in 154 patients. It was noted that
the restoration of bone tissue in the area of operation occurs faster after direct filling
of root canals during tooth resection. My observations fully confirm the authors'
research on the inexpediency of retrograde filling.
B.D. Kabakov, A.S. Ivanov (1978) indicate a high (91.6%) positive effect of the
resection of the apex of the roots of small and large molars. We have not seen such
success in our practice. However, in some cases, we have observed a positive effect.
Failures after root apex resection are most often caused not by errors in the technique
of the operation, but by errors in determining the indications and contraindications
to the mentioned surgery. The indications for the operation are: fracture of the upper
third of the root; curvature of the root apex, which interferes with the administration
of apical therapy; fracture of the instrument in the root canal; lack of success from
apothecary therapy; excessive introduction of filling material and its spread under
the periosteum; subperiosteal granulomas; peri-root cysts, in the cavity of which the
tops of the roots of the teeth are located.
Contraindications to the operation are: periodontitis, acute and exacerbated chronic
periodontitis, tooth mobility; exposure of the anatomical neck of the tooth;
involvement in the pathological process of more than one third of the apex of the
tooth with cysts; apicolateral and lateral granulomas; discoloration of the tooth root;
the absence of a part of the anterior wall of the alveoli; old age of the patient.
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The essence of this operation is that after tooth extraction with minimal trauma, it is
immersed in a warm (37 ° C) isotonic sodium chloride solution with the addition of
antibiotics. The replantable tooth cannot be stored in formalin and alcohol. The well
is cleaned of granulations, washed with a solution of antibiotics. Under aseptic
conditions, mechanical cleaning of the root canals and carious cavity of the tooth is
performed, they are sealed with phosphate cement or fast-hardening plastic. Then
the apex of the root is resected, the root stump is expanded and deepened with a
boron within the cement border and filled with amalgam or plastic, after which it is
inserted into the hole. Single-rooted teeth are fixed for two weeks with a quick-
hardening plastic or metal splint. Multi-rooted teeth may not need to be fixed. For
the entire period of immobilization of the tooth, strict hygienic care for the oral
cavity and a gentle diet are prescribed. Even with an impeccable operation technique,
the roots of the replanted tooth, on average, dissolve after 8-10 years, the tooth
becomes mobile and has to be removed. Even so, this interference is justified. The
most significant periods of replantable teeth are observed during the transplantation
of an accidentally dislocated or accidentally removed healthy tooth (according to our
data, more than 10 years).
There are 3 types of fusion of the transplanted tooth with the alveoli: periodontal -
occurs when the periosteum of the alveoli and the remnants of the periodontium on
the roots of the tooth are completely preserved; periodontal fibrous - with partial
preservation of the periosteum of the alveoli and the remnants of the periodontium
at the root of the tooth; osteoid - with complete removal of the periosteum of the
alveoli and the periodontium of the tooth root. The prognosis of the viability of the
replanted tooth is the most favorable for periodontal disease and the least for the
osteoid type of engraftment. The replanted tooth, according to our observations, can
function for 10 years or more.
BC Moroz (1969) proposed to transplant a tooth from a corpse to a patient -
orthotopic allotransplantation. However, due to the complexity of material
collection, the operation was not used in clinical practice.
The surgical methods used for the treatment of chronic periodontitis include
hemisection, root amputation, coronary-radicular separation.
Hemisection means the removal of the root together with the adjacent coronal part
of the tooth. Amputation means removing the entire root while preserving the crown
of the tooth.
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The indications for these surgical interventions are: the presence of bone pockets in
the area of one of the roots of the premolar or molar; cervical caries of one of the
roots; fracture of the tooth root, vertical split of the tooth; the presence of inter-root
granulomas, rarefaction of the apex of the interalveolar septum after perforation of
the bottom of the pulp chamber during tooth treatment; cases when a tooth is used
as a support (under a bridge) and when an X-ray image shows a significant bone loss
at one of its roots, as well as the impossibility of performing a root apex resection.
Carrying out amputation of the tooth root can hardly be considered advisable, since
food accumulates under the remaining part of it, which causes chronic inflammation
of the soft tissues.
Contraindications for hemisection and root amputation include: a significant defect
in the bone tissue of the hole; the case when the tooth does not represent functional
and cosmetic value; the presence of accrete roots, as well as acute inflammation of
the oral mucosa and impassable root canals of the teeth to be preserved.
Hemisection and amputation of the tooth root is carried out in two ways - with
exfoliation of the muco-periosteal flap and without its exfoliation. The technique for
carrying out these surgical interventions is simple and described in sufficient detail
in the literature. Therefore, we do not consider it necessary to dwell on its
presentation. It should only be recalled that after these operations, with the existing
mobility of the remaining fragment, it is necessary to splinter it with orthodontic
splints or splints made of composite materials. Literature data (V.P. Poltavsky, 1976;
V.P. Pochivalin, 1984, and others) indicate a high efficiency (in 90-100% of
patients) of such surgical interventions in the treatment of patients with chronic
periodontitis.
Under coronary-radicular separation, it is necessary to understand the dissection of
the tooth into two parts (used in the treatment of molars of the lower jaw) in the
bifurcation area, followed by careful smoothing of the overhanging edges, curettage
of the inter-root pathological pocket and covering each of the root segments with a
crown.
The indications for the operation are: the presence of small inter-root granulomas,
perforation of the bottom of the pulp chamber with rarefaction of the apex of the
inter-root septum. The operation is contraindicated in pathological processes in the
area of the interroot septum, the elimination of which can lead to exposure of more
than 1/3 of the length of the roots.
After performing the surgical intervention, a protective bandage is applied to the
area of the formed pathological pocket and the tooth fragments are fixed with a
splint, strengthening it by the adjacent teeth.
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In cases where the above methods of surgical treatment do not give a positive effect,
they resort to tooth extraction surgery.
Surgical treatment of periodontitis in elderly people includes one type of surgery -
tooth extraction. Replantation of the tooth and resection of the apex of its root in the
elderly are inapplicable and even contraindicated.
I would like to briefly dwell on the operation, which is not directly related to chronic
periodontitis, but is performed on the alveolar process of the jaw and associated with
the previously described.
Compactosteotomy is an operation aimed at reducing the resistance of the compact
bone substance before the upcoming movement of an abnormally located individual
tooth or a group of teeth.
The indication is deformation of the dentition, in which orthodontic treatment is
ineffective: implantation of teeth, their rotations and movements. A contraindication
is considered to be diseases that inhibit the regeneration processes (rickets,
osteodysplasia, diabetes mellitus, etc.).
Preparation for the operation consists in sanitation of the oral cavity. The technique
of intervention is as follows: cutting out a muco-periosteal (trapezoidal flap;
perforation of the compact layer of the jaw in the form of a "comb", the teeth of
which enter between the moved teeth (interalveolar osteotomy), and the base is
located above this group of teeth (performed with the vestibular and palatine or
lingual sides); wound suturing; fixation and instrumental movement of teeth.
Tooth autotransplantation - transplanting it into another alveolus, is rarely used.
When a dystopic tooth is introduced into the edentulous areas of the alveolar process,
it is necessary to create a hole for the tooth to be moved with the help of a bur, and
after the transplantation of the latter, fix it with a splint, which is held for at least two
weeks. When performing compactosteotomy or autologous transplantation, care
must be taken to avoid opening the floor of the maxillary sinus or nasal cavity. When
moving or osteotomy over the tops of the teeth, the pulp should be removed,
followed by filling of the root canals according to generally accepted methods.
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3. Educational technologies
- Lecture with multimedia visualization.
- Problem lecture.
4. Literature:
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Throughout a person's life, there is a loss of enamel and dentine as a result of their
abrasion. This natural process begins as soon as the teeth erupt and begin to perform
their function. Its severity depends on the type of teeth closing, the hardness of the
enamel and dentin, the magnitude of the chewing pressure and the properties of the
food consumed.
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Natural erasure of enamel occurs in two planes - horizontal and vertical. Abrasion
in the horizontal plane is observed along the incisor edge of the incisors, tubercles
of the canines, premolars and molars. The associated decrease in the height of the
crowns of the teeth should be considered as an adaptive response of the body. The
fact is that the vascular system and other tissues of the periodontium and
temporomandibular joint change with age. However, the decrease in the
functionality of these organs is compensated by a decrease in the size of the external
lever of the tooth and the flat shape of the occlusal surface.
In some people, natural functional abrasion is slowed down or absent. This could be
explained by the use of soft food, a deep bite that impedes the lateral movement of
the lower jaw, weakness of the chewing muscles. However, there are patients who
have a normal bite and they eat a wide variety of food, and the abrasion is so poorly
expressed that at the age of 40, the tubercles of the molars and premolars remain
almost unchanged. The reasons for this are unknown, but it is believed that this
phenomenon is associated with tooth mobility caused by congenital or acquired
functional periodontal deficiency. Often these patients suffer from periodontitis.
Increased abrasion violates the anatomical shape of the teeth: the tubercles
disappear, the cutting edges of the incisors, the height of the crowns decreases. With
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a direct bite, the cutting edges and chewing surface of all teeth are erased, with a
deep bite - the labial surfaces of the lower and palatal - the upper apron of the teeth.
Increased abrasion, having arisen once, steadily grows, it deepens in places where
dentin is exposed, and lingers somewhat where enamel is preserved. As a result, the
erasure facets have the appearance of smoothly polished cups or crater-shaped
recesses, the edges of which are bounded by sharp enamel projections. Formation:
crater-like facets due to unequal hardness of enamel and dentin. The latter is softer
and therefore wears off faster.
Etiology.
1) partial loss of teeth (decrease in the number of antagonizing pairs of teeth, mixed
function, etc.);
3) hypertonicity of the masticatory muscles of central origin and associated with the
profession (vibration, physical stress);
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III. Occupational hazards acid and alkaline necrosis, dustiness, hydrochloric acid
intake for achilia.
Some of the reasons listed can cause generalized abrasion, and some - only local
damage. For example, with congenital insufficiency of enamel and dentin, a
generalized form of increased abrasion should be expected, while with functional
overload, only the teeth that maintain the interalveolar height are involved in the
process.
Pathogenesis.
With pathological abrasion of the hard tissues of the teeth, various morphological
and functional disorders in the dentoalveolar system are observed: hyperesthesia of
hard tissues of the teeth, deformation of the dentition, shortening of the interalveolar
distance and the lower third of the face, the function of the masticatory muscles, pain
dysfunction of the temporomandibular joints. Dysfunction of the masticatory
muscles is manifested by pain during their contraction. Their bioelectric activity
increases, and it is also noted in the phase of physiological rest, asynchronous
contractions appear, regional blood circulation in the periodontium is disrupted.
This is due to the blockage of sagittal and transverse movements of the lower jaw.
Our data indicate that three factors play an important role in the pathogenesis of
dysfunctions of the masticatory muscles and temporomandibular joints: a decrease
in the height of the bite and shortening of the lower third of the face, blocking of
movements of the lower jaw, its lateral and distal displacement.
2) extensive defects of the dentition in the lateral regions (areas of the premolars and
molars),
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I degree - abrasion of the enamel of the cutting edges of the incisors and canines and
the upper part of the masticatory tubercles of premolars and molars. Abrasion occurs
within the enamel of the teeth and partially dentin.
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III degree - reduction as a result of erasure of the height of the crown of the tooth to
2/3 of the normal size. Erasure of tooth tissues occurs within the replacement dentin
with translucence of the tooth cavity.
IV degree - erasure to the level of the tooth neck. The entire crown of the tooth is
erased.
-Horizontal
-Vertical
-Faceted
-Patterned
-Stepped
-Cellular
-Mixed
Clinic.
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Localized compensated wear also causes a decrease in the height of the crowns of
individual teeth. At the same time, worn teeth retain contact with antagonists due to
hypertrophy of the alveolar part (vacate hypertrophy) in this zone, which leads to
dentoalveolar lengthening. The interalveolar height and face height remain
unchanged.
The facial skeleton in patients with this form of abrasion is characterized by:
2) the absence of changes in the position of the lower jaw and the preservation of
the vertical dimensions of the face;
3) deformation of the occlusal surface and a decrease in the depth of the incisal
overlap;
8) a decrease in the length of the roots of the anterior teeth and the first premolars.
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Increased abrasion can be combined with the loss of part of the teeth,
The treatment of patients with abnormal tooth wear presents significant difficulties.
This is due to the fact that the pathogenesis of the disease has not been finally
clarified. In this regard, conservative methods of pathogenetic therapy have not been
developed that would stop the process of erasing hard dental tissues. For the
treatment of pathological abrasion of teeth, two methods have been proposed -
medication and orthopedic. Drug treatment is aimed mainly at eliminating
hyperesthesia of hard tissues and is effective only in the initial stages of the
pathological process.
When planning orthopedic treatment, one should take into account the clinical type
of pathological tooth wear (horizontal, vertical, localized, generalized, compensated,
uncompensated), the depth of the lesion (I, II and III degree of wear), the presence
of complications (dysfunction of the chewing muscles and temporomandibular joint)
... The principles of planning and carrying out orthopedic treatment in the presence
of temporomandibular joint dysfunction and in its absence differ significantly.
In case of pathological abrasion of hard tooth tissues of the 1st degree (up to 1/3 of
the crown length), treatment depends on the clinical type of abrasion, the patient's
age and the functional state of the masticatory muscles. In patients with generalized
abrasion of teeth without dysfunction of the masticatory muscles (uncomplicated
form), treatment consists in rational prosthetics with one-stage restoration of the bite
height without prior restructuring of the myotatic reflexes of the masticatory
muscles.
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The main goals of prosthetics for pathological abrasion of hard dental tissues are:
To achieve these goals, both fixed (crowns, bridges) and removable clasp prostheses
with occlusal overlays can be used. It is inappropriate to use stamped crowns and
brazed bridges, since crowns, especially gold crowns, are rubbed over the occlusal
surface, and bridges often break at adhesions.
Over time, the plastic wears out, which leads to a decrease in the interalveolar
distance with all its complications. Fixed dentures should be solid and made of
materials that are not subject to increased abrasion. These primarily include metal
and porcelain. You can also use high-strength plastic (isosit).
It should be borne in mind that in the area of the anterior teeth the occlusal load is
significantly less than in the area of the second premolars and molars, therefore, the
risk of porcelain spalling is less. In addition, in the manufacture of solid metal
crowns on molars, instead of metal-ceramic, much less hard tissues of abutment teeth
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are ground, which in turn reduces the risk of pulp overheating and the occurrence of
traumatic pulpitis.
This significantly reduces the risk of chipping of the porcelain. To avoid functional
traumatic overload of the periodontium and the introduction of abutment teeth, it is
important to correctly draw up a plan of orthopedic treatment and choose a rational
design of prostheses. Of great importance for the prevention of recurrence of the
shortening of the interalveolar distance after the installation of prostheses are the
choice of their design and the sequence of prosthetics.
When choosing a design, it is necessary to take into account the size and topography
of dentition defects, as well as the condition of the tissues of the marginal
periodontium. In patients with intact periodontal disease or periodontitis of mild and
moderate severity in the presence of small (1-2 teeth) included defects in the
dentition, one-piece bridges can be used in the lateral sections. To achieve multiple
contact, single crowns can be additionally fabricated.
Clasp prostheses transfer part of the occlusal load to the mucous membrane of the
alveolar process of the jaw. Thus, the periodontium of the abutment teeth is relieved
and their loosening and penetration into the alveolar process is prevented. The
gingivomuscular reflex, which was mentioned above, also helps to prevent the
functional overload of the periodontium, the introduction of abutment teeth and the
recurrence of the shortening of the interalveolar distance (declining bite) when using
clasp prostheses.
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3. Educational technologies
- Lecture with multimedia visualization.
- Problem lecture.
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Many defects and lesions of hard tooth tissues cannot be restored (restored) with the
help of filling materials. To recreate the anatomical shape of the teeth and replace
the defects in the dentition, it is necessary to use fixed orthopedic structures. It
follows from this that knowledge of the basics of preparing teeth for artificial crowns
is necessary and relevant in modern conditions.
An artificial crown is a fixed prosthesis that restores the anatomical shape and
function of the tooth and prevents its further destruction.
In 1984, I. Yu. Milikevich proposed to use the index, which he called IROPZ (index
of destruction of the occlusal surface of the tooth), to select the method of restoration
of the crown part of the tooth. The entire area of the occlusal surface of the tooth is
taken as a unit. The fracture index (surface area of the cavity or filling) is calculated
from one (the entire occlusal surface). When the IROPZ is equal to 0.55-0.6 or more,
that is, when the surface is destroyed by more than 55%, the use of inlays is shown
in order to prevent further destruction. With an index of 0.6-0.8, the use of artificial
crowns is shown, when the index is greater than 0.8, the manufacture of pin
structures is shown.
artificial crowns
restorative fixing
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temporary permanent
2.
By design:
artificial crowns
3.
Depending on the material:
artificial crowns
4.
Depending on the manufacturing method:
artificial crowns
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During the preparation of teeth, the doctor has the following tasks:
● create a tooth shape that would satisfy all the requirements for the chosen
design of artificial crowns, taking into account the removal of the minimum amount
of hard tissue;
● perform grinding without injuring adjacent teeth, marginal periodontium,
without destroying or injuring the pulp of the tooth.
● prepare in such a way that the patient experiences as little discomfort as
possible.
The volume of hard tooth tissues to be ground off depends on the wall thickness of
the chosen artificial crown:
For odontopreparation of teeth for artificial crowns, shaped heads of various shapes
and separation discs are used (Fig. 1, 2).
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Scheme of the indicative basis of actions when preparing teeth for stamped crowns.
1 2 3
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4. Dissection of the The same, cylindrical Grinding of the edges of the tooth
oral, vestibular, heads, separation disc. in the places of transition of the
occlusal surfaces labial and oral surfaces into
and incisal edges, contact surfaces, the tooth
grinding the acquires a shape close to a
equator of the tooth. cylinder, the diameter of which
does not exceed the diameter of
the neck of the tooth, the
anatomical shape of the occlusal
surface and the incisal edge is
preserved, but reduced by the
thickness of the artificial crown.
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A ledge is a platform that bears a load and prevents the crown from sinking under
the gum and injuring the periodontal joint.
The choice of the ledge shape depends on the shape, size, inclination, topography of
the tooth and the state of the tissues of its coronal part. The shoulder width ranges
from 0.5 to 1 mm. An uneven shoulder width is allowed in cases where there is a
narrowing of the side surfaces and there are no favorable conditions for creating a
shoulder with a width of 1 mm. The ledge can be straight (Fig. 3, b), with a notch
(Fig. 3, c), with a beveled edge (Fig. 3, d), with a top (Fig. 3, e) or beveled (Fig. 3, f
).
Rice. 3. Variants of the formation of the gingival part of the tooth stump.
In the cervical region of the teeth of the anterior group and premolars at the level of
the gums, a width of 0.3-0.5 mm (up to 0.8 mm) is created.
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Scheme of the indicative basis for the preparation of teeth for artificial crowns made
of plastic.
1 2 3
3. Dissection of the Probe, mirror, tips, The walls are smooth, even,
vestibular and oral diamond heads there are no overhanging edges.
surfaces. Hard (cylindrical, conical, The ledge is at the level of the
fabrics are sanded to end, marking). gums or slightly below.
a thickness of up to 1
mm, a shoulder is
formed.
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Scheme of the OOD "Preparation of teeth for solid metal and combined artificial
crowns."
1 2 3
1. Separation. The Probe, mirror, dental The walls are smooth, even,
contact surfaces are handpieces, separation converging at an angle of 5-
prepared on a cone at discs, shaped diamond 7o. The ridge is created
an angle of no more heads. partially, follows the contours
than 5-7o. Grinding up of the gingival papilla.
to 1 mm of hard tooth
tissues with the
formation of a ledge.
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The main (structural) materials are called the materials from which dentures,
apparatus, and fillings are made.
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1. 6.
impression; insulating;
2. modeling; 7. low-melting alloys;
3. molding; 8. solder;
4. abrasive; 9. fluxes;
5. polishing; 10. whitened.
1. impression materials;
2. filling materials;
3. waxes and wax compositions.
More than 500 alloys are used in dentistry. International standards (ISO, 1989)
divide all metal alloys into the following groups:
gold;
gold-palladium;
silver-palladium.
2.
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alloys,
used in orthopedic dentistry
● ● ● ● ●
for removable two-component mechanical fusible; heat
prosthetics; (Argident 3: Au- mixture; ● refract treatment;
● for cermet Pt); ● solid ory; ● therm
prostheses; ● three- solution; o-
● for metal- component ● chemi mechanical;
polymer (Rebar 3: Au- cal ● chemi
prostheses; Pd-Ag); compounds. cal-thermal.
● for solid ● multicom
metal structures; ponent (Viron-
● for stamped 99).
crowns.
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3. Educational technologies
- Lecture with multimedia visualization.
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The most common prostheses used to restore a decayed tooth crown are full artificial
crowns. Due to the fact that they have a different design and are intended for
different purposes, they are systematized according to certain criteria:
By manufacturing method:
● stamped;
● cast;
● brazed (seam) - now they are practically not used.
3. Depending on the material:
● metal (gold alloys, stainless steel, cobalt-chromium alloys (KHS), silver-
palladium, titanium);
● non-metallic (plastic, porcelain);
● combined, that is, lined with plastic, porcelain or other ceramic masses (metal-
plastic and cermet).
4. By appointment:
● restorative;
● supporting (in bridges or other types of prostheses);
● fixing (for holding drugs, orthodontic or maxillofacial appliances);
● splinting;
● temporary and permanent.
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Contraindications:
1. Covering intact teeth with crowns, if this is not caused by the design features
of the dentures.
2. Untreated foci of chronic inflammation in the region of the marginal or apical
periodontium.
3. Pathological mobility (III degree according to Entin).
4. General grave health condition.
1st clinical stage After examining the oral cavity, making a diagnosis, the doctor
should issue an order, carry out anesthesia, since odontopreparation is accompanied
by significant trauma to dental tissues. Preparing a tooth for a stamped crown
consists in giving it a certain shape, most often resembling a cylinder and providing
a free overlay of a crown, the edge of which, entering the gingival groove, should
tightly cover the neck of the tooth. The position of the patient and the doctor during
odontopreparation When grinding, a certain sequence should be observed. It is most
advisable to start with the preparation of the contact surfaces. A thin one-sided
separation disc is inserted into the straight handpiece, with the abrasive surface
facing the mesial or distal tooth surface. The disc is mounted above the contact point
parallel to the tooth axis. Processing is carried out intermittently at minimum speed,
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without strong pressure of the cutting tool on the tooth in order to avoid disk
jamming and soft tissue injury. With short-term touches, hard tissues are removed
from the tooth until a visible gap appears between the teeth. Keeping the disk parallel
to the long axis of the tooth, grind the contact surface until the cutting tool touches
the neck of the tooth. Excessive removal of tooth tissue with a ledge should be
feared. It is also possible to use a fine fissure bur (diamond or carbide) on a turbine
unit. Mechanical preliminary separation is possible in very tight teeth. For this, a
loop of ligature bronze-aluminum wire is applied around the interdental contact, the
ends of which are twisted until light pressure is felt on the teeth. After a day, the
teeth move away from each other, and the resulting gap makes it easy to prepare the
contact surface with a separation disc. Grinding of the contact surfaces is considered
complete if all overhanging edges in the cervical region have been removed from
the contact sides. The accuracy of the preparation is controlled by a probe. The
smoothness of its movement under the gum indicates a flat surface. The ground
surfaces should be parallel to the long axis of the tooth. For a novice doctor, it is
difficult to determine the parallelism of the sides of the tooth, therefore, the shape
of not a cylinder, but a truncated cone with the apex facing the antagonist teeth, may
be recommended. The contact walls of the tooth will converge to each other, but
their inclination should be barely noticeable (1-3 °). The thickness of the hard tissue
layer removed from the contact surfaces will be minimal at the neck and more
significant at the equator, chewing or cutting surface of the tooth. Grinding of the
vestibular and oral surfaces is carried out approximately according to the same rules.
The thickness of the removed tissue layer depends on the severity of the equator,
anatomical shape, size and position of the crown in the dentition. First, the most
prominent parts of the tooth in the equator are removed with large heads. And then
the vestibular and oral surfaces are aligned with diamond, cylindrical or wheel-
shaped heads, achieving a smooth transition between them, without sharp edges: In
the front teeth, the labial surface can be ground with a separation disc. Having given
the patient's head a more horizontal position, the separation disc is placed parallel to
the long axis of the crown and the necessary tissue layer is gradually removed with
intermittent movements. Particular attention should be paid when processing the
pre-gingival ridge. To prevent injury to the gingival margin, when grinding the pre-
gingival ridge, a conical or reverse-truncated diamond head is used. Preparation
control is carried out visually or using an angled probe. After grinding the lateral
walls of the anterior teeth, the cylindrical shape is obtained only in the cervical
region. In other areas, the anatomical shape inherent in the tooth is preserved. The
oral surface of the anterior teeth is ground down to the thickness of the stamped
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crown. The preparation of the side walls is finished by grinding the edges of the
tooth at the transition points of the labial and oral surfaces to the contact surfaces.
The smoothness of the transition should correspond to the curvature of the cervical
part of the tooth in the corresponding areas. When grinding the occlusal surface or
incisal edge, it is necessary to maintain their inherent anatomical shape. To do this,
in premolars and molars, a layer of hard tissue is alternately removed in the area of
tubercles and fissures, and the incisor and canines are ground off the incisal edge
and, additionally, the vestibular and oral sides. The control of the amount of
removed fabrics is carried out using carbon paper folded in 16 layers. This
corresponds approximately to a crown thickness of 0.25-0.3 mm. When placed
between the prepared tooth and the antagonists, it stains the areas of the chewing
surfaces that are not sufficiently disconnected from each other when the dentition is
closed. Grinding is carried out until the carbon paper easily passes between the teeth.
Abrasion of the hard tissues of the chewing surfaces of molars and premolars leads
to separation from the antagonists. In the anterior teeth, separation from the
antagonists in the central occlusion position is achieved only with direct or
orthognathic bites with minimal overlap. With a deeper overlap, the separation can
be achieved by grinding the palatal surface from the tubercles to the incisal edge of
the upper anterior teeth, as well as the labial surface and the incisal edge of the lower
anterior teeth. the upper teeth and from the lingual side of the lower teeth, as well as
the transition of the chewing surface to the lateral ones. Otherwise, the volume of
the artificial ladybug will be too large, and she will protrude from the dentition.
When preparing a tooth for a crown, one should pay attention to its position in the
dentition. For example, when turning along the axis, you can correct the position of
the tooth by grinding the most protruding areas - mesio-vestibular and oral-distal.
1. refusal of the patient due to fear of pain - it is necessary to find contact and
give anesthesia, if there are no contraindications.
● Anesthesia involves more careful adherence to the preparation regimen due to
the danger of pulp overheating;
damage to the soft tissues of the cheek, tongue, especially when working with
separation discs (more often the gums and tongue are cut).
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grinding during separation not to the neck of the tooth, but higher, penetrating into
the thickness of dentin
Prevention of complications:
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(extremely rarely), double impressions taken with the help of silicone, polysulfide,
polyester masses are especially accurate. When taking an impression, you must
follow the correct sequence of actions.
Provisional crowns The prepared tooth becomes sensitive to thermal, chemical and
infectious irritants. To avoid this, the tooth should be covered with a temporary
crown or a celluloid cap.Method: before preparation, impressions are taken from the
teeth with an alginate material, into which a self-polymerizing (cold polymerization)
plastic is applied, and after processing the teeth is introduced into the oral cavity
until final hardening. Then the provisional crown is processed. It is strengthened on
the tooth immediately after preparation with temporary cement. 1st laboratory stage
Impressions from the alginate mass are immersed in a solution of potassium
permanganate for 5 minutes. Plaster prints are glued together with boiling wax,
immersed in cold water for 5-10 minutes, and plaster models are cast. In order to fix
them in the position of the central occlusion in the occluder, wax bases with bite
rollers are used. On plaster models along the boundaries outlined with a chemical
pencil, templates or bases are made from dental wax. In the area of dentition defects,
rollers are installed, the width of which in the lateral sections is not more than 1-1.2
cm, and in the area of the front teeth - 0.6-0.8 cm, the height of the rollers in the
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lateral sections is 1-2 mm above the chewing surface natural teeth, and in front of
their occlusal plane should be located at the level of the cutting edges. Obtaining a
plaster model. The impression taken out of the water is shaken off, put on a table
with a spoon upwards so that all the remaining water is glass, and proceeds to stir
the plaster, adhering to the rules. Pour as much water into the rubber cup as needed
to saturate the plaster. This is roughly one part water for two parts gypsum. Gypsum
is poured into the water in small portions until there is no free water on top. Only
after that, the gypsum is vigorously stirred with a special spatula so that a
homogeneous thick mass without lumps is obtained. A model cast from too liquid
gypsum will be brittle, since the gypsum will bind only the amount of water that is
required according to the chemical formula, the rest of the water will evaporate, and
the model will become porous, loose. Excessively thick gypsum will not give an
accurate impression, since it will not display the relief of the mucous membrane
(when taking an impression) or all the indentations in the impression (when
receiving a model). In such gypsum, pores can turn out, since air bubbles will not
have time to escape through its thickness before it hardens. It is necessary to strictly
observe the rules for storing gypsum, arising from its properties described above. It
is necessary to protect the gypsum from moisture and contamination. Gypsum is
stored in thick paper bags or in resealable barrels in a dry place. Before use, it is
recommended to sift the gypsum through a sieve in order to remove lumps and
incidental impurities.To better fill all the depressions in the impression and to
eliminate air bubbles, it is recommended to pour the gypsum in small portions onto
the protruding parts of the impression and shake it at the same time. There are special
vibrators for this purpose, but good results can be achieved without a vibrator by
tapping the impression spoon against the edge of the rubber cup. The impression is
filled with plaster of paris slightly above its edges; a little plaster is poured on a table
covered with oilcloth and, turning the print over with a spoon up, put it on the plaster
in a horizontal position. Model height must be 1, 5-2cm at its thinnest part. Smooth
the edges of the model with a spatula and wait until the plaster has completely
hardened. Separation of the model from the plaster impression. When the plaster has
hardened, the impression tray is removed with light blows with a hammer or spatula.
The finished model is trimmed, giving it more accurate contours, while trying to
keep all its anatomical details intact. The lower model does not have a recess on the
lingual side so as not to weaken it. Any damage, such as a fracture of the model,
breakage of the alveolar ridge, scratches in the area of the working part, etc., make
the model unsuitable for further work. In some cases, it is permissible to stick a
broken off plaster tooth or part of it. The broken tooth must be precisely attached to
the model and reinforced with cement or waterproof glue (celluloid solution in
acetone). 2nd clinical stage To compare the models in the occluder, it is necessary
in the clinic to determine the central occlusion or the central ratio of the jaws.
Depending on the presence of antagonizing pairs of teeth and their location, the
establishment of the dentition in the central occlusion is carried out differently. With
the existing teeth antagonists, the height of the bite is fixed by natural teeth. Is a
fixed bite, anatomical and physiological norm for a given patient. In case of non-
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fixed bite, wax templates are fitted in the oral cavity and the mesiodistal position of
the lower jaw is fixed in such a way that the bite height obtained using the occlusal
rollers is 2-4 mm less than the height of relative physiological rest. Determination
of the central ratio of the jaws. According to the degree of difficulty in determining
the central ratio of the jaws, four groups of defects in the dentition are distinguished:
When making 1-2 crowns, the central occlusion is determined by its signs, guided
by which the models are put into the occluder, that is, they are most often dealing
with the 1st group. drawn up in the central occlusion, fixed in the articulator. Having
received an idea of the nature of the closure of the dentition and the ratio of the jaws,
they proceed directly to the manufacture of the prosthesis. 2nd laboratory stage
Methods of stamping metal crowns. Plaster models, fixed in the articulator, examine
and check the degree of separation of the prepared tooth from the antagonists. With
an eye scalpel, plaster is removed, which violates the clarity of the contours of the
neck of the tooth. Engraving is done carefully to avoid damaging the cervical part
of the tooth. With inaccurate removal of excess gypsum, its perimeter shrinks or
expands. You cannot deepen the gingival groove, just mark its precise contours. If
the interdental spaces are not wiped out by the impression material and are filled
with plaster, it is carefully removed with a fine file or an eye scalpel. The contours
of the gingival margin should be clearly defined along the entire perimeter of the
tooth neck. With a sharpened chemical pencil, outline the clinical neck of the tooth.
The resulting line serves as a guideline for determining the length and width of the
crown edge, as well as the degree of its immersion in the gingival groove. The
anatomical shape of the artificial crown is restored with a special modeling wax and
a modeling spatula. Cut the wax with the sharp edge of the spatula, rounded - to melt
it on the fire of a burner To obtain the first layer, boiling wax is poured onto the
stump of a plaster tooth, holding the model with the base up, the tip of a spatula with
boiling wax is applied to the surface of the tooth from the neck to the cutting edge
or chewing surface, preventing the molten wax from getting into the neck area and
maintaining the accuracy of its contours. By layering molten wax on the surface of
a plaster tooth, an increase in volume is achieved, which is necessary to restore the
anatomical shape. To obtain an imprint of antagonist teeth on the simulated tooth,
their occlusal surface is lubricated with a thin layer of oil, petroleum jelly, or simply
moistened with water. Having received the imprint of the antagonists on warm wax,
they proceed to modeling the artificial crown. The wax is cooled and the excess is
scraped off. The volume of the modeled tooth is reduced by the thickness of the
stamped crown metal - 0.25-0.3 mm. The relief of the occlusal surface is modeled
taking into account the age characteristics of natural teeth. After modeling, the wax
surface should be smooth. All surfaces in contact with the antagonists are separated
by the thickness of the metal. The modeled tooth is cut out from the plaster model.
The crown part of the tooth in the direction of the longitudinal axis should continue
approximately to the height of two more crowns. The thickness of the so-called root
part of the plaster die must exactly match the cross-sectional profile in the neck area,
a violation will lead to the manufacture of an artificial crown with an inaccurate
entrance hole. The marking of a plaster die is carried out in several ways: The relief
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of the chewing surface is modeled taking into account the age characteristics of
natural teeth. After modeling, the wax surface should be smooth. All surfaces in
contact with the antagonists are separated by the thickness of the metal. The modeled
tooth is cut out from the plaster model. The crown part of the tooth in the direction
of the longitudinal axis should continue approximately to the height of two more
crowns. The thickness of the so-called root part of the plaster die must exactly match
the cross-sectional profile in the neck area, a violation will lead to the manufacture
of an artificial crown with an inaccurate entrance hole. The marking of a plaster die
is carried out in several ways: The relief of the chewing surface is modeled taking
into account the age characteristics of natural teeth. After modeling, the wax surface
should be smooth. All surfaces in contact with the antagonists are separated by the
thickness of the metal. The modeled tooth is cut out from the plaster model. The
crown part of the tooth in the direction of the longitudinal axis should continue
approximately to the height of two more crowns. The thickness of the so-called root
part of the plaster die must exactly correspond to the cross-sectional profile in the
neck area, a violation will lead to the manufacture of an artificial crown with an
inaccurate entrance hole. in contact with antagonists, they are separated by the
thickness of the metal. The modeled tooth is cut out from the plaster model. The
crown part of the tooth in the direction of the longitudinal axis should continue
approximately to the height of two more crowns. The thickness of the so-called root
part of the plaster die must exactly match the cross-sectional profile in the neck area,
a violation will lead to the manufacture of an artificial crown with an inaccurate
entrance hole. The marking of a plaster die is carried out in several ways: in contact
with antagonists, they are separated by the thickness of the metal. The modeled tooth
is cut out from the plaster model. The crown part of the tooth in the direction of the
longitudinal axis should continue approximately to the height of two more crowns.
The thickness of the so-called root part of the plaster die must exactly match the
cross-sectional profile in the neck area, a violation will lead to the manufacture of
an artificial crown with an inaccurate entrance hole. The marking of a plaster die is
carried out in several ways:
1. stepping back about 1 mm from the line of the clinical neck of the tooth,
indicated with a chemical pencil, a groove 0.5 mm deep is made parallel to it. - it
serves as a guide for determining the length of the edge of the metal crown.
2. first mark the second line with a chemical pencil, located at a distance of 1
mm from the first, and then engrave the groove, stepping back from the second line
by another 1 mm. Advantage - preliminary shortening of the crown along the groove
allows you to subsequently clarify its length along the second line drawn with a
chemical pencil. The pre-built headroom significantly reduces the likelihood of
over-shortening the crown and thus ensures the highest precision in the fabrication
of the prosthesis.
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With a spatula, remove excess gypsum in the entire cervical part and give it a cross-
sectional profile equal to the neck contour. Metal stamps are prepared using gypsum
stamps. To obtain an exact copy of a gypsum stamp from the metal, it is soaked in
water or in a solution of office glue. Liquid gypsum is poured into a rubber ring with
a diameter of 3-4 cm and a height of 4-5 cm. The wet gypsum stamp is preliminarily
coated with a thin layer of gypsum, removed and re-immersed in it, and then
completely placed in liquid gypsum so that the stamp is located strictly vertically
and is located in the very center of the rubber ring. This makes it easier to remove
the plaster die from the mold and maintains its accuracy. The hardened plaster block
is pushed out of the rubber ring, give the block the shape of a rectangle and make
wedge-shaped longitudinal grooves with a depth of 3-4 mm on two opposite sides,
leaving a layer up to the plaster die of at least 3-5 mm. The longitudinal grooves
should be oriented to the plaster die so that the fracture line passes exactly through
its middle. To split the plaster mold, it is placed on the palm of the left hand, and the
blade of a plaster knife is inserted into a longitudinal wedge-shaped groove. If the
fracture line is misaligned, it may be difficult to remove the plaster die from the
mold. In this case, on the half of the mold where the gypsum stamp remains, an
additional wedge-shaped groove is made strictly in the direction of the stamp and
the remaining part of the gypsum block is split along it. After releasing the plaster
die, all parts of the plaster mold are folded, placing in a rubber ring and pouring into
it a molten low-melting alloy, melted in a special spoon on the flame of a gas or
alcohol burner at a temperature of 65-95 ° C. In our country, they use the Melott
alloy: tin, lead, bismuth (5: 3: 8), the melting point is 65 ° C. For each tooth, 2 stamps
are cast: the first is used for final stamping, the second for preliminary. Defects
appearing on the surface of the stamp are removed with a file carefully in the area
of the neck. Excess metal on the chewing surface is removed with burs or circles
without disturbing its relief. In this form, a metal stamp is ready for stamping a
crown. In the manufacture of several crowns, the following technique is used: liquid
gypsum is poured into a manufactured metal frame 5-6 cm wide, 2 cm high and 15-
20 cm long, into which the previously prepared teeth are lowered in a horizontal
position at half their thickness and at a distance of 1 cm from each other After the
gypsum has hardened, "locks" are made at both ends of the block in the form of
recesses and immersed in cold water for soaking, then the second half of the mold
is cast. After the gypsum of the second half has hardened, they are separated from
each other with light hammer blows and the plaster teeth are removed. Then both
halves of the mold are connected, the inlet of each plaster tooth is slightly widened
and the mold is filled with a molten low-melting alloy. For preliminary stamping, a
metal stamp under No. 2 is used. For the manufacture of stainless steel crowns,
standard sleeves of various diameters and thicknesses are used (0.20-0, 28 mm)
manufactured by industry. For stamping crowns made of gold or platinum alloy,
disks with a diameter of 23-30 mm and a thickness of 0.25-0.28 mm are used. .If
there are no sleeves of a suitable diameter and they are larger than the diameter of a
metal tooth, then they are pulled through the "Samson" or "Sharp" apparatus. In the
same way, sleeves are obtained from discs (gold, platinum). this hole of the matrix
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and is pulled with a punch. Transferring the sleeve from one hole to another, they
achieve the required diameter so that the sleeve is put on a metal stamp with some
effort. Repeated pulling of the sleeve through the holes leads to a change in the
structure of the metal and its properties, therefore, to restore the previous structure
of the metal and its properties, the sleeve must be subjected to heat treatment after
pulling and during operation. the sleeve is calcined in an oven or in the flame of a
soldering apparatus to a temperature of 1100 ° C (straw-yellow color) and quickly
cooled in water or in air. This achieves the fixation of the most stable structure of
the steel sleeve, the so-called austenitic, which is a solid solution of carbon in iron.
For an anvil, a lead base and hammers are used: copper - for steel, horn - for an alloy
of gold. An approximate shape is first given on the anvil. Putting the sleeve on a
stamp made of low-melting alloy No. 2, it is upholstered with a hammer, bringing it
closer to the shape of the stamp; hammer blows should be directed to the most
convex sections of the sleeve, driving them towards the neck of the tooth. You can
create a bed in a lead block and hammer the stamp into the sleeve with a hammer
until the first imprints of the chewing surface or the incisal edge of the tooth appear
at the bottom of the sleeve. If the advance of the sleeve meets an obstacle from the
side of the protrusion on the stamp near the neck of the tooth, then the sleeve is
removed and trimmed. Instead of lead, a low-melting alloy is used to form the
occlusal surface, pouring it into a mold, as shown. With hammer blows, the sleeve
is given an approximate shape of the future crown, achieving a tighter fit to the entire
surface of the metal stamp. A metal stamp of a tooth with a pre-stamped crown put
on it is wrapped with a linen cloth or thick paper (to prevent the ingress of moldin
between the crown and the stamp) and after placing it strictly in the center with the
chewing surface down with hammer blows or pressing in a special press, hammer it
into the mass. press and abruptly release, while the cylinder, entering the base,
strikes the pin-peak, and the molding or rubber acts as a counter-stamp, evenly
transmitting pressure in all directions and contributing to a tight fit of the crown to
the surface of the metal stamp. the surface of the crown, then they are broken with
a hammer, the crown is removed from the stamp by melting it, holding the crown
with tweezers. This concludes the laboratory stage. Single crowns are bleached,
boiled, wiped before being sent to the clinic; if the crown is intended for anchoring
a bridge, then it is not bleached due to the danger of thinning. These crowns are
whitened after the final fabrication of the bridge, so that in the case of external
stamping, the stamp is the tooth we have prepared from a fusible alloy, and the
counter stamp is a mold or unvulcanized rubber. The combined stamping method
combines elements of external and internal stamping - it is not used, The method of
combined stamping of crowns. The method of making a metal stamp was borrowed
from the outer stamping, and a metal counter stamp from the inner stamping. It is
also called punching according to the MMOM method (Moscow Medical Dental
Institute). The device consists of a steel cuvette, the inner surfaces of which are
tapered and have two protrusions in the middle line, facilitating the splitting of the
counter-stamp. The metal stamp of the tooth is prepared in exactly the same way as
for the outer stamping. After that, the surface of the metal stamp is wrapped with
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one layer of adhesive plaster, leaving the occlusal surface or incisal edge free. This
corresponds to the thickness of the metal crown. To do this, you can grease the
surface of the stamp with oil and sprinkle with talcum. Installing the holder with a
stamp in the center of the cuvette, pour a molten fusible alloy into it, after which the
cuvette is set upside down on the stand, remove the cotton wool from the hole in the
bottom of the cuvette and insert a pestle into the hole, the counter-stamp is removed
with hammer blows. The splitting of the counter-stamp and the release of the metal
stamp from it is carried out using a chisel or a plaster knife, which are inserted into
the resulting recess on the lateral surface of the stamp. When using a rod with
triangular teeth, the counter-stamp is split at the moment of its release from the
cuvette. The adhesive plaster is removed from the surface of the metal tooth, a pre-
stamped crown is put on, and, having installed in the recess of the counter-stamp,
the latter is inserted into the cuvette with light tapping with a hammer so that it
returns to its previous position. you can use the pestle with the hollow side. After
that, stamping is carried out by blows of a hammer on the stamp and pestle. The
stamp with the crown is freed from the counter-stamp in the manner described
above, that is, by knocking out the counter-stamp, melting it and releasing the crown.
grinding and polishing the crown. The crown, tested in the patient's oral cavity, is
again transferred to the dental laboratory for bleaching, grinding and polishing. In
the process of manufacturing dentures, metal parts are heat treated, which increases
and accelerates their chemical interaction with atmospheric oxygen. As a result of
this effect, an oxide film (scale) is formed on the metal surface, which impairs the
appearance of the metal, complicating the processes of processing, grinding and
polishing the surface. Removal of the oxide film from the entire surface of metal
parts is carried out using chill. As bleaching, aqueous solutions of many acids
(hydrochloric, sulfuric, nitric, etc.) and their mixtures are used. To whiten prostheses
made of gold and silver-palladium alloys, a 40% aqueous solution of hydrochloric
acid is used. The product is heated red-hot, and then dipped into a vessel with
hydrochloric acid solution and closed with a lid. After 1-2 minutes, the product is
removed from the solution and washed in running water. All work with hydrochloric
acid should be carried out in a fume hood, since its vapors have a harmful effect on
the mucous membrane of the respiratory tract. Do not allow it to come into contact
with clothing, skin or tools. After stamping crowns made of gold alloys in molds
made of low-melting metals, their particles remain on the crowns in the form of
plaque, which is removed by immersing the crown in hydrochloric acid for 2-3
minutes. Then it is thoroughly washed with water and wiped off. As chill for
processing crowns made of stainless steel, a mixture is used, most often consisting
of 6 parts of nitric acid, 47 parts of hydrochloric acid, 47 parts of water. In this
solution, steel products are boiled for 1-2 minutes. To avoid damage to the crown,
it is necessary to strictly observe the whitening regime. 3rd clinical stage. Checking
the quality of the stamped crown and the requirements for it. The quality of the
stamped crown made in the laboratory is carefully checked in the oral cavity. on a
prepared natural tooth, which is greatly facilitated by a preliminary assessment of
the artificial crown on a plaster stamp. The smooth, even surface of the crown
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indicates the high quality of the stamped prosthesis. Folds and dents on the metal
surface, poor coverage of the plaster die neck with the edge of the crown indicate
the poor quality of the stamping. The gap between the edge of the crown and the
plaster die will result in poor retention of the crown. Several crowns, Due to poor
quality stamping, easily removable from the plaster die, it is easy to confuse in the
oral cavity before checking. Checking the length of the crown. On a plaster die, the
edge of the crown should overlap the line of the clinical tooth neck to a minimum,
that is, by 0.3-0.5 mm. The issue is resolved individually according to clinical
indications:
● if the edge of the crown overlaps the clinical neck line more than is required
for the patient, the crown is carefully shortened with a silicon carbide stone or shaped
head.
● the crown, which turned out to be obviously short on the plaster die and the
prepared tooth, must be reworked.
The finished crown should have an anatomical shape with a well-defined equator to
match the tooth. The cutting edge and chewing surface are carefully stamped, their
relief corresponds to the patient's age. Checking the stamped crown on the prepared
tooth in the oral cavity. The artificial stamped crown is removed from the plaster
stamp, washed with hydrogen peroxide, disinfected with alcohol and placed on the
abutment tooth. If the crown is not applied, the quality of the tooth preparation must
be checked. The stump of a ground tooth, which is widened in comparison with the
perimeter of the neck, can interfere, requiring additional grinding of hard tooth
tissues and bringing the stump to the required shape. A narrow crown does not fit
over a properly prepared natural tooth due to:
1. inaccurate impression;
2. sloppy engraving of the neck of the tooth on a plaster working model;
3. narrowing the neck of a gypsum die or removing part of a low-melting alloy
when processing a metal die.
Regardless of the reason, the crown is sent to the dental laboratory for re-stamping.
The immersion depth is checked with an angled probe, feeling the edge of the crown
under the gum along the entire perimeter of the neck of the tooth. Carefully examine
the position of the edge of the crown in the area of the interdental gingival papillae.
Excessive immersion of the crown in this area is caused by:
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on the tissues of the marginal periodontium. The harmful effect of the crown, like
any other prosthesis, can be exacerbated by non-compliance with the manufacturing
rules.
1. Anatomical shape characteristic of this tooth. Correctly modeled cusps and
equator provide a normal relationship with the teeth of the opposite jaw and adjacent
ones. Crown Equator:
● creates interdental contacts that protect the interdental papilla;
● protects the vestibular and oral edges of the gums from food damage. One of
the main conditions for the existence of the dental arch is its continuity, restored by
interdental contacts.
2. Dense coverage of the neck of the tooth with immersion in the gingival pocket
by no more than 0.3-0.5 mm. A crown wider than the neck of the tooth irritates and
pushes the gum back, causing its atrophy - there is a gap between such a crown and
the tooth, which is initially filled with cement, but later saliva dissolves it and food
penetrates into the resulting gap, the decomposition products of which cause
necrosis of the tooth tissues, this is favored by a violation of the integrity enamel
during preparation. Deep immersion of the crown under the gum injures the marginal
periodontium.
3. An artificial crown should not increase the interalveolar height.If not, then
with central occlusion, the entire force of the contracting muscles falls only on the
tooth covered with a crown and its antagonists. Such occlusion will be traumatic,
manifesting itself in the form of pain in the tooth when it bites into its mobility.
4. When modeling the cusps of the chewing teeth, age characteristics should be
taken into account. In young people, the tubercles are well pronounced, in the
elderly, on the contrary, due to physiological wear, they are poorly represented and
for this reason the lateral movements of the lower jaw become smoother.
Fitting the crown The crown is placed on the tooth without much effort, gradually
bringing it to the gingival margin. With a tight coverage of the crown edge of the
neck of the tooth and compliance with the edge of the gum, it is advanced into the
gingival groove. After the probe, the depth of the crown edges is checked: in the
case of deep penetration into the pocket, corrections are made. The edges of the
crown cannot be bent wider than the neck of the tooth - the crown is re-stamped. A
narrow crown should not be shortened - this can make it inappropriate to the neck
of the tooth.It is also necessary to make sure that there are no premature contacts
with lateral occlusions, for which the patient is asked to move the lower jaw to the
right and left while maintaining the contact of the teeth - the crown should not
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interfere with the occlusal relationship during the movements of the lower jaw ...
The identification of possible premature contacts is based on the nature of the
occlusion of the posterior teeth prior to prosthetics.If the artificial crown breaks the
usual occlusion, it should be corrected or redone. Avoid the appearance of premature
contacts also in case of anterior occlusion. 3rd laboratory stage The crown is ground
with an elastic wheel, then a polishing paste is applied to a hard brush or felt (fixed
in a grinder), depending on the material of which the crown is made, and polishing
is completed with a soft thread brush "Fluff". The following are used as polishing
masses: iron oxide, or crocus (PerOz), Chromium oxide is a fine-grained green
powder. Pastes are widespread,
Composition, Rough Average Thin
%
Chromium 81 76 74
oxide
Silikogel 2 2 1.8
Split fat 5 5 5
Oleic acid - - 2
Bicarbonate - - 0.2
soda
Kerosene 2 2 2
4th clinical stage Before fixation, the crown is thoroughly treated with hydrogen
peroxide, degreased with alcohol and dried. The abutment tooth is covered with
cotton swabs and subjected to medication: it is cleaned of plaque, washed with
antiseptics, disinfected with alcohol, dried with ether or warm air. Liquid cement is
kneaded on a sterile glass plate. The rules for preparing the cement and its
consistency depend on the brand and the goal that needs to be achieved when
strengthening the crown. The prepared cement is applied to the crown with a spatula,
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filling it by about two-thirds. The inner walls are coated to the edge of the crown.
Narrow incisor crowns are filled with a trowel. The crown is placed on the tooth,
making sure that the cotton swabs do not fall under the edge of the crown. After the
cemented crown has been placed, the occlusal relationship for central occlusion
should be checked immediately. If the crown is in close contact with the antagonist
teeth, the patient is asked to keep the teeth closed for 10-15 minutes until the cement
hardens. When using restorative crowns, it is necessary to control their position on
the tooth. To do this, in the final phase of application, approximately 3-4 mm from
the edge of the crown to the gum, the patient is asked to close the teeth. When fixing
the stamped crown, one should not immediately check the nature of occlusal
contacts in lateral occlusions. This can cause a displacement of the crown and a
violation of the occlusion. Only after the cement has completely hardened is it
necessary to verify the accuracy of the restoration of the occlusal relationship. The
remains of the cement are carefully removed from the surface of the crown and
adjacent teeth. It is especially necessary to carefully remove the cement filling the
interdental space, the movement of the instrument should be directed from the gum
to the incisal edge or occlusal surface. Avoid using too much force that can cause
the crown to move. The remains of cement on the surface of the polished crown can
be easily removed with a cotton swab soaked in a liquid of phosphate-cement, only
after that it is necessary to rinse the mouth with a soda solution or a solution of
potassium permanganate. After removing the remains of cement, the patient is
advised not to eat for 1-2 hours until the fixing material has completely hardened.
Cements for permanent fixation of fixed dentures; Avoid using too much force that
can cause the crown to move. The remains of cement on the surface of the polished
crown can be easily removed with a cotton swab soaked in a liquid of phosphate-
cement, only after that it is necessary to rinse the mouth with a soda solution or a
solution of potassium permanganate. After removing the remains of cement, the
patient is advised not to eat for 1-2 hours until the fixing material has completely
hardened. Cements for permanent fixation of fixed dentures; Avoid using too much
force that can cause the crown to move. The remains of cement on the surface of the
polished crown can be easily removed with a cotton swab soaked in a liquid of
phosphate-cement, only after that it is necessary to rinse the mouth with a soda
solution or a solution of potassium permanganate. After removing the remains of
cement, the patient is advised not to eat for 1-2 hours until the fixing material has
completely hardened. Cements for permanent fixation of fixed dentures;
1. Zinc-phosphate cements: Unifas, Adgezor (Czech Republic), Phosphacap,
Poscal (Germany), Septocell (France).
2. Polycarboxylate cements: Carboco, Aqualox (Germany), Selfast (France).
3. Glass ionomer cement: "Meron", "Aqua Meron". KemPhil Superior, Base
Line (Germany), Ionocell (France), Fuji ionomer (Japan).
4. Polymer cements: "Resiment" (France), "Bifix", "Dualcement", "Vario-link",
"F-21" (Germany), etc.
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3. Educational technologies
- Lecture with multimedia visualization.
- Problem lecture.
1. Surgical dentistry head. edited by T.P. Osokin. Editors L.V. Levushkina, M.G.
Fomina, N.V. Kirsanov. Art editors S.L. Andreev, T.S. Tikhomirov. Technical
editor at NA. Birkin. Proofreader A.F. Matveychuk
Cocaine is an anesthetic that ushered in the era of modern local pain relief. However,
it is toxic and is currently practically not used in dental practice. Dikain is a white
crystalline powder, readily soluble in water and alcohol. Its solutions are sterilized
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by boiling. This is a strong local anesthetic used for surface anesthesia (tissue is
lubricated with a 0.25-2% solution). For adults, the highest single dose of dicaine is
0.09 g (3 ml of a 3% solution).
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When using a 0.25% solution of trimecaine, up to 800 ml, 0.5% - up to 400 ml, 1%
- up to 100 ml and 2% - up to 20 ml of anesthetic are administered. Lidocaine
(xycaine, xylocaine) is a white crystalline powder, highly soluble in water and
alcohol. For anesthesia, lidocaine hydrochloride is used. It is a strong anesthetic,
causes a deeper and more prolonged anesthesia than novocaine (up to 3-5 hours),
surpassing its analgesic effect by 2-3 times * Provides a good efficacy of anesthesia
in inflamed tissues. Allergic reactions are extremely rare. The toxicity of lidocaine
depends on the concentration of the solution: its 0.5% solution does not differ in
toxicity from novocaine, 1-2% solutions are 1.4-1.5 times more toxic than
novocaine. Contraindicated in cardiovascular failure, atrioventricular block II-III
degree, liver and kidney disease. For infiltration anesthesia during surgical
interventions on soft tissues, 0.25% and 0.5% solutions are used, and for conduction
anesthesia (and infiltration during operations on the alveolar ridge, other areas of the
upper and lower jaws) - 1-2% solutions. For application anesthesia, 1-2% aerosol of
lidocaine is used. Maximum doses of anesthetic: 0.15% solution - 1000 ml, 0.5% -
500 ml, 1-2% - no more than 50 ml. Bupivacaine (marcaine, carbostezin) is 6 times
more potent than novocaine in terms of its analgesic effect, but 7 times more toxic.
More effective than lidocaine. Anesthesia occurs in 4-10 minutes, reaching a
maximum in 15-35 minutes. The duration of anesthesia is 12-13 hours. In surgical
practice, 0.25%, 0.5% and 0.75% solutions are used. The maximum dose is 175 mg.
Articaine (ultracaine D-S, ultracaine D-S forte) is a local anesthetic of the amide
group, is available in cylindrical ampoules of 1.7 ml and in vials of 20 ml in the form
of a 4% solution. Less toxic than lidocaine, and only IV2 times higher than that of
novocaine. However, the analgesic effect of articaine is 5 times higher than that of
novocaine. The anesthetic has a high degree of protein binding and low fat solubility,
which is the basis for its choice in pregnant women (the least toxic to the fetus).
Ultracaine D-S contains adrenaline at a dilution of 1: 200,000, and ultracaine D-S
forte - 1: 100,000. The very low concentration of adrenaline in ultracaine D-S
determines its safety in persons with concomitant cardiovascular diseases, as well as
in pregnant women. Anesthesia occurs within 1-3 minutes after injection into the
tissue. The duration of action is 45-75 minutes. Contraindicated in paroxysmal
tachycardia, tachyarrhythmias, glaucoma.
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4 ml, pour 2 ml of a light pink solution of potassium permanganate (1:10 000). Then,
2 ml of the test solution is poured into one of the test tubes from a vial or syringe.
After 1-2 minutes, the solution of potassium permanganate from novocaine acquires
an orange or straw-yellow color. If the color of the solution has not changed, then
this indicates the presence in the vial or syringe of another substance, not novocaine.
TOOLS
NON-INJECTION ANALYSIS
Anesthesia with cooling. When tissues are cooled, the excitability of nerve receptors
decreases, and when frozen, the transmission of a nerve (pain) impulse stops.
Chlorethyl is used for pain relief.
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INJECTION ANALYSIS
For infiltration anesthesia during operations on the soft tissues of the face and in the
oral cavity, 0.25-1% solutions, novocaine are used, and for interventions on the
alveolar ridge or in the area of the jaw body
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The organs of the oral cavity receive innervation from the motor, sensory, autonomic
(sympathetic and parasympathetic) nerves. The sensory nerves that innervate the
skin of the face, soft tissues and organs of the oral cavity, jaw include the trigeminal,
glossopharyngeal, vagus nerves and branches extending from the cervical plexus
(large ear nerve and small occipital). In the area of the face along the branches of the
trigeminal nerve are located
The orbital nerve (n. Ophtalmicus) is sensitive, does not participate in the
innervation of the jaws and tissues of the oral cavity.
The maxillary nerve (n. Maxillaris) is sensitive, leaves the cranial cavity through a
round hole (foramen rotundum) into the pterygopalatine fossa (fossa
pterigopalatina), where it gives off a number of branches (Fig. 8).
The infraorbital nerve (n. Infraorbitalis) is a continuation of the maxillary nerve and
gets its name after the departure from the last zygomatic and pterygopalatine nerves.
From the pterygopalatine fossa, through the lower orbital fissure, it enters the orbit,
where it lies in the infraorbital sulcus (sulcus infraorbitalis) and through the
infraorbital fossa (foramen infraorbitalis) leaves the orbit, dividing into terminal
branches that form a small crow's feet (pes anserinus minor). The latter branches out
in the area of the skin and mucous membrane of the upper lip, lower eyelid,
infraorbital region, wing of the nose and the cutaneous part of the nasal septum.
In the pterygopalatine fossa, the posterior upper alveolar branches (rami alveolares
superiores posteriores) depart from the infraorbital nerve in
an amount from 4 to 8. A smaller part of them does not enter the thickness of the
bone tissue and spreads down the outer surface of the tubercle towards the alveolar
process. End up
Rice. 7. The structure of the trigeminal nerve (scheme
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56
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they are in the periosteum of the upper jaw, adjacent to the alveolar process, the
mucous membrane of the cheeks and gums from the vestibular side at the level of
large and small molars. Most of the posterior upper alveolar branches through the
foramina alveolaris posteriora penetrate into the canalis alveolaris, from which they
exit to the outer surface of the upper jaw and enter its bony tubules. These nerves
innervate the tubercle of the upper jaw, the mucous membrane of the maxillary sinus,
the upper large molars, the mucous membrane and the periosteum of the alveolar
process within these teeth. The posterior upper alveolar branches take part in the
formation of the posterior part of the upper dental plexus.
In the pterygopalatine fossa, less often in the posterior part of the infraorbital sulcus,
the middle superior alveolar branch departs from the infraorbital nerve
(ramus alveolaris superior medius). Sometimes the latter is represented by two
stems. It passes through the thickness of the anterior wall of the upper jaw and
branches into the alveolar process. This branch takes part in the formation of the
middle section of the upper dental plexus, has anastomoses with the anterior and
posterior upper alveolar branches, innervates the upper small molars, the mucous
membrane of the alveolar process and the gums from the vestibular side in the area
of these teeth.
In the anterior part of the infraorbital canal, the anterior superior alveolar branches
(rami alveolares superiores anteriores) - 1-3 stems depart from the infraorbital nerve.
These branches can, however, branch off from the infraorbital nerve along the entire
length of the infraorbital canal or sulcus, at the level of the infraorbital foramen and
even after the main trunk leaves it. The trunks of the anterior alveolar nerve can exit
in one canal (infraorbital)
with the infraorbital nerve or located in a separate bone canal. Passing through the
thickness of the anterior wall of the upper jaw medial to the middle upper alveolar
branch, the anterior upper
+
the alveolar branches take part in the formation of the anterior part of the upper
dental plexus. They innervate the incisors and canines, the mucous membrane and
periosteum of the alveolar process and the mucous membrane of the gums from the
vestibular side in the area of these teeth. From the anterior superior alveolar
branches, the nasal branch departs to the mucous membrane of the anterior fundus
of the nose, which anastomoses with the nasopalatine nerve.
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The posterior, middle and anterior upper alveolar branches, passing through the
thickness of the walls of the upper jaw, anastomosing with each other, form the upper
dental plexus - plexus dentalis superior. It anastomoses with the same plexus of the
other side. The plexus is located in the thickness of the alveolar process of the upper
jaw along its entire length above the tops of the roots of the teeth, as well as in the
upper parts of it in the immediate vicinity of the mucous membrane of the maxillary
sinus.
3. Educational technologies
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- Problem lecture.
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1. Surgical dentistry head. edited by T.P. Osokin. Editors L.V. Levushkina, M.G.
Fomina, N.V. Kirsanov. Art editors S.L. Andreev, T.S. Tikhomirov. Technical
editor at NA. Birkin. Proofreader A.F. Matveychuk
The most common indication for tooth extraction is the need to sanitize the oral
cavity in the presence of chronic periodontitis in the acute stage, when it is
impossible to eliminate the inflammatory focus at the apex of the tooth. Multi-rooted
teeth, which are the cause of odontogenic osteomyelitis, are also removed. The
dentist can remove teeth when fitting a removable denture, but there must be strict
indications for this.
So, the indications for tooth extraction are the following situations and groups of
diseases:
5. A tooth located in the area of the fracture of the alveolar process is removed, since
it interferes with the reposition of fragments or is a conductor of infection.
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7. A tooth is removed, which causes permanent trauma to the tongue and oral
mucosa, if this cannot be removed by grinding or prosthetics.
10. Lower macrognathia (in children, the rudiments of 8 | 8 teeth are removed to
delay the growth of the lower jaw).
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• Hemorrhagic diathesis.
• Acute infections.
• I, II, IX months of pregnancy due to the risk of miscarriage and premature birth.
• Menstruation.
• Stomatitis.
In order to reduce bacterial contamination, remove food debris, prevent the ingress
of dental plaque into the socket of the removed tooth, the following measures are
taken:
+
suggest the patient to rinse the mouth with an antiseptic solution: 100-150 ml of
potassium permanganate solution 1: 1000, furacilin 1: 5000.
Tooth Extraction Tools
To remove teeth, various types of forceps are used, the design of which takes into
account the anatomical features of the structure of a certain group of teeth (Fig. 109,
A, B). To remove teeth with a destroyed crown (roots), both forceps with narrow
converging cheeks and elevators are used.
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Rice. 109. Instruments for tooth extraction: A - forceps for removing teeth of the
upper jaw, B - forceps for removing teeth of the lower jaw, C - elevators for
removing roots of teeth
Extraction of teeth with forceps
The tooth extraction operation includes a number of preparatory and main stages.
Preparatory stages:
1. Giving the patient a position convenient for the operation of tooth extraction.
2. Selection of the optimal instrument (forceps) for the extraction of the target tooth.
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Rice. 114. Fixation with the left hand of the alveolar process of the upper jaw (A) of
the lower jaw (B) during tooth extraction
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5. Extraction of the tooth from the socket. After complete separation of the tooth
from the surrounding tissues, it is removed by a rotational or "eversion" movement.
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Rice. 118. Instruments recommended for the extraction of upper jaw teeth with
preserved coronal part
Rice. 119. Instruments recommended for the extraction of teeth of the upper jaw
with a destroyed coronal part (roots)
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Rice. 120. Instruments recommended for the extraction of teeth of the lower jaw
with preserved coronal part
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Rice. 121. Instruments recommended for the extraction of teeth of the lower jaw
with a destroyed coronal part (roots)
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The periodontium includes a complex of tissues that have a genetic and functional
commonality: periodontium, alveolar bone, gum with periosteum. Some authors
include in the periodontal complex and tooth tissue (cement, for example). But in
this case, the meaning of the word (para - about iodontos - tooth) changes. Thus, the
concept of "periodontium" is an artificially isolated structural element of the
masticatory system (periodontal tissues), by which N.K. Loginova [1995] suggests
understanding the interaction of various elements (teeth, jaw bones,
temporomandibular joints, chewing muscles, tongue , salivary glands), aimed at
achieving the final result - the formation of a food lump suitable for swallowing.
When reading this section in the literature, you may come across such terms (see
"Appendix 1 - Glossary of modern terms for characterizing the state of periodontal
disease in health and disease" - report of the WHO scientific group No. 621, Geneva,
1980), as:
1. alveolar ridge - the crown edge of the alveolar process, ending close to the
contours of the enamel-cement border;
2. connective tissue attachment - fibers of the connective tissue of the gums and
periodontal ligament, growing into the cement of the root;
3. periodontal junction - a bridge between the gum tissue and the part of the tooth
that is covered by the gum;
4. gum - the epithelial-connective tissue surrounding the tooth and the alveolar
ridge attached to them and extending to the mucogingival junction (i.e., to the
clinically distinguishable line between the gum and the alveolar ridge mucosa);
5. gingival margin - the line of the gingival tissue, which is the junction of the
gingival epithelium and the epithelium of the groove;
6. gingival groove (gingival gap) - a small groove between the tooth and most
of the gums, located between the epithelial attachment (the biological mechanism of
attachment of epithelial cells of the connective epithelium to the surface of the
tooth), i.e. the lower part of the groove and the edge of the gums. This is an
anatomical concept;
7. clinical gingival groove - the space that is created by the introduction of a
probe between a healthy or slightly inflamed gum and the surface of the tooth. It is
always deeper than the anatomical groove;
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8. interdental papilla - part of the gum tissue protruding above the crown, filling
the space between the contact surfaces of the tooth, both from the labial (buccal) and
lingual (palatal) sides.
We will get acquainted with the terms for characterizing diseases (diseases) of the
periodontium as we further elaborate on this problem. Periodontal functions:
1. barrier function, which is provided by:
● the ability of the gum epithelium to keratinization (with periodontal disease,
this function is impaired);
● a large number and features of the direction of bundles of collagen fibers;
● turgor of the gums;
● the state of mucopolysaccharides of connective tissue formations of the
periodontium;
● structural features of the gingival groove;
● the antibacterial function of saliva due to the presence in it of such biologically
active substances as lysozyme, inhibin, etc .;
2. trophic function is provided by a wide network of capillaries and nerve
endings;
3. function of reflex regulation of chewing pressure - irritation of numerous
nerve endings is transmitted through a wide variety of reflex highways;
4. plastic function consists in the constant reconstruction of tissues lost in the
course of physiological or pathological processes (osteoblasts, fibroblasts, etc.);
5. the shock-absorbing function is provided by the presence of collagen, elastic
fibers of the periodontium and the fluid contained in the vessels and tissues.
A great contribution to the development of the problem of periodontal diseases was
made by such domestic scientists as: A. I. Evdokimov, E. E. Platonov, N. F.
Danilevsky, G. D. Ovrutsky, G. N. Vishnyak, T. F. Vinogradova, T. I. Lemetskaya,
E. V. Borovsky, V. S. Ivanov , M. M. Tsarinsky, T. V. Nikitina, A. P. Bezrukova,
L. A. Dmitrieva, A. I. Grudyanov, A. P. Kankanyan, V. K. Leontiev and others.
At the Department of Therapeutic Dentistry of the Smolensk State Medical
Academy, the following worked and are working on this problem: L. M. Tsepov, V.
G. Morozov, E. V. Petrova, N. S. Levchenkova, S. N. Lozbenev, A. I. Nikolaev, L.
B. Turgeneva, A. P. Khromchenkov, E. N. Zhazhkov, N. N. Usoltseva. They
defended dissertations, prepared and published monographs, reference manuals,
guidelines, developments, letters, received patents for inventions.
There is a lot of data on the systematization and classification of periodontal
diseases. The systematization of diseases in general and periodontal diseases in
particular provides for the assignment of certain nosological forms of pathology to
the main (typical) pathological processes - inflammation, dystrophy, tumors and
tumor-like lesions. As for the classifications, some of them are of historical interest,
some are set out in textbooks on therapeutic dentistry. It is necessary to dwell in
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more detail on the domestic classification adopted in November 1983 by the 16th
Plenum of the Board of VNOS in Yerevan. This classification is still used in Russia.
This classification provides for the allocation of inflammatory (gingivitis,
periodontitis), dystrophic (periodontal disease), idiopathic and tumor (tumor-like) in
their morphological and clinical essence of periodontal lesions.
1. Gingivitis is an inflammation of the gums caused by the unfavorable effect of
general and local factors and proceeding without violating the integrity of the
gingival attachment. Forms: catarrhal, hypertrophic (more correctly, hyperplastic),
ulcerative. Current: acute, chronic, aggravated, remission. Prevalence: localized,
generalized. : light, medium, heavy.
2. Periodontitis is an inflammation of the periodontal tissues, characterized by
progressive destruction of the periodontium and bone. As can be seen from the
definition given by the authors of the classification to this type of pathology, for
some reason an indication of the cause of its occurrence "dropped out" from it. Only
a characteristic of the pathological process is given. Course: acute, chronic,
exacerbated (including abscess), remission. Prevalence: localized, generalized.
Severity: light, medium, severe.
3. Periodontal disease is a periodontal dystrophic lesion. Here, as you can see,
there is generally no reference to the cause of the onset of the disease, and its
characteristics (except for indicating the morphological essence of the process).
Course: chronic, remission. Prevalence: generalized (it was easier to indicate this,
giving the definition of the disease). Severity: mild. , medium, heavy.
4. Idiopathic periodontal diseases with progressive lysis of periodontal tissues
(Papillon-Lefebvre syndrome, histiocytosis X, akatalasia, neutropenia,
agammaglobulinemia, uncompensated diabetes mellitus). We share the point of
view of TI Lemetskaya about the inexpediency of introducing the concept of
"idiopathic diseases", since this removes responsibility from the dentist for
clarifying the general disease that caused periodontolysis. The term "idiopathic"
implies an unclear etiology. And here not everything agrees with the logic, since the
etiology, for example, of diabetes mellitus is known.
5. Periodontomas are tumor and tumor-like processes in the periodontium.
Thus, the main categories used by the authors of this classification when
systematizing periodontal diseases were: the clinical form of the disease, indicating
the morphological nature of the lesion, its prevalence, severity and staging in this
form. At the same time, there is no consistency in its compilation: with a more than
detailed presentation of data concerning many aspects of gingivitis, the characteristic
of group 5 (periodontal disease) states the well-known fact that "periodontomas" are
tumor and tumor-like processes in the periodontium.
This classification, unfortunately, does not provide for any other forms of
periodontal pathology (for example, rapidly progressive periodontitis, other
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to the root of the tooth, the presence of supra- and subgingival dental deposits, the
level of resorption of the alveolar process according to the radiograph.
Diagnosis of periodontal disease, as a rule, is still carried out on the basis of
anamnesis and examination. Most of the periodontal indices proposed for assessing
the state of the periodontium are not standardized, therefore, the opinion of a number
of authors about them is different, but in practice, dentists show a reasonable
restrained attitude towards them. In addition, the assessment of the hygienic state of
the oral cavity and the severity of an already developed pathological process helps
little to diagnostics.+
The purpose of the diagnostic process is not to detect a disease in general, but to
identify a very specific disease. The doctor judges the disease on the basis of its
symptoms, thinks in nosological categories.
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1. Chronic
inflammation of the 1.
gums, suppuration Schiller-Pisarev test
from periodontal 2. Investigation of the parameters of a cheap liquid
pockets (DC)
3. Benzidine test
4. Bacteriological examination of the contents of
periodontal pockets (PC)
5. Gingival and PC thermometry
6. Study of water fractions in the oral fluid (OR)
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2. The presence of
periodontal pockets 1.
PC Depth Measurement
2. X-ray of alveolar processes and teeth with filling
of the PC with contrast agents
3. Supra- and
subgingival dental 1.
deposits Staining "dental" plaque
4. Resorption of bone
tissue of the alveolar 1.
bone Dental radiography
2. Panoramic X-ray
3. Orthopantomography
4. Densitometric analysis of radiographs
5. Echoosteometry
6. Determination of concentration in blood of
alkaline phosphatase, calcium ions, citric acid,
hydroxyproline
5. Mobility of teeth,
violation of occlusion 1.
Determination of the degree of tooth mobility
2. Revealing premature teeth contacts using
occludograms
3. Revealing the functional overload of teeth by the
analysis of odontoparodontograms
6. Disturbances in the
periodontal 1.
microvasculature, Biomicroscopy (vital microscopy) of the gums
changes in tissue 2. Rheoparodontography
metabolism 3. Photoplethysmography
4. Kulazhenko test
5. Rotger's test
6. Determination of tissue oxygen balance
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7. Changes in local
immunological 1.
reactivity and Determination of protective factors (lysozyme,
resistance of immunoglobulins, etc.)
periodontal tissues 2. Exfoliative cytology
3. Yasinovsky test
4. Test Kavetsky-Bazarnova
5. McClure-Aldrich test
8. Hyperesthesia of the
necks of the teeth 1.
Samples with mechanical, thermal and chemical irritants
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● research methods
● diagnostics
● treatment methods
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8. Venue
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Topic 2. Pulpitis
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periapical tissues.
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− Classification
− Pathogenesis
− Clinic
− Diagnostics
− Treatment
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4. Literature:
● Orthopedi
c dentistry, Abolmasov N.G., Abolmasov N.N.
● Therapeutic dentistry Borovsky
● Orthopedic dentistry Lebedenko, Artyusov
● Orthopedic dentistry Trezubov, Shcherbakov
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● Dental laboratory.
● Dental technician workplace.
● Equipment and tools for the manufacture of dentures.
● Dentures. Types of dentures: removable and non-removable.
● Materials for the manufacture of dentures: constructional and auxiliary.
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3. Educational technologies
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3. Surgical dentistry head. edited by T.P. Osokin. Editors L.V. Levushkina, M.G.
Fomina, N.V. Kirsanov. Art editors S.L. Andreev, T.S. Tikhomirov. Technical
editor at NA. Birkin. Proofreader A.F. Matveychuk
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3. Surgical dentistry head. edited by T.P. Osokin. Editors L.V. Levushkina, M.G.
Fomina, N.V. Kirsanov. Art editors S.L. Andreev, T.S. Tikhomirov. Technical
editor at NA. Birkin. Proofreader A.F. Matveychuk
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● Periodontal functions.
● Age-related periodontal changes.
● X-ray characteristics of the periodontium are normal
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4. Glossary
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of operation is to apply a special powder under pressure to the tooth, which gently
acts on hard tissues.
A cyst is a non-tumor formation with a cavity that is filled with cystic fluid. Most
often it develops as a result of chronic inflammation around the apex of the tooth
root.
Flap surgery is a surgical procedure to remove inflamed tissue deep under the gum
or calculus.
Non-carious lesions of teeth - pathological abrasion of teeth, malformations of hard
tissues of teeth and root surface. They arise as a result of heavy loads, endocrine
diseases, disturbances in the position of adjacent teeth, as a result of long-term use
of hard toothbrushes and poor-quality dental care. Manifested in the form of defects
on the teeth and high sensitivity to chemical and temperature influences.
Intolerance to base plastics is a complication after the placement of removable
dentures, accompanied by unpleasant sensations in the gum area. It manifests itself
in the form of: burning, pain, inflammation of the mucous membrane at the point of
contact with a removable denture, gag reflex.
Implant failure - rejection of a metal rod by the tissues of the oral cavity, which is
accompanied by an inflammatory process of soft or bone tissues.
Osteomyelitis is an inflammation of the jaw bone tissue.
Operation - tissue dissection, excision or stitching.
Operative periodontics is a set of surgical methods that allow local or complete
restoration of the gum bone tissue and form its correct and beautiful contour.
Orthodontics is a large branch of dentistry that specializes in the prevention,
diagnosis and treatment of dentoalveolar deformities and malocclusion.
Filling polishing - removal of roughness and overhanging edges of the filling
material from the tooth using special tools.
Full bracket system - a bracket system that connects all the teeth of the upper and
lower jaws.
A complete removable denture is a denture consisting of artificial teeth located on a
plate made of plastic to match the color of the gums. It is used in the complete
absence of teeth in the upper or lower jaws.
Conduction anesthesia is a modern method of pain relief, in which an anesthetic is
supplied to the nerve fiber of the branches of the trigeminal nerve and blocks it. The
advantage is that pain sensitivity is turned off over a large area of the jaw and
adjacent soft tissues.
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