Professional Documents
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CHILDREN
DEFINITION
Special Health Care Needs
AAPD (2013) -“any physical, developmental, mental, sensory, behavioral,
cognitive, or emotional impairment or limiting condition that requires medical
management, health care intervention, and/or use of specialized services or
programs. The condition may be congenital, developmental, or acquired
through disease, trauma, or environmental cause and may impose limitations
in performing daily self-maintenance activities or substantial limitations in a
major life activity. Health care for individuals with special needs requires
specialized knowledge, as well as increased awareness and attention,
adaptation, and accommodative measures beyond what are considered
routine.” 2
DENTAL HANDICAP-
AAPD (1996) states: a person should be considered dentally
differently-abled if pain, infection, or lack of functional
dentition affects following :
1. Restricts consumption of a diet adequate to support
growth and energy needs.
2. Delays or alters growth and development.
3. Inhibits performance of any major life activity including
work, learning, communication and recreation.
ORAL MANIFESTATIONS
• Advanced cases of Early Childhood Caries because of poor oral
hygiene and cariogenic diet patterns.
• Present with multiple anomalies of facial structures also eruption
time, sequence, number, presence of malocclusion and enamel
hypoplasia.
• Also higher prevalence of periodontal disease 11
Treatment Considerations
The dentist must first asses the child’s mental level so as to gauge the level of
cooperation to be expected and make adjustments accordingly -
1. Short attention span, restlessness, hyperactivity and erratic emotional
behavior characterize MR patients undergoing dental treatment.
2. Familiarize MR patients to the dental office and dental staff so as to reduce
his /her fear before undertaking any dental treatment.
3. Allow the patient to bring one favorite item into the clinic.
4. Speech must be slow, simple.
5. Only one instruction at a time should be given.
Treatment Considerations
6. Reward patient with compliments after the procedure is completed.
7. Carefully listen to these patients as they have difficulty in
communicating.
8. Appointments should be short and scheduled during the early part of
the day.
9. Children should be managed with a blend of kindness and firmness.
10. Permanent restorations and preventive procedures with regular visits.
11. TSD and TLC approach in mild cases whereas sedation in moderate
cases.
12. GA is advised in severe cases where any level of cooperation cannot
be achieved.
2. DOWN’S SYNDROME
Described by John Langdon Down (1865)
One of the most recognizable malformation syndromes.
Occurs due to Trisomy of chromosome 21(95%),
translocation (4%) and mosaicism (1%) .
Incidence/ prevalence –
1 in every 600 live births.
Prenatally diagnosed (from 3% to 60%) in younger patients.
Birth prevalence has declined from 1986 to 2004 (study
done in Australia).
Predisposing Factors
Mother’s age –
If 20 then 1 in 2300
If 34-39 then 1 in 280
If 45-50 then 1 in 45
Risk of Down’s syndrome per live birth
Advanced maternal age, with advanced maternal age
Chromosomal aberrations.
General Manifestations
1. SKULL-
1. Brachycephalic resulting in flattened face and occiput.
Lack of supraorbital ridges.
2. Presence at birth of third fontanelle
3. Flat nasal bridges with a small maxilla.
4. Obstructed and narrower passage- mouth breather
2. EARS-
1. Dysplastic ears with abnormal pinna.
3. NECK-
1. Short and broad with excess skin posteriorly.
1. EYES-
1. Oblique palpebral fissures with
prominent epicanthic folds (mongloid
features)
2. Brushfield’s spots appear on the iris in
a ring like concentric with the pupil.
3. Scanty eyelids
4. Cataract, squint, nystagmus.
HANDS-
Broad and short with a single transverse
palmar crease (simian crease)
Short incurved little finger
IQ- 25-50
Associated congenital abnormalities-
Cong. heart diseases (ASD, VSD, AVC)
Duodenal atresia
Atlantoaxial instability
Umbilical hernia
Multiple immunological defects affecting the skin, GIT and
respiratory tracts.
Acute lymphoblastic leukemia is 20 times more common.
Hypothyroidism
Alzheimer’s disease
Oral Manifestations
1. MOUTH-
1. Small drooping mouth.
2. Open mouth posture
2. TONGUE-
1. Protrusive, Fissured tongue
2. Circumvallate papillae are enlarged but filiform papillae
are absent.
3. Macroglossia
3. LIPS-
1. Thick, dry, fissured.
Oral Manifestations
4. OCCLUSION-
1. Anterior open bite and crossbite, class III tendency
2. Small maxilla
5. PALATE-
1. High with horizontal palatal shelves
2. Bifid Uvula, cleft lip and cleft palate
6. ERUPTION-
Retarded
Early shedding of deciduous teeth
Oral Manifestations
7. TEETH-
Hypodontia, especially third molars
Microdontia
Hypocalcification and hypoplastic defects
Low incidence of caries
8. PERIODONTIUM-
Severe, early onset of periodontal disease due to local factors like
poor oral hygiene, tooth morphology, and malocclusions and systemic
factors like decreased humoral response reduced chemotaxis,
impaired phagocytosis, poor circulation etc.
Dental Treatment
• These children are MR (10%) and require appropriate treatment.
• Incidence of cardiac disease associated with DS is 40% and will require
adequate prophylaxis
• Increased incidence of Leukemia and acute and chronic infections of the URTI
can also alter treatment.
• Children are usually affectionate & cooperative. Do not present problems
during management.
• N2O analgesia or TSD in mildly apprehensive patients can be used; GA in
severe cases.
• Preventive procedures along with CHX may be beneficial.
• Pulp treatment in deciduous teeth is contraindicated in cardiac patients
because of the risk of bacteremia, whereas it can be considered with an
adequate apical seal.
3. CEREBRAL PALSY
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Dental Problems
DENTAL CARIES-
Higher caries rate then normal children due to their inability to
maintain good oral hygiene, or due to parents tendency to overindulge
Soft cariogenic diet, Due to increased prevalence of Enamel
hypoplastic defects on their teeth.
PERIODONTAL DISEASE-
Occurs with a great frequency as the patient is unable to brush or
floss.
Also on Phenytoin to control seizure activity which is a cause of some
degree of gingival hyperplasia.
MALOCCLUSIONS-
occurs twice as often.
Protrusions of maxillary anterior teeth, excessive overjet and overbite,
open bites, and unilateral cross bites.
Also Class II Div 2 maloccusion is observed.
BRUXISM-
Atheoid cerebral palsy resulting in severe attrition, loss of vertical
dimension and TMJ.
TRISMUS-
Due to susceptibility to trauma.
Excessive drooling and difficulty in swallowing.
Dental Treatment
• Through medical and dental history should be taken.
• Maintain a calm friendly and professional atmosphere with patient.
• Many patients prefer to be treated in wheelchair which can be
tipped back into dentist’s lap.
AUTISM
CDC DSM-IV
ASPERGER’S
AUTISTIC
SYNDROME PERVASIVE RETT SYNDROME CHILDHOOD
DISORDER
DEVELOPMENTAL DISINTEGRATIVE
DISORDER, NOT DISORDER
OTHERWISE
SPECIFIED(PDD-NOS)
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Manifestations
Clinical:
• Inability to read and comprehend the feelings.
• Delayed language development, echolalia (repeating words).
• Repetitive motions, strict adherence to routines, attachment to
unusual objects.
• Self injurious behaviours.
Dental Findings
• Higher susceptible to caries.
• Bruxism
• Non- nutritive chewing
• Erosion
• Damaging oral habits such as tongue thrusting, lip biting.
• Gingivitis and poor oral hygiene
• Texture sensitivity - it leads to consumption of refined and high sugar diet. 43
TREATMENT
• Allow autistic child to bring comfort items such as toys.
• Make a first appointment short and positive.
• Approach the child in quiet, non-threatening manner.
• Avoid loud noises.
• Invite child to sit alone in dental chair to become familiar with
treatment setting.
• Talk in direct , short phrases . Talk calmly.
• Behaviour modifications technique that is use of positive
reinforcement to promote desirable behaviour.
• Some autistic child can be calm by moderate pressure by using a
papoose board to wrap the child.
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