10 YEARS CHAPTERWISE QUESTION PAPERS

2nd PROFESSIONAL MBBS

2010 – 2020

PATHOLOGY
Compiled by: Apurva Himmatsingka
Edited by: Soumyadeb Roy
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 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

GENERAL PAT HOLOGY

Group-B (COMMENT ON)
1. Pathogenesis of Septic Shock (2021 P1)
2. Difference between apoptosis and necrosis. (2021 P1)
3. Apoptosis differs from necrosis. (2020 P1)
4. Dystrophic calcification and metastatic calcification are synonymous. (2020 P1)
5. Physiological and pathological giant cells are different. (2019 P1)
6. Cellular changes in irreversible injury. (2018 P1)
7. Metaplasia is not same as neoplasia. (2018 P1)
8. Granuloma and granulation tissue. (2017 P1)
9. Hyperplasia and Hypertrophy are different. (2017 P1)
10. Direct spread is different from metastasis. (2016 P1)
11. Role of complements in acute inflammation. (2016 P1)
12. Thrombus differs from clot. (2015 P1)
13. Inflammation is beneficial. (2015 P1)
14. Healing by primary infection versus healing by secondary infection. (2014 P1)
15. Teratoma and hamartoma are two different lesions. (2014 P1)
16. Dystrophic versus metastatic calcification. (2014 P1)
17. Phlebothrombosis and thrombophlebitis are not same. (2013 P1)
18. Importance of paraneoplastic syndrome. (2013 P1)
19. Transudate and exudates indicate different clinical conditions. (2012 P1)
20. Presence of chronic inflammatory cells does not always indicate chronic
inflammation; it has otherhallmarks too. (2012 P1)
21. Routes of spread of malignant tumours are different. (2012 P2)
22. Necrosis differs from apoptosis. (2011 P1)
23. Amyloidosis is the result of long standing chronic diseases. (2011 P1)
24. Tobacco smoking and diseases. (2010 P1)
25. Grading and staging of malignant tumours and its relevance. (2010 P1)

Group-C (ANSWER THE FOLLOWING)

1. Trisomy 21 (2021 P1)
2. Differentiate between healing by primary intention and secondary intention. (2020
P1)
3. Tobacco related human health hazards. (2020 P1)
4. Philadelphia chromosome. (2019 P1)
5. Different between primary and secondary amyloidosis. (2018 P1)
6. Difference between dystrophic calcification and metastatic calcification. (2018 P1)
7. Difference between necrosis and apoptosis. (2007 P1)
8. Healing by primary union and secondary union differ in process of wound healing.
(2016 P1)
9. Pathogenesis of oedema in cardiac disease. (2016 P1)
10. Difference between coagulative necrosis and liquefactive necrosis. (2015 P1)
11. Pathogenesis of thrombosis. (2014 P1)
12. Carcinoma is a multistep process. (2014 P1)
13. Difference between carcinoma and sarcoma. (2013 P1)
14. Pathogenesis of amyloidosis. (2013 P1)
15. Pathogenesis of renal oedema. (2012 P1)
16. Cause of unconjugated hyperbilirubinemia. (2012 P1)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

17. Antibody dependent cytotoxic cell. (2011 P1)

18. Difference between carcinoma and sarcoma. (2011 P1)
19. Arterial and venous thrombosis. (2010 P1)
20. Transudate and exudates. (2010 P1)
21. Type III hypersensitivity. (2010 P1)

Group- D (SHORT NOTES)

1. Granuloma (2021 P1)
2. Tumour Markers. (2021 P1)
3. Type 2 hypersensitivity (2021 P1)
4. Karyotyping. (2020 P1)
5. Fate of a thrombus. (2020 P1)
6. Type I hypersensitivity reaction. (2019 P1)
7. BCR-ABL fusion gene. (2019 P1)
8. Vascular change in acute inflammation. (2018 P1)
9. Chemical carcinogens (2018 P1)
10. Klinefelter syndrome (2018 P1
11. Tobacco related human health hazard. (2018 P1)
12. Langhan’s Giant cell. (2017 P1)
13. Philadelphia chromosome. (2017 P1)
14. Turner syndrome. (2017 P1)
15. Septic Shock. (2017 P1)
16. Radiation injury. (2016 P1)
17. Hypovolemic shock. (2016 P1)
18. Transcoelomic space. (2015 P1)
19. Fine Needle Aspiration Cytology. (2015 P1)
20. Conjugated hyperbilirubinemia. (2015 P1)
21. Klinfelter syndrome. (2014 P1)
22. Decompression sickness. (2014 P1)
23. Radiation injury. (2014 P1)
24. Gaucher’s disease. (2013 P1)
25. Turner syndrome. (2013 P1)
26. Karyotyping. (2013 P1)
27. Difference between hypertrophy and hyperplasia. (2012 P1)
28. Coagulation necrosis. (2012 P1)
29. Carcinoma in situ. (2012 P2)
30. Down’s syndrome. (2011 P1)
31. Carcinoma in-situ. (2011 P1)
32. Giant cell. (2011 P1)
33. Dystrophic calcification. (2010 P1)
34. Apoptosis. (2010 P1)
35. X linked disease. (2010 P1)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

HAEMAT OLOGY
Group-A (LONG QUESTIONS)
1. A 5 year old boy presented with hepatosplenomegaly, stunted growth, Mongolian face
and severe pallor of chronic onset. (2021 P1)
i. What is provisional diagnosis?
ii. Laboratory investigation of the case.
iii. Pathogenesis of anaemia in such case
iv. Complications of such illness.

2. A 10 year old male child presented with massive swelling at knee joint following minor
trauma. His maternaluncle also had similar episodes in the past and subsequently died
of severe GIT bleeding. 2+5+3 (2020 P1)
a) What is your probable diagnosis?
b) What investigations are to be done to confirm the diagnosis?
c) Is it likely that his sister is also affected?

3. A 30 years old lady presented with haemorrhagic spots in the skin & mucosa, onset is
insidious. Thereis no organomegaly or lymphadenopathy. 2+5+3 (2019 P1)
a) What is your provisional diagnosis?
b) How will you proceed for diagnosis?
c) What is the basic mechanism of this disease?

4. 7 years old girl presented with sudden onset of fever, week ness severe
pallor, generalizedlymphadenopathy, hepatomegaly, sternal tenderness
and gum bleeding. 2+6+2 (2018P1)
i. What is provisional diagnosis?
ii. How will you proceed to confirm the diagnosis?
iii. What are the important prognostic factors in this case?
5. A 60 year old male patient with Low back pain and anaemia, X-ray reveals multiple
osteolytic lesion.
a. What is your provisional diagnosis? 2+8 (2017 P1)
b. How will you processed to confirm the diagnosis?

6. A 45-year-old male with history of partial gastrectomy frequently has anaemia and
neurological symptoms.
a. What is provisional diagnosis? 2+5+3 (2016 P1)
b. How you will proceed to investigation so as to establish your diagnosis?
c. Discuss the pathogenesis of this anaemia.

7. A 7year old male patient presented with fever, pallor, gum bleeding,
lymphadenopathy. Peripheral blood,smear revealed fair number of abnormal
lymphocytes. 2+6+2 (2015P1)
i. What is your provisional diagnosis?
ii. How will you diagnose the case in laboratory?
iii. Enumerate four prognostic factor.

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

8. A 4 year old female patient presented with severe pallor and splenomegaly with history
of multiple blood transfusion. Biochemical investigation revealed unconjugated
hyperbilirubinemia. 2+5+3 (2014 P1)
i. What is your provisional diagnosis?
ii. What is the examination you have done in lab?
iii. What is the basic genetic defect?

9. A male child of 5 years age presented with epistaxis and petechiae following fever 2
weeks back. 1+5+4(2013 P1)
i. What is your provisional diagnosis?
ii. What laboratory investigations are to be preform to establish your
diagnosis?
iii. Explain in short the pathogenesis of the condition.

10. A 60-year-old male presents with low back pain and anaemia-ray reveals multiple
osteolytic lesions. 2+8 (2011 P1)
i. What is your provisional diagnosis?
ii. How you will proceed to confirm your diagnosis?

11. A 10year old boy presented with pallor and splenomegaly. List the differential
diagnosis. Enumerate theinvestigations necessary to evaluate this case if a haemolytic
anaemia is suspected. 2+8 (2010 P1)

Group-B (COMMENTON)
1. Importance of Cross matching of Blood. (2021 P1)
2. Rational use of blood. (2020 P1)
3. Significance of reticulocyte count. (2019 P1)
4. Hyperaemia and congestion. (2019 P1)
5. Blood comportment therapy is beneficial than whole blood transfusion. (2018 P1)
6. Presence of spherocytosis in not pathognomonic of hereditary spherocytosis. (2017
P1)
7. Leukamoid reaction and leukaemia are different. (2016 P1)
8. Significance of peripheral blood smear. (2015 P1)
9. Transfusion related diseases can be avoided. (2013 P1)
10. Significance of peripheral blood smear. (2012 P1)
11. Presence of spherocytosis in not pathognomonic of hereditary spherocytosis. (2011
P1)
12. FAB classification of acute leukaemia and basis. (2010 P1)

Group-C (ANSWER THE FOLLOWING)

1. Role of protein and urine electrophoresis in multiple myeloma. (2021 P1)
2. Causes of thrombocytopenia (2021 P1)
3. Thrombophlebitis & Phlebothrombosis. (2019 P1)
4. Reverse blood grouping. (2019 P1)
5. Laboratory diagnosis of beta-thalassemia (2018 P1)
6. Blood picture of Iron deficiency anaemia and biochemical findings. (2017 P1)
7. Principal and utility of Coomb’s test. (2017 P1)
8. Lab diagnosis of multiple myeloma. (2016 P1)
9. Pathogenesis of β thalassemia. (2015 P1)
10. Diagnostic criteria of blastic phase of chronic myeloid leukaemia. (2014 P1)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

11. Peripheral blood picture in haemolytic anaemia. (2013 P1)

12. Peripheral blood and bone marrow picture of megaloblastic anaemia. (2012 P1)
13. Pathogenesis of Disseminated Intravascular Coagulation. (2011 P1)

Group- D (SHORT NOTES)

1. Peripheral blood picture of CLL (2021 P1)
2. Peripheral blood picture of CML(2020 P1)
3. PCV(2020 P1)
4. Hodgkins Lymphoma (2019 P1)
5. Haemorrhagic infarct (2019 P1)
6. ITP (2019 P1)
7. Peripheral Picture od CML (2016 P1)
8. Significance of reverse blood grouping (2016 P1)
9. Reed Sternberg Cells (2017 P1)
10. Transfusion Reaction (2015 P1)
11. Tubercular Lymphadenitis. (2015 P2)
12. Coomb’s test. (2014 P1)
13. Leukamoid reaction. (2013 P1
14. Reed Sternberg cell. (2013 P2)
15. Haemophilia (2012 P1)
16. Rh Incompatibility (2012 P1)
17. Fresh frozen plasma (2011 P1)

SYSTEMIC PATHOLOGY
CVS
Group-A (LONG QUESTIONS)
1. A 52 years old hypertensive developed acute precordial pain with sweating and
breathlessness. (2018 P2)
a) What could be the privational diagnosis? 2+6+2
b) What investigation will help to establish the
diagnosis?
c) What may be the complications in this case?
2.A 59 years male presented with acute pain on the left anterior chest wall, severe
dysopnia, profusesweating, and rapid thread pulse. 2+5+3 (2015 P2)
a) What is your provisional diagnosis?
b) Enumerate risk factors and pathogenesis?
c) Enumerate important diagnostic biochemical markers?
3. 70 years old man was brought to hospital emergency room at dawn with severe
precordial pain, profuse sweating and respiratory distress. On examination radial pulse was
not palpable. BP 80/? mm of Hg. 2+6+2 (2013 P2)
a) What is your Provisional diagnosis?
b) How do You Proceed to investigate the case in the laboratory?
c) What are the complications?
4. A 45 year old hypertensive developed acute precordial chest pain. 2+4+4 (2010 P2)
a) What are the possible diagnoses?
b) Describe the various laboratory findings in such a case?
c) What are the gross and microscopic changes that you would expect to find in the heart?

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

Group-B (COMMENTON)
1. Large joint pain can be a clinical manifestation of cardiac disease. (2019 P2)
2. Pathogenesis of atherosclerosis integrate risk factors and response to injury. (2018 P2)
3. Rheumatism licks the joint but bites the whole heart. (2014 P2)
4. Pancarditis in rheumatic heart disease. (2011 P2)

Group-C (ANSWER THE FOLLOWING)

1. Lab investigation of Myocardial Infarction. (2021 P2)
2. Pathogenesis and pathology of Rheumatic heart disease. (2020 P2)
3. Lesions in the heart due to acute rheumatic fever. (2016 P2)
4. Investigations of myocardial infarction. (2012 P2)

Group- D (SHORT NOTES)

1. Capillary Haemangioma. (2020 P2)
2. Complications of atheroma. (2016 P2)

KIDNEY
Group-A (LONG QUESTIONS)
1.A 60 year old male presented with gradual weight loss, heaviness in loin, mild fever and
microscopichaematuria on routine examination. 2+4+4 (2020 P2)
a) What is your provisional diagnosis?
b) How do you proceed to investigate to confirm the diagnosis?
c) Describe the gross and microscopical features of the lesion.
2.A 6-year-old boy developed puffiness of face, oliguria and mild hypertension about
two weeks afterrecovery from sore throat. 1+6+3 (2019 P2)
a) What is your provisional diagnosis ?
b) Which laboratory investigation are to be done to confirm the diagnosis?
c) Describe the pathogenesis of the condition.
3.A five years old boy developed puffiness of face, oliguria and mild hypertension
two weeks after an attack of sore throat . (2019 P1)
a) What is your provisional diagnosis?
b) What laboratory investigations and other investigations need to be done to confirm the
diagnosis?
c) Mention fates of this condition?
4. A 10 years old child is admitted with puffiness of face, oliguria and smoky urine. 2+6+2
(2014 P2)
a) What is your provisional diagnosis?
b) Describe the etiopathogenesis and morphological features observed in the target organ?
c) Which investigations should be done to reach the diagnosis?
5.A seven year old boy abruptly developed puffiness of face, oliguria and oedema about
two weeks afterrecovery from sore throat. 2+8 (2012 P2)
a) What is your provisional diagnosis ?
b) Which laboratory and other investigation to be done to establish the diagnosis ?

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

Group-C (ANSWER THE FOLLOWING)

1. Classification of glomerulonephritis. (2021 P2)
2. Rapidly progressive glomerulonephritis (RPGN). (2013 P2)
3. Diabetic Nephropathy. (2011 P2)
4. Renal function tests in chronic renal failure. (2010 P2)

Group- D (SHORT NOTES)

1. Polycystic kidney disease. (2018 P2)
2. Rapidly progressive glomerulonephritis. (2017 P2)
3. Renal cell carcinoma. (2015 P2)

NOTES

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

GASTROINTESTINAL TRACT
Group-B (COMMENT ON)
1. Ulcerative lesion of the stomach is not always benign. (2020 P2)
2. Ulcerative colitis and crohns disease differs in many aspects. (2018 P2)
3. Etiology of peptic ulcer and gastric carcinoma is same. (2015 P2)
4. Ulcerative colitis and crohn’s disease are different macroscopically and
microscopically. (2015 P2)
5. Carcinoma colon is a genetic disorder. (2014 P2)
6. Colorectal carcinoma. (2010 P2)

Group-C (ANSWER THE FOLLOWING)

1. Pathogenesis of peptic ulcer. (2018 P2)
2. Familial adenomatous polyposis. (2015 P2)
3. Macroscopic difference between peptic ulcers of stomach with ulcerative type of
gastric carcinoma.
(2013 P2)
Group- D (SHORT NOTES)
1. Pleomorphic salivary adenoma. (2021 P2)
2. Peptic Ulcer disease. (2021 P2)
3. Pleomorphic salivary adenoma. (2019 P2)
4. Barret’s oesophagus. (2017 P2)
5. Acute appendicitis and its fate and complications. (2014 P2)
6. Pleomorphic salivary adenoma. (2014 P2)
7. Crohn’s disease. (2013 P2)
8. H pylori and gastric disease. (2010 P2)

LIVER
Group-A (LONG QUESTIONS)
1. A 60 year old male developed deep jaundice, clay colored stool, weight loss, since
two months and dull aching pain in and around epigastric region. (2021 P1)
a) What is the possible differential diagnosis?
b) What should be the approach to investigate the patient to arrive at diagnosis?
c) Enumerate the various laboratory finding in the blood and Urine.

Group-B (COMMENTON)
1. A small percentage of hepatitis B infections lead to hepatocellular carcinoma. (2016
P2)
2. Alcoholism is not prerequisite for cirrhosis. (2014 P2)
3. Viral hepatitis leading to cirrhosis. (2013 P2)]
4. Serological markers of HBV induced infective hepatitis. (2011 P2)
5. Alcoholic cirrhosis. (2010 P2)

Group-C (ANSWER THE FOLLOWING)

1. Complications of cirrhosis of liver. (2020 P2)
2. Pathogenesis of Alcoholic cirrhosis of liver. (2019 P2)
3. Pathogenesis of alcoholic cirrhosis (2017 P2)
4. Enumerate causes of cirrhosis of liver. (2015 P2)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

5. Complications of cirrhosis of liver. (2013 P2)

6. Ascites in cirrhosis of liver. (2011 P2)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

Group- D (SHORT NOTES)

1. Gall stone. (2017 P2)
2. Complication resulting due to gallstones. (2012 P2)

NERVOUS SYSTEM
Group-B (COMMENT ON)
1. CSF findings in pyogenic meningitis differ from tuberculous meningitis. (2020 P2)
Group-C (ANSWER THE FOLLOWING)
1. Pyogenic meningitis vs. viral meningitis. (2017 P2)
2. Diagnosis and complications of pyogenic meningitis. (2014 P2)
Group- D (SHORT NOTES)
1. Lab investigation of Meningitis(2021 P2)
2. CSF in acute pyogenic meningitis vs. tuberculous meningitis. (2015 P2)
3. CSF in Tuberculous meningitis. (2011 P2)

RESPIRATORY SYSTEM
Group-A (LONG QUESTIONS)
1.A 64 year old man who is a chain-smoker is suffering from chronic cough, a 5 Kg weight
loss in last 3 months. Physical examination shows clubbing of finger. A chest radiograph
shows an ill-defined 3 cm massinvolving left hilum of the lung. Serum calcium level is
12.3 mg/dl. 2+6+2 (2011 P2)
a) What is the provisional diagnosis?
b) Describe the laboratory procedure for diagnosis of the case.
c) Why serum calcium level is elevated in this case?
Group-B (COMMENT ON)
1. Morphological changes of lung in case of tuberculosis of adults. (2021 P1)
2. Bronchopneumonia and lobar pneumonia are not same are not same. (2020 P2)
3. Primary Tuberculosis can involve any segment of Lung Parenchyma. (2019 P2)
4. Primary and secondary tuberculosis of lungs have distinct morphology. (2016 P2)
5. Pneumoconiosis is an inhalation disorder of mineral dust, organic and inorganic
dusts particles. (2015P2)
6. Fate of primary complex of primary tuberculosis. (2013 P2)
7. Primary tuberculosis of the lung. (2010 P2)
Group- D (SHORT NOTES)
1. Neuroendocrine tumour of lung. (2014 P2)

ENDOCRINE SYSTEM
Group-A (LONG QUESTIONS)
1.A person suffering from type-I Diabetes Mellitus develop symptoms over 24 hrs
nausea, vomiting, severe thirst, polyuria. Complaining of abdominal pain, kushmal
breathing with fruity odour on the patients breath and abdominal tenderness
revealed on examination. 1+5+4 (2012 P1)
a) What is your provisional diagnosis?
b) What are laboratory examination will be perform to establish the diagnosis.
c) Explain in short the pathophysiology of that condition.
Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka
 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

Group-B (COMMENT ON)

1. Thyroid swelling is not always due to colloid goitre. (2017 P2)

Group-C (ANSWER THE FOLLOWING)

1. Long term complications of Diabetes Mellitus. (2020 P1)
2. Laboratory diagnosis of Diabetes Mellitus. (2019 P2)
3. Long term complications of diabetes mellitus. (2018 P2)
4. Long term complications of diabetes mellitus. (2016 P2)
5. Classification of Diabetes Mellitus. (2015 P1)
6. Laboratory findings in diabetes mellitus. (2010 P2)

Group- D (SHORT NOTES)

1. Graves disease. (2018 P2)
2. Hashimoto’s thyroiditis. (2016 P2)
3. Hyperparathyroidism. (2014 P2)
4. Glycosylated (Hb A,C). (2011 P2)
5. Hashimoto’s thyroiditis. (2010 P2)

BREAST
Group-A (LONG QUESTIONS)
1.A 55 year old lady presented with a hard non tender mass of about 5 cm in
diameter in left breast andretraction of nipple. 2+4+4 (2017 P2)
a) What is your provisional diagnosis?
b) Describe the plan of investigation for the case.
c) Enumerate the important prognostic factors.

Group-C (ANSWER THE FOLLOWING)

1. Role of FNAC in breast cancer. (2010 P2)

Group- D (SHORT NOTES)

1. Phyllodes tumour of breast (2021 P2)
2. Fibroadenoma of breast. (2020 P2)

BONE
Group-B (COMMENTON)
1. Tumour arising from end of long bone may be benign or malignant. Comment with at
least one example ineach case. (2012 P2)
Group-B (COMMENT ON)
1. Pathogenesis of Chronic Osteomyelitis (2021 P2)
Group-C (ANSWER THE FOLLOWING)
1. Sequestrum. (2020 P2)
2. Classification of bone tumours. (2018 P2)
3. Pathogenesis of osteomyelitis. (2016 P2)
4. Radiological features of important bone tumours. (2014 P2)
5. Pathogenesis of chronic osteomyelitis. (2012 P2)
6. Sequestrum leads to involucrum formation. (2011 P2)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

Group- D (SHORT NOTES)

1. Osteogenic sarcoma. (2019 P2)
2. Giant cell tumour of bone. (2015 P2)
3. Osteoclastic giant cell containing lesions of the bone. (2013 P2)
4. Ewings sarcoma. (2010 P2)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

FEMALE GENITALIA
Group-B (COMMENT ON)
1. Screening test prevents Carcinoma Cervix (2021 P1)
2. Role of PAP smears in screening carcinoma of cervix. (2018 P2)
3. Cervical intraepithelial neoplasia. (2017 P2)
4. Screening based on cervical cytology is useful in the prevention of carcinoma cervix.
(2016 P2)
Group-C (ANSWER THE FOLLOWING)
1. Benign mucinous tumour of Ovary (2021 P2)
2. Cervical intraepithelial neoplasia. (2019 P2)
3. Germ cell tumour of ovary. (2017 P2)
4. Pathogenesis of carcinoma of cervix. (2015 P2)
5. Cervical intraepithelial neoplasia. (2012 P2)

Group- D (SHORT NOTES)

1. Benign cystic teratoma of ovary. (2020 P2)
2. Dermoid cyst of ovary. (2012 P2)
3. Cervical intraepithelial neoplasia. (2011 P2)

MALE GENITA LIA

Group-B (COMMENT ON)
1. Seminoma and dysgerminoma are synonymous. (2019 P2)
Group- D (SHORT NOTES)
2. Benign hyperplasia of prostate. (2019 P2)
3. Seminoma testis. (2018 P2)
4. Seminoma testis. (2016 P2)
5. Nodular hyperplasia of prostate. (2014 P2)
6. Benign hyperplasia of prostate. (2012 P2)
7. Seminoma testis. (2011 P2)

SKIN
Group- D (SHORT NOTES)
1. Basal cell carcinoma. (2018 P2)
2. Basal cell carcinoma. Seminoma testis. (2016 P2)

DIAGNOSTIC CYT OLOGY

Group-B (COMMENT ON)
1. FNAC cannot replace histological study. (2017 P2)
2. Role of exfoliative cytology in the diagnosis of neoplasm. (2013 P2)
3. Fine needle aspiration cytology cannot replace histological study of tissue. (2012 P2)
4. Fine needle aspiration cytology of a thyroid nodule. (2011 P2)
MISCELLANEOUS
Group- D (SHORT NOTES)
1. Reactive Hyperplasia of Lymph Node. (2020 P2)

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka

 10 YEARS CHAPTERWISE by TMCP STUDENTS’ UNIT, MsdMCH --- PATHOLOGY

Edited by: Soumyadeb Roy Compiled by: Apurva Himmatsingka