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Pathophysiology of osteoporosis
Osteoporosis is a disorder in which mineralized bone
density is below normal, resulting in a bone structure that
is vulnerable to fractures.
Type I
Affects women and is associated with estrogen deficiency occurring
5 to 10 years after menopause
Trabecular osteogenesis is primarily affected and results in a
diminished capacity to support compressive loads.
vertebral bodies, the distal radius, and the hip are common sites at
greater risk for fractures.
Type II
osteoporosis affects both males and females.
It is due to calcium deficiency and is associated with aging.
Both trabecular and cortical bone are affected.
Because cortical bone provides support, especially for bending and
torsional loads, the femoral neck, proximal tibia, humerus, pelvis,
and vertebral bodies are at higher risk for fractures.
Vertebral compression fractures (VCFs)
Can present with acute pain, or they can be silent and have
no associated pain.
VCFs typically are caused by falls, but they may result from
as a cough or sneeze.
Mechanisms of action
Anterior compression forces on the spine are reduced by the
countermoment produced by the posterior weight.
The device encourages active back extension through
proprioceptive input and helps increase back extensor strength.
Dorsolumbosacral corset
Dorsolumbosacral corset
They increase the intracavity pressure and provide
resistance to axial loading.
They also serve as a kinesthetic reminder in all three
anatomical planes.
Overall well-being.
Significant decreases in
kyphosis angle, postural sway
magnitude and velocity, and
pain also were observed by
using of Spinomed.
Posterior shell TLSO
Posterior shell TLSO
consists of
a plastic posterior shell,
a soft corset front,
and a system of straps
Similar to the Spinomed, the shoulder straps provide
posteriorly directed forces that help correct the unbalanced
anterior posture in the sagittal plane