Professional Documents
Culture Documents
Result of their curiosity at this stage of life – explore using their senses + common problem
BURNS
Skin damage that results from thermal, chemical, electrical, and radioactive agents
Injuries to body tissue caused by excessive heat (<40’C)
2nd greatest cause of unintentional injury in children 1-4 years of age
Incidence:
o Thermal burns are the most common type of burns
o 50% of cases occur in children 5 years and younger
Moderate 2nd degree burn between 10% - 20% or on the face, hands, feet or genitalia
3rd degree burn <10% body surface
Smoke inhalation has occurred
Emergency Management of Burns
Minor Burns
Pain and death of skin cells – must be treated seriously
Immediately apply ice to cook skin and prevent further burning (public health – toothpaste – temporarily relief)
Analgesic-antibiotic ointment, gauze bandage – to prevent infection (if blistered has not ruptured, gauze bandage is
not necessary) (avoid pricking of the blister
– skin covers the burn serves a protection from infection)
Ff-up in 2 days – inspect are for secondary infection and dressing change
Keep dressing dry – no swimming or getting the are wet for 1 week
o 1st degree burns usually heal by this time
Moderate Burns
Do not rupture blisters - invites infection
Cover w/ topical antibiotic - silver sulfadiazine, bulky dressing to prevent damage to denuded skin
Ff-up in 24 hours to assess that pain control is adequate and no s/sx of infection
Broken blisters - debrided (cut away) to remove possible necrotic tissue
Severe Burns
Fluid therapy, systemic antibiotic therapy, pain management and physical therapy
o To survive w/o disability caused by scarring, infection, or contracture
Electrical Burns of the mouth
Child puts the prongs of a plugged-in electrical cord in the mouth or chews on an electric cord = severe burn ulcer
Blood vessels burn active bleeding
Immediate Tx:
o Unplug electric cord and control bleeding
o Apply pressure to the site
o Admit to hospital for at least 24 hrs
– edema in the mouth airway obstruction
Adequate pain relief
Clean wound OlD w/ antiseptic sol.
o To reduce possibility of infection
Eating a problem
o Mouth is sore
o May be able to drink fluids from cup
o Bland fluids - fruit drinks, ginger ale
Mouth turns black = local tissue necrosis
o May cause deformity of lip and cheeks
– Lip appliance – helps maintain mouth contour
Nursing Diagnoses
Pain r/t trauma to body cells
o Outcome Evaluation: Child states pain is at tolerable level
o Morphine sulfate: IM, IV or epidural administration
Deficient fluid volume r/t fluid shifts from severe burn
o Outcome evaluation: skin turgor remains good; hourly urine output > 1 mL/kg/hr w/ specific gravity bet.
1.003-1.030; VS are w/in acceptable parameters
Risk for ineffective breathing pattern r/t respiratory edema from burn injury
o RR remains w/in 16-20 breaths/min; lung auscultation reveals no rales
o Fluid shifts after burn injury – 1st 24 hours
Remobilization of fluid after 48 hours
–
Risk for imbalanced nutrition, less than body requirements r/t burn injury
o Outcome evaluation: weight remains w/n normal age-appropriate growth percentiles; skin turgor remains
normal; urine specific gravity: 1.003 - 1.030
o Diet high in calories (IV)- MR es as body begins to pool its resources to adjust
– If not enough - begin to break down CHON - dangerous coz CHON needed for burn healing; can lead
to acidosis
o NG tube - nauseated from systemic shock; adequate nutrition
Risk for injury r/t effects of burn, denuded skin surfaces, and lowered resistance to infection w/ burn injury
o Neutrophils cannot phagocytize bacteria after burn injury
o Formation of immunoglobulin G (IgG) antibodies also apparently fails
– Child has reduced protection against infection
– Staphylococcus aureus, group A B-hemolytic streptococci (gram +) organisms; Pseudomonas
deruginosa (gram -) commonly invede burn tissue
Escharotomy
Eschar -- tough, leathery scab formed over moderately or severely burned areas
oFluid accumulates rapidly under eschar= pressure on underlying blood vessels and nerves
– cutting off circulation
o distal parts -- feel cool to touch, appears pale, tingling, numbness; pulses difficult to
opalpate, capillary refill slow (>5 secs)
"cut into the eschar"
Some bleeding after pack wound and apply pressure
Debridement
o Removal of necrotic tissue from burned area
o Reduces possibility of infection -- reduces amt. of dead tissue present on w/c microorganism could thrive
o 30 mins of hydrotherapy -- to soften and loosen eschar
o Then w/c then can be gently removed with forceps and scissors -- allows granulation tissue to form
o Prepa for skin grafting
o GRAFTING
– Homografting (allografting)
Placement of skin (sterilized and frozen) from cadavers or donor on cleaned burn site
These do not grow but provide protective covering for the area
– Heterografts (xenografts)
From other sources - e.g. porcine (pig)
Used in small children
– Autografting
Process in w/c a layer of skin of both epidermis and part of dermis (called a split-
thickness graft) is removed from a distal unburned portion of child's body and placed at
the prepared burn site where it will grow and replace burned skin
Cultured epithelium
o Can be grown into coherent sheet and supply unlimited source for autografts
Nursing Care
Provide priority admission care: ensure patent airway
o Hospitalization for burns 5% - 12% of body surface or more
o Prepare for intubation -- for laryngeal edema
o O2 as ordered
o NGT - to prevent vomiting, aspiration, and abdominal distension
Maintain adequate oxygenation
o Monitor carefully intubate child; humidified air as ordered
o Monitor respiratory fxn q hourly or PRN
o Observe/monitor:
– Signs of respiratory distress: rales, wheezes, flaring, stridor, tachypnea, dyspnea, and air hunger
– ABGS
o Prevent hypostatic pneumonia thru turning, coughing, deep breathing
o Check neck and chest for eschars - may constrict
Relieve shock, maintain fluid and electrolyte balance
o Assess and estimate accurately percentage of body surface burned -- Lund-Browder Classification
o Administer prescribed volume of fluids accurately
o Solutions used:
– Crystalloid solutions (NSS, LRS) - 1st 24 hrs to promote dieresis
– Colloid solutions (albumin, plasma) ff. dieresis
o For the computation of F&E, body weight (kg) and percent (%) of body surface area are used
– Parkland/Baxter Formula
Meticulous wound care
Strict I&O
o Indwelling catheter
o Daily weight
o Diaper count and weighing
o Check hematocrit
– Lund-Browder Classification
–
POISONING
A state of ill-health due to swallowing of common non- nutritive materials
Etiology
Developmental curiosity - developmental age (2 – 3 y.o)
Faulty storage of poisonous substances
Incidence: Peak during toddler period
– Acetaminophen poisoning
In large doses can cause liver destruction
Immediately after ingestion: nausea and vomiting
Soon after:
o Serum aspartate transaminase (AST, GOT) and serum alanine transaminase (ALT, SGPT) – liver enzymes -
become elevated
o Liver feel tender = liver toxicity
Antidote: acetylcysteine
o Prevents hepatoxicity by binding w/ the breakdown, product of acetaminophen so it will not bind to liver
cells
o Has offensive odor
– Administer via carbonated drinks
– For small children - into an NG tube
o Continue to observe for jaundice and tenderness over liver
o Assess ALT and AST levels as ordered
Caustic poisoning
Ingestion of a strong alkali, such as LYE (toilet bowl cleaners, hair care products)
May cause burns and tissue necrosis in the mouth, esophagus and stomach
Do not try to make child vomit – cause additional burning
Assessment
Immediate pain in mouth and throat and drools saliva = oral edema and inability to swallow
Mouth turns white brown (edema and ulceration)
Vomiting of blood, mucus, and necrotic tissue
Loss of blood (from denuded surface) systemic signs of tachycardia, tachypnea, pallor and hypotension
Chest radiograph
Esophagoscopy
Therapeutic Management
Immediately take child to health care facility
Assess VS closely esp. RR*
Watch out for increasing restlessness (infants) -- oxygen want
Intubation may be necessary -- ensure patent airway
Assess degree of pain involved -- Morphine may be ordered
Iron poisoning
When ingested corrosive to the gastric mucosa s/sx of gastric irritation
Immediate effects: nausea, vomiting, diarrhea, and abdominal pain
By 6 hrs:
o s/sx fade, child's condition appears to improve
o By this time, hemorrhagic necrosis of the lining of the Gl tract has occurred
By 12 hrs:
o Melena and hematemesis present
o Lethargy and coma, cyanosis, and vasomotor collapse may occur
o Coagulation defects, hepatic injury
o Shock from in peripheral vascular resistance and
o ed cardiac output
Long-term effects
o Gastric scarring from fibrotic tissue formation
Therapeutic Management
Stomach lavage -- to remove any pills not yet absorbed
Soothing compound Maalox (aluminum hydroxide) or Mylanta (magnesium hydroxide) -- to help gastric irritation
and pain
Antidote: deferoxamine
o IV or IM
o Chelating agent that combine w/ metals and allows its excretion
o Caution parents: urine turns orange as iron is excreted
Exchange transfusion – to remove excess iron
Upper Gl series and liver studies may be ordered week after ingestion
o To screen for long-term effects
Assist w/ emergency measures: lavage, meds
Stool exam for next 3 days for occult blood
o To assess for stomach irritation and Gl bleeding
LEAD Poisoning
Poisoning due to Ned levels of lead resulting from ingestion and absorption of lead-containing substances
Also referred to as plumbism
Etiology
Most common cause: lead-based paint (used in houses before 1950s, US)
Lead value of more than 15 mcg (ug)/dL - health hazard
Incidence
Most common in toddlers and preschoolers
o Prevalent in children 1 - 6 yrs of age who live in old deteriorated houses
Pica - tendency to eat non-edible substance
Pathophysiology
Lead enters the body interferes w/ RBC fn by blocking the incorporation of iron into the proporphyrin compound that
makes up the heme portion of hemoglobin in RBC
Leads to hypochromic, microcytic anemia
Kidney destruction may occur excess excretion of amino acids, glucose, and phosphates in urine
Most serious effect: LEAD encephalitis
o Inflammation of brain cells because of toxic lead content
Assessment
(+) lead poisoning: 2 successive blood lead levels >10 ug/dL
Usual sources of ingested lead:
o Paint chips or paint dust, home-glazed pottery, or fumes from burning or swallowed batteries
o Crib, windowsills - child teething
o Lead plumbing - may contaminate drinking water
Signs and Symptoms
May be asymptomatic; symptoms appear when lead blood level is 70 mcg/dL
Fatigue, pallor, listlessness
Gl complaints: anorexia, vomiting, weight loss
Irritability, clumsiness
Ataxia, loss of coordination, seizures
Acute lead colic and encephalopathy: most severe forms
o Early sx: lethargy, impulsiveness, learning difficulties
o As lead blood level es: seizures and permanent neurologic damage
Diagnostic Tests
Serum ferritin - blood lead determination
erythrocyte protoporphyrin (EP) levels
o Fingerstick
o Protoporphyrin is blocked from entering heme by the lead = elevated
Basophylic stippling (odd striation of basophils) apparent on blood smear
Radiograph:
o Abdomen: reveals paint chips in the intestinal tract
o Long bones: "lead lines" (areas of fed density) near epiphyseal line
– Thickness of line = length of time ingestion has been occurring
Treatment
Widespread screening programs
Prevention: reduction of lead from environment (responsibility of health agency)
o Reduce lead from air (by eliminating lead additives from automotive fuels), drinking water (enforcing
standard for lead content), and reduce lead content of foods (canned foods)
Prompt separation of child from sources of lead: cornerstone of tx
Chelation therapy - remove lead from soft tissue and bone (tho' more from RBC), allowing it to be eliminated into
the urine
o > 45ug/dL: BAL (dimercaprol) or edetate calcium disodium (CaEDTA)
– Given as multiple IM injections (large muscle mass)
– Painful - may be combined w/ procaine 0.5 mL
– EDTA removes calcium from body = calcium measured periodically
o Chelation therapy before onset of symptom lessens risk of encephalopathy
Fluid and electrolyte; 10% D5W and mannitol - to decrease ICP as necessary
For seizures:
o Initially Diazepam
o Repeated doses of paraldehyde, and
o Long-term anticonvulsant therapy w/ phenobarbital
Nursing Care
Prevention is the best nursing care. Provide teaching:
o iron and calcium in the diet - low dietary calcium and iron enhances toxic effects
o Observe care in toy selection for infants and young children -- shd not be lead-painted
Provide care to child and parents for drug administration
Administer BAL and CaEDTA as recommended
o BAL and CaEDTA injected simultaneously t separate IM sites in 6 ÷ doses/day for 5 days
o Pain relief at injection sites using warm soaks and site rotation
Maintain hydration; fluids and measure
I&O because chelating therapy is renal toxic & Monitor VS, I&0, and signs of ICP
CHILD ABUSE
Physical and sexual abuse and emotional neglect of children
Etiology: Exact cause
unknown Predisposing factors
Child factors
Product of unexpected, unwanted, difficult pregnancy, labor, and delivery
Physically ill or defective, the "different child"
Child may not fulfill the need of the caretaker
Parental factors
o Abused when they were kids; lacking in basic sense of trust; yearning for love
o Young age, dependent, but w/ little or no support system
o Loners (no social affiliation)
o Immature emotional responses and coping patterns
o Lack of knowledge of normal G&D of the child
Environmental factors
o Chronic financial stress
o Divorce, parental separation
o Chronic social and emotional stress
Incidence: younger children under 4 years old
Diagnostic tests/ procedures
Careful history-taking and physical examination
Complications
o Further abuse
o Psychological problems or disorders
o Death
Signs of physical abuse
Burns, bruises, scars, cigarette marks on the skin
Fractures, dislocations (unreported)
Poor hygiene, w/ head lice or skin disease
Malnutrition, retarded physical growth and devt.
Delayed language devt.
Absence of immunization
Inappropriate reactions: withdrawal from people, passivity, or restlessness
Absence of protest on admission of toddler
– Emotional abuse/neglect
A pattern of behavior that attacks a child's emotional development and sense of self-worth
Includes excessive, aggressive or unreasonable demands that place expectations on a child beyond his or her capacity
Constant criticizing, belittling, insulting, rejecting and teasing are some of the forms these verbal attacks can take
Also includes failure to provide the psychological nurturing necessary for a child's psychological growth and
development -- providing no love, support or guidance
o FTT
o Sleep disturbances
o Withdrawal and fearfulness
o Sucking fingers, biting nails
o Delayed language development
o Delinquency or runaway behavior
o Shows provocative behavior that generates anger from others
o Does not seek out parents for comfort and affection
o Reactions indicate tendency to avoid
Sexual abuse
Being overly affectionate or knowledgeable in a sexual way inappropriate to the child's age
Medical problems such as chronic itching, pain in the genitals, STIs
Other extreme reactions, such as depression, self- mutilation, suicide attempts, running away, overdoses, anorexia
Personality changes such as becoming insecure or clinging
Regressing to younger behavior patterns such as thumb sucking or bringing out discarded cuddly toys
Sudden loss of appetite or compulsive eating
o Being isolated or withdrawn
o Inability to concentrate
o Lack of trust or fear of someone they know well, such as not wanting to be alone with a babysitter or child
minder
o Starting to wet again, day or night/nightmares
o Become worried about clothing being removed
o Suddenly drawing sexually explicit pictures
o Trying to be 'ultra-good' or perfect; overreacting to criticism
CEREBRAL PALSY
Neuromuscular disorder characterized by lack of control of the voluntary muscles, spastic and involuntary
movements, abnormal muscle tone and incoordination
Etiology
Anoxia to the brain: most significant factor
Infection
Predisposing factors
Prenatal: heredity, X-ray, and any factor that results in
ed blood flow/oxygenation to fetal brain
Intranatal: difficult labor/delivery; birth trauma/injury; all causes of fetal anoxia, including analgesia/anesthesia
Postnatal: devtal brain anomalies, and CNS, trauma and infections like meningitis and encephalitis
Signs and symptoms
An infant who was subjected to prenatal or intranatal factors may manifest early signs of CP:
o Abnormal posturing
o Difficult feeding
o Tremors, seizures
o Persistence of primitive reflexes: Moro,
tonic neck and fisting
Later, signs of delayed motor development
Treatment
Exercises: passive and active
Medications:muscle relaxants,
anticonvulsants, tranquilizers
Braces, ambulation devices: crutches, walkers
o Prevention of contractures – major goal in the tx
o Braces are used to reduce possibility of contractures
Nursing care
Promote adequate nutritional intake
o calories to meet addt'l demands of
constant muscle movement
o Feed slowly; Calm, peaceful environment
o Adequate fluid intake: maintain 1&0
o Use special silverware and dishes, as
needed (padded spoon, nonskid dishes)
o Modify feeding technique to compensate
or deal w/ tongue thrusting or extrusion
reflex
Promote maximum mobility and development
of self- help skills
o Teach use of braces, splints, support
chair or wheelchair as needed
o Teach self-help skill -- one skill at a time
o Avoid movements, incidents, and
situations that trigger abnormal
movements; calmness and tranquility are
favorable
o Encourage participation
o Value of proper alignment, ROM exercises
and stretching 3 x a day
Ensure safety when ambulating
o Use safety prec and equipment when ambulating
o Observe seizure prec
o Provide safe toys: nothing that can cause
falls, skidding, slipping
Administer drugs, as ordered, noting positive
effects, toxic effects, and other untoward effects