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[Presumed prior knowledge: physiological triggers of vomiting e.g. via central nervous system,
neurohormonal etc.; embryological, histological and anatomical features of normal gastrointestinal
tract]
Describe the above congenital causes of gastrointestinal obstruction with respect to: basic
pathophysiology, presenting features, key diagnostic radiological features, principles of pre-surgical
stabilization, description of corrective surgical procedure in layman’s terms and long-term outcome.
UMBILICAL HERNIA
A) DEFINITION:
Hernia:
Protrusion of an organ from the cavity where it normally resides, through a defect in the wall
of that cavity
Umbilical hernia:
Abdominal contents herniate through a defect in the umbilicus as a result of the incomplete
closure of the umbilical ring following the return of the bowel into the abdominal cavity
during development
B) PATHOPHYSIOLOGY:
Incidence:
Common: at least 3% of newborn;
5-10% higher incidence in black patients
C) CLINICAL PRESENTATION:
D) MANAGEMENT
Procedure:
Peri-umbilical incision
Mobilisation of hernia sac
Herniotomy and closure of defect
E) DIFFERENTIAL DIAGNOSIS:
Para-umbilical hernia:
The para-umbilical hernia arises from a defect in the linear alba, above the umbilicus
The defect is elliptical in shape
This hernia seldom closes spontaneously
There is a higher risk of intestinal obstruction
It is surgically repaired
RECTUS-SHEATH DIASTATIS:
Normal variant of abdominal wall
F) POTENTIAL COMPLICATIONS:
Cosmetic concerns
Almost no recurrence
Incarceration of omentum or bowel is rare. There is an increased risk of bowel
incarceration in the following patients:
Patients with pica who eat sand
During pregnancy (girls)
UMBILICAL DISCHARGES
A) DEFINITION:
B) PATHOPHYSIOLOGY:
Causative organisms:
Usually Staphylococcus or a haemolytic Streptococcus.
Fungal infections may also occur.
Tetanus due to use of traditional dressings to newborn umbilical cord e.g. cow
dung have declined
C) CLINICAL PRESENTATION:
Foul-smelling umbilicus
Purulent discharge
Erythema, oedema and cellulitis of the abdominal wall surrounding the
umbilicus
D) COMPLICATIONS:
Infection of the umbilicus is always regarded as a severe infection. The congenital
connections to the umbilicus may result in dissemination of the infection.
The impaired immunity of the baby may result in septicaemia and other serious infection
related complications
Septicaemia
babies have a higher chance of developing septicaemia and must be admitted
for intravenous antibiotics administration
Cellulitis spreads to the abdominal wall
Umbilical gangrene
Necrotising fasciitis can spread out of the umbilicus, a very severe infection
which requires aggressive management:
Abscesses from infections spreading along congenital tract
Omphalomesenteric duct remnants
Urachal remnants – urachal abscesses
Umbilical vein thrombosis: From infection; results in long-term extra-hepatic
portal vein obstruction and portal hypertension
E) MANAGEMENT:
IV fluid
Correction of electrolytes
Broad-spectrum antibiotics
Surgical debridement of necrotizing fasciitis
Immunoglobulin (passive immunity)
UMBILICAL GRANULOMA
A) DEFENITION:
Red granuloma of the umbilicus develops as a result of a low-grade infection. May drain a
little pus
B) MANAGEMENT:
A) DEFINITION:
B) CLINICAL PRESENTATION:
C) MANAGEMENT:
A) EMBRIOLOGY:
MECKEL’S DIVERTICULUM
A) DEFINITION:
B) CLINICAL PRESENTATION:
C) SPECIAL INVESTIGATIONS:
D) MANAGEMENT:
E) COMPLICATIONS:
Rectal bleeding (40%)- due to peptic ulceration resulting from ectopic gastric mucosa
Lead point in intussusception 20%
Diverticulitis or free perforation 15%
Intestinal obstruction – volvulus around Meckel’s band 7%
PATENT URACHUS
A) DEFENITION:
B) CLINICAL PRESENTATION:
C) MANAGEMENT: