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Glaucoma
1. . SEMINAR ON GLAUCOMA PRESENTED BY Ms. Jinumol First year M.sc Nursing NUINS
2. .
3. INTRODUCTION The term Glaucoma is a term used to refer group of ocular condition characterized by the optic nerve damage. Glaucoma occurs as a result of increased intraocular pressure (IOP) caused by a
malformation or malfunction of the eyes drainage system. Normal IOP is 19 – 21 inches of mercury. The increased pressure causes compression of the retina and the optic nerve, and causes progressive , permanent loss of
eyesight if le untreated.
4. DEFINITION Glaucoma is a group of disorder characterized by an abnormally high intraocular pressure , optic nerve dystrophy, and peripheral filed loss. (BRUNNER) Glaucoma is a symptomatic condition of the eye
where the intraocular pressure is more than normal. ( L.P AGARWAL)
5. .• Normal IOP is 16 to 23 mmHg. INCIDENCE Globally 6 to 67 million. More common in peoples older than 40 years.
6. ETIOLOGY Increased intraocular pressure.( more than 24 mmhg) Optic nerve dystrophy.
7. RISK FACTORS Genetic Factors : Family history Aging : above 40 Eye trauma Hypertension Severe Myopia Ocular surgery Diabetics mellitus Migraine headache
8. . • Emotional excitement • Ca eine consumption (increases IOP) • Ethnicity ( More common in Black people compared to white people) • Prolonged use of corticosteroid
9. PATHOPHYSIOLOGY Excess production of aqueous humor Decreased outflow of aqueous humor Sed IOP
10. . . Ischemia of Nerve. Damage to optic nerve LOSS OF VISION
11. CLASSIFICATION GLAUCOMA 1. Primary Glaucoma 2. Secondary Glaucoma CONGENTIAL ACQUIRED 1. True congenital. 2. Infantile 3. Juvenile
12. CONGENTIAL GLUCOMA Rare disease. Occurs when a congenital defect in the angle of the anterior chamber obstructs the outflow of aqueous humor. If remains untreated causes damage to optic nerve damage
and blindness. 1. True Congenital Glaucoma occur when IOP increases during intrauterine period. Child is born with ocular enlargement. occur in 40 % of cases
13. .• INFANTILE GLAUCOMA occurs during third birthday occurs about 10% of cases. • JUVENILE GLAUCOMA Occurs during 3 to 16 years of life. occurs in about 10 % of casas
14. Clinical features of congenital glaucoma • Lacrimation • Photophobia • Corneal oedema • Raised IOP • Eyes Become MYOPIC • Corneal diameter more than 13mmhg
15. ACQUIRED GLAUCOMA • PRIMARY GLUCOMA. o Primary Open angle glaucoma. o Primary angle closure Glaucoma o Chronic angle closure glaucoma. • SECONDARY GLAUCOMA o Lens induced glaucoma o Glaucoma
due to uveitis o Neurovascular Glaucoma o Glaucoma associated with intraocular tumor o Steroid induced glaucoma
16. . • PRIMARY GLAUCOMA. Primary Glaucoma is a progressive condition and is most common cause of irreversible blindness across world wide. a. Primary Open Angle Glaucoma Also Called as open angle Glaucoma
or chronic simple Glaucoma or simple complex Glaucoma. Results from the overproduction of aqueous humour through trabecular Mesh work results in increased IOP and Damage to optic nerve, results in loss of vision.
17. . • The clinical features of Primary Open angle glaucoma includes. Mild ache in the eyes Headache Increased IOP ( more than24 mmhg) Loss of Peripheral vision Reduced visual acquity at night. Corneal
edema Visual field deficit.
18. b. Primary angle closure glaucoma • It is also called as Primary closed angle glaucoma, Narrow angle glaucoma, Pupil block glaucoma and acute congestive glaucoma. • Onset : rapid, ophthalmic emergency. Unless
treated promptly the causes blindness in 3 or 5 days. • This is due to the abnormality of structure infront of the eyes. This result from the obstruction to the outflow of aqueous humor.
19. Clinical features of primary angle closure glaucoma • Pain and redness in eyes • Increased IOP • Blurred vision • Headache • Nausea • Vomiting • Oedematous cornea • Decreased visual acuity • Moderate Pupillary
dilation.
20. c. Chronic angle closure Glaucoma • Chronic angle closure Glaucoma may develop as the sequelae to an attack of acute angle glaucoma. • Clinical features include Increased IOP. visual field defect. Decreased visual
acquity.
21. 2. Secondary glaucoma • Secondary glaucoma occurs as a result of either diseases within the eyes such as uveitis, Inflammation , Trauma, intra ocular haemorrhage, previous surgeries, diabetics and steroid
medication etc • The major types include
22. . • TYPES LENS INDUCED GLAUCOMA GLAUCOMA DUE TO UVEITIS NEUROVASULAR GLAUCOMA GLAUCOMA ASSOCIATED WITH INTRA OCULAR TUMOR. STEROID INDUCED GLAUCOMA
23. . 1. LENS INDUCED GLAUCOMA o It occur due to trabecular blockage. o it occur due to clogging of trabeculae by inflammatory material. 2. GLAUCOMA DUE TO UVEITIS o IOP is raised due to clogging by inflammatory
material & associated trabeculitis.
24. 3. Neurovascular glaucoma • Uncommon type of glaucoma • Di icult to treat • Caused by proliferative diabetic retinopathy. • Individual with poor blood flow to the eyes are highly at risk for this condition. 4. Glaucoma
associated with intra ocular Tumor. Intraocular tumor such as retinoblastoma & Malignant melanoma may rise IOP.
25. 5. Steroid induced Glaucoma • Developed due to sensitivity to steroid. • Sudden rise in IOP may occur, this can be prevented by judicious use of steroid. 6. Pigmentary Glaucoma • Rare Condition • This is caused by
pigment cells sloughing o from the back of the iris & floating around the aqueous humor.
26. DIAGNOSTIC MEASURES o History collection o Patient ocular & Medical history. o Tonometry ( to measure IOP) o Ophthalmoscopy ( To show the cupping of the optic disc ) o Gonioscopy (To determine the angle of the
eyes anterior chamber) o Perimetry or visual field test.( To detect loss of peripheral vision)
27. . o Slit Lamp Examination. o Fundus Photography ( To monitor the Disc for changes) o Pachymetry o Nerve fiber analysis ( to asses the thickness of nerve fiber layer)
28. MANAGEMENT • Goal of Management measures include To reduce IOP To prevent the damage of optic nerve. Management measures mainly include Medical Management Surgical management Nursing
Management
29. MEDICAL MANAGEMENT • Beta adrenergic blockers : decreases aqueous humor production , eg Timolol, betaxolol. • Cholinergic ( Miotics) : Reduces IOP by facilitating the outflow of aqueous humor. eg Pilocarpine,
Carbacol. • Carbonic anhydrase inhibitor : Decreases the formation & secretion of aqueous humor.
30. . • Prostaglandin Analogs : Reduces IOP by increasing uveoscleral Flow. • Osmotic Agents : Iv mannitol 20% or oral glycerine 50% is used to reduce IOP by creating an osmotic pressure between blood and intraocular
fluid
31. SURGICAL MANAGEMENT • Argon Laser Trabeculoplasty : Used to treat open angle glaucoma. Thermal Argon laser burns are applied to the inner surface of trabecular Meshwork to open intra trabecular spaces ,
thus reduces outflow of aqueous humor and decreases IOP.
32. • Laser Iridotomy : In this surgical procedure, an opening is made by the laser beam in the iris to eliminate pupillary block. • Relive the pressure & preserve the vision by promoting outflow of the aqueous humor. •
Cyclocryotherapy Application of a freezing probe to the sclera over the Cillary body that destroy some of the cillary processes , results in the reduction of the amount of the aqueous humor
33. . • Cyclodialysis Through a small incision in the sclera a spatula type instrument is passed into the anterior chamber, creating an opening in the angle. • Filtering Procedure For Chronic Glaucoma filtering procedure
are used to create an opening or fistula in the trabecular meshwork to drain aqueous humor. This allow the aqueous humor to flow & exit di erent route.
34. . • Trabeculotomy In this procedure a partial thickness incision is made in the sclera. Section of sclera is removed to produce an opening for outflow of aqueous humor. • Drainage implants and shunts Used to
Shunt the aqueous humor in the Conjunctival space. Implants and shunts are the open tubes implanted in the anterior chamber through sclerotomy
35. NURSING MANAGEMENT ASSESSMENT • History collection ( positive family history) • Risk factors such as tumor of eyes. • Intraocular haemorrhage. • Inflammatory intraocular uveitis. • Eyes contusion from trauma.
36. . General Physical examination • Assess for Sudden severe pain in eyes, Halo light, blurred vision etc. Check for diagnostic Measures Assess the patient Understanding and emotional response to the condition.
37. NURSING DIAGNOSIS • Acute pain related to increased IOP. • Self care deficit related to loss of vision. • Fear and anxiety related to Pain and potential loss of vision. • Risk for injury.
38. NURSING INTERVENTION • Assess the IOP. • Elevate the head end of the bed at 30 degree angle. ( Patient should be placed in un operated side) • Instruct the patient not to touch the eyes . • Encourage the patient to
wear eye shield. ( To prevent infection). • Administer medications as prescribed.
39. Complications • Complete loss of vision • Choroidal detachment • Retinal detachment • Chronic hypotony • Anterior Segment necrosis
40. Research studies Safety & e iciency of Biomatoprost sustained – release in patients with open angle glaucoma or Ocular hypertension.
41. CONCLUSION • Glaucoma is a condition that causes damage to the eyes optic nerve& gets worse over the time. Without treatment the glaucoma can cause permanent blindness within few years. So the treatment
should be given at right time to prevent complications.
42. BIBIOGRAPHY • K Khurana, Textbook of Ophthalmic Nursing, CBS Publishers, Banglore,6th edition 2008, PG NO 141 – 153. • Black.M.Joyce.Text book of Medical Surgical Nsg.Elsevier Publication.8 th edition. Pg no 567
– 568. • Chintamani, Lewis, Text book of Medical surgical Nursing, Elsevier Publication 13 th edition. volume 1. 2011.pg no 1723-1725.
43. .

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