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اخوكم حٌدرالموسوي
4. Defecation:
External anal sphincter opens
Intra-abdominal pressure increases with and stool moved out
Why It Is Done
Stool sampling
مالحظات للمرٌض
بالنسبة لألطفال ٌفضل التبول أوال لبل أخد عٌنه االبراز حتى ال ٌحدث اختالط .1
) لبل اإلمسان بعٌنه البراز حتى تتجنب نمل العدوى أو استخدم أداه لنملgloves( البد من ارتداء لفازات .2
العٌنة فى الوعاء
)toilet bowl(ال تأخذ عٌنه البراز من لاعدة الحمام .3
ال تخلط العٌنة بالماء أو الصابون .4
اغسل ٌدٌن جٌدا بعد أخد العٌنة .5
مالحظات للمعمل
Note:: Exposing the stool sample to air or room temperature will affect the test result.
عٌنة الصباح الباكر هً األفضل (الن البراز هنا مجمع طوال اللٌل و فرصه ظهور الطفٌلٌات و البوٌضات اكبر) .4
مراعاة عدم اختالط البول بالبراز خاصة عند األطفال ألنها تمتل األطوار النشطة .5
مراعاة عدم اختالط البراز بالدم عند النساء أثناء الدورة الشهرٌة حتى ال ٌكون التحلٌل ثم التشخٌص خطأ .6
ترفض العٌنة من الشخص الذي أجرى أشعة بالصبغة خالل ٌومٌن بعد األشعة .7
.8
Note:: sample rejected from patient recently had an X-ray test using barium contrast material,
such as a barium enema
.9عٌنة األطفال فى الحفاضات ترفض إذا كان البراز جاف نتٌجة تشرب البامبرز .
Note:: Samples from babies and young children may be collected from diapers (if the stool is
not contaminated with urine) or from a small-diameter glass tube inserted into the baby's
rectum.
ٌجب االمتناع عن أخد بعض األدوٌه المعٌنة لبل أجراء التحلٌل مثل: .11
فى حالة اخذ أى أدوٌة تؤثر على البراز البد من إخطار المعمل بها والسؤال عنها حسب الحالة .11
Note:: Sometimes a stool sample is collected using a rectal swab that contains a preservative
(if patient is unable to give stool). The swab is inserted into the rectum, rotated gently, and then
withdrawn. It is placed in a clean, dry container and sent to the lab right away.
ملحوظه
أهم عامل فى فحص البراز هو خبرة االخصائً الذي ٌموم بالفحص وطرٌمة تركٌز العٌنة ،فمن المعروف أنه كلما
كانت اإلصابة شدٌدة كلما سهل التشخٌص وكلما كانت اإلصابة طفٌفة أو كان الطفٌل فى حالة سكون كلما كان
التشخٌص أصعب وٌحتاج إلى خبرة أكبر .
ملحوظه
أثبتت األبحاث أنه ٌجب فحص ثالثة عٌنات براز على األلل فى ثالثة أٌام متتالٌة حٌث أن األعتماد على عٌنة واحدة
فمط ال ٌكفى وذلن لزٌادة الفرصة فى رؤٌة الطفٌلٌات وٌفضل ثالثة معامل مختلفة حتى ٌتم تالفى خطأ المعمل وإذا
كان الفحص سٌتم فى نفس المعمل ٌفضل أن ٌرى العٌنات أكثر من شخص.
Preparation methods
Direct smear
Concentration technique
Sedimentation method
Saline sedimentation Tech.
Formal Ether Concentration Tech.
Floatation method
Kato technique
Direct smear
الطرٌمه
.1ضع لطعة صؽٌرة من البراز على شرٌحه نظٌفه و امزجها مع نمطه من محلول الملح ثم افحصها تحت
المٌكروسكوب مستخدما العدسه 10ثم العدسه 40
.2ادا كان بالعٌنه جزء به دم او صدٌد او مخاط ٌجب البدء بفحص هدا الجزء
ٌ .3جب فحص عٌنة من سطح البراز و عٌنه من داخله الحتمال وجود طفٌلٌات باالمعاء الدلٌمه او االمعاء
الؽلٌظه
ٌ .4مكن استخدام الٌود لصبػ العٌنة لرؤٌة االطوار المتحوصله لكن الٌود ٌمتل الطور النشط فلذلن افحص
بدون الٌود اوال
الممٌزات
الطرٌمة سهلة وسرٌعة وتستخدم أساسا لفحص االكٌاس المتحوصله للطفٌلٌات
العٌوب
طرٌمه ؼٌر دلٌمه و تحتاج ان ٌكون االصابه بالطفٌل شدٌده اما لو كانت اعداد الطفٌلٌات فى العٌنه للٌله ففرصه
رؤٌتها بهده الطرٌمه ضعٌفه جدا
Sedimentation method
الممٌزات
هده الطرٌمه مناسبة للطفٌلٌات االثمل وزنا من محلول الملح و بالتالى تترسب فى لاع االنبوبه مثل:
Ascaris
Shistosoma mansoni
Fasciola
Strongyloides stercoralis
العٌوب
ال تصلح لجمٌع الطفٌلٌات بنفس الكفاءه
ٌ .9تم التخلص من الطبمات العلوٌة الثالثة من األنبوبة وٌترن الراسب فمط وٌمزج هذا الراسب جٌدا.
تفحص نمطه من الراسب تحت المٌكرسكوب. .10
تمزج نمطه من الراسب مع الٌود و تفحص تحت المٌكرسكوب. .11
العٌوب
طرٌمه طوٌله و صعبه
Floatation method
الطرٌمه
.1تؤخذ لطعة من البراز وتوضع فً كوب نظٌؾ ثم ٌضاؾ محلول الملح الى الكوب.
ٌ .2ملب البراز فً المحلول جٌدا.
ٌ .3صفً البراز فى انبوبه باستخدام لطعه شاش او ؼطاء الكوب.
.4اترن اانبوبه لمده 10دلائك.
.5اكمل االنبوبه حتى فوهتها العلٌا بمحلول الملح.
.6توضع شرٌحة فوق فوهة االنبوبه.
.7تترن االنبوبه لمدة 30 – 20دلٌمة.
.8ترفع الشرٌحة باحتراس وتملب وتوضع تحت المٌكروسكوب و تفحص.
الممٌزات
هده الطرٌمه مناسبة للطفٌلٌات االخؾ وزنا من محلول الملح و بالتالى تطفو على سطح السائل فى االنبوبه مثل:
Entrobius
Ankylestoma
Tricuris
العٌوب
ال تصلح لجمٌع الطفٌلٌات بنفس الكفاءه
Kato technique
الطرٌمه
.1حضر شرٌحه زجاجٌه نظٌفه
.2ضع على الشرٌحه مربع كاتو
.3امالء فراغ مربع كاتو بعٌنه البراز
.4اصبػ العٌنه بصبؽه مالكٌت جرٌن
.5تؽطً العٌنة بالسٌلوفان المشبع وتفرد جٌدا وتحضن علً 37درجة لمدة ساعتٌن ثم تفحص
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الممٌزات
تستخدم للفحص وكذلن لعد البوٌضات لتمدٌر شدة اإلصابة .
ٌمكن االحتفاظ بالشرائح المصبوؼة لمدة حوالً 6شهور .
العٌوب
ال تصلح للكشؾ عن االنتامٌبا أو ٌرلات االسترنجلوٌدس ،لذا ٌجب الفحص بطرٌمة اخرى باإلضافة إلً كاتو .
Physical examination
Microscopic examination
State of digestion
Starch granules + +
Fat globules ++ +
Vegetable cells +++ +
Muscle fibers + +
Parasites
Protozoa : Vegetative Absent Absent
: Cysts Absent Absent
Helminthes : Larvae Absent Absent
: Ova H. nana Absent
Gross examination
Color
Normal:
Brown color is the normal color of stool
Why stool color is brown? The characteristic brown color of feces is due to
stercobilin and urobinin, both of which are produced by bacterial degradation of
bilirubin.
Abnormal:
Black color: indicate iron medication (for treatment of anemia) or upper GIT
bleeding (due to peptic ulcer, stomach carcinoma or esophageal varices).
Bright red color: indicate lower GIT bleeding (due to piles and anal fissure).
Clay color(gray-white): indicate obstructive jaundice
Silver color: indicate combination of jaundice and blood (cancer of ampulla of
Vater).
Red brown color: indicate drugs as Tetracyclines, and Rifambicin antibiotics
Green color: indicate medications as Diathiazine, Mercurous chloride also
vitamins cause green color of stool and in some cases of intestinal putrefaction.
Note: red stool color may be seen if there are undigested beets or tomatoes.
Note: if stool color is black and there is no history of iron medication FOBT is recommended.
Ampulla of Vater
The ampulla of Vater, also known as the hepatopancreatic ampulla, is formed by the
union of the pancreatic duct and the common bile duct.
Function
Various smooth muscle sphincters regulate the flow of bile and pancreatic juice through
the ampulla: the sphincter of the pancreatic duct, the sphincter of the bile duct, and the
الموسوي اعداد وتقديم حيدر
hepatopancreatic sphincter (Sphincter of Oddi), which controls the introduction of bile and
pancreatic secretions into the duodenum, as well as preventing the entry of duodenal
contents into the Ampulla.
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Odour
Normal:
Normally offensive
Why stool odour is offensive? Fecal odor results from gases produced by
bacterial metabolism, including skatole, mercaptans, and hydrogen sulfide.
Abnormal:
Odorless: usually seen if there is no fecal matter as in bacterial infection.
Very offensive: usually seen in cases of constipation and with certain types of
food that produce excessive gases.
pH ( reaction )
Normal:
Normally variable
Why stool pH is variable? Because stool pH mainly depends on the type of diet.
Abnormal:
High alkaline stool
Physiological Pathological
High protein diet Poor fat absorption
Abnormal:
Abnormal consistency may be graded as follow:
Very hard: seen in cases of constipation
Semi formed: seen in the cases of parasitic infection
Soft: seen in the cases of parasitic infection
Loose: seen in the cases of diarrhea
Watery: mostly seen in cases of bacterial infection
Mucus
Normal:
Normally undetectable amount produce by GIT and found in the stool.
Abnormal:
Abnormal mucus in the sample appears as white patches and usually present
with appearance pus and indicate bacterial infection.
According to the amount of mucus it can be graded using signs (+, ++, +++)
Note: mucus with fresh blood in stool usually indicate amoebic dysentery
Appearance pus
Normal:
Normally not found (pus can be found normally in the stool but you cannot see
it by naked eye).
Abnormal:
Usually detected with mucus and appear as white patches in the stool, it
indicate ulcerative colitis or bacterial infection as bacillary dysentery.
Also it can be graded using signs (+, ++, +++).
Presence of detectable pus by naked eye means that the microscopic pus
must be over 100.
Gross blood
Normal:
Normally no blood seen in the stool (you cannot see it by naked eye).
Abnormal:
Abnormal fresh blood seen in cases of lower GIT bleeding caused by:
Bilharzias (schistosoma mansoni infection)
anal fissure
piles
فى الفحص المٌكروسكوبىRBCs اذا وجدت دم فى العٌنة بالفحص الظاهرى فالبد من كتابه عدد ال: ملحوظه
.)over 100( 111 اكثر من
Abnormal:
In some cases the whole worm or parts of its body appear in the stool and can be
seen by naked eye.
Three worms can be seen by naked eye in the stool:
Ascaris lumbericoides
Entrobius vermicularis
Taenia sp.
Microscopic examination
تحضٌر الشرٌحة للفحص
ٌجب التأكد ان المٌكروسكوب صالح للعمل من حٌث سالمة ونظافة العدسات ومن حٌث اإلضاءة والمسرح.1
. التأكد من نظافة الشرائح المٌكروسكوبٌة وكذلن غطاء الشرائح.2
مالحظات هامة
.1ابدا الفحص من نمطة معٌنة على الشرٌحه و اتبع خط سٌر محدد zigzag
.2حاول ان تفحص الشرٌحة بطرٌمة متداخلة overlappingحتى ٌتم فحص الشرٌحه كامله دون ترن
فراغات.
.3افحص شرٌحتٌن على االلل لكل عٌنة و ادا كنت تستخدم طرٌمه اللطخه اختار عٌنه من أماكن مختلفة حتى
ٌمكن الحكم علً نتٌجة العٌنة جٌدا.
.4عند وجود صدٌد او دم فى الشرٌحة افحص اكثر من شرٌحتٌن للتأكد التام من عدم وجود دٌدان
.5التضع فى تفكٌرن شىء معٌن و انت تبحث فى العٌنة مثل ان تبحث عن ovaمعٌنه مثال
ٌ .6فضل استخدام العٌنٌن فى الفحص ( مٌكروسكوب ( binocularحتى الٌصاب الشخص الذى ٌفحص
بالصداع اذا استخدم عٌن واحدة
.7البراز ٌحتوى على اشكال كثٌرة جدا للطعام الغٌر المهضوم لدلن البد ان ٌكون عندن الخبره للتفرٌك بٌن
االشكال الطبٌعٌه و البوٌضات.
.8عند الشن فى احتمال وجود بوٌضة ما ال تعرفها اترن العٌنة لغٌرن ٌفحصها او استعٌن بالصور من الكتاب
او الكمبٌوتر واذا لم ٌتوفر ذلن فمن االفضل عدم كتابة البوٌضة المشكون فٌها و اطلب من المرٌض عٌنة
اخرى فى الصباح للفحص مرة اخرى.
.9بعض االعراض المرضٌه على المرٌض تمكنن من التأكد من صحه تحلٌلن و تولع الطفٌل الموجود فى
العٌنه.
لٌس شرطا وجود طفٌلٌات بالبراز لتشخٌص مشكلة المرٌض بل ان احد وسائل التشخٌص هى .11
استبعاد وجود طفٌلٌات لذلن عند الفحص الجٌد جدا للعٌنة و المتأنى و بعد التأكد من عدم وجود بوٌضات
او larvaبها اكتب التحلٌل بدون طفٌلٌات.
Overlapping Zigzag
Cells
RBCs
Appearance:
تظهر كرات الدم الحمراء تحت المٌكروسكوب كخلٌه صغٌره الحجم ( اصغر من خلٌه الصدٌد) و تبدو بغشاء مزدوج
(لكن فى الحمٌمه غشائها لٌس مزدوج و انما ٌظهر هكدا تنٌجه امتالئها بالهٌموجلوبٌن الدى ٌعطى هدا االنطباع).
ملحوظه
وجود دم فى البراز ٌحدث فى الغالب نتٌجةة نزٌةف و ٌظهةر اٌضةا اثنةاء الفحةص الظةاهرى ممةا ٌؤكةد الرؤٌةه
تحت المٌكروسكوب
لون كةرات الةدم الحمةراء تحةت المٌكروسةكوب لةٌس احمةر بةل ال لةون لهةا و فةى بعةض االحٌةان تظهةر تحةت
المٌكروسكوب شوائب حمراء على شكل لطخه او خط طوٌل و هى لٌس دم ابدا بل هى الٌاف من المالبس او
من االكل خاصة البطٌخ و الطماطم.
Normal:
Normally few amounts of RBCs (0-3) are seen under high power field of
microscope.
Counting:
RBCs are counted under HPF of microscope and the range of count is written for
example 10-12 or 30-40 and so on.
Interpretation:
Abnormal RBCs under microscope is seen in:
)Bilharzias (schistosoma mansoni infection
anal fissure
piles
Entameba histolytica infection
PUS
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Appearance:
. تظهر كرات الدم البٌضاء او الصدٌد تحت المٌكروسكوب كخلٌه كبٌره الحجم و بها نواه
ملحوظه
الصدٌد دائما ٌوجد فى تجمعات
فى بعض االحٌان تكون العٌنة كلها صدٌد و دلن فى حاالت االصابات البكتٌرٌه و هنا الحظ انه ال توجد ماده
.برازٌه فال ٌمكن الحكم على حاله الهضم
ابحث دائما عن االماكن التى تظهر بٌضاء بالعٌن المجردة على الشرٌحة النهةا غالبةا مةا تكةون مخةاط و علٌةه
. تجمع من الصدٌد
Normal:
Normally few amounts of pus (0-5) are seen under high power field of microscope.
Counting:
Pus is counted under HPF of microscope and the range of count is written as in
the case of RBCs.
Interpretation:
Abnormal pus under microscope is seen in:
Bacterial infection as Shigellosis , Salmonellosis
Inflammation of the intestines, such as Ulcerative colitis
Note: usually pus is absent or few in cases of Viral diarrheas (Rotavirus infection) and
Parasitic infestations.
Epithelial cells
Appearance:
الخالٌا الطالئٌه ال ٌوجد شكل ممٌز لها فهةى مةن الممكةن ان تكةون دائرٌةه او مخروطٌةه و غٌةر منتظمةه لكةن حجمهةا
.الكبٌر و النواه بداخلها ٌجعل من السهل تمٌٌزها تحت المٌكرسكوب
Normal:
Normally it is few or absent.
Interpretation:
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Epithelial cells increased with gastrointestinal tract irritation
Note: absence of epithelial cells in meconium of newborn may aid in diagnosis of intestinal
obstruction in the newborn.
Meconium is the earliest stools of an infant. Unlike later feces, meconium is composed of
materials ingested during the time the infant spends in the uterus: intestinal epithelial
cells, , mucus, amniotic fluid, bile, and water. Meconium is almost sterile, unlike later
feces, is viscous and sticky like tar, and has no odor.
Digestive state
Starch
Appearance:
ٌظهر من تناول اطعمة مثل الممح و االرز و وله اشكال لٌاسٌة و لكن ال توجد بشكل كبٌر فى الحمٌمة بةل تكةون علةى
.هٌئة شكل عشوائى و تأخذ اللون البنى الممٌز للبراز
Normal:
Normally found in the stool and graded as (+) when seen under HPF.
Interpretation:
Increased starch in stool (++ or +++) indicate a case of indigestion.
Fat
Appearance:
ٌظهةةر مةةن تنةةاول الةةدهون فةةى الطعةةام سةةواء كانةةت مةةن مصةةدر نبةةاتى او مصةةدر حٌةةوانى و لةةه احجةةام مختلفةةة تشةةبه
. الكرستالت تأخذ اللون االصفر البرتمالى
Note: Neutral fat globules can be stained with Sudan stain and counted.
Normal:
Normally found in the stool and graded as (+) when seen under HPF.
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Interpretation:
Increased fat in stool may indicate:
Pancreatic insufficiency as in pancreatitis due to reduced pancreatic juice
secretion.
Disorders affect the absorption of fats as sprue (celiac disease) and infection with
Giardia.
Vegetable cells
Appearance:
.ٌظهر من تناول االطعمة الغنٌة بااللٌاف مثل الخس
الحظ انها تشبه البوٌضات لدلن تحتاج خبرة للتفرٌك بٌنها وبٌن البوٌضات و عامة ادا وجدت الخالٌا الخضةرٌة توجةد
.بكمٌة كبٌرة اكثر بكثٌر من تواجد اى بوٌضات فى العٌنة
Normal:
Normally found in the stool and graded as (+) when seen under HPF.
Interpretation:
Increased vegetable cells in stool have no clinical significance and considered as
residual food.
Muscle fibers
Appearance:
تظهر من تناول اللحوم و شكلها ممٌز وواضح(مثل شكل الحصٌره) و ال تختلط مع اى شةىء اخةر فةى البةراز وٌمكةن
.تمدٌرها بسهولة
ملحوظه
النةه ال ٌأكةل طعةام طبٌعةى و لةدلن تكتةبmuscle fibers اشةهر الٌحتةوى علةى6 براز االطفةال الرضةع فةى اول
. اشهر فٌأكل الطفل بصورة طبٌعٌة وٌكون البراز مثل الشخص العادى6 اما بعدabsent
Normal:
Normally found in the stool and graded as (+) when seen under HPF.
Interpretation:
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Increased muscle fibers in stool have no clinical significance and considered as
residual food.
Note: the presence of large amount of undigested meat fibers in the stool may be caused by
pancreatitis.
Note: Some studies have shown an excellent correlation between excessive fecal muscle
fibers and hypo- or achlorhydria.
Other findings
Note: pathogenic bacteria may be found in stool (such as Salmonella, Shigella, and
Staphylococcus aureus) and this will leads to pus formation, stool culture can differentiate
between pathogenic and non pathogenic bacteria.
Yeast: normally stool contain harmless yeast cells such as blastocystis hominis
Fibers: normally stool contains many fibers that may arise from clothes or
undigested plant food.
Oil droplets: normally found, they are very bright and completely rounded.
Note: some times oil droplets are muddled with cysts but cysts are not so bright and not so
rounded and if you stain the smear with iodine nucleus of cyst will be seen.
Parasites
Definitions:
Medical parasitology:
The study of the parasites of man and their medical consequences
Parasitism:
Is an ecological relationship between two different organisms, one designated the
parasite, the other the host.
Host:
organism harboring a parasite.
Infective stage:
It is a stage in the life cycle of the parasite that can infect human.
Reservoir host:
An animal that harbors a species of parasite that can be transmitted to and infect
man.
Vector:
an arthropod or other living carrier that transports a pathogenic organism from an
infected to a non-infected host.
Carrier:
a host that harbors a parasite but exhibits no clinical signs or symptoms.
Zoonosis:
a disease involving a parasite for which the normal host is an animal, and wherein
man can also be infected.
Helminthes
Trematoda
Schistosoma mansoni
Fasciola gigantica
Cestoidea
Taenia solium
Taenia saginata
Hymenolepis nana
Nematoda
Common questions
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-2ان اإلصابة بالطفٌلٌات سهل جدا ً ألنه ٌأتى عن طرٌك ممارسات غٌر صحٌحة وٌنتج عن تلوث البٌئة ،لذلن نجد
أنه ٌجب :
طهى الطعام ( اللحوم واألسمان ) لولاٌة االنسان من إصابات مثل الدٌدان الشرٌطٌة والتكسوبالزما .
غسل الخضروات جٌدا ً لبل أكلها ( السالطه ) لولاٌة االنسان من اإلصابة بالكثٌر من الدٌدان مثل االسكارس
والبروتوزوا مثل األمٌبا .
حفظ الطعام فى المنازل واألسواق من الذباب الذى ٌحمل أكٌاس األمٌبا .
عدم تلوث مصادر المٌاه بالبراز والبول مما ٌمى االنسان من اإلصابة بالبلهارسٌا أو الدٌدان أو البروتوزوا
توعٌة الناس لمعرفة طرٌمة رعاٌة الحٌوانات األلٌفة صحٌا ً للولاٌة من األمراض الطفٌلٌة التى لد تنتمل من
الحٌوان إلى االنسان .
كذلن الممارسات الصحٌة السلٌمة مثل غسل األٌدى لبل األكل وبعد الخروج من الحمام وكذلن النظافة الشخصٌة
ولاٌة أكٌدة من اإلصابة باألمراض الطفٌلٌة
اٌام ثم تكرر بعد اسبوعٌن ثم بعد شهر وذلن النها تمتل الدٌدان وال تمتل3 فى حالة الدٌدان تؤخذ الجرعة: ملحوظه
.الٌرلات و لذلن تعطى بعد مدة حتى تكون الٌرلات اخرجت الدٌدان
Procedure
1) Weigh 4 g of faeces and place into container.
2) Add 56 ml of saline.
3) Mix the contents thoroughly with a stirring device.
4) Filter the faecal suspension.
5) Take a sample with pasture pipette.
6) Fill the both slides of the counting chamber with the samples
7) Allow the counting chamber to stand for 5 minutes.
8) Examine the sample of the filtrate under a microscope.
9) Count all eggs within the engraved area of both chambers.
10) The number of eggs per gram of faeces can be cal calculated as
follow:
Note: caffeine ingestion increases blood pressure transiently, but is not thought to cause
chronic hypertension. Alcohol can also cause alcoholic liver disease leading to portal
hypertension.
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Hemorrhoids usually are not dangerous or life threatening. In most cases,
hemorrhoidal symptoms will go away within a few days.
Although many people have hemorrhoids, not all experience symptoms.
The most common symptom of internal hemorrhoids is bright red blood
covering the stool.
an internal hemorrhoid may protrude through the anus outside the body,
becoming irritated and painful. This is known as a protruding hemorrhoid.
Symptoms of external hemorrhoids may include painful swelling or a hard lump
around the anus that results when a blood clot forms. This condition is known
as a thrombosed external hemorrhoid.
microcytic hypochromic anemia occurs due to chronic bleeding.
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For many people, hemorrhoids are mild and temporary conditions that heal
spontaneously.
There is no medicine that will cure hemorrhoids, but local treatments such as
topical analgesic, can provide temporary relief.
In the case of external hemorrhoids with a visible lump of small size, the
condition can be improved with warm bath causing the vessels around rectal
region to be relaxed.
Eating fruit helps avoid conditions that lead to hemorrhoids.
Eating fiber-rich bulking agents to help create a softer stool that is easier to
pass,
to lessen the irritation of existing hemorrhoids.
Surgical treatments
Some people require the following medical treatments for chronic or severe
hemorrhoids:
Rubber band ligation
Elastic bands are applied onto an internal hemorrhoid to cut off its
blood supply. Within several days, the withered hemorrhoid is
sloughed off during normal bowel movement.
Hemorrhoidolysis/Galvanic Electrotherapy
Desiccation of the hemorrhoid by electrical current.
Anal fissure
Anal fissure is a crack or tear in the anus skin. As a fissure, these tiny tears may
show as bright red rectal bleeding and cause severe periodic pain after
defecation.
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Most anal fissures are caused by stretching of the anal mucosa beyond its
capability.
Anal fissures are common in women after childbirth; following excessive anal
intercourse; after difficult bowel movements; and in infants following constipation.
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Treating constipation by eating food rich in dietary fiber, avoiding caffeine
(which can increase constipation) , drinking a lot of water and taking stool
softener.
Treating diarrhea.
Avoiding straining or prolonged sitting on the toilet.
Keeping the anus dry and hygienic.
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Treatment included warm baths, topical anesthetics, stool bulking agents,
mechanical anal stretching, and, sometimes, surgery.
Obstructive jaundice
Jaundice, also known as icterus is yellowish discoloration of the skin, sclerae
(whites of the eyes) and mucous membranes caused by hyperbilirubinemia.
Post-hepatic jaundice
The most common causes are gallstones in the common bile duct, and pancreatic
cancer in the head of the pancreas.
Also, a group of parasites known as "liver flukes" live in the common bile duct,
causing obstructive jaundice.
Other causes include strictures of the common bile duct, biliary atresia,
Fat malabsorption
Malabsorption is a state arising from abnormality in digestion or absorption of food
nutrients across the gastrointestinal (GI) tract.
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Bowel mucosal damage
Bacterial and viral infections
Parasites infections e. g. Giardia lamblia
Diarrhea
Diarrhea, also spelled diarrhoea, is frequent loose or liquid bowel movements.
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Diarrhea is most commonly caused by viral infections, parasites or bacterial
toxins.
Diarrhea can also be a symptom of more serious diseases, such as dysentery,
cholera, or botulism, and can also be indicative of a chronic syndrome such as
Crohn's disease.
Diarrhea can also be caused by dairy intake in those who are lactose intolerant.
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Secretory diarrhea
Osmotic diarrhea
Osmotic diarrhea occurs when too much water is drawn into the bowels. This
can be the result of maldigestion (e.g., pancreatic disease or Coeliac disease),
in which the nutrients are left in the lumen to pull in water.
Motility-related diarrhea
Inflammatory diarrhea
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In many cases of diarrhea, replacing lost fluid and salts is the only treatment
needed.
من الخطأ ان ٌأخذ مرٌض االسهال ادوٌة تولف االسهال( اال اذا كان شدٌد جدا ) مباشرة الن ذلن ٌؤخر: ملحوظه
من فرص الشفاء من المرض باحتجاز العامل المسبب فى االمعاء و عدم خروجه
constipation
Constipation, is a condition of the digestive system in which a person experiences
hard feces that are difficult to expel. This usually happens because the colon
absorbs too much water from the food.
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In common constipation, the stool is hard, difficult, and painful to pass.
Usually, there is an infrequent urge to void.
the abdomen may become distended , tender and crampy, occasionally with
enhanced bowel sounds
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Straining to pass stool may cause hemorrhoids and anal fissures, which are
painful in themselves.
Also constipation can lead to headache and testicular varices.
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The main causes of constipation include:
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Prevention:
Drinking orange juice.
Diet rich in fibers as vegetables and fruit and whole meal bread
Increase the intake of fluids (preferably water)
Walking to facilitate bowl movement
No coffee or tea.
Medications:
Laxatives as apilaxin, laxin and cisapride.
spastic colon
In gastroenterology, irritable bowel syndrome (IBS) is a functional bowel disorder
characterized by abdominal pain, discomfort or bloating relieved by defecation and
alteration of bowel habits. Diarrhea or constipation may predominate, or they may
alternate (classified as IBS-D, IBS-C or IBS-A, respectively).
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The primary symptoms of IBS are abdominal pain or discomfort in association with
frequent diarrhea, constipation, or a change in bowel habits.
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Initially, IBS was considered a psychosomatic illness but new evidences show that
organic factors contribute for the disease such as immune reaction and infections.
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Initial treatments
Antispasmodics
The use of antispasmodic drugs (e.g. colospasmine and librax) may help patients,
especially those with cramps or diarrhea.
Crohn's disease
Crohn's disease (also known as regional enteritis) is a chronic, episodic,
inflammatory bowel disease (IBD) and is generally classified as an autoimmune
disease. It can affect any part of the gastrointestinal tract from mouth to anus;
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Many people with Crohn's disease have symptoms for years prior to the diagnosis.
The usual onset is between 15 and 30 years of age but can occur at any age.
Gastrointestinal symptoms
Abdominal pain may be the initial symptom of Crohn's disease. The pain is
commonly cramp-like and may be relieved by defecation. It is often
accompanied by diarrhea, which may or may not be.
Perianal discomfort may also be prominent in Crohn's disease. Itchiness or
pain around the anus may be suggestive of inflammation.
The mouth may be affected by non-healing sores (aphthous ulcers).
Systemic symptoms
Crohn's disease, like many other chronic, inflammatory diseases, can cause a
variety of systemic symptoms.
Inflammation of the interior portion of the eye, known as uveitis, can cause eye
pain, especially when exposed to light.
Inflammation may also involve the white part of the eye (sclera), a condition
called episcleritis. Both episcleritis and uveitis can lead to loss of vision if
untreated.
Crohn's disease is associated with inflammation of one or more joints (arthritis).
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Crohn's disease can lead to several mechanical complications within the
intestines, including obstruction, and abscesses. Obstruction typically occurs from
strictures or adhesions which narrow the lumen, blocking the passage of the
intestinal contents.
Crohn's disease also increases the risk of cancer in the area of inflammation.
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The exact cause of Crohn's disease is unknown. However, environmental and
genetic factors have been invoked in the pathogenesis of the disease.
Mutations in the CARD15 gene (also known as the NOD2 gene) are associated
with Crohn's disease.
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A complete blood count may reveal anemia, which may be caused either by
blood loss or vitamin B12 deficiency. T
Erythrocyte sedimentation rate, or ESR, and C-reactive protein measurements
can also be useful to gauge the degree of inflammation.
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Currently there is no cure for Crohn's disease
Treatment for Crohn's disease is only when symptoms are active and involve first
treating the acute problem, then maintaining remission.
Acute treatment uses medications to treat any infection (normally antibiotics) and
to reduce inflammation (normally aminosalicylate anti-inflammatory drugs and
corticosteroids).
Ulcerative colitis
Ulcerative colitis is a form of inflammatory bowel disease (IBD), a disease of the
intestine, specifically the large intestine or colon, that includes characteristic
ulcers, or open sores, in the colon.
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While the cause of ulcerative colitis is still unknown, several, possibly interrelated,
causes have been suggested:
Genetic factors
Environmental factors
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Many hypotheses have been raised for environmental contributions to the
pathogenesis of ulcerative colitis. Include the following:
Autoimmune disease
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GI symptoms
The clinical presentation of ulcerative colitis depends on the extent of the disease
process.
Patients usually present with diarrhea mixed with blood and mucus, of gradual
onset.
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Standard treatment for ulcerative colitis depends on extent of involvement and
disease severity. The goal is to induce remission initially with medications,
followed by the administration of maintenance medications to prevent a relapse of
the disease.
Drugs used
Aminosalicylates as sulfasalazine
Corticosteroids as prednisone
Immunosuppressive drugs as azathioprine
Surgery
Surgery is indicated for patients with severe colitis or patients with symptoms that
are disabling and do not respond to drugs.
Dyspepsia
Dyspepsia (Indigestion) is a medical condition characterized by indigestion with
chronic or recurrent pain in the upper abdomen, upper abdominal fullness and
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The characteristic symptoms of dyspepsia are upper abdominal pain, bloating,
fullness and tenderness on palpation.
Note: Pain worsened by exertion and associated with nausea and sweating may also indicate
angina.
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over eating
spicy food
too fast eating
alcohol drug eat
stress
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Drugs that facilitate digestion as Amraze or digestin.
Proton pump inhibitors (PPIs), which are effective for the treatment of
heartburn.
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Include chronic diarrhea, failure to thrive (in children), and fatigue.
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The only effective treatment is a lifelong gluten-free diet.
Achlorhydria
Achlorhydria and hypochlorhydria refer to states where the production of gastric
acid in the stomach is absent or low, respectively.
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The decreased acid level itself causes few symptoms, but low acid levels in the
stomach are linked with bacterial overgrowth (as the stomach does not kill
microbes normally present in food), which can manifest as diarrhoea or decreased
absorption of nutrients or vitamins.
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Autoimmune disorders where there is antibody production against parietal cells
which normally produce gastric acid.
The use of antacids or drugs that decrease gastric acid production
A symptom of Helicobacter pylori infection which neutralizes and decreases
secretion of gastric acid to aid its survival in the stomach.