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HYPOIHYROIDISII VSHYPERIHYROIDLI

THYROID DYSFUNCTION TM BALANCE oF THyRO1D HORMoN ES (T. 14


IT CAUSES
HYPOTHYRDI DI SM AND HYPERTHYROIDISM

HPERIAYRODIS
IT
HVPOTHYROIDISN
OCCURS
DOE To EXCESSIVEE OccRS pUE TO INVFFIEIENT
PRODUCTION OF THYRO(D HORMONES PRoDUCTION OF THYRO ID HORMONES

GRAVE'S DiSEASE JS oMoN CAusE HASH IMOTOs PISEASE IS MoN CAUSE


TSH EVEL WILL BE ELEVATED T3H LEVEL WILL BE NoRMAL OR
REDUCE D
CHARACTERISED BY loss oF WE IGHT oAPACTER1SD BY NCREAGED WTIGNT
PEspITE A
INCREASED APETITE DESPaTE A pook ApeT TE
INDICATES 3IGS OF TNCREASED METABOLISM N DICATES sIGNs OF SOW METABOLISM
INCREASED HEART RATE
DELREASED HEART RATE
INCREASED CARDUc coNTRACILTY DECREASE
D ARME ONTRACT)LITY
INCREASED CARDUC CTpUT PECREASED CARDIAC OrpuT

INCREASED RENAL BLOOD LOW


INCRBASEP B-ADRENERGIC RECaPTORS DECREASED RENL BLOOD FLOW

KiPNEY DECRBASED ADRE NERGIe E


N KIDNy
TNCREASED RAAS ACTIVITY
DE CREASEED RMS ACTIwITY
INCREASED FILTRATioN PRESSURE
DECREASED PLTRAON PRESSURE
INCREASED GLOMERULAR HLTRATION RATE
DECREASED GoE RULAR FILTRANN
RATE
TNCREASED TUBLO GLoME RUAR FERDBACK
PECREASED TULOGtaMERAR PRersek
TNCREASE D TUBUAR MASS DELREALED TUBUAR MASS

U5UALLY TREATED wTH CAN BE TREATED WITH


ANTI -
LEVOTHY ROANE
(LFVOTH YROXINE) PARCEUTCAL AGENTS
PADIOTHERApy oR 6RaICAL REMOVL oF THE
TH yRDID GAND

B R.yA IN
Y.SVBHABHRSE Ms8S2yAR)

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