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Communicable Disease Part 2 • Fly control and screening to protect


foods against fly contamination
BACILLARY DYSENTERY (Shigellosis)
• Protection of purified water supplies
• An acute bacterial infection of the
intestine characterized by diarrhea, • Control of infected individual contacts
fever, tenesmus and in severe cases and environment
bloody mucoid stools.
• Isolation of patient during acute illness
• Severe infections are frequent in infants
TYPHOID
and in elderly debilitated persons.
• Systemic infection characterized by
Causative agent:
continued fever, slow pulse,
Shigella, there are four main groups: involvement of lymphoid tissue,
Splenomegaly, rose spots on trunks and
• Shigella sonnei
diarrhea.
• Shigella boydii
• Many mild typical infections are often
• Shigella flexneri unrecognized.

• Shigella dysenteriae Causative agent:

Source of Infection: • Salmonella enterica serotype TYPHI

• Feces of infected persons, many in Source of infection:


apparent mild and unrecognized
• Feces and urine of infected person.
infection
Family contact may be transient carrier.
Mode of transmission: Carrier state is common among person
over 40 years of age especially female.
• Eating of contaminated foods or
drinking contaminated water and by Mode of transmission:
hand to mouth transfer of contaminated
• Direct or indirect contact with patient or
materials; by flies, by objects soiled
carrier. Principal vehicles are food and
with feces of a patient or carrier.
water. Contamination is usually by
Susceptibility, Resistance and Occurrence: hands of carrier. Flies are vectors.

• Disease is more common and more Method of Prevention and Control:


severe in children than in adults
• Same as preventive and control
Methods of prevention and Control: measures as in Dysentery in addition,
immunization with vaccine of high
• Sanitary disposal of human feces antigenicity.
• Sanitary supervision of processing, • Education of the general public and
preparation and serving of food particularly the food handlers.
particularly those eaten raw
• Adequate provision for safe washing
facilities HEPATITIS A (Infectious hepatitis, Epidemic
hepatitis, Catarrhal jaundice)
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• A form of hepatitis occurring either • Proper disposal of urine and feces


sporadically or in epidemics and caused
• Washing hands very well before eating
by virus introduced by focally
and after using the toilet
contaminated water or food.
• Separate and proper cleaning of articles
• Young people especially school children
used by patient.
are most frequently affected
CAUSATIVE AGENT:
PARALYTIC SHELLFISH POISONING
• Hepatitis A virus
• A syndrome of characteristic symptoms
Predisposing factors:
predominantly neurologic which occur
• Poor sanitation within a minute or several hours after
ingestion of poisonous shellfish.
• Contaminated water supplies
Causative agent:
• Unsanitary method of preparing and
serving of food • Single-celled organism called
Dinoflagellates: it is commonly referred
• Malnutrition
as plankton
• Disaster and War time condition
• The organism that causes red tide in the
Signs and Symptoms seas around Manila bay, Smar, Bataan
and Zambales is the Pyromidium
• Influenza-like symptom and headache bahamense var. compressum
• Malaise and easy fatigability
• Anorexia and abdominal discomfort  Mode of Transmission
• Nausea and vomiting • Ingestion of raw or inadequately cooked
• Fever seafood usually bi-valve shellfish or
mollusk during red tide season
• Lymphadenopathy
 Signs and Symptoms
• Jaundice
• Numbness of face especially around the
• Bilirubinemia with clay colored stool mouth
Management: • Vomiting and dizziness
• Prophylaxis- IM injection of gamma • Headache
globulin
• Tingling sensation and eventually
• Complete bed rest paralysis of hands and feet
Prevention and Control: • Floating sensation and weakness
• Ensure safe water or drinking • Rapid pulse
• Sanitary method in preparing, handling • Difficulty of speech (ataxia) and
and serving food difficulty in swallowing (dysphagia)
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• Total muscle paralysis with respiratory Signs and Symptoms:


arrest and death occur in severe cases
1. Leptospiremic phase-
 Management and Control
 Leptospires present in blood and
• No definite medication indicated CSF. Onset of symptoms are
abrupt with fever, headache,
• Drinking pure coconut milk and sodium myalgia, nausea, vomiting,
bicarbonate solution weakened the toxic cough and chest pain
effect of red tide. It is advised to take
these solutions in early stage of 2. Immune phase
poisoning only.
 correlates with the appearance
• Shellfish affected by red tide must not of circulating IgM.
be cooked with vinegar as the Toxin
Treatment
Pyromidium increases when mixed
with acid.  Penicillin
• Toxin of red tide is not totally destroyed  Tetracycline
upon cooking hence consumers must be
educated to avoid bi-valve mollusk such  Erythromycin
as tahong, talaba, halaan, kabiya when
red tide warning has been issued by
proper authority. Prevention and Control
 Improved education of people at particular
risk
LEPTOSPIROSIS
 Use of protective clothing boots and
• (Weil’s disease, Mud fever, Trench gloves especially by worker with
fever, Flood fever, Spiroketal jaundice, occupational hazards
Japanese seven days fever)
 Rats and other potential hosts control
• It is a Worldwide zoonotic disease. Rat
is the main host of the disease although  Investigation of contacts and source of
pigs, cattle, rabbits and other wild infection
animals can also serve as reservoir
hosts.
RABIES (Hydrophobia, Lyssa)
• It is an occupational disease affecting
veterinarians, miners, farmers, sewer,  Is an acute viral encephalomyelitis
workers, abattoir workers, etc. caused by the Rabies virus, a
rhabdovirus of the genus Lyssavirus
Causative agent
 It is fatal once the signs and symptoms
• Leptospira interrogans
appear
Mode of transmission
 There are two kinds urban or canine
• Through contact of skin, especially rabies is transmitted by dogs while
open wounds with water, moist soil or sylvatic rabies is a disease of wild
vegetation contaminated with urine of animals and bats which sometimes
infected host spread to dogs, cats, and livestock
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 Rabies remains a public health problem  The wound must be immediately and
in the Philippines. Approximately 300- thoroughly washed with soap and water.
600 Filipinos die of rabies every year Antiseptic such as povidone iodine or
alcohol may be applied.
 Philippines has the highest prevalence
rate of rabies in the world.  The patient must be given antibiotics and
anti-tetanus immunization
Mode of Transmission:
 Post exposure treatment is given to persons
 Usually by bites of a rabid animal who are exposed to rabies. It consists of
whose saliva has the virus, The virus local wound treatment, active
may also be introduced into a immunization and passive immunization.
scratch or in fresh breaks in the skin
(very rare) transmission from man to  Consult a veterinarian or trained personnel
man is possible. to observe your pet for 14 days for sign of
rabies
Incubation period:
 Be a responsible pet owner
• The usual incubation period is 2 to 8
 Consult for rabies diagnosis and surveillance
weeks. It can be as long as a year or
of the area
several years depending on the severity
of wounds, site of the wound as distance  Mobilize for community participation
from the brain, amount of virus
introduced and protection provided by
clothing. NATIONAL RABIES PREVENTION AND
CONTROL PROGRAM

 Susceptibility and Resistance • Goal: Human rabies is eliminated in the


Philippines and the country is declared
 All warm blooded mammals are rabies-free.
susceptible. Natural immunity in man is
unknown General objectives:

 Signs and Symptoms • To reduce the incidence of human rabies


from 7 per million to 1 per million
 Headache population by 2010 and eliminate
human rabies by 2015
 Fever
• To reduce the incidence of canine rabies
 Sensory change near the site of animal bite
from 70 per 100,000 to 7 per 100,000
 Spasm of muscles or deglutition on dog by 2010, and eliminate canine
attempt to swallow (fear of water) rabies by 2015.

 Paralysis
 Delirium and convulsion SCABIES

 Without medical intervention, the rabies • A communicable disease of the skin


victim would usually last only 2 to 6 days. characterized by eruptive lesions
Death is often due to respiratory paralysis produced by the burrowing of the female
parasite into the skin.
 Management and Prevention
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Causative organism  Treatment is limited entirely to


the skin
• Sarcoptes scabiei, itch mite. The female
parasite is easily visible with a  Benzyl benzoate emulsion to
magnifying glass. Burrows beneath the clean the lesion and it has a
epidermis to lay her eggs, and set up an more rapid effect
intense irritation.
Prevention and Control
Mode of transmission:
 Good personal hygiene- daily bath,
• The disease is transmitted by direct washing of hands before and after eating
contact with infected individuals or their and after using the toilet, cutting of
clothing and bedding, fingernails
 Predisposing factors  Regular changing of clean clothing
beddings and towel
 Close crowding as in tenement
districts  Eating the right kind of food and taking
plenty of fluids
 Lack a personal cleanliness
 Keeping the house clean
 Signs and Symptoms
 Improving sanitation of the surroundings
• Itching
• Minor discomfort-skin may feel hot and
burning ANTHRAX
• When large areas are involve and  An acute bacterial disease usually affecting
secondary infection is severe, there will the skin but which may vary rarely involves
be fever, headache and malaise the oropharynx. Lower respiratory tract,
mediastinum or intestinal tract.
• Secondary dermatitis is common
 ac agent
 Diagnosis
 Bacillus anthracis
 Appearance of the lesion, and
the intense itching and finding  Mode of transmission
the causative mite
 Cutaneous infection is by contact
 Scraping from its burrow with a with tissues of animals (cattle,
hypodermic needle or curette, sheep, goats, horses, pigs) dying of
and then examined under low the disease
power objective or hard lens.
 Possibly by biting flies that had
 Treatment partially fed on such animals;
contaminated hair, wool, hides or
 The whole family must be products made from them such as
examined before undertaking drums or brushes; contact with soil
treatment, as long as one associated with infected animals.
member of the family remains
infected, other members will get Signs and Symptoms:
the disease
 Cutaneous anthrax
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 Most common and is contracted


by contact with infected animals
MENINGOCOCCEMIA
 The exposed part of the skin
 The disease is usually sporadic (cases
begins to itch and a papule
occur alone or may affect household
appears in the inoculation site
members with intimate contact)
 This papule becomes a vesicle
 Although primarily a disease of
and then evolves into depressed
children, It may occur among adult
black eschars
especially in condition of forced
 The lesion is not painful and overcrowding such as institution, jail
often intreated which will result and barracks.
to septicemia and death when
 There is an increase rate in smokers,
not treated early.
overcrowded households and military
 Pulmonary anthrax recruits.
 Become acute, with fever, shock Causative agent
and death results
 Neisseria meningitidis
 Contracted by inhalation of
Source of infection
spores of Bacilllus anthracis
 Respiratory droplets from nose and
 At the onset of illness, the
throat of infected persons
symptoms are mild and
resemble that of common upper Mode of transmission
respiratory tract infection
 Direct contact with respiratory
 Gastrointestinal anthrax droplets from nose and throat of
infected persons. Carrier may exist
 Contracted by ingestion of meat
without cases of meningitis.
from infected animals and is
manifested as violent
gastroenteritis with vomiting
and bloody stools  Signs and Symptoms

 Methods of Control  High grade fever for first 24 hours

 Immunize high-risk persons  Weakness, joint and muscle pain

 Educate employees handling  Hemorrhagic rash, progressing from


potentially contaminated articles few petichiae to widespread purpura
about modes of anthrax  Headache, nausea and vomiting, stiff
transmission neck, seizure or convulsion and
 Personal cleanliness sensorial changes

 Control dusts and proper  Methods of Prevention and Control


ventilation in hazardous  Respiratory isolation of patients for the
industries especially those that first 24 hours upon admission to
handle raw animal materials prevent exposure of hospital staff.
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 Respiratory clothing for hospital staff  Yearly vaccination of poultry


with patient suspected of workers with regular of periodic
meningococcemia direct contact with poultry.
 Public should be educated to avoid
overcrowded placed to reduce the risk
SARS (Severe Acute Respiratory Syndrome)
of exposure particularly among children
and elderly.  It is a newly recognized form of a
typical pneumonia that had been
described in patient in Asia, North
“BIRD FLU” or AVIAN INFLUENZA America and Europe. The earliest
known cases were identified in
 It is an infectious disease of birds
Guangdong Province, China in
ranging from mild to severe form of
November 2002
illness. All birds are thought to be
susceptible to infection with avian  The WHO issued the global alert on the
influenza, though some species are more outbreak on March 2003 and instituted
resistant than others worldwide surveillance.
 Some forms of bird flu infections can Causative agent
cause illness to humans. It is due to
 It is a novel human corona virus based on
highly pathogenic influenza virus H5N1.
serological and molecular tests done on
It is the subtype that can be transmitted
specimens from SARS patients
from infected poultry to human.
Mode of Transmission
 Source of Infection
 Close contact with respiratory droplet
 Viruses that normally infect only
secretion from SARS patient
birds and less commonly pigs
 Transmission occurs when another
 Signs and Symptoms
person’s mucous membrane are exposed to
 Fever droplet secretions when a SARS patient
coughs, sneezes or talks.
 Body weakness and muscle pain
 Cough
FILARIASIS
 Sore throat
 Public health concern in many endemic
 May have difficulty in breathing in
areas ( 45 out of 78 provinces are
severe cases
endemic)
 Sore eyes
 Chronic parasitic infection caused by
 Preventive Measure in Human nematode parasite

 All workers directly or indirectly  Wuchereria bancrofti


involved in handling live poultry
 Brugia malayi and/or Brugia timori
should be given the curren t season’s
influenza vaccine to reduce the  Loa loa
possibility of dual infection with
Mode of Transmission
human and influenza viruses.
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 The disease is transmitted to a person  Thick smear about the size of


through bites from an infected female 25 centavo coin, blood is
mosquito. taken from the patient 10pm
to 2 am for Wuchereria and
Vectors
anytime Brugia but more
 Wuchereria bancrofti preferred at night (due to
parasite periodicity)
 Aedes poecilus, Anopheles
minismus flavirostris  Treatment

 Brugia malayi • The drug of choice is


Diethylcarbamazine (DEC)
 Mansoni bonnaea, Mansoni
uniformis
 Asymptomatic stage  Supportive care for Filariasis

 Characterized by the presence of  Patients are advised to observe


microfilariae in the peripheral blood personal hygiene by washing the
affected area with soap and water
 No clinical signs and symptoms of twice a day or prescribe antibiotics or
the disease anti-fungal for super infection.
 Acute stage  Prevention and Control
 Lympadenitis- inflammation of the  A. Measures aimed to control the
lymph nodes vector
 Lymphangitis- inflammation of  Environmental sanitation such as
lymph vessels in some cases, the proper drainage and cleanliness of
male genitalia is affected. the surroundings
 Use of insecticides.
 Chronic stage
• Developed 10-15 years from the  B. Measures aimed to protect the
onset of first attack people in endemic areas
• Hydrocoele- swelling of the scrotum • Use of mosquito nets
• Lymphedema- swelling of the upper • Application of insect repellants
and lower extremities
• Screening of houses
• Elephantiasis- enlargement or
thickening of the skin of the lower • Health education
and/or upper extremities, scrotum,
MALARIA
breast
 Continue to be a major Public health
 Laboratory examinations
concern having an annual parasite
• Blood examination incidence of 5.1 per 1000 population
Infectious agents
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 Plasmodium falciparum starting from 1-2 weeks before


entering endemic areas.
 Plasmodium vivax
 Plasmodium ovale
Prevention and Control
 Plasmodium malariae
 Sustainable preventive and vector
 The severity and characteristic
control
manifestation of the disease are govern
by the infecting species  Insecticide-treatment of mosquito nets
Mode of Transmission  House spraying
 Through the bite of an infected female  Protective clothing
mosquito or directly from one person to
 Educate the people on endemic areas
another by passage of blood containing
erythrocytic parasites  Chemoprophylaxis
Signs and Symptoms Etiologic agent
 Recurrent chills  Dengue virus types 1,2,3, and 4
 Fever Source of Infection
 Profuse sweating  Immediate source is a vector mosquito,
the Aedes aegypti or the common
 Anemia
household mosquito.
 Hepatomegaly
Mode of transmission
 Splenomegaly
 Through bite of infected female
Laboratory Examinations mosquito
 Thick and Thin blood smear- look for the Signs and Symptoms
presence of malarial parasite
 An acute febrile infection of sudden
 Para Sight F Test- dipstick test for simple onset with clinical manifestation of 3
and rapid diagnosis of Plasmodium stages.
falciparum
 First 4 days- invasive stage starts
 Serological test- IHA (indirect abruptly as high fever, abdominal pain
hemagglutination test), ELISA and headache
Treatment  4th to 7th days- toxic or hemorrhagic
stage-lowering of temperature, severe
 Drug of choice is Chloroquine.
abdominal pain, vomiting, frequent
Pyrimethanime/Sulfadoxine combination
bleeding, death may occur
may be used in areas with high levels of
resistance to Chloroquine  7th to 10th day- convalescent or recovery
 Chemoprophylaxis Diagnostic test
 Only Chloroquine should be given.  Torniquet test (Rumple Leede Capillary
It must be taken at weekly interval Fragility Test)
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 Platelet count d. Avoid certain sexual practices involving anal


or fecal contact.
Supportive and Symptomatic treatment
2. Barrier methods of contraception (use of
 Paracetamol, analgesic for fever, muscle
condoms, diaphragms and spermicides)
pain or headache.
3. Prophylactic antibiotics taken before and after
 DON’T GIVE ASPIRIN
exposure should not be done because:
 Rapid replacement of body fluids
a.. No single antibiotic covers all potential STIs
Control measures
b. Allergic reaction may occur
 Vector elimination
c. They may lead to emergence or resistant
 Changing water and scrubbing sides of organism
lover vases once a week
 Destroy breeding places of mosquito
I. BACTERIAL STI
 Keep water container clean and covered
A. Gonorrhea
 Avoid too many hanging clothes inside
Causative agent
the house
 Neisseria gonorrheae
 Spray with insecticides
Mode of transmission
 Health education of the public
• Sexual contact in adult, transmission in
neonates is during birth
SEXUALLY TRANSMITTED INFECTIONS
 Sexually Transmitted Infection (STI)
Signs and Symptoms
and their complications belong to the top
five categories for which adults seek • Genital (penis or cervix), anus, throat
health care in the developing countries. and eyes can be infected
 Many STIs can be treated and cured o Males- burning urination and pus
relatively easily and cheaply if discharges from infection of urethra
diagnosed early enough. From among
the STI, the most common treatable are: o Females- vaginal discharge
Chlamydia, Gonorrhea, Trichomoniasis, Treatment
and Syphilis
Cefriaxone for uncomplicated cases. If resistant,
PRIMARY PREVENTION OF STI spectinomycin is used
1. Modification of sexual activity – this would Prevention
decrease the like hood of exposure to or contact
with infectious agents No drugs or vaccine. Condom offer protection.
Trace contacts and treat to interrupt
a. Engage in mutually monogamous relationship transmission.
b. Limit the number of sexual partners B. Syphilis
c. Inspect and question new partners
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Causative agent  In females


 Treponema pallidum o slight vaginal discharge, itching and
burning sensation of vagina, painful
Signs and Symptoms
intercourse, abdominal pain.
 Primary syphilis
Treatment
o (Chancre)painless sore at the site of
 Tetracycline and Erythromycin
entry of germs, swollen glands
Prevention
 Secondary syphilis
 No vaccine available. Erythromycin is
o (condylomata lata)- maculopapular effective in infected mother to prevent
rash notably on the palms and soles, neonatal disease.
or as moist papules on the skin and
mucous membranes. Moist lesion of II. PARASITIC STI
the genitals
A. Trichomoniasis
 Tertiary syphilis
Causative agent
o (Gummas)- varies from no
 Trichomonas vaginalis
symptoms to indication of damage
to body organs such as brains, heart Mode of transmission
and liver
 Usually passed by direct sexual contact.
Treatment Can be transmitted through contact with
wet objects such as towels, wash
 Penicillin is effective in all stages of
clothes, etc.
syphilis
Signs and Symptoms
Prevention
 Males
 Same as other bacterial STI. No vaccine
available. Benzathine penicillin s given • slight itching of penis, painful
to contacts. urination, clear discharge from penis
C. Chlamydia  Females
Causative agent • white or greenish-yellow odorous
discharge, vaginal itching, painful
 Chlamydia trachomatis
urination
Mode of Transmission
Treatment
 Through sexual contact and birth
 Metronidazole for both partners
Signs and Symptoms
Prevention
 In males
 Condoms limit transmission
o discharge from penis, burning and
III. VIRAL STI
itching of the urethral opening,
burning sensation during urination A. AIDS (Acquire Immunodeficiency
Syndrome)
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 The first case of AIDS in the Philippines • Herpes simplex Virus type 2 (HSV2)
was reported in 1984. As of May 2000
Mode of transmission:
based on the Philippine National AIDS
council record, there were 1,385 HIV • Sexual contact in adult and during
positive and AIDS cases, 206 deaths. passage through the birth canal in
neonates
Causative agent:
Signs and Symptoms
 HIV (Human Immunodeficiency Virus)
• Painful vesicular lesion of the male and
Mode of Transmission
female genitals and anal area.
 Sexual contact, Transfer of body fluids
Treatment
(blood, semen, discharges), contaminated
syringe, transplacental • Acyclovir is useful in the treatment of
both primary and recurrent diseases.
Signs and Symptoms
Prevention
 Physical
• Primary disease can be prevented by
 Rashes
protection from exposure to vesicular
 Loss of appetite lesion. There is no vaccine.
 Weight loss C . Hepatitis B
 Fever of unknown origin Causative agent:- Hepatitis B Virus (HBV)
 Pneumocystis carinii pneumonia Mode of Transmission:
(Pneumocystis jiroveci)
• Sexual intercourse, also transmitted
 Persistent diarrhea through blood and during birth
 Esophageal candidiasis Signs and Symptoms:
 Kaposi’s Darcoma (skin cancer) a. Loss of appetite
Treatment b. Easy fatigability
 Azidothymidine (AZT) and Didanosine c. Joint and muscle pain
inhibit HIV replication by inhibiting
d. Low grade fever
reverse transcriptase. Clinical
improvement occurs but the virus persists. e. Right sided abdominal pain
Treatment of opportunistic infection
depends on the organism f. Jaundice

Prevention g. Dark colored urine

 Screening of blood prior to transfusion, Treatment:


safe sex. There is no vaccine • No specific treatment
B. Genital herpes Prevention:
Causative agent a. Vaccine that contains HBsAg as the
immunogen
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b. Hyper immune serum globin obtained from


donors with high titers of HBsAb
c. Education of chronic carriers regarding
precautions

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