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Journal of Family Psychology

© 2023 American Psychological Association 2023, Vol. 37, No. 5, 720–730


ISSN: 0893-3200 https://doi.org/10.1037/fam0001100

Continuity in Hostile Family Relationships Mediated by Family


Psychopathology: An Application of Systems Perspective
Tae Kyoung Lee1, Kandauda A. S. Wickrama2, and Catherine Walker O’Neal2
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1
Department of Child Psychology and Education/Social Innovation Convergence Program, Sungkyunkwan University
2
Department of Human Development and Family Science, University of Georgia

Grounded in the family systems theory within the life course systems perspective, this study sought to
investigate (a) long-term transmission processes of hostility from interparental relationships to parent–
young adult relationships and (b) the mediational roles of parents’ and adolescents’ psychopathology in
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these long-term processes. Research has examined the long-term transmission of hostility from interparental
relationships to young adults’ relationships with their parents. However, less is known about how this
transmission process differs for father–young adult and mother–young adult relationships and if psycho-
pathology is a mechanism that contributes to the transmission of hostility. Utilizing a structural equation
model and prospective, longitudinal data from couples in enduring marriages and their offspring (n = 345
families), we examined long-term associations between interparental hostility when offspring were
adolescents and young adults (1990 and 2001), adolescents’ hostile relationships with their fathers and
mothers (1991), and young adults’ hostile relationships with their fathers and mothers (2003) as well as the
mediating roles of fathers’, mothers’, and adolescents’ psychopathology (1992 and 1994). Interparental
hostility was more strongly linked to hostility between mothers (rather than fathers) and their young adult
offspring. Fathers’, mothers’, and adolescents’ psychopathology uniquely mediated the transmission
processes. This study elucidated transmission processes of interparental hostility to young adults’
relationship hostility with their parents and offers insights into the unique mediational roles of each family
member’s psychopathology.

Keywords: hostile family relationships, transmission, psychopathology, adolescence, young adulthood

Supplemental materials: https://doi.org/10.1037/fam0001100.supp

Interparental relationships are an important family context that hostility into later life stages, such as young adulthood, and for both
influence offspring’s subsequent relationships (e.g., Conger, et al., father–young adult and mother–young adult relationships.
1994). In particular, previous studies suggest that hostility from Studies suggest that interparental hostility and parent–adolescent
interparental relationships is transmitted to offspring’s relation- hostility (hereafter referred to together as hostile family relation-
ships with their parents (Bryant et al., 2017; Smith, et al., 2019). ships) increase both parents’ and adolescents’ psychological dis-
However, studies have mainly focused on the associations between tress (e.g., Bryant, et al., 2017). Other studies suggest that family
interparental hostility and children’s or early adolescents’ hostile members’ psychological distress reinforces family relational con-
relationships (Mark & Pike, 2017), which limits understanding of flicts (e.g., Constantine & Flores, 2006). Thus, in the longitudinal
more long-term transmissions of hostile relationships. Further, contexts of family relationships, there may be reciprocal associa-
given that multiple family dyads exist within a family (e.g., father– tions between hostile family relationships and family members’
mother, father–offspring, and mother–offspring), more research is psychological distress, which warrant an examination of family
needed to examine dynamic transmission processes of interparental members’ psychopathology as a dynamic mediational process

This article was published Online First May 15, 2023. funding was received for the publication of this article.
Tae Kyoung Lee https://orcid.org/0000-0001-8712-3182 Informed consent was obtained from all individual participants included in
The authors declare that they have no conflict of interest. There are also no the study.
financial disclosures to report. Data are proprietary but can be requested from This study was not preregistered. Data presented in the article have not
Tae Kyoung Lee. been disseminated previously (including conferences or shared online). Data,
This research was supported by a grant from the National Institute of Mental study materials, and study analysis code are available from Tae Kyoung Lee
Health (MH00567, MH19734, MH43270, MH59355, MH62989, MH48165, upon reasonable request.
MH051361); the National Institute on Drug Abuse (DA05347); the National Correspondence concerning this article should be addressed to Tae
Institute of Child Health and Human Development (HD027724, HD051746, Kyoung Lee, Department of Child Psychology and Education/Social
HD047573, HD064687); the Bureau of Maternal and Child Health (MCJ Innovation Convergence Program, Sungkyunkwan University, 32415,
−109572); and the MacArthur Foundation Research Network on Successful Dasan Hall of Economics, 25-2 Sungkyunkwan-ro, Jongno-gu, Seoul
Adolescent Development Among Youth in High-Risk Settings. However, no 03063, Republic of Korea. Email: ltk501@skku.edu

720
CONTINUITY IN HOSTILE FAMILY RELATIONSHIPS 721

responsible for the long-term transmission of hostile relationships prospective data from 345 families over 13 years. Figure 1 presents
across multiple family dyads. the theoretical framework.
The present study was grounded in the family systems theory As shown in Figure 1, the long-term transmission (T) of hostile
within the life course perspective. Family systems theory (Cox & family relationships to young adults’ hostile relationships with
Paley, 1997) conceptualizes the family as hierarchical (i.e., where parents may occur indirectly through three possible pathways: (a)
the broader family system is comprised of dyadic subsystems) and continuity in interparental hostility (C1) and transmission of inter-
dynamic (e.g., interdependence within and across dyadic subsys- parental hostility to parent–young adult hostility (T3), (b) transmis-
tems). Further, recent family research extends the family systems sion of interparental hostility to parent–adolescent hostility (T1) and
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theory by locating it within the life course perspective, termed the continuity in parent–adolescent hostility from adolescence into
life course systems perspective (Wickrama et al., 2020). The young adulthood (C2), and (c) sequential transmission of interpar-
perspective emphasizes long-term, dynamic processes of family ental hostility to parent–adolescent hostility (T1), transmission of
relationships within the family system over time (Wickrama et al., parent–adolescent hostility to interparental hostility (T2), and trans-
2020). Further, the life course perspective emphasizes the impor- mission of interparental hostility to parent–young adult hostility
tance of individuals’ social relationships in early life stages, which (T3). Further, these three transmission pathways may be mediated
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can influence one’s roles and social relationships in subsequent by parents’ and adolescents’ psychopathology (see grayed-colored
developmental stages ( Wickrama et al., 2020). However, previous paths in Figure 1). We first describe previous research on the
studies have typically focused on dynamics within the family transmission of hostile family relationships followed by research
subsystems at a single life stage (e.g., when their offspring is in supporting psychopathology as a linking mechanism in these trans-
adolescence; Jager, et al., 2014). Less is known about how in- mission processes.
dividuals’ roles and relationships within the family context change
as offspring transition from adolescence to young adulthood. In Transmission Processes of Hostility From
particular, more studies are needed to examine how hostile family
Interparental Relationships to Young Adults’
relationships unfold over time. In the present study, we examine (a)
Relationships With Parents
the long-term transmission of hostility from interparental relation-
ships to young adults’ relationships with their fathers and mothers Past studies provide consistent evidence of the transmission of
over adolescence and young adulthood and (b) the roles of parents’ hostility from interparental relationships to parent–offspring rela-
and adolescents’ psychopathology as mediators in the transmission tionships (Amato & Booth, 2001; Smith, et al., 2019). For example,
of hostility in family relationships over time. The present study uses one study suggests that hostile couple relationships are emotionally

Figure 1
Hypothesized Transmission Processes of Hostility From Interparental Relationships to Young Adults’ Relationships With
Their Parents
Adolescence Young adulthood

Marital Interparental Interparental


Continuity in interparental hostility (C1)
subsystem Hostility Hostility

Long-term
Transmission (T)
Family members’
psychopathology

Transmission of interparental hostility to Psy Psy Transmission of interparental hostility to


parent-adolescent hostility (T1) (F) (M)
parent-young adult hostility (T3)

Psy
(A)

Transmission of parent-
adolescent hostility to
interparental hostility (T2)

Parent- Adolescents’ Hostile Young Adults’ Hostile


adolescent Relationships With Parents Relationships With Parents
Continuity in parent-adolescent hostility (C2)
subsystem (Fathers & Mothers) (Fathers & Mothers)

Note. Psy = Psychopathology. The subscripts (F), (M), and (A) represent father, mother, and adolescent, respectively. Gray paths reflect
mediation processes of psychopathology on the transmission processes of hostile family relationships.
722 LEE, WICKRAMA, AND O’NEAL

draining for parents and erode their sensitivity and attentiveness more responsive and provided more emotional reassurance to their
toward adolescents, which increases conflict with their adolescents children even during periods of heightened interparental conflict
(e.g., Bryant, et al., 2017). According to family systems theory, this (Cox et al., 2001). In contrast, other research demonstrated that, in
transmission reflects how the couple subsystems affect the parent– stressful marital situations, fathers were more likely to withdraw
adolescent subsystems. Further, research suggests that interparental emotionally from their children (e.g., Smith, et al., 2019). Adoles-
relationships continue to play a significant role in the development cents may interpret this withdrawal as dismissal or abandonment,
of the parent–offspring relationship into the child’s early adult years resulting in strained relationships between fathers and adolescents
(e.g., Amato & Booth, 2001; Johnson & Galambos, 2014). This is (Lewis & Lamb, 2003). Given that adolescence is a sensitive period
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consistent with the life course systems perspective in recognizing for building individual relationships with each parent (Branje, 2018),
how dyadic subsystems may affect each other and how these effects the transmission processes linking interparental hostility to adoles-
may unfold over time. Thus, the transmission of interparental cents’ hostile relationships may be distinct for relationships with
hostility to parent–offspring hostile relationships is not expected fathers and mothers. Furthermore, these distinctions in adolescence
to be age-dependent and may persist past adolescence and into may contribute to differential consequences for hostility in the
young adulthood, particularly if couples consistently experience parent–young adult relationships with fathers and mothers (see
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interparental conflict during these years. corresponding pathway T1 and C2 in Figure 1). Given that enduring
These ideas align with the enduring dynamics model (Caughlin & couples represent a stable dyadic system (Wickrama et al., 2020), it is
Huston, 2006), which emphasizes relationship continuity of the also possible that interparental hostility occurring when their off-
couple subsystem over time. The enduring dynamics model posits spring is an adolescent may be stable until the offspring transitions to
that a spouse who brings negative behaviors and beliefs into the young adulthood, which, in turn, may create changes in the hostility
marriage is unlikely to change their behaviors and beliefs over time, present in young adults’ relationships with their parents (see corre-
resulting in relatively stable (negative) levels of marital attributes sponding pathway C1 and T3 in Figure 1). It is also possible that
over time. These findings suggest continuity in interparental hostil- these types of sequential processes may vary across adolescents’
ity over time. Further, past research suggests that interparental relationships with fathers and mothers (see corresponding pathway
problems in young adulthood influence young adults’ relationship T1, T2, and T3 in Figure 1). We aim to examine whether and to what
problems (e.g., M. Cui, et al., 2008). Therefore, the transmission extent three possible transmission pathways occur within- and across
process of hostile family relationships may occur through C1 and T3 three dyadic subsystems (couple, father–offspring, and mother–
pathways in Figure 1. offspring dyads).
Another possible explanation for the transmission of hostile family
relationships may be that interparental hostility in adolescence can
have a proximal influence on adolescents’ hostile relationships with The Mediating Roles of Psychopathology: Linking
their parents (e.g., Smith, et al., 2019). Similar to the couple dyadic Transmission Processes Underlying Interparental
subsystem, poor-quality parent–adolescent relationships tend to expe- Hostility to Young Adults’ Hostile Relationships
rience similar relational difficulties over time (Branje, 2018). There- With Parents
fore, when interparental hostility results in a hostile parent–adolescent
In addition to recognizing how dyadic subsystems can influence
relationship, hostility in the parent–offspring relationship may con-
one another, family systems theory (Cox & Paley, 1997) also
tinue into young adulthood (see corresponding pathway T1 and C2 in
emphasizes how individual family members’ functioning can be
Figure 1). Last, sequential processes of hostile family relationships
affected by, and affect, dyadic subsystems. In the present context,
are also possible. Research suggests that negativity in the parent–
hostile family relationships (dyadic subsystem) may reciprocally
offspring relationship can shape parents’ hostility (Newland et al.,
influence a family member’s, affective symptoms ( functioning).
2015). Following this research, parent–adolescent hostile relation-
The focus of the present study is on the role of family members’
ships may reinforce interparental hostility in young adulthood, which,
general psychopathology in the context of hostile family relation-
in turn, may predict hostile parent–young adult relationships (see
ships. According to the common mental disorder perspective (South
corresponding pathway T1, T2, and T3 in Figure 1).
et al., 2011), comorbidity among various psychological symptoms
reflects underlying general psychopathology (hereafter referred as to
Dynamic Transmission Processes Linking psychopathology). Consistent with this perspective, studies have
Interparental Hostility to Young Adults’ Hostile reported strong associations among multiple specific affective symp-
toms, such as depressive symptoms and anxiety in both adolescence
Relationships With Their Fathers and Mothers
(Lee et al., 2017) and middle adulthood (South et al., 2011).
Given that multiple dyadic subsystems exist within the family: (a) Further, studies reported the positive associations between hostile
couple dyad, (b) father–offspring dyad, and (c) mother–offspring family relationships and family members’ psychopathology. For
dyad, long-term transmission of hostile family relationships may example, interparental hostility has been shown to influence the
occur through unique dyadic processes (e.g., Jager, et al., 2014). For psychopathology of both fathers and mothers (South et al., 2011)
example, recent studies emphasized the unique transmission pro- and their adolescents (Smith et al., 2019). In turn, when one, or both,
cesses of interparental hostility to both mothers’ and fathers’ relation- parents have high levels of psychopathology, interparental hostility
ships with their offspring (e.g., Smith, et al., 2019). Relatedly, some is also often high (Woods et al., 2019). Similarly, hostility in the
research has noted gender differences in the degree and manner to parent–adolescent relationship can result in increases in both the
which interparental conflict spills over to the parent-offspring rela- parent’s and adolescent’s psychopathology (Lee et al., 2017). In
tionship. In one study, compared to fathers, mothers were generally turn, parents who have high levels of psychological distress may not
CONTINUITY IN HOSTILE FAMILY RELATIONSHIPS 723

provide sufficient emotional support, which is critical for the • Sequential processes involving adolescents’ hostile rela-
formation, and continuity, of positive parent–adolescent relation- tionships with their fathers and mothers and interparental
ships (Kamis, 2021). Adolescents with high levels of psychological hostility (paths T1, T2, and T3).
distress are at risk for interpersonal stressors (X. J. Cui & Vaillant,
1997), which can contribute to hostile relationships with parents Hypothesis 3: Fathers’, mothers’, and adolescents’ psychopa-
(e.g., Pineda, et al., 2007). thology will mediate the long-term transmission processes of
As shown in Figure 1, these mediation processes between hostile interparental hostility in adolescence on hostility in young
family relationships and psychopathology may involve three possi- adults’ relationships with their fathers and mothers. Including
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ble transmission pathways depicted by (a) C1 and T3 pathways, (b) transmission through:
T1 and C2 pathways, and (c) T1, T2, and T3 pathways. For example,
• Interparental hostility in young adulthood (paths C1 and T3).
interparental hostility may negatively impact both fathers’ and
mothers’ psychopathology. In turn, their psychopathology may result • Adolescents’ hostile relationships with their fathers and
in more interparental hostility, which may in turn cause more hostility mothers (paths T1 and C2).
in parent–young adult relationships. As another example, adoles-
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cents’ hostile relationships with fathers and/or mothers (transmitted • Sequential processes involving adolescents’ hostile rela-
from interparental hostility) may influence fathers’, mothers’, and/or tionships with their fathers and mothers and interparental
adolescents’ psychopathology, which, in turn, may result in more hostility (paths T1, T2, and T3).
hostility in young adults’ relationships with their parents. Further,
these mediation processes may be occurred in the context of the Method
dynamics (interdependencies) of three dyadic subsystems (i.e., cou-
ple dyad, father–offspring dyad, and mother–offspring dyad) (e.g., The data used to evaluate the hypotheses are from the Family
interparental hostility → father-adolescent hostility → adolescent’s Transitions Project (FTP). The FTP originated from the Iowa Youth
psychopathology → mother–young adult hostility). In this manner, and Families Project (IYFP), which studied target adolescents and
the present investigation illustrates the dynamic web of associations their families in north-central Iowa in 1989 (Wave 1), 1990 (Wave 2),
that can exist between family dyadic subsystems and members 1991 (Wave 3), 1992 (Wave 4), and 1994 (Wave 6) and then
comprising various subsystems. continued in 2001 (Wave 13) and 2003 (Wave 15). Both parent
and adolescent data were collected in 1989, 1990, 1991, 1992, 1994,
and 2001. However, only adolescent data were collected in 2003.
Families selected to participate in the study included a target seventh-
The Present Study grade child who lived with his or her two biological parents and had a
sibling within 4 years of the target child’s age (target adolescents’
Figure 1 depicts the specific hypotheses of the present study.
mean age = 12.7 years in 1989 (Wave 1), 53% female). For both
Based on the family systems theory (Cox & Paley, 1997) within
projects, trained field interviewers visited the families in their homes.
the life course systems perspective (Wickrama et al., 2020), the
During the visit, a trained interviewer asked each family member to
present study was directed at understanding (a) long-term trans-
complete a detailed questionnaire about family life, work, finances,
mission processes hostility from interparental relationships to
friends, and mental and physical health status. Family members
young adults’ relationships with fathers and mothers and (b)
completed the questionnaires independently so that they could not
the mediational roles of parents’ and adolescents’ psychopathol-
see each other’s answers. Additional information on the study
ogy in these long-term processes. Given that adolescents’ hostile procedures is available from Conger and Conger (2004). The project
relationships with parents have been shown to vary depending on was approved by the university ethics committee. We report how we
parents’ socioeconomic characteristics and adolescents’ gender determined our sample size, all data exclusions, all manipulations,
(e.g., Lee, et al., 2017), family income, parents’ education levels, and all measures in the study.
and adolescents’ gender were included as covariates in the analy-
ses. We hypothesized:
Participants
Hypothesis 1: Long-term transmission processes will link This study is a prospective, longitudinal study of 451 White
interparental hostility when their offspring is an adolescent European American families from eight counties in rural Iowa. At
to hostility in young adults’ relationships with their fathers and the first wave of data collection, the median ages were 39 and 34 for
mothers (path T). fathers and mothers, respectively. The median number of years of
education for fathers and mothers was 13 years, and couples had
been married for at least 17 years in 1989 (Wave 1). The median
Hypothesis 2: Dynamic pathways will emerge in the long-term yearly income in 1989 was $22,000 and $10,000 for fathers and
transmission processes of interparental hostility in adolescence mothers, respectively. Almost all of the fathers were employed
on hostility in young adults’ relationships with their fathers and (99.6%), and 66% of the mothers were employed. The attrition rate
mothers. Including transmission through: was 11% across the 14-year period. The final sample for this study
• Interparental hostility in young adulthood (paths C1 and T3). came from 345 families with parents who were consistently married
(since 1989) and continued to participate through 2001 (Wave 13)
• Adolescents’ hostile relationships with their fathers and where the average ages of fathers and mothers, 52 and 50, respec-
mothers (paths T1 and C2). tively. In 2003 (Wave 15), the average age of adolescents was 27.
724 LEE, WICKRAMA, AND O’NEAL

Participants were excluded if they had relocated outside the study Higher scores reflect more hostile relationships between parents
area, if the parents divorced, or the family indicated that they were and offspring.
no longer able to participate in the study.

Psychopathology
Measures
Twenty-eight items from the Symptom Checklist-90-R (Derogatis
Interparental Hostility & Melisaratos, 1983) were used to assess self-reported ratings of
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Interparental hostility was assessed separately for husbands and fathers’, mothers’, and adolescents’ psychopathology in 1992 (Wave
wives based on each partner’s reports of their spouse’s hostile 4) and 1994 (Wave 6) on a 5-point Likert scale from 1 (not at all) to 5
interactions in 1990 (Wave 2 when the target offspring was an (extremely). Sample items include nervousness or shakiness inside,
adolescent) and 2001 (Wave 13 when the target offspring was a feeling tense or keyed up, feeling fearful, and crying easily. The items
young adult). The five-item assessment was developed for the IYFP were validated in other studies assessing general psychopathology
(Conger, 1989a). Sample items include how often in the past year (e.g., Lee, et al., 2017). For each timepoint and each family member,
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did your spouse get angry at you, call you bad names, and criticize a mean score was computed with higher values indicating more
you or your ideas. The items were rated using a 7-point Likert scale psychopathology (Lee et al., 2021). The internal consistencies were
ranging from 1 (never) to 7 (always). The internal consistencies for more than .90 for fathers, mothers, and adolescents across timepoints.
interparental hostility were .91 and .90 for 1990 (Wave 2) and .89
and .90 for 2001 (Wave 13), for husbands and wives, respectively.
Covariates
Correlations between fathers’ and mothers’ reports of interparental
hostility were .69 and .63 in 1990 and 2001, respectively. For each Using data from 1990 (Wave 2), our analyses accounted adoles-
time point, the father’s and mother’s reports of interparental hostility cent’s gender (0 = female, 1 = male) and parents’ socioeconomic
were averaged to create a single indicator of interparental hostility. characteristics, including annual total household income (including
This single construct (or variable) approach to capturing the couple husband’s, wife’s and children’s wages, salaries, and other sources
subsystem (i.e., interparental hostility) has been commonly used in of income such as supplemental security income) and parents’
previous family studies (e.g., Bryant, et al., 2017). Higher scores college completion based on fathers’ and mothers’ reports of the
reflect more hostility in the context of their marital relationship. highest grade they completed (1 = 1+ parent completed college; 0 =
neither parent completed college).
Adolescents’/Young Adults’ Hostile Relationships With
Their Fathers and Mothers Statistical Analyses
Hostile relationships with parents (separately for fathers and First, correlations were examined among study variables. Second,
mothers) were assessed using offspring’s reports in 1991 (Wave 3 in a structural equation modeling framework (Lee et al., 2021),
for adolescence) and 2003 (Wave 15 for young adulthood) with five- cross-lagged autoregressive models were used to examine the three
items developed for the IYFP (Conger, 1989b). To assess hostility hypothesized transmission pathways linking interparental hostility
expressed by both the parent and offspring (Bartle-Haring & when offspring were adolescents to parent–offspring relationship
Gavazzi, 1996), offspring were asked to report their hostile beha- hostility when offspring were young adults. Cross-lagged autore-
viors toward each parent in the past year (e.g., got angry at your gressive models allow for the examination of time-sequential pro-
parent, called him or her bad names, and criticized his or her ideas; cesses across variables capturing different domain (Lee et al., 2021).
offspring-to-parent report) as well as the hostile actions of parents Consequently, in this instance, this model allows us to examine
toward themselves in the past year (e.g., got angry at you, called you multiple transmission processes of interparental hostility. To exam-
bad names, and criticized your ideas; parent-to-offspring report). ine dynamic transmission processes of interparental hostility to
The items were rated using a 7-point Likert scale ranging from 1 young adult’s hostile relationships with father and mother, the
(never) to 7 (always). The internal consistencies for reports of father- model included interparental hostility in 1990 and 2001 and off-
to-offspring hostility and offspring-to-father hostility in 1991 spring’s hostile relationships with their father and mother in ado-
(Wave 3) and 2003 (Wave 15) ranged from .84 to .87. Reports lescence (1991) and young adulthood (2003).
of father-to-offspring and offspring-to-father hostility were highly Third, a latent variable capturing psychopathology was created to
correlated (r = .83 and .84 in 1991 and 2003, respectively). Given estimate each respondent’s (i.e., father, mother, and adolescent)
the strong correlations, these scores were averaged to create an psychopathology from two repeated measures (1992 and 1994) of
overall measure of hostility in the father–offspring relationship in psychopathology. Factor loadings for both measurement occasions
adolescence (Wave 3) and young adulthood (Wave 15). were constrained to 1 (Wickrama et al., 2016). Correlations were
Similarly, the internal consistencies for reports of both mother-to- estimated between these latent constructs, enabling an assessment of
offspring hostility and offspring-to-mother hostility in 1991 (Wave 3) the co-occurrence of fathers’, mothers’, and adolescents’ psycho-
and 2003 (Wave 15) were acceptable (ranging from .87 to .92). pathology. Invariance tests in estimated mean parameters of latent
Correlations for reported mother-to-offspring hostility and offspring- factors across family members’ psychopathology were performed
to-mother hostility were .83 and .84 in 1991 (Wave 3) and 2003 using Wald tests ( Wickrama et al., 2016). A significant p value of a
(Wave 15), respectively. These scores were averaged to create a Wald test indicates that the estimated mean parameters of latent
measure of overall hostility in the mother–offspring relationship factors of psychopathology are statistically different across family
during adolescence (Wave 3) and young adulthood (Wave 15). members.
CONTINUITY IN HOSTILE FAMILY RELATIONSHIPS 725

Fourth, latent factors of family members’ psychopathology were young adults’ hostile relationships with their fathers and mothers
included in the cross-lagged model (described in the second step) to are shown in Figure 2 (CFI/TLI = 1.00/1.00; RMSEA = .00;
examine psychopathology as a mediator of the transmission of inter- because the model is fully saturated, these overall fit indices are
parental hostility to parent–young adult relationship hostility. All not interpretable). In the cross-lagged autoregressive models (Figure 2)
possible mediation processes involving family members’ psychopa- interparental hostility when the offspring were adolescents (1990)
thology and hostile family relationships were estimated. All mediation was not directly associated with hostile relationships with their
effects were tested using a percentile bootstrapping approach (with father and mother as young adults (2003). However, results con-
1,000 iterations; MacKinnon, et al., 2007). Standardized coefficients firmed two transmission pathways of hostility. One pathway
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(β) are reported as effect sizes (Cohen, 1988). All analyses accounted involved interparental hostility (1990) → interparental hostility
for the effects of adolescents’ gender and parents’ socioeconomic (2001) → young adults’ hostile relationships with their fathers/
characteristics (i.e., annual family income and parents’ education mothers (2003) (mediation β = .07, 95% CI [.01, .13], p < .05 for
level) (1990) on interparental hostility (2001) and offspring’s hostile father–young adult model; β = .14, 95% CI [.05, .23], p < .01 for
relationships with parents (1991 and 2003). The average rate of mother–young adult model). A second pathway involved interpar-
missingness among study variables was 10.71% (ranged from ental hostility (1990) → adolescents’ hostile relationships with their
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5.3% to 16.73%). R. J. A. Little (1988) missing at completely random fathers/mothers (1991) → young adults’ hostile relationships with
(MCAR) test resulted in a χ2/df ratio (Byrne, 1991) of 1.535, indicating the fathers/mothers (2003) (mediation β = .05, 95% CI [.02, .09],
that the MCAR assumption can be retained (ratio values ≤ 2 are p < .01 for father–young adult model; β = .04, 95% CI [.01, .07], p <
considered nonsignificant, reflecting the absence of systematic miss- .01 for mother–young adult model). However, sequential transmis-
ingness patterns). In light of the nonsignificant MCAR test, missing sion pathways of interparental hostility to young adults’ hostile
data were accounted for using full information maximum likelihood relationships with both parents (through adolescents’ hostile rela-
(FIML) procedures (Enders & Bandalos, 2001). For model evaluation, tionships with their fathers/mothers (1991) → interparental hostility
the comparative fit index (CFI; acceptable fit > .90; T. D. Little, 2013), (2001)) were not statistically significant (mediation β = .001, 95%
Tucker–Lewis fit index (TLI; acceptable fit > .90; T. D. Little, 2013), CI [−.001, .003], p = .49 for father–young adult model; β = .001,
and root-mean-square error of approximation (RMSEA; acceptable fit 95% CI [−.002, .003], p = .54 for mother–young adult model).
< .08; T. D. Little, 2013) were utilized. Analyses were conducted Further, all other possible dynamic transmission processes of inter-
using Statistical Package for Social Sciences (Version 26) and Mplus parental hostility to young adult’s hostile relationships with parents
(Version 8.00) (Muthén & Muthén, 1998–2018). This study was not were not significant. The paths from the control variables were also
preregistered. Data, study materials, and study analysis code are not statistically significant.
available from the first author upon reasonable request.
Parent’s and Adolescent’s Psychopathology
Results The fit indices for the model incorporating latent variables for
Preliminary Analysis fathers’, mothers’, and adolescents’ psychopathology suggested an
acceptable fit (CFI/TLI = .93/.93; RMSEA=.07; see Figure 3). All
Supplemental Material Table S1 displays correlations among study factor loadings were greater than .70, suggesting acceptable psy-
variables. Correlations among interparental hostility when offspring chometric properties for assessing psychopathology of each family
were adolescents (1990) and offspring’s hostile relationships with member in this manner (Kline, 2016). Results indicated positive
their father and mother as young adults (2003) were statistically associations among family members’ psychopathology (r = .15,
significant and positive, suggesting a long-term transmission of 95% CI [.05, .25], p < .01 between father’s and mother’s psycho-
interparental hostility to young adults’ hostile relationships with pathology; r = .12, 95% CI [.04, .21], p < .01 between father’s and
both parents (corresponding path T in Figure 1). Further, fathers’, adolescent’s psychopathology; r = .10, 95% CI [.03, .17], p < .01
mothers’, and adolescents’ psychopathology in 1992 and 1994 were between mother’s and adolescent’s psychopathology). Further,
positively correlated. Also, interparental hostility in 1990 and 2001 adolescents averaged the highest estimated mean levels of psycho-
were significantly and positively correlated with fathers’ and mothers’ pathology (estimated mean = 1.50), followed by mothers (estimated
psychopathology in 1992 and 1994. Offspring’s hostile relationships mean = 1.35), and then fathers (estimated mean = 1.24). Wald tests
with their mothers in 1991 and 2003 (adolescence and young for mean difference indicated statistically significant differences
adulthood, respectively) were significantly and positively correlated between the mean scores of psychopathology for adolescents and
with both mothers’ and adolescents’ psychopathology in 1992 and mothers (wald value = 32.30, p < .001), mothers and fathers (wald
1994. Adolescents’ (but not young adults’) hostile relationship with value = 22.27, p < .001), and adolescents and fathers (wald value =
their father (1991) were significantly and positively associated with 97.84, p < .001). Results indicated that male adolescents showed
adolescents’ psychopathology in 1992 and 1994, whereas fathers’ lower psychopathology compared to female adolescents (β = −.21,
psychopathology in 1992 and 1994 were more likely to be signifi- 95% CI [−.32, −.10], p < .001). The effects of other control
cantly and positively correlated with hostility in their relationship with variables were not significant.
their offspring in young adulthood (2003) than adolescence (1991).
Mediating Roles of Family Members’ Psychopathology
Transmission Processes of Hostile Family Relationships
Figure 4 shows the cross-lagged autoregressive models after
Results pertaining to transmission processes of hostility from incorporating family members’ psychopathology as a mediator of
interparental relationships when offspring were adolescents to the transmission processes of hostility from interparental relationships
726 LEE, WICKRAMA, AND O’NEAL

Figure 2
Long-Term Transmission Processes of Interparental Hostility to Young Adults’ Hostile Relationships With Parents

Adolescents’ Hostile Young Adults’ Hostile


Relationships With .28*** Relationships With
Their Fathers (1991) Their Fathers (2003)
(R2=.07) (R2=.15)
.04
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.12
.17***
.11*
.64*** .11 .52***
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Interparental Interparental
Hostility (1990) .60*** Hostility (2001)
(R2=.29)

.02
.14**
.01 .24***

.01
Adolescents’ Hostile Young Adults’ Hostile
Relationships With Relationships With
Their Mothers (1991) Their Mothers (2003)
(R2=.05) .31*** (R2=.18)

Note. Standardized coefficients are shown. χ2(0) = 0.00. CFI/TLI = 1.00/1.00. RMSEA = .00. RMSEA = root-mean-square error of
approximation; CFI = comparative fit index; TLI = Tucker–Lewis fit index.
* p < .05. ** p < .01. *** p < .001.

to young adults’ hostile relationships with their parents (CFI/TLI = hostility in 2001 to young adults’ hostile relationships with their fathers
.96/.93; RMSEA = .05). For the transmission pathways of interpar- (2003) was nonsignificant (β = .11, 95% CI [−.02, .23], p = .11; not
ental hostility (1990) to young adults’ hostile relationships with their shown in Figure 4). However, the transmission process involving
fathers (2003), after adjusting for the effects of family members’ interparental hostility (1990) → adolescents’ hostile relationships with
psychopathology, results indicated that the path from interparental their fathers (1991) → young adults’ hostile relationships with their

Figure 3
Psychopathology (PSY) Among Father, Mother, and Adolescent

.01(.15**)

.01(.12**) .02(.10**)

M= 1.24*** M= 1.50*** M= 1.35***


Var.=.06*** Father Var.=.14*** Var.=.09***
Adolescent Mother
PSY PSY PSY

1(.79***) 1(.76***) 1(.71***) 1(.72***) 1(.81***) 1(.76***)

PSY PSY PSY PSY PSY PSY


(1992) (1994) (1992) (1994) (1992) (1994)

Note. PSY = Psychopathology. Standardized coefficients are shown in parentheses. M = Mean. Var. = Variance. χ2(24) =
44.38. CFI/TLI = .93/.93. RMSEA = .07. RMSEA = root-mean-square error of approximation; CFI = comparative fit index;
TLI = Tucker–Lewis fit index.
** p < .01. *** p < .001.
CONTINUITY IN HOSTILE FAMILY RELATIONSHIPS 727

Figure 4
Mediation Processes Between Hostile Family Relationships and Psychopathology

Adolescents’ Hostile Young Adults’ Hostile


Relationships with .27*** Relationships with
Their Fathers (1991) Their Fathers (2003)
(R2=.07) (R2=.21)
.11***
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Father PSY
.13*** .17** (1992 & 1994)

(R2=.10)
.64*** .43***

.10*
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.29*** .10***
.10*
Interparental Interparental
Adolescent PSY
Hostility (1990) .54*** Hostility (2001)
(1992 & 1994)
(R2=.34)
(R2=.13)
.18*** .17***
.11*

Mother PSY
.14** .19**
.16*** (1992 & 1994)
2
(R =.10)
.10***
.21***

Adolescents’ Hostile .08*** Young Adults’ Hostile


Relationships with Relationships with
Their Mothers (1991) Their Mothers (2003)
(R2=.05) .21*** (R2=.27)

Note. PSY = Psychopathology. Standardized coefficients are shown. Nonsignificant coefficients are not shown. χ2(41) = 85.57. CFI/TLI =
.96/.93. RMSEA = .05. RMSEA = root-mean-square error of approximation; CFI = comparative fit index; TLI = Tucker–Lewis fit index.
* p < .05. ** p < .01. *** p < .001.

fathers (2003) remained significant even after adjusting for signifi- 1994) (mediation β = .02, 95% CI [.005, .04], p < .01), whereas the
cant effect of family members’ psychopathology (mediation β = .03, second mediation process included mothers’ psychopathology (1992
95% CI [.01, .07], p < .01). Related to the mediating roles of and 1994) and interparental hostility (2001) (mediation β = .01, 95%
psychopathology, results indicated one statistically significant medi- CI [.001, .01], p < .05). The third mediation process included
ation effect involving fathers’ psychopathology (interparental hos- adolescents’ hostile relationships with their mothers (1991), mothers’
tility (1990) → fathers’ psychopathology (1992 and 1994) → young and adolescents’ psychopathology (1992 and 1994) (mediation β =
adults’ hostile relationships with their fathers (2003); mediation β = .01, 95% CI [.001, .006], p < .05 for mothers’ psychopathology;
.03, 95% CI [.01, .05], p < .01). mediation β = .01, 95% CI [.001, .007], p < .05 for adolescents’
The mediation effects of psychopathology on the transmission psychopathology).
processes of interparental hostility (1990) to young adults’ hostile Further, Figure 4 displays two additional transmission processes of
relationships with their mothers (2003) are also shown in Figure 4. interparental hostility to young adults’ hostile relationships with the
The two transmission pathways of interparental hostility reported in mothers emerged: (a) interparental hostility (1990) → fathers’ psy-
Figure 2 remained statistically significant: (a) interparental hostility chopathology (1992 and 1994) → interparental hostility (2001) →
(1990) → interparental hostility (2001) → young adults’ hostile young adults’ hostile relationships with their mothers (2003) (medi-
relationships with their mothers (2003) (mediation β = .07, 95% ation β = .02, 95% CI [.001, .004], p < .05) and (b) interparental
CI [.05, .10], p < .001) and (b) interparental hostility (1990) → hostility (1990) → adolescents’ hostile relationships with their fathers
adolescents’ hostile relationships with their mothers (1991) → young (1991) → adolescents’ psychopathology (1992 and 1994) → young
adults’ hostile relationships with their mothers (2003) (mediation β = adults’ hostile relationships with their mothers (2003) (mediation β =
.02, 95% CI [.001, .04], p < .05). Further, results indicated three .004, 95% CI [.001, .005], p < .01). All other possible mediation
additional mediation processes involving mothers’ and/or adoles- processes were not significant. Results indicated that male adoles-
cents’ psychopathology. The first mediation process linked inter- cents showed lower psychopathology compared to female adoles-
parental hostility (1990) to young adults’ hostile relationships with cents (β = −.24, 95% CI [−.34, −.12], p < .001). The effects of
their mothers (2003) through mothers’ psychopathology (1992 and control variables were not significant.
728 LEE, WICKRAMA, AND O’NEAL

Discussion directionality in transmissions of hostile family relationships. That


is, interparental relationships are more likely to influence parent–
To date, there has been a lack of research examining the dynamic offspring relationships rather than the reverse (parent–adolescent
(interdependence) and long-term transmission of hostile family hostility to interparent hostility). This directionality was confirmed
relationships within a family system, particularly focused on inter- in previous research on the transmission of hostile family relation-
parental relationships and how these relationships affect young ships for adolescent relationships (Conger et al., 1994), and the
adults’ relationships with their parents (both father–young adult current findings suggest this directionality extends to when off-
and mother–young adult relationships). Moreover, less is known spring are young adults.
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about the mediating roles of family members’ psychopathology in Further, results indicated positive correlations between fathers’,
relation to the transmission of interparental hostility. Based on mothers’, and adolescents’ psychopathology, which is important for
family systems theory (Cox & Paley, 1997) within the life course identifying similarity and influences between members (see Figure 3).
systems perspective (Wickrama et al., 2020), the present study Importantly, these correlations remained significant even after ad-
addressed these research gaps with longitudinal data from a sample justing effects of hostile family relationships (see Figure 4). The mood
of White American families. convergence hypothesis posits that one family member’s psycho-
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Consistent with family systems theory (Cox & Paley, 1997), results logical distress is inextricably linked to the psychological distress
confirm how dyadic subsystems influence each other longitudinally by of other family members (Meyler et al., 2007). The findings of the
identifying long-term transmission processes of hostility from inter- present study supported this hypothesis. In addition, these positive
parental relationships (i.e., the couple subsystem) to young adults’ associations in psychopathology among family members suggest that
hostile relationships with their parents (i.e., the parent–offspring functioning (psychopathology) of each family member are interde-
subsystem). In general, consistent with previous findings (e.g., pendence within a family. Interestingly, adolescents averaged higher
Johnson & Galambos, 2014), long-term associations were evident levels of psychopathology than mothers and fathers, which may
between interparental hostility and parent–young adult hostility (see suggest that adolescence is a sensitive developmental period with a
corresponding correlations in Supplemental Material Table S1). How- heightened risk of psychological distress (Lee et al., 2017).
ever, the present study extends previous findings by examining three Results also confirmed the mediating roles of family members’
transmission pathways connecting interparental hostility to young psychopathology in relation to long-term transmission processes of
adults’ hostile relationships with their parents. Further, the study interparental hostility to parent–young adult relationships with par-
examined whether interparental hostility in adolescence uniquely ents. According to the family systems theory, this mediation process
influenced young adults’ hostile relationships with fathers and mothers. reflects dynamic associations (or interdependencies) between dyadic
Results confirmed two transmission pathways: (a) one involving subsystems and functioning of individual family members. Further,
interparental hostility in young adulthood (see paths C1 and T3 in consistent with the life course systems perspective, this mediation
Figure 1) and (b) the other involving adolescents’ hostile relationships process occurred over time. That is, hostile family relationships (either
with their father and mother (see paths T1 and C2 in Figure 1). in the couple subsystem or parent–offspring subsystem) were influ-
Importantly, those two transmission processes were found for both ential for family members’ subsequent psychopathology, yet indivi-
father–young adult and mother–young adult relationships. Consistent duals’ psychopathology was also consequential for subsequent family
with previous findings (Branje, 2018; Caughlin & Huston, 2006), the relationships. Further, the present study was able to examine media-
study found evidence of continuity in hostile relationships within a tion processes for both father–young adult hostility and mother–
family, particularly continuity in interparental hostility (C1 in Figure 1) young adult hostility.
and (b) parent–offspring hostility (C2 in Figure 1). Whereas previous Regarding mediation processes involved in father–young adult
studies mainly focused on the proximal transmission of interparental hostility (see Figure 4), results indicated that fathers’ psychopathol-
hostility to adolescents’ hostile relationships with their parents (T1 in ogy (when their offspring were in adolescents) mediated the associa-
Figure 1; e.g., Smith, et al., 2019), the present study findings confirm tions between interparental hostility in adolescence (1990) and
that this proximal transmission pathway continues into the offspring’s father–young adult hostility (2003). Importantly, interparental hos-
adulthood (T3 in Figure 1). Taken together, these results suggest two tility when offspring were young adults (2001) did not predict young
types of longitudinal stabilities in dyadic subsystems within a family: adults’ hostile relationships with their fathers (2003) after adjusting
(a) stability within each dyadic subsystem (C1 and C2 in Figure 1) and for the effects of fathers’ psychopathology when the offspring was an
(b) stability across dyadic subsystems (T1 and T3 in Figure 1). Also, adolescent (1992 and 1994). These results suggested that fathers’
these two longitudinal stabilities existed for the transmission process of affective symptoms when their offspring was an adolescent were
interparental hostility to young adult’s hostile relationship with both highly salient in shaping fathers’ relationships with their offspring at
parents. These results are consistent with the life course perspective, a later developmental stage (e.g., Low & Stocker, 2005).
which emphasizes that individuals’ roles and social relationships in Given the positive associations among fathers’, mothers’, and
one life stage are, at least in part, a product of their experiences at adolescents’ psychopathology, adolescents’ and mothers’ psycho-
previous life stages (Wickrama et al., 2020). Furthermore, the results pathology may be indirectly associated with father–young adult
contribute to the life course perspective by identifying long-term relational hostility through fathers’ psychopathology. Indeed, a
transition processes of hostile relationships both within and across previous study found that parents who experienced higher levels
two dyadic subsystems. of depressive symptoms were more likely to have children with
However, results did not support the hypothesized sequential elevated rates of anxiety and depressive symptoms from early
transmission pathways of interparental hostility to young adult’s adolescence to young adulthood (Tyrell et al., 2019). More research
hostile relationships with parents were not found (T1, T2, and T3 in is needed to examine why, and how, family members’ symptoms
Figure 1). These nonsignificant sequential pathways may suggest impact transmission processes of hostile family relationships.
CONTINUITY IN HOSTILE FAMILY RELATIONSHIPS 729

Regarding mediation processes for mother–young adult relational number of children, and occupations are necessary. Second, self-
hostility (see Figure 4), similar mediation processes were identified as reported information regarding psychopathology may be a source of
found for father–young adult hostility (i.e., interparental hostility → bias. Future research should extend this research using clinical
mothers’ psychopathology → young adults’ hostile relationships with measures of mental health outcomes. Third, the study only examined
their mothers). Two additional mediation processes involving both offspring’s reports of a hostile relationship with parent due to the lack
mothers’ and adolescents’ psychopathology were also identified. One of corresponding parent data. Thus, future studies should incorporate
mediation process included mothers’ psychopathology as a mediator of both parents’ and offspring’s reports. Fourth, previous studies have
the continuity in interparental hostility from 1990 to 2001, which, in suggested that nonobserved personal-level factors (e.g., genetic
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turn, influenced young adults’ hostile relationships with their mothers components) can play critical roles in influencing family interactions
(2003). The other mediation process indicated that interparental hos- (Horwitz et al., 2011). Future studies should explore these risk factors
tility influenced adolescents’ hostile relationships with their mothers, in greater detail. Fourth, given the limits of the data, the present study
which, in turn, influenced both mothers’ and adolescents’ psychopa- examined long-term transition processes of hostile family relations
thology. Then, both mothers’ and adolescents’ psychopathology influ- with unequal time intervals (interparental hostility in 1990 and 2001;
enced young adults’ hostile relationships with their mothers. These two parent–offspring hostility in 1991 and 2003). Therefore, it is unclear
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mediation processes suggest that young adults’ hostile relationships if the identified transmission processes of hostile family relationships
with their mothers are shaped through dynamics (interdependencies) are stable across longer time periods. Finally, the present study was
between dyadic subsystems (i.e., interparental hostility and parent– limited to couples in enduring marriages and their offspring. The
adolescent hostility) and functioning of individual family members results may be different for divorced families as they have more
(i.e., mothers’ and adolescents’ psychopathology) across adolescence exposure to both hostile family relationships and psychopathology
and young adulthood. Also, these additional mediation processes for (e.g., Wickrama, et al., 2013).
mother–young adult hostile relationships, compared to hostile relation- The present study contributes to knowledge about the longitudinal
ships with fathers, suggest that mother–offspring relationships are more mediation processes between hostile family relationships and family
sensitive (i.e., influenced by more contributing factors) compared to members’ psychopathology over an extended period of time (1990–
father–offspring relationships (e.g., Bryant, et al., 2017). This may 2003) in a sample of rural families. Importantly, the findings indicate
relate to mothers generally being more involved with their offspring that interparental hostility and parent–adolescent hostility are linked
(e.g., Pineda, et al., 2007), particularly for this sample of fairly to hostility in the parent–young adult relationship through several
traditional families from a rural area in the Mid-west. transmission pathways, and these transmission pathways can be
Last, results indicated that adolescents’ hostile relationships with mediated by family members’ psychopathology. Although many
their fathers (1991) and fathers’ psychopathology (1992 and 1994) of the identified processes were similar for mothers and fathers, some
were involved in the transmission processes of interparental hostility distinct processes emerged. These findings emphasize the importance
to mother–young adult hostility (see detailed paths in Figure 4). of intervention efforts accounting for dynamic family relationships to
These findings suggest dynamic transmission processes of inter- improve family health. Such efforts must address how dyadic
parental hostility to young adults’ hostile relationships with parents. subsystems affect each other over time and how individual family
In other words, fathers–offspring relationships and fathers’ psycho- members affect and are affected by family subsystems.
pathology when their offspring were adolescents appear to have
long-term effects on young adults’ relationships with their mothers.
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