Blood

Visit ID : MITD18523 Registration : 01/May/2023 09:59AM
UHID/MR No : AITD.0000018488 Collected : 01/May/2023 10:00AM

Patient Name : Mr.PRIYANSHU GUPTA Received : 01/May/2023 10:26AM
Age/Gender : 17 Y 0 M 0 D /M Reported : 01/May/2023 01:52PM
Ref Doctor : Dr.BIOCITY HEALTHCARE Status : Final Report
Client Name : BIOCITY HEALTH CARE Client Code : 78
Client Add : DELHI Barcode No : 10078056
DEPARTMENT OF HAEMATOLOGY
BIOCITY COMPREHENSIVE PACKAGE
Test Name Result Unit Bio. Ref. Range Method
CBC+ ESR
Sample Type : WHOLE BLOOD EDTA
HAEMOGLOBIN (HB) 13.2 gm/dl 13.00-17.00 Non-Cyanmethemoglobin
TOTAL LEUCOCYTE COUNT (TLC) 5440 cell/cmm 4000-10000 Flow cytometry
DLC (by Flow cytometry/Microscopy)
NEUTROPHIL 50.4 % 40-75
LYMPHOCYTE 41.0 % 20-40
EOSINOPHIL 1.0 % 01-07
MONOCYTE 7.4 % 2-10
BASOPHIL 0.2 % 00-02
ABSOLUTE NEUTROPHIL COUNT 2.74 x10^3 Cells/uL 1.5-7.8 Automated Calculated
ABSOLUTE LYMPHOCYTE COUNT 2.23 x10^3 Cells/uL 2.0-3.9 Automated Calculated
ABSOLUTE EOSINOPHIL COUNT 0.05 x10^3 Cells/uL 0.2-0.5 Automated Calculated
ABSOLUTE MONOCYTE COUNT 0.4 x10^3 Cells/uL 0.2-0.95 Automated Calculated

ABSOLUTE BASOPHIL COUNT 0.01 x10^3 Cells/uL 0.02-0.2 Automated Calculated
RBC COUNT(RED BLOOD CELL COUNT) 4.9 million/cmm 4.50-5.50 Optical Flowcytometry
PCV/HAEMATOCRIT 39.1 % 40-50 RBC pulse height
detection
MCV 79.7 fL 80-100 Automated/Calculated
MCH 27 pg 27-32 Automated/Calculated

MCHC 33.8 g/dl 32-36 Automated/Calculated
RDW-CV 14.6 % 11.5-14.5 Automated/Calculated
RDW-SD 38.7 fL 39-46 Calculated
PLATELET COUNT 2.30 lac/mm3 1.50 - 4.50 Optical Flowcytometry

PDW 16.6 fL 8.30-25.00 Calculated
MPV 11 fL 8.60-15.50 Calculated
PCT 0.256 % 0.15-0.62
ERYTHROCYTE SEDIMENTATION RATE 11 mm/1st hr 1-10 Westergren
Page 1 of 13
DEPARTMENT OF HAEMATOLOGY
HBA1C
Sample Type : WHOLE BLOOD EDTA
HBA1c 5.2 % Normal Glucose tolerance (non- HPLC
diabetic): <5.6%~Pre-diabetic:
5.7-6.4%~Diabetic Mellitus:
>6.5%
ESTIMATED AVG. GLUCOSE 102.54 mg/dl 62.30 - 142.72
INCREASED IN
1. Chronic renal failure with or without hemodialysis.

2. Iron deficiency anemia. Increased serum triglycerides.
3. Alcohol.
4. Salicylate treatment.
DECREASED IN
1. Shortened RBC life span (hemolytic anemia, blood loss), Pregnancy.

2. Ingestion of large amounts (>1g/day) of vitamin C or E.
3. Hemoglobinopathies (e.g.: spherocytes) produce variable increase or decrease.
4. Results of %HbA1c are not reliable in patients with chronic blood loss and consequent variable erythrocyte life span.
Page 2 of 13
Age/Gender : 17 Y 0 M 0 D /M Reported : 01/May/2023 11:10AM
DEPARTMENT OF BIOCHEMISTRY
LIVER FUNCTION TEST

Sample Type : SERUM
TOTAL BILIRUBIN 0.51 mg/dl 0.1-1.2 Diazotized, Sulfanilic
CONJUGATED ( D. Bilirubin) 0.21 mg/dl 0.00-0.30 Jendrassik & Groff
UNCONJUGATED ( I.D. Bilirubin) 0.3 mg/dl 0.1-1.0 Calculated
SGOT 35 U/L 8.0-35.0 Enzymatic,IFFC
S.G.P.T 35 U/L 10.0-35.0 Enzymatic,IFFC
ALKALINE PHOSPHATASE 136 U/I 30-120
TOTAL PROTEINS 7.59 gm/dl 6.40-8.30 Biuret
ALBUMIN 4.20 gm/dl 3.5-5.0 BCG
GLOBULIN 3.39 gm/dl 2.0-4.1 Calculated
A/G RATIO 1.24 1.0-2.0 Calculated
GGT 27 U/L 8.0-55.0 Colorimetric Method
Comments and Interpretation:

The liver filters and processes blood as it circulates through the body. It metabolizes nutrients, detoxifies harmful substances, makes blood clotting proteins, and performs
many other vital functions. The cells in the liver contain proteins called enzymes that drive these chemical reactions. When liver cells are damaged or destroyed, the
enzymes in the cells leak out into the blood, where they can be measured by blood tests Liver tests check the blood for two main liver enzymes.
Aspartate aminotransferase (AST),SGOT: The AST enzyme is also found in muscles and many other tissues besides the liver.
Alanine aminotransferase (ALT), SGPT: ALT is almost exclusively found in the liver. If ALT and AST are found together in elevated amounts in the blood,
liver damage is most likely present.
Alkaline Phosphatase and GGT: Another of the liver's key functions is the production of bile, which helps digest fat. Bile flows through the liver in a system
of small tubes (ducts), and is eventually stored in the gallbladder, under the liver. When bile flow is slow or blocked, blood levels of certain liver enzymes rise:
Alkaline phosphatase Gamma-utamyl transpeptidase (GGT) Liver tests may check for any or all of these enzymes in the blood. Alkaline phosphatase is by far
the most commonly tested of the three. If alkaline phosphatase and GGT are elevated, a problem with bile flow is most likely present. Bile flow problems can be
due to a problem in the liver, the gallbladder, or the tubes connecting them.
Proteins are important building blocks of all cells and tissues. Proteins are necessary for your body's growth, development, and health. Blood contains two classes
of protein, albumin and globulin. Albumin proteins keep fluid from leaking out of blood vessels. Globulin proteins play an important role in your immune system.
Low total protein may indicate: 1.bleeding 2.liver disorder 3.malnutrition 4.agammaglobulinemia
High Protein levels 'Hyperproteinemia: May be seen in dehydration due to inadequate water intake or to excessive water loss (eg, severe vomiting, diarrhea,
Addison's disease and diabetic acidosis) or as a result of increased production of proteins
Low albumin levels may be caused by: 1.A poor diet (malnutrition). 2.Kidney disease. 3.Liver disease.
High albumin levels may be caused by: Severe dehydration.
Page 3 of 13
LIPID PROFILE
Sample Type : SERUM
TOTAL CHOLESTEROL 170.32 mg/dl Desirable : 80-200~Borderline: Cholesterol
200 – 239~High : >=240 oxidase/peroxidase
TRIGLYCERIDES 127 mg/dl 40-149~BorderLine : 150- Glycerol phosphate

199~High : 200-499~Very High : oxidase/peroxidase
>=500
H D L CHOLESTEROL 52.0 mg/dl 40-60 Phosphotungstate/Mg-
Cholesterol oxidase/
peroxidase
VLDL 25.4 mg/dl 15-30 Calculated
L D L CHOLESTEROL 92.92 mg/dl 70-106~Above Optimal : 100- Calculated
129~Borderline High : 130-
159~High : 160-189~Very High :
>=190
NON HDL CHOLESTEROL 118.32 mg/dl Desirable: <130~BorderLine : Calculated
150-199~High : 200-499~Very
High : >=500
LDL / HDL RATIO 1.79 Calculated
T. CHOLESTEROL/ HDL RATIO 3.28 Calculated
Comment and interpretation:

A Lipid test can help determine your risk of the buildup of fatty deposits (plaques) in your arteries that can lead to narrowed or blocked arteries throughout your body
(atherosclerosis).High cholesterol usually causes no signs or symptoms. A complete cholesterol test is done to determine whether your cholesterol is high and to estimate
your risk of heart attacks and other forms of heart disease and diseases of the blood vessels.
A complete Lipid test includes the calculation of four types of fats in your blood:
Total cholesterol. This is a sum of your blood's cholesterol content.

Low-density lipoprotein (LDL) cholesterol. This is called the "bad" cholesterol. Too much of it in your blood causes the buildup of fatty deposits (plaques) in
your arteries (atherosclerosis), which reduces blood flow. These plaques sometimes rupture and can lead to a heart attack or stroke.
High-density lipoprotein (HDL) cholesterol. This is called the "good" cholesterol because it helps carry away LDL cholesterol, thus keeping arteries open
and your blood flowing more freely.
Triglycerides. Triglycerides are a type of fat in the blood. When you eat, your body converts calories it doesn't need into triglycerides, which are stored in fat
cells. High triglyceride levels are associated with several factors, including being overweight, eating too many sweets or drinking too much alcohol, smoking,
being sedentary, or having diabetes with elevated blood sugar levels
Page 4 of 13
IgG (IMMUNOGLOBULIN IgG )

Sample Type : Serum
Immunoglobulin IgG 890.20 mg/dl 700 - 1600 Turbidimetry
INTERPRETATION:
IgG is the most abundant immunoglobulin in normal adult plasma. It is the only immunoglobulin that crosses the placental barrier to
provide protection to the fetus. Decreased IgG concentrations occur in primary immunodeficiency conditions as well as in secondary immune
insufficiencies. Increased IgG concentrations occur due to polyclonal or oligoclonal immunoglobulin proliferation. In serum, IgG exists in
monomeric and polymeric forms. Monoclonal immunoglobulinemia requires detailed differential diagnostic investigation in addition to the
quantitative determination.
Page 5 of 13
KFT WITH ELECTROLYTE

Sample Type : SERUM
SERUM UREA 19.96 mg/dL 15-39 Urease GLDH
Blood Urea Nitrogen (BUN) 9.33 mg/dl 5-25
SERUM CREATININE 0.7 mg/dl 0.60–1.30 Jafees
SERUM URIC ACID 6.44 mg/dl 3.7-9.20 URICASE
BUN/CREATININE RATIO 13.33 10-20
Estimated Glomerular Filtration Rate (eGFR) 115.00 mL/min/1.73m2 REFER INTERPRETAION
Electrolyte Profile
SERUM CHLORIDE 102.3 mmol/L 98.0-109.0 ISE
SERUM SODIUM 144.5 mmol/L 136.0-149.0 ISE
SERUM POTASSIUM 4.32 mmol/L 3.5-5.0 ISE
Comment and Interpretation:

Healthy kidneys remove wastes and excess fluid from the blood. Blood and urine tests show how well the kidneys are doing their job and how quickly body wastes are being
removed. Urine tests can also detect whether the kidneys are leaking abnormal amounts of protein, a sign of kidney damage.
Blood Urea / Urea nitrogen comes from the breakdown of protein in the foods you eat. As kidney function decreases, the BUN level rises
Serum Creatinine is a waste product that comes from the normal wear and tear on muscles of the body. Creatinine levels in the blood can vary depending on
age and body size. A creatinine level of greater than 1.2 for women and greater than 1.4 for men may be an early sign that the kidneys are not working properly.
As kidney disease progresses, the level of creatinine in the blood rises
Uric Acid is produced by the breakdown of purines, chemicals that enter the bloodstream during digestion of foods or from normal breakdown of some of the
body's cells. The kidneys filter out most of the uric acid in the blood and eliminate it from the body in the urine. Some uric acid also leaves the body in the
feces. Uric acid can accumulate when the body produces too much or fails to excrete enough of it. Excess uric acid can also form crystals or kidney stoneth at
can damage the kidneys. Rarely, excess uric acid in kids can cause gout, a very painful inflammation caused by uric acid crystals in joint fluid (also called synovial
fluid). Gout most often affects the joints of the ankles, feet, and toes
Electrolytes including sodium and potassium, are lost in sweat during exercise. A rapid loss of fluids, such as after a bout of diarrhea or vomiting, can also affect
the concentration of electrolytes. In these types of situations, the balance of electrolytes in the body needs to be restored. The kidneys and several hormones
regulate the concentration of each electrolyte. If the level of one is too high, the kidneys filter it from the body, and different hormones act to restore a balance.
An imbalance causes a health issue when the concentration of a certain electrolyte becomes higher than the body can regulate. Low levels of electrolytes can also
affect overall health.
Page 6 of 13
BONE PROFILE
Sample Type : SERUM
CALCIUM 9.02 mg/dl 8.1-10.4
ALKALINE PHOSPHATASE 136 U/I 30-120
Phosphorus 3.01 mg/dl
FBS
Sample Type : Plane Blood
Blood Sugar 106.90 mg/dl 70 - 110
INTERPRETATION:
Increased In
Diabetes Mellitus
Stress (e.g., emotion, burns, shock, anesthesia)
Acute pancreatitis
Chronic pancreatitis
Wernicke encephalopathy (vitamin B1 deficiency)
Effect of drugs (e.g. corticosteroids, estrogens, alcohol, phenytoin, thiazides)
Decreased In
Pancreatic disorders
Extrapancreatic tumors
Endocrine disorders
Malnutrition
Hypothalamic lesions
Alcoholism
Endocrine disorders
Page 7 of 13
IRON PROFILE
Sample Type : Serum
SERUM IRON 108.50 ugm/dl 65-175 Ferrozine
TOTAL IRON BINDING CAPACITY 365.2 ugm/dl 250-450 Calculations
UIBC 256.70 ugm/dL 130 - 336 Ferrozine
TRANSFERRIN SATURATION 29.71 % 16-50
INTERPRETATION:
SERUM IRON INCREASED IN:
Hemosiderosis of excessive iron intake (e.g. repeated blood transfusion, iron therapy, iron containing vitamins)
Decreased formation of RBCs (thalassemia, pyridoxal deficiency anaemia).
Increased destruction of RBCs (hemolytic anaemia).
Acute liver damage
Acute iron toxicity
SERUM IRON DECREASED IN:
Iron deficiency anaemia

Normochromic anaemia of infections & chronic diseases
Nephrosis
Menorrhagia
Diurnal variation: Normal in mid morning, low values in mid afternoon, and very low values near midnight.
TIBC/UIBC INCREASED IN:
Iron deficiency anemia

Acute & Chronic blood loss
Acute liver damage
Progesterone birth control pills
TIBC/UIBC DECREASED IN:
Hemochromatosis
Cirrhosis of the liver
Thalassemia
Anemia of infective & chronic disease
Nephrosis
TRANSFERRIN SATURATION INCREASED IN:
Page 8 of 13
High Values in iron overload

Raised transferrin saturation is an early indicator of Iron accumulation in hemochromatosis.
TRANSFERRIN SATURATION DECREASED IN:
Low Values in iron deficiency
Page 9 of 13
DEPARTMENT OF HORMONE ASSAYS
THYROID PROFILE (T3,T4,TSH)

Sample Type : SERUM
T3 1.52 ng/ml 0.80-2.10 CLIA
T4 9.80 ug/dl 5.01-12.45 CLIA
TSH 0.91 ulU/mL 0.70-6.40 CLIA
INTERPRETATION:
1. Serum T3, T4 and TSH are the measurements form three components of thyroid screening panel and are useful in diagnosing various
disorders of thyroid gland function.
2. Primary hyperthyroidism is accompanied by elevated serum T3 and T4 values along with depressed TSH levels.
3. Primary hypothyroidism is accompanied by depressed serum T3 and T4 values and elevated serum TSH levels.
4. Normal T4 levels accompanied by high T3 levels are seen in patients with T3 thyrotoxicosis. Slightly elevated T3 levels may be found in
pregnancy and in estrogen therapy while depressed levels may be encountered in severe illness, malnutrition, renal failure and during
therapy with drugs like propanolol and propylthiouracil.
5. Although elevated TSH levels are nearly always indicative of primary hypothyroidism, rarely they can result from TSH secreting pituitary
tumors (secondary hyperthyroidism).
6. Low levels of Thyroid hormones (T3, T4 & FT3, FT4) are seen in cases of primary, secondary and tertiary hypothyroidism and sometimes
in non-thyroidal illness also.
7. Increased levels are found in Grave’s disease, hyperthyroidism and thyroid hormone resistance.
8. TSH levels are raised in primary hypothyroidism and are low in hyperthyroidism and secondary hypothyroidism.
9. REFERENCE RANGE :
TSH in uIU/mL
PREGNANCY
1st Trimester 0.60 - 3.40
2nd Trimester 0.37 - 3.60
3rd Trimester 0.38 – 4.04
Age TSH in uIU/mL

0 – 4 Days 1.00 - 39.00
2 Weeks to 5 Months 1.70 – 9.10
6 Months to 20 Yrs. 0.70 – 6.40
>55 Yrs. 0.50 - 8.90
( References range recommended by the American Thyroid Association)
Comments:
1. During pregnancy, Free thyroid profile (FT3, FT4 & Ultra-TSH) is recommended.
2. TSH levels are subject to circadian variation, reaches peak levels between 2-4 AM and at a minimum between 6-10 PM. The variation of
the day has influence on the measured serum TSH concentrations.
Page 10 of 13
VITAMIN D25 HYDROXY

Sample Type : SERUM
VITAMIN D 24.62 ng/ml 30-100 CLIA
INTERPRETATION:
LEVEL REFERENCE RANGE

Deficiency (serious deficient) < 10 ng/ml
Insufficiency (Deficient) 10-30 ng/ml
Sufficient (adequate) 30-100 ng/ml
Toxicity > 100 ng/ml
DECREASED LEVELS:
-Deficiency in children causes Rickets and in adults leads to Osteomalacia. It can also lead to Hypocalcemia and Tetany.
-Inadequate exposure to sunlight.
-Dietary deficiency.
-Vitamin D malabsorption.
-Severe Hepatocellular disease.
-Drugs like Anticonvulsants.
-Nephrotic syndrome.
INCREASED LEVELS:
-Vitamin D intoxication.
COMMENTS:
-Vitamin D (Cholecalciferol) promotes absorption of calcium and phosphorus and mineralization of bones and teeth. Vitamin
D status is best determined by measurement of 25 hydroxy vitamin D, as it is the major circulating form and has longer half
life (2-3 weeks) than 1, 25 Dihydronxy vitamin D (5-8 hrs).
-The assay measures D3 (Cholecaciferol) metabolites of vitamin D.
-25 (OH) D is influenced by sunlight, latitude, skin pigmentation, sunscreen use and hepatic function.
-Optimal calcium absorption requires vitamin D 25 (OH) levels exceeding 75 nmol/L.
-It shows seasonal variation, with values being 40-50% lower in winter than in summer.
-Levels vary with age and are increased in pregnancy.
-This is the recommended test for evaluation of vitamin D intoxication.
Page 11 of 13
VITAMIN B12
Sample Type : SERUM
VITAMIN B12 413.1 pg/mL 187-883 pg/mL CLIA
COMMENTS:
Results may differ between laboratories due to variation in population and test method. Vitamin B12 is implicated in the
formation of myelin, and along with Folate is required for DNA synthesis. The most prominent source of B12 for humans is
meat while untreated fresh water can also be a source.
Megaloblastic anaemia has been found to be due to B12 deficiency, a major cause being Pernicious anemia due to poor B12
uptake resulting in below normal serum levels. Other conditions related to low B12 levels include iron deficiency anemia,
pregnancy, vegetarianism, partial gastrectomy, ileal damage, oral contraceptives, parasitic infestations, pancreatic
deficiency, treated epilepsy and advancing age. The correlation of serum B12 levels and Megaloblastic anemia however is
not always clear - some patients with high MCV may have normal B12 levels, while some individuals with B12 deficiency may
not have megaloblastic anemia. Disorders renal failure, liver diseases and myeloproliferative diseases may have elevated
vitamin B12 levels.
LIMITATIONS:
For diagnostic purposes, the B12 results should be used in conjunction with other data; e.g.; symptoms results of other
testing, clinical impressions, etc.
If the B12 level is inconsistent with clinical evidence, additional testing is suggested to confirm the result.
Page 12 of 13
DEPARTMENT OF CLINICAL PATHOLOGY
URINE EXAMINATION COMPLETE

Sample Type : URINE
PHYSICAL EXAMINATION
QUANTITY 25 ml 0-50
COLOUR PALE YELLOW PALE YELLOW
TRANSPARENCY CLEAR Clear
SPECIFIC GRAVITY 1.020 1.010 - 1.030
CHEMICAL EXAMINATION
pH 6.0 5-7 Double Indicator
PROTEIN NIL Nil Protein - error of
Indicators
REDUCING SUGAR NIL Nil GOD-POD
UROBILINOGEN NIL Nil Ehrlichs Reaction
KETONE BODIES NIL Nil Legals Nitroprasside
BILIRUBIN NIL Nil Azo-coupling Reaction
BLOOD NIL Nil Pseudo-peroxidase
LEUCOCYTE NIL Nil by an azo-coupling
reaction
NITRITE NIL Nil Diazotization Reaction
MICROSCOPIC EXAMINATION
PUS CELLS 2-4 cells/HPF 0-5
RBCs NIL Cells/HPF Nil
EPITHELIAL CELLS 0-1 Cells/HPF 0-5
CRYSTALS NIL Nil Nil
CASTS NIL /HPF Nil
OTHER NIL
*** End Of Report ***
Page 13 of 13

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Visit ID : MITD18523 Registration : 01/May/2023 09:59AM

UHID/MR No : AITD.0000018488 Collected : 01/May/2023 10:00AM

ABSOLUTE MONOCYTE COUNT 0.4 x10^3 Cells/uL 0.2-0.95 Automated Calculated

MCH 27 pg 27-32 Automated/Calculated

RDW-SD 38.7 fL 39-46 Calculated

PLATELET COUNT 2.30 lac/mm3 1.50 - 4.50 Optical Flowcytometry

1. Chronic renal failure with or without hemodialysis.

1. Shortened RBC life span (hemolytic anemia, blood loss), Pregnancy.

LIVER FUNCTION TEST

Comments and Interpretation:

TRIGLYCERIDES 127 mg/dl 40-149~BorderLine : 150- Glycerol phosphate

Comment and interpretation:

Total cholesterol. This is a sum of your blood's cholesterol content.

IgG (IMMUNOGLOBULIN IgG )

KFT WITH ELECTROLYTE

Comment and Interpretation:

SERUM IRON DECREASED IN:

Iron deficiency anaemia

TIBC/UIBC INCREASED IN:

Iron deficiency anemia

TIBC/UIBC DECREASED IN:

TRANSFERRIN SATURATION INCREASED IN:

High Values in iron overload

TRANSFERRIN SATURATION DECREASED IN:

Low Values in iron deficiency

THYROID PROFILE (T3,T4,TSH)

Age TSH in uIU/mL

VITAMIN D25 HYDROXY

LEVEL REFERENCE RANGE

URINE EXAMINATION COMPLETE

*** End Of Report ***

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