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STROKE

KELOMPOK 7
MAFTUHATUL AFIAH - 11020150061
MUH. ILHAM KAHARU - 11020150062
ARIDAYANA - 11020150063
NUR FADILLAH ARIESA - 11020150064
AFRILIA CHAERUNNISA - 11020150065
DZULFACHRI KURNIAWAN - 11020150066
RESKY DARMAWAN - 11020150067
DESI UTAMI SARI - 11020150068
FATURAHMAN MUBARAK - 11020150069
AMALIAH HAKIM - 11020150070
NUR FADHILLAH AZIZAH -11020150157
PENDAMPING: DIAN EKA SAPUTRI

DEFINITION

a syndrome which has


the characteristic
clinical signs and
symptoms of focal or
global neurological
growing rapidly
disturbance in cerebral,
with symptoms lasting
more than 24 hours or
leading to death
without cause signs
other than those
derived from vascular

EPIDEMIOLOGY
Stroke cases in Indonesia showed an
increase. both in incidence, disability
or death.
Amounted to 100,000 people have
suffered a stroke and about 4.3% of
stroke patients have disabilities are
advancing.

Stroke Facts

Stroke is the
fourth leading
cause of death in
the United States
795,000 people in
the U.S. suffer
strokes each year

A leading cause of adult


disability
Up to 80 percent of all
strokes are preventable
through risk factor
management
On average, someone
suffers a stroke every
40 seconds in the United
States

133,000 deaths in
the U.S. each
year
1998 to 2008,
the stroke death
rate fell
approximately 35
percent and
number of deaths
fell by 19 percent
7,000,000 stroke
survivors

ETIOLOGY

A risk factors for stroke


1.
2.
3.
4.
5.
6.
7.

Genetic
History with cardiovascular
Hypertension
Smoking
Atrial fibrillation
Dyslipidemia
Condition of infection

PHATHOFISIOLOGY
Stroke can be categorised into two main groups, namely :

A. Stroke Ischemic
B. Stroke Hemorrhagic
Ischemic is caused by occlusion of the arteries in the brain that
can be caused by thrombosis or embolism. thrombosis is an
obstruction of blood flow due to luminal narrowing or blockage of
blood vessels. the most common cause is atherosclerosis. The
symptoms gradually become heavy.
hemorrhagic caused by a ruptured artery, either intracerebral or
subarachnoid. the most common cause of intracerebral
hemorrhage where the small blood vessels that have been
damaged by chronic hypertension tear. Subarachnoid
hemorrhage due to rupture of the aneurysm into the
subarachnoid cavity causing cerebrospinal fluid fills with the
blood

CLINICAL MANIFESTATIONS

- Focal neurological deficits such as hemiparesis, hemihypestesia,


aphasia, dysphagia, disturbance of consciousness, and so on.
- In haemorrhagic, there are signs of increased cranial pressure, such
as headache and loss of consciousness
- In ischemic, the clinical symptoms are usually calmer, rarely found
the signs of increased intracranial pressure, except in case of
occlusions in the large arteries or heavy hypoxia to cause edema

DIAGNOSTICS APPROACHES

Anamnesis

Sudden symptoms at the start /


active during attacks Describes
the symptoms which is occurred
with the seque Associated
symptoms and the presence or
absence of risk factors
Basically the goal of a therapy
itself is
a) efforts that disability is not limited
b) prevent complications
c) help the recovery of the patients
d) prevent deaths

Physical examination
a) vital signs
b) examination of the head and neck
c) general physical examination
d) neurological examination

Supporting Examination
a) electrocardiographic
b) lab
c) radiographic
d) ct scan
e) transcranial doppler
f) serebrospital fluid analysis

THERAPHY
As we know that the stroke is divided into two, while
the therapy should be performed in ischemic and
hemorrhagic stroke is Medical Therapy of Ischemic
StrokeDead cells cannot be regained anymore. What we
are trying to do is that do not let the dying cells die. After
the ischemic brain occurs in a wide variety of advanced for
example the formation of edema in parts of the brain,
changes in the composition of neurotransmitters, changes
in regional vascularization and changes in metabolic rate.
Many kinds of actions which have been done but they are
still controversial.
As for the drug to be used in ischemic stroke:
a) Medication for swollen brain (cerebral edema)
b) Antiagregaji platelet drugs
c) Thrombolytic drugs (may destroy the thrombus)
d) Other actions

Medical Therapy of Hemorrhagic Stroke


Medical treatment of subarachnoid hemorrhagic by
rupture of aneurism or arteriovenous malformation
rupture is not yet standard. Treatment or containment
procedures include
a) bed rest is not in a quiet room,
b) getting people not straining
c) maintaining fluid and electrolyte balanced

COMPLICATION
Stroke complcations may appear as a result stroke
attack itself and the risk of other diseases. Stroke
canoccur after or accompanies the other diseases such
as high cholesterol, heart disease, hypertension,
diabetes and obesity. With the complications of a stroke,
the patient's recovery will be depleting. If we analyze
multiple concomitant diseases or the trigger of
complications, the disease is caused due to our daily
habits especially regarding the food we eat. So that this
habit will lead to disease which blockage of blood flow
which causes stroke. Therefore, paying attention to the
diet and the nutrition that we shallow into the body will
reduce the risk of stroke and its complications.

PROGNOSIS
The Journey of stroke is vary. There is a stroke
patient who has recovered perfectly, but
those survivors who recovered with mild
disability, or moderate and severe disabilities.
Stroke is a disease that is most likely to cause
defects in the age group of 45 years.General
recovery of neurological disorders in stroke
occurs during the first days or weeks. If the
patient still has the defect after 6 months, the
repair will not be striking, but mild
improvement can still be expected in 2 years.

REFERENCE

Tanto Chris , liwang frans , Hanifati sonia , pradipta eka adit ( 2014 ) , Kapita Selekta
Kedokteran Jakarta :Media Aescculapius

https://www.google.co.id/webhp?sourceid=chrome-instant&ion=1&espv=2&ie=UTF8#q=national+stroke+association+slide

THANK YOU

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