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Ariwibowo, SpJP
ECG Interpretation
I. Calibration : Standard recording : speed 25 mm/sec & amplitude 1 mV.
II. Basic rhythm : (after no III-VI)
Sinus / Supraventricular (atrial/junctional)/
Ventricular rhytm.
III. QRS (R-R) Regularity : Regular Brady/Normal/Tachy.
& Rate Irregular Fibrilation.
IV. P wave : Presentation/ Regularity/ Rate/ Axis/ followed by QRS/
Width/ Height/ Morphology.
V. PR interval : Normal / Short / Long.
VI. QRS complex : Width/ Height/ Morphology.
QRS axis : Normal / LAD / RAD / Superior
VII. ST segment : Normal / Depression / Elevation
(Morphology & Location).
VIII. T wave : Normal / Tall / Inverted (Morphology & location)
IX. QT Interval : Normal / Short / Long.
X. U wave : Normal / Prominent/ Inverted
Conclusion Diagnosis
I. Calibration the standard recording
1 300
2 150
3 100
4 75
5 60
6 50
Example 1: What is the heart rate?
(300 / 6) = 50 bpm
Example 2: What is the heart rate?
(300 / ~ 4) = ~ 75 bpm
Example 3: What is the heart rate?
12 x 10 = 120 bpm
IV. P Wave
Sequential activation of the RA & LA.
It is common to see notched or
biphasic P waves.
Normal (Lead II):
1. Wide < 2.5 mm (< 0.11 sec).
2. Notched Duration < 1 mm (0,045 sec).
3. Amplitudo < 2.5 mm ( 0.25 mV)
P Wave deflection
Should be upright in I, II, and V2 to V6
P wave axis
V. PR Interval
Time interval from onset of atrial depolarization to onset
of ventricular depolarization.
Normal 0.12-0.20 sec.
PR Interval abnormalities
I. Short PR interval (< 0.12 sec) Pre-excitation syndrome.
A. Lown-Ganong-Levine Syndrome.
B. Wolff-Parkinson-White Syndrome.
II. Long PR interval (> 0.20 sec)
A. AVB grade I.
B. AVB grade II type 2.
C. Trifascicular block
III. Irregular PR interval:
A. AVB grade II type 1.
B. TAVB
C. AV dissociation
VI. QRS complex
Simultaneous activation of the right and left ventricles.
Normal < 0.12 sec
Qr Rs rS qs rSr’ rSR’
QRS Morphology: Limb leads.
The QRS complex dominantly upright in I & II.
QRS & T waves tend to have the same general
direction in the limb leads.
QRS Morphology: Precordial leads
q-waves reflect normal septal activation (<0.04 sec & <25% R
amplitude) in I, II, V2 to V6
r-waves begin in V1 / V2 & progress in size to V5 at least V4 (R-V6 is
usually < R-V5).
S-waves begin in V6 or V5 and progress in size to V2. (S-V1 is usually
smaller than S-V2).
V1 V2 V3
V4
Sternum
V5
V6
Spine
QRS Axis
The normal QRS axis range -30 o to 110 o
Implies that the QRS be mostly positive (upright) in leads I
& II.
VII. ST Segment
Normal ST segment elevation occurs in leads with large S
waves (e.g., V1-3) with concave upward configuration .
The ST segment should start isoelectric except in V1 and
V2 where it may be elevated
Also be seen in other leads called early repolarization.
ST Segment Changes
ST Elevation
1. Acute myocardial Infarction
2. Myocarditis
3. Pericarditis
4. Aneurysm
5. Early repolarisation
ST Depression
1. Myocardial ischaemia.
2. Ventricular hypertrophy ("strain“).
3. Hypokalaemia
4. Digoxin effect
5. Non-specific
VIII. T Wave
Ventricular repolarisation.
The normal T wave :
1. Asymmetrical.
The first half more gradual slope than the second
half.
2. No widely accepted criteria exist regarding T
wave amplitude.
Generally ≥ 1/8 but < 2/3 of the the corresponding R
wave, rarely > 10 mm.
The tallest in leads V3 and V4.
T Wave
3. The orientation ~ the QRS complex.
Inverted in lead aVR.
May be inverted in lead III & V1 occasionally accompanied by V2
(isolated inversion in V2 is abnormal).
The T wave must be upright in I, II, V2 to V6.
Tall T wave
1. Acute myocardial ischaemia (hyper acute T).
2. Hyperkalaemia.
3. Early repolarization (normal variant).
Inverted T waves
1. Myocardial ischaemia
Symmetrical inverted highly suggestive.
Asymmetrical inverted non-specific finding.
2. Acute – recent myocardial Infarction.
3. Pericarditis.
4. Digoxin effect.
5. Hypokalaemia.
6. LV strain.
7. Persistent juvenile pattern (normal variant).
Inversion in V1-V2, common in blac k people.
IX. QT Interval
Duration of ventricular depolarization and repolarization
Normal QTc: < 0.44 sec (Roughly < ½ RR interval)
QTC = QT / RR.
Best measured in aVL
QT Interval Changes
Short QT
1. Hypercalcaemia
2. Digoxin
Long QT
1. Congenital LQTS
2. AMI
3. Cardiomyopathy
4. Myocarditis
5. MVP
6. Electrolyte disorders
7. Drugs
8. Hypothermia
9. CVA
10. Neck surgery
X. U Waves
Normal
1. Usually present in V2-3.
2. Height ± 80% T waves
U Waves changes
Prominent U wave (≥ 1 mm, or > T wave height)
1. Hypokalemia
2. Hypomagnesemia
3. Hyperthyroidism
4. Class I antiarrhythmics
5. Tricyclics, phenothiazines
6. CNS disease
7. Bradycardia
8. LVH
9. Digoxin
Inverted U wave
1. Ischaemic heart disease
2. Anterior ischaemia
3. Hypertensive heart disease
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