Professional Documents
Culture Documents
MODERN DRESSINGS
Presenter: Angela
Mentor: Dr Siek
Definition
• A disruption in the continuity of epithelium, with or without
loss of underlying connective tissue, following external
injury or intrinsic factors.
Phases of wound healing
Principles of Wound Bed Preparation
• Tissue
• Infection/inflammation
• Moisture
• Edge of wound
Falanga V. Classifications for wound bed preparation and stimulation of chronic wounds. Wound Repair Regen, 2000; 8(5): 347-52.
Schultz, G. S., Sibbald, R. G., Falanga, V., Ayello, E. A., Dowsett, C., Harding, K., Romanelli, M., Stacey, M. C., Teot, L. and
Vanscheidt, W. (2003), Wound bed preparation: a systematic approach to wound management. Wound Repair Regen, 11: S1–S28.
• Devitalized tissue/Debridement
• Infection/inflammation
• Moisture balance
• Wound Edge preparation
Sibbald RG, Woo KY, Ayello E. Wound bed preparation: DIM before DIME. Wound Healing Southern Africa 2008; 1: 29- 34.
Triangle of Wound Assessment (TOWA)
World Union of Wound Healing Societies (2016) Position Document. Advances in Wound Care: The Triangle of Wound Assessment.
Wounds International, London.
Factors affecting wound healing
Local factors Systemic factors
Mechanical trauma Advanced age
Infection Malnutrition
Edema Obesity
Dessication and dehydration Diabetes mellitus
Lack of oxygen Anemia
Continued pressure Vascular insufficiency
Presence of necrotic tissue Immunosuppression
Winter, GD. Formation of the Scab and the Rate of Epithelization of Superficial Wounds in the Skin of the Young Domestic
Pig. Nature. 1962;193:293-294.
Hydrocolloids
Foam
Hydrofibers
Negative pressure wound therapy (NPWT)
• Mechanism of action
– Removal of excess exudates
– Reduction of edema
– Increase in perfusion
– Promotes granulation tissue formation
– Reduction in bacterial load
– Enhances epithelial migration
• Contraindications
– Malignant wounds
– Untreated osteomyelitis
– Presence of necrotic and eschar tissue
– Non-enteric and unexplored fistulas
– Exposed blood vessels, anastomotic
sites, organs, or nerves
Wound edge assessment
• Edge of wound
Periwound skin assessment
• Surrounding skin
– Periwound skin is defined as skin within 4cm of the wound edge,
or any skin under the dressing
Modern Dressings
• Main characteristic of maintaining the moisture level is the foremost
advantage of modern dressings.
• Primary dressing: Dressing that comes directly in contact with the wound bed
• Secondary dressing: Dressing used to cover a primary dressing when the
primary dressing does not protect the wound from contamination
• Occlusive dressing: Covers a wound from the outside environment and keep
nearly all wound vapors at the wound site
• Semi-occlusive dressing: Allows some oxygen and moisture vapour to
evaporate
Choice of dressing
Purpose Dressings
Debridement Hydrogels
Hydrocolloids
Films
Antimicrobial Silver dressings
Iodine based dressings
Exudate control Hydrocolloids
Hydrofibers
Foam
Rehydration Hydrogels
Inert NA cotton wool • Protect new tissue growth Dry or low-exuding wounds • Use as contact layer on superficial
dressings • Absorb minimal exudate low-exuding wounds
• Will not cope with moderate or higher
levels of exudate
Polyurethane film • Moisture control • Primary dressing over superficial, • Do not use on patients with
• Breathable bacterial low-exuding wounds fragile/compromised periwound skin
barrier • Secondary dressing over alginate or • Do not use on moderate- to high-
• Transparent (allow hydrogel for rehydration of wound exuding wounds
visualisation of wound) bed
Hydrogels • Rehydrate wound bed • Dry/low- to moderate-exuding • Do not use on highly exuding wounds
• Moisture control wounds or where anaerobic infection is
• Promote autolytic • Combined presentation with silver suspected
debridement for antimicrobial activity • May cause maceration
Hydrocolloids • Absorb fluid Clean, low- to moderate-exuding • Do not use on dry/necrotic wounds or
• Promote autolytic wounds high-exuding wounds
debridement • May encourage overgranulation
• May cause maceration
Alginates/CMC • Absorb fluid • Moderate- to high-exuding wounds • Do not use on dry/necrotic wounds
• Promote autolytic • Special cavity presentations in the • Use with caution on friable tissue
• debridement form of rope or ribbon (may cause bleeding)
• Moisture control • Combined presentation with silver • Do not pack cavity wounds tightly
• Conformability to wound for antimicrobial activity
bed
Types Actions Indications Precautions/Contraindications
Foams • Absorb fluid • Moderate- to high-exuding wounds • Do not use on dry/necrotic wounds
• Moisture control • Special cavity presentations in the or those with minimal exudate
• Conformability to wound form of strips or ribbon
bed • Low-adherent versions available for
patients with fragile skin
• Combined presentation with silver
or PHMB for antimicrobial activity
Foam-like • Absorb fluid • Moderate- to high-exuding wounds • Do not use on dry/necrotic wounds
hydroactive • Moisture control • Special cavity presentations in the or those with minimal exudate
dressings • Conformability to wound form
bed • Low-adherent versions available for
• Similar but not the same patients with fragile skin
as a foam
Silver Antimicrobial action • Critically colonised wounds or • Some may cause discolouration
clinical signs of infection • Known sensitivity to silver
• Low- to high-exuding wounds • Discontinue after 2 weeks if no
• Combined presentation with foam improvement and re-evaluate
and alginates/CMC for increased
absorbency
Iodine • Antimicrobial action • Critically colonised wounds or • Do not use on dry necrotic tissue
• Debrider clinical signs of infection • Known sensitivity to iodine
• Healing stimulation • Low- to high-exuding wounds • Short-term use recommended
• 3 months (there is a risk of systemic
absorption in larger wound with
prolonged use)
References
1. Leaper DJ, Harding KG. (1998) Wounds: Biology and Management. Oxford University Press.
2. Hutchinson J (1992). The Wound Programme. Centre for Medical Education: Dundee.
3. Classification of surgical procedures by risk of infection (Mangram et al., 1999)
4. Lazarus GS, Cooper DM, Knighton DR, et al. Definitions and guidelines for assessment of wounds
and evaluation of healing. Arch Dermatol. 1994;130(4):489–493.
5. Falanga V. Classifications for wound bed preparation and stimulation of chronic wounds. Wound
Repair Regen, 2000; 8(5): 347-52.
6. Schultz, G. S., Sibbald, R. G., Falanga, V., Ayello, E. A., Dowsett, C., Harding, K., Romanelli, M.,
Stacey, M. C., Teot, L. and Vanscheidt, W. (2003), Wound bed preparation: a systematic approach to
wound management. Wound Repair Regen, 11: S1–S28.
7. Sibbald RG, Woo KY, Ayello E. Wound bed preparation: DIM before DIME. Wound Healing Southern
Africa 2008; 1: 29- 34.
8. World Union of Wound Healing Societies (2016) Position Document. Advances in Wound Care: The
Triangle of Wound Assessment. Wounds International, London.
9. Winter, GD. Formation of the Scab and the Rate of Epithelization of Superficial Wounds in the Skin of
the Young Domestic Pig. Nature. 1962;193:293-294.
10. Thomas A, Harding KG, Moore K Biomaterials. 2000 Sep; 21(17):1797-802.