Professional Documents
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Respiratory System
Primary functions
a. provides oxygen for metabolism in the tissues
b. removes carbon dioxide, the waste product of metabolism
2 Main Parts
NOSE
1. Filters, warms and humidifies air
2. First defense against foreign particles
3. Inhalation for deep breathing is to be done via nose
4. Exhalation is done through the mouth
5. Serves as passageway for incoming and outgoing air, filtering, warming, moistening, and chemically examining it.
6. Organ of smell (Olfactory receptors located in the nasal mucosa
7. Aids in phonation
PHARYNX
1. Serves as a passageway and entrance to the respiratory and digestive tracts
2. Aids in Phonation
3. Tonsils function to destroy incoming bacteria and detoxify certain foreign proteins
LARYNX
1. Voice production: during expiration, air passing through the larynx cause the vocal cords to vibrate; short, tense cords
2. Serves as a passageway for air and as the entrance to the lower respiratory tract
TRACHEA”windpipe”
1. tube about 4 inches long
2. begins just under the larynx (voice box) and runs down behind the breastbone (sternum).
3. divides into two smaller tubes called bronchi: one bronchus for each lung.
4. composed of about 20 rings of tough cartilage
5. widens and lengthens slightly with each breath in, returning to its resting size with each breath out
BRONCHUS
1. a passage of airway in the respiratory tract that conducts air into the lungs
2. branches into smaller tubes, which in turn become bronchioles
3. No gas exchange takes place in this part of the lungs
BRONCHIOLES
1. first passageways by which the air passes through the nose or mouth to the air sacs of the lungs in which branches no longer
contain cartilage or glands in their submucosa
2. they are branches of the bronchi. The bronchioles terminate by entering the circular sacs called alveoli
LUNGS
1. a pair of spongy, air-filled organs located on either side of the chest (thorax).
2. covered by a thin tissue layer called the pleura. The same
3. A thin layer of fluid acts as a lubricant allowing the lungs to slip smoothly as they expand and contract with each breath.
Diagnostic Tests
1. Spirometry
2. Arterial blood gas determination
3. Oximeters
4. Exercise tolerance
5. Radiography
6. Bronchoscopy
7. Culture, sensitivity tests
PNEUMONIA
– inflammation of lung parenchyma leading to pulmonary consolidation as alveoli is filled with exudates.
High risk
-elderly & children below 5 y/o
Etiologic agents
1. Streptococcus pneumoniae (pnemococcal pneumonia)
2. Hemophilus pneumoniae(Bronchopneumonia)
3. Escherichia coli
4. Klebsiella P.
5. Diplococcus P.
Predisposing factors
1. Smoking
2. Air pollution
3. Immuno-compromised
4. Prolonged immobility – CVA- hypostatic pneumonia
5. Aspiration of food
6. Over fatigue
Diagnostic
1. Sputum (GSCS)- gram staining & culture sensitivity - Reveals (+) cultured microorganism.
2. Chest X-ray – pulmo consolidation
3. CBC – increase WBC
4. Erythrocyte sedimentation rate
5. ABG – PO2 decrease
Nursing Management
1. Enforce Complete Bed Rest
2. Strict respiratory isolation
3. Meds:
a.) Broad spectrum antibiotics
Penicillin or tetracycline
Macrolides – ex azythromycin (zythromax)
b.) Anti pyretics
c.) Mucolytics or expectorants
4. Force fluids – 2 to 3 L/day
5. Institute pulmonary toilet-
a.) Deep breathing exercise
b.) Coughing exercise
c.) Chest physiotherapy – cupping
d.) Turning & reposition - Promote expectoration of secretions
6. Semi-fowler
7. Nebulize & suction
8. Comfortable & humid environment
9. Diet: increase CHO or calories, CHON & Vit. C
10. Postural drainage - To drain secretions using gravity
Predisposing factors
1. Malnutrition
2. Overcrowding
3. Alcoholism
4. Ingestion of infected cattle (mycobacterium BOVIS)
5. Virulence
11. Over fatigue
Diagnosis
1. Skin test – Mantoux test – infection of Purified CHON Derivative PPD
DOH – 8-10 mm induration
WHO – 10-14 mm induration
Result within 48 – 72h
(+) Mantoux test – previous exposure to tubercle bacilli
Nursing Management
1. Complete Bed Rest
2. Strict respiratory isolation
3. O2 inhalation
4. Semi fowler’s position
5. Force fluid to liquefy secretions
6. Nebulize & suction
7. Comfortable & humid environment
8. Diet – increase CHO & calories, CHON, Vits., minerals
9. Short course chemotherapy
a. Isoniazid (INH)
b. Rifampin (RIF)
c. Ethambutol
d. Pyrazinamide
e. Streptomycin
HISTOPLASMOSIS
- acute fungal infection caused by inhalation of contaminated dust with histoplasma capsulatum transmitted to birds manure.
Signs and Symptoms
Same as pneumonia & PTB – like
1. Productive cough
2. Dyspnea
3. Chest & joint pains
4. Cyanosis
5. Anorexia, gen body malaise, wt loss
6. Hemoptysis
Diagnostic
1. Histoplasmin skin test = (+)
2. ABG – pO2 decrease
Nursing Management
1. Complete Bed Rest
2. Meds:
a.) Anti fungal agents
b.) Corticosteroids
c.) Mucolytic/ or expectorants
3. O2 – force fluids
4. Nebulize, suction
5. Complications:
a.) Atelectasis
b.) Bronchiectasis COPD
6. Prevent spread of histoplasmosis:
a.) Spray breeding places or kill the bird
CHRONIC BRONCHITIS
- called BLUE BLOATERS inflammation of bronchus due to hypertrophy or hyperplasia of goblet mucus producing cells leading to narrowing of
smaller airways.
Predisposing factors
1. Smoking – all COPD types
2. Air pollution
Diagnostic
1. ABG
Nursing Management
(Same as emphysema)
1. Complete Bed Rest
2. Administer medications as ordered
a.) Bronchodilators
b.) Corticosteroids
c.) Antimicrobial agents
d.) Mucolytics/ expectorants
3. O2 – Low inflow
4. Force fluids
5. High fowlers
6. Nebulizer & suction
7. Institute
a. P – posture
b. E – end
c. E – expiratory
d. P – pressure
8. Health Teaching:
a.) Avoid smoking
b.) Prevent complications
1.) Cor pulmonary – R ventricular hypertrophy
2.) CO2 narcosis – lead to coma
3.) Atelectasis
4.) Pneumothorax – air in pleural space
c.) Adhere with the medications
BRONCHIAL ASTHMA
- reversible inflammation lung condition due to hyerpsensitivity leading to narrowing of smaller airway
2 Types
1. Extrinsic asthma
a. Acute episodes triggered by type I hypersensitivities
b. Onset in childhood
2. Intrinsic asthma
a. Onset during adulthood
b. Stimuli target hyperresponsive tissue = acute attack
Predisposing factor
1. Extrinsic Asthma – called Atropic/ allergic asthma
a.) Pallor
b.) Dust
c.) Gases
d.) Smoke
e.) Dander
f.) Lints
2. Intrinsic Asthma -
Cause:
a.) Herediatary
b.) Drugs – aspirin, penicillin, blockers
c.) Food additives – nitrites
d.) Foods – seafood, chicken, eggs, chocolates, milk
e.) Physical/ emotional stress
f.) Sudden change of temp, humidity &air pressure
Diagnostic
Nursing Management
1. Complete Bed Rest for all types of COPD
2. Meds-
a.) Bronchodilator through inhalation or metered dose inhaled / pump. Give 1st before corticosteroids
b.) Corticosteroids – due inflammatory. Given 10 min after administration of bronchodilator
c.) Mucolytic/ expectorant
d.) Mucomist – at bedside put suction machine.
e.) Antihistamine
3. Force fluid
4. O2 – all COPD low inflow to prevent respiratory distress
5. Nebulize & suction
6. Semifowler – all COPD except emphysema due late stage
7. Health teachings
a.) Avoid predisposing factors
b.) Complications:
a.) Status astmaticus- give epinephrine & bronchodilators
b.) Emphysema
c.) Adherence to medications
BRONCHIECTASIS – abnormal permanent dilation of bronchus resulting to destruction of muscular & elastic tissues of alveoli.
Predisposing factors
1. Recurrent upper & lower RI
2. Congenital anomalies
3. Tumors
4. Trauma
Diagnostic
1. ABG – PO2 decrease
2. Bronchoscopy – direct visualization of bronchus using
fiberscope
Nsg Mgt: before bronchoscopy
1. Consent, explain procedure – MD/
lab explain RN
2. NPO
3. Monitor VS
Nsg Mgt after bronchoscopy
1. Feeding after return of gag reflex
2. Instruct client to avoid talking, smoking or coughing
3. Monitor signs of frank or gross bleeding
4. Monitor of laryngeal spasm
DOB
Prepare at bedside tracheostomy set
PULMONARY EMPHYSEMA
– irreversible terminal stage of COPD
– Characterized by inelasticity of alveolar wall leading to air trapping, leading to maldistribution of gases.
– Body will compensate over distension of thoracic cavity
– Barrel chest
Predisposing factor
1. Smoking
2. Allergy
3. Air pollution
4. High risk – elderly
5. Hereditary
Diagnosis
1. Pulmonary function test – decrease vital lung capacity
2. ABG
Nursing Management
1. Complete Bed Rest
2. Administer medications as ordered
a. Bronchodilators
b. Corticosteroids
c. Antimicrobial agents
d. Mucolytics/ expectorants
3. O2 – Low inflow
4. Force fluids
5. High fowlers
6. Nebulize & suction
7. Institute
P – posture
E – end
E – expiratory
P – pressure
8. Health teachings
a.) Avoid predisposing factors
b.) Complications:
c.) Status astmaticus- give epinephrine & bronchodilators
d.) Emphysema
c.) Adherence to medications
PNEUMOTHORAX
– partial / or complete collapse of lungs due to entry or air in pleural space.
Types:
1. Spontaneous Pneumothorax – entry of air in pleural space without obvious cause
Eg. rupture of bleb (alveoli filled sacs) in pt with inflammed lung conditions
Eg. open pneumothorax – air enters pleural space through an opening in chest wall
-Stab/ gun shot wound
2. Tension Pneumothorax – air enters plural space with at inspiration & can’t escape leading to over distension of thoracic cavity
resulting to shifting of mediastinum content to unaffected side.
Eg. flail chest – “paradoxical breathing”
Predisposing factors
1.Chest trauma
2.Inflammatory lung conditions
3.Tumor
Diagnosis
1. ABG – pO2 decrease –
2. Chest X-ray – confirms pneumothorax
Nursing Management
1. Endotracheal intubation
2. Thoracenthesis
3. Meds – Morphine SO4
- Anti microbial agents
4. Assist in test tube thoracotomy