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Epidemiology of Stuttering in

the Community Across the


Entire Life Span

Ashley Craig
Karen Hancock
A randomized and stratified investigation was conducted into the epidemiology of
Yvonne Tran stuttering in the community across the entire life span. Persons from households in
Magali Craig the state of New South Wales, Australia, were asked to participate in a telephone
Karen Peters interview. Consenting persons were given a brief introduction to the research, and
Department of Health Sciences details were requested concerning the number and age of the persons living in
University of Technology the household at the time of the interview. Interviewees were then given a
Sydney, Australia description of stuttering. Based on this description, they were asked if any person
living in their household stuttered (prevalence). If they answered “yes,” a number
of corroborative questions were asked, and permission was requested to tape
over the telephone the speech of the person who stutters. Confirmation of
stuttering was based on (a) a positive detection of stuttering from the tape and (b)
an affirmative answer to at least one of the corroborative questions supporting the
diagnosis. Results showed that the prevalence of stuttering over the whole
population was 0.72%, with higher prevalence rates in younger children (1.4–
1.44) and lowest rates in adolescence (0.53). Male-to-female ratios ranged from
2.3:1 in younger children to 4:1 in adolescence, with a ratio of 2.3:1 across all
ages. The household member being interviewed was also asked whether anyone
in the household had ever stuttered. If the answer was “yes,” the same corrobora-
tive questions were asked. These data, along with the prevalence data, provided
an estimate of the incidence or risk of stuttering, which was found to range from
2.1% in adults (21–50 years) to 2.8% in younger children (2–5 years) and 3.4%
in older children (6–10 years). Implications of these results are discussed.
KEY WORDS: stuttering, incidence, prevalence, fluency disorders

I
t is important that clinicians, researchers, and health administra-
tors know the prevalence and incidence (risk) of a disorder in the
community in order to allocate sufficient resources for managing prob-
lems associated with that disorder. Stuttering is a communication dis-
order involving involuntary disfluency. It is ordinarily diagnosed early—
around age 2 in the majority of cases—and it can become a chronic
condition for up to 20% of those who stutter in their childhood (Andrews
et al., 1983; Bloodstein, 1995). Therefore, resources must be allocated to
manage stuttering in young children, adolescents, and adults. However,
the extent of the population who stutter over the total life span is not
clear, as the prevalence of stuttering in the community has only been
estimated based on studies of children (Bloodstein, 1995).
In this paper, prevalence is defined as the number of confirmed cases
of stuttering in a sample at the time the sample is interviewed. This is
known as point prevalence (Slome, Brogan, Eyres, & Lednar, 1986, p. 34).

Journal of Speech, Language, and Hearing Research • Vol. 45 • 1097–1105 • December 2002 • ©AmericanCraig
Speech-Language-Hearing
et al.: Epidemiology ofAssociation
Stuttering 1097
1092-4388/02/4506-1097

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Incidence is usually defined as the occurrence of new 0.23% in girls). Aron (1962), using teacher referral to
cases during a specified period of time (Slome et al., 1986, select children who stutter, surveyed 6,581 schoolchil-
p. 98). Although the research reported in this paper is dren (3,105 males and 3,476 females from 6 to 21 years
not prospective in design and is therefore not primarily old) in 13 schools in Johannesburg and found an over-
designed to assess the risk of stuttering (Moscicki, 1984), all prevalence rate of 1.26% (83 who stuttered, 62
it may be possible to use the data to compute an esti- males and 21 females). Gillespie and Cooper (1973)
mate of the risk. This estimate was determined from surveyed junior high and high school students in Ala-
the number of confirmed stuttering cases at the time of bama. They interviewed 5,054 students (grades 7–12
interview (prevalence) and the number of persons who by convenience sampling) and found a prevalence rate
stuttered in the past but who did not stutter at the time of 2.1%. Boyle, Decoufle, and Yeargin-Allsopp (1994)
of the interview. Incidence is, therefore, defined as the conducted a large survey of 17,110 children, from the
risk of the person in the sample population ever stutter- very young to age 17. Children were invited to partici-
ing over the period of the person’s life span; it is based pate from a register established by the National Health
on confirmed cases of current stuttering as well as cases Interview Survey (NHIS). The NHIS involves an on-
of past stuttering. going survey of households within the United States
This paper does not attempt to review the large and uses a sampling procedure that is believed to be
number of prevalence studies that have been conducted representative of the civilian non-institutionalized
over the past century. For this, the reader is referred to population (Boyle et al., 1994). The study was designed
Bloodstein (1995, pp. 105–107), who has provided a suc- to assess a number of disabilities, such as developmen-
cinct review. The aim of this paper is to analyze a small tal disability, deafness, learning disability, and stut-
number of more recent studies that have contributed to tering. The researchers found a stuttering prevalence
the current understanding of the epidemiology of stut- of 1.89% (N = 297). In contrast, Brady and Hall (1976)
tering. The review concentrates on prevalence rates in conducted a survey on a very large sample (N =
children, adolescents, and children/adolescents with dis- 187,420) of schoolchildren in Illinois and found only a
ability in addition to stuttering, in that order. The sex 0.35% prevalence rate.
ratio and risk of stuttering are also addressed. Of the above seven studies, the mean prevalence
Although a number of studies determined childhood rate in children is 1.29%. However, Bloodstein (1995)
stuttering prevalence, there are few, if any, randomized, listed 17 U.S. studies that report a mean prevalence rate
stratified studies that investigated the extent of stut- of 0.97% for school-age children and 21 non-U.S. stud-
tering in the total population (Andrews et al., 1983; ies that report a mean prevalence rate of 1.28%. Andrews
Bloodstein, 1995). Currently, estimates of prevalence et al. (1983) suggested the difference between the two
are based on cross-sectional or survey research on rates may be due to the higher proportion of U.S. chil-
schoolchildren. For example, Andrews and Harris (1964) dren who remain in school after puberty. Conversely,
surveyed 7,358 schoolchildren from 9 to 11 years old in the difference may also be due to error arising from the
Newcastle upon Tyne, England. They relied on school- sampling and assessment methods used by many of
teachers to identify children who stuttered and then these studies. When the sample is not randomized and
assessed each child selected by the teacher. Although stratified, it is not a reliable representation of the
86 children were identified by teachers, only 80 chil- broader population.
dren were subsequently confirmed as children who stut- Although adolescent prevalence rates have rarely
ter. Bloodstein (1995, p. 107) reports a prevalence of been studied, Ardila et al. (1994) found a 2% prevalence
1.2% for this study (based on 86 children), but the cor- rate in 1,879 Spanish-speaking university students.
rect prevalence rate is almost 1.1% based on 80 chil- Porfert and Rosenfield (1978) found a 2.1% prevalence
dren. Leavitt (1974) conducted two surveys among rate in 2,107 students at a U.S. university. However,
Puerto Rican schoolchildren. The first was in 12 schools both studies employed non-randomized sampling,
in San Juan, Puerto Rico (N = 10,449, comprising 5,476 thereby increasing the likelihood of obtaining unrepre-
boys and 4,973 girls). The second involved Puerto Rican sentative samples, and both studies failed to confirm
children from 19 schools in New York (N = 10,455, with the stuttering cases.
5,270 boys and 5,185 girls). Leavitt also relied on school- Prevalence rates have varied widely in studies con-
teachers to identify children who stuttered and then ducted on populations with disabilities. Stansfield (1990)
conducted assessments to confirm the diagnosis. She conducted a non-randomized survey of adult psychiat-
found an overall prevalence rate of 1.5% in the Puerto ric wards and relied on medical staff to send back infor-
Rican children living in San Juan (2.17% in boys and mation on patients who had speech problems. Stansfield
0.76% in girls) and an overall rate of 0.84% in the Puerto then assessed the speech of a percentage of those believed
Rican children living in New York (1.44% in boys and to have speech problems and found a 6.3% prevalence

1098 Journal of Speech, Language, and Hearing Research • Vol. 45 • 1097–1105 • December 2002

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rate of stuttering in the adult psychiatric population. In summary, little is known about the prevalence or
Montgomery and Fitch (1988) sent survey forms to 150 incidence of stuttering throughout the entire life span.
schools for the hearing impaired. Only children who had It is important to conduct research designed to estimate
been diagnosed as having a stutter by a qualified speech reliably the number across all ages as well as the sex
professional were counted in the survey. Just over half ratio of those persons who stutter in the community. This
the schools replied. Only 12 children out of a total of paper presents a study that investigated the prevalence
9,930 were reported to have a stutter, and all but one and sex ratio of stuttering in an ethnically heterogeneous
had congenital hearing loss. Montgomery and Fitch sample. The study also provided an estimate of the inci-
found a prevalence rate of 0.12% in school-age children dence of stuttering.
with hearing disorders. Due to the non-randomized sam-
pling methods used, these two studies do not provide
reliable stuttering population rates for disabled persons. Method
Male-to-female ratios were investigated by Aron Participants and Random
(1962), who found a 2.9:1 (male:female) child ratio;
Andrews and Harris (1964) found a similar ratio.
Sampling Procedure
Gillespie and Cooper (1973) found a child male-to-fe- The study consisted of a random and stratified selec-
male ratio of 2.7:1, and Porfert and Rosenfield (1978) tion of households in New South Wales (NSW), Australia.
found a ratio of 2.4:1 in university students. The major- The population of NSW consisted of approximately 6 mil-
ity of studies have found child stuttering male-to-female lion people during 1995 to 1996 when the data were col-
ratios of about 2.5:1 to 3:1 (Bloodstein, 1995). lected. The population was composed of primarily city and
Incidence studies are less common than prevalence suburban dwellers (74%). Almost 77% of people in NSW
studies. Andrews and Harris (1964) investigated inci- were born in Australia, though the population is ethni-
dence in a sample of 1,000 children followed from birth cally diverse, with the most common groups in NSW in
up to age 15 in England (incidence was defined as the rank order being people of European/British, Asian,
percentage of the 1,000 children who stuttered at any Middle Eastern, and Indian descent. Families living in
time in their life). However, there was a 25.8% attri- city, suburban, and rural areas across NSW were randomly
tion rate over the first 5 years. The resulting sample of selected, so that (a) all families had equal chances of be-
875 included 43 children who were thought to stutter, ing selected and (b) the distribution of people in the sample
providing a 4.9% risk estimate. Ingham (1976) ques- from these three types of areas was proportional to the
tioned the reliability of the incidence data from this known population spread in NSW.
study for several reasons, including uncertainty Our hypothesized proportion of people who stutter
whether 16 of the 43 children actually stuttered. If the in the community was 1% (0.01), and we planned to
16 are removed, the estimate of the lifetime risk of stut- identify up to 100 people who stutter (as this would
tering in the Andrews and Harris (1964) study reduces provide sufficient and stable numbers of people who
to about 3.1%. stutter to determine the epidemiology of stuttering).
Mansson (2000) studied the incidence of stuttering This implies we needed a sample size of 10,000 (100/
in Denmark. All children born on the island of Bornholm 0.01) people from whom to collect epidemiological data
in 1990 and 1991 were assessed for stuttering when they regarding stuttering. As stuttering is now believed to
turned 3 years old. The total population of this island be genetic in origin (Ambrose, Cox, & Yairi, 1997), the
was 45,000. Although 1,042 children were born, only randomness of the selection process could be negatively
1,021 participated in the third year. Fifty-one were be- influenced by cluster sampling. Therefore, we aimed to
lieved to stutter, and only two additional cases were iso- collect a larger sample than 10,000 in order to over-
lated in surveys conducted 2 years later (age 5) and 4 come any possible influence of cluster sampling. The
years later (age 9). As a result, Mansson suggested the final number of participating families was 4,689, con-
incidence for this group of children was 5.19%. This study sisting of 12,131 individuals.
is to be commended for its prospective design and sam- Families were selected (using 1995/96 telephone
pling rate. However, Mansson acknowledged that the directories) until enough households had been contacted
island’s geographic isolation and homogeneous ethnic who agreed to participate in the survey. In accordance
population limit the generalizability of the findings. with the population distribution of NSW (Australian
Mansson also failed to report reliability of the assess- Bureau of Statistics, 1998), three-quarters of the sample
ment of stuttering or a definition of stuttering. Based were from city/suburban areas in NSW, whereas the
on a number of studies, Bloodstein (1995) and Andrews remaining sample came from rural areas in NSW. Table
et al. (1983) concluded that the risk of stuttering is 1 shows the regions sampled. If a survey is strategi-
around 4 to 5%. cally conducted according to known distributions of a

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Table 1. Breakdown of regions in New South Wales that were was followed (see results for response rate). The time and
selected for the survey. day of interviews was varied through the week to ensure
a high penetration rate.
Region Males Females Total

Sydney region 3,625 3,648 7,274


Central coast 277 259 536
Stuttering Definition and
South coast 111 115 226 Interviewing Procedure
Windsor area 85 80 165
Five professionals were trained to conduct the in-
Campbelltown area 289 272 561
terviews. An interview began with a brief scripted state-
Penrith area 298 312 610
Southern tablelands 103 110 213
ment of the purpose of the survey and identified the
Albury area 96 112 208 university represented. The scripted introduction in-
Bathurst area 176 169 346 cluded informed verbal consent—the interviewer asked
Cooma area 78 79 157 the person who answered the telephone for permission
Singleton area 288 269 557 to be interviewed. If the person gave consent but said
Northern rivers 186 173 359 that another time was more convenient, a new time was
New England area 158 186 344 arranged for the interviewer to call. If a young child
Western plains 102 119 221 answered the telephone, the interviewer asked to speak
Southwest plains 128 160 288 to a parent for the interview. All refusals were recorded
Broken Hill area 35 31 66
as missing data as were “no answers.” The interview
Total 6,035 6,096 12,131
was structured to include forced-choice questions so that
Note. The number of persons interviewed in each region was based on responses could be categorized. The results from each
the population distribution across the state so that the numbers sampled telephone interview were recorded on provided response
in each area of the study were proportional to the population spread forms (see the questionnaire in the Appendix).
(total N = 12,131).
Stuttering was defined to all interviewees in detail
using a standard definition (Craig et al., 1996). It was
defined as repetitions of syllables, part or whole words
population, then regional coverage rates are higher and
or phrases; prolongations of speech; or blocking of
therefore are believed to be valid representations of
sounds. Associated symptoms such as embarrassment
the population (Cannell, 1985).
and anxiety were also discussed. If requested, the inter-
Households were contacted by telephone and inter- viewer gave a demonstration over the telephone of a
views were then conducted (either during this initial con- repetition and a block. The interviewees (if not children)
tact or at a convenient follow-up time). As over 95% of were asked whether they or a member of their house-
Australian households had a telephone in 1995/96, a high hold stuttered. If they were not sure, they were encour-
penetration rate in the community was assured. There- aged to speak to other members of the family and an
fore, only a small chance existed of introducing popula- alternative time was arranged to call to complete the
tion bias into the sample. The procedure used (Dillman, interview. If the people answering the telephone believed
1978) involved the estimation of the total length of col- they or a member of their household stuttered, a num-
umns of listings for each of the telephone directories used ber of corroborative questions were then asked directly
in NSW. A sampling distance (in centimeters) was com- of the person believed to stutter, or of parents if a young
puted by dividing this estimated length by the proposed child stuttered. Questions included (a) “Has the stut-
sample size from that region. For each telephone direc- tering persisted for the last three months?”, (b) “Has
tory, a different starting point was selectd using random the stuttering caused fear and avoidance of situations?”,
numbers. This proposed sample size was then used as (c) “Has the person consulted a speech professional?”,
the sampling interval between each pair of selected tele- and (d) “Has the person had therapy for stuttering?” If
phone numbers in the telephone books. Using this proce- someone in the family was believed to stutter, the inter-
dure ensures that most of names in each directory have viewer asked for permission to speak to that member
the potential to be selected for the study (Dillman, 1978). and to tape the person’s speech over the telephone for
Table 1 shows the breakdown for the regions sampled as up to 5 minutes. If the person was a child, the inter-
well as the final sample sizes surveyed from each region. viewer asked the parents for permission to tape their
Families in which no member could speak English well child’s speech. Taping involved the interviewer engag-
enough to complete the interview were not included in ing the person believed to stutter in conversation for at
the study. If this occurred, the interviewer noted this and least 5 minutes, during which they taped the person’s
proceeded to another number. Telephone numbers that speech. During this interview, the interviewer also lis-
were disconnected or where there was no answer after tened for stuttering.
three attempts were also noted and the same procedure

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As a person who stutters may be less likely to an- the interviewer decided whether the person stuttered.
swer the telephone, it is probable that someone in the The interviewer taped the speech using digital technol-
house who does not stutter will answer. If the person ogy (Sony Pro II TCD D10, Sony condenser microphone,
answering believed he or she or someone else in the ECM-MS5) to ensure high-quality recording. The inter-
household had stuttered at some previous time (that is, viewer then passed the tape to a co-researcher “rater”
is not presently stuttering), the interviewer asked for who had over 15 years of experience in treating and di-
permission to speak to the person who formerly stut- agnosing stuttering. The rater qualitatively and quan-
tered and asked this person similar corroborative ques- titatively evaluated the tape of the participant’s speech.
tions. In most cases, the interviewer was able to speak If both the interviewer and the rater agreed that the
to the person who was believed to have formerly stut- person stuttered, they determined the frequency of stut-
tered. If not, the interviewer requested the information tering (percent syllables stuttered or %SS) and speech
from a person in the household who could answer the rate (syllables per minute or SPM). This was the pri-
corroborative questions. mary measure of stuttering. To ensure reliability, an
independent rater (that is, neither the interviewer nor
the original rater) assessed a proportion of the rated
Reliability of the Interviews tapes for %SS and SPM. The interrater reliability of the
Reliability of the survey itself was also an impor- rater was demonstrated. The interrater reliability, us-
tant consideration, and a 1-week test-retest reliability ing Pearson correlation coefficients with the second ex-
measure conducted on 15 interviews showed 100% agree- perienced rater, was 0.93 for %SS and 0.91 for SPM on
ment on the prevalence interview. This involved recon- 15 randomly selected tapes (that is, 17% of the total
tacting families who had participated in the study. In- number of tapes). If stuttering was diagnosed from the
terviewers were trained in the interview protocols, with tape, the corroborative evidence mentioned above was
emphasis placed on establishing rapport with the re- then used to confirm the diagnosis of stuttering. The
spondent. Interview structure and rapport are impor- rater and independent rater demonstrated over 96%
tant in ensuring the validity and reliability of telephone agreement on the diagnosis of stuttering from the tape.
sampling compared to face-to-face and self-administered When there was disagreement or no corroborative evi-
modes (Quine, 1985). Traditionally, personal interviews dence, the case was not considered confirmed as a per-
are regarded as more valid and reliable than telephone son who stuttered. Only confirmed stuttering cases were
interviews. However, several studies reported no signifi- used in the analysis to determine prevalence and to es-
cant differences between these modes of interview in timate incidence. Obviously, taping speech on the tele-
outcome and also in socio-demographic data obtained phone will not assess secondary aspects of stuttering
(Aneshensel, Frerichs, Clark, & Yokopenic, 1982; such as facial grimace, and so on. However, secondary
Cannell, 1985; Paulsen, Crowe, Noyes, & Pfohl, 1988; symptoms are extremely variable and are not generally
Quine, 1985). Low response rate is a possible disadvan- considered necessary to measure frequency of stutter-
tage when conducting telephone interviews, but it is a ing (Bloodstein, 1995).
disadvantage that besets all modes of interview. Stud-
ies conducted in the United States report lower response
rates of only 3–5% for telephone interviewing compared Results
with face-to-face interviewing (Cannell, 1985). Telephone
Response rate for telephone calls was considered
samples give higher response rates than mail surveys
satisfactory, with 63% of the telephone numbers initially
and share many of the advantages of the face-to-face
selected resulting in completed interviews (for a minor-
interview over the self-completed questionnaire. In this
ity of numbers interviews were completed after 2–3
study, no one who stuttered refused to be interviewed.
calls). This meant that for 37% of initially selected tele-
phone numbers, a second or third alternate telephone
Validation and Reliability of number had to be randomly selected. Of the 37% of calls
Stuttering Assessment where interviews did not occur, 19% of respondents re-
fused, giving no reason, 0.15% were too busy or unwell,
An accurate determination of prevalence and risk and 6.9% did not speak English well enough to com-
depends on a valid and robust method of detecting stut- plete an interview. The remaining 10.95% could not be
tering. The five interviewers were trained in the inter- contacted due to disconnected lines or unanswered calls.
view protocol and in the detection of stuttering. All fam- After three attempts without contact, a new number was
ily or household members who were believed to stutter selected.
were consequently interviewed for up to 5 minutes or
The mean %SS for the sample of those who stut-
until at least 500 syllables were taped. During the in-
tered (n = 87) was 5.04 %SS (range of 0.5 to 24.7, SD =
terview, and based on the above definition of stuttering,

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3.8), and the mean SPM was 169 (range of 88 to 218, Prior cases of stuttering (those believed to stutter
SD = 26). All interviewees whose stuttering was con- in the past) are also shown in Table 2 . This allows es-
firmed were asked if they had ever sought treatment timation of the incidence of stuttering. Risk over the
(e.g., speech pathology, medical, psychological, hypno- entire sample was 2.2%, with, as would be expected,
therapy) for their stuttering, and around 30% replied the highest risk in young children (2.8% in 2- to 5-year-
that they had at some time received treatment (just olds) and older children (3.4% in 6- to 10-year-olds).
over 50% had never received treatment and about 20% Lowest risk was in those over 50 years old (1.8%). The
were unsure). Frequency of stuttering was shown to risk in 5- to 18-year-olds (n = 2553 with 71 current-
correlate significantly and positively with seeking treat- stuttering and prior-stuttering cases) was determined
ment (point biserial r = 0.23, p < .05). This suggests to be 2.8% (95% CI of 2.16 to 3.44). Data on recovery
that those with more severe stuttering had sought treat- rates can also be derived from this research. Table 2
ment. Table 2 also shows age and sex breakdowns in shows that there were 87 people identified as currently
the sample. The sample of 4,689 families consisted of stuttering and 176 identified as stuttering in the past.
12,131 persons (6,023 males, mean age 35.5 years, SD Therefore, these data provide a 67% recovery rate over
= 21.6, age range from less than 1 year to 95 years; the entire life span (176/263).
6,108 females, mean age 37.3, SD = 22.1; age range
from less than 1 year to 99 years). There was an aver-
age of 2.9 members in each of the 4,689 families inter- Discussion and Conclusion
viewed. There was a total of 1,638 participants age 10
or younger (13.5%)—though only children who were at This paper presents epidemiological data on the
least 2 years old were included, as stuttering usually prevalence, sex ratio, and estimated risk of stuttering.
begins around age 2—resulting in a sample of 1,622. The study was conducted within an ethnically hetero-
There was a total of 1,881 participants age 11 to 20 geneous population in Australia, strengthening its ap-
(15.5%), and the majority of participants (n = 8,612) plicability to other populations that are also ethnically
were age 21 or over (71%). heterogeneous. Furthermore, it provides the first at-
tempt to estimate the prevalence of stuttering across
Table 2 also shows prevalence rates as well as male- the entire life span using randomly selected households
to-female stuttering ratios for all age categories. Preva- that are stratified for region (city, suburban, and rural).
lence was highest in the 2–5 (1.4%) and 6–10 (1.44%)
age groups and lowest in the 11–20 (0.53%) and 51 and The prevalence of stuttering over the entire life span
older (0.37%) age groups. The overall prevalence rate (from age 2 on) was found to be 0.72% with at least a
for the entire sample was 0.72% (95% confidence inter- 50% higher prevalence rate of stuttering in males (2.3:1
vals (CIs) of 0.57 to 0.87). Bloodstein (1995) presented male-to-female ratio). A 0.72% prevalence rate is rea-
data on the prevalence in school-age children (approxi- sonable given that it is accepted that many children
mately 5–18 years) of around 1%. In this study, the naturally recover from stuttering (Bloodstein, 1995). A
prevalence of stuttering in this same age group (5–18 higher prevalence rate of around 1.4% was found in chil-
years inclusive; n = 2553 with 23 persons who stuttered) dren (2 to 10 years), with male children again having a
was found to be 0.9% (95% CI of 0.54 to 1.26%). Table 2 higher prevalence of stuttering (2.3 to 3.3:1). In adoles-
also shows sex ratios by age group. Over the total sample, cence (11 to 20 years), the prevalence rate fell substan-
the male-to-female sex ratio was 2.3:1. It was highest in tially to 0.53%, with boys much more likely to stutter
the 11–20 age group (4:1 male:female) and lowest in the (4:1 ratio). However, prevalence increased in adulthood
51 and older age group (1.4:1). (21–50 years) to 0.78% (2.2:1 ratio), falling once again

Table 2. Breakdown of age by stuttering cases (SC), prior stuttering cases (PSC), prevalence, male-to-female stuttering ratios, and an estimate
of the incidence or risk of stuttering.

Age (yrs) SC PSC Total N # males Prevalence (95% CI) M:F ratios Risk (95% CI)

2–5 10 10 720 389 1.4% (0.54–2.26) 2.3:1 2.8% (1.6–4.0)


6–10 13 18 902 465 1.44% (0.66–2.22) 3.3:1 3.4% (2.2–4.6)
11–20 10 32 1,881 1006 0.53% (0.20–9.86) 4:1 2.2% (1.5–2.9)
21–50 42 71 5,405 2607 0.78% (0.55–1.01) 2.2:1 2.1% (1.7–2.5)
51+ 12 45 3,207 1556 0.37% (0.16–0.58) 1.4:1 1.8% (1.3–2.3)
All ages 87 176 12,131 6023 0.72% (0.57–0.87) 2.3:1 2.2% (1.3–2.3)

Note. There are 16 missing cases in the 2–5 age breakdown as children less than 2 years old were not included in the analysis.

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in late middle to older age (0.37% for 51+ years), with also be due to relapse (Craig et al., 1996), or to other
males again stuttering more frequently than females factors not yet understood. Perhaps it also reflects a
(1.4:1 ratio). It is interesting to note the 95% confidence greater willingness to discuss ones’ stuttering. The de-
intervals for all the prevalence data in Table 2. The crease in prevalence in older ages cannot yet be ex-
prevalence rates found by the majority of studies re- plained. As found elsewhere, more males than females
viewed in this paper fall within these 95% confidence stuttered, but the sex ratio also changed over the life
intervals. It is also interesting to note that the 95% con- span (a 3 or 4 to 1 male-to-female ratio in older chil-
fidence intervals for the prevalence of school-age chil- dren, with an expected smaller ratio in younger chil-
dren found in this study (5–18 years; 0.9% prevalence, dren and perhaps unexpectedly, down to 2.2:1 in early
CI range from 0.54% to 1.26%) covers the estimates to middle adulthood, and further down to 1.4:1 in older
quoted by Bloodstein (1995), who suggested that the adults).
prevalence in the United States was 0.97% and outside Risk of stuttering is best generated from prospec-
of the United States was 1.28%. tive designs in which new cases of stuttering are deter-
The results of this study highlight two possibilities, mined over a designated period of time in a population
that (a) the accepted prevalence of around 1% in younger that is initially nonstuttering. This study provides only
children is too low and (b) the accepted prevalence of an estimated risk of stuttering, as risk was partly based
around 1% in adolescence is too high. The prevalence on the memories of those interviewed (those who were
rates in Table 2 suggest that more realistic figures may believed to stutter in the past). As a result, this study
be 1.4% in younger children and only 0.5% in adoles- could only provide a lower bound on the risk of stutter-
cents. The 1.4% prevalence rate in children found in the ing due to the above design limitations and the assump-
current study is substantially higher than Bloodstein’s tion that people are unlikely to stutter after adolescence.
(1995) estimate and is closer to the mean prevalence The estimated risk in children ranged from 2.8% to 3.4%
rate of 1.3% from the seven studies critiqued in the first in the present study. This is lower than expected if one
section of this paper. The results of the current study accepts the findings of Andrews and Harris (1964) or
strongly suggest a higher prevalence rate in younger Mansson (2000) that the risk is closer to 5% in children.
children be accepted as the status quo. Significant clini- Inspection of Table 2 shows that the 95% confidence in-
cal issues arise that will affect the management of stut- tervals came close (upper limit of 4%) to the figures found
tering in younger children if the prevalence of the disor- by the above authors. However, it may be, as argued
der is underestimated. For example, assuming that 14% above, that risk in the Andrews and Harris study is closer
of a population of 200 million people falls in the age range to 3%, a figure that concurs with the findings in the
of 2 to 10 years, a 1% prevalence rate of the number of present study. The Mansson finding of 5% may be over-
children who stutter at any one time is 300,000. How- estimated due to design limitations or may truly reflect
ever, if the prevalence is at least 1.4%, then the number a high risk in an isolated population. Further research
of children stuttering is underestimated by 120,000 is needed to determine the risk of stuttering, using a
(420,000 rather than 300,000 children may be expected prospective study conducted on a randomly selected
to stutter). If this underestimation of prevalence were heterogeneous population in which stuttering is care-
the case, then the clinical load for clinicians treating fully diagnosed and assessed. There are no existing data
children would be high, with long wait times. Services on risk throughout the life span, so the findings of the
for adults who stutter would be severely reduced as a current study await replication. However, as expected,
result. This clinical scenario is a reality in Australia. the adult risk of stuttering was around 2%, almost 1%
The lower prevalence rate in adolescents should not less than the childhood risk. The risk was higher in older
be unexpected given that the trend in the treatment of children (3.4%) than in young children (2.8%), perhaps
children who stutter is to treat the stutter early rather due to new cases emerging in this older age group. Natu-
than wait hoping that spontaneous remission will occur ral recovery seems to have influenced risk in adolescents
(Lincoln & Onslow, 1997). In addition, a significant pro- (2.2%). In a population of 140 million adults (if adults
portion of children’s stuttering is thought to spontane- represent 70% of 200 million people), a potential 2.8
ously remit before adolescence (Yairi & Ambrose, 1992). million people have a risk of stuttering. Furthermore,
Recovery from stuttering has been estimated to be at there may be from 600,000 up to 1 million people stut-
least 70% from longitudinal research (Yairi & Ambrose, tering at any one time in such a population. Given the
1992) and is now believed to be both more frequent in potential for stuttering to hinder the psychosocial and
females and genetically transmitted (Ambrose, Cox, & employment prospects for persons who stutter through
Yairi, 1997). This study shows an estimated recovery their lifetime (Craig, 1990; Craig & Calver, 1991), these
rate over the entire life span of almost 70%. The increase figures argue for the allocation of substantial resources
in prevalence through early to middle adulthood may to treatment facilities for adults who stutter.

Craig et al.: Epidemiology of Stuttering 1103

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Acknowledgments Craig, A., Hancock, K., Chang, E., McCready, C.,
Shepley, A., McCaul, A., et al. (1996). A controlled trial
This research was funded by The University of Technol- for stuttering in persons aged 9 to 14 years. Journal of
ogy, Sydney, and a Commonwealth Department of Health Speech and Hearing Research, 39, 808–826.
Grant (NHMRC). Thanks also to the following funding Dillman, D. A. (1978). Mail and telephone surveys. New
bodies who contributed financially to the research: the Big York: John Wiley.
Brother Movement, the Australian Rotary Health Research
Gillespie, S. K., & Cooper, E. B. (1973). Prevalence of
Fund, the Sunshine Foundation, and the Inger Rice Founda- speech problems in junior and senior high schools. Journal
tion. of Speech and Hearing Research, 16, 739–743.
Ingham, R. J. (1976). “Onset, prevalence, and recovery from
References stuttering”: A reassessment for findings from the Andrews
and Harris study. Journal of Speech and Hearing Disor-
Ambrose, N. G., Cox, N. J., & Yairi, E. (1997). The genetic ders, 41, 280–281.
basis of persistence and recovery in stuttering. Journal of Leavitt, R. R. (1974). The Puerto Ricans: Culture change
Speech, Language, and Hearing Research, 40, 567–580. and language deviance. Tucson: University of Arizona
Andrews, G., Craig, A. R., Feyer, A. M., Hoddinott, S., Press.
Howie, P., & Neilson, M. (1983). Stuttering: A review of Lincoln, M., & Onslow, M. (1997). Long-term outcome of
research findings and theories circa 1982. Journal of early intervention for stuttering. American Journal of
Speech and Hearing Disorders, 48, 226–246. Speech-Language Pathology, 6(1), 51–58.
Andrews, G., & Harris, M. (1964). The syndrome of Mansson, H. (2000). Childhood stuttering: Incidence and
stuttering. London: Heinemann Medical Books. development. Journal of Fluency Disorders, 25, 47–57.
Aneshensel, C. S., Frerichs, R. R., Clark, V., & Montgomery, B. M., & Fitch, J. L. (1988). The prevalence of
Yokopenic, P. A. (1982). Telephone versus in-person stuttering in the hearing-impaired school age population.
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Public Health, 72, 1017–1021.
Moscicki, E. K. (1984, August). The prevalence of ‘inci-
Ardila, A., Bateman, J. R., Nino, C. R., Pulido, E., dence’ is too high. Proceedings of the American Speech and
Rivera, D. B., & Vanegas, C. J. (1994). An epidemiologic Hearing Association, 39–40.
study of stuttering. Journal of Communication Disorders,
27, 37–48. Paulsen, A. S., Crowe, R. R., Noyes, R., & Pfohl, B. (1988).
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Aron, M. L. (1962). The nature and incidence of stuttering disorders. Archives of General Psychiatry, 45, 62–63.
among a Bantu group of school-going children. Journal of
Speech and Hearing Disorders, 27, 116–128. Porfert, A. R., & Rosenfield, D. B. (1978). Prevalence of
stuttering. Journal of Neurology, Neurosurgery, and
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resident population by age and sex in statistical local areas
in NSW 30 June 1991. Sydney, Australia: Author. Quine, S. (1985). Does the mode matter? A comparison of
three modes of questionnaires completion. Community
Bloodstein, O. (1995). A handbook on stuttering (5th ed.). Health Studies, 9, 151–156.
San Diego, CA: Singular.
Slome, C., Brogan, D., Eyres, S., & Lednar, W. (1986).
Boyle, C. A., Decoufle, P., & Yeargin-Allsopp, M. (1994). Basic epidemiological methods and biostatistics. Boston:
Prevalence and health impact of developmental disabili- Jones and Bartlett.
ties in US children. Pediatrics, 93, 399–403.
Stansfield, J. (1990). Prevalence of stuttering and clutter-
Brady, W. A., & Hall, D. E. (1976). The prevalence of ing in adults with mental handicaps. Journal of Mental
stuttering among school-age children. Language, Speech, Deficiency Research, 34, 287–307.
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Yairi, E., & Ambrose, N. G. (1992). A longitudinal study of
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niques. New York: George, Allen and Unwin.
Craig, A. (1990). An investigation into the relationship Received May 15, 2002
between anxiety and stuttering. Journal of Speech and Accepted July 15, 2002
Hearing Disorders, 55, 290–294.
DOI: 10.1044/1092-4388(2002/088)
Craig, A., & Calver, P. (1991). Following up on treated
Contact author: Ashley Craig, PhD, Department of Health
stutterers: Studies of perceptions of fluency and job status.
Sciences, University of Technology, Sydney, Broadway,
Journal of Speech and Hearing Research, 34, 279–284.
New South Wales, Australia 2007.
E-mail: a.craig@uts.edu.au

1104 Journal of Speech, Language, and Hearing Research • Vol. 45 • 1097–1105 • December 2002

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Appendix

Prevalence Questionnaire
Date ___ / ___ / ___

Stuttering Telephone Survey

Demographics (d) Did you have therapy for stuttering?


Do you know what types of therapy? ____________
(a) May I ask how many females live in your household at
present? (e) Do you know whether anyone in your close family
ever stuttered? (as above)
(b) What are their ages?
Who are they? _____________
(c) How many males live in your household at present?
(d) What are their ages?
3. Does anyone in your household stutter?
If answer to Q3 is no, go to Q4.
The lettered questions could be answered Yes, No, or Not sure.
(a) Who are the persons in your household who stutter?
(label F1, M2, etc.)
1. Do you stutter?
(b) Has the stuttering persisted for the past 3 months?
If answer to Q1 is no, go to Q2.
(c) Has the stuttering caused fear and avoidance of
(a) Has the stuttering persisted for the past 3 months?
situations?
(b) Has the stuttering caused fear and avoidance of
(d) Has the person(s) consulted a speech professional?
situations?
(e) Has the person(s) had therapy for stuttering?
(c) Have you consulted a health professional? (speech
therapist, psychologist, psychiatrist, GP, etc.) (f) Does the person(s) have close family who have ever
stuttered?
(d) Have you had therapy for stuttering?
If a person in the household stutters, ask permission to
Do you know what types of therapy?____________.
tape them for up to 5 min.
(e) Do you know whether anyone in your close family
ever stuttered? (parents, grandparents, sisters,
brothers, children) 4. Has anyone in your present household ever stuttered?
Who are they? ____________________ (a) Who are the persons in your household who have
stuttered in the past? (label F1, M2, etc.)
If the person stutters, ask permission to tape for up to 5 (b) Did the stuttering persist for at least 3 months?
min.
(c) Did the stuttering cause fear and avoidance of
situations?
2. Have you ever stuttered?
(d) Did the person(s) consult a health professional?
If answer to Q2 is no, go to Q3. (speech therapist, psychologist, GP, psychiatrist,
(a) Did the stuttering persist for at least 3 months? etc.)
(b) Did the stuttering cause fear and avoidance of (e) Did the person(s) have therapy for stuttering?
situations? (f) Do you know whether anyone in your close family
(c) Did you consult a health professional? (speech ever stuttered?
therapist, psychologist, GP, psychiatrist, etc.) Who are they?

Craig et al.: Epidemiology of Stuttering 1105

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