Christ the King College

9014 Gingoog City

ACUTE GLOMERULONEPHRITIS
NCM 103

Submitted to:

Mrs. Vilma S. Ramoso

Submitted by:

Stephanie A. Reyes
 Acknowledgement
This dissertation would not have been possible without the guidance and the help
of the people who contributed and extended their valuable assistance in the
preparation and completion of this study. Many helped shaped and completed
this work and I wish to acknowledge them whose help is instrumental in the
realization of this endeavor:

To our school, Christ the King College, the source of our understanding and the
residence of our learning.

To our acting dean of the Nursing Program, Mr. Tristan Jay I. Galarrita, RN, for his
resilient leadership and organization for the likelihood of our hospital exposure.

To my dear beloved Clinical Instructor Mrs. Vilma S. Ramoso for extending and
sharing her knowledge about nursing care and also for abiding with me during our
area work; and for your constructive insights and motivation.

To the staff of the hospital, for leading me with my performance, And to the client
for allowing me to do my assessment during her Hospitalization.

To my dear mother, brother, sister, classmates, and friends, for their spirit-
warming inspiration and unceasing prayers.

And above all, to our Almighty God, our creator, who gives me abundant wisdom
and countless love for the strength and guidance in the preparation and
completion of this work.
Table of Contents:

I. Introduction

II. Objectives

III. Developmental Theories

IV. Client’s Profile

V. Nursing History of Illness

VI. Anatomy and Physiology

VII. Pathophysiology

VIII. Laboratory Examinations

IX. Nursing Management

X. Discharge Planning

XI. Health Teaching

XII. Nursing Care Plan

XIII. Drug Study

XIX. Bibliography
I. Introduction

Acute glomerulonephritis refers to a specific set of renal diseases in which an immunologic

mechanism triggers inflammation and proliferation of glomerular tissue that can result in
damage to the basement membrane, capillary endothelium. Acute nephrotic syndrome is a
group of disorders that cause inflammation of the internal kidney structures (specifically, the
glomeruli). In acute glomerulonephritis, the kidneys are normal in size or enlarged and
edematous, and the surface of the kidney may show punctuate hemorrhages. With the
development of the microscope, Langhans was later able to describe these pathophysiologic
glomerular changes. Acute glomerulonephritis is defined as the sudden onset of hematuria,
proteinuria, and red blood cell casts. This clinical picture is often accompanied by hypertension,
edema, and impaired renal function. As will be discussed, acute glomerulonephritis can be due
to a primary renal or systemic disease. Symptoms of acute glomerulonephritis include the
following: Hematuria is a universal finding, even if it is microscopic. Gross hematuria is reported
in 30% of pediatric patients. Edema (peripheral or periorbital) is reported in approximately 85%
of pediatric patients; edema may be mild (involving only the face) to severe, bordering on a
nephrotic appearance .Headache may occur secondary to hypertension; confusion secondary to
malignant hypertension may be seen in as many as 5% of patients. Shortness of breath or
dyspnea on exertion secondary to heart failure or pulmonary edema; usually uncommon,
particularly in children. Possible flank pain secondary to stretching of the renal capsule.
Hypertension is seen in as many as 80%of affected patients. Hematuria, either macroscopic
(gross) or microscopic, may be noted.

Skin rashes may be observed. Abnormal neurologic examination or altered level of

consciousness occurring, because of malignant hypertension or hypertensive encephalopathy.
Arthritis may be noted. The most common cause is post infectious. Cases of acute nephritis
often progress to a chronic form. This progression occurs in as many as 30% of adult patients
and 10% of pediatric patients. Glomerulonephritis is the most common cause of chronic renal
failure (25%).The mortality rate of acute glomerulonephritis in the most commonly affected age
group, pediatric patients, has been

Reported at 0-7%. A male-to-female ratio of 2:1 has been reported. Most cases occur in
patients aged 5-15 years. Only 10% occur inpatients older than 40 years. Acute nephritis may
occur at any age, including infancy.

On the other hand, a urinary tract infection (UTI) is a bacterial infection that affects any part of
the urinary tract. The main etiologic agent is Escherichia coli. Although urine contains a variety
of fluids, salts, and waste products, it does not usually have bacteria in it. When bacteria gets
into the bladder or kidney and multiply in the urine, they may cause a UTI. Infections of the
urinary tract are the second most common type of infection in the body. Urinary tract
infections (UTIs) account for about 8.3 million doctor visits each year. Women are especially
prone to UTIs for reasons that are not yet well understood. One woman in five develops a UTI
during her lifetime. UTIs in men are not as common as in women but can be very serious when
they do occur. The most common type of UTI is acute cystitis often referred to as a bladder
infection. An infection of the upper urinary tract or kidney is known as pyelonephritis, and is
potentially more serious. Although they cause discomfort, urinary tract infections can usually
be easily treated with a short course of antibiotics.

Symptoms include frequent feeling and/or need to urinate, pain during urination, and cloudy
urine. The first step is to identify the infecting organism and the drugs to which it is sensitive.
Usually, doctors recommend lengthier therapy in men than in women, in part to prevent
infections of the prostate gland. As a group, we decided to study this kind of disease for us to
know more about the complications. As a nursing students, we must not only focus to one
corner or merely by just taking care of our patients but to know their underlying condition as
well for the better and good nursing intervention done to promote maximum living ability.
Furthermore, we have chosen this case study in order to identify and determine the general
health problems and needs of the patient with an admitting diagnosis of acute
glomerulonephritis. This study also intends to help patient as well as its significant others to
promote health and medical understanding of such condition through the application of the
nursing theories and nursing skills.
II. Objectives

General Objectives:
At the end of this case study, I will be able to gain knowledge and ideas, practice my skills and
deal appropriate attitudes towards client and everyone.

Specific Objectives:
Student Centered Objectives:
At the end of the case study, I will be able to:

Affective

1. Appreciate the importance of health.

2. Appreciate the importance of significant other’s support in all aspects.

3. Give value to one’s life.

Psychomotor

1. Appreciate the importance of providing extensive care to clients with kidney disease.

2. Perform effectively the nursing procedures to clients with kidney disease.

3. Promote health teachings for betterment of the health of the client.

Cognitive

1. Identify and determine general health problems of the client.

2. Formulate care plans related to psychological and physiological aspects of the disease.

3. Analyze the effectiveness in meeting the needs of the client.

Client Centered Objectives:

At the end of the case study, the client will able to:

Affective:

1. Appreciate essence of health.

2. Appreciate the support of significant others.

3. Give value and importance to one’s health.

Psychomotor

1. Avoid things that will complicate the disease.

2. Participates with the appropriate nursing interventions.

3. Demonstrate and apply health practices concerning the disease.

Cognitive

1. Identify and determine general health problems.

2. Analyze the effectiveness in meeting the needs.

3. Verbalize understanding regarding different complications of the disease.

III. DEVELOPMENTAL THEORIES

Erik Erikson’s Psychosocial Theory
According to Erikson every stage must be complete and must be obtain by a person. Ms. X is 14
years old so she is in the stage of Adolescence, which is the identity vs. role confusion phase.
Ms. X shows feelings of confusion, indecisiveness which indicates negative resolution, she is still
in the role confusion phase, maybe it’s because she is still very young to make decisions.

Robert Havighurst Developmental Task Theory

Ms. X belongs to the adolescence stage. She is achieving feminine social role, accepting one’s
physique and using the body effectively and achieving new and more mature realtions with
age-mates of both sexes.

Sigmund Freud’s psychosexual Theory

Ms. X is in her Genital stage which means she is developing to her full sexual maturity. Her
energy is directed toward full maturity and function and development of skills needed to cope
with the environment. She is highly sociable and friendly because many of her friends and
classmates visited her during her hospitalization.
IV. Clients Profile
A. Socio-demographic data
Patient X is a 14 year old female, Roman Catholic; a resident of Brgy. Nawasa Gingoog City.
Patient X was admitted for the first time at Misamis Oriental Provincial Hospital at 5:30 pm. due
to difficulty of breathing, facial and pedal edema. She arrived at the hospital awake, conscious
and coherent with a chief complaint of difficulty of breathing, facial and pedal edema.

B. Vital Signs
The patient’s vital signs are essential because it provides a baseline data in determining
alteration in the patient’s body that may suggest underlying disease. Any changes from the
normal are considered to be an indication of the person’s state of health and provide cues to
the physiological functioning of the client. The patient had the following vital signs: blood
pressure: 100/70 mmHg, pulse rate: 104 bpm, respiratory rate: 32 cpm, temperature: 36.9ºC.
She currently weighs 42kilograms from the previous weight of 45 kilograms and he is 5’1 tall.

C. Health Patterns Assessment

V. Nursing History of Illness

1. History of Present illness
The client was brought to the hospital due to difficulty of breathing, facial and pedal edema.
Three weeks prior to admission, onset of edema with no other associated symptoms noted.
One week prior to admission, persistence of facial edema associated with pallor. 3 days prior to
admission, difficulty of breathing, pallor and decreased urine output was noted which
prompted patient to seek medical consultation. Patient has no previous hospitalization and
surgeries. Client has no family history of kidney-related diseases. Patient X was not taking any
medication. She’s a non tobacco user and a non alcoholic drinker. She has no drug allergies but
has a food allergy specifically “uwang”.

2. Nutrition

During pre-hospitalization, the client used to eat junk foods which are high in sodium and likes
to drink soft drinks most of the time and seldom drinks water. During hospitalization, Patient X
was on a low salt, low fat diet. She has a poor appetite. She was not taking in any vitamins. The
client is hooked with an intravenous fluid of D5 .3% solution running at 10 gtts/min.

3. Elimination Pattern

Pre-hospitalization, Patient X defecates once to twice daily with formed, brownish stool and
soft in consistency. No discomfort felt during defecation but during hospitalization, the client
has difficulty in defecating, thus, making her at risk to have constipation. While confined, the
client defecates after 2-3 days. Pre-hospitalization, a month before the admission to the
hospital, client has difficulty urinating thus, decreasing the urinary frequency from 6-8 times to
1 time per day. It’s yellowish in color and clear.

4. Activity -Exercise Pattern (pre – hospitalization)

Patient X is incorporating her exercise when walking from her house to the highway every
morning and she’s going home from the school. Playing is her leisure activity together with few
of her friends but mostly, she loves to play with her other siblings.

A. Activity-Exercise Pattern (while confined)

Describe the patient’s functional abilities

a. Feeding: independent

b. Bathing: independent

c. Toileting: independent

d. Bed mobility: independent

e. Dressing: independent

f. Grooming: dependent

g. General mobility: independent

h. ROM: independent

i. Ambulation: independent

The patient can do independently all activity- exercise but then, it is limited and controlled due
to disease condition and client prefer to stay in the bed than ambulating. Patient does not like
to take a bath during hospitalization, her mother cleans wipe out dirt in the body which made
her dependent in Grooming.

5. Cognitive-Perceptual Pattern

Patient X understands and speaks Visayan language and she didn’t have any speech deficit.
Patient is a 4th year High School Student without any learning difficulties. There is pain felt in
the abdominal area and patient usually guards the location of pain.

6. Sleep -Rest Pattern

Pre-hospitalization, Patient X usually sleeps for 8 hours. She doesn’t have any sleep
disturbances but while confine, she verbalized problem of sleeping disturbances at night.
Imagery is one of the effective tools for her in order to sleep at night.

7. Self-perception and Self-concept Pattern

Patient X says that she is good as well a responsible student because she usually gets an award
after each school year. She feels that she was weak and fear to be behind in their classroom
lessons and scared not to get an award this school year due to the series of absences because
of her hospitalization.

8. Role-Relationship Pattern

Her family specifically her mother and grandfather who helped her during hospitalization. Her
father and other members in the family also visit her in the hospital and also her classmates
and friends.

9. Coping -stress Tolerance Pattern

Patient X seldom experiences any stress during hospitalization due to difficulty of breathing,
she just subject herself to sleep.

10. Value -Belief Pattern

Patient X is a Roman Catholic. To her it is important as it had helped her when she has a
problem. She goes to church every Sunday together with her family. The client also prays
frequently as part of her religious practices.

D. Physical Assessment
1. Neurologic Assessment

Level of consciousness Conscious

Orientation Oriented
Emotional state Worried/anxious (sometimes); restless

2. Head

Head Normocephalic
Facial movement Symmetrical
Fontanels Closed
Hair Fine
Scalp Clean

3. Eyes

Lids Symmetrical
Periorbital region Edema
Conjunctiva Pale
Cornea & Lens Opacity R/L
Sclera Anicteric
Pupils Equal in size
Reaction to light Brisk R/L
Reaction to accommodation Uniform to constriction
Visual acuity Grossly normal
 Peripheral vision Intact/full

4. Ears

External pinnae Normoset

External canal No discharge
Tympanic membrane Intact
Gross hearing Normal

5. Nose

Mucosa Pinkish
Patency Both patent
Gross smell Normal/symmetrical
Sinuses No tenderness presence

6. Mouth

Lips Pallor
Mucosa Pinkish
Tongue Midline
Teeth Complete
Gums Pinkish

7. Pharynx

Uvula Midline
Tonsils Not inflamed
Posterior pharynx No inflammation

8. Neck

Trachea Midline
Thyroids Non-palpable

9. Skin

General Color Pallor

Texture Rough
Turgor Firm
Temperature Warm

10. Abdomen

Configuration Globular
Bowel sound Hypoactive (3 counts)
Percussion Tympanic

11. Cardiovascular Status

Pericardial area Flat

Point of maximal impulse (PMI) 5th intercostals
Apical & rhythm Normal (100bpm)
Heart sound Regular
Peripheral pulse Symmetrical & regular but weak
Capillary refill 2 seconds

12. Respiratory Status

Breathing pattern Irregular (Tachypneic)

Lung expansion Symmetrical
Percussion Resonant
Breath sound Vesicular
Cough Non-productive

HOME VISIT
Post hospitalization ( August 13,2012)
VI. ANATOMY AND PHYSIOLOGY
The Kidney

The main functional unit of the kidney is thenephron. There are approximately onemillion
nephrons per kidney. The role of nephrons is to make urine by:

 Filtering blood of small molecules and ions such as water, salt, glucose and other solutes
including urea. Large “macromolecules” like proteins are untouched.
 Recycling the required quantities of useful solutes which then re-
e n t e r t h e bloodstream. (A process called reabsorption)
 Allowing surplus or waste molecules/ions to flow from the tubules/ureter as urine.
Nephrons are the basic structural and functional units of the kidney. They consist of a network
of tubules and canals specialized in filtration.

The kidney is responsible for maintaining fluid balance within the body. The basic structural and
functional units of the kidneys are the nephrons. Each nephron is made of intricately
interwoven capillaries and drainage canals to filter wastes, macromolecules, and ions from the
blood to urine. The approximately 1 million nephrons in each human kidney form 10-20 cone-
shaped tissue units called renal pyramids that span both the inner and outer portions of the
kidney, the renal medulla and renal cortex.

A. Renal Vein
 This has a large diameter and a thin wall. It carries blood away from the kidney and back to the
right hand side of the heart. Blood in the kidney has had all its urea removed. Urea is produced
by your liver to get rid of excess amino-acids. Blood in the

renal vein also has exactly the right amount of water and salts. This is because the kidney gets
rid of excess water and salts. The kidney is controlled by the brain. A hormone in our blood
called Anti-Diuretic Hormone (ADH for short) is used to control exactly how much water is
excreted. This blood vessel supplies blood to the kidney from the left hand side of the heart.
This blood must contain glucose and oxygen because the kidney has to work hard producing
urine. Blood in the renal artery must have sufficient pressure or the kidney will not be able to
filter the blood. Blood supplied to the kidney contains a toxic product called urea which must
be removed from the blood. It may have too much salt and too much water. The kidney
removes this excess material that is its function.

B. Renal Artery

This blood vessel supplies blood to the kidney from the left hand side of the heart. This blood
must contain glucose and oxygen because the kidney has to work hard producing urine. Blood
in the renal artery must have sufficient pressure or the kidney will not be able to filter the
blood. Blood supplied to the kidney contains a toxic product called urea which must be
removed from the blood. It may have too much salt and too much water. The kidney removes
these excess materials; that are its function.

C. Pelvis

This is the region of the kidney where urine collects. If you are very unlucky, you may develop
kidney stones. Sometimes the salts in the urine crystallize in the pelvis and form a solid mass
which prevents urine from draining out of the medulla of the kidney. You will need treatment:
see your doctor.

D. Ureter

This one is easy peasy: the ureter carries the urine down to the bladder. It does this 24 hours
per day, but fortunately the urine can be stored in a bladder so that it is not necessary to wear
a nappy!

E. Medulla

The medulla is the inside part of the kidney. It is shown in green in the diagram, but in real life it
is a very dark red color. This is where the amount of salt and water in your urine is controlled. It
consists of billions of loops of Henlé. These work very hard pumping sodium ions. ADH makes
the loops work harder to pump more sodium ions. The result of this is that very concentrated
urine is produced. The opposite of an anti-diuretic is a "diuretic". Alcohol and tea are diuretics

F. Cortex

The cortex is the outer part of the kidney. This is where blood is filtered. We call this process
"ultra-filtration" or "high pressure filtration" because it only works if the blood entering the
kidney in the renal artery is at high pressure. Billions of glomeruli are found in the cortex. A
glomerulus is a tiny ball of capillaries. Each glomerulus is surrounded by a "Bowman's Capsule".
Glomeruli leak. Things like red blood cells, white blood cells, platelets and fibrinogen stay in the
blood vessels. Most of the plasma leaks out into the Bowman's capsules. This is about 160 liters
of liquid every 24 hours. Most of this liquid, which we call "ultra-filtrate" is re-absorbed in the
medulla and put back into the blood.

G. Glomerulus and Bowman's Capsule

This is where ultra-filtration takes place. Blood from the renal artery is forced into the
glomerulus under high pressure. Most of the liquid is forced out of the glomerulus into the
Bowman's capsule which surrounds it. This does not work properly in people who have very low
blood pressure. Proximal Convoluted Tubules Proximal means "near to" and convoluted means
"coiled up" so this is the coiled up tube near to the Bowman's capsule. This is the place where
all that useful glucose is re-absorbed from the ultra-filtrate and put back into the blood. If the
glucose was not absorbed it would end up in your urine. This happens in people who are
suffering from diabetes.

H. Loop of Henlé

This part of the nephron is where water is reabsorbed. Kidney cells in this region spend all their
time pumping sodium ions. This makes the medulla very salty; you could say that this is a region
of very low water concentration. If you remember the definition of osmosis, you will realize
that water will pass from a region of high water concentration (the ultra-filtrate and urine) into
a region of low water concentration (the medulla) through cell membranes which are semi-
permeable.

I. Distal Convoluted Tubules

Distal means "distant" so it is at the other end of the nephron from the Bowman's capsule. This
is where most of the salts in the ultra-filtrate are re-absorbed.

J. Collecting Duct
Collecting ducts run through the medulla and are surrounded by loops of Henlé. The liquid in
the collecting ducts (ultra-filtrate) is turned into urine as water and salts are removed from it.
Although our kidneys make about 160 liters of urine every 24 hours, we only produce about ½
liter of urine. It is called a collecting duct because it collects the liquid produced by lots of
nephrons.

Nephron Function

The blood is filtered and urine formed by the actions of the nephrons. In each nephron, high
pressure in the glomerulus pushes water and small dissolved materials into the extra vascular
space of the Bowman’s capsule and into the tubule. The proximal tubule reabsorbs water, salts,
glucose, and amino acids to maintain electrolyte levels in the body. The interstitial of, that is
the tissue space surrounding, the loop of Henle concentrates salts that will be excreted in the
urine, creating a concentration gradient in the medulla. The limbs of Henle’s loop are
permeable to particular ions (descending, water and some urea; thin ascending, general ions;
medullary thick ascending –sodium, potassium, chloride), with the cortical thick ascending limb
draining into the distal convoluted tubule. The distal tubule contains cells specialized in active
transport and maintains urine and blood pH levels, particularly through the regulation of
sodium and potassium. Fluid then passes from the distal tubule to the collecting ducts, a tubule
system that can become permeable or impermeable to water depending on the body’s needs.
Ultra filtration also occurs in the cortex in the cortical collecting ducts, which is regarded by
some anatomy references as not being a portion of the nephron, and by others as being the
final portion of the nephron. The urine then passes from the collecting ducts through the
drainage system of the kidney to the ureters and bladder for urination.

Tubular Secretion in the Kidneys

Another, less familiar, mechanism for urine production in the kidneys is tubular secretion.
Specialized cells move solutes directly from the blood into the tubular fluid. For example,
hydrogen and potassium ions are secreted directly into the tubular fluid. This process is
“coupled” or balanced by the re-uptake of sodium ions back into the blood. Tubular secretion
of hydrogen ions, augmented by control of bicarbonate levels is important in maintaining
correct blood pH. When the blood is too acidic (acidosis) more hydrogen ions are secreted. If
the blood becomes too alkaline (alkalosis), hydrogen secretion is reduced. In maintaining blood
pH within normal limits (about7.35–7.45) the kidney can produce urine with pH as low as that
of acid rain or as alkaline as baking soda!

The Kidney as an Endocrine Gland

In addition to its excretory and homeostatic roles, the kidneys also release two important
hormones into the blood. These are:

• Erythropoietin which acts on bone marrow to increase the production of red blood cells

• Calciterol which promotes the absorption of calcium from food in the intestine and acts
directly on bones to shift calcium into the blood stream. Finally the kidney produces the
enzyme renin, an important regulator of blood pressure.

THE RENIN ±ANGIOTENSIN MECHANISM

 Decreased blood pressure stimulates the kidney to secrete renin.

 Renin splits the plasma protein angiotensinogen (synthesized by the liver) to
angiotensin I
 Angiotensin I is converted to angiotensin II by an enzyme (called converting enzyme)
 Secreted by the lung tissue and vascular endothelium.

Angiotensin II:

-causes vasoconstriction- stimulates the adrenal cortex to secrete aldosterone which maintains
normal blood levels of sodium and potassium and contributes to the maintenance of normal
blood pH, blood volume, and blood pressure.
VIII. Laboratory Examinations
Complete Blood Count
PARAMETERS NORMAL VALUES ACTUAL VALUES ANALYSIS
WBC 4.5-11.0 x 10mm 11.1 Increase indicates
infection,
leukemia, tissue
necrosis.
Hemoglobin Female: 11.7 – 10.9 Inadequate
14.5 g/dl production of
RBCs, which may
result to
anemia,fluid
overload, recent
hemorrhage.
Hematocrit Female: 34.1 – 33.0 Decrease may
44.3 vol% result
hemodilation,
anemia, acute
massive blood
loss.
Platelet 175,000 – 256,000 NORMAL
350,000/mm

PARAMETERS NORMAL VALUES ACTUAL VALUES ANALYSIS

Segmenters 45-70% 67 Increased;
Indicates high
glucose level in the
blood.
Lymphocytes 18-45% 24 Increased;
Indicates infection
Monocytes 4-8% 7 NORMAL
Eosinophils 2-3% 2 NORMAL
 Blood Chemistry Report
PARAMETER NORMAL VALUES ACTUAL VALUES ANALYSIS
Increased BUN
levels suggest
impaired kidney
BUN/ Urea 1.7-8.3 Mmol/L 10.10 function. This may
be due to acute or
chronic kidney
disease, damage,
or failure.

Urinalysis Report
PARAMETER NORMAL VALUES ACTUAL VALUES ANALYSIS
Color Yellow/Amber Light yellow Normal
Transparency Clear to slightly cloudy
turbid
Reaction 4.5-8 6.0 Normal
Specific Gravity 1.005-1.030 1.025 Normal
Sugar Negative Trace Indicative of
glucosuria
Protein Negative Positive (+1) Indicative of
proteinuria
Squamous Few Few Normal
Epithelial Cells
Red Blood Cells Few 7.50 Hematuria,
indicative of
possible
glomerular
damage
Pus Cells Few 3-5 Indicative of
bacterial infection
Mucous Few Few Normal
Bacteria Few Many Indicative of
presence of
 bacteria.
Amorp. Few Moderate Indicative of
Urates/Phosphates bacterial infection

IX. Nursing Management

According to book
•Review fluid and diet restrictions measure and record intake and output.

•Instruct patient to schedule follow up evaluations of blood pressure, urinalysis for protein and
BUN and creatinine studies to determine if disease has worsened.

•Instruct patient to notify physician if infection or symptoms of renal failure occur: fatigue,
nausea, and vomiting, diminishing urinary output.

•Refer to home care nurse as indicated for assessment and detection of early symptoms and
follow-up evaluations.

According to patient
•Vital sign was monitored timely and was recorded in TPP chart.

•An electrolyte value was monitored.

•Renal function was assessed

•Intake and output chart was strictly maintained.

•Acute renal failure was monitored.

•Oliguria

•Azotaemia (Increased blood urea)

•Acidosis and hyperkalemia (K+increased)

•Health education was provided about the nature of illness, diet, and medicines

IX. Discharge Planning

D– iscipline regarding the diet

I – nstruct to avoid excessive use of salt

S– eek for medical assistance for further complications

C- ontinue to take medication for maintenance

H– ealth teaching regarding the importance of restricting diet

A– ssess for any adverse effect of the medication

R- egular check-up

G – ive the importance in taking the medication

E – ncourage to follow the prescribed medication at home

 Explain to the patient and family members the importance of taking medicines.
 Discuss to the patient and family the dosage, frequency and adverse effects of the
drugs.
 Encourage to follow the dosages and proper timing of her meds. Such as the
Furosemide 40mg once a day x 3 doses, Prednisone 20mg tab 1 tab Twice a day oral,
Ranitidine 50mg x 6 doses. As prescribed by her physician.

Economic status
  Explain to significant others that the rehabilitation may be prolonged to be able for the
family to prepare financial needs
 Have occupational therapist to help re- learn everyday activities or ADL
 Inform the patient to avail to some government programs such as Philhealth.

Treatment
 Tell the patient that she should have self-monitoring by checking his vital signs and
weighing regularly.
 Encourage/instruct to keep the edematous extremities to elevate as often
 Limit of water intake; monitor intake and output
 Provide warm environment
 Provide egg white a day
 Weight the pt. daily, at the same time.
 Avoid eating foods high in sodium and drinking soft drinks.

X. HEALTH TEACHINGS

 Instruct the patient to take medications religiously.

 Improve nutritional status.
 Importance of proper hygiene for comfort.

OUT-PATIENT

 The patient could avail her medication from government hospitals that she could get
some benefits.
 She will also be able to avail the services offered by the Barangay health center and at
the “Botika ng barangay”.
 Instruct patient to seek regular medical check-up

DIET
 Eat five or more servings of vegetables and fruit daily.
 Intake of fluids 8-10 glasses a day to avoid constipation and to maintain skin turgor.
  Instruct patient to eat low fat and low sodium foods that will help and will not
complicate her condition that is ordered by the physician.

XIX. Bibliography

WEB:
http://generalmedicine.suite101.com/article.cfm/the_human_kidney_structure_and_function#
ixzz0wIXUzTtr

http://cellstissuesmembranes.suite101.com/article.cfm/nephron_structure_and_function

http://emedicine.medscape.com/article/777272-overview;http://www.total-health-
care.com/illness/acute-glomerulonephritis.htm)

BOOKS:
Nurse’s Pocket Guide 11th edition (Diagnoses, Prioritized interventions, and Rationales)
By:Marilyn E. DoengesMary Frances Moorhouse Alice C. Murr

Nursing 2003 Drug Handbook 23rd edition By: Springhouse Lippincott Williams and Wilkins

Medical Surgical Nursing 8th Edition, (Clinical Management for Positive Outcomes) by: Joyce M.
Black, Jane Hokanson Hawks
Kozier & Erb’s Fundamentals of Nursing 8th Edition (Concepts, Process and Practice) by:
Berman, Synder, Kozier, Erb