Ataxia

Uncoordinated ,
shaky voluntary movements open Lesions of the cerebellar hemispheres result in problems
anointed with cerebellar dysfunction or
impaired with voluntary unis movements . This is appendicitis an
vestibular 1 propriocaptive all event input to the cerebellum . ataxia .
Finger -
nose testing , and rapid alternating
movements show difficulties with these areas .

It is characterised by a wide , unsteady gait , errors of

extremity trajectory or
placement ,
errors in motor Patients have dysmaria
-

problems judging distances and

sequence or
rhythm , and dysannria .
Tone is unruly
are unable to maintain rhythm or amplitude of movements

dysdiadocnokinesia Speech
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decreased ,
and stretch reflexes may be pendulum . known as . can also be

attend , with variations in

Nystagmus , skin deviation , disoonjugate saccades and intensity and shining at words .

altered ocular
pursuit can be present .

Tnlncal instability and tremor of the head or body The ftoccul modular lobe or vestibule cerebellum cause

may occur ,
especially with midline disorders .
postural instability , and impaired eye movement

control .

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one side Cerebellar ataxia can result from vascular lesions

Of the body inflammation tumors,

limb such as stroke , ,
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infections disorders drugs alcohol

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hereditary , or .

sensory Ataxia

Problems with lesions wheeling this

Vestibular system proprioception .

Disorders cause disequilibrium and unsteadiness .

pathway result in an inability to stand with eyes
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If it is unilateral ,
the patient will have profound closed
Romberg sign) .
They will www. heavily ,

with amounted nausea and

as
they do not know where to place their feet They
Veni
go vomiting
.
.
,

Drugs t alcohol are common causes at this type of

will stumble it walking in the dark .

ataxia . Also consider maniacs dieare .

May be caused by inflammation ,

dymetinah.cn ,
Cerebellum infections ,
hereditary disorders and vitamin deficiencies .

The cerebellum is organised in a somato trophic

distribution with the trunk control centrally , and Treatment
units more laterally .
Requires an individualised approach -

may need a

vemnis combination of 0T I PT1 SALT interventions , as well as

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Lesions whetting the area medications to manage

\ I symptoms .

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drunk movement retraining ,

reducing the degrees of
limbs trunk limbs freedom and optimising the environment .

wide boned
thincat ataxia - A gait characterised by starts
2) Restorative -
aim to improve function by improving
and stops lateral deviations and unequal steps
, .
It is a tmncal the underlying impairment .

instability and often seen when sitting ,

or
walking hen to

toe .
Rehabilitation

Physio avin to improve gait ,

balance and trunk control

for those with ataxia , and can reduce

activity
limitations and support increased participation .

The prevention of falls is important to consider in

those with ataxia

progressive .

Dynamic task practice which challenges stability ,

aims
explores stability limits and to reduce upper limb-

weight bearing seems an

important intervention to

improve gait and balance .

strength training is also

indicated .

Example exercises :

lying bent knee rotations

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kneeling press ups

-

Superman in 4 -

point kneeling

Tandem stand 1
walking
-