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The Spatial Diffusion of Homicide in Mexico City: A Test of Theories in Context
The Spatial Diffusion of Homicide in Mexico City: A Test of Theories in Context
To cite this article: Carlos Vilalta, Pablo Lopez-Ramirez & Gustavo Fondevila (2021): The
spatial diffusion of homicide in Mexico City: a test of theories in context, Global Crime, DOI:
10.1080/17440572.2021.1909480
Article views: 58
ARTICLE
Introduction
In this study we tested three complementary hypotheses with regards to the spatial
diffusion of homicidal violence in Mexico City. First, we tested whether there was
a contagious spread of homicide across neighborhoods. The simplest of the spatial
diffusion hypotheses predicts that fluctuations in homicidal violence in one neighbor
hood will affect homicide fluctuations in adjacent neighborhoods. Second, we tested for
the contagious spread of homicide to neighborhoods more susceptible to drug market
activity between organized crime groups. Although this hypothesis has been partially
confirmed for the case of aggravated assault crimes in the U.S. (Contreras & Hipp, 2020),
this is an ad-hoc hypothesis for the case of Mexico City which is explained in the next
section. Third, we tested whether homicide spread to more structurally criminogenic
neighborhoods characterized by high levels of social disorganization, broken windows,
signs of disorder, and crime pattern covariates.
We tested these hypotheses using homicide data for the 2016–2019 period in Mexico City
neighbourhoods. We present evidence that homicidal violence has spread to neighbour
hoods troubled with drug dealing crimes and concentrated disadvantage, yet
CONTACT Carlos Vilalta cvilalta@centrogeo.edu.mx Centre for Research in Geospatial Information Sciences
(Centrogeo), Contoy 137, Lomas De Padierna, Tlalpan, Mexico City, 14240 Mexico
© 2021 Informa UK Limited, trading as Taylor & Francis Group
2 C. VILALTA ET AL.
independently of signs of disorder and crime opportunities. In other words, the spatial
diffusion of homicide is likely to happen in concurrence with drug market competition
among criminal organisations operating in disadvantage neighbourhoods.
These findings are important for two main reasons. First, because they show that social
disorganisation, broken windows and crime pattern theories are able to explain the
spatial clustering of homicidal violence, but only social disorganisation, and more speci
fically, concentrated disadvantage, can explain the spatial diffusion process behind this
crime. This finding alone offers a shift from the traditional focus of studying homicide
change independently of its spatial spillover effects. Second, the experience of Latin
American cities is largely absent from criminological theory and research. However,
much of the homicidal violence problem is, and is likely to continue to be, a Latin
American problem (Vilalta & Muggah, 2020). Criminological theory thus clearly needs to
be empirically tested and expanded based on the Latin American case. In this sense, while
we do not update any theory, we present one novelty regarding the Mexican context,
namely, that homicidal violence cannot be studied independently of drug dealing crimes.
types of homicides in terms of victims’ and suspects’ journeys – the victims and suspects
of gang homicides travel the longest distances, whereas suspects in domestic homicides
travel the shortest distances (Pizarro, Corsaro, Yu, 2007). In the same vein, previous studies
have found that offenders in general prefer to travel to areas of low social cohesion
(Johnson & Summers, 2015).
To sum up, it seems that the spatial diffusion of homicide may be due to (1) crime
opportunity differentials in connection with gang violence (e.g. new versus saturated
drug markets), (2) emulation and copycat crime (i.e. social diffusion processes), and/or (3)
mobility and networking of offenders (i.e. journey to crime).
2015; Sampson & Raudenbush, 2004; Taylor, 2018; Vilalta, Lopez, Fondevila, Siordia, 2020;
Williams, Burnap, Sloan, 2017; Yang, 2010). Some studies have used principal component
analyses (PCA) and indexes to create measures of social disorder and physical disorder
(Gau & Pratt, 2008; 2010; O’Brien & Sampson, 2015; Sampson & Raudenbush, 1999; 2004;
Vilalta et al., 2019; Wilcox, Quisenberry, Cabrera, Jones, 2004; Yang, 2010).
With regard to empirical evidence, while some studies have found social disorder to
increase homicide (Alves, Lenzi, Mendes, Ribeiro, 2015; Culyba et al., 2016; César Fuentes,
2015; Patino, Duque, Pardo-Pascual, Ruiz, 2014; Rosenfeld, Fornango, Rengifo, 2007;
Vilalta et al., 2019), others have not found such a relationship (Cerdá et al., 2009;
Sampson & Raudenbush, 1999; 2001).
Crime pattern theory combines rational choice and routine activity theory to help
explain spatial variations of crime activity (Eck & Weisburd, 1995; Wilcox & Cullen, 2018).
Crime pattern theory predicts that land use characteristics create opportunities for crime
(Vânia Ceccato, Haining, Kahn, 2007; L. E. Cohen & Felson, 1979; McCord, Ratcliffe, Garcia,
Taylor, 2007; Vilalta, Castillo, Torres, 2016; Vilalta, Lopez, Fondevila, Siordia, 2020; Vilalta,
Muggah, Fondevila, 2020). Evidence shows that entertainment venues, bars and restau
rants correlate with more homicide (Vânia Ceccato, Haining, Kahn, 2007; Henderson,
Morgan, Patel, Tiplady, 2005; Hohl et al., 2017; Swart, Seedat, Nel, 2018; Vilalta, Castillo,
Torres, 2016; 2019; 2020). Liquor outlets and convenience stores selling alcohol also seem
to impact violent crime (Norza, Vargas, Avendaño, Rincón, Ospino, 2018; Snowden,
Stucky, Pridemore, 2017; Valasik, Brault, Martinez, 2018) including homicide in Mexico
City (Vilalta, Lopez, Fondevila, Siordia, 2020; Vilalta, Muggah, Fondevila, 2020). Non-
residential land uses also correlates positively with homicide rates (Cesar Fuentes &
Sanchez, 2015). Likewise, mass transportation facilities, such as metro stations or bus
stops, with a concentration of high numbers of motivated offenders with suitable targets,
have been found to be statistically associated with homicidal violence at the census block
level (Vilalta, Muggah, Fondevila, 2020), although not at the neighbourhood level (Vilalta,
Lopez, Fondevila, Siordia, 2020).
Clearly, these theories have been able to explain the spatial clustering of homicidal
violence in cities across the world by providing different views of criminogenic conditions
of places. One need not subscribe to one particular theory to hypothesise some type of
spatial effect associated with the social processes explained by these theories. What
remains to be seen is whether any of these theories can help explain the spatial diffusion
of homicide.
Hypotheses
In this study, we propose the following hypotheses:
● The spatial diffusion by ‘proximity’ Hypothesis (H1): The geographically closer neigh
bourhoods are, the more positively correlated time fluctuations in homicide will be.
Our expectation is that the spatial lag parameter, Rho, will be positive, indicating that
the closer the neighbourhoods, the more spatially dependent the year-to-year
fluctuations in homicide.
● The spatial diffusion by ‘drug market activity’ Hypothesis (H2): Homicide violence will
not spread to neighbourhoods randomly, but as a consequence of organised crime
6 C. VILALTA ET AL.
groups fighting for drug market territories. Our expectation is that the spatial lag
parameter, Theta, will be positive, indicating that the closer the neighbourhoods
with more drug dealing crimes, the more spatially dependent the year-to-year
fluctuations in homicide.
● The spatial diffusion by ‘criminogenic characteristics’ Hypothesis (H3): Spatial diffu
sion effects will be strongest in neighbourhoods with similar criminogenic condi
tions, measured by their levels of social disorganisation, signs of disorder, and crime
pattern opportunities. Our expectation is that the spatial lag parameters, Theta, will
be positive, indicating that the closer and more criminogenic the neighbourhoods,
the more spatially dependent the year-to-year change in the homicide rate.
Dependent variable
Criminal investigation data are managed by the local Office of the Attorney General of
Mexico City (Procuraduria General de Justicia de la Ciudad de Mexico). These data can be
downloaded directly from the Portal de Datos de la Ciudad de Mexico of Mexico City3. This
study used 4,510 criminal investigations for homicide (i.e. homicidio doloso) recorded
between January 2016 and December 2019, using geographic coordinates (point data) of
the incident. Homicide data were aggregated to neighbourhood level (polygon data) and
homicide rates were calculated per a population of 1,000 residents. Homicide rates (plus
a value of 1 to adjust zeros) were transformed to their natural logs to reduce skewness4.
Using a first-order queen-type neighbour matrix (i.e. strict contiguity matrix), the global
Moran’s I coefficient indicated significant positive spatial autocorrelation in the natural
logs of the homicide rates (I = 0.170, p = 0.001).
Independent variables
Hypothesis two (H2) states that homicide violence will spread across neighbourhoods as
a consequence of organised crime groups disputing drug markets. To represent drug
market activity, we used the number of criminal investigations for drug dealing crimes (i.e.
GLOBAL CRIME 7
narcomenudeo) also recorded between January 2016 and December 2019 and by geo
graphic coordinates (point data) of the incident. Previous studies have used similar
measures (e.g. drug arrests) as indicators of local drug activity in city case studies
(Contreras & Hipp, 2020; Warner & Coomer, 2003) including Mexico City (Vilalta, 2009).
After georeferencing, drug dealing crime rates (plus a value of 1 to adjust zeros) were
also transformed to their natural logs to reduce skewness. The source of data is the same
as with the dependent variable. We followed the same georeferencing procedures as
above.
A number of census-based variables were used to test Hypothesis 3 (H3). H3 is the
spatial diffusion by ‘criminogenic potential’ hypothesis. Following previous studies, we
used a variety of measures representing the criminogenic potential of neighbourhoods.
First, a concentrated disadvantage single component was constructed using principal
component analysis (PCA) that included the 15–29 year old (%) population of
a neighbourhood, Native indigenous speaking population (%), population without health
insurance (%), population without elementary schooling (%), and the average number of
persons per bedroom as proxy for crowding conditions5. These 2010 census-based
variables have already been used to measure concentrated disadvantage as a proxy for
social disorganisation in previous studies based in Mexico City (Vilalta, Lopez, Fondevila,
Siordia, 2020; Vilalta, Muggah, Fondevila, 2020). The PCA statistics are given in the
Appendix (Table A1).
Second, neighbourhood signs of disorder were operationalised through two variables:
social disorder and physical disorder (Sampson and Raudenbush, 2004; Yang, 2010). PCA
were also used to simplify the complexity of the social disorder and physical disorder
concepts. The data source for these components was the Mexico City 911 emergency call
system. This approach has already been used to create component measures of disorder
and informal social control (O’Brien, Sampson, Winship, 2015; O’Brien, Farrell, Welsh,
2019a; O’Brien & Sampson, 2015). We identified four types of 911 incidents for the
construction of the social disorder component: public intoxication with drugs, public
intoxication with alcohol, street scandals, and loud partying; and five types of 911
incidents for the construction of the physical disorder component: graffiti signs, drinking
water leaks, sewage spills, gas leaks, and downed power lines. Similar components have
also been used to predict homicide crimes in previous Mexico City studies (Vilalta, Lopez,
Fondevila, Siordia, 2020; Vilalta, Muggah, Fondevila, 2020).
Third, we used PCA to create a crime opportunity index at the neighbourhood level.
This index was calculated based on data from the 2017 National Statistical Directory of
Economic Units (DENUE), the Mexico City Open Data (CDMX) source, and the
OpenStreetMaps data source. The DENUE provided the geographical location of bars,
restaurants, and convenience stores with alcohol sales permits while the CDMX provided
the location of the metro and metrobus stations. These data were aggregated at the
neighbourhood level. The OpenStreetMaps data source was used to create a street
permeability index which was also included in the crime opportunity index. The street
permeability index was obtained by the weighted sum of the streets entering each
neighbourhood. Different weights were used to represent the different street capacity,
where a greater weight (i.e. 2) represents a main avenue while a lesser weight (i.e. 1)
represents a residential street. The higher the value of the neighbourhood index, the
higher its street capacity. The permeability index and metro and metrobus transportation
8 C. VILALTA ET AL.
variables were included in the crime opportunity index for two reasons: (1) to proxy
daytime population concentration, and (2) because mass transit tends to attract and
generate crime as it gathers large crowds of people (Gallison, 2016). The alcohol avail
ability variables (i.e. bars, restaurants, and convenience stores with alcohol sales permits)
were included in the crime opportunity index as previous studies have found a positive
spatial correlation with homicide in Mexico City (Vilalta, Lopez, Fondevila, Siordia, 2020;
Vilalta, Muggah, Fondevila, 2020).
Statistical analysis
One non-spatial and three spatial panel regression models were used to test our three
hypotheses. The random effects non-spatial panel regression was used as a baseline model
to compare the effectiveness of spatial models given that the residuals of this model were
spatially autocorrelated (I = 0.164, p = 0.001). One spatial panel model (SAR) was employed to
test for the spatial autocorrelation of the DV. Following this, one spatial Durbin (SDM) panel
and one spatial Durbin Error model (SDEM) were used to test for spillover effects of the IVs.
The former includes a spatial lag of the DV (i.e. Rho coefficient) as well as spatial lags of the IVs
(i.e. Theta coefficient), whereas the latter nests the spatial interaction of the IVs with the error
term (i.e. Lambda coefficient) to indicate whether there is spatial autocorrelation in errors
which may be due to the omitted bias. Among the advantages of SDM and SDEM is that they
include both endogenous (direct or local) and exogenous effects (indirect or spillovers),
produce unbiased coefficient estimates even in the case of misspecification, and do not
impose prior restrictions on the magnitude of spatial spillover effects (Elhorst, 2010;
Rüttenauer, 2019). Another advantage of the SDEM is that the spatial dependence of the
error terms is modelled separately from spillover effects.
Random effects were used since our DENUE and census-based independent variables (i.e.,
concentrated disadvantage and crime opportunity components) contain measures that are
not available on a yearly basis (e.g. population between 15 and 29 years old or bars and
restaurants). As such, these measures are time-invariant. One advantage of using random
effects is that we have hypothesised that differences across (rather than within) neighbour
hoods influence homicide rates6. For instance, H2 tests whether yearly fluctuations in homi
cide rates across neighbourhoods are due to differences between these neighbourhoods in
terms of previous levels of drug dealing activity (Contreras & Hipp, 2020).
Spatial panel models were tested using the queen first-order neighbour contiguity
matrix. One neighbourhood in the southernmost part of the city is not contiguous to any
other neighbourhood; however, it was forced into the regression analyses. The spatial
weight matrix was row normalised given that the number of neighbours for each spatial
unit was not the same. Row normalisation brought two benefits: first, the total effect of
neighbours remained unchanged across all spatial units no matter how many neighbours
a spatial unit has, and second, the spatial lags have the same metric as their original
variables, allowing for comparable interpretation.
SDM and SDEM regression coefficients are not directly interpretable as marginal
changes because of the infinite feedback loop created by the use of spatial lags (Pace &
Lesage, 2010). Thus, we calculated the direct (own-neighbourhood), indirect (spatial
spillover), and total impacts (i.e. partial derivatives) of each variable on the dependent
GLOBAL CRIME 9
variable. The software utilised were STATA v.15.1 and Geoda v.1.14. Results with alpha less
than 0.05 were considered statistically significant.
Results
Table 1 shows the descriptive statistics. The mean neighbourhood homicide rate
increased by 34% from 2016 to 2019. The coefficient of variation (CV) shows
a decreasing trend in variation in neighbourhood homicide rates over time, suggesting
a convergence trend in homicide risk across neighbourhoods.
Panel regression results of the SAR, SDM and SDEM are shown in Table 2. Estimated
direct, indirect, and total impacts of the SDM are presented in Table 3. With regard to H1,
we see that the spatial lag coefficient for homicide (Rho) is positive and highly significant
in the SAR and SDM, indicating that homicide spread over time to adjacent neighbour
hoods. This result confirms H1, that is, homicidal violence spread by proximity indepen
dently of other factors. However, some variables are missing in the model as indicated by
the statistical significance of the Lambda coefficient in the SDEM. The Lambda coefficient
indicates that homicide dynamics during the period of study can be characterised by both
spatial dependence and error (omitted variable) processes.
The Theta coefficient of drug dealing crimes is positive and statistically significant in
the SAR, SDM and SDEM, indicating that homicide spread over time to adjacent and
neighbourhoods more troubled by drug dealing crimes. This result confirms H2, namely,
that homicide spread to neighbourhoods suffering from drug dealing crimes, likely as
a consequence of organised crime groups fighting for drug markets. While the direct
effect of drug dealing is larger than the spillover effect, neither effect is small (see Table 3).
SDM and SDEM panels show positive own-neighbourhood and spillover effects of
concentrated disadvantage on homicide in adjacent neighbourhoods. However,
neither social and physical signs of disorder nor crime opportunity components
had significant spillover effects on homicide. According to previous studies in Latin
American cities, a positive and significant direct effect of concentrated disadvantage
on homicidal violence is expected (César Fuentes, 2015; Cesar Fuentes & Sanchez,
2015; Pereira, Mota, Andresen, 2017; Santos, Barcellos, Sacarvalho, 2006; Vilalta,
Castillo, Torres, 2016; Vilalta, Lopez, Fondevila, Siordia, 2020; Vilalta, Muggah,
Fondevila, 2020; Vilalta & Muggah, 2014).
The standard deviation of the time-invariant neighbourhood-specific effects
(Sigma_u) is about one-third the standard deviation of the residuals (Sigma_e) in
all regression models, suggesting that a small fraction of the variance of the model is
due to unobserved heterogeneity across neighbourhoods. In other words, even with
an omitted variable, the selection of variables provided a good fit to the data. SAR,
SDM and SDEM panel regressions offered similar AIC values, although the value for
the SDM was comparatively lower, suggesting a better fit.
Table 3. Direct, indirect and total impact estimates and marginal changes in homicide rates (LN).
Model (1)
SDM
Direct Indirect Total
Drug dealing 0.211*** 0.171*** 0.383***
(0.027) (0.042) (0.048)
Conc. disadvantage 0.052*** 0.034*** 0.086***
(0.007) (0.011) (0.008)
Social disorder 0.031*** 0.009 0.039**
(0.009) (0.016) (0.016)
Physical disorder 0.023*** −0.020 0.003
(0.010) (0.016) (0.018)
Crime opport. 0.048*** −0.017 0.032**
(0.008) (0.012) (0.012)
Standard errors are in parenthesis
*** p < 0.01, ** p < 0.05
presented evidence that homicidal violence spread by proximity across Mexico City
neighbourhoods, contingent on neighbourhood drug crime incidence and concentrated
disadvantage – but independent of disorder and crime opportunity. Likewise, we have
confirmed Contreras and Hipp (2020) argument that illegal drug markets can induce
a spatial spillover effect on homicidal violence.
While our selection of variables and PCA components depicting social disorganisation,
broken windows, and crime pattern theories were able to explain the spatial clustering of
homicide, only social disorganisation by means of concentrated disadvantage was able to
predict its spatial diffusion. Thus, only social disorganisation theory explains the spatial
process of homicidal violence – the other theories appear to only explain spatial patterns,
at least in the case of Mexico City.
The increasingly negative interaction between drug-dealing crimes and year in all
models is substantively important as it indicates that the neighbourhood-level relation
ship between drug activity and criminal homicide weakened over time7. This finding may
suggest that homicides either caused by or associated with organised crime drug dealing
operations declined in number during the period of study when analysed at the neigh
bourhood level. This remains speculative yet represents an interesting hypothesis for
future study.
In this respect, if we are to test and advance criminological theory based on the Latin
American case, some modifications need to be made that respond to context. For the
Mexican case, we know homicidal violence is strongly associated with organised crime
(Flores & Rodriguez-Oreggia, 2014; Flores & Villarreal, 2015), and organised crime is
strongly associated with drug dealing (Shirk & Wallman, 2015; Vilalta, 2014). In this
study, we confirmed the contagious spread of homicidal violence in neighbourhoods
with higher rates of drug dealing crimes. While this finding is not entirely new, given the
Contreras and Hipp (2020) study, it is nevertheless one of the least discussed processes of
spatial elements of crime. Our study compensates for this lack of research.
One limitation of this study is that while we have estimated the spatial effect of drug
dealing crimes on the spatial diffusion of homicide, we do not know why some neigh
bourhoods have high rates of drug dealing crimes in the first place. Another limitation is
that our study focuses on a single city. There might be something specific about Mexico
12 C. VILALTA ET AL.
City that explains the results of this study. In addition, we did not include the neighbour
hoods in the State of Mexico surrounding Mexico City as neighbourhood crime data for
the surrounding area were unavailable. Future research may also provide direct evidence
of the causes of the spread of violence, perhaps through interviews with drug dealers and
drug users.
In conclusion, criminological theories on the geography of homicide have provided
clear explanations for the spatial clustering of homicide, but spatial diffusion processes of
homicidal violence remain extremely understudied. In addition, most empirical evidence
to date is based on the developed world, yet, most of the homicidal violence problem is
essentially a Latin American one. Clearly, criminological theory cannot disregard the
examination of the spatial patterns and processes of crime and violence in the developing
world, given the gravity of the problem and its capacity to provide theoretical insights.
Notes
1. This report was not made publicly and we were not able to access it. However, some
journalists were able to access and report on it.
2. This cartography is a heavy data file, and thus can only be obtained by CD via a specific
request to the INE authority. See: https://portalanterior.ine.mx/archivos3/portal/historico/
contenido/interiores/Detalle_geografia_electoral_y_cartografia_transparencia-id
-02d4aee2ccdf0210VgnVCM1000000c68000aRCRD/
3. See: https://datos.cdmx.gob.mx/pages/home/
4. As suggested by one of the reviewers.
5. We are aware that previous studies include income (e.g. median family income) measures in
Concentrated Disadvantage indexes. As no such data exists in the Mexican Census, we were
unable to do this.
6. In a fixed-effects model, time-invariant variables are taken up by the intercept.
7. As correctly pointed out by one of the reviewers.
Disclosure statement
No potential conflict of interest was reported by the author(s).
Notes on contributors
Carlos Vilalta is Professor at the Center for Research in Geospatial Information Sciences (CentroGeo)
in Mexico City. He studies the geography of crime and fear of crime, prevention policies, criminal
statistics, and prison populations. He has been visiting researcher in UC San Diego, U. of Florida,
Cambridge, McGill, U of Missouri in St. Louis, Washington University in St. Louis, U. of Houston and
UNC-Chapel Hill. He holds a PhD in Urban Studies from Portland State University.
Pablo Lopez-Ramirez is coordinator of the master's degree at the Center for Research in Geospatial
Information Sciences (CentroGeo) and responsible for the DataLab project of the National
Geointelligence Laboratory. He has taught the subjects of Spatial Analysis, Geographic
Information Systems and Geoinformatics. His research focuses on developing links between tradi
tional research in Human Geography, especially from the perspective of Spatial Analysis, and Data
Science.
Gustavo Fondevila is a professor and researcher at the Centro de Investigación y Docencia
Económicas (CIDE) in Mexico CIty, where he concentrates on empirical and comparative quantita
tive criminology. Specifically, he focuses on criminal justice and prisons in Latin America by use of
GLOBAL CRIME 13
surveys of prisons, court records and more. His most recent work examines the relationship
between prison violence and criminal government within prisons of the region. He also studies
criminal justice institutions from a quantitative perspective, such as prosecution, defense and
courts.
ORCID
Carlos Vilalta http://orcid.org/0000-0002-6030-7018
Gustavo Fondevila http://orcid.org/0000-0003-4818-0584
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