Professional Documents
Culture Documents
NCM74
Care of Clients with Problems in Nutrition and Gastrointestinal,
Metabolism, and Endocrine, Perception and
Coordination, Acute and Chronic
Submitted by:
AMBAL, KISHA LONE G.
Submitted to:
Ms. Neda Joy L. Espina MAN, RN
Clinical Instructor
Gastroesophageal reflux disease (GERD) occurs when stomach acid frequently flows
back into the tube connecting your mouth and stomach (esophagus). This backwash
(acid reflux) can irritate the lining of your esophagus.
Precipitating factors
Obese- People who are obese or overweight seem to have an increased risk of
GERD. While the reasons for this are not well understood, it is thought to be related
to increased pressure in the abdomen.
Hiatal hernia- This is a condition in which part of the upper stomach pushes up
through the diaphragm (the large, flat muscle at the base of the lungs). The
diaphragm has an opening for the esophagus to pass through before it joins with the
stomach (called the "diaphragmatic hiatus"); in people with a hiatal hernia, part of
the stomach also squeezes up through this hole.
Pregnant- Many women experience acid reflux during pregnancy. This usually
resolves after delivery, and complications are rare.
Predisposing factors
Smoking- The nicotine from tobacco relaxes the valve between the esophagus and
stomach (lower esophageal sphincter). This can allow stomach acid and juices, the
chemicals that break down food in the stomach, to back up (reflux) into the
esophagus, which causes heartburn.
Alcohol- alcohol can relax the lower part of the esophagus, making it easier for your
stomach acid to back up.
Medication- medication that irritates your esophagus like ibuprofen, aspirin etc.
Eating large meals- Large meals make your stomach expand. This increases
upward pressure against the lower esophageal sphincter (LES). The LES is the
valve between your esophagus and stomach
Etiology
The presence of reflux implies lower esophageal sphincter (LES) incompetence, which
may result from a generalized loss of intrinsic sphincter tone or from recurrent
inappropriate transient relaxations. Transient LES relaxations are triggered by gastric
distention or sub threshold pharyngeal stimulation. TLESR) is defined as lower
esophageal sphincter relaxation that is induced spontaneously without swallowing.
TLESR is a physiological mechanism that enables venting of gas from the stomach.
Pathophysiology
Lower esophageal sphincter (LES) is weak and allows the stomach to reflux up into the
esophagus Gastroparesis contributes acidic stomach contents irritate the esophageal
epithelium Hiatal hernia interferes with the closure of the LES and results in reflux gastric
Clinical diagnosis- assess for signs and symptoms, physical exam and health
history to determine GERD.
Endoscopy- detects the inflammation of the esophagus or other complication.
24- Hour pH monitoring- performed to measure the pH or amount of acid that flows
into the esophagus from the stomach during a 24-hour period.
Prognosis
GERD is treated it lower the risk for long term damage of esophagus
Nursing Dx
Risk for aspiration r/t impaired swallowing risk for intragastric pressure
• Assess patient’s ability to swallow and the presence of gag reflex. Have the
patient swallow a sip of water.
• Avoid placing patient in supine position, have the patient sit upright after meals.
• Instruct patient to avoid highly seasoned food, acidic juices, alcoholic drinks,
bedtime snacks, and foods high in fat
• If the patient has dysphagia, put the patient on NPO and notify physician
• Fundoplication Surgery
• Assess the patient vital sign and characteristics at least 30 minutes after
administration of medication
• Help the patient to select appropriate dietary choices to follow a low fat diet with
minimal fried foods, alcohol and caffeine.
References