Professional Documents
Culture Documents
• Nonfluoride hypomineralization.
• Cheese molars.
Etiology
Oxygen shortage
combined with low Parentally risks:
birth weight infection,
Children with suspected to be maternal
systemic contributing psychological
conditions in factor stress and
their first 3 frequent exposure
years to ultrasonic scans
Multifactorial
Children born
with poor
general health
Respiratory
diseases and
oxygen shortage of
the ameloblasts
Prevalence
• Worldwide ranges from 3.6% to 40.2%
• Difficult to anaesthetize
• Aesthetic problems
Fluorosis :
Diffuse enamel opacities which
affect more than one tooth
Enamel hypoplasia:
The edge of enamel is smooth while in MIH
the post eruptive loss of enamel renders the
enamel edges sharper and more irregular.
Amelogenesis imperfecta:
•More symmetrical than MIH .
•Affect both sets of dentition.
Hypomineralization defect:
Etiological factors are due to local causes
like trauma or infection of the primary
predecessors.
Prevention
• Risk identification.
• Early diagnosis.
• Remineralization and
desensitization.
• Maintenance.
Treatment
Mild MIH
• Fissure sealants
( problem in retention) ??
N.B. Pretreatment of the pits and
fissures with 5% sodium
hypochlorite
• Resin infiltration (ICON)
• GI in difficult isolated or
partially erupted teeth
Moderate MIH
• extensive areas more
fracture
• If no enamel loss .. Ttt like
mild cases
• If enamel loss :
• Composite or GI
restoration .. Same
technique for sealants
• Amalgam could also be
used but not end on
severely affected areas
Severe MIH
The most challenging to treat
??
• Extreme sensitivity
Treated by chemical or light-cured
or resin-modified GI as temporary
restoration till full eruption of teeth
Provide effective barrier to thermal
and chemical stimulation
• Later SCC or Cast crowns
• RCT or apexification
• Extraction
Prognosis
• The quality of enamel supporting the restoration is fail so the
prognosis of restorations is poor.