OSTEOARTHRITIS

Secondary Cause TAKE NOTE:

- Give pain relievers before activity
Primary Cause - In OA- pain worsens at activity
- RA- Pain worsens at rest
Inflammatory - NURSING MANAGEMENT: Make
Obesity Age Wear & Tear High Impact Trauma Disease (IA, RA, Infection (SA, Anatomic Abnormalities sure to assess functional ability
Vitamin D Deficiency
Increased Physical Activities ReA) VA, BA) (Dyplasias, ehler danlos)
Activity for Weight Loss
especially s/sx flares up
Acetaminophen Muscle Pain
Liver Function Decreased absorption of Generates excess fat
Test calcium and phosphorous Synovial fluid inside the
Diclofenac Bone Pain Weight Reduction joints decreases and Repetitive stress
Program cartilage becomes on joints
Bones cannot mineralize Stress on cells
thinner
Consult a Nutritionist Changes in anatomical
maintain good minimize exessive
Plaster/Fiberglass Bone Fracture Over production of Decreased activity architecture
Subchondral Bone T2DM posture
Cast Glucose Hyaluronic Acid Soft tissue damage
Remodeling
Bariatric Surgery molecules

Risk for injury related to PBMC Promotion abnormal load on joints

decreased bone function as Cartilage Damage Increases friction Increased ESR (<50
in joints chrondocyte proliferate
evidenced by decreased mm/hr)
Swelling
calcium absorption Arthroscopic
Proinflammatory
Surgery bouchard node-
Cytokines Pain Naproxen Sodium hsCRP
Glucosamine Production Damages the excessive cartilage proximal
Stiffnes SKELAN <2 normal
cartilage proinflammatory damage> Chondrocyte heberden's node-
Hyaluronic Acid
MRI
3-9 acute inflammation mediators are remodeling distal
Administration >10 severe inflammation
Stimulate Synovial CT Scan Duloxetine Pain and inflammation
produced (cytokines)
Osteochondral Autograft Fibroblasts Cymbalta plain radiograph
Transplantation (Mosaicplasty) RICE Regimen reduction Chondrocyte
Decreases Cartilage proteoglycans attract senescence
Thickness Secrete enzymes that increased synovial decreased synovial
water, and articular increased bone
Microfracture degrades Proteoglycans fluid production fluid viscosity crepitus
and Collagen
Blocks cartilage expands palpable bony to bone contact
Inflammation of Joint Alternate Ice
Neurotransmitters hypertrophy
Cartilage Compress with impaction of formation of
Hot/Warm Compress +patellar Reduced osteophyte during osteophyes and joint surgery,
Joint Pain joint effusion MRI lubrication movement subchondral sclerosis osteotomy
Soft Tissue Destruction Reduces pain ballottement
Legends
Pain naproxen
Risk Factors/ Causes Swelling Acute pain related to
rest, ice or hot
Etoricoxib Impaired physical mobility cartilage degeneration as compress, and stiffness exercise increased bone disruption of normal
Signs and Symptopms Liver Function related to pain as evidenced by facial grimaces joint architecture supportive
Test
raising the joint to bone contact
evidenced by limited range shoes
Imaging Studies
of motion Arthrocentesis
Surgical Management Diclofenac Capsaicin Cream muscle contracture pain in motion, increased
stimulation of muscular Risk for falls as evidence
decreased ROM antalgic gait chances of
Pathophysiology nociceptors atrophy by antalgic gait
falls
Laboratory Results breathing colchicine
exercises (tab) Impaired physical mobility
Pharmacologic tx related to stiffness as
evidenced by limited range breathing
worsening pain sensitivity of Strenghtening
Medical Management
of motion exercises balancing ROM exercise
glucocorticoids surrounding nerves exercise stretching exercise
Nursing Diagnosis (via intra-articular
injection)
Impaired physical mobility
radiating pain related to pain as
Local Inflammation evidenced by limited range
treatment NSAIDS
of motion
(Ibuprofen,
Celecoxib) Gastric Upset

Nausea
Vomiting