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Active phase labour

75cm
I
Every z hrs

line
Alert reaction
She came in active
phasethus no need

Activethusfullyeffaced Donit
O position unknown
circle
haveto
only
indica
2ndstage
It pushing but no delivery

Assisted delivery fullydilated do an amnio


bony
Every curs Every w

Strongercontr
Headdescending Thee
Dilating perogession
Options
to i Reassess in this
Allow labour progress
Rupture membranes t Give oxytocin
I usually measted
othercontraction
when at 8 9cm
is acceleration
MI feetuns toolong

curs
at labour Reassess in 1
Allow progression
no 2 crest
Make sure

No evidence at total distress AFAR


CPD no HOBdescent
caput moulding
5 dilated
Fully
precognition

strongcontr
adequate

justcolour
the first le
MI
Prepare for delivery

Indicationsforc aFetal
distress
CPD
NB in porogram
Poorprogress at labour
Placenta pervia etc
110160
stor 30 59 mins star so mus
g 25
Early
can
variable
spontaneous
controlled SNS PNS
Variability is
by

Fetaldistress
due to acidosis
Baseline FAR 1406pm or hypoxia
wakecycle
stays
Decreased
t
variability pethidine
for 15 mins
non mas hmm
y No accelerations dugout immune

e Nodecelerations
Tocobelt not on

keep on Ctg
for 60 mins
To if variability
Ff
see
Suspicious stays h
g
In labour engaged fullydilated s oxytocin
If pathological tassived
not in labor 45
Pre eclampsia and 26wks feta agnes deny
INCR
Nor in labour we only Spontaneous decelerations Abnormal
i
at nanny
pt b No accelerations
m

T T 4The

iI
lines
look Badbility
M 9
straight

Absent contr

Tocobelt is on

It Pathological Mx Ifook
Iv line www
my
forCls s deliver
Poor variability
Spontaneousdecelerations

Bt 1256pm

Absent conor

fat Pathological
root spontaneous decelerations
variability

we assume
no contr
even tho no tocobelt

Cat Pathological
compromises
Coupled contr
y
bloodhow to
Uterus not tears
many
Conto
paths not part

I
ii
I Einstein I

4 3 2 1

count contr over 30 mins


In Analgesic
Always
then 3 Cat Reassuring
Nodecelerations

Goodvariability erosion

Absent contr

Eat Reassuring
Mmff variability
Poor
variability
Noaccelerations

Whan acceleration Laddecelerations


Abnormal

refs
BL 1656pm Non swing

partat
decal
acceleration returns
s FYI conn
I 1
Ether
I
8 a 10
2 3 4 5 6 7

toped Excessive t coupled conto


not return to But

fat Pathological
decels
Excessive uterine
e activity
Poorvariability for comin
No accelerations

Alamofraight
line

I 9 9 Bl 1406pm q
end
and
s de
Assume 1cm min

coupled
5
prolonged
gang
far Pathological
indicated forrenal distress
Ftgffmmontistroz to
baby datong'd
Poor
variability Iv fluids
Noaccel
Stopoxytocin
Spontaneousdecal Tocolytic it strongcoupled conto
plan to a

fÉÉÉf
t lo mins
t t e was

NoHOB
app

BL 1405pm Rom
Fullydilated

y a
e

No tocobelt
Assume no contr
Latedeeds
e Poorvariability
Excessive uterine conto
t Moderate
tachy

BL1606pm

0 O
coupled conn

G Pathological
e Excessive uterine contr

t decels
might
even be variable

Be No accels
DOModerate tachy
BL 1656pm

Ok
variability
or

coupled contr

fat Pathological
Byi3obpm 1406pm
r r r a
Good variability Accelerations

It Reassuring
Poor
variability
Excessive uterine contr

BL 1506pm

0
coupled
Ma Run for 30 60 mins
Most
likely cause
is HYPOXIA
to see it an
paralog presence at
excessive caw
Sinusoidal pattern very poor variability

Pathological
Poor variability

Excessive uterine activity


Late decels
Spontaneous decels
dvariability
dvariability
Abels

coupled conn
BL 170

late decels
Lale decels
Poor varabiling
Decels

v u v

Pathological
Lane deals
Tachy
Excessive uterine contr
Late decels

Tacky

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