Professional Documents
Culture Documents
vii
Foreword
Acquiring knowledge and developing clinical competency research and scientific knowledge specific to each region
are two of the greatest challenges facing orthopedic manual are analyzed and correlated to support the tests, tech-
therapists. Knowledge provides the solid foundation for evi- niques, and clinical reasoning presented. Each chapter
dence-enhanced practice and increasingly establishes the pa- provides the reader with an extensive bibliography to
rameters that define the scope of orthopedic physical therapy. facilitate further investigation and underscore the sup-
Clinical competency requires the succinct synthesis of porting evidence.
anatomy, physiology, and current research to serve as a A major strength of the text is the extensive and compre-
framework for the utilization and development of appropri- hensive technique section, which is representative of a wide
ate evaluation and treatment techniques. variety of manual therapy philosophies. This integrated
An orthopedic manual therapist cannot be complacent approach allows the therapist to compare and investigate
in the pursuit of knowledge. Regardless of the therapist’s the most appropriate method of intervention for each
governing regulations, all orthopedic manual therapists patient, rather than being directed into one specific system
must function as a direct access practitioner in the sense of or viewpoint. In addition, over 700 color pictures supple-
competency and responsibility. The rapidly evolving disci- ment this section, facilitating a more efficient understanding
pline of manual therapy dictates the aggressive pursuit of and application of the techniques presented.
updated knowledge and skills. The second edition of The first edition of Orthopedic Manual Therapy immedi-
Orthopedic Manual Therapy: An Evidence-Based Approach ately became required reading for our Functional Manual
effectively presents the foundations of patient manage- Therapy Fellowship program in 2007. This text has pro-
ment, sound clinical reasoning, reflective practice, and vided the fellows in training with a comprehensive
problem solving, which assists in the management of the resource for information that guides the appropriate evalu-
unique challenge presented by each patient. Our patients ation, testing, and treatment of each patient, in addition to
require and deserve our ongoing commitment to excellence being the primary resource in studying for their examina-
and development of our knowledge of human behavior tions. I eagerly anticipate the release of the expanded and
and function to achieve optimum resolutions. enhanced version, bringing to our profession an up-to-date
Dr. Cook’s comprehensive, regional approach to the synopsis of relevant research and techniques for all ortho-
body allows the practicing therapist to synthesize current pedic patient populations. I am confident the second edi-
research, didactic knowledge, and clinical expertise in one tion of Orthopedic Manual Therapy: An Evidence-Based
resource. Chapters 1–4 provide detailed information per- Approach will continue to augment the advanced training
taining to orthopedic manual therapy assessment, evalua- of not only Manual Therapy Fellows and Residents, but all
tion, treatment, and contraindications. This carefully physical therapists dedicated to excellence in clinical man-
organized preamble to the regional technique section is agement and patient care. I am honored to write a Fore-
necessary reading for all physical therapists, regardless of word to such a high-quality and important text.
one’s area of specialty or experience. A therapist’s ability
to recognize and appropriately address the orthopedic Gregory S. Johnson, PT
issues with any patient requires a knowledge base sup- Co-Founder, Institute of Physical Art, Inc.
ported by a comprehensive understanding of anatomy, President, Johnson and Johnson Physical Therapy, Inc.
pathology, and applicable research. The following 12 chap- Vice Chairman, Functional Manual Therapy Foundation
ters offer an in-depth, regional insight into the prevalence Program Director, FMT Fellowship
of musculoskeletal conditions supported by the relevant Program Administrator, FMT Orthopedic Residency
anatomy, biomechanics, clinical examination protocols, Secretary, FMT Certification Board
treatment techniques, and outcomes. Within this section, Associate Professor, Touro College
viii
Preface
It is with great pleasure that I present the second edition within- and between-session findings, and a brief discus-
of Orthopedic Manual Therapy: An Evidence-Based sion on classification. Gone from the second edition is the
Approach. As is appropriate for any text espousing evi- level of detail on special tests, which isn’t truly germane to
dence-based elements, I have updated and expanded the care provided by a manual therapist. In its place are the
accordingly. As I mentioned in the preface to the first edi- presentations of more home exercise activities for carry-
tion,1 literature describing and measuring “evidence- over of care and mobilization with movement techniques
based” care has grown significantly over the last decade.2 to further broaden the scope of the text.
The core components of evidence-based concepts were What remains a bastion of this text is its emphasis on the
developed in the 1970s and 1980s with the application of debunking of myths and its polarizing discussion on weak-
epidemiological principles of patient care.3,4 These epi- nesses of certain manual therapy approaches. The text still
demiological principles advocate that using evidence- exposes faulty philosophies, theories, and other clinical
based care allows clinicians to apply the current best provisos that are advocated and does so for the sake of sim-
evidence from research to the clinical care of the individ- plicity. Good manual therapy (provided with the appropri-
ual patient.3,5 The overwhelming collective evidence is ate motor training) should not be so complex that all
daunting and the ability of one textbook to capture all clinicians can’t use it. And if I’m wrong in my take of the
aspects is simply unattainable. evidence, then I do so in the spirit of simplicity and clinical
But it’s not as if we haven’t tried. The second edition has utility for all therapists.
enlisted the assistance of a number of new collaborators,
including Christopher Fiander, Amy Cook, Megan Donald- Chad E. Cook
son, and Roy Coronado. Ken Learman has contributed an Professor and Chair, Walsh University
additional chapter and Bob Fleming has returned to update
the knee chapter. In addition, two new chapters have been
added to expand the material within the textbook. A neuro-
dynamics chapter (Chapter 15) and a soft tissue mobiliza- References
tion chapter (Chapter 16) should improve the
comprehensiveness of the text, which now covers all ele- 1. Cook C. Orthopedic manual therapy: An evidence-based
ments of manual therapy. Chapters 5–16 each have two or approach. Upper Saddle River, NJ; Prentice Hall: 2007.
three dedicated patient cases and we’ve included videos of 2. Cohen AM, Stavri PZ, Hersh WR. A categorization and
selected techniques to improve the understanding and analysis of the criticisms of evidence based medicine. Int
carryover to the clinic. Visit www.myhealthprofessionskit. J Med Informatics. 2004;73:35–43.
com to view these videos. To further improve the text- 3. Sackett DL. The fall of clinical research and the rise of clin-
book’s ease of application in a clinical setting, we’ve placed ical practice research. Clin Invest Med. 2000;23:331–333.
the anatomy and arthrological elements (with new illustra- 4. Buetow MA, Kenealy T. Evidence based medicine: the
tions) online at www.myhealthprofessionskit.com. need for new definition. J Evaluation Clin Pract. 2000;
The most notable difference from the first edition is the 6:85–92.
further emphasis on clinical decision making. Different 5. Sackett DL, Strauss SE, Richardson WS, Rosenberg W,
models of decision making are discussed as are clinical Haynes RB. Evidence-based medicine. In: How to practice
decision-making aids such as clinical prediction rules, and teach EBM. Edinburgh; Churchill Livingstone: 2000.
ix
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Acknowledgments
I’d like to acknowledge the following individuals who have Chris Showalter and my friends at MAPS: Toga! Toga!
significantly impacted the material within this textbook: Phillip Sizer, Jr.: The man never sleeps!
Special thanks to Steve Houghton who created the
Jean-Michel Brismee: A great clinician, but an even better
majority of the tables for the Anatomy and Biome-
person.
chanics sections of each chapter.
Amy, Zach, Jaeger, and Simon Cook: “Dad, quit check-
My former students from Duke University: Intellectual
ing your emails!”
juggernauts.
Bob Fleming and Ken Learman: Solid friends and col-
Geoff Maitland, Gregory Grieve, Bob Sprague, Bob Elvey,
laborators.
and the numerous other manual therapy pioneers that
Eric Hegedus: You are making progress, Sisyphus!
have created the framework in which manual therapy
John Medeiros and the people involved with the Journal
stands: We are riding your wave—and it’s a BIG one.
of Manual and Manipulative Therapy: A gentlemanly
leader of an unpretentious journal.
xi
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Contributors
Amy Cook, PT, MS Christopher Fiander, DPT, OCS
Contract Physical Therapist Senior Physical Therapist
North Canton, Ohio Department of Physical Therapy and Occupational Therapy
Duke University
Rogelio Coronado, PT, MS, FAAOMPT Durham, North Carolina
PhD Student
Department of Rehabilitative Sciences Robert Fleming, Jr., PT, DPT, MS, OCS, FAAOMPT
University of Florida Rehabilitation Services Manager
Gainesville, Florida Ellis Hospital
Schenectady, New York
Megan Donaldson, PT, PhD, FAAOMPT
Assistant Professor Ken Learman, MPT, PhD, OCS, FAAOMPT
Department of Physical Therapy Associate Professor
Walsh University Department of Physical Therapy
North Canton, Ohio Youngstown State University
Youngstown, Ohio
xiii
Reviewers
Second Edition
Dr. Jason A. Craig, MCSP, DPhil, PT John Leard, EdD, PT, ATC
Marymount University University of Hartford
Arlington, Virginia West Hartford, Connecticut
Michelle Dolphin, PT, DPT, MS, OCS Marcia Miller Spoto, PT, DC, OCS
SUNY Upstate Medical University Nazareth College
Syracuse, New York Rochester, New York
Megan Donaldson, PT, PhD Clare Safran-Norton, PhD, MS, MS, PT, OCS
Walsh University Simmons College
North Canton, Ohio Boston, Massachusetts
Marcia Epler, PhD, PT, ATC Michael P. Reiman, PT, DPT, OCS, ATC, CSCS
Lebanon Valley College Wichita State University
Annville, Pennsylvania Wichita, Kansas
Lisa T. Hoglund, PT, PhD, OCS, CertMDT Toni S. Roddey, PT, PhD, OCS, FAAOMPT
University of the Sciences in Philadelphia Texas Woman’s University
Philadelphia, Pennsylvania Houston, Texas
First Edition
Stephania Bell, MS, PT, OCS, CSCS Kenneth E. Learman, MEd, PT, OCS, COMT, FAAOMPT
Kaiser Hayward Orthopedic Manual Therapy Fellowship Youngstown State University
Union City, California Youngstown, Ohio
Jean-Michel Brismee, ScD, PT, OCS, FAAOMPT Christopher R. Showalter, LPT, OCS, FAAOMPT
Texas Tech University Health Sciences Center Maitland-Australian Physiotherapy Seminars
Odessa, Texas Cutchogue, New York
xiv
Chapter
Orthopedic Manual
Therapy
Chad E. Cook 1
Objectives
• Define orthopedic manual therapy.
• Outline the mechanical changes associated with manual therapy intervention.
• Compare and contrast the effects of static stretching, manually assisted movements, mobilization, and manipulation.
• Outline the neurophysiological changes associated with manual therapy intervention.
• Outline the proposed psychological changes associated with manual therapy intervention.
• Compare and contrast the different methods of reporting of evidence.
• Outline the levels of evidence used to judge quality of information.
What Constitutes Orthopedic ual therapy may reflect selected passive or active
assistive techniques such as stretching, mobilization,
Manual Therapy? manipulation, and muscle energy–related methods
(Table 1.1 ■). Each application is used for the purposes
Construct
of modulation of pain, reducing or eliminating soft
Within the professional fields of medicine and reha- tissue inflammation, improving contractile and non-
bilitation, orthopedic manual therapy is best defined contractile tissue repair, extensibility, and/or stabil-
by the description of the application. Orthopedic man- ity, and increasing range of motion (ROM) for
■ TABLE 1.1 Definitions of the Most Common Applications of Orthopedic Manual Therapy
Construct/Application Definition
Passive stretching Passive technique involving application of a tensile force to tissue in an effort to increase the
extensibility of length (and resultant range of motion) of the targeted tissue.
Mobilization Passive technique designed to restore full painless joint function by rhythmic, repetitive passive
movements, well within the patient’s tolerance, in voluntary and/or accessory ranges.
Manipulation An accurately localized or globally applied single, quick, and decisive movement of small amplitude, fol-
lowing a careful positioning of the patient.
Muscle energy technique A manually assisted method of stretching/mobilization where the patient actively uses his or her
muscles, on request, while maintaining a targeted preposition against a distinctly executed
counterforce.
Passive mobilization with A passive technique that consists of a rhythmic, repetitive passive movement to the patient’s tolerance,
an active movement in voluntary and/or accessory ranges, performed concomitantly with an active movement of the patient
at the same region.
1
2 Chapter 1: Orthopedic Manual Therapy
facilitation of movement and return to function. Other from the scientifically pertinent to the inexplicably
types of “manual therapy” that make curative claims strange. To date, most theories remain hypothetical,
beyond those I have just outlined are outside the have involved investigations that were poorly
scope of this textbook and often offer dubious claims designed, or were predominantly promoted by per-
of mental, physical, and behavioral changes. The sonal opinion. There are no shortages of hypotheses
validity of other methods requires further study. Truly, driven primarily by researchers and theoreticians in chi-
the variations in the types of manual therapies are ropractic, physical therapy, osteopathic, and massage-
extraordinary, specifically when one explores the based fields. Hypotheses have included; movement of
incongruent philosophical and theoretical constructs the nucleus pulposis5,6 activation of the gate-control
of each type. mechanism,7 neurophysiological and biomechanical
responses,8,9 and resultant reductions in paraspinal mus-
cle hypertonicity.10,11
Terminology
The constructs behind the use of stretching, mobi-
Simply stating that the terminology of manual ther- lization, manipulation, and muscle energy–related
apy is inconsistent among its users may be consid- methods are similar and share comparable indica-
ered the penultimate understatement. The variations tions and contraindications for use. Most impor-
in language have prompted a call for consistency1 tantly, the application of each treatment method
and the development of standardized manipulation results in similar functional outcomes and compa-
terminology in practice.2,3 The proposed terminology rable hypothesized effects. 12 These hypothesized
reflects descriptive language designed to homoge- effects are frequently categorized as biomechanical
nize how one describes an application or technique. and neurophysiological,8,13 with an understanding that
The terms advocated are (1) rate of force application, the two effects have significant interactions that
(2) location in range of available movement, (3) direc- improve one another (Table 1.3 ■). Additionally,
tion of force, (4) target of force, (5) relative structural manual therapy may provide measurable psycho-
movement, and (6) patient position (Table 1.2 ■). In logical changes such as relaxation, decreased anxi-
nearly all circumstances these terms are adopted by ety, or improved general well-being. The majority
this textbook during the descriptions of each of the of this chapter is dedicated to analysis of these
techniques. three areas.
Term Definition
Rate of force application The rate at which the force was applied during the procedure.
Location in range of available Where in the availability of range of the segment the application was applied.
movement
Direction of force The direction in which the force is applied.
Target of force The location in which the therapist applied the force (e.g., level of the spine, area of the periphery).
Relative structural movement The movement of a targeted structure in comparison to the stable structure.
Patient movement The position of the patient during the application of the procedure.
Term Definition
Biomechanical
synovial fluid within the joint cavity, which results in The amount of movement necessary for reduction
an increase in intra-articular pressure during of symptoms is also unknown. Overall, most studies
movement.14 Consequently, the distance between have either been poorly performed,26,27 have used
articular surfaces declines and reduces the lubricating spines from cadavers for the experimental analysis,28 or
properties of the joint, thus increasing irregular have reported the effect of manipulation on the spine of
collagen cross-links.15,16 Cross-links between collagen- a canine.29 Additionally, one well-cited study used sur-
based fibers inhibit normal connective tissue gliding, face markers during assessment of joint-related move-
which leads to restricted joint movement17 and ments.24 The use of surface markers is associated with
corresponding range-of-motion loss and impairment. a high degree of error since the measurement of skin
Additional contributors such as intra-articular displacement is a component of the movement. Sub-
meniscoids,18 entrapment of a fragment of posterior sequently, the findings of studies that have investigated
annular material from the intervertebral disc,19 and movement using skin markers or other erroneous
excessive spasm or hypertonicity of the deep intrinsic devices may provide misleading results.
musculature20,21 may further the impairment of joint When explaining the biological benefit of manual ther-
mobility. Consequential debilitating changes include apy for biomechanical improvements it is important to
impaired strength, endurance, coordination, and note that most changes reported have been short-term
alterations in the autonomic nervous system.22 in nature.30 Lasting structural changes are rarely identi-
Some evidence exists that mobilization and/or fied (if at all) and immediate benefits are likely reflective
manipulation techniques solicit joint displacement.23 of muscle-reflexogenic changes or neurophysiological
In theory, this joint displacement solicits a temporary alterations of pain. Furthermore, whether or not true
increase in the degree of displacement that is pro- positional faults are corrected is also unknown as the reli-
duced with force due to hysteresis effects.24 Chiro- ability and validity behind this concept is questionable.30
practors suggest that when joint structures are
rapidly stretched, cavitation internally occurs and
an audible “pop” may be heard, resulting in
increased range of motion after the cavitation.25 Summary
What is unknown is whether the movement or • Although very limited in gross amount, joint displacement does
the corresponding neurophysiological changes occur during manipulation and mobilization.
are responsible for the increased movement and • Joint displacement may be associated with an audible pop.
whether the new range of movement is maintained
• An audible pop is not necessary for neurophysiological changes.
over time.
4 Chapter 1: Orthopedic Manual Therapy
mediators at the region of the application. During an Other explanations have included the activation of
injury, a chemical reaction occurs that produces a the gate-control mechanism proposed by Melzack and
cascade of chemically related pain. Injury may Wall,69 neural hysteresis, and release of endogenous
stimulate the release of proteoglycans, metalo-matrix opioids. Small-diameter nociceptors tend to open the
protease inhibitors, and other factors that trigger an “gate,” thus facilitating perception of pain, whereas
autoimmune reaction and the influx of spinal cord larger-diameter fibers tend to close the gate of pain.
mediators such as bradykinin, serotonin, histamines, Gating pain is a mechanism in which afferent and
and prostaglandins that irritate surrounding Type C descending pathways modulate sensory transmission
nerve endings. The result is a diffuse pain that is by inhibitory mechanisms within the central nervous
activated during “normal” activity that usually would system. Some have suggested that manual therapy
not stimulate pain.61 The passive movement associated movements may stimulate afferent fibers in the joint,
with mobilization and manipulation may change the muscle, skin, and ligaments, potentially providing an
pH structure and alter the acute inflammatory effective overstimulation response, although further
response of the area, thus resulting in decreased pain, work is needed to confirm this theory.
although further study is needed for substantiation.
Temporal Effects The temporal effects of manual
Central-Mediated Mechanisms Manual therapy
therapy procedures such as manipulation, mobilization,
may affect the central and peripheral mechanisms of
or muscle energy techniques, when not combined with
pain control and create neurophysiological responses
another intervention, are short term.59,70,71 Studies sug-
and changes in pain perception.41,62,63 Central facili-
gest a carryover effect of 20–30 minutes only. Con-
tation occurs when the dorsal horn is hyperresponsive
sequently, to maximize the benefits of manual therapy,
to afferent input.64 This process may cause a lowering
follow-up exercises that strengthen or move the patient
of the pain threshold and results in lower levels of pain-
into the newly gained range of movement may be
producing stimuli. Central facilitation may occur
necessary for long-term outcomes. Further investigation
regionally at the injured site or in the brain’s pain
is needed.
processing centers. Manual therapy may provide
alterations in pain “experience” in the anterior cingular
cortex, amygdala, and rostral ventromedial medulla, Summary
including a lessening of temporal summation, a central • It is suggested that manual therapy demonstrates pain reduction
nervous system (CNS) condition that demonstrates an through inhibition of nociceptors, dorsal horn, and inhibitory
increase in perception of pain to repetitive painful descending pathways of the spinal cord.
stimuli.30 Passive mobilization forces arouse descending • Manual therapy may improve chemical alterations secondary to
inhibitory systems that originate in the lateral injury and CNS thresholds.
periaqueductal gray matter of the brainstem.41 • Both manipulation and nonthrust mobilization forces have demon-
There also appears to be a reduction of afferent noci- strated neurophysiological changes in discriminatory analysis.
ceptive input into the CNS, thus evoking descending • Manual therapy may improve altered pain thresholds.
pain inhibitory systems43,59 consequently resulting in
• Manual therapy, specifically nonthrust mobilization, has been
hypoalgesia. The reduction of pain through descending
shown to demonstrate a sympathoexcitatory effect.
mechanisms appears to happen by two separate path-
ways. The primary (rapid-onset) analgesic effect is from • Sympathoexcitatory activity and hypoalgesia appear to function
concurrently and are considered positive-responsive during an
the dorsal periaqueductal gray (PAG) area and is sym-
application of manual therapy. A primary role of skin, muscle, and
pathoexcitatory in nature.65,66 This is a nonopioid mech- articular joint receptors is to detect the presence of movement or
anism because it is unaffected by the administration of energy input and provide the central nervous system with pro-
naloxone.67 The secondary mechanism is from the ven- prioceptive or nociceptive information.
tral PAG and is sympathoinhibitory in nature and is • There are four primary articular receptors.
referred to as an opioid mechanism.66 It is described as
• There are theories dictating why reflexogenic changes occur,
opioid because administration of naloxone will attenu-
including stimulation of mechanoreceptors, resetting reflex
ate the effect.67 The preceding mechanisms of pain con- responses, and the gate control theory.
trol have been clearly linked to spinal manipulation but
• One theory is that manual therapy techniques “reset” the reflex
not as strongly linked to spinal mobilization according
activity by stimulating the muscle spindles and Golgi tendon organs.
to Wright’s review article.43 There is moderate evidence
to support that spinal manual therapy has a hypoalgesic • Measurable alterations in electromyographical (EMG) activity in
local and distant spinal muscles and depression of the H reflex
effect specific to mechanical nociception.39,40,68 However,
have been documented after use of mobilization and/or manipu-
the majority of studies were poorly designed or resulted lation methods.
in conflicting findings among authors.8
6 Chapter 1: Orthopedic Manual Therapy
pain. There is some evidence to support that reduction accurate distinction between chronic pain and a
in levels of distress, pain, tension, discomfort, and chronic pain syndrome. Theoretically, the most
mood are possible with treatments such as massage.83 effective treatments designed to improve coping
Because development of a chronic pain syndrome strategies should incorporate both psychological and
appears to reflect a failure to adapt to the change in physical components and require intervention by an
condition,82 treatment of pain exclusively may still interdisciplinary team. Generally, early treatment
result in a regression of the patient’s status. Instead of pain syndromes may improve employment-related
of true pain-related changes, most individuals fail to outcomes, but even those with long-standing
cope with the unimproved symptoms and the syndromes generally improve dramatically.89
decrease in function. The presence of selected psy- Improvements in coping include the use of a
chosocial factors that interfere with adaptation may biopsychosocial model. A biopsychosocial model
promote the development of pain syndromes. These assumes an interaction between mental and physical
factors include the derivatives of emotion, beliefs, and aspects of disability, assumes that the relationship
coping strategies. between impairment and disability is mediated
by psychosocial factors, and that beliefs about
Emotions Main and Watson78 identify anxiety, illness/disability are as important as illness. Presence
fear, depression, and anger as the four emotions that of a chronic pain syndrome strongly suggests that
best characterize the distress of those with chronic medical interventions (including surgery) may not
pain. Much of the patient anxiety may be traced to be effective.89 In some instances of physical
unmet expectations. Anxiety is often present in improvements, separate psychological interventions
patients who have not received a clear explanation for may be necessary for reducing back pain incidence.90
the origin or cause methods to manage pain.78
Fear is an emotional response that stems from a
belief that selected movements or interventions may
damage one’s present condition.78 Fear has been asso- Summary
ciated with catastrophizing behavior and may increase
patients’ self-report of pain intensity.84 Most notably, • The placebo effect could potentially explain some of the pain
fear of movement may reduce a patient’s buy-in to a reduction benefit associated with manual therapy.
particular treatment, specifically if pain is reproduced • It is difficult to design a study in which an effective and compa-
within the treatment process. Fear of movement or rable placebo sham is used during manual therapy intervention.
reinjury and subsequent hypokinesis is highly corre- • Treatments that consist of manual therapy techniques routinely
lated with an increased pain report.85 display better patient satisfaction scores than other nonmanual
Depression is more difficult to acknowledge. Main therapy–related methods.
and Watson78 suggest that it is important to distinguish • Manual therapists may improve the likelihood of meeting patient
between dsyphoric moods from depressive illness. expectations secondary to the nature of the physical intervention.
Dysphoric behavior is common in patients who have • Failure to meet patient expectations is associated with poor patient
experienced long-term pain but will most likely be satisfaction.
absent of the debilitating effects of depression. Depres- • Anxiety, fear, depression, and anger are common emotional com-
sion often leads to a learned helplessness, dependency ponents that may alter a manual therapist’s outcome.
of pharmaceuticals, and other debilitating behaviors. • A manual therapist may reduce the anxiety associated with
The complex relationship between anger and frus- unknown symptoms.
tration is not well understood78 but is believed to alter • Fear is commonly associated with decreased movement and trep-
judgment and may reduce the internal commitment the idation of reinjury.
patient has to improving his or her own condition. • Depression coexists with numerous other variables, all which can
Recent evidence suggests that expressive anger style is lead to poor patient outcomes.
associated with elevated pain sensitivity, secondary to
• Anger and outcome are poorly understood, yet there does appear
dysfunction within the body’s antinociceptive system.86 to be a relationship between higher report of pain and increased
anger.
Coping Strategies During their discussion of • Coping strategy is reportedly a reason why some disorders
coping strategies, DeGood et al.87 distinguish three progress to chronic pain syndrome.
distinct fields of inquiry: (1) specific beliefs about
• There is little evidence to suggest that manual therapy interven-
pain and treatment, (2) the thought processes tion will decrease the progression to chronic pain syndrome.
involved in judgment or appraisal, and (3) coping
• Purportedly, a biopsychosocial model should demonstrate effec-
styles or strategies. Schultz et al.88 report that effective
tiveness in treating patients with chronic pain syndrome.
treatment to improve coping strategies requires
8 Chapter 1: Orthopedic Manual Therapy
Case series
Case reports
Animal research
questions are defined at a relevant baseline point exists that lack statistical power.102 The calculations
(prior to when outcomes occur). Retrospective require essential methodological elements for com-
cohorts involve a longitudinal study where a group bining data and statistical information across
or groups of patients are involved in prospective sources96–102 such as an estimate of effect size and
data collection but the research questions (and vari- assessment of heterogeneity of data available for com-
ables) were defined retrospectively. parison.96 Meta-analyses enhance precision by improv-
For individual trials, randomized controlled trials ing effect estimation. Effect estimation is enhanced by
(RCT) are considered the highest level of evidence.95 providing comparisons of characteristics not involved
However, although strong with respect toward pro- in the original root studies and by answering ques-
viding utility of a particular intervention, an RCT tions about whether conflicting studies exist.97 Cumu-
should demonstrate strong internal, external, and lative results from the meta-analysis can display the
model validity. Internal validity reflects the ability of relative change in magnitude of the effect size or
the independent variable to affect the dependent vari- empirical evidence on how the treatment effect has
able. If the experiment can clearly establish that the changed over time.97
treatment causes an effect, then the experiment has RCTs are the framework of meta-analyses, although
internal validity. External validity reflects the ability observational cohort studies have been used in the
to generalize beyond the specific study, including the past as well. Low-quality RCTs result in poorly
ability to translate the findings to other settings, with homogenous meta-analyses and potentially biased
other subject populations, and with other, but related data for health-care clinicians.
variables. Model validity is a component within
external validity, and reflects how well the study
Managing the Quantity of Information
design actually models real practice settings. In
essence, a model resembles the target system in some The 5S Model One of the most significant
aspects while at the same time it differs in other roadblocks to the use of evidence-based information is
aspects that are not considered essential. If a study the shear quantity of evidence available that the
design is technically correct, but differs so signifi- clinician must consume. Success in delivering an
cantly from actual clinical practice, the study may evidence-based method relies heavily on the ability to
lack external validity. take information and transpose it toward evidence-
When a series of studies are available, systematic based health-care practice for each individual patient.
literature reviews or meta-analyses are more useful Although no information is perfect and the transfer of
tools for assessing the effect of a particular interven- information is often altered in methods that impact
tion. A meta-analysis is a commonly used systematic clinical decisions, recent mechanistic suggestions may
reviewing strategy for addressing health-related sci- improve our ability to consume the large quantities of
entific research96 and involves a systematic statistical information.
explanation of available evidence in multiple studies.97 Brian Haynes suggests the use of the 5S model
Meta-analyses are used for public and health-care pol- for organization of evidence-based information103
icy decision making,98–102 and are especially helpful in (Figure 1.2 ■). The 5S model begins at the top of the
making decisions when a number of small studies hierarchy of evidence, and adds three additional steps
Systems
Summaries
Synopses (abstracts)
■ TABLE 1.4 Methodological Guidelines Outlined by the U.S. Clinical Practice Guidelines
for Acute Low Back Problems in Adults
Category Description
1. Strong evidence: Level A Includes interventions deemed either effective or ineffective with strong support in the literature as
determined by consistent findings/results in several high-quality randomized controlled trials or in at
least one meta-analysis.
2. Moderate evidence: Level B Includes interventions deemed either effective or ineffective with moderate support in the literature as
determined by consistent findings/results in one high-quality randomized controlled trial and one or
several low-quality randomized controlled trials.
3. Limited/contradictory evidence: Includes interventions with weak or conflicting support in the literature as determined by one
Level C randomized controlled trial (high or low quality), or inconsistent findings between several randomized
controlled trials.
4. No known evidence: Level D Includes interventions that have not been sufficiently studied in the literature in terms of
effectiveness and no randomized controlled trials in this area.
for information synthesis. The lowest levels (studies) that the stratification and scoring used in the study
reflect clinical trials and the syntheses reflect system- often affects the outcome of the analyses.105 Thus, dif-
atic literature reviews or meta-analyses. Synopses usu- ferent tools will provide different results and recom-
ally provide a summary of evidence from one or more mendations even during use of the same studies!
articles, and may include a clinical “bottom line.” Sum- Nonetheless, the use of a quality tool such as those
maries draw upon the syntheses of information to for- outlined by the U.S. Clinical Practice Guidelines for Acute
mulate the best evidence regarding the information. Low Back Problems in Adults106 provides a benchmark
Systems involve decision support services that match for performance in an attempt not to over support
information and tests and measures from a specific interventions that lack validity.
patient to best-evidence practices of diagnosis and The guidelines106 (Table 1.4 ■) reflect four primary
treatment.104 levels of evidence (Levels A, B, C, and D). Level A
includes findings from a number of high-quality RCTs
or at least one well-designed meta-analysis. Level B
includes information from only one high-quality RCT
Assigning Levels of Evidence or one or more low-quality RCTs. Level C outlines con-
flicting information from a number of studies. Level D
Methodological Guidelines
indicates that the intervention has not been properly
There are a number of methodological guidelines for investigated. This four-level quality measure is used
report of the quality of existing evidence. One chal- throughout the book to outline the evidence behind
lenge to using a methodological quality guideline is dedicated interventions.
32. Mohammadian P, Andersen OK, Arendt-Nielsen L. 49. Shamus J, Shamus E, Gugel R, Brucker B, Skaruppa C.
Correlation between local vascular and sensory The effect of sesamoid mobilization, flexor hallucis
changes following tissue inflammation induced by strengthening, and gait training on reducing pain and
repetitive application of topical capsaicin. Brain Res. restoring function in individuals with hallux limitus: A
1998;792(1):1–9. clinical trial. J Orthop Sports Phys Ther. 2004;34:368–376.
33. Bialosky JE, George SZ, Bishop MD. How spinal 50. Raftis K, Warfield C. Spinal manipulation for back pain.
manipulative therapy works: Why ask why? J Orthop Hosp Pract. 1989;15:89–90.
Sports Phys Ther. 2008;38(6):293–295. 51. Denslow JS. Analyzing the osteopathic lesion. 1940.
34. Glover J, Morris J, Khosla T. Back pain: A randomized J Am Osteopath Assoc. 2001;101(2):99–100.
clinical trial of rotational manipulation of the trunk. 52. Sran MM. To treat or not to treat: New evidence for the
Br J Physiol. 1947;150:18–22. effectiveness of manual therapy. Br J Sports Med.
35. Terrett AC, Vernon H. Manipulation and pain toler- 2004;38(5):521–525.
ance: A controlled study of the effect of spinal manip- 53. McLain R, Pickar J. Mechanoreceptor ending in human
ulation on paraspinal cutaneous pain tolerance levels. thoracic and lumbar facet joints. Spine. 1998;23:168–173.
Am J Phys Med.1984;63(5):217–225. 54. Sakamoto N, Yamashita T, Takebayashi T, Sekine M,
36. Vernon H, Dhami M, Howley T, Annett R. Spinal Ishii S. An electrophysiologic study of mechanorecep-
manipulation and beta-endorphin: A controlled study tors in the sacroiliac joint and adjacent tissues. Spine.
of the effect of a spinal manipulation on plasma beta- 2001;26:468–471.
endorphin levels in normal males. J Manipulative Phys- 55. Korr IM. Proprioceptors and somatic dysfunction.
iol Ther. 1986;9:115–123. J Amer Osteopath Assoc. 1975;74:638–650.
37. Wright A, Thurnwald P, Smith J. An evaluation of 56. Zusman M. Spinal manipulative therapy: Review of
mechanical and thermal hyperalgesia in patients with some proposed mechanisms and a hew hypothesis.
lateral epicondylalgia. Pain Clin. 1992;5:199–282. Australian J Physio 1986;32:89–99.
38. Wright A, Thurbwald P, O’Callaghan J. Hyperalgesia in 57. Sung P, Kang YM, Pickar J. Effect of spinal manipula-
tennis elbow patients. J Musculoskel Pain. 1994;2:83–89. tion duration on low threshold mechanoreceptors in
39. Zusman M. Mechanisms of musculoskeletal physio- lumbar paraspinal muscles. Spine. 2004;30:115–122.
therapy. Phys Ther Rev. 2004;9:39–49. 58. Keller T, Collaca C, Guzburg R. Neuromechanical
40. Sterling M, Jull G, Wright A. Cervical mobilization: characterization of in vivo lumbar spinal manipula-
Concurrent effects on pain, sympathetic nervous sys- tion. Part 1. Vertebral motion. J Manipulative Physiol
tem activity and motor activity. Man Ther. 2001;6:72–81. Ther. 2003;26:567–578.
41. Shacklock M. Neural mobilization: A systematic review 59. Dishman J, Bulbulian R. Spinal reflex attenuation
of randomized controlled trials with an analysis of ther- associated with spinal manipulation. Spine. 2000;
apeutic efficacy. J Man Manip Ther. 2008;16(1):23–24. 25:2519–2525.
42. Simon R, Vicenzino B, Wright A. The influence of an 60. Murphy B, Dawson N, Slack J. Sacroiliac joint manip-
anteroposterior accessory glide of the glenohumeral joint ulation decreases the H-reflex. Electromyog Clin Neuro-
on measures of peripheral sympathetic nervous system physiol. 1995;35:87–94.
function in the upper limb. Man Ther. 1997;2(1):18–23. 61. Sizer PS, Matthijs O, Phelps V. Influence of age on the
43. Wright A. Hypoalgesia post-manipulative therapy: A development of pathology. Curr Rev Pain 2000;4:362–373.
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14 Chapter 1: Orthopedic Manual Therapy
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at: http://www.cebm.net/
Chapter
Orthopaedic Manual
Therapy Assessment
Chad E. Cook 2
Objectives
• Compare and contrast the clinical decision-making models.
• Compare and contrast selected manual therapy backgrounds and their assessment philosophies.
• Determine if any of the philosophical elements of manual therapy are best supported by scientific evidence.
• Describe the purposes, types, and necessities of a manual therapy diagnosis.
• Describe the strengths and weaknesses of the patient response model.
15
16 Chapter 2: Orthopaedic Manual Therapy Assessment
Model Description
Hypothetical-deductive Hypothetical-deductive decision making involves the development of a hypothesis during the clinical
examination, and the refuting or acceptance of that hypothesis that occurs during the process of the
examination.
Heuristic Heuristic decision making involves pattern recognition and the ability to lump useful findings into coherent
groups.
Mixed The mixed model involves decision-making elements of hypothetical-deductive, heuristic, and
pathognomonic.
after accumulation and processing clinical findings the use of selected testing methods specifically
and confirming or refuting preexisting hypotheses. designed to determine if the patient would benefit
The process is considered a bottom-up approach, as it from additional medical consultation.
allows any pertinent finding to be a qualifier during The most routine situation in which a pathogno-
the decision-making process. monic diagnosis is used is during assessment of comor-
A benefit of the hypothetical-deductive model is bidities, which may contribute or potentially harm a
that most examination processes tend to be compre- patient’s recovery and/or function. Comorbidities such
hensive, focused, and quite extensive. Pertinent find- as high blood pressure, arthritis, or depression are com-
ings are rarely unexplored. Clinicians are allowed to monly encountered in practice,5,6 whereas other disor-
address a number of potential options because the data ders such as a neurological illness, fracture, or neoplasm
captured during the examination are extensive and are less common, but represent comorbidities or “red
detailed. flags” that are potentially threatening to the patient.
There are a number of challenges with this partic- Red flags are signs and symptoms that may tie a disor-
ular model. For starters, the model assumes that all der to a serious pathology.4,7 When combinations or sin-
findings are essential and can equally impact the gular representations of selected red flag features are
hypothesis exploration. In truth, a number of clinical encountered during an examination, a clinician may
findings do not provide useful information toward improve his or her ability to assess the risk of a serious
diagnosis or patient care management. In addition, underlying pathology.8 Differential assessment for red
although the hypothetical-deductive model is a logi- flags in individual patients by a clinician involves the
cal model and can be used in a number of noncom- use of special tests or standardized examinations in order
plicated circumstances, the process is less efficient for to identify individuals needing special intervention. A
experienced clinicians and may lead to a significant recent study demonstrated that < 5% of primary care
investment in refuting or confirming irrelevant physicians routinely examine for red flags during an
hypotheses. initial screening,9 whereas physical therapists document
An element of the hypothetical-deductive model is and screen at a much higher rate (>60%).10
the pathognomonic diagnosis. A pathognomonic diag-
nosis involves a decision based on a sign or symptom
Heuristic Decision Making (Clinical Gestalt)
that is so characteristic of a disease or an outcome that
the decision is made on the spot. In essence, a patho- Heuristic decision making, or clinical gestalt, is a
gnomonic diagnosis is an immediate “threshold”-level process that assumes health-care practitioners actively
finding prompting immediate action such as referral organize clinical perceptions into coherent construct
out or further testing. It is predicated on the assump- wholes. This implies that clinicians have the ability to
tion that all conditions are diagnosable in nature. indirectly make clinical decisions in absence of com-
The pathognomonic diagnosis is a constituent of plete information and can generate solutions that are
the history-taking, database analysis (patient intake characterized by generalizations that allow transfer
forms), physical examination, and monitoring of the from one problem to the next. In essence, clinical
patient’s condition during follow-up.4 A pathogno- gestalt is pattern recognition and is characterized as a
monic diagnosis is generally considered for use in the heuristic approach to decision making.11 At present,
early stages of an examination but can be used at any the literature suggests that experience does positively
time, specifically if new findings are present. Discern- influence decision-making accuracy, as experienced
ing the meaning of each of the findings may warrant clinicians have better pattern recognition skills.11
Assessment Modifiers of Decision Making 17
There are a number of benefits associated with also useful in capturing the true value of a pathogno-
heurism or gestalt. The method allows a quick global monic test finding. Because of the strengths and weak-
interpretation within seconds of data collection.12 This nesses of each area, it is advised that the manual
process is considered “top down,” that is, clinicians therapy clinician use a mixed model during decision
organize data in a manner that creates the most coher- making. A careful clinician always recognizes the
ent, seamless perception possible.13 Seasoned clinicians weaknesses of each model and compensates by using
often advocate the usefulness of heuristic decision the strengths of the other.
making. Arguably, without a working knowledge of
gestalt principles, clinicians would be hopelessly
bogged down with “bottom-up” assessments (hypo- Summary
thetical-deductive) of their patients, begrudgingly
• A threshold approach is the accumulation of information to a spec-
plowing through reams of clinical data to form a work- ified level that triggers a clinician to make a decision.
able hypothesis. Yet despite the utility of clinical
• There are two primary clinical decision-making models in man-
gestalt, it is important to realize that this method is not
ual therapy: (1) hypothetical-deductive and (2) heuristic decision
without error. For example, at present, most health- making.
care providers use tools for decision making that
• Hypothetical-deductive decision making involves the development
demonstrate only marginal value.14 Most clinicians
of a hypothesis during the clinical examination and the refuting or
also make errors in diagnosis when faced with com- acceptance of that hypothesis that occurs during the process of
plex and even noncomplex cases14 and up to 35% of the examination.
these errors can cause harm to patients.15
• Heuristic decision making involves pattern recognition and the
Although intuitive, heuristic decision making is rid- ability to collate useful findings into coherent groups.
dled with five tangible errors16: (1) the representative
• Most true models are mixed models of decision making.
heuristic (if it’s similar to something else, it must be
like that); (2) the availability heuristic (we are more
inclined to find something if it’s something we are
used to finding); (3) the confirmatory bias (looking for Assessment Modifiers
things in the exam to substantiate what we want to of Decision Making
find); (4) the illusory correlation (linking events when
there is actually no relationship); and (5) overconfidence. Assessment modifiers are elements that are used in
Of these five decision-making errors, overconfidence any of the three decision-making models that are
may be the most predominant. Most diagnosticians designed to improve the accuracy of the outcome.
feel that they are better decision-makers than what Assessment modifiers are not decision-making mod-
they demonstrate in actual clinical practice.15 In fact, els, because each does not contain a unique decision-
the least skilled diagnosticians are also the most over- making characteristic and is not exclusive within one
confident and most likely to make a mistake!15 particular model. Modifiers do affect decision-making
These mistakes can occur in two domains: (1) the uniquely when evaluated under specific circum-
empirical aspect (real-world observation of findings, stances. Examples of assessment modifiers include
or the data collection phase) and (2) the rational aspect probabilistic statistics and clinical prediction rules.
(the clinical decision-making phase during which clin- Within epidemiological literature, the two most
icians make sense of the data at hand).17 Although both commonly discussed assessment modifiers are
are common, the reasoning (rational) aspect is by far moderators and mediators. While referred to as
the most common.17 “assessment modifiers” in this textbook, modera-
tors and mediators are actually outcomes modifiers,
which can influence the outcome of the assessment
Mixed Model Decision Making
or intervention.
In reality, most decision-making models are mixed. Moderators and mediators are useful in explaining
For examination, most clinicians use a hypothetical- why some patients change and others do not. Both
deductive approach to identify variables that support involve dedicated variables that influence the causal-
a pre-examination hypothesis. During the develop- ity link between treatment and outcome.18 Both are
ment of most clinical prediction rules (CPR), gestalt essential when considering the true effect of an inter-
has been a driver in capturing most variables for study. vention during an assessment of therapeutic outcome.
Using assessment modifiers such as probabilistic Failure to consider moderators and mediators during
decision-making analyses, one can assign predictive observational or randomized controlled designs may
values to pertinent findings captured during a hypo- lead to an overestimation or underestimation of the
thetical-deductive approach. Probabilistic tools are effect magnitude.
18 Chapter 2: Orthopaedic Manual Therapy Assessment
An outcome mediator functions to partially iden- directionality of moderation and mediation. Modera-
tify the possible mechanisms through which a treat- tors clarify the magnitude of treatment effects through
ment might achieve its effects. These mechanisms are adjustment of estimates for imbalances in the group
causal links between treatment and outcome19 in with respect to covariables.20 Mediators establish ‘how’
which the treatment affects the mediator and the con- or ‘why’ one variable predicts or causes an outcome
sequence of the mediator (after exposure to the treat- variable.19,21 Consider an example using graded expo-
ment) affects the targeted outcome. Mediators can sure for low back and the effects of fear-avoidance
positively or negatively affect an outcome by virtue of beliefs on low back pain perceived disability.22 One
a “change” in the variable during the time frame of could argue that if stratified, fear avoidance could dic-
the intervention. To qualify as an outcome mediator, tate change scores for specific outcomes. Higher fear-
the variable of interest must (1) change during expo- avoidance scores should relate to lower outcomes,
sure to treatment, (2) be correlated with the treatment, moderate fear avoidance scores should relate to
and (3) explain all or a portion of the effect of the treat- slightly better outcomes, and very low fear-avoidance
ment on the desired outcome measure. scores should relate to excellent outcomes. If this is the
In contrast, an outcome moderator is a baseline case then fear-avoidance strata could be considered a
variable that (1) precedes the treatment temporally, moderator. Yet prior to determining whether fear
(2) is independent of the treatment (is not affected by avoidance should function as a moderator or media-
the treatment), but (3) influences the outcome (e.g., tor, one must examine the affect of the treatment on
Oswestry or Short Form 36) when stratified by selected the moderator or mediator. Graded exposure is
values. Potential moderators may be sociodemo- hypothesized to positively improve fear avoidance
graphic variables, genotype, or baseline clinical char- behavior, which in turn should improve overall out-
acteristics (comorbidities) that are not influenced by comes.23 When a treatment influences the variable, the
active treatment mechanisms. variable must be considered a mediator. Figure 2.1 ■
It is somewhat challenging to differentiate moder- provides a graphic example of how moderators and
ators from mediators when evaluating outcomes of a mediators influence outcomes.
clinical trial, when designing a study, or when con- Why is this information useful? Moderators, when
sidering the appropriate treatment for clinical care. As stratified, assist in defining which groups are likely to
discussed by Kraemer and colleagues,19 there is ambi- benefit from a particular form of manual therapy. For
guity between a moderator and a mediator and in the example, the moderators associated with the clinical
High
1 (FAB/Q15)
2 (FAB/Q 15-30)
Low
3 (FAB/Q30)
(c) Moderator strata (a) Treatment (b) Outcome
Example of Fear Avoidance Beliefs Questionnaire (FABQ) strata as a moderator
variable; treatment moist hot pack application 4 weeks. represents
change over time.
High
Low
prediction rule for manipulation of the thoracic spine Although modifiers such as these have shown
in patients with mechanical neck pain are used to pre- improved outcomes over gestalt methods, it is worth
dict who is likely to improve from a specific interven- noting that CPRs are only as good as the derivation/
tion. In contrast, findings such as high fear avoidance validation sequence of the study.26,27 At present, there
behaviors, poor coping strategies, low self-efficacy, are a number of CPRs that have been published in the lit-
and depression are considered mediating variables. erature that are reflective of a manual therapy proce-
These are considered mediators if a specific exercise dure, and the majority has exceptionally weak
approach changes the finding of the mediator (e.g., if methodology.27,28 Although the development of a CPR
graded exposure reduces the fear-avoidance behav- is recommended to improve outcomes, a careful and
iors). This information can be used to target specific iterative process is necessary to assure clinicians we are
interventions that can affect the mediators and subse- using the proper predictive tools. In addition, CPRs are
quently improve the outcome. not a substitute for good clinical decision making; CPRs
are a “process modifier” within the clinical decision-
making sequence.
Probabilistic Decision Making
Health-care providers always make decisions in the Patient Response Triggers
face of uncertainty.24 Probabilistic modification is the A patient response trigger is a finding within an
use of a probability estimate for determining diagno- examination that facilitates a dedicated care response,
sis, prognosis, or treatment of a patient and is a form expectation of prognosis, or diagnosis, and is a form of
of moderator. Probabilistic modification is an induc- mediator. Patient response triggers differ from patho-
tive method that uses a statistically oriented overview gnomonic findings because these are generally not
to determine decisions. This method of modification solely associated with a negative finding. Typically,
uses given information from the dedicated circum- patient response triggers are gathered during a phys-
stance of the patient, and specific statistical laws asso- ical examination that consists of pain reproduction and
ciated with probability, to determine the occurrence of reduction (using pain provocation and reduction
the event that is expected with high logical or induc- methods). During this examination, various move-
tive probability.25 This modification suppresses many ments are found that alter patient report of symptoms
of the errors associated with heuristic decision making (either improving or worsening findings). The process
by allowing the strength of a giving decision-making assumes that the clinical findings are relevant toward
instrument (i.e., clinical tool) to dictate outcomes, ver- the outcome of the patient and ties each treatment
sus internal judgment. intervention that may be unique to each particular
Bayesian assessment is considered a form of prob- patient.
abilistic modification. Bayesian assessment is some- Germane to the patient response trigger is the
times referred to as “knowledge-based decision expectation that the patient response triggers are pre-
making” and is predicated on prior estimates of prob- dictive of within-session (during the same session)29
abilities, based on additional experience, and influ- or between-session (after the patient returns)30
enced by additive information. In essence, a prior changes during the care of that patient. Many clini-
estimate of a condition is fixed, and a finite set of revi- cians use the within- and between-session changes to
sionist tests and measures are performed that revise adjust their treatment dosage, intensity, and applica-
the initial probability estimate. Appropriate tests and tion for the optimal targeted result advocating pri-
measures significantly modify the probability estimate. marily the use of within-session (immediate response)
Although probability modifications provide com- changes toward a positive long-term outcome. In
plexity to hypothetical-deductive and heuristic deci-
truth, within- and between-session changes have been
sion making, the process can be simplified through the
shown to be useful in predicting a positive outcome
use of clinical prediction rules or decision rules. Clin-
for acute lumbar spine pain, impairments, and neck
ical prediction rules capture selected variables that
pain. Surprisingly, there is little research on between-
have demonstrated the ability to influence the post-
session changes, although it is arguably the more
test probability of a positive (or a negative) change in
important of the two.
a patient’s condition. Most are obtained by capturing
conditionally independent measures that are associ-
Classification or Clusters
ated toward an outcome. A high-quality clinical pre-
diction rule allows clinicians to improve their Classification is a mechanism of labeling or placing a
probability of success with a diagnosis or an inter- patient into a known group to target a preconceived,
vention when patient characteristics and examination directed clinical approach to that patient. The process
findings match the identified rules. uses both probabilistic and heuristic influences, but
20 Chapter 2: Orthopaedic Manual Therapy Assessment
also dwells heavily on pre-existing prognostic litera- adjustments toward emphasis on each category based
ture toward what benefits that patient. In most cases, on changes in findings or the patient’s condition.
classifications are developed through regression (sta- Consider the following examples. Suppose a patient
tistical) modeling, which captures subgroups of seen for general low back pain provides encouraging
patients who benefit from a dedicated procedure. outcomes after administration of a lumbar manipula-
Treatment by classification has demonstrated a bet- tive technique on the first visit. The manipulation tech-
ter outcome than independent clinician decision mak- nique was selected secondary to the patient’s
ing and as a whole provides a fairly well-vetted classification as a candidate for passive movement
mechanism for general treatment of patients. Com- (assessment modifier) and a positive response during
bining hypothetical-deductive, patient-response find- mobilization during the examination (hypothetical-
ings within a classification allows a specified approach deductive/assessment modifier–patient response).
to target the most effective treatment to the patient. In The patient’s report of pain and range of motion are
Chapter 4, we discuss the use of general versus specific markedly improved (assessment modifier, between-
techniques. Specific techniques are those gathered session change) yet the patient still reports a low level
during combined hypothetical-deductive, patient- of function and fear in returning to work. Heuristic
response findings such as “pain during closing” or influences (pattern recognition) would suggest an
“pain during dorsiflexion.” active approach may benefit this patient versus a
purely passive approach. In addition, on the third visit,
the patient reports bladder retention, bilateral leg pain,
Summary and rapid neurological changes34 (pathognomonic
• Probabilistic modification is an inductive method that uses a sta- findings) prompting the clinician to immediately refer
tistically oriented overview to determine decisions and can be the patient for diagnostic work-up. The scenario
used to enhance or modify any decision-making model. describes a mixed model where assessment modifiers
• A patient response trigger is a finding within an examination that alter the clinical decision-making process. The process
facilitates a dedicated care response, expectation of prognosis, or as a “whole” is clinical reasoning.
diagnosis.
• Classification is the process of labeling or placing a patient into a
known group to target a preconceived, directed clinical approach Summary
to that patient.
• Clinical reasoning is a thinking process to direct a clinician to
take “wise” action or to take the best judged action in a specific
context.
Clinical Reasoning • Clinical reasoning is the overarching act of appropriate clinical
decision making and may involve any of the models of clinical
Clinical reasoning is a thinking process to direct a cli- decision making.
nician to take “wise” action or to take the best judged
action in a specific context.31 It is a process in which
the therapist, who interacts with the patient and other
appropriate parties, helps patients develop health Decision Making in Manual
management strategies for their condition based on Therapy
the unique findings for that patient and the patient’s
own response to their condition.32 Clinical reasoning There are significant philosophical variations in deci-
is the overarching element of clinical decision making sion making between practicing manual clinicians.
and involves processes beyond diagnosis and inter- The philosophical variations robustly harbor influen-
vention, such as assessment and management during tial internal biases that affect decision making in man-
continued care. ual therapy. Components of one’s philosophy are
As stated previously, most clinicians use a mixed likely affected by a number of factors, including the
model for decision making, which combines the ele- overwhelming amount of information that is present
ments of heurism and hypothetical-deductive think- in the literature and the difficulty as a clinician in
ing. The ability to partition, combine, or modify the maintaining current understanding of that knowl-
decision-making components of each is clinical rea- edge. Clinicians often use mechanisms such as con-
soning. This requires a patient-centered approach that tinuing education programs, colleagues, or textbooks
is grounded in a biopsychosocial framework,33 to gather “new” material, all of which have potential
requires storing of findings that are based either on weaknesses.
pattern recognition, probabilistic importance, or that The most dominant mechanism is one’s back-
have influenced within-session findings, and allows ground exposure (or experience) toward a specific
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DANCE ON STILTS AT THE GIRLS’ UNYAGO, NIUCHI
I see increasing reason to believe that the view formed some time
back as to the origin of the Makonde bush is the correct one. I have
no doubt that it is not a natural product, but the result of human
occupation. Those parts of the high country where man—as a very
slight amount of practice enables the eye to perceive at once—has not
yet penetrated with axe and hoe, are still occupied by a splendid
timber forest quite able to sustain a comparison with our mixed
forests in Germany. But wherever man has once built his hut or tilled
his field, this horrible bush springs up. Every phase of this process
may be seen in the course of a couple of hours’ walk along the main
road. From the bush to right or left, one hears the sound of the axe—
not from one spot only, but from several directions at once. A few
steps further on, we can see what is taking place. The brush has been
cut down and piled up in heaps to the height of a yard or more,
between which the trunks of the large trees stand up like the last
pillars of a magnificent ruined building. These, too, present a
melancholy spectacle: the destructive Makonde have ringed them—
cut a broad strip of bark all round to ensure their dying off—and also
piled up pyramids of brush round them. Father and son, mother and
son-in-law, are chopping away perseveringly in the background—too
busy, almost, to look round at the white stranger, who usually excites
so much interest. If you pass by the same place a week later, the piles
of brushwood have disappeared and a thick layer of ashes has taken
the place of the green forest. The large trees stretch their
smouldering trunks and branches in dumb accusation to heaven—if
they have not already fallen and been more or less reduced to ashes,
perhaps only showing as a white stripe on the dark ground.
This work of destruction is carried out by the Makonde alike on the
virgin forest and on the bush which has sprung up on sites already
cultivated and deserted. In the second case they are saved the trouble
of burning the large trees, these being entirely absent in the
secondary bush.
After burning this piece of forest ground and loosening it with the
hoe, the native sows his corn and plants his vegetables. All over the
country, he goes in for bed-culture, which requires, and, in fact,
receives, the most careful attention. Weeds are nowhere tolerated in
the south of German East Africa. The crops may fail on the plains,
where droughts are frequent, but never on the plateau with its
abundant rains and heavy dews. Its fortunate inhabitants even have
the satisfaction of seeing the proud Wayao and Wamakua working
for them as labourers, driven by hunger to serve where they were
accustomed to rule.
But the light, sandy soil is soon exhausted, and would yield no
harvest the second year if cultivated twice running. This fact has
been familiar to the native for ages; consequently he provides in
time, and, while his crop is growing, prepares the next plot with axe
and firebrand. Next year he plants this with his various crops and
lets the first piece lie fallow. For a short time it remains waste and
desolate; then nature steps in to repair the destruction wrought by
man; a thousand new growths spring out of the exhausted soil, and
even the old stumps put forth fresh shoots. Next year the new growth
is up to one’s knees, and in a few years more it is that terrible,
impenetrable bush, which maintains its position till the black
occupier of the land has made the round of all the available sites and
come back to his starting point.
The Makonde are, body and soul, so to speak, one with this bush.
According to my Yao informants, indeed, their name means nothing
else but “bush people.” Their own tradition says that they have been
settled up here for a very long time, but to my surprise they laid great
stress on an original immigration. Their old homes were in the
south-east, near Mikindani and the mouth of the Rovuma, whence
their peaceful forefathers were driven by the continual raids of the
Sakalavas from Madagascar and the warlike Shirazis[47] of the coast,
to take refuge on the almost inaccessible plateau. I have studied
African ethnology for twenty years, but the fact that changes of
population in this apparently quiet and peaceable corner of the earth
could have been occasioned by outside enterprises taking place on
the high seas, was completely new to me. It is, no doubt, however,
correct.
The charming tribal legend of the Makonde—besides informing us
of other interesting matters—explains why they have to live in the
thickest of the bush and a long way from the edge of the plateau,
instead of making their permanent homes beside the purling brooks
and springs of the low country.
“The place where the tribe originated is Mahuta, on the southern
side of the plateau towards the Rovuma, where of old time there was
nothing but thick bush. Out of this bush came a man who never
washed himself or shaved his head, and who ate and drank but little.
He went out and made a human figure from the wood of a tree
growing in the open country, which he took home to his abode in the
bush and there set it upright. In the night this image came to life and
was a woman. The man and woman went down together to the
Rovuma to wash themselves. Here the woman gave birth to a still-
born child. They left that place and passed over the high land into the
valley of the Mbemkuru, where the woman had another child, which
was also born dead. Then they returned to the high bush country of
Mahuta, where the third child was born, which lived and grew up. In
course of time, the couple had many more children, and called
themselves Wamatanda. These were the ancestral stock of the
Makonde, also called Wamakonde,[48] i.e., aborigines. Their
forefather, the man from the bush, gave his children the command to
bury their dead upright, in memory of the mother of their race who
was cut out of wood and awoke to life when standing upright. He also
warned them against settling in the valleys and near large streams,
for sickness and death dwelt there. They were to make it a rule to
have their huts at least an hour’s walk from the nearest watering-
place; then their children would thrive and escape illness.”
The explanation of the name Makonde given by my informants is
somewhat different from that contained in the above legend, which I
extract from a little book (small, but packed with information), by
Pater Adams, entitled Lindi und sein Hinterland. Otherwise, my
results agree exactly with the statements of the legend. Washing?
Hapana—there is no such thing. Why should they do so? As it is, the
supply of water scarcely suffices for cooking and drinking; other
people do not wash, so why should the Makonde distinguish himself
by such needless eccentricity? As for shaving the head, the short,
woolly crop scarcely needs it,[49] so the second ancestral precept is
likewise easy enough to follow. Beyond this, however, there is
nothing ridiculous in the ancestor’s advice. I have obtained from
various local artists a fairly large number of figures carved in wood,
ranging from fifteen to twenty-three inches in height, and
representing women belonging to the great group of the Mavia,
Makonde, and Matambwe tribes. The carving is remarkably well
done and renders the female type with great accuracy, especially the
keloid ornamentation, to be described later on. As to the object and
meaning of their works the sculptors either could or (more probably)
would tell me nothing, and I was forced to content myself with the
scanty information vouchsafed by one man, who said that the figures
were merely intended to represent the nembo—the artificial
deformations of pelele, ear-discs, and keloids. The legend recorded
by Pater Adams places these figures in a new light. They must surely
be more than mere dolls; and we may even venture to assume that
they are—though the majority of present-day Makonde are probably
unaware of the fact—representations of the tribal ancestress.
The references in the legend to the descent from Mahuta to the
Rovuma, and to a journey across the highlands into the Mbekuru
valley, undoubtedly indicate the previous history of the tribe, the
travels of the ancestral pair typifying the migrations of their
descendants. The descent to the neighbouring Rovuma valley, with
its extraordinary fertility and great abundance of game, is intelligible
at a glance—but the crossing of the Lukuledi depression, the ascent
to the Rondo Plateau and the descent to the Mbemkuru, also lie
within the bounds of probability, for all these districts have exactly
the same character as the extreme south. Now, however, comes a
point of especial interest for our bacteriological age. The primitive
Makonde did not enjoy their lives in the marshy river-valleys.
Disease raged among them, and many died. It was only after they
had returned to their original home near Mahuta, that the health
conditions of these people improved. We are very apt to think of the
African as a stupid person whose ignorance of nature is only equalled
by his fear of it, and who looks on all mishaps as caused by evil
spirits and malignant natural powers. It is much more correct to
assume in this case that the people very early learnt to distinguish
districts infested with malaria from those where it is absent.
This knowledge is crystallized in the
ancestral warning against settling in the
valleys and near the great waters, the
dwelling-places of disease and death. At the
same time, for security against the hostile
Mavia south of the Rovuma, it was enacted
that every settlement must be not less than a
certain distance from the southern edge of the
plateau. Such in fact is their mode of life at the
present day. It is not such a bad one, and
certainly they are both safer and more
comfortable than the Makua, the recent
intruders from the south, who have made USUAL METHOD OF
good their footing on the western edge of the CLOSING HUT-DOOR
plateau, extending over a fairly wide belt of
country. Neither Makua nor Makonde show in their dwellings
anything of the size and comeliness of the Yao houses in the plain,
especially at Masasi, Chingulungulu and Zuza’s. Jumbe Chauro, a
Makonde hamlet not far from Newala, on the road to Mahuta, is the
most important settlement of the tribe I have yet seen, and has fairly
spacious huts. But how slovenly is their construction compared with
the palatial residences of the elephant-hunters living in the plain.
The roofs are still more untidy than in the general run of huts during
the dry season, the walls show here and there the scanty beginnings
or the lamentable remains of the mud plastering, and the interior is a
veritable dog-kennel; dirt, dust and disorder everywhere. A few huts
only show any attempt at division into rooms, and this consists
merely of very roughly-made bamboo partitions. In one point alone
have I noticed any indication of progress—in the method of fastening
the door. Houses all over the south are secured in a simple but
ingenious manner. The door consists of a set of stout pieces of wood
or bamboo, tied with bark-string to two cross-pieces, and moving in
two grooves round one of the door-posts, so as to open inwards. If
the owner wishes to leave home, he takes two logs as thick as a man’s
upper arm and about a yard long. One of these is placed obliquely
against the middle of the door from the inside, so as to form an angle
of from 60° to 75° with the ground. He then places the second piece
horizontally across the first, pressing it downward with all his might.
It is kept in place by two strong posts planted in the ground a few
inches inside the door. This fastening is absolutely safe, but of course
cannot be applied to both doors at once, otherwise how could the
owner leave or enter his house? I have not yet succeeded in finding
out how the back door is fastened.