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PHYSIO MMURMMURRS

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ejection
inp Diastasis Eagle ejection

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MURMUR PHONOCARDIOGRAM

81 Split
d
valveclosing
signifantly
before the
Pit
canbephysiologic
or
mneme
isexaggeratedphysiologically
thiswhen
expiration
i nspiration
duringinspiration layingsupine that
wide delayedemptyingortheright

5
expirationinspiration
exaggeratednorma ventricle eg rBBBor
splitting pulmonic stenosis it

Fixed novariationinsplit delayedclosingorpulmonary


valve partonshunt
expirationinspiration on mpen Mp
splitting duringexpirationinspiration createdin Asp tittt
Paradoxical splittingduringexpiration closingofpulmonaryvalve
before expirationinspiration

Butnotinspiration theaorticvalve delayedventricular at m


splitting mm
heard beginingormidalethirao
diastole
canbephysiologic in
childrenadolescents youngadults
53 M Mm
h rorthfigqntraicfs.am
altoscilitationorbloodback mm
over inaauitscgensys.gg heartfailure

heardinptswho need
atrialcontractiontocompensate Olderpatients
gy for aventricularwallcompliance
resistopengtovenmanar w ventricularHypertrophy
Mm M TMT
mostcommonheartvalvedisease

Aortic ouamurmur
systole
autonomicerectduring usuallyelderlypts to
duetoaging calcification
ofvalve
NNNAhNM
Mh
Stenosis Rheumaticfever learn concentrichypertrophy
myocardialischemia

resurge aventricular
aortic
bloodfictitidiitaratrom diastolicpressurewhich
Aortic thenignpressure aortaintothe
nextventricle
compresses
muscle
theinnerlayerorthe
giggarincoronary Mn mummmmmunammut
Regurgitation aorticdiastolicpressure
myocardialischemia

back'Eatatrointo'the
stfu
Ming duringsystole
high lowing
mild mitralregurgitation
associatedw mitralvalve
prolapse mummmmmun immunumanm

Regurgitation
apicmairmansystolic
pint valveprolapseheardasmid
mitral
systolicclick
bestheardatapexor
mitralregurg pulmonaryedema

bloodhasaimicuitypassing
fromtheleftatriumtotheleft virtuallyonlycauseof
Mitra ventricle
usuallyweakmurmur
mostlybelowthefrequency
mitralstenosis Mm mhm immmmmmmunffy

Stenosis thathumannearing
any mitralstenosis pulmonaryedema
aribiniatestages
Acute
INFLECTIVE LENNDOCCAARRDDITIS
rapidonset
days
mostcommon 2 Mostcommoncauseofdeath
is.io mostcommoncause
toweeks

slowprogression BUG CLINICAL


weekstomonths
Dt
ETIOLOGY
PRESENTATION T TEST TREATMENT
ptsthatrecently subacute ofsubacuteI.E Blood
n cultures or
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that
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catalase b hygienesteriletechniques

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ENT
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orgenitourinary d
pm names MildFever ampicillin gentamicin
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new orworsening
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em Transmission weightloss resistant


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Linezoid

the valvesartected mannitol


saltagar fatal intreated
weeks if not
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piddgiefiff.im
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musthave
airferentblood native war pennies raging
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waricinin
aureus Reggggitispouman invasiveprocedures
underlying disease
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Respiratorydroplets Mrs
endogenous native
nm valve
vancomycin

vancomycin
Grant or.IE common coagulase Piatti rifampicin gentamicin

CATALASE pts w peripheral SubacuteInfective forptswcause


prosthetic
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catheters

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underlying disease
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respiratorydroplets
endogenous
mm

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lesions
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membranes rormspores
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innibitsmansomicolase
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injury platelets Boundingpulse stiffs'tfitttistifis
fibrinaccumulate Traubesign giggynne capsorungin
vegetarians micarungin
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aniaularungin
valveanected complications popinnibitingtranspenaasemoniaggig.ge
crosslines p eptidoglycan agyeai monirreversibly raise
inhibits
immunecomplexglomerulonephritis
by I e mitral mycoticaneurysm
chains
ProtennsummeasonBOORS stucanagggggendearn

septicemboli abscess aqgm.sn 50s


aneroid
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I
moninnibitsianosterointa
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