Professional Documents
Culture Documents
Management
1
Contents
• Introduction
• Colour and colour Perception
• Classification and Etiology of discoloration
• Diagnosis
• Conservative management
• Conclusion
• References
2
Introduction
3
Colour and Colour Perception
Colour of healthy teeth is determined by:
⮚ The colour of enamel
⮚ The translucency of enamel
⮚ Thickness of enamel – Greater: Occlusal/Incisal edge
- Thinner : Cervical third
Normal colour:
⮚ Primary teeth: bluish white
⮚ Permanent teeth: grayish yellow, grayish white or yellowish white
⮚ Elderly persons: more yellow or grayish yellow
•Intrinsic
•Combination
5
Internalized discolouration(Vimal K Sikri)
• Enamel defects and in porous surface of exposed dentin
• Developmental defects like Fluorosis, Hypoplasia, Enamel
calcification
• Tooth wear and gingival recession
6
Classification of extrinsic Discoloration –
I. According to cause -
• Direct Stain –
- compounds incorporated into the pellicle layer
- basic color of the chromogen
- dietary sources and substances habitually placed in the mouth
Heavy tobacco and coffee stains reddish black stains - habit of chewing pan
Manuel ST; Etiology of tooth discoloration- a review; Nig Dent J Vol 18 No. 2 July - Dec 2010 7
• Indirect stain:
- Cationic antiseptics – Chlorhexidine(brown to black),
- Cetylpyridinium chloride
- Benzalkonium chloride
Manuel ST; Etiology of tooth discoloration- a review; Nig Dent J Vol 18 No. 2 July - Dec 2010
8
II. According to presence of metals:
❑ Non-metallic Stains:
❖ Brown Stain:
- Thin,translucent, bacteria-free, pigmented pellicle
- Buccal surface of maxillary molars and on the lingual surface of the
mandibular incisors
- Deposition of tannin found in tea, coffee, and other beverages
❖ Tobacco stain:
- Tenacious dark-brown or black discoloration
- Cervical one- third to one-half of most teeth
- Deposition of coal tar products on the tooth surface
9
❖ Black stain:
- Facial and lingual surfaces of the teeth near the gingival margin
and diffuse patches on proximal surfaces
- Actinomyces species and Prevotella melaninogenica
- Stain - ferric sulfide - reaction between hydrogen sulfide
❖ Green Stain:
-Children (boys – 65% ; girls – 63%)
- Stained remnants of enamel cuticle
- Gingival half of maxillary anterior teeth
- Penicillium(fluorescent bacteria) and Aspergillus(fungi)
10
• Orange Stain (3%) -
- Serratia marcescens and Flavobacterium lutescens
-both facial and lingual surfaces of anterior teeth
11
❑ Metallic Stains:
❖ Black stain: iron, manganese, and silver
❖ Grayish stain : Mercury and lead dust
❖ Green to blue-green stain : copper and nickel
❖ Deep orange : Chromic acid fumes
❖ Black stain: Iron-containing drugs (in solution form)
❖ Brown stain: iodine solution and stannous fluoride
❖ Violet black: potassium permanganate mouthwash
❖ Greenish-black : mercury
❖ Golden brown: stannous fluoride
12
13
14
Intrinsic Stains
1. Pre-eruptive (during Odontogenesis)
a. Disease: Alkaptonuria
Haematological diseases
Liver disease
Disease of Enamel and Dentin
b. Medication: Tetracycline stains
Other Antibiotics use
Fluorosis stain
2. Post eruptive (after Odontogenesis)
a. Trauma e. Smoking
b. primary and secondary caries f. Chemicals
c. Dental restorative materials g. functional and parafunctional
d. Ageing h.Minocycline
15
:
Pre-eruptive (during Odontogenesis):
Haematological diseases
1. Erythroblastosis Fetalis
-Green, brown or bluish
-Cause: lysis of erythrocytes and incorporation
of hemosiderin pigment in forming dentin
2. Congenital Porphyria:
⮚ Red/brownish discoloration
⮚ Under ultraviolet light – red fluorescence
⮚ Cause- overproduction of Uroporphyrin
16
3. Sickle cell anemia:
⮚ Cause: increased hemolysis of red blood cells
⮚ Green, brown or bluish – more severe than erythroblastosis fetalis
17
• Liver diseases:
⮚ Bilirubinaemia:
-Liver dysfunction
- Bilirubin pigmentation in deciduous teeth
Alkaptonuria:
⮚ Cause: complete oxidation of Phenylalanine and Tyrosine causing
increased level of Homogentisic acid
⮚ Dark brown in primary teeth
-
18
Develpomental defects in Enamel formation:
1. Amelogenesis Imperfecta
3 types: Hypoplastic
Hypomineralization
Hypomaturation
⮚ Yellow to brownish
⮚ Snow capped appearance
19
2. Fluorosis:
Oral manifestations:
⮚ Mild: Gray or white opaque area on teeth
⮚ Yellow – brownish discolouration
⮚ Moderate – pitting and brownish discoloration
⮚ Severe – corroded with dark brown discoloration
3. Vitamin deficiency:
Vit D deficiency: white patch hypoplasia
Vit C and Vit A deficiency: pitting type
20
4. Enamel Hypoplasia:
General :
⮚ Amelogenesis imperfecta
⮚ Prenatal or congenital Syphilis
⮚ Endemic Fluorosis
⮚ Vit C deficiency
21
Defects in Dentin Formation:
1. Dentinogenesis imperfecta:
- Gray to brownish violet to yellowish brown
with a characteristic usual translucent or
opalescent hue
2. Dentin Dysplasia:
- slight amber translucency of teeth
22
Tetracycline stain:
Acc to Jordan and Boksman1984 -
23
Post eruptive:
Minocycline stains:
⮚ Chelates with iron to form insoluble complexes
with secondary dentin
⮚ Does not resolve after discontinuation
of the therapy
⮚ Milder than Tertracycline
⮚ Black
24
Pulpal changes: Pulpal necrosis
Pulpal haemorrhagre – grayish
- non-vital appearance
Calcific Metamorphosis – anteriors
- yellow-brown
25
Dental caries:
• Around areas of bacterial stagnation and leaking restorations
• Arrested caries- brown discoloration similar to that of pellicle
26
Restorative materials:
• Silver amalgam- corrosive changes and degradation
• Gold fillings – dark hues
• Composite resin- leak at margins
• Metal pins and pre fabricated posts – anteriors – visible underneath
composite
• Silver points- grey/pink appearance
27
Aging: Enamel changes – thinning and texture
Dentin deposition: secondary and tertiary
pulp stones
Salivary changes – salivary content and composition changes
(Solheim 1988)
28
Daily acquired stains:
• Plaque
• Tobacco use
• Food and beverages
• Poor oral hygiene
• Good oral hygiene – black type – children
• Swimmer’s calculus-
- yellow to dark brown stain
- facial/lingual/palatal surfaces of anteriors
Prolonged exposture to pool water (Rose and Carey 1995)
29
30
• Based on cause; Tooth discoloration usually occurs owing to
patient- or dentist-related causes.- [INGLE]
• Dental stains
• Dentist- related causes
• Endodonticallyrelated
• Pulp tissue remnantsIntracanalmedicamentsObturatingmaterials
• Restoration related
• Patient-related causes
• Pulp necrosis,Intrapulpalhaemorrhage, Dentinhypercalcification, Tooth formation
defects: -Developmental defects -Drug-related defects
31
Diagnosis of Discoloration
• Dental history - previous dental treatment, oral hygiene practices,
use of mouthwashes, amount and scheduling of fluoride intake,
history of dental trauma
• Medical history (history of maternal or childhood diseases, use of
medications
• Family history (genetic disorders)
• Diet history (nutritional deficiencies, diet that can cause staining of
the teeth
• Social history (occupational exposure to metals, use of tobacco)
32
Scratch Test: - This test differentiates between extrinsic and intrinsic stains
⮚ Armamentarium used is dental explorer, scalars, or any other sharp
instruments
⮚ An extrinsic stain can be removed by use of these instruments while
intrinsic cannot be removed
Usually, if the removal is difficult the stain is considered tenacious
33
Conservative Management
• Prevention
• Scaling
• Microabrasion
• Macroabrasion
• Bleaching
1. Extra- coronal Bleaching
2. Intra – coronal Bleaching
34
Prevention
Joiner, Whitening toothpastes: A review of the literature Jl of dentistry 38s (2010) e17-e2
36
Phosphate materials, such as A toothpaste containing 5% Hexa
pyrophosphate, tripolyphosphate meta phosphate as anion
and hexametaphosphate, tend to was shown in clinical studies to
have a strong binding significantly remove
affinity for enamel, dentine and chlorhexidine/tea induced stain
tartar, and during adsorption versus control toothpastes
they have been shown to desorb after 3 and 6 weeks product use
stain components.
Gerlach et al,2002
Shellies et al,2007
37
Microabrasion
• Conservative method for removing enamel to improve
discolorations limited to the outer enamel layer
• Sundfeld, et al (2007) - enamel microabrasion technique results in a
loss of enamel of around 25 to 200 μm, depending on the number
of applications and acids concentration
PREMA:
18%, 10% or 6.6 % 37%phosporicacid
hydrochloric acid gel and extrafine HCl +Silicon
and pumice pumice gel + Silicon
Carbide
+Silica gel
38
Sundfeld et al.; Microabrasion in tooth enamel discoloration defects: three cases with long-term
follow ups; J Appl Oral Sci; 2014
39
•Fragosoet al,microabrasionfollowed by polishing with diamond paste or
fluorideprophylaticpasteprovided higher hardness and bettersurface smoothnessof the enamel
•Segura, et al.showed thatenamel aftermicroabrasiontechnique is more resistant to
demineralization, it was also observed that there isless colonization by Streptococcusmutans
• Miereles et al. showed thatenamel treated with Phosphoric acidproduced a rougher surface than enamel
treatedwithHCl
• Bezarraet alshowed thatboth acids (37% H 3 PO 4,18%HClwith pumice) can be used successfullywithout any
statistically significant clinical differences
40
McInnes Microabrasion Technique
41
Macroabrasion
• When the stains are very deep and intense some times the more
conservative treatments are not totally effective
• This also is a technique for the treatment of surface stains & defects,
but utilizes a 12- fluted carbide bur or a micron diamond point at
high speed
• Microabrasion(gross) + Macroabrasion(finer)
microabrasion and macroabrasion By Dr. Vishaal Bhat 42
Bleaching
According to Grossman-
Bleaching is defined as the lightening of the color of the teeth through
the application of chemical agent to oxidize the organic pigmentation
in the teeth
43
Indications Contraindications
•Guarded
PatientPrognoses for bleaching
dissatisfaction with • Poor case selection
1. Sensitive teeth
tooth color • Dentin hypersensitivity
2. Extremely
• To extend dark gingival
esthetic lifethird
of of • Extensively restored teeth
tooth – visible
existing when smiling
crown • Hypoplastic marks and cracks
3. Exposed root surfaces
• Psychological benift – 10-14
4. Extensive • Defective and leaky
years –white spots self image
individual’s restorations-
5. Temoromandibular disorders/
Bruxism -metallic salts – silver amalgam
6. Translucent teeth -defective obturation
• Pregnancy/ lactation
• Unwilling to tolerate taste
45
Mechanism Of Action
• Hydrogen Peroxide:
• Sodium Perborate:
superoxol
Sodium Perborate Sodium Metaborate + Hydrogen Peroxide +O₂
46
Carbamide Peroxide:
10% Carbamide Peroxide
Ammonia Carbon
dioxide oxygen Water
47
Composition of commercial bleaching agents
1. Active Ingredient: Hydrogen peroxide and Carbamide Peroxide
2. Inactive ingredient:
a. Thickening agents:
❖ Carbopol (carboxypolymethylene)
⮚ 0.5% and 1.5%
⮚ Advantages :- Increases viscosity - better retention of bleaching gel in tray
(approx – 29 ml per arch)
-Increases active oxygen-releasing time of bleaching material
e. Flavourings :
⮚ Peppermint, spearmint, wintergreen, sassafras, anise
⮚ Sweetener such as saccharin
50
2.Tooth Whitening Pen:
⮚ Fast and effective whitening
⮚ Whitening gel – pen like applicator
Problems with OTC
⮚ Listerine whitening pen– erosion
• Over use and white ice surface
of labial whitner stick
of teeth
- dissolution of enamel and loss of anatomy
• Exposed Dentin appears darker
3. H2O2 Strip system:
• More sensitivity
⮚ Trayless bleaching system
• These kits contain:
⮚ Thin strip precoated with an adhesive 5.3% Hydrogen peroxide gel
1. Acid rinse – citric or phosphoric acid –erosion
⮚ Worn for 30 min – removed
2. Bleaching and discarded
gel: acidic – twice
pH – between 1 and 2a day for 14 days
3. Post – bleach polishing cream: Titanium dioxide
( temporary painted white appearance)
Chewing gum
51
Mechanism Of Action
H 2 O2 H+ + HO2-
56
Thermocatalytic Bleaching:
⮚ Use of heat alone or heat and light both
⮚ Heat units:
1. Heat light unit:
- A narrow beam of light is concentrated in one
section of mouth at 13-15 inches distance
- Calibrated rheostat controls it
2. Heat unit:
- Non-vital teeth: 60-70 degree celsius
- Vital teeth : 46-60 degree celsius
57
• Sources of heat:
• Photoflood lamp
• Polymerization light
• Spirit lamp
• Commercial Bleaching Lights
• Light heat lamp
• Lasers – Argon and Diode
•3 appointments
•Scheduled 2-4 weeks apart
59
Power Bleach:
Indications:
Single tooth bleaching within arch
• Developmental or acquired stains
• Stains in enamel and dentin
• Mild to moderate Tetracycline stains
Advantages:
• Immediate result
• Preferred over home bleaching – wearing trays
- distaste
Disadvantages:
• Expensive
• Dehydration – falsely lighter shade immediately post treatment
Rehydration – slightly darker discoloration (rebound discoloration)
60
• Equipment needed:
1.Power bleach materials: 35% H2O2 , 35% Carbamide Peroxide
- Dual activated (35% H2O2 – light and chemically)
2. Tissue protector: Light activated liquid resins- (Paint-on-dam)
3. Energizing and activating source: -Conventional halogen lights
- Plasma arc lamps
- LED lights
- Xenon power arc light
- Lasers: Argon and CO2
4. Heat source: may not be necessary
5. Mechanical timer
61
Plasma arc light: 6-7mm
Light cured protective away from gel
resin is placed around - Emits 3-s bursts of light
gingival margins - 3 times at 3 min
Bleaching material is intervals
applied (10-15 min)
63
Dual Activated Technique:
⮚ Hi – Lite system: Light and chemical activation( Tetracycline stains)
⮚ Constituents:
1. Ferrous Sulphate: chemical activator (7-9min)
2. Manganese Sulphate: light activated(2-4 min)
3. H2O2: 19 – 35%
4. Blue – green indicator dye
Procedure:
⮚ Material placed on teeth
⮚ Left – 6 to 10 min – removed
⮚ Repeated again – 6 times per visit if required
64
Non-Thermocatalytic Bleaching:
⮚ Does not utilize heat sources
⮚ Solutions:
1. Superoxol(5 parts of H2O2+1 part of ether)
2. McInnes solution: 5 parts of 36%H2O2 +5 parts 30%H2O2+1 part
of 0.2%anaesthetic ether
3. Modified McInnes solution: 30%H2O2and 20%NaOH in 1:1
ratio with 0.2%ether
4. Self activating bleaching agents
5. Matsuba et al- 35%H2O2 0.4ml, CaO 0.12gm, Aerosil 0.32, 0.48 or
0.64 gms
65
After rubber dam application
66
Microabrasion assisted bleaching:
⮚ Indications:
- Superficial stains but not for deeper stains
- Areas of Enamel Fluorosis
- Decalcification lesions – orthodontic treatment
- Multicolored stains
67
Dentist prescribed bleaching(night guard)
Matrix bleaching/ At- home bleaching
⮚ Success rate for - Non- tetracycline stained teeth : 98%
- Tetracycline stained teeth: 86%
⮚ Commercial preparations:
1. 10% carbamide peroxide with carbopol(Proxigel, Ultralite)
2. 10% carbamide peroxide without carbopol(fast oxygen-releasing-
Glyoxide, Dentalite)
3. 15% carbamide peroxide- Nu smile
Indications:
4. 1-10% H2O2
• Age yellowing discoloration
• Mild tetracycline staining
• Very mild fluorosis
• Stains from smoking tobacco 68
Home Bleaching Trays:
Reservoir(spacer):
• Void or space in bleaching tray
• Receptacle for bleaching material
• Viscous material – better retained
• Disadvantage: tray – less retentive
- more bulbous and slightly thicker
- more material to fill tray
- occlusal interferences(mandible)
Placed on : Buccal/facial surfaces atleast 1mm from gingiva
70
1. Full vestibule upper and lower trays – not used
2. Scalloped/ Reservoir: minimal tissue contact desired
maxillary arch
3. Non – Scalloped/Non-Reservoir: maximum retention
mandibular arch
4. Facial/Buccal Scalloped Reservoir: taste and tongue irritation- problem
5. Scalloped Reservoir buccal and lingual surface: TMD patients
6. Scalloped non reservoir: fluid or paste materials
7. Non-Scalloped reservoir: tray with viscous material(mandibular)
8. Trays with a foam liner: may impinge on occlusion – not recommended
72
Dentist prescribed bleaching(night guard )
Take an
alginate Pour the
impression of impression
the whole with plaster
arch.
Trim the tray
in a straight
pattern on the Trim the cast
facial and
lingual side Disinfect the
tray with a
Mark the
cleaning
tooth to be
Instruct the solution in an
bleached
patient to ultrasonic
Deliver
cleaner the
place the
tray and home
bleaching gel
bleaching gel
only at the
(10%–
discolored
20%carbamid
tooth and
eperoxide gel)
wear the tray
overnight Evaluate the
color of the
tooth
73
Side effects of Vital Bleaching:
• Sensitivity
• H2O2 and HCl- affect adhesion of composites and GIC
• Caries progression
• Minor ulceration of gingival tissues
74
Non vital tooth bleaching
75
Barriers:
• Guide : Labial Cemento-enamel junction
curves in incisal direction on proximal sides of teeth
• Flat barrier: proximal dentinal tubules unprotected
Cervical resorption
76
Barrier Transfer
77
• Facial view: Bobsled
Tunnel outline
• Proximally: Ski slope
outline
78
Preparation of the patient:
• Diagnose
• Record shade and photographs
• Record barrier probings
• Isolate the tooth
• Prepare and clean access cavity- necrotic debris etc
• Approx – 2-3mm of root canal restorative material is removed in
apical direction beyond CEJ
• Irrigate properly
• Transfer barrier probings
• Place barrier material – GIC , Cavit
- GIC: straight lentulo spiral mounted in latch type or slow hand piece
- Cavit: small cone on end of Plugger
• Introduce bleaching solutions
79
Thermocatalytically Activate Bleach:
•Superoxol
•Sodiumperborate
•Activate bleaching agent by repeatedly placing instrument against saturated cotton pellet
until bleach has evaporated
•Repeat this processupto3 times
82
•Familiarize the patient the causes ,procedure expected outcome
83
•Evaluate tooth color with shade guide / clinical photographs for future
reference
•Isolate tooth with rubber dam ( prevent leakage of bleaching agent onto
gingival tissue
•Interproximal wedges and ligatures
84
•Remove materials just belowlabio-gingival margin1–2mm below the CEJ
•Orange solvent/chloroform/xylene – dissolve sealer remnants
•Barrier transfer:
•Apply sufficient thick layer 2 mm ( protective cement –
polycarboxylate,zincphosphate,glassionomer,IRM,Cavitto
cover endodonticobturation
85
•Thick well sealed temporary filling material ( IRM) to be
given( 3mm thick – good seal )
•Evaluate patient 2 weeks later and repeat it if necessary
86
Modified Walking Bleach:
⮚ This technique is modified with a combination of 30% hydrogen
peroxide and sodium perborate sealed into the pulp chamber for
one week
87
Combination Bleaching technique:
⮚ Thermocatalytic in office non vital bleaching + walking bleach
regimen(sodium perborate and superoxol)
⮚ Synergistic effect
88
Inside/Outside Bleaching:
• Intracoronal + Home bleaching Technique
• More effective
• Difficult stains – single dark
• Decrease incidence of Cervical Resorption
Procedure:
⮚ Place mechanical barrier
⮚ Place cotton pellet in chamber
⮚ Check fitting of bleaching tray
⮚ Instructions : remove pellet before bleaching
- bleaching syringe directly placed in pulp chamber before seating tray
and excess – on tray
89
Side effects of non vital bleaching:
▪ External root resorption
- Irritating chemical diffuses via unprotected dentinal tubules and
cementum defects
- necrosis of cementum, inflammation of periodontal ligament,
finally- root resorption
90
Suggestions for safer intracoronal non vital bleaching:
• Isolate tooth effectively
• Protect oral mucosa
• Verify adequate endodontic obturation
• Use protective bariers
• Avoid acid etching
• Avoid strong oxidizers
• Avoid heat recall periodically
91
Laser assisted tooth whitening:
❑ Argon laser(488nm)
❑ Carbondioxide(10,600nm)
92
Argon Laser:Blue light - 480nm
es fall apart into extremely reactive ionic fragments – combine withchromophilicstructure of organ
93
Carbon dioxide laser(10,600nm)
⮚ Enhancing effect of argon laser
⮚ The laser penetrats only 0.1mm into water and H2O2 – absorbed
MOA-
Argon laser- activate bleaching gel
94
Diode Laser:
⮚ Semiconductor lasers(980nm)
⮚ Forms :
1. Infrared diode – 790nm
2. laser with blue light emission diode- 467mn
3. GaAIAs diode- works at different watts
(Gallium-Aluminium-arsenide)
95
Photochemical Laser whitening:
⮚ KTP Smart Bleach- new smart bleach technique
⮚ Photochemical not photothermal
⮚ Bleaching gel- alkaline(9.5)
⮚ Perhydroxyl radical – more reactive than superoxide
⮚ Tetracycline stains
Red quinone product – dimethylamino tetracycline
colorless
96
Effects of Bleaching Process
• Effects on soft tissues:
- In-office bleaching (30–35% hydrogen peroxide) - produce soft-tissue
burns - tissue white
- Reversible
• Systemic effects:
- Gastrointestinal and mucosal irritation - burning palate and throat,
minor upsets in the stomach or intestines
• Effects on Enamel surface morphology and texture
-Increased porosity , demineralization and decreased protein concentration
-Organic matrix degradation
- Calcium loss
97
• Effects on Enamel surface hardness and wear resistance:
⮚ 10% carbamide peroxide and 7.5% hydrogen peroxide - change the
surface micromorphology of enamel(Sasaki et al.2009)
98
• Effects on enamel chemical composition
- Increase H2O2 - microhardness of enamel decreased significantly
(Al-Salehi et al. (2007)
99
• Effects on dentin:
- Pecora et al. (1994) - dentin microhardness decreased after
application of a 10% carbamide peroxide agent for 72 h
100
• Effects of dental bleaching on composite resin restorations
101
- Heated 30% hydrogen - composite color changes (Canay and
Cehreli, 2003)
102
Effects on the bonding of composite resin restorations to tooth
structure
⮚ Vital bleaching - alter protein and mineral content of enamel,
reduced bond strength (Perdigao et al., 1998)
103
⮚ (Adebayo et al., 2007; Gurgan et al., 2009; Moule et al., 2007)- BS
of bleached enamel was best when etch-and-rinse adhesives were
used
104
Recent advances
105
3.Tooth Bleaching with non thermal atmospheric plasma:
• Effective bleaching without thermal change
107
References
• Ingles endodontics 6 th edition
• Cohens pathways of pulp -11 th edition
• Summits Fundamentals of Operative Dentistry -4 th edition
• Grossman’s Endodontic Practice- 12th Edition
• Complete Dental Bleaching- Goldstein
• Bleaching Techniques in restorative dentistry – Martin Dunitz
• Tooth whitening- indications and outcomes of night guard
bleaching – van B. Haywood
• Textbook of operative dentistry – Vimal K sikri
• Dahl et al, Tooth bleaching- a critical review. crit rev oral biol med
14(4):292-304 (2003)
• Samir et al, White Spot Lesions: Formation,Prevention and
Treatment, Vol 14, No 3,2008: pp 174-182
108
• Sulieman M. An overview of bleaching techniques: 3. In-surgery or
power bleaching. Dent Update. 2005;32(2):101-4
• Esthetic rehabilitation of discolored anterior teeth with porcelain
veneers Kamble Vaibhav et al, 2013
• Microabrasion and macroabrasion By Dr. Vishaal Bhat
• Sundfeld et al.; Microabrasion in tooth enamel discoloration
defects: three cases with long-term follow ups; J Appl Oral Sci; 2014
• The Chemistry and Mechanisms of Extrinsic and Intrinsic
Discolouration; Nathoo; 1997
• Watts, and M. Addy: Tooth discolouration and staining: a review of
the literature. British Dental Journal Volume 190 No.6 March 24
2001
• Manuel ST, Abhishek P, Kundabala M. Etiology of tooth
discoloration-a review. Nigerian Dent J 2010;18(2):56-63
109
110