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  • Soal Retina

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    drheri
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    1. A 50-year-old man with a history of long-term chloroquine therapy presented with blurry vision and was found to have signs of chloroquine retinal toxicity including bull's eye maculopathy and crystalline retinopathy. 2. A 60-year-old patient presented with intraretinal hemorrhages, microaneurysms, and venous changes and was recommended for follow-up every 2-4 months and possible focal laser treatment. 3. A 19-year-old patient presented with blurry vision and was found to have macular atrophy surrounded by flecks, with findings on FFA consistent with a diagnosis of Stargardt disease.

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    retina

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    soal retina

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    1. A 50-year-old man with a history of long-term chloroquine therapy presented with blurry vision and was found to have signs of chloroquine retinal toxicity including bull's eye maculopathy and crystalline retinopathy. 2. A 60-year-old patient presented with intraretinal hemorrhages, microaneurysms, and venous changes and was recommended for follow-up every 2-4 months and possible focal laser treatment. 3. A 19-year-old patient presented with blurry vision and was found to have macular atrophy surrounded by flecks, with findings on FFA consistent with a diagnosis of Stargardt disease.

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    87 views2 pages

    Soal Retina

    Uploaded by

    drheri
    1. A 50-year-old man with a history of long-term chloroquine therapy presented with blurry vision and was found to have signs of chloroquine retinal toxicity including bull's eye maculopathy and crystalline retinopathy. 2. A 60-year-old patient presented with intraretinal hemorrhages, microaneurysms, and venous changes and was recommended for follow-up every 2-4 months and possible focal laser treatment. 3. A 19-year-old patient presented with blurry vision and was found to have macular atrophy surrounded by flecks, with findings on FFA consistent with a diagnosis of Stargardt disease.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    of 2

    1.

    a man 50 years old, consulted from internal department with hystory of long term chloroquin therapy. This patient complain blur vision in the both eye. From examination we could find a. granular depigmentation of central RPE b. bulls eye maculophaty c. central scotoma d. numular retinal hyperpigmentation e. crystalline retinopathies

    2.

    60 years-old patient present diffuse intraretinal hemorrhage and microaneurims in 4 quadrants, venous beading in inferotemporal and inferonasal quadrants, and intraretinal microvascular abnormalities in inferotemporal quadrant. Which of treatment option is most correct ? a. suggested follow-up every 6 month b. suggested follow-up every 2-4 month c. focal laser photocoagulation d. grid laser fotocoagulation e. scatter pan retinal photocoagulation

    3.

    19 years old patient came to the eye clinic complain blur vision in both of his eyes. VODS 6/30 ph-, anterior segmen clear. From fundus examination found macular athropy surounded by yellowish fleck. FFA shows dark choroid. The most possible diagnosis: a. Stagart disease b. Chloroquin retinal toxicity c. ARMD d. Cone-Rod dystrophies e. Ceroid lipofuscinosis

    4.

    A 68 years old man complained of sudden onset of decreased vision, metamorphosia, paracentral scotomata in both of his eyes. There was elevation of the RPE, subretinal lipid deposit, and PED. In order to diagnosing whether theres also choroidal neovascularization in this patient, its best to us to perform A. Indocyanine green angiography B. Contrast Sensitivity test C. Farnsworth Panel D-15 test D. Fluorescent Angiography E. Dark adaptation test

    5.

    A 50 years old woman came to Eye clinic of RSMH with main complain blurred vision in both eye. metamorphopsia and central scotoma since 6 month ago. The examination found that: RLVA 6/21 ph (-), normal anterior segment and Posterior segmen found: round yellow subfoveal lesion with central pigmented spot. What is the diagnosis a. best disease b. adult onset vitelliform lesion c. familial drusen d. Pattern dystrophies e. Sorsby macular dystrophy

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