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    Phillip Go
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    The document discusses the pathophysiology of dengue hemorrhagic fever (DHF). It describes how the dengue virus enters the body via the bite of an infected Aedes aegypti mosquito. This leads to an interruption in homeostasis as the immune system responds by producing cytokines and prostaglandins. This causes physiological changes like increased vascular permeability and fragility. This can result in bleeding, decreased blood volume, hypotension, shock, and potentially death if not treated. The summary provides an overview of the key stages and processes involved in the pathophysiology of DHF.

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    PATHO DHF

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    The document discusses the pathophysiology of dengue hemorrhagic fever (DHF). It describes how the dengue virus enters the body via the bite of an infected Aedes aegypti mosquito. This leads to an interruption in homeostasis as the immune system responds by producing cytokines and prostaglandins. This causes physiological changes like increased vascular permeability and fragility. This can result in bleeding, decreased blood volume, hypotension, shock, and potentially death if not treated. The summary provides an overview of the key stages and processes involved in the pathophysiology of DHF.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    403 views2 pages

    Patho DHF

    Uploaded by

    Phillip Go
    The document discusses the pathophysiology of dengue hemorrhagic fever (DHF). It describes how the dengue virus enters the body via the bite of an infected Aedes aegypti mosquito. This leads to an interruption in homeostasis as the immune system responds by producing cytokines and prostaglandins. This causes physiological changes like increased vascular permeability and fragility. This can result in bleeding, decreased blood volume, hypotension, shock, and potentially death if not treated. The summary provides an overview of the key stages and processes involved in the pathophysiology of DHF.

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    of 2

    VI.

    Pathophysiology
    A.Theoretical-based
    Non-modifiable factors: Modifiable factors:
    AGE and SEX – both are Poor envi’t sanitation (presence of
    affected since dengue is a PATHOPHYSIOLOGY OF DHF stagnant water container junk tires
    universal infection and bottle)
    SEASON – Aedes aegypti is Poor practice to protect self against
    more commonly found in mosquitoes
    tropical and subtropical
    countries Other lifestyle practices

    Mosquito bite (AEDES AEGYPTI)

    Dengue virus enters the body carried by circulation and multiplies

    Interruption in the normal homeostatic environment of the body

    Immune response by the body (defense mechanism of the body to foreign bodies)

    Body induces the action of the cytokines and prostaglandin

    The body responds by producing physiologic changes aimed at elevating body temperature.
    FEVER

    Cytokine and prostaglandin

    Increase capillary or vascular permeability and fragility

    Ruptured capillaries

    Oral mucosa nasal mucosa .petechiae Thrombocytopenia GIT bleeding vomiting


    gum bleeding epistaxis ulceration

    Severe Bleeding

    Decrease blood volume

    Hypotension

    Shock

    DEATH
    B.Client-based

    PATHOPHYSIOLOGY OF DHF
    Non-modifiable factors: Modifiable factors:
    AGE and SEX – both are Environment- Community is
    affected since dengue is a exposed to dengue cases
    universal infection Presence of stagnant waters and
    Age- 8 y/o uncovered containers near their
    Sex- Female house which is a possible
    sources of vectors.

    Mosquito bite (AEDES AEGYPTI)

    Dengue virus enters the body carried by circulation and multiplies

    Interruption in the normal homeostatic environment of the body

    Immune response by the body (defense mechanism of the body to foreign bodies)

    Body induces the action of the cytokines and prostaglandin

    The body responds by producing physiologic changes aimed at elevating body temperature.
    FEVER for 4 days jan. 12-15 2010

    Cytokine and prostaglandin

    Increase capillary or vascular permeability and fragility

    Ruptured capillaries

    nasal mucosa(epistaxis) Thrombocytopenia(low CBC PC) petechiae

    Treated early with doctor @ MDH and was given prescribed


    medications and managed to prevent the
    virus ( ranitidine, paracetamol, hydraxyzine)

    With IVF d5Lr 1L x 8 hours for fluid replacement


    and prevention of dehydration

    Patient recovers improvement


    in the CBC PC last January 16,2009
    patient advised MGH.

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