Professional Documents
Culture Documents
Acknowledgement
Would like to acknowledge SAI and NIS for the opportunity to share
my thoughts on this topic
ACSEP
WHY SHOULD I LEARN ABOUT FOP INJURY
AND PREVENTION. Why do we have the medical
COACH team then……
DOCTOR
& medical team
Mechelen et al
Establish the
extent of the
problem
Assesseign
the
effectiveness
Injury Establish
etiology and
of the
measure Prevention mechanism
Introduce
preventive
measure
Athlete injury
What do1.you
Training do:big Is the injury? (And not what is the injury)
level
How
ISL
Equipment
Immediately level
Athlete level or medical room ( Short term)
On the side-line
2. When
In the coming can
days the Athlete
( Long term) return to sport ?
PREVIEW
Collapsed Athlete
Runner with Midfoot pain
Apophyseal Injuries
Rower with Chest pain
Wrestler with Skin rash
Parathlete with HTN
CASE 1
What do you do ?:
Immediately
Short term
Long term
Immediate action
d ay
sa me
o n the
to p lay
et ur n
No r
Send to hospital immediately if:
b) Seizures
e) Skull Fracture
OR
COGSPORTS
Early Neuro-vestibular rehab
CONCUSSION RECOGNITION TOOL 5 ©
To help identify concussion in children, adolescents and adults Role of helmets & mouthguards
RECOGNISE & REMOVE in Australian Football
Head impacts can be associated with serious and
potentially fatal brain injuries. The Concussion
Recognition Tool 5 (CRT5) is to be used for the
STEP 3: SYMPTOMS Helmets Mouthguards
designed to diagnose concussion.
• Headache • “Don’t feel right” There is no definitive scientific Mouthguards have a definite role in
• “Pressure in head” • More emotional
evidence that helmets prevent preventing injuries to the teeth and face
STEP 1: RED FLAGS — CALL AN AMBULANCE • Balance problems • More irritable
concussion or other brain injuries and for this reason they are strongly
• Nausea or vomiting • Sadness
If there is concern after an injury, including
• Drowsiness • Nervous or anxious
in Australian Football. recommended at all levels of football.
whether ANY of the following signs are observed Mouthguards should be worn for all
or complaints are reported, then the player • Dizziness • Neck pain Helmets may have a role in the
• Blurred vision • games and training sessions.
should be safely and immediately removed from protection of players on return to play
play/game/activity. If no licensed healthcare • Sensitivity to light •
following specific injuries (e.g. face or Dentally-fitted laminated mouthguards
professional is available, call an ambulance for • Sensitivity to noise • Feeling slowed down
urgent medical assessment: skull fractures). offer the best protection. ‘Boil and
• Fatigue or low energy • Feeling like “in a fog“
bite’ type mouthguards are not
• Neck pain or • Loss of Overall, however there is insufficient
STEP 4: MEMORY ASSESSMENT recommended for any level of play as
http://www.sportsconcussion.co.za/Concussion/CogSportFlyer.pdf
tenderness consciousness
(IN ATHLETES OLDER THAN 12 YEARS)
scientific evidence to make a
• Double vision • Deteriorating they can dislodge during play and block
• Weakness or conscious state recommendation for the use of helmets
the airway.
tingling/burning in • Vomiting for the prevention of concussion in
arms or legs • Increasingly appropriately for each sport) correctly may suggest
a concussion: Australian Football. There is some preliminary scientific
• Severe or increasing restless, agitated
headache or combative • “What venue are we “What team did
evidence that mouthguards may
• Seizure or convulsion at today?” you play last prevent concussion or other brain
• “Which half is it now?” week/game?” injuries in Australian Football.(3)
• “Who scored last in • “Did your team win
: this game?” the last game?”
• In all cases, the basic • Do not attempt to
move the player (other Athletes with suspected concussion should: References
(danger, response, than required for
airway, breathing, airway support) unless • Not be left alone • Not be sent home
circulation) should trained to so do. initially (at least by themselves. They 1. McCrory P, Meeuwisse W, Dvorak J, et al. Consensus statement on concussion
be followed. • Do not remove a
need to be with a in sport-the 5th international conference on concussion in sport held in Berlin,
• Not drink alcohol. responsible adult.
• Assessment for a helmet or any other October 2016. Br J Sports Med 2017 doi: 10.1136/bjsports-2017-097699
spinal cord injury equipment unless • Not use recreational/ • Not drive a motor
is critical. trained to do so safely. prescription drugs. vehicle until cleared to 2. Echemendia RJ, Meeuwisse W, McCrory P, et al. The Concussion Recognition Tool
do so by a healthcare
5th Edition (CRT5). Br J Sports Med 2017 doi: 10.1136/bjsports-2017-097508
professional.
Athlete with Concussion ABC, Stop participation SCAT-5 Neurological Prevent Post Concussion
Cervical spine assessment Symptomatic recovery Syndrome
Assess if hospital transfer is Cogsport, ImPACT Neuropsychological testing
required Neurovestibular rehab
CASE 2
What do you do ?:
Immediately
Short term
Long term
EVALUATION
• Where exactly is the pain
• Previous hx
• Diet
RULE OUT NAVICULAR STRESS FRACTURE
• Training
• Footwear
• Surface
Important sign: “ N- Spot” tenderness
Mechanism of Injury
Staging of stress Fracture
Treatment
CAUTION
NON-OPERATIVE Operative
• Screw across #. Percutaneous or
open and bone graft.
PLEASE DON’T TAKE THIS
• IMMOBILISE
• Complete # two cortices (Saxena
INJURY LIGHTLY
• Graduate RTP
III) probably
• ADL, water running/swimming à
jogging à running
• NON UNION
àsprint/takeoff/cutting
• High level athletes
CAN BE predisposing
• Correct CAREERfactors
ENDING
• FAILED CONSERVATIVE RX
INJURY
• Maybe quicker RTP?
Prevention Strategies
TRAINING LEVEL ATHLETE LEVEL
Training error – repetitive high level or rapid
increase training load Decreased ankle DF
• Esp. jumping, push-off, sprinting, cutting
Stiff subtalar joint
Short 1st MT plus long 2nd MT
Red flags
Red flags Infection, osteomyelitis, Bony tumour
Features to suggest Calcaneal Apophysitis- Sever’s disease
• Posterior inferior heel pain
• 23 yr
• Participating in camp
• High volume (200 km / 3 days)
• High intensity
• Rough water
Immediate Action
• Stopped immediately
• Night pain
• Pain with ADLs
• Pain with deep
inspiration
• Cough
• Sit up
• Push up
• Thoracic spring
Courtesy D Huges AIS
INVESTIGATION
EQUIPMENT LEVEL
• Weight and Size of Equipment
ATHLETE LEVEL
• Biomechanical issues
• Kinetic chain
• Warm-up and land-based training (Ergometers)
Key messages
Athlete with Concussion ABC, Stop participation SCAT-5 Neurological Prevent Post Concussion
Cervical spine assessment Symptomatic recovery Syndrome
Assess if hospital transfer is Neurovestibular rehab Neuropsychological testing
required
Runner with Midfoot pain Assessment to rule out Staging of the stress fracture Rehab and Return to play
Navicular stress fracture Non-operative Rx Address risk factors
N-Spot Operative Rx
Stop running
Adolescent with heel pain Assessment Intial treatment Biomechanical correction
POLICE Activity modification Training loads
Education
Rower with Chest pain Stop activity Healing of the fracture Correct biomechanics
Initial assessment
Bone scan/CT
Staging of the stress #
Case 5
Wrestler with
Skin rash
Primary Herpes
Gladiatorum
Herpes
(1991) Gladiatorum-
Prevalence in wrestlers:
Presentation
2.6-29% High School Herpes Gladiato
7.6-12.8% Collegiate
20-40% Division I
Location
Presentation
73% on Head and Face
42% on Extremities Location
Herpes Gladiatorum-
28% on Trunk 73% on Head and Face
contact
Presentation
Appear 3-8 days after 42% on Extremities
28% on Trunk
Primarily atLocation
locations of Appear 3-8 days after
‘Lock-up’ position
73% on Head and Face contact
42% on Extremities
Only from skin-to-skin Primarily at locations of
28% on Trunk
contact Appear 3-8 days after
‘Lock-up’ position
No association with
contact Only from skin-to-skin
mats Primarily at locations of contact
‘Lock-up’ position
Only from skin-to-skin
No association with
contact mats
No association with
mats
HERPES GLADIATORUM
Symptoms:
Herpes Gladiatorum-Recurrent
Outbreaks
• Painful Vesicles with red base
• Malaise
• Pharyngitis
Latency
• Feverand Reactivation are the rule
Usual reoccurrences
• Lasts 10-14 days last 3-5 days
Less signs and symptoms than primary
outbreak
Brought on by stress, i.e. weight
cutting, abrading or rubbing facial skin,
sun exposure, suppressed cell-mediated
Primary HG: Note grouped vesicles on forehead and along jawline
immunity
TREATMENT PREVENTION
Personal Hygiene
REMOVE ATHLETE FROM PLAY Equipment Hygiene
Avoid overtraining and Fatigue
CANNOT PLAY TILL ALL VESCICLE GET DRY and no General conditioning
Consider treatment for the whole team
NEW VESCICLES in 48 HRS
CANNOT COVER THE RASH AND PLAY
CAN SPREAD TO OTHER ATHLETES MYTHS
ANTI VIRAL DRUGS
CONSIDER TREATING OTHER TEAM MEMBERS Not Sexually Transmitted
It can occurs even if you wash the mat
EQUIPMENT AND PERSONAL HYGIENE Its different from Herpes- cold sore
Its different from other skin infections which are
more common in athletes
SUMMARY
Condition Immediate action Short term Action Long term Action
Athlete with Concussion ABC, Stop participation SCAT-5 Neurological Prevent Post Concussion Syndrome
Cervical spine assessment Symptomatic recovery Neuropsychological testing
Assess if hospital transfer is required Neurovestibular rehab
Runner with Midfoot pain Assessment to rule out Navicular Staging of the stress fracture Rehab and Return to play
stress fracture Non-operative Rx Address risk factors
N-Spot Operative Rx
Stop running
Adolescent with heel pain Assessment Initial treatment Biomechanical correction
POLICE Activity modification Training loads
Education
Rower with Chest pain Stop activity Healing of the fracture Correct biomechanics
Initial assessment
Bone scan/CT
Staging of the stress #
Wrestler with skin rash Assessment Wait for healing of the lesions Personal and Equipment hygiene
Differentiate form other infection Anti-viral Education
Stop participation Team assessment Prevention of recurrence
Case 6
Parathlete
with
hypertension
Spinal cord Injury
wheelchair racing athlete
Examination:
Sharp elevation of blood pressure
Bradycardia
Pressure sores
Distended bladder, blocked catheter
Constipation, faecal impaction
Video- Disclaimer
From You tube in the Public domain
Source is identifiable on the video and only for Educational use.
BOOSTING
Autonomic Dysreflexia and Boosting: Non-pharmacological
doping in disability sport
If BP >180 mm Systolic, retest after few minutes
- If still high: Athlete is disqualified
Management:
- No sanctions, but IPC Ethical committee Remove the patient from competition
- Disqualification is to prevent serious injury
Including death of the athlete Sit the patient up
Examine for the common causes - bowel and bladder
-Insert a catheter if one is not in use
Anti-hypertensive if systolic blood pressure >150mmHg
(Nifedipine 10mg rapid release or GTN)- Rule out
VIAGRA
Avoid beta blockers - may cause peripheral
vasoconstriction resulting in prolonged hypertension
Low threshold to transfer to hospital
Prevention Strategies
TRAINING LEVEL
- Education: recognize symptoms early ATHLETE LEVEL
- rewards and Punishment
- Avoid constipation
EQUIPMENT
-Regular catheterization (avoid bladder distension)
- Wheel chairs
- Monitor for UTI’s
- no sharp areas - Avoid pressure areas
Athlete with Concussion ABC, Stop participation SCAT-5 Neurological Prevent Post Concussion Syndrome
Cervical spine assessment Symptomatic recovery Neuropsychological testing
Assess if hospital transfer is required Neurovestibular rehab
Runner with Midfoot pain Assessment to rule out Navicular stress Staging of the stress fracture Rehab and Return to play
fracture Non-operative Rx Address risk factors
N-Spot Operative Rx
Stop running
Rower with Chest pain Stop activity Healing of the fracture Correct biomechanics
Initial assessment
Bone scan/CT
Staging of the stress #
Wrestler with skin rash Assessment Wait for healing of the lesions Personal and Equipment hygiene
Differentiate form other infection Anti-viral Education
Stop participation Team assessment Prevention of recurrence
Questions?