TROPICAL PATHANATOMY

TOPIC 1
1)The largest changes with giardiasis found in- duodenum and small intestine.
2)Amoebic abscess are found in- Right lobe of the liver
3)Extraintestinal changes are characteristics of- Amoebiasis
4)Morphological changes characteristics of Amebiasis- Erosive and ulcerative
lesions of the colon, abscess in the liver
5)The form of amoeba excreted exclusively from the feces of sick people with
acute amebiasis- tissue form
6)The form of entamoeba in asymptomatic amoebiasis and in feces of carriers-
Cyst and lumen
7)Fecal matter in amoebiasis looks like- raspberry jelly
8)The main route of infection with extraintestinal amoebiasis, which is
accompanied by the appearance of abscesses in the organs- fecal-oral route
9)The development malabsorption syndrome syndrome is due to damage- small
intestine
10)Gastroenteritis, reminiscent of the course of cholera is characteristic of-
Cryptosporidiosis
11)Ameboma- granuloma
12)Sclerosing cholangitis, cholecystitis and hepatitis occur with- Cryptosporidiasis
13)The predominant lesion of the colon occurs with- Amoebiasis, balantidiasis
14)Primary lesion of the small intestine occurs with- Cryptosporidiasis, giardiasis
15)Giardiasis characterized by-
Astheno-neurotic syndrome, pancreatitis, allergic dermatitis
16)The causes of death in Cryptosporidiosis – dehydration, malabsorption,
intoxication
17)Trichomonads cause changes in- small and large intestine
18)The main routes of transmission of intestinal protozoal infections are:
Fecal-oral, contact-house hold,sexual
19)In HIV infected, the causative agent of Cryptosporidiosis- C.bailey.
20)The form of Entamoeba in the affected organ in extraintestinal amoebas-
luminal
TOPIC 2
21)Cholera infection source- Sick person
22)Clinical form of melioidosis- pulmonary
23)Leproms are usually localized in such organs as- organs of the endocrine
system, the brain, kidneys, lungs, heart, skin.
24)Melioidosis is characterized by development of- purulent inflammation with
formation of multiple abscesses
25)Specific cells in the diagnosis of Leprosy- Virchow cells
26)Causative agent of cholera- Vibrio cholera
27)Causative agent of Leprosy- Mycobacterium leprae
28)Causative agent of Melioidosis- Burkholderia pseudomallei
29)The characteristic morphological sign of cholera- serous-hemorrhagic enteritis,
gastritis.
30)The duration of the incubation period of Leprosy- 4-5 years
31)Mechanism of transmission of cholera- fecal-oral
32)The mechanism of transmission of Leprosy- Transmissive
33)The mechanism of transmission of Melioidosis- Contact
34)The natural reservoir of the causative agent of Melioidosis- water and soil
35)The nature of skin inflammation with leprosy- granulomatous
36)The necrosis in Leprosy- caseous
37)The pathogenesis of cholera is directly related to the action- Endotoxin
38)The primary localization of the pathological process in cholera- The small
intestine and stomach
39) The septic form of melioidosis is characterized by- fever with chills and
sweats, cough with thick greenish-yellow sputum, chest pain.
40)The syndrome ,determining the severity of the condition caused by
hypovolemia in cholera is- dehydration.
TOPIC 3
41) Another name Onchocerciasis- River blindness
42)Clinical sign of Onchocerciasis- Conjunctivitis
43)Early clinical manifestations of Dracunculiasis- Sepsis
44)Final host for Wuchereriasis- Person
45)Helminthiasis in filariasis- loaos
46)Loaosis is characterized by the presence of- Calabar tumor
47)Localization of the parasite in Dracunculiasis- Subcutaneous tissues
48)Loasis due- Roundworms
49)Lymphostasis in the external genitalia and lower extremities are characteristic
for- Onchocerciasis
50)Main treatment for Drancunculiasis- Mechanical removal of parasites
51)The Dracunculiasis- Zoonotic infection
52)The causative agent of Dracunculiasis- Dracunculiasis medinensis
53)The causative agent of Loasis- Worm Loa loa(African eye worm)
54)The causative agent for Wuchereriosis- Wuchereria bancrofti
55)Duration of the incubation period of Drancunculiasis- 8-12 montha
56)The duration of the incubation period of loasis- 1-3 years
57)The duration of the incubation period of Wuchereriosis- 3-18 months
58)The main complications of Loaosis- Cardiomyopathy, nephropathy,
encephalopathy
59)Wuchereriosis is characterized by- Elephantiasis
60)The causative agent of Onchocerciasis- Onchocerca volvulus
Topic 4
61)Changes characterizing the tertiary clinical-morphological form of nonvenereal
syphilis- gummy(nodular or ulcerative) rashes, damage to bones, ligaments and
joints
62)Changes in the late yaws- nodular or ulcerative rashes, damage to bones and
joints
63)Characteristic symptoms of non-venereal syphilis- solid buboes, chancre
64)Disease characterized by mucosal lesions in the form of pink spots and whitish
nodules- Bejel
65)Lesions multiple ring-shaped, discoid, sickle-shaped- cutaneous early yaws(the
secondary lesions)
66)Localization of yaws: Initial lesion are- skin of legs,arms,face and neck
67)Morphological elements of the late stage of yaws- hyperkeratosis of palms and
soles with deep fissuring
68)Necrotic destructive lesions of the skin and gummatic lesions of bones and
tissues…- The late stage
69)Pathological changes in the late stage of yaw- the long bones, phalanges of the
fingers and nasopharynx.
70)Pathomorphological elements on the skin that make walking painful (crab
yaws)- plantar papillomas
71)first Bejel disease was describes in children living in areas- Asia, in the middle
Euphrates
72)The initial/primary lesion appears after 21 day incubation period of yaws-
“mother-jaw”
73)The localization of the pathological process in Bejel is- skin, mucous
membrane, bones, joints
74)The main route of transmission of Bejel disease- through broken skin or sexual
contact
75) Outcomes of Pinta- acanthosis, atrophy and hyperpigmentation of skin
76)The pathological process in the late stage of pinta characterized by the
appearance- achromic or hyperpigmented skin lesions
78)The primary element in Bejel disease are- inflammatory papule
79)The reservoir of the pathogen Begala- a sick man
80)The spread of the disease may contribute to the presence of- cuts and
abrasions
81)Yaws occur commonly among children aged- 2-15 years.
TOPIC 5
82)The outcomes of Ebola virus- intoxication, bleeding and hemorrhage, sepsis
83)The mechanism for the development of common hemorrhagic phenomena of
Dengue fever- endothelial swelling and mononuclear infiltration of small vessels
84)Main source of Ebola for humans- direct contact with gorilla, chimpanzees,
monkeys.
85)Main mechanism of Jaundice in Yellow fever- obstructive and parenchymal
86)The main characteristic of the liver in yellow fever- necrosis of hepatocytes
with the bodies of Councilman, mononuclear infiltration.
87)The main characteristic of hemorrhagic fevers is- hemorrhagic diathesis , fever,
intoxication and damage to internal organs.
88)The first symptom of Ebola- severe headache, muscle pain, diarrhea,
abdominal pain
89)The causes of death in hemorrhagic fevers- Thrombohemorrhagic syndrome
90)The causative agent of Ebola is a group virus- Filoviridae
91)Sources of Q fever- cattle
92)Pathomorphological changes characteristic of Q fever- Multiple organ failure,
progressive thrombocytopenia.
93)Coxiella burnetii- Q-fever
94)Characteristic symptoms of Ebola fever- DIC-syndrome, bleeding and
hemorrhage, sepsis
95)Changes characterizing Lassa fever- pneumonia, encephalitis, meningitis,
convulsive syndrome.
96)The localization of the pathological process of the initial period of yellow fever-
cells of the liver, spleen, bone marrow, lymph nodes and kidneys.
97)A characteristic symptom of dengue- Exanthema (roseola, papules, spots),
hemorrhages in the skin of various sizes.
98)The acute phase of a disease occurs with intoxication, myalgia, exanthema,
lymphadenopathy, leukopenia and hemorrhagic syndrome- Dengue
99)The organ in which specific pathological changes are detected with Q-fever-
liver
100) The syndrome in Ebola virus- Kasabach-Merritt syndrome.