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CASE STUDY
ON
SUBMITTED TO SUBMITTED BY
SUBMITTED ON –
IDENTIFICATION DATA
Name : Saida Begam
Age : 68 years
Gender : Female
PID No : 2133759
Diagnosis : ACS(Acute coronary syndrome)
Name of the physician : Dr. Fazal Karim / Dr. Irshad Wani
Date of admission : 02/03/2020
Date of surgery : Nothing significant
Date of discharge : Nothing significant
Education : Graduate
Marital status : Married
Religion : Muslim
Nationality : Indian
Address : Mejarganj, Sultanpur UTTER PRADESH
FAMILY TREE
Male
Female
Patient
PERSONAL HISTORY
SOCIOECONOMIC HISTORY
Consciousness : Conscious
Orientation : Altered
Nourishment : Well nourished
Body built : Moderately built
Activity : Dull
Look : Dull
Hygiene : Power
Speech : Normal
Height : 5.2 inch
Weight : 68 kg
INTEGUMENTARY SYSTEM
SKIN
Colour : Fair
Texture : Normal
Skin turgor : Absent
Hydration : Moderate
Lesions / Masses : No lesions present
NAILS
HAIR
HEAD
EYES
EARS
NOSE
NECK
Alignment : Normal
Movement : Normal
Trachea : Normal
Glands and lymph nodes : No enlargement present
Thorax : Normal
Thorax expansion : Normal
Heart sound : S1 AND S2 sound heard
Breath sound : Normal breath sound heard.
Apical pulse : 106 b/min
Cough : Absent
Sputum : No sputum present
ABDOMEN
MUSCULO – SKELETAL
UPPER EXTERMITIES
Symmetry : Normal
Range of motion : No restriction of range of motion , normal flexion
LOWER EXTERMITIES
Symmetry : Normal
Range of motion : No restriction of range of motion , normal flexion
Oedema / swelling : Absent
Cyanosis : No cyanosis present
Joints : No joint pain present
Deformity : No any deformity present
Gait : Normal
GENTIO- URINARY
Lesions/ Scars : No scar and lesions present
Discharge / Infection : No discharge present
INVESTIGATION
Day 1
2. KFT
Urea 52 mg /dl 15- 36 mg/dl
Creatinine 1.8 mg /dl 0.5- 1.25 mmol/L
Na+ 136 mmol/L 135-145 mmol/L
K+ 4.1 mmol/L 3.5- 5.1 mmol/L
3. LFT
Serum Bilirubin ( Total ) 0.8 mg/ dl 0.2- 1.3 mg / dl
S.G.P.T 73 U/L < 50 U/L
S.G.O.T 46 U/L 17 – 59 U/L
Serum Alkaline Phosphatase 164 U/L 38- 126 U/L
4. CALCIUM 8.5 mg/dl 8. 4 – 10.2 mg /dl
Day 2 -
DAY 3-
DAY 4-
2- KFT
Urea 52 mg /dl 15- 36 mg/dl
Creatinine 1.8 mg/dl 0.5- 1.25 mg /dl
Na+ 136 mmol/L 135-145 mmol/L
K+ 4.1 mmol/L 3.5- 5.1 mmol/L
DAY 5-
TREATMENT
S. Trade Phar Action Do Route Freq Indication Contraindic Adverse Nsg.
N name ma se uenc ation effect Responsibi
O name y lities
1- Tab Ramip Ramistar 2.5 0ral BD Hypertension Kidney Blurred Inform the
Ramist ril Tablet is an mg disease vision doctor if
ar Angiotensin Congestive Liver disease Sweating have a
converting Pregnant Dizziness history of
enzyme Heart failure Marfan Unusual allergy in
(ACE) syndrome tiredness this
inhibitor. Heart disease and medicine.
weakness Report any
Diabetes Back pain undesirable
Chest side effect
tightness to the
Increased doctor
heartbeat promptly.
Cough Check the
Diarrhea vital sign.
Vomiting
2. Tab. Cardiz Anti – 30 Oral TDS Hypertension Hypotension Body pain Check BP
Dilze em anginal mg AV block Cough and ECG
m agent, Anti – Angina Sick sinus Fever before
arrhythmics, syndrome Headache initiation of
Calcium Certain heart Hepatic Diarrhea therapy and
channel rhythm disease Constipatio monitor
blocker disorder Aortic n particularly
stenosis Skin rash during.
Irregual heart Gastroesopha Monitor for
beat geal reflux headache
disease
(GERD)
Pregnancy
CASE STUDY
Aorta
PHYSIOLOGY OF THE HEART The main function of heart is pumping blood it bring out
oxygenated blood from lung to all the body part.The oxygenated blood from body to lungs.
Pulmonary vein
Left atrium
BICUSPID VALVE
Left ventricle
AORTIC VALVE
Diastole - During diastole, the cardiac muscle cells relax to allow the chamber to fill
with blood. Blood pressure increases in the major arteries during ventricular systole and
decreases during ventricular diastole. This leads to the 2 numbers associated with blood
pressure—systolic blood pressure is the higher number and diastolic blood pressure is
the lower number. For example, a blood pressure of 120/80 describes the systolic
pressure (120) and the diastolic pressure (80).
The Cardiac Cycle - The cardiac cycle includes all of the events that take place during
one heartbeat. There are 3 phases to the cardiac cycle: atrial systole, ventricular systole,
and relaxation.
Atrial systole- During the atrial systole phase of the cardiac cycle, the atria contract and
push blood into the ventricles. To facilitate this filling, the AV valves stay open and the
semi lunar valves stay closed to keep arterial blood from re-entering the heart. The atria
are much smaller than the ventricles, so they only fill about 25% of the ventricles during
this phase. The ventricles remain in diastole during this phase.
Ventricular systole: During ventricular systole, the ventricles contract to push blood into
the aorta and pulmonary trunk. The pressure of the ventricles forces the semi lunar valves
to open and the AV valves to close. This arrangement of valves allows for blood flow
from the ventricles into the arteries. The cardiac muscles of the atria depolarize and enter
the state of diastole during this phase.
Relaxation phase: During the relaxation phase, all 4 chambers of the heart are in diastole
as blood pours into the heart from the veins. The ventricles fill to about 75% capacity
during this phase and will be completely filled only after the atria enter systole. The
cardiac muscle cells of the ventricles depolarize during this phase to prepare for the next
round of depolarization and contraction. During this phase, the AV valves open to allow
blood to flow freely into the ventricles while the semi lunar valves close to prevent the
regurgitation of blood from the great arteries into the ventricles.
DEFINITION
Impedance or blockage of one or more arteries that supply blood to the heart usually due
to atherosclerosis is known as CAD.
INCIDENCE
Coronary Artery diseases (CAD) have now become the leading cause of mortality in
India. A quarter of all mortality is attributable to CAD. Ischemic heart disease and stroke are the
predominant causes and are responsible for >80% of CAD deaths. The Global Burden of Disease
study estimate of age-standardized CVD death rate of 272 per 100 000 population in India is
higher than the global average of 235 per 100000 population.
CAUSES
RISK FACTORS
Book Picture Patient Picture
High blood pressure Obesity
High cholesterol levels High blood pressure
High triglyceride levels High cholesterol levels
Diabetes and high blood sugar levels High triglyceride levels
Obesity
Smoking
Age
Family history
CLINICAL MANIFESTATION
DIAGNOSTIC EVALUATION
MANAGEMENT
Medical Management:
THEORY APPLICATION
“OREM’S theory” Self - care deficit will be appropriate to apply in this condition, as because
in Coronary
Artery Disease most of the patients are unable to take care of themselves.
2. Nutrition The nutritional status of the Imbalance nutrition less than body
patient will decrease. requirement related to less intake of
food.
6. Social Patient will be having low self- Risk for situational low self-esteem
interaction esteem. related to unfamiliar environment.
7. Prevention of ----------- -----------
hazard
8. Promotion of Patient is unable to understand Ineffective therapeutic regimen
human the treatment modalities. related to complexity of treatment.
function Patient will be having anxiety. Anxiety related of hospitalization.
NURSING DIAGNOSIS
Acute Pain related to coronary artery occlusion as evidenced by reports of chest pain with
radiation.
Imbalance nutrition status less than body requirement related to less intake of food as
evidence by weight loss.
HEALTH EDUCATION
Home care
Take medicines exactly as directed. Don’t skip doses. Talk with healthcare provider if
medicines aren't working for.
Recovery after a heart attack takes time. Plan to rest for at least 4 to 8 weeks while
recover. Then return to normal activity when doctor says it’s OK.
Ask doctor about joining a heart rehabilitation program. This can help strengthen heart
and lungs and give more energy and confidence.
Tell doctor if are feeling depressed. Feelings of sadness are common after a heart attack.
But it is important to speak to someone or seek counseling if feeling overwhelmed by these
feelings.
Ask family members to learn CPR. This is an important skill that can save lives when it's
needed.
Learn to take own blood pressure and pulse. Keep a record of results. Ask doctor when
should seek emergency medical attention.
Lifestyle changes
This will help the heart disease from getting worse. These changes will most likely be a
combination of diet and exercise.
Diet
Patient may need to see a registered dietitian for help with these diet changes. These changes
may include:
Exercise
Depending on case, patient may recommended that get moderate to vigorous physical activity for
at least 40 minutes each day, and for at least 3 to 4 days each week. A few examples of moderate
to vigorous activity include:
Other changes
Lose weight.
Stop smoking.
Learn to manage stress.
Follow-up
BIBLIOGRAPHY
Hinkle L. Janice , Cheever H. Kerry . Brunner & Suddarth’s Textbook of Medical
Surgical Nursing. 10th edition. page no 1094 – 1103.
Chintamani and Mani Mrinalini. Medical surgical nursing Assessment and management
of clinical problems. Third south Asia edition.2018: ELSEVIER India private limited;
new delhi. Pg-674-678.
shodhganga.inflibnet.ac.in/bitstream/10603/.../12_summary%20and%20conclusion.p.
https://www.ncbi.nlm.nih.gov/books/NBK235823/
vivaessay.net/custom-written-medical-essay-example-on-myocardial-infarction.php
https://www.mayoclinic.org/diseases-conditions/heart-attack/...causes/syc-20373106
https://www.health.harvard.edu/a_to_z/heart-attack-myocardial-infarction-a-to-z