Inhibits muscle contractions by blocking calcium receptors Produces a peripheral (predominantly arteriolar) vasodilation thus reducing the blood pressure buttocks / 1 45 mins. May decrease acetylcholine released by nerve impulses, but anticonvulsant mechanism is unknown.
Inhibits muscle contractions by blocking calcium receptors Produces a peripheral (predominantly arteriolar) vasodilation thus reducing the blood pressure buttocks / 1 45 mins. May decrease acetylcholine released by nerve impulses, but anticonvulsant mechanism is unknown.
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Inhibits muscle contractions by blocking calcium receptors Produces a peripheral (predominantly arteriolar) vasodilation thus reducing the blood pressure buttocks / 1 45 mins. May decrease acetylcholine released by nerve impulses, but anticonvulsant mechanism is unknown.
Copyright:
Attribution Non-Commercial (BY-NC)
Available Formats
Download as DOC, PDF, TXT or read online from Scribd
DRUG CLASSIFICA ACTION/INDICATI DOSAGE/ROUTE/FREQ NURSING EVALUATION
NAME TION ON UENCY CONSIDERATION
S BRAND: Anticonvulsa Inhibiting muscle 1st dose: Assess Patient responds nt contractions by 4 mL / SIVP / 1 ̊ patient’s well to therapy, blocking calcium condition doesn’t GENERI receptors 2nd dose: before experience C: Magnesi 5 mL / ® buttocks / 1 therapy and convulsions. Patient does not um ̊ 45 mins. reassess experience injury Sulfate Produces a regularly related to drug peripheral thereafter induced adverse (predominantly to monitor reactions. arteriolar) drug Patient and vasodilation thus effectivene family state reducing the blood ss. understanding pressure Monitor about drug respiration, therapy. watch for respiratory May decrease depression acetylcholine and signs released by nerve of heart impulses, but block. anticonvulsant Monitor mechanism is renal unknown. status. Assess INDICATIONS: input- Used as an output anticonvuls ratio. ant. Monitor for Lowers possible intracranial drug pressure. induced Prevention adverse and control reactions: of seizures CNS: in toxemia drowsiness, (preeclamps depressed ia, reflexes, eclampsia). flaccid Inhibits paralysis, uterine hypothermi contractions a. CV: in tocolysis hypotensio (preterm n, flushing, labor). circulatory collapse, depressed cardiac function, heart block. Metabolic : Hypocalce mia Respirato ry: paralysis Skin: diaphoresis Monitor for signs of magnesium toxicity: disappeara nce of knee jerk and patellar reflexes are signs of early magnesium toxicity. Assess prior drug history; there are many drug interaction s
DRUG CLASSIFICATIO ACTION/INDICATIO DOSAGE/ROUTE/FREQU NURSING EVALUATION
NAME N N ENCY CONSIDERATIO NS BRAND: Histamine H2 Inhibits histamine at Use antagonists H2 receptor site in caution in Ranitidine presence the gastric parietal of renal or GENERIC: cells, which inhibits hepatic gastric acid impairme Zantac secretion. nt. Monitor INDICATIONS: AST, ALT, serum Used in the creatinine management ; when of various use to gastrointestin prevent stress- al (GI) related GI disorders. bleeding, Prophylaxis of occult GI blood with hemorrhage GI bleeding, from stress monitor ulceration renal and in functionto patients at correct risk of dose; monitor developing for side acid effects aspiration Evaluate during results of general laboratory anaesthesia. tests, therapeuti c effectiven ess and adverse reaction. Assess knowledg e/teach patient appropriat e use, possible side effects / appropriat e interventi ons and adverse symptoms to report. Drug name Classification Action/Indication Dosage/Route/Fre Nursing considerations Evaluation quency
◦ history of improves with 12mgl/IM/ q12 Generic: Enters target cells underlying drug therapy. Dexamethasone and binds to specific condition before receptors therapy Patient has no 2nd dose: Monitor patients injury as a result ◦ weight and of drug therapy. 12mgl/IM/ q12 glucose level Initiating many (notify physician Patient and family complex reactions of weekly gain state that are responsible >5lbs. or understanding of for its anti- hyperglycaemia. drug therapy. inflammatory and Assess potassium immune-suppressive depletion or effects. hypokalemia; fatigue, nausea, Indications: vomiting, depression, dysrhythmias, weakness. Monitor cardiac status; blood pressure, edema, and chest pain. Assess carefully for infection because drug can mask infection; fever and WBC even after withdrawal of medication. Monitor input- output ratio, urinary output and increasing edema. Asssess mental status; affect, mood, behavioral, changes, aggression. Assess patient’s and family’s knowledge on drug therapy.