Professional Documents
Culture Documents
is a hollow tube, the upper 1/3 is - it begins before food enters the
composed of skeletal muscles, the rest is stomach
smooth muscle - is mediated by the vagus nerve,
lined with mucous membrane - secretes releasing acetylcholine which
mucoid substance for protection stimulates the parietal
the bolus of food arrives at the cardiac cells and chief cells to secrete
sphincter of the stomach w/in 5-10 secs acid, pepsin and mucus
after ingestion
the lower esophageal sphincter (LES) B. Gastric phase begins with the arrival of
prevents reflux of food in the stomach food in the stomach
back into the lower esophagus
STOMACH: Contains the cardia, fundus, the - distention of the stomach and
body, and the pylorus presence of digested proteins
stimulate gastrin hormone
a. Mucous glands secretion
mucous glands are located in the - Gastrin stimulates the parietal
mucosa
cells of the stomach to secrete
mucous glands prevent
HCL
autodigestion by providing an
- this phase continues for several
alkaline protective covering.
b. lower esophageal (cardiac) sphincter hours, until the acidity of gastric
prevents reflux of gastric contents into contents reaches pH of 1.5
the esophagus C. Intestinal Phase is stimulated by food
c. pyloric sphincter regulates the rate of
entering the duodenum
stomach emptying into the small
intestine - a substance similar to gastrin is
d. Hydrochloric acid kills microorganisms, released from the intestines it
breaks food into small particles, and stimulates gastric secretion of
provides a chemical environment that is pepsin and mucus
required by the gastric enzymes.
- when the pH in the duodenum or contraction of external anal sphincter
decreases (increase acidity) this (voluntary control)
results to release of Secretin
hormone - w/c inhibit gastric
PANCREATIC INTESTINAL JUICE ENZYMES
acid secretion and slows gastric
motility and gastric emptying 1. Amylase digests starch to Maltose
2. Maltase reduces maltose to
SMALL INTESTINE monosaccharides glucose
3. Lactase splits lactose into galactose and
2.5 cm (1 inch) wide and 6 meters (20 feet) long
glucose
– fills most of the abdomen
4. Sucrase reduces sucrose to fructose and
3 parts: glucose
5. Nucleuses split nucleic acids to
a. Duodenum - First (10 inches) of small
nucleotides
intestine which connects to the stomach
6. Enterokinase activates trypsinogen to
Receives chyme from the stomach
trypsin
through the pyloric sphincter
Secretion and digestion – major portion of
Fluids from the pancreas and gall
digestion occurs in the small intestines by the
bladder via the common bile duct
action of pancreatic and intestinal secretions
Manufactures intestinal juice (enzymes) and bile
Susceptible to inflammatory
processes a. Carbohydrate digestion start in the
Neutralizes the acidic rhyme from mouth Ptyalin — breakdown
the stomach polysaccharides to disaccharides
b. Jejunum - middle portion (8 feet long) - intestinal enzymes (maltase, lactase,
Absorption of Magnesium, sucrase)
Calcium, Iron Breakdown disaccharides to
c. Ileum - with connects to the large monosaccharides (glucose, galactose
intestine (12 feet long) fructose)
Chyme moves slowly towards the b. Protein digestion
ileocecal valve (3 - 10 hours) - start in the stomach pepsin –
LARGE INTESTINE breakdown of proteins to polypeptides
- small intestines trypsin – breakdown
1. The large intestine is about 5 feet long of polypeptides into peptides and amino
2. The large intestine absorbs water and acids
eliminates wastes c. Fat digestion
3. Intestinal bacteria play a vital role in the - fats require emulsification into small
synthesis of some B vitamins and vitamin droplets before it can be broken down
K. into glycerol and fatty acids
3 parts: Bile - from liver; emulsify fats so that it
a. cecum - which connects to the small could be broken down
intestines PERITONEUM
b. colon - 4 parts (ascending, transverse, 1. The peritoneum lines the abdominal
descending, sigmoid colon) cavity
c. rectum - 17-20 cm. (7-8 inches) long, 2. The peritoneum forms the mesentery
anal canal that supports the intestines and blood
4. The ileocecal valve prevents contents of supply.
large intestine from entering ileum LIVER
5. The anal sphincters guard the anal canal
Defecation reflex 1. The liver is the largest gland in the body,
weighing 3 to 4 lbs.
Feces enter the rectum and cause 2. The liver contains Kupffer's cells, which
distention of wall of the rectum > send remove bacteria in the portal venous
impulses to the sacral segment of the blood.
spinal cord - then back to the colon, 3. The liver removes excess glucose and
sigmoid and rectum > initiate relaxation amino acids from the portal blood
of the internal anal sphincter > relaxation 4. The liver synthesizes glucose, amine
acids and fats
5. The liver aids in the digestion of fats,
carbohydrates and proteins
6. The liver stores and filters blood (200 to
400 ml of blood stored)
7. The liver stores vitamins A, D and B and
iron.
8. The liver secretes bile to emulsify fats 90% of absorption occurs within the
(500 to 1000 ml of bile a day) small intestines by active transport or
diffusion
9. Hepatic Duct amino acids, monosaccharides, Na+, Ca+
The hepatic duct delivers bile to + are transported by active transport w/
the gallbladder via the cystic duct the expenditure or use of energy
the hepatic ducts deliver bile to GIT role in Fluid and Electrolyte Balance
the duodenum via the common
GIT secretions contain electrolytes
bile duct
severe fluid and electrolyte imbalance
the common bile duct opens into
may occur with excessive losses of
the duodenum, with the
gastrointestinal fluids
pancreatic duct at the ampulla of
Example:
Vater
the sphincter prevents the reflux Na+ and K+ deficits: vomiting, diarrhea,
of intestinal contents into the gastric suctioning, intestinal fistula
common bile duct and pancreatic Ca++ & M9++ deficits: malnutrition, mal-
duct. absorption, intestinal fistula
Detoxifies ammonia into urea Metabolic alkalosis: loss of gastric acid by
GALLBLADDER suctioning or persistent vomiting
Metabolic acidosis: loss of bicarbonate-
The gallbladder stores and
rich intestinal secretions by severe
concentrates bile
diarrhea or fistula
The gallbladder contracts to force
Other functions of the GIT
bile into the duodenum during the
digestion of fats the GIT supports bacterial growth and
The cystic duct joins the hepatic has a role in antibody formation
duct to form the common bile duct intestinal bacteria synthesize Vit. K
The sphincter of Oddi guards the required for production of clotting
entrance into the duodenum factors 11 (Prothrombin), VII, IX, X
The presence of fatty materials in ASSESSMENT OF GIT
the duodenum stimulates the
Nursing History: Subjective Data
liberation of cholecystokinin,
which causes contraction of the General Data
gallbladder and relaxation of the a. presence of dental prosthesis, comfort of
sphincter of Oddi usage
PANCREAS b. difficulty eating or digesting food
c. nausea or vomiting
EXOCRINE GLAND
d. weight loss
a. the pancreas secretes sodium e. pain - may be caused by distention or
bicarbonate to neutralize the acidity of sudden contraction of any part of the GIT
the stomach contents that enter the
- specify the area,
duodenum. b. Pancreatic juices contain
describe the pain
enzymes for digesting carbohydrates,
fats, and proteins. Specific data if symptoms are Present
ENDOCRINE GLAND a. situations or events that effect
symptoms
a. the islets of Langerhans secrete insulin.
b. onset, possible cause, location, duration,
b. insulin secreted into the bloodstream
character of symptoms
and is important for carbohydrate
c. relationship of specific foods, smoking or
metabolism
alcohol to severity of symptoms
c. the pancreas secretes glucagon to raise
d. how the symptoms were managed
blood glucose levels
before seeking medical help
d. the pancreas secretes somatostatin to
Normal pattern of bowel elimination
exert a hypoglycemic effect
a. frequency and character of stool
b. use of laxatives, enemas
ABSORPTION
> white patches - due to candidiasis (oral thrush) to determine size of liver, spleen, uterus,
kidneys - if enlarged\
> white plaques w/in red patches may be determine presence and character. of
malignant lesions abdominal masses determine degree of
tenderness and muscle rigidity (rebound
5. tongue- color, mobility, symmetry,
or direct
ulcerations / lesions or nodules 6.
pharynx - observe the uvula, soft palate, RECTUM
tonsils, posterior pharynx
- signs of inflammation (redness, perineal skin and perianal skin
edema, ulceration, thick yellowish assess for presence of pruritus, fissures,
secretions), assess also for external hemorrhoids, rectal prolapse
symmetry of uvula and palate 3 Phases of Diagnostic testing
ABDOMEN Pretest: Client preparation
- assess for the presence or absence of Intra-test: specimen collection and VS monitoring
tenderness, organ enlargement, masses, spasm
or rigidity of the abdominal muscles, fluid or air Post-test: Monitoring and follow-up nursing care
in the abdominal cavity
Related Nursing Diagnoses
Anatomic Location of Organs
Anxiety
RUQ liver, gallbladder, duodenum, right kidney, Fear
hepatic flexure of colon Impaired physical mobility
Deficient knowledge
RLQ cecum, appendix, right ovary and fallopian
tube Stool Analysis
- assess the skin for color, texture, scars, striae, FECAL ANALYSIS
engorged veins, visible peristalsis (intestinal
Fecal Occult Blood Test (FOBT)
obstruction), visible pulsations (abdominal
aorta), visible masses (hernia) Detects GI bleeding
↑ Fiber diet 48-72 hours
- assess contour (flat, protuberant, globular)
(+) colon cancer and other forms of cancer Client may resume normal activities.
Refrigerate gastric samples if not tested
CEA - recurrence or spread of tumor within 4 hours.
effectiveness of therapy
Avoid Heparin
GI TRACT VISUALIZATION
Exfoliative Cytology
RADIOLOGY AND IMAGING STUDIES
Detect malignant cells
UPPER GASTROINTESTINAL SERIES AND SMALL
Liquid diet
BOWEL SERIES (Barium swallow)
UGI: NGT insertion - saline lavage
1. Upper GI series and small-bowel series administered after the patient is given an
are fluoroscopic x-ray examinations of enema of barium sulfate.
the esophagus, stomach, and small 2. Can visualize structural changes, such as
intestine after the patient ingests barium tumors, polyps, diverticula, fistulas,
sulfate. obstructions, and ulcerative colitis.
2. As the barium passes through the GI 3. Air may be introduced after the barium
tract, fluoroscopy outlines the GI mucus to provide a double-contrast study.
and organs.
Nursing and patient care considerations
3. Spot films record significant findings.
4. Double-contrast studies administer 1. Explain to the patient:
barium first followed by a radio lucent A. What the x-ray procedure
substance, such as air, to produce a thin involves.
layer of barium to coat the mucosa. B. That proper preparation
provides a more accurate view
This allows for better visualization of any type of
of the tract and that
lesion.
preparations may vary.
Nursing and patient care considerations
C. That it is important to retain the
1. Explain procedure to patient. barium so all surfaces of the
2. Instruct patient to maintain low-residue tract are coated with opaque
diet for 2 to 3 days before test and a solution.
clear liquid dinner the night before the 2. Instruct the patient on the objective of
procedure. having the large intestine as clear of fecal
3. Emphasize NPO after midnight before material as possible:
the test. A. The patient may be given a low-
4. Encourage patient to avoid smoking, residue, low-fat diet, 1 to 3 days
alcohol, caffeine before the test. before the examination.
5. Explain that the health care provider may B. The day before the examination,
prescribe all narcotics and intake may be limited to clear
anticholinergics to be held 24 hours liquids (no drinks with red dye).
before the test. C. The day before the examination,
6. Tell the patient that he or she will be an oral laxative, suppository,
instructed at various times throughout and/or cleansing enema may be
the procedure to drink the barium (480 prescribed
to 600 mL). 3. The patient will be on NPO after
7. Explain that a cathartic will be prescribed midnight the day of procedure.
after the procedure. 4. An enema or cathartic may be ordered
8. Instruct the patient that stool will be after the barium enema.
light in color for the next 2 to 3 days 5. Inform the patient that barium may
from the barium. cause light-colored stools for several
9. Instruct patient to notify health care days after the procedure.
provider if he or she has not passed the
Git Visualization
barium in 2 to 3 days
10. Note that water-soluble iodinated Barium Enema- LGIS
contrast agent (such as Gastrografin)
may be used for a patient with a Pretest: Informed consent, NPO the night, enema
suspected perforation or colonic the morning
obstruction. Intratest: position on left side, administer enema,
GIT Visualization then X-ray follow
- X-ray visualization of the gallbladder after Under fluoroscopy, the bile duct is
administration of contrast media intravenously entered percutaneously and injected
with a dye to observe filling of hepatic
Pre-test: Allergy to iodine and sea- foods
and biliary ducts
Intra-test: ensure patent IV line
Paracentesis
Post-test: increase fluid intake to flush out the
dye, Assess for delayed hypersensitivity reaction Transabdominal removal of fluid from
to the dye like chills and N/V the peritoneal cavity for analysis.
Risk Factors
Alternative Feeding:
Indications of NGT:
NG TUBE
USE GRAVITY
BE SURE TO ASPIRATE