This document discusses common health problems in toddlers, including poisoning from ingestion of household products. It outlines signs and symptoms of poisoning from corrosives, hydrocarbons, and salicylates. The emergency treatment section describes assessing the victim, terminating exposure, identifying the poison, and contacting poison control. Child maltreatment is also discussed, defining physical and sexual abuse as well as risk factors for abusers and victims.
This document discusses common health problems in toddlers, including poisoning from ingestion of household products. It outlines signs and symptoms of poisoning from corrosives, hydrocarbons, and salicylates. The emergency treatment section describes assessing the victim, terminating exposure, identifying the poison, and contacting poison control. Child maltreatment is also discussed, defining physical and sexual abuse as well as risk factors for abusers and victims.
This document discusses common health problems in toddlers, including poisoning from ingestion of household products. It outlines signs and symptoms of poisoning from corrosives, hydrocarbons, and salicylates. The emergency treatment section describes assessing the victim, terminating exposure, identifying the poison, and contacting poison control. Child maltreatment is also discussed, defining physical and sexual abuse as well as risk factors for abusers and victims.
Care of Toddlers with Problem Severe burning pain in the mouth, throat,
COMMON HEALTH PROBLEMS IN TODDLER stomach
1. Ingestion of injurious agents most cases White, swollen, and/or ulcerated mucous membranes; edema of lips, tongue, pharynx occurring in children under 6 years of age (respiratory obstruction) MOST COMMONLY INGESTED POISONS: Violent vomiting, hemoptysis Drooling and inability to clear secretions Cosmetics and personal care products (perfume, Signs of shock cologne, aftershave) Anxiety and agitation Cleaning products Analgesics 2. HYDROCARBONS
Alterations in sensorium; weakness Foreign bodies, toys, miscellaneous Tachypnea, cyanosis, retractions, grunting Arts, crafts, and office supplies 3. SALICYLATES Hydrocarbons Nausea, vomiting, disorientation, dehydration, COMMON SIGNS OF POISONING diaphoresis, hyperpnea, hyperthermia, oliguria, tinnitus, coma, seizures *GENERAL SIGNS 1. GASTOINTESTINAL EMERGENCY TREATMENT - abdominal pain 1. ASSESS THE VICTIM -vomiting Take vital signs; reevaluate regularly -diarrhea Initiate cardiorespiratory support if needed -anorexia Treat other symptoms, such as seizure 2. TERMINATE EXPOSURE RESPIRATORY AND CIRCULATORY SYSTEMS Empty mouth of pills, plant parts, or other Depressed respirations material Labored respirations Flush eyes continuously with normal saline for Unexplained cyanosis 15 to 20 minutes Signs of shock Flush skin and was with soap and a soft cloth; Delayed capillary refill remove contaminated clothes Increased, weak pulse Bring victim of an inhalation poisoning into Decreased blood pressure fresh air Increased, shallow respiration Give one sip of water to dilute ingested poison Pallor 3. IDENTIFY THE POISON Cool, clammy skin Question the victim and the witnesses CENTRAL NERVOUS SYSTEM Look for environmental clues Be alert to signs and symptoms of ocular or Seizures dermal exposure Overstimulation Loss of consciousness 4. CALL POISON CONTROL CENTER FOR IMMEDIATE ADVICE Dizziness REGARDING TREATMENT Stupor, lethargy Coma CHILD MALTREATMENT Specific Signs it includes intentional physical abuse or 1. COROSIVE neglect, emotional abuse or neglect, and sexual abuse of children, usually by adults it is one of the significant social problems Problems of divorce, poverty, unemployment, affecting children poor housing, frequent relocation, alcoholism, most sexual abuse is committed by family and drug addiction members or by individuals who are close to Lower socioeconomic population the child TYPES OF SEXUAL ABUSE INCEST- any physical sexual activity between THE CAPTA DEFINITON family members the CAPTA(Child Abuse Prevention and MOLESTATION- “indicent liberties’ such as Treatment Act), defines child abuse and touching, fondling, kissing, single or mutual neglect as, at a minimum, any recent act or masturbation, or oral-genital contact failure to act that results in imminent risk of EXHIBITIONISM- indecent exposure, usually death, serious physical or emotional harm, exposure of the genitalia by an adult to sexual abuse, or exploitation of a child by a children parent or caretaker who is responsible for the child’s welfare. CHILD PORNOGRAPHY- arranging and photographing sexual acts involving children, FOUR MAJOR TYPES OF MALTREATMENT: either alone or with adults or animals, regardless of consent by the child’s legal PHYSICAL ABUSE- infliction of physical injury as a guardian result of punching, beating, kicking, biting, burning, CHILD PROSTITUTION- involving children in a shaking, or otherwise harming a child sex act for profit and usually with changing partners CHILD NEGLECT- characterized by failure to provide PEDOPHILIA- “love of child” the sexual for the child’s basic needs-physical, educational or preference for prepubertal children as the means emotional) of achieving sexual excitement PHYSICAL NEGLECT -deprivation of basic necessities CHARACTERISTIC OF ABUSERS AND VICTIMS ABUSER EMOTIONAL NEGLECT- failure to meet the child’s need for affection, attention and emotional Typical abuser is a male that the victim knows nurturance Are in position that involve working closely with SEXUAL ABUSE-includes fondling a child’s genitalia, children intercourse, incest, rape, sodomy, exhibitionism, and commercial exploitation through prostitution or production of pornographic materials VICTIM EMOTIONAL ABUSE- includes acts or omissions by the parents or other caregivers that have caused, or could Tend to be runaways-young adolescents who cause, serious behavioral, cognitive, emotional or engage in prostitution and pornography to obtain mental disorders money for food Incentuous relationship between father or FACTORS PREDISPOSING TO PHYSICAL ABUSE stepfather and daughter are generally prolonged and victims are reluctant to report the situation Parental characteristics because they fear of retaliation and that they will Abusive parents mostly have low self-esteem not be believed and less adequate maternal functioning METHODS USED TO PRESSURE CHILDREN INTO Tend to have difficulty controlling aggressive SEXUAL ACTIVITY impulses, and the free expression of violence The child is offered gifts or privileges Characteristics of the child The adult misrepresents moral standards by Occasionally, the abuse child is illegitimate, telling the child that it is “okay to do this” unwanted, brain damage, hyperactive or Isolated and emotionally and socially physically disabled impoverished children are enticed by adults Environmental characteristics who meet their needs for warmth and human Burns contact Fracture or dislocation The successful offender pressures the victim Lacerations into secrecy regarding the activity by describing CHARACTERISTICS OF SEXUAL ABUSE it as a “secret between us” that other people may take away if they find out Torn or bloody underclothing The offender plays on the child’s fears, Bruises, bleeding, lacerations of external including fear of punishment by the offender, genitalia, anus, mouth, or throat fear of repercussions if the child tells, and fear Genital discharge or odor of abandonment or rejection by the family Recurrent urinary tract infection NURSING INTERVENTIONS FOR THE MALTREATED WARNING SIGNS OF ABUSE CHILD Physical evidence of abuse or neglect, including A thorough physical examination and a careful, detailed previous injuries history are the diagnostic tools needed to identify abuse Conflicting stories about the “accident” or injury from the parents or others Observe child for physical and behavioral Injury blamed on the sibling or other party evidence of abuse An injury inconsistent with child’s development Report suspicions to appropriate authorities level Assist in removing child from unsafe A complaint other than the one associated with environment signs of abuse Refer family to social agencies Inappropriate response of caregiver, such as an Collaborate with multidisciplinary team exaggerated or absent emotional response, Keep factual, objective records refusal to sign for an additional tests or to agree Be aware of signs for continued abuse or to necessary treatment, excessive delay in neglect seeking treatment, absence of the parents for Help parents identify circumstances that questioning precipitate an abuse act Inappropriate response of the child, such as little or no response to pain, fear of being CEREBRAL PALSY touched, excessive or lack of separation anxiety, indiscriminate friendliness to strangers -neurologic disorders characterized by early onset Child’s report of physical or sexual abuse and impaired movement and posture Previous reports of abuse in the family Repeated visits to emergency facilities with -it is non progressive and may be accompanied by injuries perceptual problems, language deficits, and intellectual involvement
CHARACTERISTICS OF PHYSICAL NEGLECT CAUSES OF CEREBRAL PALSY
Failure to thrive 1. PRENATAL
Malnutrition Maternal Poor hygiene - DM or hyperthyroidism Poor health care Frequent injuries -exposure to radiation or toxins
CHARACTERISTICS OF EMOTIONAL NEGLECT Gestational
Failure to thrive -chromosome abnormalities
Enuresis -genetic syndromes Sleep disorders -teratogens 2.LABOR AND DELIVERY CHARACTERISTICS OF PHYSICAL ABUSE -premature deliver Bruises -prolonged rupture of membranes Stiff or rigid arms or legs Pushing away or arching back -Abnormal presentation Floppy or limp body posture 3. PERINATAL Cannot sit without support at 8 months Uses only one side of the body -sepsis or CNS infection Clenched hands after 3 months - seizures Persistence of primitive reflexes past 6 months Hand preference demonstrated before 18 -meconium stain months 4. CHILDHOOD/POSTNATAL Leg scissoring Seizures -brain injury Sensory impairment (hearing, vision) -meningitis BEHAVIORAL CHANGES
-traumatic brain injury Extreme irritability or crying
CLINICAL CLASSIFICATION OF CEREBRAL PALSY Feeding difficulties after 6 months of age, 1. SPASTIC- may involve one or both sides persistent tongue thrusting Little interest in surroundings Hypertonicity with poor control of posture, Excessive bleeding balance and coordinated motion Impairment of fine and gross motor DIAGNOSTIC EVALUATION The neurologic examination and history are the primary modalities for diagnosis Types of Spastic Cerebaral Palsy MRI (Magnetic Resonance Imaging) Hemiparesis- one side of the body affected Metabolic and genetic testing is recommended Quadriparesis-(or tetraparesis)- all four if no structural abnormality is identified by extremities involved, lower affected more than neuroimaging the upper limb Diplegia –similar parts on both sides of the THERAPEUTIC INTERVENTIONS FOR CEREBRAL body involved, such as both arms PALSY Monoplegia- involving only one extremity Triplegia- involving three extremities PHYSICAL THERAPY Paraplegia- pure cerebral paraplegia of lower Orthotic devices (braces, splints, casting) extremities
2. DYSKENETIC OR ATHETOID-abnormal involuntary ADAPTIVE EQUIPMENT
movement Scooters, bicycles, wheelchairs, boards, and Athetosis, characterized by slow, wormlike, tricycles writhing movements that usually involve the OCCUPATIONAL THERAPY extremities, trunk, neck, facial muscles, and tongue Adaptive equipment utensils for functional use (eating, writing) 3. ATAXIC switches, computers Wide based gait, rapid, repetitive movement SPEECH LANGUAGE THERAPY performed poorly Oral-motor skills 4. MIXED TYPE OR DYSTONIC- combination of spasticity and Adaptive communication techniques athetosis SPECIAL EDUCATION Early intervention programs POSSIBLE SIGNS OF CEREBRAL PALSY Specialized learning programs and support services PHYSICAL SIGNS in school Socialization to promote self-concept development Poor head control after 3 months of age Source: primary secretions of respiratory tract of infected SPECIAL EDUCATION persons; to a lesser degree; skin lesions (scabs not infectious) Early intervention programs Specialized learning programs and support services Transmission: direct contact, droplet (airborne) spread, in school and contaminated objects Socialization to promote self-concept Incubation: 2-3 weeks, usually 14-16 days development Period of communicability: probably 1 day before NURSING INTERVENTIONS/ MANAGEMENT eruption of lesions to 6 days after the first crop vesicles Reinforce Therapeutic Plan and Assist in when crusts have formed Normalization Assist the family in devising and modifying Clinical manifestations: equipment and activities to continue the therapy program in the home Prodromal stage: slight fever, malaise Address health maintenance needs and anorexia for first 24 hours, rah highly More frequent rest periods should be arranged pruritic, begins as macule, rapidly progresses to Diet should be tailored to the child’s activity papule and the vesicles; all 3 stages present at and metabolic needs (eg. Gastrostomy feeding) varying degrees at 1 time Advice parents on how to administer Distribution: centripetal, spreading to medication through gastrostomy tube face and proximal extremities but sparse on Safety precautions are implemented such as distal limbs and less on areas not exposed to having helmet if the child is subject to falls heat Support family THERAPEUTIC MANAGEMENT Help family cope with emotional aspects SPECIFIC: Antiviral agent Acyclovir (Zovirax); Acknowledging and addressing their concerns varicella zoster immunoglobulin (VZIG) after and frustrations and by noting and appreciating exposure in a high risk children their problem-solving skills and their SUPPORTIVE: diphenhydramine hydrochloride approaches to helping the child or antihistamines to relieve itching Find help and support from parent groups, where they can share their experiences, COMPLICATIONS accomplishments, problems, and concerns Support hospitalized child Secondary bacterial infections (eg. Pneumonia) Should be approached and treated in the same Encephalitis manner as any child in the hospital Varicella pneumonia Therapy program should be continued while Hemorrhagic varicella they are hospitalized Maintain strict isolation, until vesicles have Encourage parents to room-in and actively dried participate in the child’s care facilitates a continuation of the home therapy program and NURSING MANAGEMENT helps the child adjust to unfamiliar Administer skin care; bath and change clothes environment and linen daily; apply calamine lotion; keep INFECTIOUS DISEASES fingernails short and clean An illness caused by a specific infectious agent Keep child cool (may decrease the number of or its toxic products through a direct or lesions) indirect mode of transmission that agent from Teach child to apply pressure on the pruritic a reservoir area rather than scratching it Reason with the child regarding danger of scar Chicken pox (Varicella) formation from scratching DIPTHERIA Agent: Corynebacterium diptheriae Agents : Varicella-zoster virus (VZV) Source: discharges from mucous membranes PRODROMAL STAGE: fever and malaise, of nose and nasopharynx, skin and other followed in 24 hour by coryza, cough, lesions of infected person conjunctivitis, Koplik spots ( small, irregular red Transmission: direct contact with infected spots with a minute, bluish white center first person, carrier, or contaminated articles seen on buccal mucosa opposite molars 2 days Incubation period: usually 2-5 days, possibly before rash) longer RASH: appears 3-4 days after onset of Period of communicability: variable, until prodromal stage; begins as erythematous virulent bacilli are no longer present (3 maculopapular eruption on face and gradually negative cultures);usually 2 wks but as long as 4 spreads downward; after 3-4 days assumes week brownish appearance THERAPEUTIC MANAGEMENT CLINICAL MANIFESTATIONS NASAL: resembles common colds; Vitamin A supplementation serosaguineous mucopurulent nasal discharge Bedrest during febrile period TONSILLAR/PHARYNGEAL: malaise, anorexia; Antibiotics to prevent secondary bacterial sore throat; low-grade fever,; in severe cases infection septic shock Complications: otitis media, pneumonia, LARYNGEAL: Fever, hoarseness, cough, bronchiolitis, obstructive laryngitis, encephalitis potential airway obstruction, dyspnea, retractions, cyanosis NURSING MANAGEMENT THERAPEUTIC MANAGEMENT Antitoxin (IV); preceded by skin or conjunctival test to rule out sensitivity to horse serum Isolate child until 5th day of rash Antibiotics (penicillin or erythromycin) Maintain bed rest Complete bed rest FEVER: instruct parents to administer Tracheostomy for airway obstruction antipyretics Agent: paramyxovirus Complications: myocarditis (2nd week), neuritis Source: Saliva of infected persons NURSING MANAGEMENT Transmission: direct contact with or droplet Maintain strict isolation spread from infected person Bed rest Incubation period: 14-21 days Use suctioning as needed Period of communicability: before and after Observe respirations for signs of obstruction swelling begins Administered humidified oxygen as needed EYE CARE: dim light if photophobia is present; clean eyelids with warm saline solution to MEASLES (RUBEOLA) remove secretions Agent: virus CORYZA/COUGH: use cool-mist vaporizer; protect skin around nares with layer of Source: respiratory tract secretions, blood, and urine petrolatum; encourage fluids and soft, bland of infected person foods Transmission: usually by direct contact with droplets SKIN CARE: keep skin clean, use tepid bath as of infected person necessary Incubation period: 10-20 days MUMPS Agent: paramyxovirus Period of ommunicability:; from 4 days before to 5 Source: Saliva of infected persons days after rash appears, but mainly during Transmission: direct contact with or droplet prodromal stage spread from infected person Incubation period: 14-21 days CLINICAL MANIFESTATIONS Period of communicability: before and after swelling begins CLINICAL MANIFESTATIONS Antimicrobial therapy (erythromycin) PRODROMAL: fever, headache, malaise and anorexia for Bed rest 24 hr, followed by “earache” that is aggravated by Increase oxygen intake and humidity chewing Adequate fluids Intubation possibly PAROTITIS: by 3rd day, parotid gland enlarges and Complications: pneumonia, atelectasis, otitis reaches maximum size in 1-3 days, accompanied by media, seizures, hemorrhage (epistaxis), weight pain and tenderness loss and dehydration, hernia, prolapsed rectum THERAPEUTIC MANAGEMENT NURSING MANAGEMENT Analgesics for pain and antipyretics for fever Isolate the child during catarrhal stage IV therapy for child who refuses to drink or maintain bed rest vomits encourage fluids Complications: sensoneural deafness, provide high humidity, suction gently but postinfection encephalitis, myocarditis, often to avoid choking on secretions arthritis, hepatitis, meningitis, sterility in men observe for signs of airway obstruction NURSING MANAGEMENT Isolate child during period of communicability POLIOMYELITIS Maintain bed rest Agent: Enterovirus Give analgesics for pain Encourage fluids and soft, bland foods; avoid Source: feces and oropharyngeal secretions of foods requiring chewing infected persons Apply hot or cold compresses to the neck Transmission: direct contact with apparent or To relieve orchitis, provide warmth and local inapparent active infection; spread via fecal-oral support with tight-fitting and pharyngeal-oropharyngeal routes Incubation period: 7-14 days, with range of 5-35 days PERTUSSIS (WHOOPING COUGH) Period of Communicability: virus persist about 1 week in throat and about 4-6 weeks in feces Agent: Bordetella pertussis Clinical Manifestations Source:discharge from respiratory tract of infected person -ABORTIVE OR INAPPARENT- fever, uneasiness, sore Transmission: direct contact or droplet spread from throat, headache, anorexia, vomiting, abdominal infected person; indirect contact with freshly pain; last a few hours to a few days contaminated articles -NONPARALYTIC- more severe than abortive, with Incubation period: 6-20 days, usually 7-10 days pain and stiffness in neck, back, and legs Period of communicability: greatest during -PARALYTIC- initial course similar to nonparalytic catarrhal stage before onset of paroxysm type, followed by recovery and then signs of CLINICAL MANIFESTATIONS central nervous system paralysis CATARRHAL STAGE: begins with symptoms of THERAPEUTIC MANAGEMENT upper respiratory tract infection, such as SUPPORTIVE TREATMENT: CBR during acute phase coryza, sneezing, lacrimation, cough and low- Assisted respiratory ventilation in case of grade fever; continue for 1-2 weeks, when dry respiratory paralysis hacking cough become more severe Physical therapy for muscles after acute stage PAROXYSMAL STAGE: cough most often occurs at night and consists of short, rapid coughs followed by sudden inspiration associated with RUBELLA (GERMAN MEASLES) high pitched crowing sound or “whoop”;cheeks Agent: rubella virus becomes flushed or cyanotic, eyes bulge, and Source: nasopharyngeal secretions of person tongue protrudes, last for 4-6 weeks with apparent or inapparent infection; virus also THERAPAETIC MANAGEMENT present in blood, stool and urine Incubation period: 14-21 days Period of communicability: 7 days before to Clinical Manifestations about 5 days after appearance of rash PRODROMAL STAGE: abrupt high fever, increased pulse, vomiting, headache, chills, malaise and Complications: permanent paralysis, respiratory arrest, abdominal pain hypertension, kidney stones due to prolonged ENANTHEMA: tonsils enlarged, edematous, immobility reddened, and covered with patches of exudates; in severe case, pharynx is edematous and beefy CLINICAL MANIFESTATION red; during the first 1-2 days tongue is coated and papillae become red and swollen(white PRODROMAL STAGE: Absent in children, present in strawberry tongue); 4th to 5th day white coat adults and adolescents; consist of low grade sloughs off, leaving prominent papilla (red fever, headache, malaise, anorexia, mild strawberry tongue); palate covered with lesions conjunctivitis, coryza, sore throat, cough and lymphadenopathy; last 1-5 days, subsides 1 day EXANTHEMA: rash appearing within 12 hours, red after appearance of rash pinhead-sized punctuate lesions rapidly becoming generalized but absent on face, which RASH: first appears on face and rapidly spreads becomes flushed with striking circumoral pallor, downward to neck, arms, trunk, and legs; by end rash more intense in folds of joints of first day, body covered with discrete, pinkish red maculopapular exanthema; disappears in THEREPAEUTIC MANAGEMET same order it began and is usually gone by 3rd Full course of penicillin day Antibiotic therapy for newly diagnosed carriers THERAPEUTIC TREATMENT No treatment necessary other than antipyretics for Bed rest low-grade fever and analgesics for discomforts Analgesics for sore throat Complications: rare, greatest danger is teratogenic Complications: otitis media, peritonsillar and effect on fetus retropharyngeal abscess, sinusitis, Nursing management glomerulonephritis, carditis, polyarthritis Reassure parents of benign nature of illness NURSING INTERVENTION Use comfort measures as necessary Institute respiratory precautions until 24 hrs after initiation of treatment Isolate child from pregnant women Ensure compliance of antibiotic therapy Maintain bed rest Encourage fluids SCARLET FEVER Agent: Group A B-hemolytic streptococci Source: from nasopharyngeal secretions of infected persons and carriers Transmissions: direct contact with infected person or droplet spread; indirectly by contact with contaminated articles or ingestion of contaminated milk or other food Period of communicability: approximately 10 days, during first 2 weeks of carrier phase, although may persist for months Incubation period: 2-5 days, with range of 1-7 days