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  • Turun: Sekresi Protein Terganggu Sekresi Eritropoitis Turun Retensi Na

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    Naniandayani12
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    The document discusses the pathophysiology of kidney failure. It notes that kidney failure can be caused by things like toxic substances, blood vessel obstructions, inflammation/injury, large kidney stones, and retained urine. This can lead to decreased blood supply to the kidneys, lowered glomerular filtration rate, and impaired protein secretion and sodium retention among other things. Ultimately, this causes a buildup of waste products in the blood and issues with fluid, electrolyte, and acid-base balance in the body.

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    Pat ttt

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    Patoflo CKD 12

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    The document discusses the pathophysiology of kidney failure. It notes that kidney failure can be caused by things like toxic substances, blood vessel obstructions, inflammation/injury, large kidney stones, and retained urine. This can lead to decreased blood supply to the kidneys, lowered glomerular filtration rate, and impaired protein secretion and sodium retention among other things. Ultimately, this causes a buildup of waste products in the blood and issues with fluid, electrolyte, and acid-base balance in the body.

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    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    9 views1 page

    Turun: Sekresi Protein Terganggu Sekresi Eritropoitis Turun Retensi Na

    Uploaded by

    Naniandayani12
    The document discusses the pathophysiology of kidney failure. It notes that kidney failure can be caused by things like toxic substances, blood vessel obstructions, inflammation/injury, large kidney stones, and retained urine. This can lead to decreased blood supply to the kidneys, lowered glomerular filtration rate, and impaired protein secretion and sodium retention among other things. Ultimately, this causes a buildup of waste products in the blood and issues with fluid, electrolyte, and acid-base balance in the body.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
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    of 1

    infeksi vaskuler zat toksik Obstruksi saluran kemih

    arteriosklerosis iritasi / cidera


    reaksi antigen tertimbun ginjal batu besar jaringan
    retensi urin
    antibodi dan kasar
    suplai darah ginjal
    turun
    hematuria
    menekan saraf perifer
    GFR turun

    nyeri pinggang anemia


    GGK

    sekresi protein terganggu retensi Na sekresi eritropoitis turun

    resiko suplai nutrisi dalam


    sindrom uremia urokrom produksi Hb turun
    tertimbun di kulit
    total CES naik gangguan nutrisi darah turun

    perpospatemia oksihemoglobin turun


    gang.
    tek. kapiler
    keseimbangan
    asam - basa perubahan naik gangguan intoleransi
    suplai O2 kasar
    warna kulit perfusi jaringan aktivitas
    pruritis turun
    vol. interstisial
    prod. asam naik naik payah jantung bendungan atrium
    gang. COP turun kiri kiri naik
    integritas as. lambung naik
    kulit edema
    (kelebihan volume aliran darah suplai O2 tek. vena pulmonalis
    iritasi lambung cairan) jaringan turun suplai O2 ke
    nausea, ginjal turun
    otak turun
    vomitus kapiler paru
    preload naik RAA turun metab. naik
    infeksi perdarahan anaerob syncope
    resiko (kehilangan edema paru
    retensi Na
    gangguan beban jantung kesadaran)
    gastritis & H2O
    nutrisi - hematemesis naik timb. as. laktat
    naik
    - melena naik gang. pertukaran
    gas
    mual, muntah hipertrofi kelebihan vol.
    ventrikel kiri - fatigue intoleransi
    anemia cairan - nyeri aktivitas
    sendi

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