Epinephrine

Adrenal glands will

produce adrenaline

binds in
the adrenergic
receptors

beta 1 adrenergic beta 2 adrenergic

receptor receptor

Increase cardiac
Vasa dilatory
output
 Cardiac Arrest

If treated: If not treated:

Artificial manual
Cardiac Pulmonary
breathing bag (Ambu
resuscitation Atrial Fibrillation Ventric
Bag)

deliver positive
pressure ventilation He
If not successful
w

aids in respiration Sinoatrial (SA) node

doesn't send out Vent
Defibrillation
correct electrical in
impulses

Increase gas
alpha 1 adrenergic exchange
receptors Decrea
If successful If not successful bloo

Good prognosis

Vasoconstriction Electrical pulses in No electrical pulses in

Sudden
the heart will be the heart will be
c
restored. restored.
 COVID 1

Predisposing Factors
- age
-sex
-familial tendncies
-race

Extreme Age Familial tendencies Sex

Vic

less protective
Dizzines Weak immune system mechanism for male
sex hormones

cular Fibrillation
Alpha Beta Gamma Delta

eart rhythm
will change Cough

Lodges in the
pharynx and initiates Lack of production of
Airway irritation
inflammatory immune cells
tricles pump
response
efficiently Increase in
Sore throat inflammatory
cytokines triggers
Neutrophils move to immune response
Alveolar Capillary lungs release reactive Poor Oxygenation
ases amount of Damage oxygen species and process
cytokines Cytokines induce the
od pumped hypothalamus to
release
prostaglandins
Fluid accumulates in
interstitium and Electrolyte imbalance
n stop of blood alveoli
irculation Increase body
temperature set point
9

Precipitating Factors
-History of travel
- vices
- hygiene
- environment
-lifestyle
-economical crisis

Poor Environmental Poor Hygiene

ces Economical Crisis History of Travel
Sanitation Practices

poor sanitation lack of resources

Poor Health
Maintenance

World Wide
Pandemic

Susceptible diseases

Fever
 increase in vascular
resistance and blood
pressure

Good prognosis
Restoration of heart No restoration of
rythm heart rhythm

Lost of blood flow

Good prognosis
towards the brain

Decreased Oxygen in
brain

Brain disfunction Unconscious

Comatose Brain Damage

Pons and medulla

oblongata stops
functioning.

Apnea

Death
 Increase in distance to fight infection
for oxygen to diffuse
from alveoli to Viral infection via
Death capillary ACE2 and TMPRSS2

Low sodium and

potassium
Difficulty of breathing ACE2 down
regulation

Reduction of in
cardiac re polarization
reserve
Acute Respiratory Decreased activation
Distress Syndrom of the mTOR with
increased autophagy

Intestinal dysbiosis Diarrhea

If treated If not treated

Identify and treat

Sepsis
underlying causes

Ventilatory support Blood clotting

Restore If treated
hemodynamic Pulmonary emboli
function tachyarrythmias

Prevent complications severe

by administering Pneumothorax
medications Defibrillation Perman
Procainamide
peacema

Pulmonary
Ensure adequate
fibrosis/Lung scarring
nutrition
and stiffness The heart's electrical
blocks electrolytes Sends electri
pulses will be
channels to hea
restored.

Prognosis: Good Respiratory failure

Decrease myocardial
Heart rhythm re- stables hear
excitability and slower
establish a normal
conduction velocity

Oxygen toxicity
 Impairs body
functions (muscle
contractions and fluid
regulation)

Arrhythmia

If left untreated

Abnormal cardiac
nent signals
aker

Decrease heart rate.

Decrease blood
ical pulse supply.
art Decrease O2 output

rt beat at
l rate
Hypoxemia Hypoxia
 Vasodilate
Organ failure

Cardiovascular
Good prognosis
problems

Cognitive dysfunction

Death

Prognosis: Bad
Damages brain, liver,
and other organs

Death