CARDIOVASCULAR RISK OF SMOKING AND THE BENEFITS OF QUITTING

NAME:
DAVID ALBERTO JONES PARDO

CODE:
01200021024

PRACTICE ICB

UNIVERSIDAD DE SANTANDER, BUCARAMANGA

INSTRUMENTACION QUIRURGICA
2.023
As we well know worldwide, cardiovascular diseases are an important factor that prevails in
morbidity and mortality, in this case in the United States it affects many racial and ethnic groups,
this public health problem has generated a very high cost that is estimated to be around of the
$200 billion dollars annually, in health services, medicines and productivity losses and this is due
to the lack of development is the implementation of prevention strategies and deficient control of
risk factors in cardiovascular diseases, according to To the different studies it is estimated that
smoking determines 10% of cardiovascular diseases, tobacco consumption causes approximately
6 million deaths a year worldwide, this study has also shown that it is not only for those people
who are active smokers, this affects directly or exposure to secondhand smoke weakened the
myocardium and coronary arteries, this increase in coronary diseases represents 30% of the
population
Tobacco smoking has also been shown to have a causal relationship with many cardiovascular
disease phenotypes and is primarily associated with early-onset atherosclerosis primarily in
youth and adults, which increases the risk of myocardial infarction and stroke. It's because
cigarettes are made with more than 7,000 chemical compounds of many different classes,
including at least 72 cancer-causing compounds.
Smoking also causes a lipid effect on endothelial function, smoking has an effect on serum
lipids, this has been statistically shown to increase serum total cholesterol levels, this induces
lipid oxidation, increasing foam cells, thus initiating a process of platelet formation. , a study in
children of passive smokers exposed a susceptibility to early-onset heart disease, due to a genetic
form of dyslipidemia .
Smoking also increases the risk of type 2 diabetes mellitus with pathogenic studies supporting
the link between smoking and T2DM. Factor 2DM has a multifactorial etiologic index, it causes
a detrimental effect on endothelial function, increases inflammation and oxidative stress.
In conclusion, smoking triggers many diseases that directly accelerate cardiovascular diseases in
the body, so it is important to develop strategies to reduce this impact for both active smokers
and passive smokers.