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Bacterial Meningitis

Modifiable Factors: Non-Modifiable Factors:

• Vaccination Status • Age


• Hygiene • Medical Condition (Congenital abnormalities)
• Avoiding close contact • Premature birth
• Genetics

Streptococcus Pneumonia
Neisseria meningitidis

Organisms gain entry into CNS through Inflammatory response


upper respiratory tract via blood stream
• Increase cerebrospinal fluid
• Increased intracranial pressure
May enter through penetrating wounds of
the skull or fractured sinuses in basilar skull
fractures Increased ICP early signs

• Altered LOC, restless, irritability,


Purulent secretions spread throughout agitation, decreased mental
brain through the cerebrospinal fluid and status
cover cranial nerves and other intercranial
structures

If extends to brain parenchyma or if Observe for signs of increased ICP, it can


concurrent encephalitis is present, cerebral be increased from swelling and increased
edema and increased intracranial pressure cerebrospinal fluid volume
become a problem

SIGNS AND SYMPTOMS:

• High pitch cry LABS & DIAGNOSTIC PROCEDURE:


• Bulging fontanels
Cranial UTZ: (benign enlargement of the
• Hard stiff neck (nuchal rigidity)
subarachnoid space hydrocephalus)
• High temp (fever)
• Photophobia Hematology: (low HCT, low RBC, low EO, high MCH)
• Increased intracranial pressure (could lead to seizure, CT scan
coma, death)
• Agitation
• Altered/decrease Level of Consciousness
• Severe headache
• Nausea
• vomiting

If Yes Management If No

Persistent or recurrent infections


Nursing Management: Medical Management:

• Monitor vital signs closely, including • Initiate empiric antibiotic therapy


temperature, heart rate, respiratory immediately upon suspicion of Enlargement of ventricles and increased ICP
rate, and blood pressure. bacterial meningitis, based on
local antimicrobial resistance
• Assess neurological status regularly, patterns.
including level of consciousness, Neurologic complications (motor
• Perform lumbar puncture for
pupillary response, and motor cerebrospinal fluid (CSF) analysis impairments, sensory deficits, cognitive
function. to confirm the diagnosis of dysfunction, damage to cerebral cortex or
• Implement strict infection control bacterial meningitis and guide basal ganglia)
measures to prevent the spread of antibiotic selection.
bacterial meningitis to other patients • Administer appropriate antibiotics
Neurologic complications (motor
and healthcare workers. intravenously for a duration
determined by the specific impairments, sensory deficits, cognitive
• Provide comfort measures, such as organism and clinical response. dysfunction)
positioning the infant for optimal • intensive care teams to optimize
comfort and administering pain relief management and outcomes.
as needed.
Vision and hearing impairments
• Educate parents or caregivers about
• Consider placement of an external
the signs and symptoms of bacterial Developmental delays and disabilities
ventricular drain (EVD) or
meningitis and hydrocephalus, as
ventriculoperitoneal shunt (VP
well as the importance of follow-up
shunt) for management of
care.
hydrocephalus if indicated by
• Provide emotional support to parents
imaging studies or clinical
or caregivers during this challenging Recurrent seizure and epilepsy
symptoms.
time, offering reassurance and
• Provide supportive care for
information as needed.
complications such as seizures,
• Ensure adequate hydration and respiratory distress, or
nutrition, possibly through disseminated intravascular
nasogastric or intravenous routes if coagulation (DIC). Chronic health conditions
necessary. • Perform regular neurologic
assessments to monitor for
• Monitor for signs of increased
changes in status and response to
intracranial pressure (ICP), such as
treatment.
irritability, bulging fontanelle, or
• Coordinate multidisciplinary care Mortality
changes in level of consciousness.
involving neurology, neurosurgery,
• infectious disease, and pediatric
intensive care teams to optimize
management and outcomes.

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