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THIRD Edition
Disorders of Childhood
Development and Psychopathology
Michael F. Troy
Children’s Hospitals and Clinics of Minnesota
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Disorders of Childhood: Development and © 2018, 2014 Cengage Learning
Psychopathology, Third Edition
WCN: 02-300
Robin Parritz and Michael Troy
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Dedication
Robin dedicates this book to Ari, Adam, and Jesse, with love
and gratitude for these sweet babies, exuberant children, and
remarkable men.
Mike dedicates this book to Kevin and Brendan, whose lives are
his treasured memories, and Mimi, who brings new blessings.
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Brief Contents
1 Introduction 1
2 Models of Child Development, Psychopathology, and Treatment 12
11 A
nxiety Disorders, Obsessive-Compulsive Disorder, and Somatic Symptom
Disorders 194
Glossary 275
References 283
Name Index 358
Subject Index 386
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Contents
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viii Contents
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Contents ix
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x Contents
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Contents xi
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xii Contents
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About the Authors
xiii
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Preface
Writing a textbook on the psychological disorders of in- to be explored for both typically and atypically devel-
fants, children, and adolescents involves multiple deci- oping children. In this third edition, for example, we
sions about content, emphasis, and organization. These expanded the coverage of neuroscience research, in-
decisions reinforce and extend the knowledge base of cluding brain development and function (e.g., patterns
the field and determine what is distinctive about the of connectivity) and behavior genetics and epigenetics
authors’ approach. The decisions we made while writ- (e.g., gene-by-environment processes and interactions,
ing this book were influenced by our academic and differential sensitivity). We also expanded our emphasis
clinical experiences involving both typical and atypi- on understanding the multiple environments in which
cal development. When we made the decision to write children develop (e.g., relationships, families, peer
this book, we were particularly interested in providing groups, cultures).
a text that was both relevant and compelling. Our hope The second theme focuses on developmental frame-
was to provide students with the type of meaningful works and developmental pathways, and this perspec-
framework and conceptual integration that has come tive is reflected in the sequencing of chapters, unique
to characterize our field. We also wanted to offer teach- sections that open each chapter and summarize key
ers a more practical and more true-to-life approach to developmental tasks and challenges, and our descrip-
organizing their courses. tions of disorders over time. Disorders that emerge or
In this third edition, we reorganized several chap- are diagnosed early in development are presented first,
ters to better reflect how disorders, combinations of followed by disorders that emerge or are diagnosed in
disorders, and challenging diagnostic issues present the elementary-school years, followed by those that
in real-world clinical settings. For example, we moved emerge or are diagnosed in adolescence. This sequenc-
the chapter on maltreatment and trauma- and stressor- ing serves several purposes. First, it allows students to
related disorders to the set of chapters focused on consider specific disorders and sets of disorders that
early childhood. We also included new content on the occur in a particular developmental period in proxim-
transition to psychosis and personality disorders in a ity and relation to one another. Second, this sequenc-
reorganized chapter on substance-related disorders and ing allows for an ongoing focus on the constructs of
transition to adult disorders, in order to emphasize that risk and resilience and provides a basis for coherent
the developmental psychopathology perspective does discussions of early-occurring disorders as risk factors
not end with adolescence, but rather continues to in- for later-occurring disorders. For example, the chapter
form our understanding of individuals and disorders on disorders of early childhood focuses first on under-
across the lifespan. standing the nature and course of these disorders in and
Multiple themes recur throughout the text; together, of themselves; it also previews the multiple connections
they distinguish our clinical and teaching emphases. that will be made in subsequent chapters between tem-
Each of these themes is informed by the principles perament and attachment difficulties and later forms of
and practices of developmental psychopathology, an in- psychopathology. Third, this sequencing emphasizes a
terdisciplinary approach that asserts that maladaptive more complex understanding of disorders: For example,
patterns of emotion, cognition, and behavior occur in we think differently about depression that is identified
the context of typical development. The first theme em- early and on its own than we do about depression that
phasizes multifactor explanations. Multifactor explana- follows and may be related to an anxiety disorder or at-
tions of disorders encompass biological, psychological, tention deficit/hyperactivity disorder.
and sociocultural factors. These factors are examined in The sections at the beginning of chapters that sum-
detailed analyses of etiologies, assessments, diagnoses, marize the developmental tasks and challenges experi-
developmental pathways, and interventions. Especially enced by typically developing children are especially
distinctive is the way that we make sure to discuss the relevant, given the disorders discussed in the chapter.
multiple ways that factors at every level of analysis need For instance, a detailed summary of the development of
xv
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xvi Preface
self-regulation, effortful control, and executive function information about a child. We need to appreciate the
is presented before a clinical presentation of attention everyday joys and special accomplishments that are
deficit/hyperactivity disorder. An overview of prosocial part of all children’s lives. In addition, we believe that
behavior is presented at the beginning of the chapter on this holistic focus provides a number of opportunities
oppositional defiant disorder and conduct disorder, and a to talk about the stigma associated with mental illness
review of stress and coping is provided in the chapter on and to encourage awareness, tolerance, respect, and
maltreatment and trauma- and stressor-related disorders. compassion for children and adolescents who struggle
These introductory sections help students appreciate the with disorders.
developmental contexts of disorders and their core symp- Our hope is that this book will enable students to
toms; to make distinctions among the everyday issues think about disorders in the same way that caring adults
that most children experience, more difficult types of think about disorders they encounter every day—in
problems, and clinically meaningful psychopathology; terms of an individual child who is coping with distress
and to make comparisons between the factors that influ- and dysfunction: a boy or girl of a certain age, with
ence the multiple pathways of typical development and a specific temperament, characteristic strengths, and
the multiple pathways of psychopathology. personal history and a family and a network of friends
Discussions of developmental pathways, or descrip- embedded in a community and culture. We believe that
tions of disorders over time, accurately reflect how each we have written a textbook that places the child at the
child’s psychopathology unfolds over time in real life. center of comprehensive and meaningful information,
This pathway model also emphasizes opportunities reflecting the most up-to-date understandings of child
for growth and change. For example, we describe age- and adolescent psychopathology, in a format designed
related experiences, such as the transition to middle to support learning and understanding.
school, that are associated with some struggling chil-
dren getting back on track and certain well-function-
ing children experiencing distress. In this third edition, Key Features
we continue to provide up-to-date coverage of models In addition to the previously discussed case studies
describing developmental cascades, the accumulating woven throughout, our textbook offers a variety of
consequences of multiple transactions across domains, feature boxes that highlight important topics of inter-
levels, and systems. These new constructs emphasize est for students. The themes covered in these boxes
the integrative and dynamic nature of development and are (1) The Child in Context, (2) Clinical Perspectives,
psychopathology. (3) Risk and Resilience, and (4) Emerging Science.
The third theme takes into account the child in For study and review, each chapter includes a chapter
context and calls attention to the multiple settings in summary and list of key terms that appear in boldface
which the child is embedded. Discussions throughout in the text.
the text are intended to highlight the many ways in
which children and their disorders are understood in MindTap for Parritz and Troy’s Disorders
larger social contexts (e.g., families, schools and com- of Childhood
munities, cultures, and historical eras). In the third edi- MindTap is a personalized teaching experience with rele-
tion, new summaries provide information on children’s vant assignments that guide students to analyze, apply,
mental health in global context, as well as additional and improve thinking, allowing instructors to measure
research findings comparing children’s adjustment and skills and outcomes with ease.
maladjustment from diverse cultural backgrounds and
in various countries. ●● Guide Students: A unique learning path of relevant
The fourth theme involves a broad focus on the whole readings, media, and activities that moves students
child, rather than a narrow focus on disorder, devel- up the learning taxonomy from basic knowledge and
opmental delay, or impairment. This holistic appre- comprehension to analysis and application.
ciation of the child emphasizes patterns of interests, ●● Personalized Teaching: Becomes yours with a Learn-
abilities, and strengths. We make sure that our case ing Path that is built with key student objectives. Control
studies include this kind of information to remind what students see and when they see it. Use it as is or
students as often as possible that the diagnosis of a match to your syllabus exactly—hide, rearrange, add,
particular disorder does not provide all the important and create your own content.
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Preface xvii
●● Promote Better Outcomes: Empower instructors REVIEW. After students read the chapter and un-
and motivate students with analytics and reports derstand and know what they’ve read, it’s time to
that provide a snapshot of class progress, time in review and take the Chapter Quiz.
course, engagement, and completion rates.
In addition to the benefits of the platform, MindTap Supporting Resources
for Parritz and Troy’s Disorders of Childhood includes
Cengage offers the following supplements for Disorders
the following learning path:
of Childhood:
START. Students begin their personalized learn- ●● Cognero. Cengage Learning Testing Powered by
ing plan for each chapter with Mastery Training,
Cognero is a flexible, online system that allows you
powered by Cerego. This app helps students retain
to author, edit, and manage test bank content from
knowledge as they progress through each chapter,
multiple Cengage Learning solutions, create multi-
and pass each test!
ple test versions in an instant, and deliver tests from
READ. Students read the chapter next. After each your Learning Management System (LMS), your
major section, students answer the Check Your classroom, or wherever you want. The testbank was
Understanding mini-quiz questions. These section prepared by Debra Schwiesow
quizzes help students know what they just read be- ●● Online Instructor’s Manual. This supplement,
fore progressing to the next major section. prepared by Rebecca Fraser-Thill, contains valuable
WATCH. Students watch videos, which are followed resources for preparing for class, including chapter
by thought-provoking questions related to both the outlines, lecture topics, YouTube video suggestions,
chapter that they just read and the video content. and class activities.
Each video features real people with real disorders, ●● Online Microsoft PowerPoint Lecture Outlines.
including attention deficit/hyperactivity disorder Prepared by Rebecca Fraser-Thill, these handy and
(ADHD), autism spectrum disorder, learning disor- accessible lecture outlines are a great starting point
ders, intellectual disability and more. for helping instructors prepare for class.
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Editorial review has deemed that any suppressed content does not materially affect the overall learning experience. Cengage Learning reserves the right to remove additional content at any time if subsequent rights restrictions require it.
Acknowledgments
We wish to thank the many individuals who have in- We have many people to thank at Cengage, in-
spired, challenged, encouraged, and supported us from cluding Carly McJunkin, who provided invaluable
the very beginning of this project to the final revisions advocacy, enthusiasm, and experience. We thank
of the third edition. Although our two names are on the Tangelique Williams-Grayer for her careful attention
cover, we are deeply aware that our text reflects the work to detail, patient problem solving, and positive atti-
of countless others whose research studies and clinical tude. We are grateful to the marketing team for all of
insights we have cited and summarized. We thank them their hard work in promoting the first and second edi-
for their contributions to this text and to the field of de- tions and for their excitement about the third edition.
velopmental psychopathology. We also thank all the individuals on the production
We are grateful for the exceptional educational, team, including Nick Barrows (Intellectual Property
research, and clinical experiences that have motivated Project Manager), James Finlay (Marketing Manager),
us to write this book. We are grateful to teachers and Ruth Sakata-Corley (Cengage Content Project Man-
colleagues who have shared their knowledge of child ager), and Lynn Lustberg (Project Manager) at MPS
development and psychopathology, along with their Limited.
vision of sound, compassionate intervention. We es- This text has benefited greatly from the comments and
pecially acknowledge our undergraduate, graduate, suggestions of many reviewers, including the following:
and clinical mentors, who exemplify professional
accomplishment and generosity and who model pas- Jack Bates, Indiana University–Bloomington
sionate commitment to children’s well-being: Joe Michelle Broth, Georgia Gwinnett College
Cunningham of Brandeis University, Megan Gunnar Arin Connell, Case Western Reserve University
and Alan Sroufe of the University of Minnesota, and Mary Ann Coupland, Sinte Gleska University
Ada Hegion and Vivian Pearlman of the Hennepin Carolyn Fallahi, Central Connecticut State
County Medical Center. We could not be prouder to University
be their students. Bill Frey, Castleton State College
We thank our own students and clients, who have Jennifer Green, Miami University
challenged us to be better explainers of theory and Wendy Hart, Arizona State University
more thoughtful models of practice. We also thank Steve Lee, University of California Los Angeles
our colleagues at Hamline University and at Children’s Susan Marell, St. Thomas Aquinas College
Hospitals and Clinics of Minnesota for their ongoing Paul McCabe, Brooklyn College–City University of
encouragement and support. Thanks also to Wendy New York
Werdin, the most wonderfully obliging faculty secre- Suzanne Morin, Shippensburg University
tary at Hamline University, for typing stacks and stacks Casey Tobin, University of Wisconsin–La Crosse
of references. Deborah Walder, Brooklyn College–City University
As we prepare to launch the third edition, we wish of New York
to thank all those who were so important to the suc-
cess of the first and second editions, including the Robin Parritz especially thanks Mike Troy, a dear
professors and instructors who adopted our textbook friend and admired clinician whose intellect, passion,
and provided us with valuable feedback. In particu- and perfectly timed humor have made all aspects of this
lar, we thank Alan Sroufe and Dante Cicchetti for collaboration rewarding. Robin is extraordinarily fortu-
believing in us and in the value of this endeavor. We nate that she is embedded in ever-expanding circles of
are especially grateful for their formal endorsement loving family and wonderful friends. She will always be
of the book, which has been key to its widespread grateful for their support, encouragement, and friend-
acceptance in the field and a source of great pride to ship. Robin also thanks her sons and daughters-in-law,
the authors. Ari and Rachel, Adam and Hadley, and Jesse for every
xix
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xx Acknowledgments
kind of happiness. Robin is forever indebted to her the years pass by. He is grateful to have grown up
husband, Jon Parritz, for enormous amounts of love, in the convergence of love created by his siblings—
counsel, and support. Kathie, Bill, Joe, Tim, Maureen, and Mary, who each
Mike Troy thanks Robin Hornik Parritz, a true of us miss each and every day. He thanks Paul, Tom,
friend and gifted colleague, for her grace and wis- and Brian for timeless friendship. Above all else, he
dom. Their ongoing collaboration is his great good is thankful to Cynthia Koehler Troy for the gift of
fortune. Mike’s gratitude to his parents, Bill and love, loyalty, and family. She is at the center of all
Carmen Troy, somehow manages to grow deeper as that is best in his life.
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1 Introduction
WHEN WE THINK ABOUT childhood and growing up, images of wonder, energy, excite-
ment, and joy are common. Babies sharing first smiles and taking first steps; kindergarten-
ers singing loud songs and looking forward to family vacations; children reading books, rid-
ing bikes, and sleeping over with friends; teens studying for exams, learning to drive, and
falling in love. In the midst of all this growth and change, however, we notice children who
are almost always sad, worried, afraid, or angry. We meet children who believe that they
are bad, that they have no control over their lives, that the world is an awful place. There are
children who lash out at others, and some who withdraw from relationships. Some of these
children exhibit patterns of feelings, thoughts, and behaviors that are best understood as
psychological disorders.
The goal of this textbook is to provide a basic understanding of these children and
their disorders, and of the theories, methodologies, and findings of developmental
psychopathology. We need to understand so that we may meaningfully describe the
psychological disorders of infancy, childhood, and adolescence. We need to understand
so that we can identify the numerous factors that increase vulnerability to psychopa-
thology. We need to understand so that we can design appropriate interventions for
struggling children. We need to understand so that we can increase awareness and
empathy for children who deserve to be treated with dignity and respect. And we need
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2 CHAPTER 1 Introduction
to understand so that we can provide the necessary support and resources to families,
schools, and communities.
Our approach in writing and organizing this textbook is based on the central prem-
ise of developmental psychopathology, which suggests that we gain a better un-
derstanding of children’s disorders when we think about those disorders within the
context of typical development. We believe that infant, child, and adolescent psycho-
pathology can be understood only by placing descriptions of disorders against the
background of usual emotional, cognitive, and behavioral development. We also be-
lieve that it is necessary to acknowledge the everyday problems and difficult phases
that characterize typical child development, and to make clear both the connections
and the distinctions between adaptation and maladaptation. We present discussions
of children’s disorders in a sequence that follows the child’s own growth from birth
through early adulthood and emphasizes that both children and their disorders develop
and change over time.
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What Is Normal? 3
Transition to Adulthood
Major issue: Emancipation
Proportion
Additional issues:
Launching a life course
Financial responsibility
Adult social competence Disorder Disorder
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Editorial review has deemed that any suppressed content does not materially affect the overall learning experience. Cengage Learning reserves the right to remove additional content at any time if subsequent rights restrictions require it.
4 CHAPTER 1 Introduction
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The Role of Values 5
frequent temper tantrums that often involved psychotherapist attempts to provide support for his
biting and scratching. In fact, Dylan’s parents were concerns, Antoine is adamant that he does not want
asked to withdraw him from his preschool because to talk about any possible relocation. ■
of his poor emotional and behavioral regulation.
When these issues with Dylan escalated, so did his
mother’s depression, as well as conflict between Optimal Adaptation
his parents, who disagreed on what should be done
to manage Dylan’s behavior.
Dylan’s father died just before the start of
The Case of Jenna
kindergarten. Following the unexpected loss, Jenna is a six-year-old girl who, like Dylan and
Dylan’s anxiety, always present but overshadowed Antoine, suffered an early loss. Jenna’s mother was
by his behavior problems, became increasingly a single parent who died in an automobile accident
evident. Over the next two years, both his first- when Jenna was two. Following her mother’s death,
and second-grade teachers provided Dylan with Jenna went to live with her maternal grandparents.
extra support and encouragement, but with little Although distraught at the loss of their daughter,
positive effect. At the beginning of third grade, the they dedicated themselves to caring for Jenna to the
school counselor suggested to Dylan’s mother that best of their ability.
they see a child psychologist. Although Dylan’s In addition to her grandparents, Jenna is involved
mother wanted to comply with the referral, she felt with and supported by her many relatives who
overwhelmed by the challenges of single parenting live nearby and include her in their lives. Jenna’s
and her depression and never arranged for Dylan teachers describe her as bright and enthusiastic
to see a therapist. As his classmates became more in the classroom. She is excited about learning to
focused on developing friendships and enjoying read and seems to have a special aptitude for math.
academic experiences, Dylan felt increasingly Jenna is well liked by both the girls and the boys in
isolated, lonely, and unhappy. ■ her class, and she is often invited to play dates and
birthday parties.
At home, Jenna enjoys hearing stories about her
Adequate Adaptation mother and thinking of how loving and proud her
mother would be. There are times, of course, when
Jenna and her grandparents cry together about
The Case of Antoine
Jenna’s mother. And as Jenna gets older, she may
Antoine is a six-year-old boy who is currently in his become more aware of her absent biological father
third foster home. Antoine was severely neglected and seek to learn more about him. But Jenna and her
early in his life and was removed from his biological grandparents are able to take comfort in each other
mother’s home when he was nine months old by the and in the warm and secure home that they have
county’s child protection services. After two brief created together. ■
foster placements, Antoine has been in a stable and
nurturing foster home for two years.
Even with the traumatic beginnings of their child-
Although his teachers have no concerns about
his basic academic skills, they note that Antoine
hoods, both Antoine and Jenna are moving in a positive
does have difficulty paying attention and that he is developmental direction, in contrast with Dylan. Still,
frequently impulsive. Antoine has several friends Antoine’s adequate adaptation is different from Jenna’s
that he likes to play with, but he is seldom sought optimal adaptation in the degree to which each success-
out as a playmate by other children. His feelings fully manages past traumas and current challenges, the
are hurt easily, and he sometimes misinterprets quality of caregiving and friendship, and the potential
the intentions of others, feeling that they are out for growth in coming years. Neither adequate nor opti-
to get him. Consequently, he is quicker than other mal adaptation guarantees smooth sailing throughout
children to resort to name-calling or shoving when development. Challenges are inevitable, and struggles
he is upset.
themselves are not evidence of disorder. Indeed, chal-
Antoine is more comfortable and relaxed at
lenges and struggles are viewed by most developmental
home with his foster parents, but he asks often
if he will have to move away from them. While
psychologists as forces of growth. Sameroff (1993, p. 3),
being as reassuring as possible, his foster parents in fact, suggests that “all life is characterized by distur-
have acknowledged that they do not know how bance that is overcome, and that only through distur-
long Antoine will be with them. He clearly worries bance can we advance and grow. . . . In this view, it is
about leaving his current home, and although his the overcoming of challenge that furnishes the social,
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6 CHAPTER 1 Introduction
emotional, and intellectual skills that produce all forms by a particular sociocultural group influences concep-
of growth, both healthy and unhealthy.” tualizations of pathological addiction. Whether inde-
pendence or connectedness is more valued influences
conceptualizations of pathological dependency.
The Impact of Values on Definitions
With mental health definitions, the values of psy-
of Disorder chologists, psychiatrists, and clinical social workers are
Other important judgments involving values are tied to embedded in both scientific and lay community decision
specific definitions of disorder. With statistical deviance making. Returning to the Surgeon General’s descrip-
definitions, it sometimes makes sense to examine both tion of psychological well-being, clinicians must evaluate
extremes of the continuum (e.g., too much intense emo- whether a young person’s life is characterized by a posi-
tion as well as too little) because we have made a judg- tive quality, adequate functioning, and few symptoms.
ment that there is a desirable middle course related to the Whether these particular benchmarks represent the least
characteristic in question (again, see Fig. 1:1). At other we can do for children and adolescents, or the best we can
times, it makes sense to focus only on the “bad” end of hope for, is yet another value judgment. Indeed, recent
the continuum and ignore the “good” end (e.g., too little discussions of models of mental health have emphasized
empathy, but not too much empathy; too little intelli- the difference between the absence of mental illness and
gence, but not too much intelligence). In these specific the presence of flourishing. For example, to enhance
cases, judgments are made that some types of extreme individuals’ opportunities for flourishing, Keyes (2007)
characteristics are to be accepted or even prized. argues for increased resources for programs that focus
With sociocultural definitions, value judgments are on the promotion of mental health across the lifespan,
the very basis of definitions of disorder. Whether casual as well as for programs that focus on the prevention and
use of mind-altering substances is tolerated or condemned treatment of mental illness.
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Definitions of Psychopathology and Developmental Psychopathology 7
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8 CHAPTER 1 Introduction
Sample
Outcome N studies size
Any anxiety disorder 41 63130
MDD 22 68382
ADHD 33 77297
ODD 28 69799
CD 28 73679
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The Stigma of Mental Illness 9
iStockphoto.com/Claudiad
able to access appropriate and effective mental health
services directly; (2) Child mental health should be a
major component of healthy development promotion
and attention in primary care settings such as schools,
pediatric care, community programs, and other systems
central to child development; (3) Efforts should empha- Far too many children experience displacement,
size preventive care for high-risk children and families; hardship and loss; the negative impact on physical and
(4) More attention must be paid to cultural context and psychological well-being is enormous.
cultural competence.” These kinds of proposals lay the
groundwork for resource allocation and policy imple- ensure children’s safety and well-being. Interventions
mentation that will have long-standing consequences include both prevention efforts and treatment for those
for the well-being of countless children. with various disorders. To facilitate the success of inter-
ventions, mental health professionals must consider
The Globalization of Children’s how to implement treatments in countries where the
health and welfare systems work differently (or are non-
Mental Health existent), as well as how to provide treatment to chil-
Discussions of mental health and mental illness involv- dren who are difficult to reach (Atilola, 2015; Patel,
ing resource allocation and public policy increasingly 2012). Treatments must take into account local and
emphasize global perspectives that require careful think- culture-based approaches and community caretaking
ing about Western models of development, disorder and and service models (Atilola, 2015). Holistic approaches
intervention, as well as the vastly different experiences with achievable goals, embedded in health, social,
of children who live in resource-rich versus resource- and educational networks, have been proposed. These
poor countries. Patel, Flisher, Nikapota, and Malhotra multicomponent treatments focus on children and ado-
(2007) and Omigbodun (2008) identify rapid social lescents, on families, and on communities and systems
change, urbanization and urban poverty, and inade- (Patel, 2012; Wuermli, Tubbs, Petersen, & Aber, 2015).
quate health and educational services as key factors that Finally, the development and implementation of glob-
increase children’s vulnerability to psychopathology in ally useful interventions require recognition of the cur-
resource-poor countries in Eastern and Central Europe, rent disconnect between where research takes place and
Africa, Asia, Latin America, and the Pacific region. In where the need is greatest, as well as a commitment
these countries, awareness of mental illness issues and to do better on behalf of the world’s children (Atilola,
promotion of mental health are limited by allocation of 2015; Patel, 2012; Wuermli et al., 2015).
scarce resources to urgent medical needs, a lack of for-
mal mental health policies and programs, and too few
mental health professionals. The costs of impairment The Stigma of Mental Illness
and lost potential are enormous (Belfer, 2008). A final issue concerns the continued and painfully
We must also emphasize that, across the globe, mil- unnecessary stigmatization of individuals with psy-
lions of children are struggling in the face of unimagi- chopathology (Corrigan, 2005; Hinshaw, 2005; Pes-
nable trauma, including exposure to disease and death, cosolido, 2007). For parents concerned about their
armed conflict, abandonment and homelessness, and children’s distress or dysfunction, there is almost
dislocation (Omigbodun, 2008; Vostanis, 2012). These always shame, fear, and/or blame (dos Reis, Barksdale,
terrible situations require increased awareness, advo- Sherman, Maloney, & Charach, 2010; Heflinger,
cacy, and a responsibility to provide interventions to Wallston, Mukolo, & Brannan, 2014). For children,
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10 CHAPTER 1 Introduction
Box 1:2
The Child in Context
The Stigma of Mental Illness
I gnorance and intolerance have long been identified as
critical issues for those struggling with mental illness.
Much of the available research focuses on adults’ limited
ongoing stigmatization related to views of those struggling
with mental illnesses as violent, unpredictable, blamewor-
thy, and beyond hope (Corrigan et al., 2007; Watson,
and inaccurate knowledge of and negative attitudes to- Miller & Lyons, 2005). Stigma is often associated with ex-
ward other adults with mental illness. In study after study, clusion, and evidence suggests that adolescents worry about
the data suggest that most adults tend to think primar- the perceived social and personal risks of friendships with
ily in terms of serious psychopathology (such as schizo- peers with mental health problems (O’Driscoll, Heary,
phrenia and bipolar disorder), believe that individuals Hennessy, & McKeague, 2014). It is not surprising, then,
are responsible for their disorders, and overestimate the to find that many children and adolescents with disorders
likelihood of aggression and violence in adults with men- “self-stigmatize”; that is, they internalize these negative be-
tal illness; stigmatization, in terms of ridicule, avoidance, liefs and attitudes and exhibit low levels of self-esteem and
and rejection, is rampant. self-efficacy (McKeague, Hennessey, O’Driscoll, & Heary,
Adults also exhibit distorted beliefs and harmful atti- 2015; Moses, 2009).
tudes toward children who are struggling with mental Given that children are exposed to multiple sources of
illness, as well as toward their families. Adults both trivi- information and attitudes, including parents, peers, and
alize the reality of children’s distress and dysfunction by the media, how can stigmatization be prevented or mini-
suggesting that children are overdiagnosed, overmedi- mized? Many types of programs, from those designed for
cated, and poorly parented, and exaggerate the extent to individual classrooms to those intended as national dem-
which these same children are unpredictable, dangerous, onstration projects, have shown improvements in knowl-
and deviant (Giummarra & Haslam, 2005; Pescosolido edge and attitudes (e.g., Corrigan, 2005; Pitre, Stewart,
et al., 2008). Adams, Bedard, & Landry, 2007; Watson et al., 2004).
How do children and adolescents compare to their adult Successful programs share several emphases. They must
counterparts? Sadly, their beliefs and attitudes are all too begin early; target multiple dimensions of knowledge and
similar. Surveys of children’s labels for those dealing with attitudes; be developmentally appropriate; and include in-
mental illness—including crazy, nuts, retarded, psycho, and dividuals, families, and communities. Children can learn
lunatic—reveal their aversion (Bailey, 1999; Wahl, 2002). lies or they can learn facts; they can display ugly attitudes
Although children display increasing knowledge about the or they can display compassion. The choices are theirs,
causes of mental illness as they age, their attitudes reflect and ours.
experiences of secrecy and rejection are commonplace. stereotypes, devaluation, and discrimination; (2) two
Lack of respect and lack of access to care (again) are targets of stigma, the individual and the family; and
often the results of personal, familial, social, and (3) two contexts of stigma, the general public and the
institutional stigmas (Heflinger & Hinshaw, 2010; self/individual. Both Mukolo et al. (2010) and Heflinger
Williams & Polaha, 2014). A clinician seeking paren- and Hinshaw (2010) urge researchers to continue to
tal permission to obtain information from a child’s investigate the multiple ways that stigma compli-
teacher is not surprised when a father says, “You cates the experiences of children with mental disor-
know, doctor, we’d prefer that the school not know ders and their families. Box 1:2 provides additional
anything about this. We haven’t told his brothers or perspective on this kind of stigma. Understanding
his grandparents. No one else needs to know.” Or a the development, course, and treatment of psycho-
teacher, preparing a child to begin attending a social pathology in infants, children, and adolescents rep-
skills group the following week, is asked, “Why do I resents only half the battle. Increasing our tolerance
have to leave your room, Mrs. Stern? I don’t want to and compassion for the diverse group of those who
go with those kids. They’re weird. I’m not weird. I’m are diagnosed with psychopathology and believ-
not crazy.” ing in the inherent worth of each struggling infant,
Mukolo, Heflinger, and Wallston (2010) identify child, and adolescent make up the other, far more
(1) several dimensions of stigma, including negative difficult, half.
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The Stigma of Mental Illness 11
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2 Models of Child Development,
Psychopathology, and Treatment
12
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The Role of Theory in Developmental Psychopathology 13
after the school day. Also, Max began to argue The Role of Theory
and fight more frequently with his 10-year-old
brother, and especially with his four-year-old sister. in Developmental Psychopathology
His parents report that Max still enjoys playing Models of development, psychopathology, and treat-
with friends in the neighborhood but is becoming ment allow us to organize our clinical observations
increasingly resistant, discouraged, and pessimistic
of children and our research findings into coherent,
about school. The more stressful family problems
informative accounts. In this chapter, the cases of Max
coincided with Max’s father being laid off from his
job as a master electrician. Max’s father has spent
and Anna will illustrate key concepts related to typical
increasing amounts of time at home, with escalating developmental processes, the emergence of disorder,
conflicts between him and Max’s mother (who does and intervention goals and strategies. For introduc-
not work outside the home) about child care and tory purposes, the sections on Max and Anna present
discipline. ■ somewhat simplified examples. In the next chapter,
and throughout the rest of this book, the models will
become increasingly complex, integrated, and real.
The Case of Anna Before the practices and principles of developmental
Anna is 14 years old. She spends a lot of her free psychopathology are described in Chapter 3, we summa-
time alone in her room, feeling unhappy and not rize here the historical models that have contributed valu-
doing much of anything. She rarely gets together able ideas to our contemporary understanding. Although
with other kids, who have mostly stopped asking these models are presented separately and are often con-
her to join them. Anna’s mother is worried about her ceptualized as complete and comprehensive in and of
sadness and withdrawal and has called her family themselves, they are not mutually exclusive. It is more
physician for a referral. useful to think of these models as providing different and
Anna’s mother has been a single parent since complementary perspectives on the complicated phenom-
Anna’s birth and is employed as a customer service
ena of development, psychopathology, and treatment.
representative for a health care company. Anna’s
father has a long history of hospitalizations for
both major depression and alcohol abuse. Following
Dimensional and Categorical Models
several extremely unpleasant exchanges with
Anna’s mother when he came to visit Anna as an To provide additional background for the upcoming
infant and toddler, Anna’s father has had no contact summaries, it is useful to consider how various defini-
with her. tions of disorder correspond with dimensional versus
Anna’s mother describes Anna’s infancy and categorical models of psychopathology. Dimensional
childhood as normal. Throughout elementary school, models of psychopathology emphasize the ways in
Anna was generally quiet and cooperative and which typical feelings, thoughts, and behaviors gradually
received average grades. Although not especially
become more serious problems, which then may inten-
social, she always had a few good friends and was
sify and become clinically diagnosable disorders. With
active in sports and with her church youth group.
Looking back, Anna’s mother remembers that Anna
dimensional models, there are no sharp distinctions
seemed to worry more than most other children, between adjustment and maladjustment. Dimensional
but not to the point where it interfered with her models also are referred to as continuous or quantitative.
schoolwork or social activities. Her transition to Categorical models of psychopathology, in contrast,
middle school was challenging. Anna’s mother emphasize discrete and qualitative differences in individ-
reports that Anna seemed somewhat overwhelmed ual patterns of emotion, cognition, and behavior. With
by the size of the school and had difficulty adjusting categorical models, there are clear distinctions between
to changing classes and increased homework. Anna what is normal and what is not. Categorical models are
had less contact with her elementary school friends sometimes referred to as discontinuous or qualitative.
and has had trouble making new friendships.
Important differences between dimensional and cat-
Although Anna does not talk much about her
situation, her increasing withdrawal, apathy, and
egorical models are illustrated by thinking about Max and
occasional irritability are apparent. She no longer
Anna. For instance, do the difficulties experienced by Max
participates in athletics, has dropped out of her church and Anna reflect extremes of typical difficulties (dimen-
youth group, and spends most of her time at home sional examples), or are they problems of a different sort
alone. She is increasingly behind in her schoolwork, altogether (categorical examples)? What do parents, teach-
and her grades have dropped significantly. ■ ers, and clinicians gain from the dimensional perspective,
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14 CHAPTER 2 Models of Child Development, Psychopathology, and Treatment
Courtesy of the USC Laboratory of Neuro Imaging and Athinoula A. Martinos Center for
the categorical perspective, which instead emphasizes the
particular patterns of the problematic emotions, thoughts,
and behaviors that give rise to significant maladjustment?
Physiological Models
Historical and Current
Conceptualizations
Physiological models propose that there is a physiologi-
cal (i.e., genetic, structural, biological, or chemical) basis
for all psychological processes and events. Historical
Biomedical Imaging
conceptualizations often focused on the multiple ways
in which genes, brain structure and function, and early
critical periods influenced, directed, and constrained
development. Contemporary conceptualizations are even
FIGURE 2:1 An illustration of neural connections across
more complex, taking into account behavior genetics, brain regions. From http://www.humanconnectomeproject
gene-by-environment effects, and the organization and .org/
reorganization of brain networking across development.
Using information from increasingly detailed studies of context of numbers of connections, distances between
genetics and from imaging technologies that examine the them (i.e., the path length of connections), centrality, and
structure, function, and neurobiology of the brain, we are clustering. Hubs are nodes with extensive connections
becoming more knowledgeable about “how a child builds a to other nodes. Modules are groups of nodes with strong
brain” (Cicchetti, 2002, p. 23). With respect to brain devel- interconnections. With greater connectivity within and
opment, we need to consider how children’s brains adapt to across brain regions, the medial parietal cortex, the cin-
their environments over time in ways that are similar to all gulate cortex, the superior frontal cortex, and the insula
other children, as well as in ways that are idiosyncratically are hubs in the connectome model. Wiring patterns are
distinct (Johnson, Jones, & Gliga, 2015). We must not fully in place at birth, with some connectivity tracts
appreciate both how specific brain regions are associated maturing early (related to, for example, visual, auditory
with particular types of activity (e.g., emotion, memory) and sensorimotor processing) and other tracts maturing
and how interactions and connectivity among brain later (related to, for example, more sophisticated cogni-
regions contributes to overall brain function (Johnson tive activity). There are also changes related to the balance
et al., 2015). In fact, recent work suggests that important between segregation and specialization of function and
information about psychopathology is less likely to come overall integrated functioning (Bullmore & Sporns, 2012;
from investigations of “the dysfunction of one specific Collin & van den Heuvel, 2013).
brain region” and more likely to come from studies of the Advances in neuroscience have led to the mapping
ways in which “these regions are anatomically and func- of brain structures over time and to rich descriptions of
tionally connected” (Matthews & Fair, 2015, p. 405). development: the “exuberant increase in brain connec-
Research focused on the human connectome—the tions is followed by an enigmatic process of dendritic
diagram of the brain’s neural connections—makes use of ‘pruning’ and synapse elimination, which leads to a more
graph theory, diffusion imaging, and quantitative analysis efficient set of connections that are continually remod-
to map the anatomical and functional features of com- eled throughout life” (Toga, Thompson, & Sowell, 2006,
plex brain networks (Bullmore & Sporns, 2009; Vertes & p. 148). Toga et al. (2006) suggest that some brain areas
Bullmore, 2015; http://www.humanconnectomeproject (such as the frontal cortex) develop under “tight genetic
.org/; also see Figure 2:1). In contrast to explanations of control,” whereas other areas are more influenced by the
microscopic connectivity (e.g., between neurons), explana- environment. In addition, differing levels of gray and
tions of the connectome focus on macroscopic connectiv- white matter growth are observed, with some regions dis-
ity (e.g., between brain regions), and include descriptions playing simpler growth trajectories (e.g., ending earlier)
of nodes, hubs, and modules (Collin & van den Heuvel, and others more complex ones (e.g., ending later) (Nelson,
2013; also see Figure 2:2). Nodes are understood in the 2011; Vertes & Bullmore, 2015; also see Figure 2:3).
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Editorial review has deemed that any suppressed content does not materially affect the overall learning experience. Cengage Learning reserves the right to remove additional content at any time if subsequent rights restrictions require it.
Physiological Models 15
a d
b
Pathlength Clustering Hub Module
The architecture of networks can be examined using graph theory. Four commonly used measures to probe the organization of the
human connectome are (see accompanying figure) path length, clustering, and the presence of hubs and modules (Rubinov and Sporns
2010). The characteristic path length of a brain graph is defined as the average number of steps required to travel between nodes of
the network (e.g., brain regions) and is often taken as an (inverse) metric of communication efficiency or information integration of a
region. The left-most bottom panel depicts the path length between nodes a and b measuring three steps. The clustering coefficient
of a node indicates the extent to which its neighbors are mutually connected (left middle panel), expressing a level of information
segregation in neural networks (grained lines indicate the absence of a connection between the neighbors of node c). The right middle
panel shows a network with a highly connected hub node d, indicating a node with an above average dense level of connectivity. Hub
nodes play a central position in the overall network. The most right panel indicates the presence of a clustered module, indicated by
three nodes (encircled in yellow) being mutually strongly interconnected, but sparsely connected to the rest of the network.
FIGURE 2:2 Architectural structures in the connectome.
Source: DOI: 10.1177/1073858413503712, The Neuroscientist 19(6) 616–628 © The Author(s) 2013, The Ontogeny of the Human
Connectome: Development and Dynamic Changes of Brain Connectivity Across the Life Span, Collin & van den Heuvel, 2013, p. 617, Box 1,
second figure. Reprints and permissions: sagepub.com/journalsPermissions.nav
Neurogenesis
Consolidation
Synaptogenesis
Anatomy
Axon growth
Myelination
MRI
Then Grey matter volume
Then Cortical thickness
Increasing white matter volume
Neuroimaging
DTI
Anatomical connections Maturation of white
in place at birth matter tracts
fMRI
Functional connections progressively form
first locally, then over longer distances
EEG
Increasingly small-world connectivity
16 32 4 months 2 5 10 15 20 25+
Weeks Years
Conception Birth
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Editorial review has deemed that any suppressed content does not materially affect the overall learning experience. Cengage Learning reserves the right to remove additional content at any time if subsequent rights restrictions require it.
16 CHAPTER 2 Models of Child Development, Psychopathology, and Treatment
Sensitive (or critical) periods in brain develop- Neural plasticity illustrates several physiological pro-
ment have also been identified, some of which appear cesses related to brain development, organization, and
domain or component dependent (e.g., in the audi- reorganization. It involves the development and modifi-
tory system, or for specific components of language) cation of neural circuits, with now-conclusive evidence
(Thomas & Johnson, 2008). For example, researchers that “both positive and negative experiences can influ-
have described early periods in which the development ence the wiring diagram of the brain” (Nelson, 2011,
of limbic system circuitry leads to later differences in p. 57). Numerous examples of neural plasticity, involv-
internalizing or externalizing self-regulation (Tucker, ing changes in anatomy and neurochemistry, have been
Poulsen, & Luu, 2015). A recent review of sensitive described for multiple systems (e.g., visual, motor, lan-
periods explored the role of the hormone oxytocin in guage, and learning and memory systems). The “primary
promoting social development. Feldman (2015, p. 369) sensory areas in particular—the brain’s first filters to the
suggested that the oxytocin system “cross-talks with outside world—exhibit striking examples of experience-
the stress, reward, immune, and brain stem systems” dependent plasticity” in early life (Takesian & Hensch,
and that early “synchrony, the coordination of biology 2013, p. 6). And, whereas we once believed that brain
and behavior during social contact” was the mechanism development was relatively complete by age three, and
by which the sensitive period influenced brain devel- that any damage was permanent and irreversible, we
opment, and later social behavior. While these various now understand that plasticity is associated with impor-
processes of brain development are similar for typi- tant growth after the age of three and with the lifelong
cally developing children, differences have also been potential for new, improved, and recovered function (see
observed in “perfectly normally functioning individu- Figure 2.4). As important as plasticity is across develop-
als” (Nelson, 2011, p. 52). That is, we often expect that ment, a balance between plasticity and stability is critical.
differences in brain structure, function, and develop- Indeed, “one of the outcomes of normal development is to
ment will be observed between typically and atypically stabilize the neural networks initially sculpted by experi-
developing children, but we also must understand that ence. Rather than being passively lost, the brain’s intrinsic
there are variations (e.g., deviations from the norm) potential for plasticity is actively dampened” (Takesian &
within groups of typically developing children. Hensch, 2013, p. 3). Ongoing research explores the mech-
With respect to brain–behavior relations, we have anisms underlying both plasticity (i.e., molecular trig-
shifted from earlier views that emphasized the unidi- gers) and stabilization (i.e., molecular brakes), and how
rectional influence of brain structure and function on either might be influenced or modified to treat neuro-
behavior to models that emphasize bidirectional influ- developmental or neurodegenerative disorders. Figure 2:4
ences. As one early example of brain–behavior relations, illustrates the relation across development between plas-
Bell and Fox (1996) documented patterns of physiological ticity (i.e., the brain’s ability to adapt or change) and the
and electroencephalograph (EEG) activity in groups of amount of effort required for such change.
eight-month-olds with various crawling histories. Com- Genetics play a critical role in physiological models.
parisons of noncrawling infants, beginning crawlers, and We need to understand the many ways that the genetic
experienced crawlers provide evidence that brain develop-
ment specific to crawling involves an initial overproduc- The brain’s ability to Amount of effort
change in response such change requires
tion of cortical connections that are then “pruned” with to experiences
additional crawling experience. This pattern of produc-
tion and pruning illustrates how the brain’s development
responds to environmental feedback, resulting in increas-
ingly efficient processing. Another example of experi-
ence-dependent brain development involves changes in
the connectivity tracts over the first years of life that are
associated with higher cognitive functions (Collin & van
den Heuvel, 2013). Further, while the “structural topol- 2 4 6 810 20 30 40 50 60 70
ogy of communication hubs in the human brain appears Age
Birth
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Editorial review has deemed that any suppressed content does not materially affect the overall learning experience. Cengage Learning reserves the right to remove additional content at any time if subsequent rights restrictions require it.
Physiological Models 17
makeup of an individual, or genotype, influences the Explanations for both typical development (and all
observable characteristics of an individual, or phenotype. variations of healthy children) and atypical develop-
Our understanding of genetics (i.e., genes and heredity) ment (and all variations of struggling children) depend
is ever expanding and involves work in both behavior on understanding genes, genetic variants, and the pro-
genetics and molecular genetics. Research techniques cesses by which multiple genetic and nongenetic effects
used to investigate the influence of genes include twin, lead to physiological and psychological outcomes (see
family, and adoption studies, as well as genetic sequencing, Table 2:1 for key definitions). Behavior genetics, the
genetic screening, and genome-wide association studies. study of the joint effects of genes and environments,
Additive genetic variance: Individual differences caused by the independent effects of genes that add up.
Allele: One of two or more alternative forms of a gene. These alternative forms may be relatively common or relatively
rare in the population of individuals with and without disorders.
Behavior genetics: The study of the joint effects of genetics and the environment; with respect to developmental
psychopathology, the focus is on the relationship of genetic variation and psychological traits, symptoms, and disorders.
Chromosome: A structure that contains DNA, and resides in the nucleus of cells.
Deoxyribonucleic acid (DNA): Double-stranded molecule that encodes genetic information.
DNA methylation: An epigenetic process by which gene expression is influenced by adding a methyl group to a chromosome region.
Epigenetics: The processes and mechanisms that influence gene expression.
Epigenome: Epigenetic events throughout the genome.
Gene: The basic unit of inheritance.
Gene expression: Transcription of DNA into messenger RNA (mRNA).
Gene map: Visual representation of the relative distances between genes or genetic markers on chromosomes.
Genome: The complete set of genes. All the DNA sequences of an organism. The human genome contains about
3 billion DNA base pairs.
Genomics: A field of study focused on genes and gene functions.
Genomewide association studies: A research method that involves rapidly scanning markers across the complete sets of DNA,
or genomes, of many people to find genetic variations associated with a particular phenotype (such as a disease or disorder).
Genotype: The genetic constitution of an individual.
Heritability: The proportion of phenotypic differences among individuals that can be attributed to genetic differences in a
particular population.
Mapping: Linkage of DNA markers to a chromosome and to specific regions of chromosomes.
Molecular genetics: Investigation of the effects of specific genes at the DNA level.
Nonadditive genetic variance: Individual differences due to the effects of alternate forms of genes at a particular locus
(i.e., dominance), or multiple genes at different loci (i.e., epistasis).
Nonshared environment: Environmental influences that contribute to differences between family members.
Phenotype: An observed characteristic or behavior of an individual that results from the combined effects of genotype
and environment.
Pleiotropy: The phenomenon where a single gene influences more than one phenotypic trait.
Polygenic trait: A trait influenced by many genes.
Shared environment: Environmental factors responsible for resemblance between family members.
Transcription: The synthesis of an RNA molecule from DNA in the cell nucleus.
Whole-genome sequencing: Determining the complete sequence of DNA base pairs for a genome.
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18 CHAPTER 2 Models of Child Development, Psychopathology, and Treatment
Gene-by-environment effects (or correlations) involve differential exposure to environments or experiences. There are
three types of gene-by-environment effects:
1. Passive correlations, in which children are exposed to different environments provided by their genetically related parents.
Examples: Extraverted children raised by extraverted parents are exposed to more social opportunities; highly intelligent
children raised by highly intelligent parents are exposed to more educational opportunities.
2. Active correlations, in which children select or create their own environments as a function of their genetic background.
Examples: Extraverted children seek out other children on the playground; highly intelligent children choose to read or
tinker with electronics during unstructured times.
3. Evocative correlations, in which children experience different reactions or responses to their genetically influenced
emotions or behaviors. Examples: Extraverted children who display rambunctious behavior may be disciplined more
frequently than introverted children; highly anxious children may be shielded from even mildly stressful situations by
parents and teachers, whereas less anxious children may be encouraged to deal with difficult situations.
provides a framework for understanding many sources notion that our ‘molecular selves’ are fixed across time
of genetic influence and allows researchers to estimate and situations, for example, is increasing evidence that
the heritability of many psychological characteristics. changes in the expression of literally hundreds of genes
One of the most important shifts in thinking about can occur as a function of the physical and social envi-
genetics involves moving beyond early views on nature ronments we inhabit. . . . The human genome, there-
versus nurture to current complex descriptions of gene- fore, is not a static blueprint for human potential.
by-environment effects and gene-by-environment Instead our genome appears to encode a wide variety
interactions (see Table 2.2). of ‘potential biological selves,’ and which ‘biological
Finally, we need to consider cutting-edge research self ’ gets realized depends on the social conditions we
in epigenetics, the study of how environmental fac- experience over the life course.”
tors influence gene expressivity. Epigenetics is focused What we know about psychopathology is also influ-
on the activity of the gene rather than the presence of enced by these physiological models (Cicchetti &
the gene (see Figure 2:5 and Box 2:1). To illustrate Walker, 2003). For some disorders, psychopathology
this distinction, leading researcher Moshe Szyf (2013) unfolds according to a “maturational blueprint,” with
analogizes the genome, deoxyribonucleic acid (DNA), deviance innately and inevitably related to damage or
as the script of a movie (or the score of a symphony), dysfunction (Sameroff, 2000). Certain severe forms
with epigenetics as the movie as it is actually filmed (or of intellectual developmental disorder are examples
the performance of that symphony). Epigenetics, like of this type of psychopathology (see Figure 2:6). For
synaptic pruning, is a way to think about how chil- most disorders of childhood and adolescence, how-
dren’s particular environments “get under their skin.” ever, this straightforward model of physical cause and
If we see synaptic pruning as identifying a mechanism psychopathological effect can be set aside. Genetically
by which the brain “listens” to the environment, then informed models of psychopathology must account
we can also see that epigenetics identifies a mechanism for the high heritability of many different kinds of
by which genes “listen” to the environment. In both disorder, as well as the findings related to genetic over-
cases, developmental experiences, especially in early lap. That is, rather than a correspondence between par-
life and during adolescence, and the social context in ticular genetic variants and one disorder, there appear
which they occur, have the capability to become bio- to be a more limited set of risk alleles that impair gen-
logically embedded with lifelong impacts on develop- eral processes (e.g., cognitive or emotion functions)
mental health. Slavich and Cole (2013, p. 331) provide across many disorders (Kiser, Rivero, & Lesch, 2015).
an even more compelling description: “Contrary to the Risk alleles include common variants, shared by
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Physiological Models 19
1 External Experiences
(e.g., stress, nutrition, toxins) 3 Gene Regulatory Proteins
spark signals between neurons attract or repel enzymes that
add or remove epigenetic markers
2 Neural Signals
launch production
of gene regulatory 4 Epigenetic “Markers” control where and
proteins inside cell how much protein is made by a gene, effectively
turning a gene “on” or “off,” thereby shaping
how brains and bodies develop
Gene—a specific
segment of a DNA strand
Neuron
(brain cell) DNA—strands encircle histones
that determine whether or not the
gene is “readable” by the cell
Chromosome—can pass
on genes to next generation
FIGURE 2:5 Epigenetic research provides new insights about gene-environment processes and interactions.
Source: From the http://developingchild.harvard.edu/science/deep-dives/gene-environment-interaction/ (the Gene-Environment Interaction tab
(under “Deep Dives”))
individuals with and without disorders; rare variants, imbalance does not by itself lead to disorder. Rather,
both inherited and de novo (newly appearing); and diatheses (or predispositions) such as neurological dam-
many different combinations of variants. Polygenic age at birth or genetic risk for disorder, in combination
models emphasize the likelihood that many genes have with additional stress (either physiological or environ-
small effects and attempt to account for the multiple mental), lead to the emergence of a disorder. Diathesis–
types of genetic variations and processes that result stress models call attention to the lack of a one-to-one
in genetic burdens that influence the development of correspondence between the genotype and phenotype
both mild and severe forms of disorders (Kiser et al., for most forms of psychopathology, and they are an
2015; Wray et al., 2014). Even with these exciting data, example of gene-by-environment effects or interactions.
we need to be cautious about overstating our hypoth- Two variations of the diathesis–stress model are illus-
eses and findings. The phrase “X is a gene for Y” is trated in the following cases. In the first case, a child
widely used, but it is inappropriate for psychology and with phenylketonuria (PKU) is born with a particu-
psychiatry. lar metabolic dysfunction, an inactive liver enzyme (a
Physiological models suggest that there are inborn or physiological diathesis of genetic origin). The presence
acquired vulnerabilities to disorders—including genetic of phenylalanine (a physiological stressor) in the child’s
abnormalities, structural pathologies, and biochemical diet and the subsequent metabolic abnormalities result
disturbances—that may lead to psychological dis- in severe intellectual dysfunction. Treatment of this
tress and dysfunction. According to this physiological condition involves a diet low in phenylalanine, begin-
diathesis–stress model, structural damage or chemical ning shortly after birth; this intervention is associated
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20 CHAPTER 2 Models of Child Development, Psychopathology, and Treatment
Box 2:1
Emerging Science
Behavior Genetics, Epigenetics, and Developmental
Psychopathology
R emarkable advances in scientific knowledge and tech-
nology have enabled investigators from many dis-
ciplines to ask, and begin to answer, questions about the
Indeed, epigenetics is the bridge between the fixed
genome and the dynamic world in which the genome ex-
ists. Epigenetic processes maintain fundamental genetic
biopsychosocial nature of human experience. Behavior stability on the one hand, while allowing for flexible
genetics is “the study of the relationship between ge- genetic expression in the face of a changing environment
netic variation and psychological traits,” including per- on the other. Thinking about developmental psycho-
sonality and psychopathology (Chabris, Lee, Cesarini, pathology, then, what looks like maladaptation may
Benjamin, & Laibson, 2015). Variant forms of specific actually be the best possible strategy in a challenging en-
genes are known as alleles (or polymorphisms), and geneti- vironment. For example, environmental stress is associ-
cists study whether these variations are associated with ated with early puberty. And although early puberty is
particular physical or psychological characteristics or with associated with a range of long-term negative conse-
increased risk for disorders. Combining data from decades quences, it is—at a biological level—an adaptive solution
of twin, family, and adoption studies with new data from to difficult circumstances.
the Human Genome Project (a collaborative effort by the The most frequently studied epigenetic mechanism is
U.S. Department of Energy and the National Institutes of DNA methylation (e.g., Szyf & Bick, 2013). “Levels of
Health (NIH) that sequenced the approximately 20,000 methylation are associated with how well DNA is
genes in human DNA, genomics.energy.gov), researchers transcribed. . . . As methylation levels increase, there is
have agreed upon a number of “big” findings. These big less transcription until the level of DNA methylation
findings include the following (Plomin, DeFries, Knopik, reaches the point at which the gene is switched off. . . . In
& Neiderhiser, 2016): the absence of DNA methylation, gene transcription is
allowed to occur. Although DNA methylation is often
●● All psychological traits show significant and substan-
described as an “on-off ” switch, it is, in fact, more like a
tial genetic influence.
“dimmer” switch that gradually decreases gene expres-
●● No traits are 100% heritable.
sion as methylation increases. In other words, if all the
●● Genetic impact is caused by many genes with small
cells associated with a particular gene are unmethylated,
effects.
the population of cells can produce the amount of pro-
●● Environments matter.
tein consistent with a fully active gene. Conversely, if the
In addition to ongoing work in behavior genetics, gene is fully methylated, it will produce very little or
complementary research on epigenetics holds enormous none of the protein. . . . The amount of methylation re-
promise for helping to explain the ways in which the envi- lated to behavior varies by gene. Some behaviors may be
ronment “gets under the skin” and facilitates or hinders affected by only slight changes in DNA methylation,
genetic expression (see the NIH Roadmap Epigenome while others may require a larger percent change” (Lester
Project; www.roadmapepigenomics.org). Underlying et al., 2016, pp. 30–31). With respect to methylation,
these research efforts is the understanding that human what is especially important to understand is that it is the
genetic variability is essential, so that individuals are able expression of the gene, rather than its presence that mat-
to adapt to a wide range of environmental challenges and ters (O’Connor, 2014).
opportunities. Epigenetics “refers to processes and mech- Epigenetics research may examine individual (or can-
anisms . . . that affect the activity of the DNA but do not didate) genes, small sets of genes, or much larger gene sets
change the DNA itself” (Lester, Conradt, & Marsit, (researched using genome-wide assays). Much of the work
2016, p. 29). DNA molecules contain genetic information in developmental psychopathology examines “how sig-
stored as codes. “Gene expression is the process by which nals from the environment (prenatal or postnatal) trigger
genes . . . make the specific proteins that determine the molecular changes” (Lester et al., 2016, p. 33), with a par-
structure and function of the individual gene. Gene ex- ticular focus on early adversity and stress (Keating, 2016;
pression is initiated by transcription factors. . . . Epigenetic Slavich & Cole, 2013; Szyf, 2013) and on disorders such
mechanisms regulate this transcriptional machinery, and as intellectual developmental disorder, autism spectrum
in so doing control gene expression. Thus, epigenetics con- disorder, and ADHD (O’Connor, 2014). Findings from
trols the activity of the gene or how the gene functions” these types of research studies are discussed in upcoming
(Lester et al., 2016, p. 30, emphasis added). chapters.
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Physiological Models 21
Bipolar disorder
Pediatric/developmental
diseases Severe phenotypes
(core)
Epilepsy
Observable if assessed
Autism Discovery of rare adequately
CNV associated Mutation carrier (no
Mental retardation
with disease observable phenotype)
Birth defects
Spectrum of variable expression
Core phenotypes differ for specific CNVs
Key
Schizophrenia
Epilepsy
Autism
Mental retardation
22q11.2 deletion 15q13.3 deletion 1q21.1 deletion Birth defects
FIGURE 2:6 Neurodevelopmental phenotypes associated with copy number variations (CNVs).
Source: https://www.researchgate.net/figure/51783053_fig1_Figure-1-Neuropsychiatric-phenotypes-associated-with-copy-number-variations-CNVs
with more typical intellectual development. In the sec- treatment of psychopathology. Examples of these atypi-
ond case, a child’s physical and psychological well-being cal processes will be provided in upcoming chapters.
may be adversely affected by maternal drug abuse dur-
ing pregnancy (again, a physiological diathesis, but this
one of nongenetic origin). After birth, poor parenting (a Thinking about Max
psychosocial stressor) of these health-compromised chil- From a physiological perspective, we emphasize the
dren may lead to a number of clinical disorders. High- role of brain structure and function and consider the
quality parenting, in contrast, may buffer or protect the likelihood of atypical biochemical processes in the
child from especially negative outcomes. Of course, we development of disorder in Max’s case. Specifically,
still must consider the possibility that brain structure physiologically-oriented clinicians conceptualize Max’s
or function is, in some clinically significant way, differ- difficulties as primarily due to underarousal of key
ent in children and adolescents with disorders. As noted parts of his brain; because of this, he lacks sufficient
previously, it appears that many forms of psychopa- focus and sustained engagement with the environment,
thology are associated with abnormal patterns of brain resulting in inattentive and impulsive behavior. These
network organization. Models of developmental brain difficulties, consistent with a diagnosis of attention-
network disorders have been described for schizophre- deficit hyperactivity disorder (ADHD), are not typical
nia and for autism spectrum disorder (Collin & van den of other similar-age children.
Heuvel, 2013; Vertes & Bullmore, 2015). With respect Because the disorder is physiologically based, the
to neural plasticity, gene-by-environment processes, and first choice for intervention is a physiological treatment.
brain–behavior relations in psychopathology, current The clinical literature suggests that stimulant drugs
physiological models of psychopathology seek to explain such as Ritalin are effective for children with ADHD,
the interplay of physical and biological factors, neuro- so a trial of stimulant medication would be prescribed.
logical processes, development, and life experiences in In addition, although pharmacological treatment is the
order to understand the emergence, the course, and the primary intervention, behaviorally influenced strategies
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Editorial review has deemed that any suppressed content does not materially affect the overall learning experience. Cengage Learning reserves the right to remove additional content at any time if subsequent rights restrictions require it.
22 CHAPTER 2 Models of Child Development, Psychopathology, and Treatment
would be routinely included in both the school and to work through developmental issues become “stuck”
home settings. in the past. Disorders themselves were rooted in trau-
mas or conflicts experienced during early childhood
(e.g., the oral, anal, and phallic stages). Psychoanalytic
Thinking about Anna interventions for children, such as those developed by
Again, from a physiological perspective, we note with Anna Freud and Melanie Klein, made special use of
special interest Anna’s family history, which includes play (using toys and games) and art to bring repressed
her father’s episodes of clinical depression, and consider traumas and unconscious conflicts into therapeutic
the possibility of a genetic vulnerability to depression. awareness.
Anna’s various problems, then, might be usefully Setting aside some of the more scientifically dubious
conceptualized as the psychological expression of a claims of early psychodynamic models, we are left with
biochemical imbalance. For example, Anna’s symptoms much to appreciate. Contemporary psychodynamic
may be a result of low levels of the neurotransmitter approaches continue to emphasize (1) unconscious cog-
serotonin, or dysregulation of multiple neurotransmitter nitive, affective, and motivational processes; (2) men-
systems. tal representations of self, other, and relationships;
Although treatment recommendations may include (3) the meaningfulness of individual (i.e., subjective)
suggestions that Anna participate in structured social experiences; and (4) a developmental perspective focused
activities in school as a way of helping her to be more on the origins of typical and atypical personality in
active and successful in friendships, the first step in her early childhood and the constantly changing psy-
intervention is the initiation of a trial of antidepres- chological challenges faced by children as they age
sant medication designed to correct the biochemical (Emde, 1992; Fonagy & Target, 2008; Westen, 1998).
imbalance. These emphases are evident in some of today’s most
significant psychodynamically informed research, such
as work on parent–child attachment and attachment’s
Psychodynamic Models enduring effects on personality and interpersonal func-
Historical and Current tioning (Cassidy, Jones, & Shaver, 2013).
Although recent psychodynamic models cer-
Conceptualizations tainly take into account recent advances in neuro-
Psychodynamic models have a rich past and a recently science (Protopopescu & Gerber, 2013), there is
revived future; they include the classic psychoanalytic still an emphasis on the importance of psychologi-
explanations set forth by Sigmund Freud, the socially cal contexts, such as relationships, when explaining
oriented explanations of Erik Erikson and Harry Stack the development of personality and psychopathology
Sullivan, the work of object-relations theorists such (Blatt & Luyten, 2009). With respect to treatments,
as Margaret Mahler and Donald Winnicott, and the today’s psychodynamic assessments and treatments
contemporary perspectives provided by Robert Emde, continue to rely on play to make connections with
Daniel Stern, and others. Psychodynamic models have troubled children, to identify the specific pathol-
historically focused on several themes, including (1) the ogy, and to effect change (Ray, Armstrong, Balkin,
impact of unconscious processes on typical and atypical & Jayne, 2015). Psychodynamic psychotherapy for
personality development; (2) conflicts among processes children and adolescents also emphasizes the impor-
and structures of the mind (e.g., id, ego, and superego); tant role of parents and family members (Palmer,
(3) the stages of development, with different ages associ- Nascimento, & Fonagy, 2013).
ated with distinctive emotional, intellectual, and social
challenges; and (4) the lasting impact of more (or less)
successful resolutions of stage-related challenges on later Thinking about Max
outcomes. Indeed, these themes were well appreciated From a psychodynamic perspective, we are concerned
by the novelist and astute observer of human nature, that the management of early developmental challenges
William Faulkner, who wrote (in 1950’s Requiem for a may have compromised Max’s current adjustment. For
Nun), “The past is never dead. It’s not even past.” example, do his inattentive and distractible activity
Psychodynamic theorists and clinicians usually and lack of school success reflect unconscious conflicts
emphasized a fixation–regression model of psychopa- about autonomy that he failed to resolve in a healthy
thology, which suggested that individuals who failed manner during his preschool years? Or has a somewhat
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Editorial review has deemed that any suppressed content does not materially affect the overall learning experience. Cengage Learning reserves the right to remove additional content at any time if subsequent rights restrictions require it.
Another random document with
no related content on Scribd:
— Bien, ma fille, dit Mme Voraud. Nous attendrons ton bon plaisir.
Nous irons voir Mme Stibel quand tu seras disposée à
m’accompagner.
Daniel se leva brusquement.
— Mademoiselle, dit-il à Berthe, vous m’excuserez de vous
quitter. Il faut que je rentre pour travailler.
— Pourquoi vous en allez-vous ? dit vivement Berthe. Ce n’est
pas vous qui nous empêchez de sortir.
— Je vous assure, répéta Daniel avec beaucoup de dignité, que
je suis obligé de rentrer chez moi.
— Si M. Daniel a des occupations… dit Mme Voraud. Pourquoi le
retiens-tu ? Tu es indiscrète.
— Au revoir, madame, dit Daniel en allant saluer Mme Voraud.
Mme Voraud répondit par un sourire aimable, qui semblait comme
rapporté sur son visage froid. Puis, elle baissa les yeux sur son
ouvrage. Berthe, à qui Daniel tendit la main, ne la prit pas. Louise
Loison sortit dans l’antichambre avec Daniel.
— Vous êtes fou de faire des scènes pareilles.
— Ça ne peut pas durer, répondit-il. Je ne veux pas qu’on me
fasse toujours des affronts. Je ne veux pas qu’on me tolère ici ; je
veux qu’on me reçoive. Je vais dire à papa, dès ce soir, qu’il vienne,
demain, voir M. Voraud, pour lui demander la main de Berthe. Si on
me la refuse, je verrai ce que j’aurai à faire.
— Attendez, dit Louise intéressée, je vais vous conduire jusqu’à
la porte du jardin.
Ils s’arrêtèrent ensemble devant la grille. Un petit ruisseau de
pluie courait le long du mur. Avec le bout de son parapluie, Daniel
faisait des petits trous dans le sable, entre les pierres ; ce qui
troublait l’eau d’amusants petits floconnements.
— Si j’ai hésité jusqu’ici, dit-il gravement à Louise, c’est que les
parents de Berthe me paraissent plus riches que les miens.
— Quelle fortune ont vos parents, sans indiscrétion ?
— Je ne l’ai jamais su, dit Daniel. Ils ne m’en ont jamais parlé. Un
jour, j’avais à peu près dix ans, papa est entré dans la chambre de
maman. Je savais qu’il était resté tard au magasin pour terminer son
inventaire. Il a dit à maman : C’est bien à peu près ce que je disais.
— Deux cent trente ? a dit maman. — Deux cent dix-sept, a dit papa.
— Maman a dit : Je croyais que c’était davantage… Depuis ce
temps, je n’ai plus rien su. Seulement, papa a dû faire de très
bonnes années. On a déménagé. Le magasin s’est agrandi. On a
deux voyageurs en plus. Mes parents auraient maintenant plus de
cinq cent mille francs que ça ne m’étonnerait pas… Mais qu’est-ce
que c’est que cinq cent mille francs auprès de la fortune de M.
Voraud ?
— Combien a-t-il, M. Voraud ? demanda Louise Loison.
— Trois millions, m’a-t-on dit.
— Papa dit beaucoup moins que ça, dit Louise. Papa m’a dit qu’il
devait avoir de douze à quinze cent mille francs, et que ce n’était
pas une fortune très sûre. Il y a des jours où M. Voraud a l’air
ennuyé. En tout cas, je sais ce qu’il donne à sa fille : quinze mille
francs de rente et le logement.
— Est-ce que c’est beaucoup ? dit Daniel.
— Il me semble, dit Louise. J’ai cent mille francs de dot, et tout le
monde dit que c’est très joli. Or, quinze mille francs de rente, c’est
certainement beaucoup plus… Mais vous n’avez pas besoin d’être
gêné parce que Berthe est plus riche que vous. Elle sait bien que
vous l’épouserez par amour.
— Oh ! ce n’est pas ça qui me gêne, dit Daniel, d’autant plus que
je suis bien sûr qu’un jour je serai très riche, et que je gagnerai
beaucoup d’argent. Mais c’est pour ses parents : est-ce qu’ils
voudront de moi ?
— Faites toujours faire la demande par votre père. C’est le seul
moyen de le savoir.
— Dites à Berthe, dit Daniel, qu’elle ne m’en veuille pas de ce qui
s’est passé aujourd’hui. Dites-lui que je ne l’ai jamais tant aimée.
XVIII
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